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Journal der Deutschen Dermatologischen Gesellschaft = Journal of the German Society of Dermatology : JDDG

Gebauer K, Schirren J, Jaeschke B, Kaufmann R, Meissner M.
PMID: 31985157
J Dtsch Dermatol Ges. 2020 Jan;18(1):47-49. doi: 10.1111/ddg.13983_g.

No abstract available.

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Gebauer K, Schirren J, Jaeschke B, et al. Neoadjuvante Therapie mit Vemurafenib bei Horner-Syndrom als sehr seltene Erstdiagnose eines CUP-Syndroms bei malignem Melanom. . J Dtsch Dermatol Ges. 2020;18(1):47-49doi: 10.1111/ddg.13983_g.
Gebauer, K., Schirren, J., Jaeschke, B., Kaufmann, R., & Meissner, M. (2020). Neoadjuvante Therapie mit Vemurafenib bei Horner-Syndrom als sehr seltene Erstdiagnose eines CUP-Syndroms bei malignem Melanom. . Journal der Deutschen Dermatologischen Gesellschaft = Journal of the German Society of Dermatology : JDDG, 18(1), 47-49. https://doi.org/10.1111/ddg.13983_g
Gebauer, Konstanze, et al. "Neoadjuvante Therapie mit Vemurafenib bei Horner-Syndrom als sehr seltene Erstdiagnose eines CUP-Syndroms bei malignem Melanom." . Journal der Deutschen Dermatologischen Gesellschaft = Journal of the German Society of Dermatology : JDDG vol. 18,1 (2020): 47-49. doi: https://doi.org/10.1111/ddg.13983_g
Gebauer K, Schirren J, Jaeschke B, Kaufmann R, Meissner M. Neoadjuvante Therapie mit Vemurafenib bei Horner-Syndrom als sehr seltene Erstdiagnose eines CUP-Syndroms bei malignem Melanom. . J Dtsch Dermatol Ges. 2020 Jan;18(1):47-49. doi: 10.1111/ddg.13983_g. German. PMID: 31985157.
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Two-Dome Superconductivity in FeS Induced by a Lifshitz Transition.

Physical review letters

Shimizu M, Takemori N, Guterding D, Jeschke HO.
PMID: 30312064
Phys Rev Lett. 2018 Sep 28;121(13):137001. doi: 10.1103/PhysRevLett.121.137001.

Among iron chalcogenide superconductors, FeS can be viewed as a simple, highly compressed relative of FeSe without a nematic phase and with weaker electronic correlations. Under pressure, however, the superconductivity of stoichiometric FeS disappears and reappears, forming two domes....

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Shimizu M, Takemori N, Guterding D, et al. Two-Dome Superconductivity in FeS Induced by a Lifshitz Transition. Phys Rev Lett. 2018;121(13):137001doi: 10.1103/PhysRevLett.121.137001.
Shimizu, M., Takemori, N., Guterding, D., & Jeschke, H. O. (2018). Two-Dome Superconductivity in FeS Induced by a Lifshitz Transition. Physical review letters, 121(13), 137001. https://doi.org/10.1103/PhysRevLett.121.137001
Shimizu, Makoto, et al. "Two-Dome Superconductivity in FeS Induced by a Lifshitz Transition." Physical review letters vol. 121,13 (2018): 137001. doi: https://doi.org/10.1103/PhysRevLett.121.137001
Shimizu M, Takemori N, Guterding D, Jeschke HO. Two-Dome Superconductivity in FeS Induced by a Lifshitz Transition. Phys Rev Lett. 2018 Sep 28;121(13):137001. doi: 10.1103/PhysRevLett.121.137001. PMID: 30312064.
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Dual roles of p62/SQSTM1 in the injury and recovery phases of acetaminophen-induced liver injury in mice.

Acta pharmaceutica Sinica. B

Qian H, Bai Q, Yang X, Akakpo JY, Ji L, Yang L, Rülicke T, Zatloukal K, Jaeschke H, Ni HM, Ding WX.
PMID: 35024307
Acta Pharm Sin B. 2021 Dec;11(12):3791-3805. doi: 10.1016/j.apsb.2021.11.010. Epub 2021 Nov 16.

Acetaminophen (APAP) overdose can induce liver injury and is the most frequent cause of acute liver failure in the United States. We investigated the role of p62/SQSTM1 (referred to as p62) in APAP-induced liver injury (AILI) in mice. We...

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Qian H, Bai Q, Yang X, et al. Dual roles of p62/SQSTM1 in the injury and recovery phases of acetaminophen-induced liver injury in mice. Acta Pharm Sin B. 2021;11(12):3791-3805doi: 10.1016/j.apsb.2021.11.010.
Qian, H., Bai, Q., Yang, X., Akakpo, J. Y., Ji, L., Yang, L., Rülicke, T., Zatloukal, K., Jaeschke, H., Ni, H. M., & Ding, W. X. (2021). Dual roles of p62/SQSTM1 in the injury and recovery phases of acetaminophen-induced liver injury in mice. Acta pharmaceutica Sinica. B, 11(12), 3791-3805. https://doi.org/10.1016/j.apsb.2021.11.010
Qian, Hui, et al. "Dual roles of p62/SQSTM1 in the injury and recovery phases of acetaminophen-induced liver injury in mice." Acta pharmaceutica Sinica. B vol. 11,12 (2021): 3791-3805. doi: https://doi.org/10.1016/j.apsb.2021.11.010
Qian H, Bai Q, Yang X, Akakpo JY, Ji L, Yang L, Rülicke T, Zatloukal K, Jaeschke H, Ni HM, Ding WX. Dual roles of p62/SQSTM1 in the injury and recovery phases of acetaminophen-induced liver injury in mice. Acta Pharm Sin B. 2021 Dec;11(12):3791-3805. doi: 10.1016/j.apsb.2021.11.010. Epub 2021 Nov 16. PMID: 35024307; PMCID: PMC8727897.
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Comparing N-acetylcysteine and 4-methylpyrazole as antidotes for acetaminophen overdose.

Archives of toxicology

Akakpo JY, Ramachandran A, Curry SC, Rumack BH, Jaeschke H.
PMID: 34978586
Arch Toxicol. 2022 Jan 03; doi: 10.1007/s00204-021-03211-z. Epub 2022 Jan 03.

Acetaminophen (APAP) overdose can cause hepatotoxicity and even liver failure. N-acetylcysteine (NAC) is still the only FDA-approved antidote against APAP overdose 40 years after its introduction. The standard oral or intravenous dosing regimen of NAC is highly effective for...

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Akakpo JY, Ramachandran A, Curry SC, et al. Comparing N-acetylcysteine and 4-methylpyrazole as antidotes for acetaminophen overdose. Arch Toxicol. 2022;doi: 10.1007/s00204-021-03211-z.
Akakpo, J. Y., Ramachandran, A., Curry, S. C., Rumack, B. H., & Jaeschke, H. (2022). Comparing N-acetylcysteine and 4-methylpyrazole as antidotes for acetaminophen overdose. Archives of toxicology, . https://doi.org/10.1007/s00204-021-03211-z
Akakpo, Jephte Y, et al. "Comparing N-acetylcysteine and 4-methylpyrazole as antidotes for acetaminophen overdose." Archives of toxicology vol. (2022). doi: https://doi.org/10.1007/s00204-021-03211-z
Akakpo JY, Ramachandran A, Curry SC, Rumack BH, Jaeschke H. Comparing N-acetylcysteine and 4-methylpyrazole as antidotes for acetaminophen overdose. Arch Toxicol. 2022 Jan 03; doi: 10.1007/s00204-021-03211-z. Epub 2022 Jan 03. PMID: 34978586.
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Classification of Microglial Morphological Phenotypes Using Machine Learning.

Frontiers in cellular neuroscience

Leyh J, Paeschke S, Mages B, Michalski D, Nowicki M, Bechmann I, Winter K.
PMID: 34267628
Front Cell Neurosci. 2021 Jun 29;15:701673. doi: 10.3389/fncel.2021.701673. eCollection 2021.

Microglia are the brain's immunocompetent macrophages with a unique feature that allows surveillance of the surrounding microenvironment and subsequent reactions to tissue damage, infection, or homeostatic perturbations. Thereby, microglia's striking morphological plasticity is one of their prominent characteristics and...

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Leyh J, Paeschke S, Mages B, et al. Classification of Microglial Morphological Phenotypes Using Machine Learning. Front Cell Neurosci. 2021;15:701673doi: 10.3389/fncel.2021.701673.
Leyh, J., Paeschke, S., Mages, B., Michalski, D., Nowicki, M., Bechmann, I., & Winter, K. (2021). Classification of Microglial Morphological Phenotypes Using Machine Learning. Frontiers in cellular neuroscience, 15701673. https://doi.org/10.3389/fncel.2021.701673
Leyh, Judith, et al. "Classification of Microglial Morphological Phenotypes Using Machine Learning." Frontiers in cellular neuroscience vol. 15 (2021): 701673. doi: https://doi.org/10.3389/fncel.2021.701673
Leyh J, Paeschke S, Mages B, Michalski D, Nowicki M, Bechmann I, Winter K. Classification of Microglial Morphological Phenotypes Using Machine Learning. Front Cell Neurosci. 2021 Jun 29;15:701673. doi: 10.3389/fncel.2021.701673. eCollection 2021. PMID: 34267628; PMCID: PMC8276040.
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Kupffer cells regulate liver recovery through induction of chemokine receptor CXCR2 on hepatocytes after acetaminophen overdose in mice.

Archives of toxicology

Nguyen NT, Umbaugh DS, Sanchez-Guerrero G, Ramachandran A, Jaeschke H.
PMID: 34724096
Arch Toxicol. 2022 Jan;96(1):305-320. doi: 10.1007/s00204-021-03183-0. Epub 2021 Nov 01.

Acetaminophen (APAP) is a widely used analgesic, but also a main cause of acute liver injury in the United States and many western countries. APAP hepatotoxicity is associated with a sterile inflammatory response as shown by the infiltration of...

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Nguyen NT, Umbaugh DS, Sanchez-Guerrero G, et al. Kupffer cells regulate liver recovery through induction of chemokine receptor CXCR2 on hepatocytes after acetaminophen overdose in mice. Arch Toxicol. 2021;96(1):305-320doi: 10.1007/s00204-021-03183-0.
Nguyen, N. T., Umbaugh, D. S., Sanchez-Guerrero, G., Ramachandran, A., & Jaeschke, H. (2022). Kupffer cells regulate liver recovery through induction of chemokine receptor CXCR2 on hepatocytes after acetaminophen overdose in mice. Archives of toxicology, 96(1), 305-320. https://doi.org/10.1007/s00204-021-03183-0
Nguyen, Nga T, et al. "Kupffer cells regulate liver recovery through induction of chemokine receptor CXCR2 on hepatocytes after acetaminophen overdose in mice." Archives of toxicology vol. 96,1 (2022): 305-320. doi: https://doi.org/10.1007/s00204-021-03183-0
Nguyen NT, Umbaugh DS, Sanchez-Guerrero G, Ramachandran A, Jaeschke H. Kupffer cells regulate liver recovery through induction of chemokine receptor CXCR2 on hepatocytes after acetaminophen overdose in mice. Arch Toxicol. 2022 Jan;96(1):305-320. doi: 10.1007/s00204-021-03183-0. Epub 2021 Nov 01. PMID: 34724096.
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Secondary Education in COVID Lockdown: More Anxious and Less Creative-Maybe Not?.

Frontiers in psychology

Patston TJ, Kennedy J, Jaeschke W, Kapoor H, Leonard SN, Cropley DH, Kaufman JC.
PMID: 33692721
Front Psychol. 2021 Feb 22;12:613055. doi: 10.3389/fpsyg.2021.613055. eCollection 2021.

Secondary education around the world has been significantly disrupted by covid-19. Students have been forced into new ways of independent learning, often using remote technologies, but without the social nuances and direct teacher interactions of a normal classroom environment....

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Patston TJ, Kennedy J, Jaeschke W, et al. Secondary Education in COVID Lockdown: More Anxious and Less Creative-Maybe Not?. Front Psychol. 2021;12:613055doi: 10.3389/fpsyg.2021.613055.
Patston, T. J., Kennedy, J., Jaeschke, W., Kapoor, H., Leonard, S. N., Cropley, D. H., & Kaufman, J. C. (2021). Secondary Education in COVID Lockdown: More Anxious and Less Creative-Maybe Not?. Frontiers in psychology, 12613055. https://doi.org/10.3389/fpsyg.2021.613055
Patston, Timothy J, et al. "Secondary Education in COVID Lockdown: More Anxious and Less Creative-Maybe Not?." Frontiers in psychology vol. 12 (2021): 613055. doi: https://doi.org/10.3389/fpsyg.2021.613055
Patston TJ, Kennedy J, Jaeschke W, Kapoor H, Leonard SN, Cropley DH, Kaufman JC. Secondary Education in COVID Lockdown: More Anxious and Less Creative-Maybe Not?. Front Psychol. 2021 Feb 22;12:613055. doi: 10.3389/fpsyg.2021.613055. eCollection 2021. PMID: 33692721; PMCID: PMC7937704.
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Hole doping and chemical pressure effects on the strong coupling superconductor PdTe.

Physical chemistry chemical physics : PCCP

Chen L, Ide A, Jeschke HO, Kobayashi K.
PMID: 34096944
Phys Chem Chem Phys. 2021 Jun 16;23(23):13331-13337. doi: 10.1039/d1cp01071a.

Chemical doping of known superconductors is a probate strategy to test and enhance our understanding of which parameters control the critical temperature Tc and the critical magnetic fields. The transition metal chalcogenide PdTe is considered a conventional type II...

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Chen L, Ide A, Jeschke HO, et al. Hole doping and chemical pressure effects on the strong coupling superconductor PdTe. Phys Chem Chem Phys. 2021;23(23):13331-13337doi: 10.1039/d1cp01071a.
Chen, L., Ide, A., Jeschke, H. O., & Kobayashi, K. (2021). Hole doping and chemical pressure effects on the strong coupling superconductor PdTe. Physical chemistry chemical physics : PCCP, 23(23), 13331-13337. https://doi.org/10.1039/d1cp01071a
Chen, Li, et al. "Hole doping and chemical pressure effects on the strong coupling superconductor PdTe." Physical chemistry chemical physics : PCCP vol. 23,23 (2021): 13331-13337. doi: https://doi.org/10.1039/d1cp01071a
Chen L, Ide A, Jeschke HO, Kobayashi K. Hole doping and chemical pressure effects on the strong coupling superconductor PdTe. Phys Chem Chem Phys. 2021 Jun 16;23(23):13331-13337. doi: 10.1039/d1cp01071a. PMID: 34096944.
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Targeting the sterile inflammatory response during acetaminophen hepatotoxicity with natural products.

Toxicology letters

Jaeschke H, Ramachandran A.
PMID: 34801638
Toxicol Lett. 2022 Feb 01;355:170-171. doi: 10.1016/j.toxlet.2021.11.006. Epub 2021 Nov 18.

No abstract available.

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Jaeschke H, Ramachandran A. Targeting the sterile inflammatory response during acetaminophen hepatotoxicity with natural products. Toxicol Lett. 2021;355:170-171doi: 10.1016/j.toxlet.2021.11.006.
Jaeschke, H., & Ramachandran, A. (2022). Targeting the sterile inflammatory response during acetaminophen hepatotoxicity with natural products. Toxicology letters, 355170-171. https://doi.org/10.1016/j.toxlet.2021.11.006
Jaeschke, Hartmut, and Ramachandran, Anup. "Targeting the sterile inflammatory response during acetaminophen hepatotoxicity with natural products." Toxicology letters vol. 355 (2022): 170-171. doi: https://doi.org/10.1016/j.toxlet.2021.11.006
Jaeschke H, Ramachandran A. Targeting the sterile inflammatory response during acetaminophen hepatotoxicity with natural products. Toxicol Lett. 2022 Feb 01;355:170-171. doi: 10.1016/j.toxlet.2021.11.006. Epub 2021 Nov 18. PMID: 34801638.
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Magnetization Process of Atacamite: A Case of Weakly Coupled S=1/2 Sawtooth Chains.

Physical review letters

Heinze L, Jeschke HO, Mazin II, Metavitsiadis A, Reehuis M, Feyerherm R, Hoffmann JU, Bartkowiak M, Prokhnenko O, Wolter AUB, Ding X, Zapf VS, Corvalán Moya C, Weickert F, Jaime M, Rule KC, Menzel D, Valentí R, Brenig W, Süllow S.
PMID: 34110224
Phys Rev Lett. 2021 May 21;126(20):207201. doi: 10.1103/PhysRevLett.126.207201.

We present a combined experimental and theoretical study of the mineral atacamite Cu_{2}Cl(OH)_{3}. Density-functional theory yields a Hamiltonian describing anisotropic sawtooth chains with weak 3D connections. Experimentally, we fully characterize the antiferromagnetically ordered state. Magnetic order shows a complex...

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Heinze L, Jeschke HO, Mazin II, et al. Magnetization Process of Atacamite: A Case of Weakly Coupled S=1/2 Sawtooth Chains. Phys Rev Lett. 2021;126(20):207201doi: 10.1103/PhysRevLett.126.207201.
Heinze, L., Jeschke, H. O., Mazin, I. I., Metavitsiadis, A., Reehuis, M., Feyerherm, R., Hoffmann, J. U., Bartkowiak, M., Prokhnenko, O., Wolter, A. U. B., Ding, X., Zapf, V. S., Corvalán Moya, C., Weickert, F., Jaime, M., Rule, K. C., Menzel, D., Valentí, R., Brenig, W., & Süllow, S. (2021). Magnetization Process of Atacamite: A Case of Weakly Coupled S=1/2 Sawtooth Chains. Physical review letters, 126(20), 207201. https://doi.org/10.1103/PhysRevLett.126.207201
Heinze, L, et al. "Magnetization Process of Atacamite: A Case of Weakly Coupled S=1/2 Sawtooth Chains." Physical review letters vol. 126,20 (2021): 207201. doi: https://doi.org/10.1103/PhysRevLett.126.207201
Heinze L, Jeschke HO, Mazin II, Metavitsiadis A, Reehuis M, Feyerherm R, Hoffmann JU, Bartkowiak M, Prokhnenko O, Wolter AUB, Ding X, Zapf VS, Corvalán Moya C, Weickert F, Jaime M, Rule KC, Menzel D, Valentí R, Brenig W, Süllow S. Magnetization Process of Atacamite: A Case of Weakly Coupled S=1/2 Sawtooth Chains. Phys Rev Lett. 2021 May 21;126(20):207201. doi: 10.1103/PhysRevLett.126.207201. PMID: 34110224.
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Convalescent Plasma in COVID-19: To what Degree should Clinicians Rely on Currently Available Data?.

Saudi journal of medicine & medical sciences

Alhazzani W, Jaeschke R.
PMID: 33519336
Saudi J Med Med Sci. 2021 Jan-Apr;9(1):1-2. doi: 10.4103/sjmms.sjmms_882_20. Epub 2020 Dec 26.

No abstract available.

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Alhazzani W, Jaeschke R. Convalescent Plasma in COVID-19: To what Degree should Clinicians Rely on Currently Available Data?. Saudi J Med Med Sci. 2020;9(1):1-2doi: 10.4103/sjmms.sjmms_882_20.
Alhazzani, W., & Jaeschke, R. (2021). Convalescent Plasma in COVID-19: To what Degree should Clinicians Rely on Currently Available Data?. Saudi journal of medicine & medical sciences, 9(1), 1-2. https://doi.org/10.4103/sjmms.sjmms_882_20
Alhazzani, Waleed, and Jaeschke, Roman. "Convalescent Plasma in COVID-19: To what Degree should Clinicians Rely on Currently Available Data?." Saudi journal of medicine & medical sciences vol. 9,1 (2021): 1-2. doi: https://doi.org/10.4103/sjmms.sjmms_882_20
Alhazzani W, Jaeschke R. Convalescent Plasma in COVID-19: To what Degree should Clinicians Rely on Currently Available Data?. Saudi J Med Med Sci. 2021 Jan-Apr;9(1):1-2. doi: 10.4103/sjmms.sjmms_882_20. Epub 2020 Dec 26. PMID: 33519336; PMCID: PMC7839581.
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Fructose Protects Against Acetaminophen-Induced Hepatotoxicity Mainly by Activating the Carbohydrate-Response Element-Binding Protein α-Fibroblast Growth Factor 21 Axis in Mice.

Hepatology communications

Zhang D, Wang S, Ospina E, Shabandri O, Lank D, Akakpo JY, Zhao Z, Yang M, Wu J, Jaeschke H, Saha P, Tong X, Yin L.
PMID: 34141985
Hepatol Commun. 2021 Feb 23;5(6):992-1008. doi: 10.1002/hep4.1683. eCollection 2021 Jun.

Acetaminophen (N-acetyl-para-aminophenol [APAP]) overdose is the most common cause of drug-induced liver injury in the Western world and has limited therapeutic options. As an important dietary component intake, fructose is mainly metabolized in liver, but its impact on APAP-induced...

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Zhang D, Wang S, Ospina E, et al. Fructose Protects Against Acetaminophen-Induced Hepatotoxicity Mainly by Activating the Carbohydrate-Response Element-Binding Protein α-Fibroblast Growth Factor 21 Axis in Mice. Hepatol Commun. 2021;5(6):992-1008doi: 10.1002/hep4.1683.
Zhang, D., Wang, S., Ospina, E., Shabandri, O., Lank, D., Akakpo, J. Y., Zhao, Z., Yang, M., Wu, J., Jaeschke, H., Saha, P., Tong, X., & Yin, L. (2021). Fructose Protects Against Acetaminophen-Induced Hepatotoxicity Mainly by Activating the Carbohydrate-Response Element-Binding Protein α-Fibroblast Growth Factor 21 Axis in Mice. Hepatology communications, 5(6), 992-1008. https://doi.org/10.1002/hep4.1683
Zhang, Deqiang, et al. "Fructose Protects Against Acetaminophen-Induced Hepatotoxicity Mainly by Activating the Carbohydrate-Response Element-Binding Protein α-Fibroblast Growth Factor 21 Axis in Mice." Hepatology communications vol. 5,6 (2021): 992-1008. doi: https://doi.org/10.1002/hep4.1683
Zhang D, Wang S, Ospina E, Shabandri O, Lank D, Akakpo JY, Zhao Z, Yang M, Wu J, Jaeschke H, Saha P, Tong X, Yin L. Fructose Protects Against Acetaminophen-Induced Hepatotoxicity Mainly by Activating the Carbohydrate-Response Element-Binding Protein α-Fibroblast Growth Factor 21 Axis in Mice. Hepatol Commun. 2021 Feb 23;5(6):992-1008. doi: 10.1002/hep4.1683. eCollection 2021 Jun. PMID: 34141985; PMCID: PMC8183176.
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Showing 25 to 36 of 259 entries