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Clinical and Functional Characteristics of Patients with Unclassifiable Interstitial Lung Disease (uILD): Long-Term Follow-Up Data from European IPF Registry (eurIPFreg).

Journal of clinical medicine

Krauss E, El-Guelai M, Pons-Kuehnemann J, Dartsch RC, Tello S, Korfei M, Mahavadi P, Breithecker A, Fink L, Stoehr M, Majeed RW, Seeger W, Crestani B, Guenther A.
PMID: 32756496
J Clin Med. 2020 Aug 03;9(8). doi: 10.3390/jcm9082499.

(1) Aim of the study: In spite of extensive research, up to 20% of interstitial lung diseases (ILD) patients cannot be safely classified. We analyzed clinical features, progression factors, and outcomes of unclassifiable ILD (uILD). (2) Methods: A total...

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Krauss E, El-Guelai M, Pons-Kuehnemann J, et al. Clinical and Functional Characteristics of Patients with Unclassifiable Interstitial Lung Disease (uILD): Long-Term Follow-Up Data from European IPF Registry (eurIPFreg). J Clin Med. 2020;9(8)doi: 10.3390/jcm9082499.
Krauss, E., El-Guelai, M., Pons-Kuehnemann, J., Dartsch, R. C., Tello, S., Korfei, M., Mahavadi, P., Breithecker, A., Fink, L., Stoehr, M., Majeed, R. W., Seeger, W., Crestani, B., & Guenther, A. (2020). Clinical and Functional Characteristics of Patients with Unclassifiable Interstitial Lung Disease (uILD): Long-Term Follow-Up Data from European IPF Registry (eurIPFreg). Journal of clinical medicine, 9(8), . https://doi.org/10.3390/jcm9082499
Krauss, Ekaterina, et al. "Clinical and Functional Characteristics of Patients with Unclassifiable Interstitial Lung Disease (uILD): Long-Term Follow-Up Data from European IPF Registry (eurIPFreg)." Journal of clinical medicine vol. 9,8 (2020). doi: https://doi.org/10.3390/jcm9082499
Krauss E, El-Guelai M, Pons-Kuehnemann J, Dartsch RC, Tello S, Korfei M, Mahavadi P, Breithecker A, Fink L, Stoehr M, Majeed RW, Seeger W, Crestani B, Guenther A. Clinical and Functional Characteristics of Patients with Unclassifiable Interstitial Lung Disease (uILD): Long-Term Follow-Up Data from European IPF Registry (eurIPFreg). J Clin Med. 2020 Aug 03;9(8). doi: 10.3390/jcm9082499. PMID: 32756496; PMCID: PMC7464480.
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Release of GLP-1 and PYY in response to the activation of G protein-coupled bile acid receptor TGR5 is mediated by Epac/PLC-ε pathway and modulated by endogenous H2S.

Frontiers in physiology

Bala V, Rajagopal S, Kumar DP, Nalli AD, Mahavadi S, Sanyal AJ, Grider JR, Murthy KS.
PMID: 25404917
Front Physiol. 2014 Nov 03;5:420. doi: 10.3389/fphys.2014.00420. eCollection 2014.

Activation of plasma membrane TGR5 receptors in enteroendocrine cells by bile acids is known to regulate gastrointestinal secretion and motility and glucose homeostasis. The endocrine functions of the gut are modulated by microenvironment of the distal gut predominantly by...

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Bala V, Rajagopal S, Kumar DP, et al. Release of GLP-1 and PYY in response to the activation of G protein-coupled bile acid receptor TGR5 is mediated by Epac/PLC-ε pathway and modulated by endogenous H2S. Front Physiol. 2014;5:420doi: 10.3389/fphys.2014.00420.
Bala, V., Rajagopal, S., Kumar, D. P., Nalli, A. D., Mahavadi, S., Sanyal, A. J., Grider, J. R., & Murthy, K. S. (2014). Release of GLP-1 and PYY in response to the activation of G protein-coupled bile acid receptor TGR5 is mediated by Epac/PLC-ε pathway and modulated by endogenous H2S. Frontiers in physiology, 5420. https://doi.org/10.3389/fphys.2014.00420
Bala, Vanitha, et al. "Release of GLP-1 and PYY in response to the activation of G protein-coupled bile acid receptor TGR5 is mediated by Epac/PLC-ε pathway and modulated by endogenous H2S." Frontiers in physiology vol. 5 (2014): 420. doi: https://doi.org/10.3389/fphys.2014.00420
Bala V, Rajagopal S, Kumar DP, Nalli AD, Mahavadi S, Sanyal AJ, Grider JR, Murthy KS. Release of GLP-1 and PYY in response to the activation of G protein-coupled bile acid receptor TGR5 is mediated by Epac/PLC-ε pathway and modulated by endogenous H2S. Front Physiol. 2014 Nov 03;5:420. doi: 10.3389/fphys.2014.00420. eCollection 2014. PMID: 25404917; PMCID: PMC4217307.
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Imatinib alters cell viability but not growth factors levels in TM4 Sertoli cells.

International journal of reproductive biomedicine

Hashemnia SM, Atari-Hajipirloo S, Roshan-Milani S, Valizadeh N, Mahabadi S, Kheradmand F.
PMID: 27738659
Int J Reprod Biomed. 2016 Sep;14(9):577-582.

BACKGROUND: The anticancer agent imatinib (IM) is a small molecular analog of ATP that inhibits tyrosine kinase activity of platelet derived growth factors (PDGFs) and stem cell factor (SCF) receptor in cancer cells. However these factors have a key...

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Hashemnia SM, Atari-Hajipirloo S, Roshan-Milani S, et al. Imatinib alters cell viability but not growth factors levels in TM4 Sertoli cells. Int J Reprod Biomed. 2016;14(9):577-582
Hashemnia, S. M., Atari-Hajipirloo, S., Roshan-Milani, S., Valizadeh, N., Mahabadi, S., & Kheradmand, F. (2016). Imatinib alters cell viability but not growth factors levels in TM4 Sertoli cells. International journal of reproductive biomedicine, 14(9), 577-582.
Hashemnia, Seyyed Mohammad Reza, et al. "Imatinib alters cell viability but not growth factors levels in TM4 Sertoli cells." International journal of reproductive biomedicine vol. 14,9 (2016): 577-582.
Hashemnia SM, Atari-Hajipirloo S, Roshan-Milani S, Valizadeh N, Mahabadi S, Kheradmand F. Imatinib alters cell viability but not growth factors levels in TM4 Sertoli cells. Int J Reprod Biomed. 2016 Sep;14(9):577-582. PMID: 27738659; PMCID: PMC5054294.
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Light-induced defect creation in hydrogenated amorphous silicon: A detailed examination using junction-capacitance methods.

Physical review. B, Condensed matter

Cohen JD, Harbison JP, Mahavadi KK, Zellama K.
PMID: 9941103
Phys Rev B Condens Matter. 1987 May 15;35(14):7776-7779. doi: 10.1103/physrevb.35.7776.

No abstract available.

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Cohen JD, Harbison JP, Mahavadi KK, et al. Light-induced defect creation in hydrogenated amorphous silicon: A detailed examination using junction-capacitance methods. Phys Rev B Condens Matter. 1987;35(14):7776-7779doi: 10.1103/physrevb.35.7776.
Cohen, J. D., Harbison, J. P., Mahavadi, K. K., & Zellama, K. (1987). Light-induced defect creation in hydrogenated amorphous silicon: A detailed examination using junction-capacitance methods. Physical review. B, Condensed matter, 35(14), 7776-7779. https://doi.org/10.1103/physrevb.35.7776
Cohen, et al. "Light-induced defect creation in hydrogenated amorphous silicon: A detailed examination using junction-capacitance methods." Physical review. B, Condensed matter vol. 35,14 (1987): 7776-7779. doi: https://doi.org/10.1103/physrevb.35.7776
Cohen JD, Harbison JP, Mahavadi KK, Zellama K. Light-induced defect creation in hydrogenated amorphous silicon: A detailed examination using junction-capacitance methods. Phys Rev B Condens Matter. 1987 May 15;35(14):7776-7779. doi: 10.1103/physrevb.35.7776. PMID: 9941103.
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Detection of Epstein-Barr virus in synovial fluid of rheumatoid arthritis patients.

Electronic physician

Mahabadi M, Faghihiloo E, Alishiri GH, Ataee MH, Ataee RA.
PMID: 27123228
Electron Physician. 2016 Mar 25;8(3):2181-6. doi: 10.19082/2181. eCollection 2016 Mar.

INTRODUCTION: Rheumatoid arthritis (RA) is one of the most common chronic inflammatory disorders. Genes and environmental factors contribute to RA. Epstein-Barr Virus (EBV) has been considered as one the RA pathogeneses. The aim of this study was to detect...

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Mahabadi M, Faghihiloo E, Alishiri GH, et al. Detection of Epstein-Barr virus in synovial fluid of rheumatoid arthritis patients. Electron Physician. 2016;8(3):2181-6doi: 10.19082/2181.
Mahabadi, M., Faghihiloo, E., Alishiri, G. H., Ataee, M. H., & Ataee, R. A. (2016). Detection of Epstein-Barr virus in synovial fluid of rheumatoid arthritis patients. Electronic physician, 8(3), 2181-6. https://doi.org/10.19082/2181
Mahabadi, Mostafa, et al. "Detection of Epstein-Barr virus in synovial fluid of rheumatoid arthritis patients." Electronic physician vol. 8,3 (2016): 2181-6. doi: https://doi.org/10.19082/2181
Mahabadi M, Faghihiloo E, Alishiri GH, Ataee MH, Ataee RA. Detection of Epstein-Barr virus in synovial fluid of rheumatoid arthritis patients. Electron Physician. 2016 Mar 25;8(3):2181-6. doi: 10.19082/2181. eCollection 2016 Mar. PMID: 27123228; PMCID: PMC4844486.
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Growth-phase dependence of bacterial membrane lipid profile and labeling for in-cell solid-state NMR applications.

Biochimica et biophysica acta. Biomembranes

Laydevant F, Mahabadi M, Llido P, Bourgouin JP, Caron L, Arnold AA, Marcotte I, Warschawski DE.
PMID: 34800428
Biochim Biophys Acta Biomembr. 2022 Feb 01;1864(2):183819. doi: 10.1016/j.bbamem.2021.183819. Epub 2021 Nov 17.

Cell labeling is a preliminary step in multiple biophysical approaches, including the solid-state nuclear magnetic resonance (NMR) study of bacteria in vivo. Deuterium solid-state NMR has been used in the past years to probe bacterial membranes and their interactions...

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Laydevant F, Mahabadi M, Llido P, et al. Growth-phase dependence of bacterial membrane lipid profile and labeling for in-cell solid-state NMR applications. Biochim Biophys Acta Biomembr. 2021;1864(2):183819doi: 10.1016/j.bbamem.2021.183819.
Laydevant, F., Mahabadi, M., Llido, P., Bourgouin, J. P., Caron, L., Arnold, A. A., Marcotte, I., & Warschawski, D. E. (2022). Growth-phase dependence of bacterial membrane lipid profile and labeling for in-cell solid-state NMR applications. Biochimica et biophysica acta. Biomembranes, 1864(2), 183819. https://doi.org/10.1016/j.bbamem.2021.183819
Laydevant, Florent, et al. "Growth-phase dependence of bacterial membrane lipid profile and labeling for in-cell solid-state NMR applications." Biochimica et biophysica acta. Biomembranes vol. 1864,2 (2022): 183819. doi: https://doi.org/10.1016/j.bbamem.2021.183819
Laydevant F, Mahabadi M, Llido P, Bourgouin JP, Caron L, Arnold AA, Marcotte I, Warschawski DE. Growth-phase dependence of bacterial membrane lipid profile and labeling for in-cell solid-state NMR applications. Biochim Biophys Acta Biomembr. 2022 Feb 01;1864(2):183819. doi: 10.1016/j.bbamem.2021.183819. Epub 2021 Nov 17. PMID: 34800428.
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Showing 49 to 54 of 54 entries