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Progression-Related Loss of Stromal Caveolin 1 Levels Mediates Radiation Resistance in Prostate Carcinoma via the Apoptosis Inhibitor TRIAP1.

Journal of clinical medicine

Ketteler J, Panic A, Reis H, Wittka A, Maier P, Herskind C, Yagüe E, Jendrossek V, Klein D.
PMID: 30871022
J Clin Med. 2019 Mar 12;8(3). doi: 10.3390/jcm8030348.

Tumour resistance to chemo- and radiotherapy, as well as molecularly targeted therapies, limits the effectiveness of current cancer treatments. We previously reported that the radiation response of human prostate tumours is critically regulated by CAV1 expression in stromal fibroblasts...

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Ketteler J, Panic A, Reis H, et al. Progression-Related Loss of Stromal Caveolin 1 Levels Mediates Radiation Resistance in Prostate Carcinoma via the Apoptosis Inhibitor TRIAP1. J Clin Med. 2019;8(3)doi: 10.3390/jcm8030348.
Ketteler, J., Panic, A., Reis, H., Wittka, A., Maier, P., Herskind, C., Yagüe, E., Jendrossek, V., & Klein, D. (2019). Progression-Related Loss of Stromal Caveolin 1 Levels Mediates Radiation Resistance in Prostate Carcinoma via the Apoptosis Inhibitor TRIAP1. Journal of clinical medicine, 8(3), . https://doi.org/10.3390/jcm8030348
Ketteler, Julia, et al. "Progression-Related Loss of Stromal Caveolin 1 Levels Mediates Radiation Resistance in Prostate Carcinoma via the Apoptosis Inhibitor TRIAP1." Journal of clinical medicine vol. 8,3 (2019). doi: https://doi.org/10.3390/jcm8030348
Ketteler J, Panic A, Reis H, Wittka A, Maier P, Herskind C, Yagüe E, Jendrossek V, Klein D. Progression-Related Loss of Stromal Caveolin 1 Levels Mediates Radiation Resistance in Prostate Carcinoma via the Apoptosis Inhibitor TRIAP1. J Clin Med. 2019 Mar 12;8(3). doi: 10.3390/jcm8030348. PMID: 30871022; PMCID: PMC6462938.
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Interdisciplinary Diagnosis, Therapy and Follow-up of Patients with Endometrial Cancer. Guideline (S3-Level, AWMF Registry Number 032/034-OL, April 2018) - Part 2 with Recommendations on the Therapy and Follow-up of Endometrial Cancer, Palliative Care, Psycho-oncological/Psychosocial Care/Rehabilitation/Patient Information and Healthcare Facilities.

Geburtshilfe und Frauenheilkunde

Emons G, Steiner E, Vordermark D, Uleer C, Bock N, Paradies K, Ortmann O, Aretz S, Mallmann P, Kurzeder C, Hagen V, van Oorschot B, Höcht S, Feyer P, Egerer G, Friedrich M, Cremer W, Prott FJ, Horn LC, Prömpeler H, Langrehr J, Leinung S, Beckmann MW, Kimmig R, Letsch A, Reinhardt M, Alt-Epping B, Kiesel L, Menke J, Gebhardt M, Steinke-Lange V, Rahner N, Lichtenegger W, Zeimet A, Hanf V, Weis J, Mueller M, Henscher U, Schmutzler RK, Meindl A, Hilpert F, Panke JE, Strnad V, Niehues C, Dauelsberg T, Niehoff P, Mayr D, Grab D, Kreißl M, Witteler R, Schorsch A, Mustea A, Petru E, Hübner J, Rose AD, Wight E, Tholen R, Bauerschmitz GJ, Fleisch M, Juhasz-Boess I, Lax S, Runnebaum I, Tempfer C, Nothacker MJ, Blödt S, Follmann M, Langer T, Raatz H, Wesselmann S, Erdogan S.
PMID: 30581199
Geburtshilfe Frauenheilkd. 2018 Nov;78(11):1089-1109. doi: 10.1055/a-0715-2964. Epub 2018 Nov 26.

No abstract available.

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Emons G, Steiner E, Vordermark D, et al. Interdisciplinary Diagnosis, Therapy and Follow-up of Patients with Endometrial Cancer. Guideline (S3-Level, AWMF Registry Number 032/034-OL, April 2018) - Part 2 with Recommendations on the Therapy and Follow-up of Endometrial Cancer, Palliative Care, Psycho-oncological/Psychosocial Care/Rehabilitation/Patient Information and Healthcare Facilities. Geburtshilfe Frauenheilkd. 2018;78(11):1089-1109doi: 10.1055/a-0715-2964.
Emons, G., Steiner, E., Vordermark, D., Uleer, C., Bock, N., Paradies, K., Ortmann, O., Aretz, S., Mallmann, P., Kurzeder, C., Hagen, V., van Oorschot, B., Höcht, S., Feyer, P., Egerer, G., Friedrich, M., Cremer, W., Prott, F. J., Horn, L. C., Prömpeler, H., Langrehr, J., Leinung, S., Beckmann, M. W., Kimmig, R., Letsch, A., Reinhardt, M., Alt-Epping, B., Kiesel, L., Menke, J., Gebhardt, M., Steinke-Lange, V., Rahner, N., Lichtenegger, W., Zeimet, A., Hanf, V., Weis, J., Mueller, M., Henscher, U., Schmutzler, R. K., Meindl, A., Hilpert, F., Panke, J. E., Strnad, V., Niehues, C., Dauelsberg, T., Niehoff, P., Mayr, D., Grab, D., Kreißl, M., Witteler, R., Schorsch, A., Mustea, A., Petru, E., Hübner, J., Rose, A. D., Wight, E., Tholen, R., Bauerschmitz, G. J., Fleisch, M., Juhasz-Boess, I., Lax, S., Runnebaum, I., Tempfer, C., Nothacker, M. J., Blödt, S., Follmann, M., Langer, T., Raatz, H., Wesselmann, S., & Erdogan, S. (2018). Interdisciplinary Diagnosis, Therapy and Follow-up of Patients with Endometrial Cancer. Guideline (S3-Level, AWMF Registry Number 032/034-OL, April 2018) - Part 2 with Recommendations on the Therapy and Follow-up of Endometrial Cancer, Palliative Care, Psycho-oncological/Psychosocial Care/Rehabilitation/Patient Information and Healthcare Facilities. Geburtshilfe und Frauenheilkunde, 78(11), 1089-1109. https://doi.org/10.1055/a-0715-2964
Emons, Günter, et al. "Interdisciplinary Diagnosis, Therapy and Follow-up of Patients with Endometrial Cancer. Guideline (S3-Level, AWMF Registry Number 032/034-OL, April 2018) - Part 2 with Recommendations on the Therapy and Follow-up of Endometrial Cancer, Palliative Care, Psycho-oncological/Psychosocial Care/Rehabilitation/Patient Information and Healthcare Facilities." Geburtshilfe und Frauenheilkunde vol. 78,11 (2018): 1089-1109. doi: https://doi.org/10.1055/a-0715-2964
Emons G, Steiner E, Vordermark D, Uleer C, Bock N, Paradies K, Ortmann O, Aretz S, Mallmann P, Kurzeder C, Hagen V, van Oorschot B, Höcht S, Feyer P, Egerer G, Friedrich M, Cremer W, Prott FJ, Horn LC, Prömpeler H, Langrehr J, Leinung S, Beckmann MW, Kimmig R, Letsch A, Reinhardt M, Alt-Epping B, Kiesel L, Menke J, Gebhardt M, Steinke-Lange V, Rahner N, Lichtenegger W, Zeimet A, Hanf V, Weis J, Mueller M, Henscher U, Schmutzler RK, Meindl A, Hilpert F, Panke JE, Strnad V, Niehues C, Dauelsberg T, Niehoff P, Mayr D, Grab D, Kreißl M, Witteler R, Schorsch A, Mustea A, Petru E, Hübner J, Rose AD, Wight E, Tholen R, Bauerschmitz GJ, Fleisch M, Juhasz-Boess I, Lax S, Runnebaum I, Tempfer C, Nothacker MJ, Blödt S, Follmann M, Langer T, Raatz H, Wesselmann S, Erdogan S. Interdisciplinary Diagnosis, Therapy and Follow-up of Patients with Endometrial Cancer. Guideline (S3-Level, AWMF Registry Number 032/034-OL, April 2018) - Part 2 with Recommendations on the Therapy and Follow-up of Endometrial Cancer, Palliative Care, Psycho-oncological/Psychosocial Care/Rehabilitation/Patient Information and Healthcare Facilities. Geburtshilfe Frauenheilkd. 2018 Nov;78(11):1089-1109. doi: 10.1055/a-0715-2964. Epub 2018 Nov 26. PMID: 30581199; PMCID: PMC6261739.
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Multimodal cell with simultaneous electrochemical quartz crystal microbalance and in operando spectroscopic ellipsometry to understand thin film electrochemistry.

The Review of scientific instruments

Gettler R, Young MJ.
PMID: 34243232
Rev Sci Instrum. 2021 May 01;92(5):053902. doi: 10.1063/5.0035309.

To inform the development of advanced electrodes for energy storage, water treatment, and catalysis, among other applications, we need to improve our understanding of how material structure evolves during electrochemical operation. Insight into the evolution of local atomic structure...

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Gettler R, Young MJ. Multimodal cell with simultaneous electrochemical quartz crystal microbalance and in operando spectroscopic ellipsometry to understand thin film electrochemistry. Rev Sci Instrum. 2021;92(5):053902doi: 10.1063/5.0035309.
Gettler, R., & Young, M. J. (2021). Multimodal cell with simultaneous electrochemical quartz crystal microbalance and in operando spectroscopic ellipsometry to understand thin film electrochemistry. The Review of scientific instruments, 92(5), 053902. https://doi.org/10.1063/5.0035309
Gettler, Ryan, and Young, Matthias J. "Multimodal cell with simultaneous electrochemical quartz crystal microbalance and in operando spectroscopic ellipsometry to understand thin film electrochemistry." The Review of scientific instruments vol. 92,5 (2021): 053902. doi: https://doi.org/10.1063/5.0035309
Gettler R, Young MJ. Multimodal cell with simultaneous electrochemical quartz crystal microbalance and in operando spectroscopic ellipsometry to understand thin film electrochemistry. Rev Sci Instrum. 2021 May 01;92(5):053902. doi: 10.1063/5.0035309. PMID: 34243232.
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Presentation acuity, induction mortality, and resource utilization in infants with acute leukemia.

Pediatric blood & cancer

Ibrahimova A, Winestone LE, Miller TP, Kettler K, Seif AE, Huang YS, Elgarten CW, Myers RM, Fisher BT, Aplenc R, Getz KD.
PMID: 33704911
Pediatr Blood Cancer. 2021 Jul;68(7):e28940. doi: 10.1002/pbc.28940. Epub 2021 Mar 11.

BACKGROUND: Treatment of infants with acute leukemia remains challenging, especially for acute lymphocytic leukemia (ALL). Infants have shown markedly higher rates of induction mortality compared with noninfants. There are limited data on presentation acuity and supportive care utilization in...

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Ibrahimova A, Winestone LE, Miller TP, et al. Presentation acuity, induction mortality, and resource utilization in infants with acute leukemia. Pediatr Blood Cancer. 2021;68(7):e28940doi: 10.1002/pbc.28940.
Ibrahimova, A., Winestone, L. E., Miller, T. P., Kettler, K., Seif, A. E., Huang, Y. S., Elgarten, C. W., Myers, R. M., Fisher, B. T., Aplenc, R., & Getz, K. D. (2021). Presentation acuity, induction mortality, and resource utilization in infants with acute leukemia. Pediatric blood & cancer, 68(7), e28940. https://doi.org/10.1002/pbc.28940
Ibrahimova, Azada, et al. "Presentation acuity, induction mortality, and resource utilization in infants with acute leukemia." Pediatric blood & cancer vol. 68,7 (2021): e28940. doi: https://doi.org/10.1002/pbc.28940
Ibrahimova A, Winestone LE, Miller TP, Kettler K, Seif AE, Huang YS, Elgarten CW, Myers RM, Fisher BT, Aplenc R, Getz KD. Presentation acuity, induction mortality, and resource utilization in infants with acute leukemia. Pediatr Blood Cancer. 2021 Jul;68(7):e28940. doi: 10.1002/pbc.28940. Epub 2021 Mar 11. PMID: 33704911; PMCID: PMC8283996.
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Neuroanatomy of the nodosaurid Struthiosaurus austriacus (Dinosauria: Thyreophora) supports potential ecological differentiations within Ankylosauria.

Scientific reports

Schade M, Stumpf S, Kriwet J, Kettler C, Pfaff C.
PMID: 34996895
Sci Rep. 2022 Jan 07;12(1):144. doi: 10.1038/s41598-021-03599-9.

Nodosauridae is a group of thyreophoran dinosaurs characterized by a collar of prominent osteoderms. In comparison to its sister group, the often club-tailed ankylosaurids, a different lifestyle of nodosaurids could be assumed based on their neuroanatomy and weaponry, e.g.,...

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Schade M, Stumpf S, Kriwet J, et al. Neuroanatomy of the nodosaurid Struthiosaurus austriacus (Dinosauria: Thyreophora) supports potential ecological differentiations within Ankylosauria. Sci Rep. 2022;12(1):144doi: 10.1038/s41598-021-03599-9.
Schade, M., Stumpf, S., Kriwet, J., Kettler, C., & Pfaff, C. (2022). Neuroanatomy of the nodosaurid Struthiosaurus austriacus (Dinosauria: Thyreophora) supports potential ecological differentiations within Ankylosauria. Scientific reports, 12(1), 144. https://doi.org/10.1038/s41598-021-03599-9
Schade, Marco, et al. "Neuroanatomy of the nodosaurid Struthiosaurus austriacus (Dinosauria: Thyreophora) supports potential ecological differentiations within Ankylosauria." Scientific reports vol. 12,1 (2022): 144. doi: https://doi.org/10.1038/s41598-021-03599-9
Schade M, Stumpf S, Kriwet J, Kettler C, Pfaff C. Neuroanatomy of the nodosaurid Struthiosaurus austriacus (Dinosauria: Thyreophora) supports potential ecological differentiations within Ankylosauria. Sci Rep. 2022 Jan 07;12(1):144. doi: 10.1038/s41598-021-03599-9. PMID: 34996895.
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Modulation of muscle redox and protein aggregation rescues lethality caused by mutant lamins.

Redox biology

Coombs GS, Rios-Monterrosa JL, Lai S, Dai Q, Goll AC, Ketterer MR, Valdes MF, Uche N, Benjamin IJ, Wallrath LL.
PMID: 34872044
Redox Biol. 2021 Nov 25;48:102196. doi: 10.1016/j.redox.2021.102196. Epub 2021 Nov 25.

Mutations in the human LMNA gene cause a collection of diseases called laminopathies, which includes muscular dystrophy and dilated cardiomyopathy. The LMNA gene encodes lamins, filamentous proteins that form a meshwork on the inner side of the nuclear envelope....

Cite
Coombs GS, Rios-Monterrosa JL, Lai S, et al. Modulation of muscle redox and protein aggregation rescues lethality caused by mutant lamins. Redox Biol. 2021;48:102196doi: 10.1016/j.redox.2021.102196.
Coombs, G. S., Rios-Monterrosa, J. L., Lai, S., Dai, Q., Goll, A. C., Ketterer, M. R., Valdes, M. F., Uche, N., Benjamin, I. J., & Wallrath, L. L. (2021). Modulation of muscle redox and protein aggregation rescues lethality caused by mutant lamins. Redox biology, 48102196. https://doi.org/10.1016/j.redox.2021.102196
Coombs, Gary S, et al. "Modulation of muscle redox and protein aggregation rescues lethality caused by mutant lamins." Redox biology vol. 48 (2021): 102196. doi: https://doi.org/10.1016/j.redox.2021.102196
Coombs GS, Rios-Monterrosa JL, Lai S, Dai Q, Goll AC, Ketterer MR, Valdes MF, Uche N, Benjamin IJ, Wallrath LL. Modulation of muscle redox and protein aggregation rescues lethality caused by mutant lamins. Redox Biol. 2021 Nov 25;48:102196. doi: 10.1016/j.redox.2021.102196. Epub 2021 Nov 25. PMID: 34872044.
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Trial design and baseline characteristics of CaLIPSO: a randomized, double-blind placebo-controlled trial of SNF472 in patients receiving haemodialysis with cardiovascular calcification.

Clinical kidney journal

Bellasi A, Raggi P, Bover J, Bushinsky DA, Chertow GM, Ketteler M, Rodriguez M, Sinha S, Salcedo C, Garg R, Gold A, Perelló J.
PMID: 33564440
Clin Kidney J. 2019 Oct 30;14(1):366-374. doi: 10.1093/ckj/sfz144. eCollection 2021 Jan.

BACKGROUND: The objective of CaLIPSO, a Phase 2b, randomized, double-blind, placebo-controlled clinical trial, is to test the hypothesis that myo-inositol hexaphosphate (SNF472) attenuates the progression of cardiovascular calcification in patients receiving maintenance haemodialysis. Here we report the trial design...

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Bellasi A, Raggi P, Bover J, et al. Trial design and baseline characteristics of CaLIPSO: a randomized, double-blind placebo-controlled trial of SNF472 in patients receiving haemodialysis with cardiovascular calcification. Clin Kidney J. 2019;14(1):366-374doi: 10.1093/ckj/sfz144.
Bellasi, A., Raggi, P., Bover, J., Bushinsky, D. A., Chertow, G. M., Ketteler, M., Rodriguez, M., Sinha, S., Salcedo, C., Garg, R., Gold, A., & Perelló, J. (2021). Trial design and baseline characteristics of CaLIPSO: a randomized, double-blind placebo-controlled trial of SNF472 in patients receiving haemodialysis with cardiovascular calcification. Clinical kidney journal, 14(1), 366-374. https://doi.org/10.1093/ckj/sfz144
Bellasi, Antonio, et al. "Trial design and baseline characteristics of CaLIPSO: a randomized, double-blind placebo-controlled trial of SNF472 in patients receiving haemodialysis with cardiovascular calcification." Clinical kidney journal vol. 14,1 (2021): 366-374. doi: https://doi.org/10.1093/ckj/sfz144
Bellasi A, Raggi P, Bover J, Bushinsky DA, Chertow GM, Ketteler M, Rodriguez M, Sinha S, Salcedo C, Garg R, Gold A, Perelló J. Trial design and baseline characteristics of CaLIPSO: a randomized, double-blind placebo-controlled trial of SNF472 in patients receiving haemodialysis with cardiovascular calcification. Clin Kidney J. 2019 Oct 30;14(1):366-374. doi: 10.1093/ckj/sfz144. eCollection 2021 Jan. PMID: 33564440; PMCID: PMC7857813.
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Investigation of USP30 inhibition to enhance Parkin-mediated mitophagy: tools and approaches.

The Biochemical journal

Tsefou E, Walker AS, Clark EH, Hicks AR, Luft C, Takeda K, Watanabe T, Ramazio B, Staddon JM, Briston T, Ketteler R.
PMID: 34704599
Biochem J. 2021 Dec 10;478(23):4099-4118. doi: 10.1042/BCJ20210508.

Mitochondrial dysfunction is implicated in Parkinson disease (PD). Mutations in Parkin, an E3 ubiquitin ligase, can cause juvenile-onset Parkinsonism, probably through impairment of mitophagy. Inhibition of the de-ubiquitinating enzyme USP30 may counter this effect to enhance mitophagy. Using different...

Cite
Tsefou E, Walker AS, Clark EH, et al. Investigation of USP30 inhibition to enhance Parkin-mediated mitophagy: tools and approaches. Biochem J. 2021;478(23):4099-4118doi: 10.1042/BCJ20210508.
Tsefou, E., Walker, A. S., Clark, E. H., Hicks, A. R., Luft, C., Takeda, K., Watanabe, T., Ramazio, B., Staddon, J. M., Briston, T., & Ketteler, R. (2021). Investigation of USP30 inhibition to enhance Parkin-mediated mitophagy: tools and approaches. The Biochemical journal, 478(23), 4099-4118. https://doi.org/10.1042/BCJ20210508
Tsefou, Eliona, et al. "Investigation of USP30 inhibition to enhance Parkin-mediated mitophagy: tools and approaches." The Biochemical journal vol. 478,23 (2021): 4099-4118. doi: https://doi.org/10.1042/BCJ20210508
Tsefou E, Walker AS, Clark EH, Hicks AR, Luft C, Takeda K, Watanabe T, Ramazio B, Staddon JM, Briston T, Ketteler R. Investigation of USP30 inhibition to enhance Parkin-mediated mitophagy: tools and approaches. Biochem J. 2021 Dec 10;478(23):4099-4118. doi: 10.1042/BCJ20210508. PMID: 34704599.
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Feasibility of Proton Beam Therapy as a Rescue Therapy in Heavily Pre-Treated Retinoblastoma Eyes.

Cancers

Biewald E, Kiefer T, Geismar D, Schlüter S, Manthey A, Westekemper H, Wulff J, Timmermann B, Ketteler P, Schönberger S, Metz KA, Ting S, Göricke S, Bechrakis NE, Bornfeld N.
PMID: 33924716
Cancers (Basel). 2021 Apr 13;13(8). doi: 10.3390/cancers13081862.

Despite the increased risk of subsequent primary tumors (SPTs) external beam radiation (EBRT) may be the only therapeutic option to preserve a retinoblastoma eye. Due to their physical properties, proton beam therapy (PBT) offers the possibility to use the...

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Biewald E, Kiefer T, Geismar D, et al. Feasibility of Proton Beam Therapy as a Rescue Therapy in Heavily Pre-Treated Retinoblastoma Eyes. Cancers (Basel). 2021;13(8)doi: 10.3390/cancers13081862.
Biewald, E., Kiefer, T., Geismar, D., Schlüter, S., Manthey, A., Westekemper, H., Wulff, J., Timmermann, B., Ketteler, P., Schönberger, S., Metz, K. A., Ting, S., Göricke, S., Bechrakis, N. E., & Bornfeld, N. (2021). Feasibility of Proton Beam Therapy as a Rescue Therapy in Heavily Pre-Treated Retinoblastoma Eyes. Cancers, 13(8), . https://doi.org/10.3390/cancers13081862
Biewald, Eva, et al. "Feasibility of Proton Beam Therapy as a Rescue Therapy in Heavily Pre-Treated Retinoblastoma Eyes." Cancers vol. 13,8 (2021). doi: https://doi.org/10.3390/cancers13081862
Biewald E, Kiefer T, Geismar D, Schlüter S, Manthey A, Westekemper H, Wulff J, Timmermann B, Ketteler P, Schönberger S, Metz KA, Ting S, Göricke S, Bechrakis NE, Bornfeld N. Feasibility of Proton Beam Therapy as a Rescue Therapy in Heavily Pre-Treated Retinoblastoma Eyes. Cancers (Basel). 2021 Apr 13;13(8). doi: 10.3390/cancers13081862. PMID: 33924716; PMCID: PMC8069965.
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Inducing micromechanical motion by optical excitation of a single quantum dot.

Nature nanotechnology

Kettler J, Vaish N, de Lépinay LM, Besga B, de Assis PL, Bourgeois O, Auffèves A, Richard M, Claudon J, Gérard JM, Pigeau B, Arcizet O, Verlot P, Poizat JP.
PMID: 33349683
Nat Nanotechnol. 2021 Mar;16(3):283-287. doi: 10.1038/s41565-020-00814-y. Epub 2020 Dec 21.

Hybrid quantum optomechanical systems

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Kettler J, Vaish N, de Lépinay LM, et al. Inducing micromechanical motion by optical excitation of a single quantum dot. Nat Nanotechnol. 2020;16(3):283-287doi: 10.1038/s41565-020-00814-y.
Kettler, J., Vaish, N., de Lépinay, L. M., Besga, B., de Assis, P. L., Bourgeois, O., Auffèves, A., Richard, M., Claudon, J., Gérard, J. M., Pigeau, B., Arcizet, O., Verlot, P., & Poizat, J. P. (2021). Inducing micromechanical motion by optical excitation of a single quantum dot. Nature nanotechnology, 16(3), 283-287. https://doi.org/10.1038/s41565-020-00814-y
Kettler, Jan, et al. "Inducing micromechanical motion by optical excitation of a single quantum dot." Nature nanotechnology vol. 16,3 (2021): 283-287. doi: https://doi.org/10.1038/s41565-020-00814-y
Kettler J, Vaish N, de Lépinay LM, Besga B, de Assis PL, Bourgeois O, Auffèves A, Richard M, Claudon J, Gérard JM, Pigeau B, Arcizet O, Verlot P, Poizat JP. Inducing micromechanical motion by optical excitation of a single quantum dot. Nat Nanotechnol. 2021 Mar;16(3):283-287. doi: 10.1038/s41565-020-00814-y. Epub 2020 Dec 21. PMID: 33349683.
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Investigation of USP30 inhibition to enhance Parkin-mediated mitophagy: tools and approaches.

The Biochemical journal

Tsefou E, Walker AS, Clark EH, Hicks AR, Luft C, Takeda K, Watanabe T, Ramazio B, Staddon JM, Briston T, Ketteler R.
PMID: 34704599
Biochem J. 2021 Dec 10;478(23):4099-4118. doi: 10.1042/BCJ20210508.

Mitochondrial dysfunction is implicated in Parkinson disease (PD). Mutations in Parkin, an E3 ubiquitin ligase, can cause juvenile-onset Parkinsonism, probably through impairment of mitophagy. Inhibition of the de-ubiquitinating enzyme USP30 may counter this effect to enhance mitophagy. Using different...

Cite
Tsefou E, Walker AS, Clark EH, et al. Investigation of USP30 inhibition to enhance Parkin-mediated mitophagy: tools and approaches. Biochem J. 2021;478(23):4099-4118doi: 10.1042/BCJ20210508.
Tsefou, E., Walker, A. S., Clark, E. H., Hicks, A. R., Luft, C., Takeda, K., Watanabe, T., Ramazio, B., Staddon, J. M., Briston, T., & Ketteler, R. (2021). Investigation of USP30 inhibition to enhance Parkin-mediated mitophagy: tools and approaches. The Biochemical journal, 478(23), 4099-4118. https://doi.org/10.1042/BCJ20210508
Tsefou, Eliona, et al. "Investigation of USP30 inhibition to enhance Parkin-mediated mitophagy: tools and approaches." The Biochemical journal vol. 478,23 (2021): 4099-4118. doi: https://doi.org/10.1042/BCJ20210508
Tsefou E, Walker AS, Clark EH, Hicks AR, Luft C, Takeda K, Watanabe T, Ramazio B, Staddon JM, Briston T, Ketteler R. Investigation of USP30 inhibition to enhance Parkin-mediated mitophagy: tools and approaches. Biochem J. 2021 Dec 10;478(23):4099-4118. doi: 10.1042/BCJ20210508. PMID: 34704599.
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Modulation of muscle redox and protein aggregation rescues lethality caused by mutant lamins.

Redox biology

Coombs GS, Rios-Monterrosa JL, Lai S, Dai Q, Goll AC, Ketterer MR, Valdes MF, Uche N, Benjamin IJ, Wallrath LL.
PMID: 34872044
Redox Biol. 2021 Nov 25;48:102196. doi: 10.1016/j.redox.2021.102196. Epub 2021 Nov 25.

Mutations in the human LMNA gene cause a collection of diseases called laminopathies, which includes muscular dystrophy and dilated cardiomyopathy. The LMNA gene encodes lamins, filamentous proteins that form a meshwork on the inner side of the nuclear envelope....

Cite
Coombs GS, Rios-Monterrosa JL, Lai S, et al. Modulation of muscle redox and protein aggregation rescues lethality caused by mutant lamins. Redox Biol. 2021;48:102196doi: 10.1016/j.redox.2021.102196.
Coombs, G. S., Rios-Monterrosa, J. L., Lai, S., Dai, Q., Goll, A. C., Ketterer, M. R., Valdes, M. F., Uche, N., Benjamin, I. J., & Wallrath, L. L. (2021). Modulation of muscle redox and protein aggregation rescues lethality caused by mutant lamins. Redox biology, 48102196. https://doi.org/10.1016/j.redox.2021.102196
Coombs, Gary S, et al. "Modulation of muscle redox and protein aggregation rescues lethality caused by mutant lamins." Redox biology vol. 48 (2021): 102196. doi: https://doi.org/10.1016/j.redox.2021.102196
Coombs GS, Rios-Monterrosa JL, Lai S, Dai Q, Goll AC, Ketterer MR, Valdes MF, Uche N, Benjamin IJ, Wallrath LL. Modulation of muscle redox and protein aggregation rescues lethality caused by mutant lamins. Redox Biol. 2021 Nov 25;48:102196. doi: 10.1016/j.redox.2021.102196. Epub 2021 Nov 25. PMID: 34872044.
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Showing 25 to 36 of 204 entries