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Showing 697 to 704 of 704 entries
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Methamphetamine and HIV-Tat Protein Synergistically Induce Oxidative Stress and Blood-Brain Barrier Damage via Transient Receptor Potential Melastatin 2 Channel.

Frontiers in pharmacology

Huang J, Zhang R, Wang S, Zhang D, Leung CK, Yang G, Li Y, Liu L, Xu Y, Lin S, Wang C, Zeng X, Li J.
PMID: 33815104
Front Pharmacol. 2021 Mar 17;12:619436. doi: 10.3389/fphar.2021.619436. eCollection 2021.

Synergistic impairment of the blood-brain barrier (BBB) induced by methamphetamine (METH) and HIV-Tat protein increases the risk of HIV-associated neurocognitive disorders (HAND) in HIV-positive METH abusers. Studies have shown that oxidative stress plays a vital role in METH- and...

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Huang J, Zhang R, Wang S, et al. Methamphetamine and HIV-Tat Protein Synergistically Induce Oxidative Stress and Blood-Brain Barrier Damage via Transient Receptor Potential Melastatin 2 Channel. Front Pharmacol. 2021;12:619436doi: 10.3389/fphar.2021.619436.
Huang, J., Zhang, R., Wang, S., Zhang, D., Leung, C. K., Yang, G., Li, Y., Liu, L., Xu, Y., Lin, S., Wang, C., Zeng, X., & Li, J. (2021). Methamphetamine and HIV-Tat Protein Synergistically Induce Oxidative Stress and Blood-Brain Barrier Damage via Transient Receptor Potential Melastatin 2 Channel. Frontiers in pharmacology, 12619436. https://doi.org/10.3389/fphar.2021.619436
Huang, Jian, et al. "Methamphetamine and HIV-Tat Protein Synergistically Induce Oxidative Stress and Blood-Brain Barrier Damage via Transient Receptor Potential Melastatin 2 Channel." Frontiers in pharmacology vol. 12 (2021): 619436. doi: https://doi.org/10.3389/fphar.2021.619436
Huang J, Zhang R, Wang S, Zhang D, Leung CK, Yang G, Li Y, Liu L, Xu Y, Lin S, Wang C, Zeng X, Li J. Methamphetamine and HIV-Tat Protein Synergistically Induce Oxidative Stress and Blood-Brain Barrier Damage via Transient Receptor Potential Melastatin 2 Channel. Front Pharmacol. 2021 Mar 17;12:619436. doi: 10.3389/fphar.2021.619436. eCollection 2021. PMID: 33815104; PMCID: PMC8010131.
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Constrained Center Loss for Convolutional Neural Networks.

IEEE transactions on neural networks and learning systems

Shi Z, Wang H, Leung CS.
PMID: 34428154
IEEE Trans Neural Netw Learn Syst. 2021 Aug 24; doi: 10.1109/TNNLS.2021.3104392. Epub 2021 Aug 24.

From the feature representation's point of view, the feature learning module of a convolutional neural network (CNN) is to transform an input pattern into a feature vector. This feature vector is then multiplied with a number of output weight...

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Shi Z, Wang H, Leung CS. Constrained Center Loss for Convolutional Neural Networks. IEEE Trans Neural Netw Learn Syst. 2021;doi: 10.1109/TNNLS.2021.3104392.
Shi, Z., Wang, H., & Leung, C. S. (2021). Constrained Center Loss for Convolutional Neural Networks. IEEE transactions on neural networks and learning systems, . https://doi.org/10.1109/TNNLS.2021.3104392
Shi, Zhanglei, et al. "Constrained Center Loss for Convolutional Neural Networks." IEEE transactions on neural networks and learning systems vol. (2021). doi: https://doi.org/10.1109/TNNLS.2021.3104392
Shi Z, Wang H, Leung CS. Constrained Center Loss for Convolutional Neural Networks. IEEE Trans Neural Netw Learn Syst. 2021 Aug 24; doi: 10.1109/TNNLS.2021.3104392. Epub 2021 Aug 24. PMID: 34428154.
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Reaction of the Liver upon Long-Term Treatment of Fluoxetine and Atorvastatin Compared with Alcohol in a Mouse Model.

Journal of toxicology

Chen Z, Chow TCH, Wang S, Leung GCT, Wu SLY, Yew DT.
PMID: 35003254
J Toxicol. 2021 Dec 30;2021:9974969. doi: 10.1155/2021/9974969. eCollection 2021.

BACKGROUND: Alcoholism is known to cause liver toxicity and is extensively researched. On the other hand, stress, depression, and obesity are interrelated conditions with alcoholism, and their medications would affect the liver itself. In this study, we investigated the...

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Chen Z, Chow TCH, Wang S, et al. Reaction of the Liver upon Long-Term Treatment of Fluoxetine and Atorvastatin Compared with Alcohol in a Mouse Model. J Toxicol. 2021;2021:9974969doi: 10.1155/2021/9974969.
Chen, Z., Chow, T. C. H., Wang, S., Leung, G. C. T., Wu, S. L. Y., & Yew, D. T. (2021). Reaction of the Liver upon Long-Term Treatment of Fluoxetine and Atorvastatin Compared with Alcohol in a Mouse Model. Journal of toxicology, 20219974969. https://doi.org/10.1155/2021/9974969
Chen, Zhiliang, et al. "Reaction of the Liver upon Long-Term Treatment of Fluoxetine and Atorvastatin Compared with Alcohol in a Mouse Model." Journal of toxicology vol. 2021 (2021): 9974969. doi: https://doi.org/10.1155/2021/9974969
Chen Z, Chow TCH, Wang S, Leung GCT, Wu SLY, Yew DT. Reaction of the Liver upon Long-Term Treatment of Fluoxetine and Atorvastatin Compared with Alcohol in a Mouse Model. J Toxicol. 2021 Dec 30;2021:9974969. doi: 10.1155/2021/9974969. eCollection 2021. PMID: 35003254; PMCID: PMC8740222.
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The miR-223/nuclear factor I-A axis regulates inflammation and cellular functions in intestinal tissues with necrotizing enterocolitis.

FEBS open bio

Wu YZ, Chan KYY, Leung KT, Lam HS, Tam YH, Lee KH, Li K, Ng PC.
PMID: 33932136
FEBS Open Bio. 2021 Jul;11(7):1907-1920. doi: 10.1002/2211-5463.13164. Epub 2021 Jun 01.

We previously demonstrated that microRNA(miR)-223 is overexpressed in intestinal tissue of infants with necrotizing enterocolitis (NEC). The objective of the current study was to identify the target gene of miR-223 and to investigate the role of the miR-223/nuclear factor...

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Wu YZ, Chan KYY, Leung KT, et al. The miR-223/nuclear factor I-A axis regulates inflammation and cellular functions in intestinal tissues with necrotizing enterocolitis. FEBS Open Bio. 2021;11(7):1907-1920doi: 10.1002/2211-5463.13164.
Wu, Y. Z., Chan, K. Y. Y., Leung, K. T., Lam, H. S., Tam, Y. H., Lee, K. H., Li, K., & Ng, P. C. (2021). The miR-223/nuclear factor I-A axis regulates inflammation and cellular functions in intestinal tissues with necrotizing enterocolitis. FEBS open bio, 11(7), 1907-1920. https://doi.org/10.1002/2211-5463.13164
Wu, Yu Zheng, et al. "The miR-223/nuclear factor I-A axis regulates inflammation and cellular functions in intestinal tissues with necrotizing enterocolitis." FEBS open bio vol. 11,7 (2021): 1907-1920. doi: https://doi.org/10.1002/2211-5463.13164
Wu YZ, Chan KYY, Leung KT, Lam HS, Tam YH, Lee KH, Li K, Ng PC. The miR-223/nuclear factor I-A axis regulates inflammation and cellular functions in intestinal tissues with necrotizing enterocolitis. FEBS Open Bio. 2021 Jul;11(7):1907-1920. doi: 10.1002/2211-5463.13164. Epub 2021 Jun 01. PMID: 33932136; PMCID: PMC8255851.
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Remdesivir induces persistent mitochondrial and structural damage in human induced pluripotent stem cell derived cardiomyocytes.

Cardiovascular research

Kwok M, Lee C, Li HS, Deng R, Tsoi C, Ding Q, Tsang SY, Leung KT, Yan BP, Poon EN.
PMID: 34609482
Cardiovasc Res. 2021 Oct 05; doi: 10.1093/cvr/cvab311. Epub 2021 Oct 05.

AIMS: Remdesivir is a prodrug of an adenosine triphosphate analogue and is currently the only drug formally approved for the treatment of hospitalised COVID-19 patients. Nucleoside/nucleotide analogues have been shown to induce mitochondrial damage and cardiotoxicity, and this may...

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Kwok M, Lee C, Li HS, et al. Remdesivir induces persistent mitochondrial and structural damage in human induced pluripotent stem cell derived cardiomyocytes. Cardiovasc Res. 2021;doi: 10.1093/cvr/cvab311.
Kwok, M., Lee, C., Li, H. S., Deng, R., Tsoi, C., Ding, Q., Tsang, S. Y., Leung, K. T., Yan, B. P., & Poon, E. N. (2021). Remdesivir induces persistent mitochondrial and structural damage in human induced pluripotent stem cell derived cardiomyocytes. Cardiovascular research, . https://doi.org/10.1093/cvr/cvab311
Kwok, Maxwell, et al. "Remdesivir induces persistent mitochondrial and structural damage in human induced pluripotent stem cell derived cardiomyocytes." Cardiovascular research vol. (2021). doi: https://doi.org/10.1093/cvr/cvab311
Kwok M, Lee C, Li HS, Deng R, Tsoi C, Ding Q, Tsang SY, Leung KT, Yan BP, Poon EN. Remdesivir induces persistent mitochondrial and structural damage in human induced pluripotent stem cell derived cardiomyocytes. Cardiovasc Res. 2021 Oct 05; doi: 10.1093/cvr/cvab311. Epub 2021 Oct 05. PMID: 34609482; PMCID: PMC8500104.
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Predicting primary treatment failure using interim FDG-PET scanning in diffuse large B-cell lymphoma.

European journal of haematology

Wight J, Wai SH, Shen E, Lee ST, Berlangieri S, Fancourt T, Hawkes E, Hannah A, Leung T, Chong G.
PMID: 34240453
Eur J Haematol. 2021 Oct;107(4):475-483. doi: 10.1111/ejh.13684. Epub 2021 Jul 22.

Interim FDG-PET (iPET) in diffuse large B-cell lymphoma (DLBCL) is increasingly practised and used in clinical trials to adapt further therapy. However, the optimum timing and methodology of iPET remains controversial. We retrospectively analysed the iPET results and outcomes...

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Wight J, Wai SH, Shen E, et al. Predicting primary treatment failure using interim FDG-PET scanning in diffuse large B-cell lymphoma. Eur J Haematol. 2021;107(4):475-483doi: 10.1111/ejh.13684.
Wight, J., Wai, S. H., Shen, E., Lee, S. T., Berlangieri, S., Fancourt, T., Hawkes, E., Hannah, A., Leung, T., & Chong, G. (2021). Predicting primary treatment failure using interim FDG-PET scanning in diffuse large B-cell lymphoma. European journal of haematology, 107(4), 475-483. https://doi.org/10.1111/ejh.13684
Wight, Joel, et al. "Predicting primary treatment failure using interim FDG-PET scanning in diffuse large B-cell lymphoma." European journal of haematology vol. 107,4 (2021): 475-483. doi: https://doi.org/10.1111/ejh.13684
Wight J, Wai SH, Shen E, Lee ST, Berlangieri S, Fancourt T, Hawkes E, Hannah A, Leung T, Chong G. Predicting primary treatment failure using interim FDG-PET scanning in diffuse large B-cell lymphoma. Eur J Haematol. 2021 Oct;107(4):475-483. doi: 10.1111/ejh.13684. Epub 2021 Jul 22. PMID: 34240453.
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Trunk muscle size and function in volleyball players with and without injuries to the head, neck and upper limb.

Physical therapy in sport : official journal of the Association of Chartered Physiotherapists in Sports Medicine

Hides JA, Leung FT, Watson K, Trojman A, Grantham B, Mendis MD.
PMID: 34922033
Phys Ther Sport. 2021 Dec 13;54:1-7. doi: 10.1016/j.ptsp.2021.12.003. Epub 2021 Dec 13.

OBJECTIVE: To investigate trunk muscle size and function in elite and community volleyball players with and without a history of head, neck or upper limb injury.DESIGN: Cross-sectional observational study.SETTING: Volleyball training camp or training sessions.PARTICIPANTS: 86 volleyball players (elite...

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Hides JA, Leung FT, Watson K, et al. Trunk muscle size and function in volleyball players with and without injuries to the head, neck and upper limb. Phys Ther Sport. 2021;54:1-7doi: 10.1016/j.ptsp.2021.12.003.
Hides, J. A., Leung, F. T., Watson, K., Trojman, A., Grantham, B., & Mendis, M. D. (2021). Trunk muscle size and function in volleyball players with and without injuries to the head, neck and upper limb. Physical therapy in sport : official journal of the Association of Chartered Physiotherapists in Sports Medicine, 541-7. https://doi.org/10.1016/j.ptsp.2021.12.003
Hides, Julie A, et al. "Trunk muscle size and function in volleyball players with and without injuries to the head, neck and upper limb." Physical therapy in sport : official journal of the Association of Chartered Physiotherapists in Sports Medicine vol. 54 (2021): 1-7. doi: https://doi.org/10.1016/j.ptsp.2021.12.003
Hides JA, Leung FT, Watson K, Trojman A, Grantham B, Mendis MD. Trunk muscle size and function in volleyball players with and without injuries to the head, neck and upper limb. Phys Ther Sport. 2021 Dec 13;54:1-7. doi: 10.1016/j.ptsp.2021.12.003. Epub 2021 Dec 13. PMID: 34922033.
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Citicoline Modulates Glaucomatous Neurodegeneration Through Intraocular Pressure-Independent Control.

Neurotherapeutics : the journal of the American Society for Experimental NeuroTherapeutics

van der Merwe Y, Murphy MC, Sims JR, Faiq MA, Yang XL, Ho LC, Conner IP, Yu Y, Leung CK, Wollstein G, Schuman JS, Chan KC.
PMID: 33846961
Neurotherapeutics. 2021 Apr;18(2):1339-1359. doi: 10.1007/s13311-021-01033-6. Epub 2021 Apr 13.

Glaucoma is a neurodegenerative disease that causes progressive, irreversible vision loss. Currently, intraocular pressure (IOP) is the only modifiable risk factor for glaucoma. However, glaucomatous degeneration may continue despite adequate IOP control. Therefore, there exists a need for treatment...

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van der Merwe Y, Murphy MC, Sims JR, et al. Citicoline Modulates Glaucomatous Neurodegeneration Through Intraocular Pressure-Independent Control. Neurotherapeutics. 2021;18(2):1339-1359doi: 10.1007/s13311-021-01033-6.
van der Merwe, Y., Murphy, M. C., Sims, J. R., Faiq, M. A., Yang, X. L., Ho, L. C., Conner, I. P., Yu, Y., Leung, C. K., Wollstein, G., Schuman, J. S., & Chan, K. C. (2021). Citicoline Modulates Glaucomatous Neurodegeneration Through Intraocular Pressure-Independent Control. Neurotherapeutics : the journal of the American Society for Experimental NeuroTherapeutics, 18(2), 1339-1359. https://doi.org/10.1007/s13311-021-01033-6
van der Merwe, Yolandi, et al. "Citicoline Modulates Glaucomatous Neurodegeneration Through Intraocular Pressure-Independent Control." Neurotherapeutics : the journal of the American Society for Experimental NeuroTherapeutics vol. 18,2 (2021): 1339-1359. doi: https://doi.org/10.1007/s13311-021-01033-6
van der Merwe Y, Murphy MC, Sims JR, Faiq MA, Yang XL, Ho LC, Conner IP, Yu Y, Leung CK, Wollstein G, Schuman JS, Chan KC. Citicoline Modulates Glaucomatous Neurodegeneration Through Intraocular Pressure-Independent Control. Neurotherapeutics. 2021 Apr;18(2):1339-1359. doi: 10.1007/s13311-021-01033-6. Epub 2021 Apr 13. PMID: 33846961; PMCID: PMC8423893.
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Showing 697 to 704 of 704 entries