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Getting closer to the dirty little secret.

Immunity

Pelka K, Latz E.
PMID: 21511178
Immunity. 2011 Apr 22;34(4):455-8. doi: 10.1016/j.immuni.2011.04.003.

The molecular mechanism behind alum adjuvanticity is probably the oldest secret of immunology. In this issue of Immunity, Kuroda et al. (2011) and Kool et al. (2011) identify NLRP3 inflammasome-independent signaling to be crucial for the Th2 cell response...

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Pelka K, Latz E. Getting closer to the dirty little secret. Immunity. 2011;34(4):455-8doi: 10.1016/j.immuni.2011.04.003.
Pelka, K., & Latz, E. (2011). Getting closer to the dirty little secret. Immunity, 34(4), 455-8. https://doi.org/10.1016/j.immuni.2011.04.003
Pelka, Karin, and Latz, Eicke. "Getting closer to the dirty little secret." Immunity vol. 34,4 (2011): 455-8. doi: https://doi.org/10.1016/j.immuni.2011.04.003
Pelka K, Latz E. Getting closer to the dirty little secret. Immunity. 2011 Apr 22;34(4):455-8. doi: 10.1016/j.immuni.2011.04.003. PMID: 21511178; PMCID: PMC3128920.
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Driving regeneration, instead of healing, in adult mammals: the decisive role of resident macrophages through efferocytosis.

NPJ Regenerative medicine

Rabiller L, Robert V, Arlat A, Labit E, Ousset M, Salon M, Coste A, Da Costa-Fernandes L, Monsarrat P, Ségui B, André M, Guissard C, Renoud ML, Silva M, Mithieux G, Raymond-Letron I, Pénicaud L, Lorsignol A, Casteilla L, Dromard Berthézène C, Cousin B.
PMID: 34344890
NPJ Regen Med. 2021 Aug 03;6(1):41. doi: 10.1038/s41536-021-00151-1.

Tissue repair after lesion usually leads to scar healing and thus loss of function in adult mammals. In contrast, other adult vertebrates such as amphibians have the ability to regenerate and restore tissue homeostasis after lesion. Understanding the control...

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Rabiller L, Robert V, Arlat A, et al. Driving regeneration, instead of healing, in adult mammals: the decisive role of resident macrophages through efferocytosis. NPJ Regen Med. 2021;6(1):41doi: 10.1038/s41536-021-00151-1.
Rabiller, L., Robert, V., Arlat, A., Labit, E., Ousset, M., Salon, M., Coste, A., Da Costa-Fernandes, L., Monsarrat, P., Ségui, B., André, M., Guissard, C., Renoud, M. L., Silva, M., Mithieux, G., Raymond-Letron, I., Pénicaud, L., Lorsignol, A., Casteilla, L., Dromard Berthézène, C., & Cousin, B. (2021). Driving regeneration, instead of healing, in adult mammals: the decisive role of resident macrophages through efferocytosis. NPJ Regenerative medicine, 6(1), 41. https://doi.org/10.1038/s41536-021-00151-1
Rabiller, Lise, et al. "Driving regeneration, instead of healing, in adult mammals: the decisive role of resident macrophages through efferocytosis." NPJ Regenerative medicine vol. 6,1 (2021): 41. doi: https://doi.org/10.1038/s41536-021-00151-1
Rabiller L, Robert V, Arlat A, Labit E, Ousset M, Salon M, Coste A, Da Costa-Fernandes L, Monsarrat P, Ségui B, André M, Guissard C, Renoud ML, Silva M, Mithieux G, Raymond-Letron I, Pénicaud L, Lorsignol A, Casteilla L, Dromard Berthézène C, Cousin B. Driving regeneration, instead of healing, in adult mammals: the decisive role of resident macrophages through efferocytosis. NPJ Regen Med. 2021 Aug 03;6(1):41. doi: 10.1038/s41536-021-00151-1. PMID: 34344890; PMCID: PMC8333253.
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A Conversation with Dawn Pratt: Her Mission to Indigenize STEM.

ACS central science

Harwitz E.
PMID: 34841050
ACS Cent Sci. 2021 Nov 24;7(11):1773-1775. doi: 10.1021/acscentsci.1c01275. Epub 2021 Oct 28.

No abstract available.

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Harwitz E. A Conversation with Dawn Pratt: Her Mission to Indigenize STEM. ACS Cent Sci. 2021;7(11):1773-1775doi: 10.1021/acscentsci.1c01275.
Harwitz, E. (2021). A Conversation with Dawn Pratt: Her Mission to Indigenize STEM. ACS central science, 7(11), 1773-1775. https://doi.org/10.1021/acscentsci.1c01275
Harwitz, Emily. "A Conversation with Dawn Pratt: Her Mission to Indigenize STEM." ACS central science vol. 7,11 (2021): 1773-1775. doi: https://doi.org/10.1021/acscentsci.1c01275
Harwitz E. A Conversation with Dawn Pratt: Her Mission to Indigenize STEM. ACS Cent Sci. 2021 Nov 24;7(11):1773-1775. doi: 10.1021/acscentsci.1c01275. Epub 2021 Oct 28. PMID: 34841050; PMCID: PMC8614100.
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A study protocol for a randomised controlled feasibility trial of an intervention to increase activity and reduce sedentary behaviour in people with severe mental illness: Walking fOR Health (WORtH) Study.

Pilot and feasibility studies

McDonough SM, Howes SC, Dillon M, McAuley J, Brady J, Clarke M, Clarke M, Lait E, McArdle D, O'Neill T, Wilson I, Niven A, Williams J, Tully MA, Murphy MH, McDonough CM.
PMID: 34782018
Pilot Feasibility Stud. 2021 Nov 15;7(1):205. doi: 10.1186/s40814-021-00938-5.

BACKGROUND: People with severe mental illness (SMI) are less physically active and more sedentary than healthy controls, contributing to poorer physical health outcomes in this population. There is a need to understand the feasibility and acceptability, and explore the...

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McDonough SM, Howes SC, Dillon M, et al. A study protocol for a randomised controlled feasibility trial of an intervention to increase activity and reduce sedentary behaviour in people with severe mental illness: Walking fOR Health (WORtH) Study. Pilot Feasibility Stud. 2021;7(1):205doi: 10.1186/s40814-021-00938-5.
McDonough, S. M., Howes, S. C., Dillon, M., McAuley, J., Brady, J., Clarke, M., Clarke, M., Lait, E., McArdle, D., O'Neill, T., Wilson, I., Niven, A., Williams, J., Tully, M. A., Murphy, M. H., & McDonough, C. M. (2021). A study protocol for a randomised controlled feasibility trial of an intervention to increase activity and reduce sedentary behaviour in people with severe mental illness: Walking fOR Health (WORtH) Study. Pilot and feasibility studies, 7(1), 205. https://doi.org/10.1186/s40814-021-00938-5
McDonough, Suzanne M, et al. "A study protocol for a randomised controlled feasibility trial of an intervention to increase activity and reduce sedentary behaviour in people with severe mental illness: Walking fOR Health (WORtH) Study." Pilot and feasibility studies vol. 7,1 (2021): 205. doi: https://doi.org/10.1186/s40814-021-00938-5
McDonough SM, Howes SC, Dillon M, McAuley J, Brady J, Clarke M, Clarke M, Lait E, McArdle D, O'Neill T, Wilson I, Niven A, Williams J, Tully MA, Murphy MH, McDonough CM. A study protocol for a randomised controlled feasibility trial of an intervention to increase activity and reduce sedentary behaviour in people with severe mental illness: Walking fOR Health (WORtH) Study. Pilot Feasibility Stud. 2021 Nov 15;7(1):205. doi: 10.1186/s40814-021-00938-5. PMID: 34782018; PMCID: PMC8590967.
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BTK operates a phospho-tyrosine switch to regulate NLRP3 inflammasome activity.

The Journal of experimental medicine

Bittner ZA, Liu X, Mateo Tortola M, Tapia-Abellán A, Shankar S, Andreeva L, Mangan M, Spalinger M, Kalbacher H, Düwell P, Lovotti M, Bosch K, Dickhöfer S, Marcu A, Stevanović S, Herster F, Cardona Gloria Y, Chang TH, Bork F, Greve CL, Löffler MW, Wolz OO, Schilling NA, Kümmerle-Deschner JB, Wagner S, Delor A, Grimbacher B, Hantschel O, Scharl M, Wu H, Latz E, Weber ANR.
PMID: 34554188
J Exp Med. 2021 Nov 01;218(11). doi: 10.1084/jem.20201656. Epub 2021 Sep 23.

Activity of the NLRP3 inflammasome, a critical mediator of inflammation, is controlled by accessory proteins, posttranslational modifications, cellular localization, and oligomerization. How these factors relate is unclear. We show that a well-established drug target, Bruton's tyrosine kinase (BTK), affects...

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Bittner ZA, Liu X, Mateo Tortola M, et al. BTK operates a phospho-tyrosine switch to regulate NLRP3 inflammasome activity. J Exp Med. 2021;218(11)doi: 10.1084/jem.20201656.
Bittner, Z. A., Liu, X., Mateo Tortola, M., Tapia-Abellán, A., Shankar, S., Andreeva, L., Mangan, M., Spalinger, M., Kalbacher, H., Düwell, P., Lovotti, M., Bosch, K., Dickhöfer, S., Marcu, A., Stevanović, S., Herster, F., Cardona Gloria, Y., Chang, T. H., Bork, F., Greve, C. L., Löffler, M. W., Wolz, O. O., Schilling, N. A., Kümmerle-Deschner, J. B., Wagner, S., Delor, A., Grimbacher, B., Hantschel, O., Scharl, M., Wu, H., Latz, E., & Weber, A. N. R. (2021). BTK operates a phospho-tyrosine switch to regulate NLRP3 inflammasome activity. The Journal of experimental medicine, 218(11), . https://doi.org/10.1084/jem.20201656
Bittner, Zsófia Agnes, et al. "BTK operates a phospho-tyrosine switch to regulate NLRP3 inflammasome activity." The Journal of experimental medicine vol. 218,11 (2021). doi: https://doi.org/10.1084/jem.20201656
Bittner ZA, Liu X, Mateo Tortola M, Tapia-Abellán A, Shankar S, Andreeva L, Mangan M, Spalinger M, Kalbacher H, Düwell P, Lovotti M, Bosch K, Dickhöfer S, Marcu A, Stevanović S, Herster F, Cardona Gloria Y, Chang TH, Bork F, Greve CL, Löffler MW, Wolz OO, Schilling NA, Kümmerle-Deschner JB, Wagner S, Delor A, Grimbacher B, Hantschel O, Scharl M, Wu H, Latz E, Weber ANR. BTK operates a phospho-tyrosine switch to regulate NLRP3 inflammasome activity. J Exp Med. 2021 Nov 01;218(11). doi: 10.1084/jem.20201656. Epub 2021 Sep 23. PMID: 34554188; PMCID: PMC8480672.
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Microglia jointly degrade fibrillar alpha-synuclein cargo by distribution through tunneling nanotubes.

Cell

Scheiblich H, Dansokho C, Mercan D, Schmidt SV, Bousset L, Wischhof L, Eikens F, Odainic A, Spitzer J, Griep A, Schwartz S, Bano D, Latz E, Melki R, Heneka MT.
PMID: 34555357
Cell. 2021 Sep 30;184(20):5089-5106.e21. doi: 10.1016/j.cell.2021.09.007. Epub 2021 Sep 22.

Microglia are the CNS resident immune cells that react to misfolded proteins through pattern recognition receptor ligation and activation of inflammatory pathways. Here, we studied how microglia handle and cope with α-synuclein (α-syn) fibrils and their clearance. We found...

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Scheiblich H, Dansokho C, Mercan D, et al. Microglia jointly degrade fibrillar alpha-synuclein cargo by distribution through tunneling nanotubes. Cell. 2021;184(20):5089-5106.e21doi: 10.1016/j.cell.2021.09.007.
Scheiblich, H., Dansokho, C., Mercan, D., Schmidt, S. V., Bousset, L., Wischhof, L., Eikens, F., Odainic, A., Spitzer, J., Griep, A., Schwartz, S., Bano, D., Latz, E., Melki, R., & Heneka, M. T. (2021). Microglia jointly degrade fibrillar alpha-synuclein cargo by distribution through tunneling nanotubes. Cell, 184(20), 5089-5106.e21. https://doi.org/10.1016/j.cell.2021.09.007
Scheiblich, Hannah, et al. "Microglia jointly degrade fibrillar alpha-synuclein cargo by distribution through tunneling nanotubes." Cell vol. 184,20 (2021): 5089-5106.e21. doi: https://doi.org/10.1016/j.cell.2021.09.007
Scheiblich H, Dansokho C, Mercan D, Schmidt SV, Bousset L, Wischhof L, Eikens F, Odainic A, Spitzer J, Griep A, Schwartz S, Bano D, Latz E, Melki R, Heneka MT. Microglia jointly degrade fibrillar alpha-synuclein cargo by distribution through tunneling nanotubes. Cell. 2021 Sep 30;184(20):5089-5106.e21. doi: 10.1016/j.cell.2021.09.007. Epub 2021 Sep 22. PMID: 34555357; PMCID: PMC8527836.
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Genomic Epidemiology of MRSA During Incarceration at a Large Inner-City Jail.

Clinical infectious diseases : an official publication of the Infectious Diseases Society of America

Popovich KJ, Thiede SN, Zawitz C, Aroutcheva A, Payne D, Janda W, Schoeny M, Green SJ, Snitkin ES, Weinstein RA.
PMID: 33395473
Clin Infect Dis. 2021 Dec 06;73(11):e3708-e3717. doi: 10.1093/cid/ciaa1937.

BACKGROUND: Congregate settings, such as jails, may be a location where colonized detainees transmit methicillin-resistant Staphylococcus aureus (MRSA). We examined MRSA acquisition during incarceration and characterized the genomic epidemiology of MRSA entering the jail and isolated during incarceration.METHODS: Males...

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Popovich KJ, Thiede SN, Zawitz C, et al. Genomic Epidemiology of MRSA During Incarceration at a Large Inner-City Jail. Clin Infect Dis. 2021;73(11):e3708-e3717doi: 10.1093/cid/ciaa1937.
Popovich, K. J., Thiede, S. N., Zawitz, C., Aroutcheva, A., Payne, D., Janda, W., Schoeny, M., Green, S. J., Snitkin, E. S., & Weinstein, R. A. (2021). Genomic Epidemiology of MRSA During Incarceration at a Large Inner-City Jail. Clinical infectious diseases : an official publication of the Infectious Diseases Society of America, 73(11), e3708-e3717. https://doi.org/10.1093/cid/ciaa1937
Popovich, Kyle J, et al. "Genomic Epidemiology of MRSA During Incarceration at a Large Inner-City Jail." Clinical infectious diseases : an official publication of the Infectious Diseases Society of America vol. 73,11 (2021): e3708-e3717. doi: https://doi.org/10.1093/cid/ciaa1937
Popovich KJ, Thiede SN, Zawitz C, Aroutcheva A, Payne D, Janda W, Schoeny M, Green SJ, Snitkin ES, Weinstein RA. Genomic Epidemiology of MRSA During Incarceration at a Large Inner-City Jail. Clin Infect Dis. 2021 Dec 06;73(11):e3708-e3717. doi: 10.1093/cid/ciaa1937. PMID: 33395473; PMCID: PMC8664465.
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Transcriptional Profiling of the Adult Hair Follicle Mesenchyme Reveals R-spondin as a Novel Regulator of Dermal Progenitor Function.

iScience

Hagner A, Shin W, Sinha S, Alpaugh W, Workentine M, Abbasi S, Rahmani W, Agabalyan N, Sharma N, Sparks H, Yoon J, Labit E, Cobb J, Dobrinski I, Biernaskie J.
PMID: 32289736
iScience. 2020 Apr 24;23(4):101019. doi: 10.1016/j.isci.2020.101019. Epub 2020 Apr 02.

The adult hair follicle (HF) undergoes successive regeneration driven by resident epithelial stem cells and neighboring mesenchyme. Recent work described the existence of HF dermal stem cells (hfDSCs), but the genetic regulation of hfDSCs and their daughter cell lineages...

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Hagner A, Shin W, Sinha S, et al. Transcriptional Profiling of the Adult Hair Follicle Mesenchyme Reveals R-spondin as a Novel Regulator of Dermal Progenitor Function. iScience. 2020;23(4):101019doi: 10.1016/j.isci.2020.101019.
Hagner, A., Shin, W., Sinha, S., Alpaugh, W., Workentine, M., Abbasi, S., Rahmani, W., Agabalyan, N., Sharma, N., Sparks, H., Yoon, J., Labit, E., Cobb, J., Dobrinski, I., & Biernaskie, J. (2020). Transcriptional Profiling of the Adult Hair Follicle Mesenchyme Reveals R-spondin as a Novel Regulator of Dermal Progenitor Function. iScience, 23(4), 101019. https://doi.org/10.1016/j.isci.2020.101019
Hagner, Andrew, et al. "Transcriptional Profiling of the Adult Hair Follicle Mesenchyme Reveals R-spondin as a Novel Regulator of Dermal Progenitor Function." iScience vol. 23,4 (2020): 101019. doi: https://doi.org/10.1016/j.isci.2020.101019
Hagner A, Shin W, Sinha S, Alpaugh W, Workentine M, Abbasi S, Rahmani W, Agabalyan N, Sharma N, Sparks H, Yoon J, Labit E, Cobb J, Dobrinski I, Biernaskie J. Transcriptional Profiling of the Adult Hair Follicle Mesenchyme Reveals R-spondin as a Novel Regulator of Dermal Progenitor Function. iScience. 2020 Apr 24;23(4):101019. doi: 10.1016/j.isci.2020.101019. Epub 2020 Apr 02. PMID: 32289736; PMCID: PMC7155209.
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Skewed endosomal RNA responses from TLR7 to TLR3 in RNase T2-deficient macrophages.

International immunology

Liu K, Sato R, Shibata T, Hiranuma R, Reuter T, Fukui R, Zhang Y, Ichinohe T, Ozawa M, Yoshida N, Latz E, Miyake K.
PMID: 34161582
Int Immunol. 2021 Aug 23;33(9):479-490. doi: 10.1093/intimm/dxab033.

RNase T2, a ubiquitously expressed RNase, degrades RNAs in the endosomal compartments. RNA sensors, double-stranded RNA (dsRNA)-sensing Toll-like receptor 3 (TLR3) and single-stranded RNA (ssRNA)-sensing TLR7, are localized in the endosomal compartment in mouse macrophages. We here studied the...

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Liu K, Sato R, Shibata T, et al. Skewed endosomal RNA responses from TLR7 to TLR3 in RNase T2-deficient macrophages. Int Immunol. 2021;33(9):479-490doi: 10.1093/intimm/dxab033.
Liu, K., Sato, R., Shibata, T., Hiranuma, R., Reuter, T., Fukui, R., Zhang, Y., Ichinohe, T., Ozawa, M., Yoshida, N., Latz, E., & Miyake, K. (2021). Skewed endosomal RNA responses from TLR7 to TLR3 in RNase T2-deficient macrophages. International immunology, 33(9), 479-490. https://doi.org/10.1093/intimm/dxab033
Liu, Kaiwen, et al. "Skewed endosomal RNA responses from TLR7 to TLR3 in RNase T2-deficient macrophages." International immunology vol. 33,9 (2021): 479-490. doi: https://doi.org/10.1093/intimm/dxab033
Liu K, Sato R, Shibata T, Hiranuma R, Reuter T, Fukui R, Zhang Y, Ichinohe T, Ozawa M, Yoshida N, Latz E, Miyake K. Skewed endosomal RNA responses from TLR7 to TLR3 in RNase T2-deficient macrophages. Int Immunol. 2021 Aug 23;33(9):479-490. doi: 10.1093/intimm/dxab033. PMID: 34161582.
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The role of follicular anti-Mullerian hormone in women undergoing IVF/ICSI with regard to size, oocyte presence, sociodemographic parameters, treatment variables, and other hormones and vitamins.

Gynecological endocrinology : the official journal of the International Society of Gynecological Endocrinology

Witzke L, Leser S, Nesbigall M, Pfefferle PI, Wagner U, Ziller V.
PMID: 34519591
Gynecol Endocrinol. 2021 Sep 14;1-6. doi: 10.1080/09513590.2021.1967313. Epub 2021 Sep 14.

AIMS: The aim of this study was to evaluate the relationship between follicular anti-Mullerian hormone (fAMH) regarding follicle size, the presence of an oocyte, sociodemographic parameters, and other hormones and vitamins in follicular fluid.MATERIALS AND METHODS: This prospective exploratory...

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Witzke L, Leser S, Nesbigall M, et al. The role of follicular anti-Mullerian hormone in women undergoing IVF/ICSI with regard to size, oocyte presence, sociodemographic parameters, treatment variables, and other hormones and vitamins. Gynecol Endocrinol. 2021;1-6doi: 10.1080/09513590.2021.1967313.
Witzke, L., Leser, S., Nesbigall, M., Pfefferle, P. I., Wagner, U., & Ziller, V. (2021). The role of follicular anti-Mullerian hormone in women undergoing IVF/ICSI with regard to size, oocyte presence, sociodemographic parameters, treatment variables, and other hormones and vitamins. Gynecological endocrinology : the official journal of the International Society of Gynecological Endocrinology, 1-6. https://doi.org/10.1080/09513590.2021.1967313
Witzke, L, et al. "The role of follicular anti-Mullerian hormone in women undergoing IVF/ICSI with regard to size, oocyte presence, sociodemographic parameters, treatment variables, and other hormones and vitamins." Gynecological endocrinology : the official journal of the International Society of Gynecological Endocrinology vol. (2021): 1-6. doi: https://doi.org/10.1080/09513590.2021.1967313
Witzke L, Leser S, Nesbigall M, Pfefferle PI, Wagner U, Ziller V. The role of follicular anti-Mullerian hormone in women undergoing IVF/ICSI with regard to size, oocyte presence, sociodemographic parameters, treatment variables, and other hormones and vitamins. Gynecol Endocrinol. 2021 Sep 14;1-6. doi: 10.1080/09513590.2021.1967313. Epub 2021 Sep 14. PMID: 34519591.
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The Specific NLRP3 Antagonist IFM-514 Decreases Fibrosis and Inflammation in Experimental Murine Non-Alcoholic Steatohepatitis.

Frontiers in molecular biosciences

Torres S, Brol MJ, Magdaleno F, Schierwagen R, Uschner FE, Klein S, Ortiz C, Tyc O, Bachtler N, Stunden J, Bertheloot D, Kitanovic A, Sanchez B, Schrum J, Roush WR, Franchi L, Byth K, Latz E, Trebicka J.
PMID: 34513923
Front Mol Biosci. 2021 Aug 13;8:715765. doi: 10.3389/fmolb.2021.715765. eCollection 2021.

No abstract available.

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Torres S, Brol MJ, Magdaleno F, et al. The Specific NLRP3 Antagonist IFM-514 Decreases Fibrosis and Inflammation in Experimental Murine Non-Alcoholic Steatohepatitis. Front Mol Biosci. 2021;8:715765doi: 10.3389/fmolb.2021.715765.
Torres, S., Brol, M. J., Magdaleno, F., Schierwagen, R., Uschner, F. E., Klein, S., Ortiz, C., Tyc, O., Bachtler, N., Stunden, J., Bertheloot, D., Kitanovic, A., Sanchez, B., Schrum, J., Roush, W. R., Franchi, L., Byth, K., Latz, E., & Trebicka, J. (2021). The Specific NLRP3 Antagonist IFM-514 Decreases Fibrosis and Inflammation in Experimental Murine Non-Alcoholic Steatohepatitis. Frontiers in molecular biosciences, 8715765. https://doi.org/10.3389/fmolb.2021.715765
Torres, Sandra, et al. "The Specific NLRP3 Antagonist IFM-514 Decreases Fibrosis and Inflammation in Experimental Murine Non-Alcoholic Steatohepatitis." Frontiers in molecular biosciences vol. 8 (2021): 715765. doi: https://doi.org/10.3389/fmolb.2021.715765
Torres S, Brol MJ, Magdaleno F, Schierwagen R, Uschner FE, Klein S, Ortiz C, Tyc O, Bachtler N, Stunden J, Bertheloot D, Kitanovic A, Sanchez B, Schrum J, Roush WR, Franchi L, Byth K, Latz E, Trebicka J. The Specific NLRP3 Antagonist IFM-514 Decreases Fibrosis and Inflammation in Experimental Murine Non-Alcoholic Steatohepatitis. Front Mol Biosci. 2021 Aug 13;8:715765. doi: 10.3389/fmolb.2021.715765. eCollection 2021. PMID: 34513923; PMCID: PMC8425476.
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