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Erratum to Targeting NOX4 alleviates sepsis-induced acute lung injury via attenuation of redox-sensitive activation of CaMKII/ERK1/2/MLCK and endothelial cell barrier dysfunction, Redox Biology 36 (2020) 101638.

Redox biology

Jiang J, Huang K, Xu S, Garcia JGN, Wang C, Cai H.
PMID: 34844898
Redox Biol. 2021 Dec;48:102200. doi: 10.1016/j.redox.2021.102200. Epub 2021 Nov 27.

No abstract available.

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Jiang J, Huang K, Xu S, et al. Erratum to Targeting NOX4 alleviates sepsis-induced acute lung injury via attenuation of redox-sensitive activation of CaMKII/ERK1/2/MLCK and endothelial cell barrier dysfunction, Redox Biology 36 (2020) 101638. Redox Biol. 2021;48:102200doi: 10.1016/j.redox.2021.102200.
Jiang, J., Huang, K., Xu, S., Garcia, J. G. N., Wang, C., & Cai, H. (2021). Erratum to Targeting NOX4 alleviates sepsis-induced acute lung injury via attenuation of redox-sensitive activation of CaMKII/ERK1/2/MLCK and endothelial cell barrier dysfunction, Redox Biology 36 (2020) 101638. Redox biology, 48102200. https://doi.org/10.1016/j.redox.2021.102200
Jiang, Jinyao, et al. "Erratum to Targeting NOX4 alleviates sepsis-induced acute lung injury via attenuation of redox-sensitive activation of CaMKII/ERK1/2/MLCK and endothelial cell barrier dysfunction, Redox Biology 36 (2020) 101638." Redox biology vol. 48 (2021): 102200. doi: https://doi.org/10.1016/j.redox.2021.102200
Jiang J, Huang K, Xu S, Garcia JGN, Wang C, Cai H. Erratum to Targeting NOX4 alleviates sepsis-induced acute lung injury via attenuation of redox-sensitive activation of CaMKII/ERK1/2/MLCK and endothelial cell barrier dysfunction, Redox Biology 36 (2020) 101638. Redox Biol. 2021 Dec;48:102200. doi: 10.1016/j.redox.2021.102200. Epub 2021 Nov 27. PMID: 34844898.
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Upregulation of miR-499a-5p Decreases Cerebral Ischemia/Reperfusion Injury by Targeting PDCD4.

Cellular and molecular neurobiology

Shan W, Ge H, Chen B, Huang L, Zhu S, Zhou Y.
PMID: 33837492
Cell Mol Neurobiol. 2021 Apr 09; doi: 10.1007/s10571-021-01085-4. Epub 2021 Apr 09.

MiR-499a-5p was significantly downregulated in degenerative tissues and correlated with apoptosis. Nonetheless, the biological function of miR-499a-5p in acute ischemic stroke has been still unclear. In this study, we found that the plasma levels of miR-499a-5p were significantly downregulated...

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Shan W, Ge H, Chen B, et al. Upregulation of miR-499a-5p Decreases Cerebral Ischemia/Reperfusion Injury by Targeting PDCD4. Cell Mol Neurobiol. 2021;doi: 10.1007/s10571-021-01085-4.
Shan, W., Ge, H., Chen, B., Huang, L., Zhu, S., & Zhou, Y. (2021). Upregulation of miR-499a-5p Decreases Cerebral Ischemia/Reperfusion Injury by Targeting PDCD4. Cellular and molecular neurobiology, . https://doi.org/10.1007/s10571-021-01085-4
Shan, Weifeng, et al. "Upregulation of miR-499a-5p Decreases Cerebral Ischemia/Reperfusion Injury by Targeting PDCD4." Cellular and molecular neurobiology vol. (2021). doi: https://doi.org/10.1007/s10571-021-01085-4
Shan W, Ge H, Chen B, Huang L, Zhu S, Zhou Y. Upregulation of miR-499a-5p Decreases Cerebral Ischemia/Reperfusion Injury by Targeting PDCD4. Cell Mol Neurobiol. 2021 Apr 09; doi: 10.1007/s10571-021-01085-4. Epub 2021 Apr 09. PMID: 33837492.
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Case Report: Cetuximab in Combination With Chemotherapy for the Treatment of Multifocal Hepatic Metastases From Colorectal Cancer Guided by Genetic Tests.

Frontiers in oncology

Qiu C, Xie S, Cheng N, Lin Q, Shen G, Xiang Z, Huang T, Zhang X, Duan J, Wei L, Zheng Z.
PMID: 33889542
Front Oncol. 2021 Apr 06;11:612171. doi: 10.3389/fonc.2021.612171. eCollection 2021.

Hepatic metastases were reported in up to 70% of colorectal cancer patients, among which multifocal hepatic metastasis represents one of the complications that lead to poor prognosis. The majority of the patients carrying multifocal hepatic metastases required pharmaceutical treatments...

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Qiu C, Xie S, Cheng N, et al. Case Report: Cetuximab in Combination With Chemotherapy for the Treatment of Multifocal Hepatic Metastases From Colorectal Cancer Guided by Genetic Tests. Front Oncol. 2021;11:612171doi: 10.3389/fonc.2021.612171.
Qiu, C., Xie, S., Cheng, N., Lin, Q., Shen, G., Xiang, Z., Huang, T., Zhang, X., Duan, J., Wei, L., & Zheng, Z. (2021). Case Report: Cetuximab in Combination With Chemotherapy for the Treatment of Multifocal Hepatic Metastases From Colorectal Cancer Guided by Genetic Tests. Frontiers in oncology, 11612171. https://doi.org/10.3389/fonc.2021.612171
Qiu, Chunhui, et al. "Case Report: Cetuximab in Combination With Chemotherapy for the Treatment of Multifocal Hepatic Metastases From Colorectal Cancer Guided by Genetic Tests." Frontiers in oncology vol. 11 (2021): 612171. doi: https://doi.org/10.3389/fonc.2021.612171
Qiu C, Xie S, Cheng N, Lin Q, Shen G, Xiang Z, Huang T, Zhang X, Duan J, Wei L, Zheng Z. Case Report: Cetuximab in Combination With Chemotherapy for the Treatment of Multifocal Hepatic Metastases From Colorectal Cancer Guided by Genetic Tests. Front Oncol. 2021 Apr 06;11:612171. doi: 10.3389/fonc.2021.612171. eCollection 2021. PMID: 33889542; PMCID: PMC8056263.
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Current status of traditional Chinese medicine for the treatment of COVID-19 in China.

Chinese medicine

Chu L, Huang F, Zhang M, Huang B, Wang Y.
PMID: 34315521
Chin Med. 2021 Jul 27;16(1):63. doi: 10.1186/s13020-021-00461-y.

An ongoing outbreak of severe respiratory illness and pneumonia caused by the severe acute respiratory coronavirus 2 (SARS-CoV-2) commenced in December 2019, and the disease was named as coronavirus disease 2019 (COVID-19). Soon after, scientists identified the characteristics of...

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Chu L, Huang F, Zhang M, et al. Current status of traditional Chinese medicine for the treatment of COVID-19 in China. Chin Med. 2021;16(1):63doi: 10.1186/s13020-021-00461-y.
Chu, L., Huang, F., Zhang, M., Huang, B., & Wang, Y. (2021). Current status of traditional Chinese medicine for the treatment of COVID-19 in China. Chinese medicine, 16(1), 63. https://doi.org/10.1186/s13020-021-00461-y
Chu, Liang, et al. "Current status of traditional Chinese medicine for the treatment of COVID-19 in China." Chinese medicine vol. 16,1 (2021): 63. doi: https://doi.org/10.1186/s13020-021-00461-y
Chu L, Huang F, Zhang M, Huang B, Wang Y. Current status of traditional Chinese medicine for the treatment of COVID-19 in China. Chin Med. 2021 Jul 27;16(1):63. doi: 10.1186/s13020-021-00461-y. PMID: 34315521; PMCID: PMC8314260.
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Nrf2 contributes to the benefits of exercise interventions on age-related skeletal muscle disorder via regulating Drp1 stability and mitochondrial fission.

Free radical biology & medicine

Yan X, Shen Z, Yu D, Zhao C, Zou H, Ma B, Dong W, Chen W, Huang D, Yu Z.
PMID: 34823019
Free Radic Biol Med. 2021 Nov 23;178:59-75. doi: 10.1016/j.freeradbiomed.2021.11.030. Epub 2021 Nov 23.

The progressive and generalized loss of skeletal muscle mass and function, also known as sarcopenia, underlies disability, increasing adverse outcomes and poor quality of life in older people. Exercise interventions are commonly recommended as the primary treatment for sarcopenia....

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Yan X, Shen Z, Yu D, et al. Nrf2 contributes to the benefits of exercise interventions on age-related skeletal muscle disorder via regulating Drp1 stability and mitochondrial fission. Free Radic Biol Med. 2021;178:59-75doi: 10.1016/j.freeradbiomed.2021.11.030.
Yan, X., Shen, Z., Yu, D., Zhao, C., Zou, H., Ma, B., Dong, W., Chen, W., Huang, D., & Yu, Z. (2021). Nrf2 contributes to the benefits of exercise interventions on age-related skeletal muscle disorder via regulating Drp1 stability and mitochondrial fission. Free radical biology & medicine, 17859-75. https://doi.org/10.1016/j.freeradbiomed.2021.11.030
Yan, Xialin, et al. "Nrf2 contributes to the benefits of exercise interventions on age-related skeletal muscle disorder via regulating Drp1 stability and mitochondrial fission." Free radical biology & medicine vol. 178 (2021): 59-75. doi: https://doi.org/10.1016/j.freeradbiomed.2021.11.030
Yan X, Shen Z, Yu D, Zhao C, Zou H, Ma B, Dong W, Chen W, Huang D, Yu Z. Nrf2 contributes to the benefits of exercise interventions on age-related skeletal muscle disorder via regulating Drp1 stability and mitochondrial fission. Free Radic Biol Med. 2021 Nov 23;178:59-75. doi: 10.1016/j.freeradbiomed.2021.11.030. Epub 2021 Nov 23. PMID: 34823019.
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GP73 is a TBC-domain Rab GTPase-activating protein contributing to the pathogenesis of non-alcoholic fatty liver disease without obesity.

Nature communications

Peng Y, Zeng Q, Wan L, Ma E, Li H, Yang X, Zhang Y, Huang L, Lin H, Feng J, Xu Y, Li J, Liu M, Liu J, Lin C, Sun Z, Cheng G, Zhang X, Liu J, Li D, Wei M, Mo Y, Mu X, Deng X, Zhang D, Dong S, Huang H, Fang Y, Gao Q, Yang X, Wu F, Zhong H, Wei C.
PMID: 34853313
Nat Commun. 2021 Dec 01;12(1):7004. doi: 10.1038/s41467-021-27309-1.

The prevalence of non-obese nonalcoholic fatty liver disease (NAFLD) is increasing worldwide with unclear etiology and pathogenesis. Here, we show GP73, a Golgi protein upregulated in livers from patients with a variety of liver diseases, exhibits Rab GTPase-activating protein...

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Peng Y, Zeng Q, Wan L, et al. GP73 is a TBC-domain Rab GTPase-activating protein contributing to the pathogenesis of non-alcoholic fatty liver disease without obesity. Nat Commun. 2021;12(1):7004doi: 10.1038/s41467-021-27309-1.
Peng, Y., Zeng, Q., Wan, L., Ma, E., Li, H., Yang, X., Zhang, Y., Huang, L., Lin, H., Feng, J., Xu, Y., Li, J., Liu, M., Liu, J., Lin, C., Sun, Z., Cheng, G., Zhang, X., Liu, J., Li, D., Wei, M., Mo, Y., Mu, X., Deng, X., Zhang, D., Dong, S., Huang, H., Fang, Y., Gao, Q., Yang, X., Wu, F., Zhong, H., & Wei, C. (2021). GP73 is a TBC-domain Rab GTPase-activating protein contributing to the pathogenesis of non-alcoholic fatty liver disease without obesity. Nature communications, 12(1), 7004. https://doi.org/10.1038/s41467-021-27309-1
Peng, Yumeng, et al. "GP73 is a TBC-domain Rab GTPase-activating protein contributing to the pathogenesis of non-alcoholic fatty liver disease without obesity." Nature communications vol. 12,1 (2021): 7004. doi: https://doi.org/10.1038/s41467-021-27309-1
Peng Y, Zeng Q, Wan L, Ma E, Li H, Yang X, Zhang Y, Huang L, Lin H, Feng J, Xu Y, Li J, Liu M, Liu J, Lin C, Sun Z, Cheng G, Zhang X, Liu J, Li D, Wei M, Mo Y, Mu X, Deng X, Zhang D, Dong S, Huang H, Fang Y, Gao Q, Yang X, Wu F, Zhong H, Wei C. GP73 is a TBC-domain Rab GTPase-activating protein contributing to the pathogenesis of non-alcoholic fatty liver disease without obesity. Nat Commun. 2021 Dec 01;12(1):7004. doi: 10.1038/s41467-021-27309-1. PMID: 34853313; PMCID: PMC8636488.
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Degree of paraxiality of an electromagnetic fractional multi-Gaussian Schell-model beam.

Journal of the Optical Society of America. A, Optics, image science, and vision

Huang J, Xiang S, Jiang W, Ji X, Wang T.
PMID: 34613132
J Opt Soc Am A Opt Image Sci Vis. 2021 Sep 01;38(9):1264-1269. doi: 10.1364/JOSAA.431772.

The degree of paraxiality (DOP) of an electromagnetic fractional multi-Gaussian Schell-model (EM-FMGSM) beam is discussed, and the effect of the properties of the light source on its DOP is also studied. It is shown from the numerical results that...

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Huang J, Xiang S, Jiang W, et al. Degree of paraxiality of an electromagnetic fractional multi-Gaussian Schell-model beam. J Opt Soc Am A Opt Image Sci Vis. 2021;38(9):1264-1269doi: 10.1364/JOSAA.431772.
Huang, J., Xiang, S., Jiang, W., Ji, X., & Wang, T. (2021). Degree of paraxiality of an electromagnetic fractional multi-Gaussian Schell-model beam. Journal of the Optical Society of America. A, Optics, image science, and vision, 38(9), 1264-1269. https://doi.org/10.1364/JOSAA.431772
Huang, Ju, et al. "Degree of paraxiality of an electromagnetic fractional multi-Gaussian Schell-model beam." Journal of the Optical Society of America. A, Optics, image science, and vision vol. 38,9 (2021): 1264-1269. doi: https://doi.org/10.1364/JOSAA.431772
Huang J, Xiang S, Jiang W, Ji X, Wang T. Degree of paraxiality of an electromagnetic fractional multi-Gaussian Schell-model beam. J Opt Soc Am A Opt Image Sci Vis. 2021 Sep 01;38(9):1264-1269. doi: 10.1364/JOSAA.431772. PMID: 34613132.
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Homology-based repair induced by CRISPR-Cas nucleases in mammalian embryo genome editing.

Protein & cell

Zhang X, Li T, Ou J, Huang J, Liang P.
PMID: 33945139
Protein Cell. 2021 May 04; doi: 10.1007/s13238-021-00838-7. Epub 2021 May 04.

Recent advances in genome editing, especially CRISPR-Cas nucleases, have revolutionized both laboratory research and clinical therapeutics. CRISPR-Cas nucleases, together with the DNA damage repair pathway in cells, enable both genetic diversification by classical non-homologous end joining (c-NHEJ) and precise...

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Zhang X, Li T, Ou J, et al. Homology-based repair induced by CRISPR-Cas nucleases in mammalian embryo genome editing. Protein Cell. 2021;doi: 10.1007/s13238-021-00838-7.
Zhang, X., Li, T., Ou, J., Huang, J., & Liang, P. (2021). Homology-based repair induced by CRISPR-Cas nucleases in mammalian embryo genome editing. Protein & cell, . https://doi.org/10.1007/s13238-021-00838-7
Zhang, Xiya, et al. "Homology-based repair induced by CRISPR-Cas nucleases in mammalian embryo genome editing." Protein & cell vol. (2021). doi: https://doi.org/10.1007/s13238-021-00838-7
Zhang X, Li T, Ou J, Huang J, Liang P. Homology-based repair induced by CRISPR-Cas nucleases in mammalian embryo genome editing. Protein Cell. 2021 May 04; doi: 10.1007/s13238-021-00838-7. Epub 2021 May 04. PMID: 33945139.
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Development of an In-House Rapid Antimicrobial Susceptibility Testing Protocol for Positive Blood Culture and Its Implementation in Routine Microbiology Laboratories.

Frontiers in microbiology

Cao M, Huang L, Hu Y, Fang Y, Zhang R, Chen G.
PMID: 34917051
Front Microbiol. 2021 Nov 30;12:765757. doi: 10.3389/fmicb.2021.765757. eCollection 2021.

Bloodstream infections (BSI) are associated with high morbidity and mortality and remain a leading cause of death. Blood culture (BC) including the identification and the antimicrobial susceptibility testing of the causative microorganisms should be performed as soon as possible....

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Cao M, Huang L, Hu Y, et al. Development of an In-House Rapid Antimicrobial Susceptibility Testing Protocol for Positive Blood Culture and Its Implementation in Routine Microbiology Laboratories. Front Microbiol. 2021;12:765757doi: 10.3389/fmicb.2021.765757.
Cao, M., Huang, L., Hu, Y., Fang, Y., Zhang, R., & Chen, G. (2021). Development of an In-House Rapid Antimicrobial Susceptibility Testing Protocol for Positive Blood Culture and Its Implementation in Routine Microbiology Laboratories. Frontiers in microbiology, 12765757. https://doi.org/10.3389/fmicb.2021.765757
Cao, Min, et al. "Development of an In-House Rapid Antimicrobial Susceptibility Testing Protocol for Positive Blood Culture and Its Implementation in Routine Microbiology Laboratories." Frontiers in microbiology vol. 12 (2021): 765757. doi: https://doi.org/10.3389/fmicb.2021.765757
Cao M, Huang L, Hu Y, Fang Y, Zhang R, Chen G. Development of an In-House Rapid Antimicrobial Susceptibility Testing Protocol for Positive Blood Culture and Its Implementation in Routine Microbiology Laboratories. Front Microbiol. 2021 Nov 30;12:765757. doi: 10.3389/fmicb.2021.765757. eCollection 2021. PMID: 34917051; PMCID: PMC8669140.
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Correction to: YM155 decreases radiation-induced invasion and reverses epithelial-mesenchymal transition by targeting STAT3 in glioblastoma.

Journal of translational medicine

Zhang X, Wang X, Xu R, Ji J, Xu Y, Han M, Wei Y, Huang B, Chen A, Zhang Q, Li W, Wang J, Li X, Qiu C.
PMID: 34579738
J Transl Med. 2021 Sep 27;19(1):407. doi: 10.1186/s12967-021-03017-8.

No abstract available.

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Zhang X, Wang X, Xu R, et al. Correction to: YM155 decreases radiation-induced invasion and reverses epithelial-mesenchymal transition by targeting STAT3 in glioblastoma. J Transl Med. 2021;19(1):407doi: 10.1186/s12967-021-03017-8.
Zhang, X., Wang, X., Xu, R., Ji, J., Xu, Y., Han, M., Wei, Y., Huang, B., Chen, A., Zhang, Q., Li, W., Wang, J., Li, X., & Qiu, C. (2021). Correction to: YM155 decreases radiation-induced invasion and reverses epithelial-mesenchymal transition by targeting STAT3 in glioblastoma. Journal of translational medicine, 19(1), 407. https://doi.org/10.1186/s12967-021-03017-8
Zhang, Xin, et al. "Correction to: YM155 decreases radiation-induced invasion and reverses epithelial-mesenchymal transition by targeting STAT3 in glioblastoma." Journal of translational medicine vol. 19,1 (2021): 407. doi: https://doi.org/10.1186/s12967-021-03017-8
Zhang X, Wang X, Xu R, Ji J, Xu Y, Han M, Wei Y, Huang B, Chen A, Zhang Q, Li W, Wang J, Li X, Qiu C. Correction to: YM155 decreases radiation-induced invasion and reverses epithelial-mesenchymal transition by targeting STAT3 in glioblastoma. J Transl Med. 2021 Sep 27;19(1):407. doi: 10.1186/s12967-021-03017-8. PMID: 34579738; PMCID: PMC8474712.
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Have COVID-19-Related Economic Shocks Affected the Health Levels of Individuals in the United States and the United Kingdom?.

Frontiers in public health

Jiang B, Liu Z, Shen R, Huang L, Tong Y, Xia Y.
PMID: 33363099
Front Public Health. 2020 Dec 09;8:611325. doi: 10.3389/fpubh.2020.611325. eCollection 2020.

This paper introduces a health index for measuring the health level of societies during the lockdown era, i. e., for the period from March 21, 2020 to April 7, 2020. For this purpose, individual-level survey data from the Global...

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Jiang B, Liu Z, Shen R, et al. Have COVID-19-Related Economic Shocks Affected the Health Levels of Individuals in the United States and the United Kingdom?. Front Public Health. 2020;8:611325doi: 10.3389/fpubh.2020.611325.
Jiang, B., Liu, Z., Shen, R., Huang, L., Tong, Y., & Xia, Y. (2020). Have COVID-19-Related Economic Shocks Affected the Health Levels of Individuals in the United States and the United Kingdom?. Frontiers in public health, 8611325. https://doi.org/10.3389/fpubh.2020.611325
Jiang, Baozhen, et al. "Have COVID-19-Related Economic Shocks Affected the Health Levels of Individuals in the United States and the United Kingdom?." Frontiers in public health vol. 8 (2020): 611325. doi: https://doi.org/10.3389/fpubh.2020.611325
Jiang B, Liu Z, Shen R, Huang L, Tong Y, Xia Y. Have COVID-19-Related Economic Shocks Affected the Health Levels of Individuals in the United States and the United Kingdom?. Front Public Health. 2020 Dec 09;8:611325. doi: 10.3389/fpubh.2020.611325. eCollection 2020. PMID: 33363099; PMCID: PMC7755999.
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Clinicopathological features of titinopathy from a Chinese neuromuscular center.

Neuropathology : official journal of the Japanese Society of Neuropathology

Huang K, Duan HQ, Li QX, Luo YB, Bi FF, Yang H.
PMID: 34553419
Neuropathology. 2021 Oct;41(5):349-356. doi: 10.1111/neup.12761. Epub 2021 Sep 22.

Titin, one of the largest proteins in humans, is a major component of muscle sarcomeres. Pathogenic variants in the titin gene (TTN) have been reported to cause a range of skeletal muscle diseases, collectively known as titinopathy. Titinopathy is...

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Huang K, Duan HQ, Li QX, et al. Clinicopathological features of titinopathy from a Chinese neuromuscular center. Neuropathology. 2021;41(5):349-356doi: 10.1111/neup.12761.
Huang, K., Duan, H. Q., Li, Q. X., Luo, Y. B., Bi, F. F., & Yang, H. (2021). Clinicopathological features of titinopathy from a Chinese neuromuscular center. Neuropathology : official journal of the Japanese Society of Neuropathology, 41(5), 349-356. https://doi.org/10.1111/neup.12761
Huang, Kun, et al. "Clinicopathological features of titinopathy from a Chinese neuromuscular center." Neuropathology : official journal of the Japanese Society of Neuropathology vol. 41,5 (2021): 349-356. doi: https://doi.org/10.1111/neup.12761
Huang K, Duan HQ, Li QX, Luo YB, Bi FF, Yang H. Clinicopathological features of titinopathy from a Chinese neuromuscular center. Neuropathology. 2021 Oct;41(5):349-356. doi: 10.1111/neup.12761. Epub 2021 Sep 22. PMID: 34553419.
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Showing 9985 to 9996 of 51057 entries