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Showing 9985 to 9996 of 25165 entries
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Multi-omic analysis of selectively vulnerable motor neuron subtypes implicates altered lipid metabolism in ALS.

Nature neuroscience

Lee H, Lee JJ, Park NY, Dubey SK, Kim T, Ruan K, Lim SB, Park SH, Ha S, Kovlyagina I, Kim KT, Kim S, Oh Y, Kim H, Kang SU, Song MR, Lloyd TE, Maragakis NJ, Hong YB, Eoh H, Lee G.
PMID: 34782793
Nat Neurosci. 2021 Dec;24(12):1673-1685. doi: 10.1038/s41593-021-00944-z. Epub 2021 Nov 15.

Amyotrophic lateral sclerosis (ALS) is a devastating disorder in which motor neurons degenerate, the causes of which remain unclear. In particular, the basis for selective vulnerability of spinal motor neurons (sMNs) and resistance of ocular motor neurons to degeneration...

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Lee H, Lee JJ, Park NY, et al. Multi-omic analysis of selectively vulnerable motor neuron subtypes implicates altered lipid metabolism in ALS. Nat Neurosci. 2021;24(12):1673-1685doi: 10.1038/s41593-021-00944-z.
Lee, H., Lee, J. J., Park, N. Y., Dubey, S. K., Kim, T., Ruan, K., Lim, S. B., Park, S. H., Ha, S., Kovlyagina, I., Kim, K. T., Kim, S., Oh, Y., Kim, H., Kang, S. U., Song, M. R., Lloyd, T. E., Maragakis, N. J., Hong, Y. B., Eoh, H., & Lee, G. (2021). Multi-omic analysis of selectively vulnerable motor neuron subtypes implicates altered lipid metabolism in ALS. Nature neuroscience, 24(12), 1673-1685. https://doi.org/10.1038/s41593-021-00944-z
Lee, Hojae, et al. "Multi-omic analysis of selectively vulnerable motor neuron subtypes implicates altered lipid metabolism in ALS." Nature neuroscience vol. 24,12 (2021): 1673-1685. doi: https://doi.org/10.1038/s41593-021-00944-z
Lee H, Lee JJ, Park NY, Dubey SK, Kim T, Ruan K, Lim SB, Park SH, Ha S, Kovlyagina I, Kim KT, Kim S, Oh Y, Kim H, Kang SU, Song MR, Lloyd TE, Maragakis NJ, Hong YB, Eoh H, Lee G. Multi-omic analysis of selectively vulnerable motor neuron subtypes implicates altered lipid metabolism in ALS. Nat Neurosci. 2021 Dec;24(12):1673-1685. doi: 10.1038/s41593-021-00944-z. Epub 2021 Nov 15. PMID: 34782793; PMCID: PMC8639773.
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IL13Rα2 Is Involved in the Progress of Renal Cell Carcinoma through the JAK2/FOXO3 Pathway.

Journal of personalized medicine

Kang MA, Lee J, Lee CM, Park HS, Jang KY, Park SH.
PMID: 33917914
J Pers Med. 2021 Apr 08;11(4). doi: 10.3390/jpm11040284.

Previously, we reported a close relationship between type II IL4Rα and IL13Rα1 complex and poor outcomes in renal cell carcinoma (RCC). In this study, we investigated the clinicopathologically significant oncogenic role of IL13Rα2, a kind of the independent receptor...

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Kang MA, Lee J, Lee CM, et al. IL13Rα2 Is Involved in the Progress of Renal Cell Carcinoma through the JAK2/FOXO3 Pathway. J Pers Med. 2021;11(4)doi: 10.3390/jpm11040284.
Kang, M. A., Lee, J., Lee, C. M., Park, H. S., Jang, K. Y., & Park, S. H. (2021). IL13Rα2 Is Involved in the Progress of Renal Cell Carcinoma through the JAK2/FOXO3 Pathway. Journal of personalized medicine, 11(4), . https://doi.org/10.3390/jpm11040284
Kang, Mi-Ae, et al. "IL13Rα2 Is Involved in the Progress of Renal Cell Carcinoma through the JAK2/FOXO3 Pathway." Journal of personalized medicine vol. 11,4 (2021). doi: https://doi.org/10.3390/jpm11040284
Kang MA, Lee J, Lee CM, Park HS, Jang KY, Park SH. IL13Rα2 Is Involved in the Progress of Renal Cell Carcinoma through the JAK2/FOXO3 Pathway. J Pers Med. 2021 Apr 08;11(4). doi: 10.3390/jpm11040284. PMID: 33917914; PMCID: PMC8068290.
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Runx3 regulates iron metabolism via modulation of BMP signalling.

Cell proliferation

Kim HY, Lee JM, Lee YS, Li S, Lee SJ, Bae SC, Jung HS.
PMID: 34611951
Cell Prolif. 2021 Dec;54(12):e13138. doi: 10.1111/cpr.13138. Epub 2021 Oct 06.

OBJECTIVES: Runx3, a member of the Runx family of transcription factors, has been studied as a tumour suppressor and key player of organ development. In a previous study, we reported differentiation failure and excessive angiogenesis in the liver of...

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Kim HY, Lee JM, Lee YS, et al. Runx3 regulates iron metabolism via modulation of BMP signalling. Cell Prolif. 2021;54(12):e13138doi: 10.1111/cpr.13138.
Kim, H. Y., Lee, J. M., Lee, Y. S., Li, S., Lee, S. J., Bae, S. C., & Jung, H. S. (2021). Runx3 regulates iron metabolism via modulation of BMP signalling. Cell proliferation, 54(12), e13138. https://doi.org/10.1111/cpr.13138
Kim, Hyun-Yi, et al. "Runx3 regulates iron metabolism via modulation of BMP signalling." Cell proliferation vol. 54,12 (2021): e13138. doi: https://doi.org/10.1111/cpr.13138
Kim HY, Lee JM, Lee YS, Li S, Lee SJ, Bae SC, Jung HS. Runx3 regulates iron metabolism via modulation of BMP signalling. Cell Prolif. 2021 Dec;54(12):e13138. doi: 10.1111/cpr.13138. Epub 2021 Oct 06. PMID: 34611951; PMCID: PMC8666273.
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Lipid-Based Nanoparticles in the Clinic and Clinical Trials: From Cancer Nanomedicine to COVID-19 Vaccines.

Vaccines

Thi TTH, Suys EJA, Lee JS, Nguyen DH, Park KD, Truong NP.
PMID: 33918072
Vaccines (Basel). 2021 Apr 08;9(4). doi: 10.3390/vaccines9040359.

COVID-19 vaccines have been developed with unprecedented speed which would not have been possible without decades of fundamental research on delivery nanotechnology. Lipid-based nanoparticles have played a pivotal role in the successes of COVID-19 vaccines and many other nanomedicines,...

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Thi TTH, Suys EJA, Lee JS, et al. Lipid-Based Nanoparticles in the Clinic and Clinical Trials: From Cancer Nanomedicine to COVID-19 Vaccines. Vaccines (Basel). 2021;9(4)doi: 10.3390/vaccines9040359.
Thi, T. T. H., Suys, E. J. A., Lee, J. S., Nguyen, D. H., Park, K. D., & Truong, N. P. (2021). Lipid-Based Nanoparticles in the Clinic and Clinical Trials: From Cancer Nanomedicine to COVID-19 Vaccines. Vaccines, 9(4), . https://doi.org/10.3390/vaccines9040359
Thi, Thai Thanh Hoang, et al. "Lipid-Based Nanoparticles in the Clinic and Clinical Trials: From Cancer Nanomedicine to COVID-19 Vaccines." Vaccines vol. 9,4 (2021). doi: https://doi.org/10.3390/vaccines9040359
Thi TTH, Suys EJA, Lee JS, Nguyen DH, Park KD, Truong NP. Lipid-Based Nanoparticles in the Clinic and Clinical Trials: From Cancer Nanomedicine to COVID-19 Vaccines. Vaccines (Basel). 2021 Apr 08;9(4). doi: 10.3390/vaccines9040359. PMID: 33918072; PMCID: PMC8069344.
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Complete chloroplast genome of .

Mitochondrial DNA. Part B, Resources

Kim SC, Shin S, Ahn JY, Lee JW.
PMID: 33365690
Mitochondrial DNA B Resour. 2019 Jul 23;4(2):2700-2701. doi: 10.1080/23802359.2019.1644223.

The complete plastid genome of

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Kim SC, Shin S, Ahn JY, et al. Complete chloroplast genome of . Mitochondrial DNA B Resour. 2019;4(2):2700-2701doi: 10.1080/23802359.2019.1644223.
Kim, S. C., Shin, S., Ahn, J. Y., & Lee, J. W. (2019). Complete chloroplast genome of . Mitochondrial DNA. Part B, Resources, 4(2), 2700-2701. https://doi.org/10.1080/23802359.2019.1644223
Kim, Sang-Chul, et al. "Complete chloroplast genome of ." Mitochondrial DNA. Part B, Resources vol. 4,2 (2019): 2700-2701. doi: https://doi.org/10.1080/23802359.2019.1644223
Kim SC, Shin S, Ahn JY, Lee JW. Complete chloroplast genome of . Mitochondrial DNA B Resour. 2019 Jul 23;4(2):2700-2701. doi: 10.1080/23802359.2019.1644223. PMID: 33365690; PMCID: PMC7706852.
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Fibromyalgia in Axial Spondyloarthritis: A Meta-analysis.

Journal of clinical rheumatology : practical reports on rheumatic & musculoskeletal diseases

Son SM, Kim DS, Lee JS.
PMID: 33298817
J Clin Rheumatol. 2022 Jan 01;28(1):e222-e227. doi: 10.1097/RHU.0000000000001612.

OBJECTIVES: This study aimed to compare the demographic characteristics, disease activity, functional status, and quality of life between patients with axial spondyloarthritis and fibromyalgia and patients with axial spondyloarthritis without fibromyalgia.METHODS: We searched MEDLINE via PubMed, Cochrane, Scopus, and...

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Son SM, Kim DS, Lee JS. Fibromyalgia in Axial Spondyloarthritis: A Meta-analysis. J Clin Rheumatol. 2022;28(1):e222-e227doi: 10.1097/RHU.0000000000001612.
Son, S. M., Kim, D. S., & Lee, J. S. (2022). Fibromyalgia in Axial Spondyloarthritis: A Meta-analysis. Journal of clinical rheumatology : practical reports on rheumatic & musculoskeletal diseases, 28(1), e222-e227. https://doi.org/10.1097/RHU.0000000000001612
Son, Seung Min, et al. "Fibromyalgia in Axial Spondyloarthritis: A Meta-analysis." Journal of clinical rheumatology : practical reports on rheumatic & musculoskeletal diseases vol. 28,1 (2022): e222-e227. doi: https://doi.org/10.1097/RHU.0000000000001612
Son SM, Kim DS, Lee JS. Fibromyalgia in Axial Spondyloarthritis: A Meta-analysis. J Clin Rheumatol. 2022 Jan 01;28(1):e222-e227. doi: 10.1097/RHU.0000000000001612. PMID: 33298817.
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Performance Assessment of the Post-Tensioned Anchorage Zone Using High-Strength Concrete Considering Confinement Effect.

Materials (Basel, Switzerland)

Lee JS, Woo BH, Ryou JS, Kim JS.
PMID: 33918152
Materials (Basel). 2021 Apr 02;14(7). doi: 10.3390/ma14071748.

Post-tensioned anchorage zones need enough strength to resist large forces from jacking forces from prestress and need spiral reinforcement to give confinement effect. High-strength concrete (HSC) has high-strength and brings the advantage of reducing material using and simplifying reinforcing....

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Lee JS, Woo BH, Ryou JS, et al. Performance Assessment of the Post-Tensioned Anchorage Zone Using High-Strength Concrete Considering Confinement Effect. Materials (Basel). 2021;14(7)doi: 10.3390/ma14071748.
Lee, J. S., Woo, B. H., Ryou, J. S., & Kim, J. S. (2021). Performance Assessment of the Post-Tensioned Anchorage Zone Using High-Strength Concrete Considering Confinement Effect. Materials (Basel, Switzerland), 14(7), . https://doi.org/10.3390/ma14071748
Lee, Jun Suk, et al. "Performance Assessment of the Post-Tensioned Anchorage Zone Using High-Strength Concrete Considering Confinement Effect." Materials (Basel, Switzerland) vol. 14,7 (2021). doi: https://doi.org/10.3390/ma14071748
Lee JS, Woo BH, Ryou JS, Kim JS. Performance Assessment of the Post-Tensioned Anchorage Zone Using High-Strength Concrete Considering Confinement Effect. Materials (Basel). 2021 Apr 02;14(7). doi: 10.3390/ma14071748. PMID: 33918152; PMCID: PMC8038034.
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Complete mitochondrial genome of the marine mysid .

Mitochondrial DNA. Part B, Resources

Choi BS, Lee YH, Hwang DS, Ma CW, Hagiwara A, Lee JS.
PMID: 33365999
Mitochondrial DNA B Resour. 2019 Oct 16;4(2):3372-3373. doi: 10.1080/23802359.2019.1673238.

The complete mitochondrial genome was sequenced from the marine mysid

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Choi BS, Lee YH, Hwang DS, et al. Complete mitochondrial genome of the marine mysid . Mitochondrial DNA B Resour. 2019;4(2):3372-3373doi: 10.1080/23802359.2019.1673238.
Choi, B. S., Lee, Y. H., Hwang, D. S., Ma, C. W., Hagiwara, A., & Lee, J. S. (2019). Complete mitochondrial genome of the marine mysid . Mitochondrial DNA. Part B, Resources, 4(2), 3372-3373. https://doi.org/10.1080/23802359.2019.1673238
Choi, Beom-Soon, et al. "Complete mitochondrial genome of the marine mysid ." Mitochondrial DNA. Part B, Resources vol. 4,2 (2019): 3372-3373. doi: https://doi.org/10.1080/23802359.2019.1673238
Choi BS, Lee YH, Hwang DS, Ma CW, Hagiwara A, Lee JS. Complete mitochondrial genome of the marine mysid . Mitochondrial DNA B Resour. 2019 Oct 16;4(2):3372-3373. doi: 10.1080/23802359.2019.1673238. PMID: 33365999; PMCID: PMC7707204.
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The complete mitogenome of the Arctic moss .

Mitochondrial DNA. Part B, Resources

Kang P, Cho SM, Lee J, Yim JH, Lee JH, Lee H.
PMID: 33366032
Mitochondrial DNA B Resour. 2019 Oct 09;4(2):3446-3447. doi: 10.1080/23802359.2019.1674204.

The Arctic moss

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Kang P, Cho SM, Lee J, et al. The complete mitogenome of the Arctic moss . Mitochondrial DNA B Resour. 2019;4(2):3446-3447doi: 10.1080/23802359.2019.1674204.
Kang, P., Cho, S. M., Lee, J., Yim, J. H., Lee, J. H., & Lee, H. (2019). The complete mitogenome of the Arctic moss . Mitochondrial DNA. Part B, Resources, 4(2), 3446-3447. https://doi.org/10.1080/23802359.2019.1674204
Kang, Pilsung, et al. "The complete mitogenome of the Arctic moss ." Mitochondrial DNA. Part B, Resources vol. 4,2 (2019): 3446-3447. doi: https://doi.org/10.1080/23802359.2019.1674204
Kang P, Cho SM, Lee J, Yim JH, Lee JH, Lee H. The complete mitogenome of the Arctic moss . Mitochondrial DNA B Resour. 2019 Oct 09;4(2):3446-3447. doi: 10.1080/23802359.2019.1674204. PMID: 33366032; PMCID: PMC7707293.
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Prevalence and factors associated with suicide ideation and psychiatric morbidity among inpatients of a general hospital: A consecutive three-year study.

The Kaohsiung journal of medical sciences

Lee JI, Burdick KE, Ko CH, Liu TL, Lin YC, Lee MB.
PMID: 33336553
Kaohsiung J Med Sci. 2021 May;37(5):427-433. doi: 10.1002/kjm2.12336. Epub 2020 Dec 17.

Using a self-administered questionnaire (the five-item Brief Symptom Rating Scale, BSRS-5), determine the prevalence of suicide ideation (SI), psychiatric morbidity (PM), and the factors associated with these conditions in a general hospital in Taiwan. All individuals aged 12 years...

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Lee JI, Burdick KE, Ko CH, et al. Prevalence and factors associated with suicide ideation and psychiatric morbidity among inpatients of a general hospital: A consecutive three-year study. Kaohsiung J Med Sci. 2020;37(5):427-433doi: 10.1002/kjm2.12336.
Lee, J. I., Burdick, K. E., Ko, C. H., Liu, T. L., Lin, Y. C., & Lee, M. B. (2021). Prevalence and factors associated with suicide ideation and psychiatric morbidity among inpatients of a general hospital: A consecutive three-year study. The Kaohsiung journal of medical sciences, 37(5), 427-433. https://doi.org/10.1002/kjm2.12336
Lee, Jia-In, et al. "Prevalence and factors associated with suicide ideation and psychiatric morbidity among inpatients of a general hospital: A consecutive three-year study." The Kaohsiung journal of medical sciences vol. 37,5 (2021): 427-433. doi: https://doi.org/10.1002/kjm2.12336
Lee JI, Burdick KE, Ko CH, Liu TL, Lin YC, Lee MB. Prevalence and factors associated with suicide ideation and psychiatric morbidity among inpatients of a general hospital: A consecutive three-year study. Kaohsiung J Med Sci. 2021 May;37(5):427-433. doi: 10.1002/kjm2.12336. Epub 2020 Dec 17. PMID: 33336553.
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Targeting the mitochondrial permeability transition pore for neuroprotection in a piglet model of neonatal hypoxic-ischemic encephalopathy.

Journal of neuroscience research

Chen MW, Santos P, Kulikowicz E, Koehler RC, Lee JK, Martin LJ.
PMID: 33675112
J Neurosci Res. 2021 Jun;99(6):1550-1564. doi: 10.1002/jnr.24821. Epub 2021 Mar 05.

Neonatal hypoxic-ischemic encephalopathy (HIE) causes significant morbidity despite treatment with therapeutic hypothermia. Mitochondrial dysfunction may drive the mechanisms underlying neuronal cell death, thereby making mitochondria prime targets for neuroprotection. The mitochondrial permeability transition pore (mPTP) is one such target...

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Chen MW, Santos P, Kulikowicz E, et al. Targeting the mitochondrial permeability transition pore for neuroprotection in a piglet model of neonatal hypoxic-ischemic encephalopathy. J Neurosci Res. 2021;99(6):1550-1564doi: 10.1002/jnr.24821.
Chen, M. W., Santos, P., Kulikowicz, E., Koehler, R. C., Lee, J. K., & Martin, L. J. (2021). Targeting the mitochondrial permeability transition pore for neuroprotection in a piglet model of neonatal hypoxic-ischemic encephalopathy. Journal of neuroscience research, 99(6), 1550-1564. https://doi.org/10.1002/jnr.24821
Chen, May W, et al. "Targeting the mitochondrial permeability transition pore for neuroprotection in a piglet model of neonatal hypoxic-ischemic encephalopathy." Journal of neuroscience research vol. 99,6 (2021): 1550-1564. doi: https://doi.org/10.1002/jnr.24821
Chen MW, Santos P, Kulikowicz E, Koehler RC, Lee JK, Martin LJ. Targeting the mitochondrial permeability transition pore for neuroprotection in a piglet model of neonatal hypoxic-ischemic encephalopathy. J Neurosci Res. 2021 Jun;99(6):1550-1564. doi: 10.1002/jnr.24821. Epub 2021 Mar 05. PMID: 33675112.
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Early hormonal recovery following endoscopic transsphenoidal surgery for silent non-functioning pituitary adenomas with hormone dysfunction.

Journal of neuro-oncology

Lee MH, Hur KY, Hong SD, Seol HJ, Choi JW, Lee JI, Nam DH, Kong DS.
PMID: 34002303
J Neurooncol. 2021 Jun;153(2):343-350. doi: 10.1007/s11060-021-03774-y. Epub 2021 May 17.

PURPOSE: The role of transsphenoidal surgery in the recovery of preexisting hormone dysfunction from pituitary tumors remains controversial. This study aimed to investigate the incidence of hormone dysfunction among asymptomatic non-functioning pituitary adenomas and their recovery following endoscopic transsphenoidal...

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Lee MH, Hur KY, Hong SD, et al. Early hormonal recovery following endoscopic transsphenoidal surgery for silent non-functioning pituitary adenomas with hormone dysfunction. J Neurooncol. 2021;153(2):343-350doi: 10.1007/s11060-021-03774-y.
Lee, M. H., Hur, K. Y., Hong, S. D., Seol, H. J., Choi, J. W., Lee, J. I., Nam, D. H., & Kong, D. S. (2021). Early hormonal recovery following endoscopic transsphenoidal surgery for silent non-functioning pituitary adenomas with hormone dysfunction. Journal of neuro-oncology, 153(2), 343-350. https://doi.org/10.1007/s11060-021-03774-y
Lee, Min Ho, et al. "Early hormonal recovery following endoscopic transsphenoidal surgery for silent non-functioning pituitary adenomas with hormone dysfunction." Journal of neuro-oncology vol. 153,2 (2021): 343-350. doi: https://doi.org/10.1007/s11060-021-03774-y
Lee MH, Hur KY, Hong SD, Seol HJ, Choi JW, Lee JI, Nam DH, Kong DS. Early hormonal recovery following endoscopic transsphenoidal surgery for silent non-functioning pituitary adenomas with hormone dysfunction. J Neurooncol. 2021 Jun;153(2):343-350. doi: 10.1007/s11060-021-03774-y. Epub 2021 May 17. PMID: 34002303.
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Showing 9985 to 9996 of 25165 entries