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How the COVID-19 pandemic highlights the necessity of animal research.

Current biology : CB

Genzel L, Adan R, Berns A, van den Beucken JJJP, Blokland A, Boddeke EHWGM, Bogers WM, Bontrop R, Bulthuis R, Bousema T, Clevers H, Coenen TCJJ, van Dam AM, Deen PMT, van Dijk KW, Eggen BJL, Elgersma Y, Erdogan I, Englitz B, Fentener van Vlissingen JM, la Fleur S, Fouchier R, Fitzsimons CP, Frieling W, Haagmans B, Heesters BA, Henckens MJAG, Herfst S, Hol E, van den Hove D, de Jonge MI, Jonkers J, Joosten LAB, Kalsbeek A, Kamermans M, Kampinga HH, Kas MJ, Keijer J, Kersten S, Kiliaan AJ, Kooij TWA, Kooijman S, Koopman WJH, Korosi A, Krugers HJ, Kuiken T, Kushner SA, Langermans JAM, Lesscher HMB, Lucassen PJ, Lutgens E, Netea MG, Noldus LPJJ, van der Meer JWM, Meye FJ, Mul JD, van Oers K, Olivier JDA, Pasterkamp RJ, Philippens IHCHM, Prickaerts J, Pollux BJA, Rensen PCN, van Rheenen J, van Rij RP, Ritsma L, Rockx BHG, Roozendaal B, van Schothorst EM, Stittelaar K, Stockhofe N, Swaab DF, de Swart RL, Vanderschuren LJMJ, de Vries TJ, de Vrij F, van Wezel R, Wierenga CJ, Wiesmann M, Willuhn I, de Zeeuw CI, Homberg JR.
PMID: 33142090
Curr Biol. 2020 Nov 02;30(21):4328. doi: 10.1016/j.cub.2020.10.033.

No abstract available.

Cite
Genzel L, Adan R, Berns A, et al. How the COVID-19 pandemic highlights the necessity of animal research. Curr Biol. 2020;30(21):4328doi: 10.1016/j.cub.2020.10.033.
Genzel, L., Adan, R., Berns, A., van den Beucken, J. J. J. P., Blokland, A., Boddeke, E. H. W. G. M., Bogers, W. M., Bontrop, R., Bulthuis, R., Bousema, T., Clevers, H., Coenen, T. C. J. J., van Dam, A. M., Deen, P. M. T., van Dijk, K. W., Eggen, B. J. L., Elgersma, Y., Erdogan, I., Englitz, B., Fentener van Vlissingen, J. M., la Fleur, S., Fouchier, R., Fitzsimons, C. P., Frieling, W., Haagmans, B., Heesters, B. A., Henckens, M. J. A. G., Herfst, S., Hol, E., van den Hove, D., de Jonge, M. I., Jonkers, J., Joosten, L. A. B., Kalsbeek, A., Kamermans, M., Kampinga, H. H., Kas, M. J., Keijer, J., Kersten, S., Kiliaan, A. J., Kooij, T. W. A., Kooijman, S., Koopman, W. J. H., Korosi, A., Krugers, H. J., Kuiken, T., Kushner, S. A., Langermans, J. A. M., Lesscher, H. M. B., Lucassen, P. J., Lutgens, E., Netea, M. G., Noldus, L. P. J. J., van der Meer, J. W. M., Meye, F. J., Mul, J. D., van Oers, K., Olivier, J. D. A., Pasterkamp, R. J., Philippens, I. H. C. H. M., Prickaerts, J., Pollux, B. J. A., Rensen, P. C. N., van Rheenen, J., van Rij, R. P., Ritsma, L., Rockx, B. H. G., Roozendaal, B., van Schothorst, E. M., Stittelaar, K., Stockhofe, N., Swaab, D. F., de Swart, R. L., Vanderschuren, L. J. M. J., de Vries, T. J., de Vrij, F., van Wezel, R., Wierenga, C. J., Wiesmann, M., Willuhn, I., de Zeeuw, C. I., & Homberg, J. R. (2020). How the COVID-19 pandemic highlights the necessity of animal research. Current biology : CB, 30(21), 4328. https://doi.org/10.1016/j.cub.2020.10.033
Genzel, Lisa, et al. "How the COVID-19 pandemic highlights the necessity of animal research." Current biology : CB vol. 30,21 (2020): 4328. doi: https://doi.org/10.1016/j.cub.2020.10.033
Genzel L, Adan R, Berns A, van den Beucken JJJP, Blokland A, Boddeke EHWGM, Bogers WM, Bontrop R, Bulthuis R, Bousema T, Clevers H, Coenen TCJJ, van Dam AM, Deen PMT, van Dijk KW, Eggen BJL, Elgersma Y, Erdogan I, Englitz B, Fentener van Vlissingen JM, la Fleur S, Fouchier R, Fitzsimons CP, Frieling W, Haagmans B, Heesters BA, Henckens MJAG, Herfst S, Hol E, van den Hove D, de Jonge MI, Jonkers J, Joosten LAB, Kalsbeek A, Kamermans M, Kampinga HH, Kas MJ, Keijer J, Kersten S, Kiliaan AJ, Kooij TWA, Kooijman S, Koopman WJH, Korosi A, Krugers HJ, Kuiken T, Kushner SA, Langermans JAM, Lesscher HMB, Lucassen PJ, Lutgens E, Netea MG, Noldus LPJJ, van der Meer JWM, Meye FJ, Mul JD, van Oers K, Olivier JDA, Pasterkamp RJ, Philippens IHCHM, Prickaerts J, Pollux BJA, Rensen PCN, van Rheenen J, van Rij RP, Ritsma L, Rockx BHG, Roozendaal B, van Schothorst EM, Stittelaar K, Stockhofe N, Swaab DF, de Swart RL, Vanderschuren LJMJ, de Vries TJ, de Vrij F, van Wezel R, Wierenga CJ, Wiesmann M, Willuhn I, de Zeeuw CI, Homberg JR. How the COVID-19 pandemic highlights the necessity of animal research. Curr Biol. 2020 Nov 02;30(21):4328. doi: 10.1016/j.cub.2020.10.033. PMID: 33142090; PMCID: PMC7605800.
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Lipolysis drives expression of the constitutively active receptor GPR3 to induce adipose thermogenesis.

Cell

Sveidahl Johansen O, Ma T, Hansen JB, Markussen LK, Schreiber R, Reverte-Salisa L, Dong H, Christensen DP, Sun W, Gnad T, Karavaeva I, Nielsen TS, Kooijman S, Cero C, Dmytriyeva O, Shen Y, Razzoli M, O'Brien SL, Kuipers EN, Nielsen CH, Orchard W, Willemsen N, Jespersen NZ, Lundh M, Sustarsic EG, Hallgren CM, Frost M, McGonigle S, Isidor MS, Broholm C, Pedersen O, Hansen JB, Grarup N, Hansen T, Kjær A, Granneman JG, Babu MM, Calebiro D, Nielsen S, Rydén M, Soccio R, Rensen PCN, Treebak JT, Schwartz TW, Emanuelli B, Bartolomucci A, Pfeifer A, Zechner R, Scheele C, Mandrup S, Gerhart-Hines Z.
PMID: 34048700
Cell. 2021 Jun 24;184(13):3502-3518.e33. doi: 10.1016/j.cell.2021.04.037. Epub 2021 May 27.

Thermogenic adipocytes possess a therapeutically appealing, energy-expending capacity, which is canonically cold-induced by ligand-dependent activation of β-adrenergic G protein-coupled receptors (GPCRs). Here, we uncover an alternate paradigm of GPCR-mediated adipose thermogenesis through the constitutively active receptor, GPR3. We show...

Cite
Sveidahl Johansen O, Ma T, Hansen JB, et al. Lipolysis drives expression of the constitutively active receptor GPR3 to induce adipose thermogenesis. Cell. 2021;184(13):3502-3518.e33doi: 10.1016/j.cell.2021.04.037.
Sveidahl Johansen, O., Ma, T., Hansen, J. B., Markussen, L. K., Schreiber, R., Reverte-Salisa, L., Dong, H., Christensen, D. P., Sun, W., Gnad, T., Karavaeva, I., Nielsen, T. S., Kooijman, S., Cero, C., Dmytriyeva, O., Shen, Y., Razzoli, M., O'Brien, S. L., Kuipers, E. N., Nielsen, C. H., Orchard, W., Willemsen, N., Jespersen, N. Z., Lundh, M., Sustarsic, E. G., Hallgren, C. M., Frost, M., McGonigle, S., Isidor, M. S., Broholm, C., Pedersen, O., Hansen, J. B., Grarup, N., Hansen, T., Kjær, A., Granneman, J. G., Babu, M. M., Calebiro, D., Nielsen, S., Rydén, M., Soccio, R., Rensen, P. C. N., Treebak, J. T., Schwartz, T. W., Emanuelli, B., Bartolomucci, A., Pfeifer, A., Zechner, R., Scheele, C., Mandrup, S., & Gerhart-Hines, Z. (2021). Lipolysis drives expression of the constitutively active receptor GPR3 to induce adipose thermogenesis. Cell, 184(13), 3502-3518.e33. https://doi.org/10.1016/j.cell.2021.04.037
Sveidahl Johansen, Olivia, et al. "Lipolysis drives expression of the constitutively active receptor GPR3 to induce adipose thermogenesis." Cell vol. 184,13 (2021): 3502-3518.e33. doi: https://doi.org/10.1016/j.cell.2021.04.037
Sveidahl Johansen O, Ma T, Hansen JB, Markussen LK, Schreiber R, Reverte-Salisa L, Dong H, Christensen DP, Sun W, Gnad T, Karavaeva I, Nielsen TS, Kooijman S, Cero C, Dmytriyeva O, Shen Y, Razzoli M, O'Brien SL, Kuipers EN, Nielsen CH, Orchard W, Willemsen N, Jespersen NZ, Lundh M, Sustarsic EG, Hallgren CM, Frost M, McGonigle S, Isidor MS, Broholm C, Pedersen O, Hansen JB, Grarup N, Hansen T, Kjær A, Granneman JG, Babu MM, Calebiro D, Nielsen S, Rydén M, Soccio R, Rensen PCN, Treebak JT, Schwartz TW, Emanuelli B, Bartolomucci A, Pfeifer A, Zechner R, Scheele C, Mandrup S, Gerhart-Hines Z. Lipolysis drives expression of the constitutively active receptor GPR3 to induce adipose thermogenesis. Cell. 2021 Jun 24;184(13):3502-3518.e33. doi: 10.1016/j.cell.2021.04.037. Epub 2021 May 27. PMID: 34048700; PMCID: PMC8238500.
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Inhibition of Cholesteryl Ester Transfer Protein Preserves High-Density Lipoprotein Cholesterol and Improves Survival in Sepsis.

Circulation

Trinder M, Wang Y, Madsen CM, Ponomarev T, Bohunek L, Daisely BA, Julia Kong H, Blauw LL, Nordestgaard BG, Tybjærg-Hansen A, Wurfel MM, Russell JA, Walley KR, Rensen PCN, Boyd JH, Brunham LR.
PMID: 33228395
Circulation. 2021 Mar 02;143(9):921-934. doi: 10.1161/CIRCULATIONAHA.120.048568. Epub 2020 Nov 24.

BACKGROUND: The high-density lipoprotein hypothesis of atherosclerosis has been challenged by clinical trials of cholesteryl ester transfer protein (CETP) inhibitors, which failed to show significant reductions in cardiovascular events. Plasma levels of high-density lipoprotein cholesterol (HDL-C) decline drastically during...

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Trinder M, Wang Y, Madsen CM, et al. Inhibition of Cholesteryl Ester Transfer Protein Preserves High-Density Lipoprotein Cholesterol and Improves Survival in Sepsis. Circulation. 2020;143(9):921-934doi: 10.1161/CIRCULATIONAHA.120.048568.
Trinder, M., Wang, Y., Madsen, C. M., Ponomarev, T., Bohunek, L., Daisely, B. A., Julia Kong, H., Blauw, L. L., Nordestgaard, B. G., Tybjærg-Hansen, A., Wurfel, M. M., Russell, J. A., Walley, K. R., Rensen, P. C. N., Boyd, J. H., & Brunham, L. R. (2021). Inhibition of Cholesteryl Ester Transfer Protein Preserves High-Density Lipoprotein Cholesterol and Improves Survival in Sepsis. Circulation, 143(9), 921-934. https://doi.org/10.1161/CIRCULATIONAHA.120.048568
Trinder, Mark, et al. "Inhibition of Cholesteryl Ester Transfer Protein Preserves High-Density Lipoprotein Cholesterol and Improves Survival in Sepsis." Circulation vol. 143,9 (2021): 921-934. doi: https://doi.org/10.1161/CIRCULATIONAHA.120.048568
Trinder M, Wang Y, Madsen CM, Ponomarev T, Bohunek L, Daisely BA, Julia Kong H, Blauw LL, Nordestgaard BG, Tybjærg-Hansen A, Wurfel MM, Russell JA, Walley KR, Rensen PCN, Boyd JH, Brunham LR. Inhibition of Cholesteryl Ester Transfer Protein Preserves High-Density Lipoprotein Cholesterol and Improves Survival in Sepsis. Circulation. 2021 Mar 02;143(9):921-934. doi: 10.1161/CIRCULATIONAHA.120.048568. Epub 2020 Nov 24. PMID: 33228395.
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Glucocorticoid excess induces long-lasting changes in body composition in male C57Bl/6J mice only with high-fat diet.

Physiological reports

Auvinen HE, Coomans CP, Boon MR, Romijn JA, Biermasz NR, Meijer OC, Havekes LM, Smit JW, Rensen PC, Pereira AM.
PMID: 24303175
Physiol Rep. 2013 Oct;1(5):e00103. doi: 10.1002/phy2.103. Epub 2013 Oct 11.

Glucocorticoid (GC) overexposure period as observed in Cushing's syndrome (CS) is associated with the metabolic syndrome and cardiovascular disease, which persist after long-term correction of GC excess. We performed a mouse study to identify factors that modulate metabolic recovery...

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Auvinen HE, Coomans CP, Boon MR, et al. Glucocorticoid excess induces long-lasting changes in body composition in male C57Bl/6J mice only with high-fat diet. Physiol Rep. 2013;1(5):e00103doi: 10.1002/phy2.103.
Auvinen, H. E., Coomans, C. P., Boon, M. R., Romijn, J. A., Biermasz, N. R., Meijer, O. C., Havekes, L. M., Smit, J. W., Rensen, P. C., & Pereira, A. M. (2013). Glucocorticoid excess induces long-lasting changes in body composition in male C57Bl/6J mice only with high-fat diet. Physiological reports, 1(5), e00103. https://doi.org/10.1002/phy2.103
Auvinen, Hanna E, et al. "Glucocorticoid excess induces long-lasting changes in body composition in male C57Bl/6J mice only with high-fat diet." Physiological reports vol. 1,5 (2013): e00103. doi: https://doi.org/10.1002/phy2.103
Auvinen HE, Coomans CP, Boon MR, Romijn JA, Biermasz NR, Meijer OC, Havekes LM, Smit JW, Rensen PC, Pereira AM. Glucocorticoid excess induces long-lasting changes in body composition in male C57Bl/6J mice only with high-fat diet. Physiol Rep. 2013 Oct;1(5):e00103. doi: 10.1002/phy2.103. Epub 2013 Oct 11. PMID: 24303175; PMCID: PMC3841039.
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Short-Term Cooling Increases Plasma ANGPTL3 and ANGPTL8 in Young Healthy Lean Men but Not in Middle-Aged Men with Overweight and Prediabetes.

Journal of clinical medicine

Janssen LGM, Jauhiainen M, Olkkonen VM, Haridas PAN, Nahon KJ, Rensen PCN, Boon MR.
PMID: 31416197
J Clin Med. 2019 Aug 14;8(8). doi: 10.3390/jcm8081214.

Angiopoietin-like proteins (ANGPTLs) regulate triglyceride (TG)-rich lipoprotein distribution via inhibiting TG hydrolysis by lipoprotein lipase in metabolic tissues. Brown adipose tissue combusts TG-derived fatty acids to enhance thermogenesis during cold exposure. It has been shown that cold exposure regulates...

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Janssen LGM, Jauhiainen M, Olkkonen VM, et al. Short-Term Cooling Increases Plasma ANGPTL3 and ANGPTL8 in Young Healthy Lean Men but Not in Middle-Aged Men with Overweight and Prediabetes. J Clin Med. 2019;8(8)doi: 10.3390/jcm8081214.
Janssen, L. G. M., Jauhiainen, M., Olkkonen, V. M., Haridas, P. A. N., Nahon, K. J., Rensen, P. C. N., & Boon, M. R. (2019). Short-Term Cooling Increases Plasma ANGPTL3 and ANGPTL8 in Young Healthy Lean Men but Not in Middle-Aged Men with Overweight and Prediabetes. Journal of clinical medicine, 8(8), . https://doi.org/10.3390/jcm8081214
Janssen, Laura G M, et al. "Short-Term Cooling Increases Plasma ANGPTL3 and ANGPTL8 in Young Healthy Lean Men but Not in Middle-Aged Men with Overweight and Prediabetes." Journal of clinical medicine vol. 8,8 (2019). doi: https://doi.org/10.3390/jcm8081214
Janssen LGM, Jauhiainen M, Olkkonen VM, Haridas PAN, Nahon KJ, Rensen PCN, Boon MR. Short-Term Cooling Increases Plasma ANGPTL3 and ANGPTL8 in Young Healthy Lean Men but Not in Middle-Aged Men with Overweight and Prediabetes. J Clin Med. 2019 Aug 14;8(8). doi: 10.3390/jcm8081214. PMID: 31416197; PMCID: PMC6723119.
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[No title available]

Annals of Indian Academy of Neurology

Chaudhari DM, Renjen PN, Sardana R, Butta H.
PMID: 29184344
Ann Indian Acad Neurol. 2017 Oct-Dec;20(4):399-402. doi: 10.4103/aian.AIAN_263_17.

By definition, a brain abscess is an intraparenchymal collection of pus. Nocardia shows to have a special tropism for the neural tissue. Solitary abscess represents the most common manifestation in the central nervous system, accounting for 1%-2% of all...

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Chaudhari DM, Renjen PN, Sardana R, et al. . Ann Indian Acad Neurol. 2017;20(4):399-402doi: 10.4103/aian.AIAN_263_17.
Chaudhari, D. M., Renjen, P. N., Sardana, R., & Butta, H. (2017). . Annals of Indian Academy of Neurology, 20(4), 399-402. https://doi.org/10.4103/aian.AIAN_263_17
Chaudhari, Dinesh Mohan, et al. "." Annals of Indian Academy of Neurology vol. 20,4 (2017): 399-402. doi: https://doi.org/10.4103/aian.AIAN_263_17
Chaudhari DM, Renjen PN, Sardana R, Butta H. . Ann Indian Acad Neurol. 2017 Oct-Dec;20(4):399-402. doi: 10.4103/aian.AIAN_263_17. PMID: 29184344; PMCID: PMC5682745.
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Lipolysis drives expression of the constitutively active receptor GPR3 to induce adipose thermogenesis.

Cell

Sveidahl Johansen O, Ma T, Hansen JB, Markussen LK, Schreiber R, Reverte-Salisa L, Dong H, Christensen DP, Sun W, Gnad T, Karavaeva I, Nielsen TS, Kooijman S, Cero C, Dmytriyeva O, Shen Y, Razzoli M, O'Brien SL, Kuipers EN, Nielsen CH, Orchard W, Willemsen N, Jespersen NZ, Lundh M, Sustarsic EG, Hallgren CM, Frost M, McGonigle S, Isidor MS, Broholm C, Pedersen O, Hansen JB, Grarup N, Hansen T, Kjær A, Granneman JG, Babu MM, Calebiro D, Nielsen S, Rydén M, Soccio R, Rensen PCN, Treebak JT, Schwartz TW, Emanuelli B, Bartolomucci A, Pfeifer A, Zechner R, Scheele C, Mandrup S, Gerhart-Hines Z.
PMID: 34048700
Cell. 2021 Jun 24;184(13):3502-3518.e33. doi: 10.1016/j.cell.2021.04.037. Epub 2021 May 27.

Thermogenic adipocytes possess a therapeutically appealing, energy-expending capacity, which is canonically cold-induced by ligand-dependent activation of β-adrenergic G protein-coupled receptors (GPCRs). Here, we uncover an alternate paradigm of GPCR-mediated adipose thermogenesis through the constitutively active receptor, GPR3. We show...

Cite
Sveidahl Johansen O, Ma T, Hansen JB, et al. Lipolysis drives expression of the constitutively active receptor GPR3 to induce adipose thermogenesis. Cell. 2021;184(13):3502-3518.e33doi: 10.1016/j.cell.2021.04.037.
Sveidahl Johansen, O., Ma, T., Hansen, J. B., Markussen, L. K., Schreiber, R., Reverte-Salisa, L., Dong, H., Christensen, D. P., Sun, W., Gnad, T., Karavaeva, I., Nielsen, T. S., Kooijman, S., Cero, C., Dmytriyeva, O., Shen, Y., Razzoli, M., O'Brien, S. L., Kuipers, E. N., Nielsen, C. H., Orchard, W., Willemsen, N., Jespersen, N. Z., Lundh, M., Sustarsic, E. G., Hallgren, C. M., Frost, M., McGonigle, S., Isidor, M. S., Broholm, C., Pedersen, O., Hansen, J. B., Grarup, N., Hansen, T., Kjær, A., Granneman, J. G., Babu, M. M., Calebiro, D., Nielsen, S., Rydén, M., Soccio, R., Rensen, P. C. N., Treebak, J. T., Schwartz, T. W., Emanuelli, B., Bartolomucci, A., Pfeifer, A., Zechner, R., Scheele, C., Mandrup, S., & Gerhart-Hines, Z. (2021). Lipolysis drives expression of the constitutively active receptor GPR3 to induce adipose thermogenesis. Cell, 184(13), 3502-3518.e33. https://doi.org/10.1016/j.cell.2021.04.037
Sveidahl Johansen, Olivia, et al. "Lipolysis drives expression of the constitutively active receptor GPR3 to induce adipose thermogenesis." Cell vol. 184,13 (2021): 3502-3518.e33. doi: https://doi.org/10.1016/j.cell.2021.04.037
Sveidahl Johansen O, Ma T, Hansen JB, Markussen LK, Schreiber R, Reverte-Salisa L, Dong H, Christensen DP, Sun W, Gnad T, Karavaeva I, Nielsen TS, Kooijman S, Cero C, Dmytriyeva O, Shen Y, Razzoli M, O'Brien SL, Kuipers EN, Nielsen CH, Orchard W, Willemsen N, Jespersen NZ, Lundh M, Sustarsic EG, Hallgren CM, Frost M, McGonigle S, Isidor MS, Broholm C, Pedersen O, Hansen JB, Grarup N, Hansen T, Kjær A, Granneman JG, Babu MM, Calebiro D, Nielsen S, Rydén M, Soccio R, Rensen PCN, Treebak JT, Schwartz TW, Emanuelli B, Bartolomucci A, Pfeifer A, Zechner R, Scheele C, Mandrup S, Gerhart-Hines Z. Lipolysis drives expression of the constitutively active receptor GPR3 to induce adipose thermogenesis. Cell. 2021 Jun 24;184(13):3502-3518.e33. doi: 10.1016/j.cell.2021.04.037. Epub 2021 May 27. PMID: 34048700; PMCID: PMC8238500.
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Elevated plasma succinate levels are linked to higher cardiovascular disease risk factors in young adults.

Cardiovascular diabetology

Osuna-Prieto FJ, Martinez-Tellez B, Ortiz-Alvarez L, Di X, Jurado-Fasoli L, Xu H, Ceperuelo-Mallafré V, Núñez-Roa C, Kohler I, Segura-Carretero A, García-Lario JV, Gil A, Aguilera CM, Llamas-Elvira JM, Rensen PCN, Vendrell J, Ruiz JR, Fernández-Veledo S.
PMID: 34315463
Cardiovasc Diabetol. 2021 Jul 27;20(1):151. doi: 10.1186/s12933-021-01333-3.

BACKGROUND: Succinate is produced by both host and microbiota, with a key role in the interplay of immunity and metabolism and an emerging role as a biomarker for inflammatory and metabolic disorders in middle-aged adults. The relationship between plasma...

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Osuna-Prieto FJ, Martinez-Tellez B, Ortiz-Alvarez L, et al. Elevated plasma succinate levels are linked to higher cardiovascular disease risk factors in young adults. Cardiovasc Diabetol. 2021;20(1):151doi: 10.1186/s12933-021-01333-3.
Osuna-Prieto, F. J., Martinez-Tellez, B., Ortiz-Alvarez, L., Di, X., Jurado-Fasoli, L., Xu, H., Ceperuelo-Mallafré, V., Núñez-Roa, C., Kohler, I., Segura-Carretero, A., García-Lario, J. V., Gil, A., Aguilera, C. M., Llamas-Elvira, J. M., Rensen, P. C. N., Vendrell, J., Ruiz, J. R., & Fernández-Veledo, S. (2021). Elevated plasma succinate levels are linked to higher cardiovascular disease risk factors in young adults. Cardiovascular diabetology, 20(1), 151. https://doi.org/10.1186/s12933-021-01333-3
Osuna-Prieto, Francisco J, et al. "Elevated plasma succinate levels are linked to higher cardiovascular disease risk factors in young adults." Cardiovascular diabetology vol. 20,1 (2021): 151. doi: https://doi.org/10.1186/s12933-021-01333-3
Osuna-Prieto FJ, Martinez-Tellez B, Ortiz-Alvarez L, Di X, Jurado-Fasoli L, Xu H, Ceperuelo-Mallafré V, Núñez-Roa C, Kohler I, Segura-Carretero A, García-Lario JV, Gil A, Aguilera CM, Llamas-Elvira JM, Rensen PCN, Vendrell J, Ruiz JR, Fernández-Veledo S. Elevated plasma succinate levels are linked to higher cardiovascular disease risk factors in young adults. Cardiovasc Diabetol. 2021 Jul 27;20(1):151. doi: 10.1186/s12933-021-01333-3. PMID: 34315463; PMCID: PMC8314524.
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A physiological glucocorticoid rhythm is an important regulator of brown adipose tissue function.

Molecular metabolism

Kroon J, Schilperoort M, In Het Panhuis W, van den Berg R, van Doeselaar L, Verzijl CRC, van Trigt N, Mol IM, Sips HHCM, van den Heuvel JK, Koorneef LL, van der Sluis RJ, Fenzl A, Kiefer FW, Vettorazzi S, Tuckermann JP, Biermasz NR, Meijer OC, Rensen PCN, Kooijman S.
PMID: 33548499
Mol Metab. 2021 May;47:101179. doi: 10.1016/j.molmet.2021.101179. Epub 2021 Feb 03.

OBJECTIVE: Brown adipose tissue (BAT) displays a strong circadian rhythm in metabolic activity, but it is unclear how this rhythm is regulated. As circulating levels of corticosterone coincide with the rhythm of triglyceride-derived fatty acid (FA) uptake by BAT,...

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Kroon J, Schilperoort M, In Het Panhuis W, et al. A physiological glucocorticoid rhythm is an important regulator of brown adipose tissue function. Mol Metab. 2021;47:101179doi: 10.1016/j.molmet.2021.101179.
Kroon, J., Schilperoort, M., In Het Panhuis, W., van den Berg, R., van Doeselaar, L., Verzijl, C. R. C., van Trigt, N., Mol, I. M., Sips, H. H. C. M., van den Heuvel, J. K., Koorneef, L. L., van der Sluis, R. J., Fenzl, A., Kiefer, F. W., Vettorazzi, S., Tuckermann, J. P., Biermasz, N. R., Meijer, O. C., Rensen, P. C. N., & Kooijman, S. (2021). A physiological glucocorticoid rhythm is an important regulator of brown adipose tissue function. Molecular metabolism, 47101179. https://doi.org/10.1016/j.molmet.2021.101179
Kroon, Jan, et al. "A physiological glucocorticoid rhythm is an important regulator of brown adipose tissue function." Molecular metabolism vol. 47 (2021): 101179. doi: https://doi.org/10.1016/j.molmet.2021.101179
Kroon J, Schilperoort M, In Het Panhuis W, van den Berg R, van Doeselaar L, Verzijl CRC, van Trigt N, Mol IM, Sips HHCM, van den Heuvel JK, Koorneef LL, van der Sluis RJ, Fenzl A, Kiefer FW, Vettorazzi S, Tuckermann JP, Biermasz NR, Meijer OC, Rensen PCN, Kooijman S. A physiological glucocorticoid rhythm is an important regulator of brown adipose tissue function. Mol Metab. 2021 May;47:101179. doi: 10.1016/j.molmet.2021.101179. Epub 2021 Feb 03. PMID: 33548499; PMCID: PMC7907824.
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Chronobiology and Chronotherapy of Osteoporosis.

JBMR plus

Winter EM, Kooijman S, Appelman-Dijkstra NM, Meijer OC, Rensen PC, Schilperoort M.
PMID: 34693186
JBMR Plus. 2021 May 05;5(10):e10504. doi: 10.1002/jbm4.10504. eCollection 2021 Oct.

Physiological circadian (ie, 24-hour) rhythms are critical for bone health. Animal studies have shown that genes involved in the intrinsic molecular clock demonstrate potent circadian expression patterns in bone and that genetic disruption of these clock genes results in...

Cite
Winter EM, Kooijman S, Appelman-Dijkstra NM, et al. Chronobiology and Chronotherapy of Osteoporosis. JBMR Plus. 2021;5(10):e10504doi: 10.1002/jbm4.10504.
Winter, E. M., Kooijman, S., Appelman-Dijkstra, N. M., Meijer, O. C., Rensen, P. C., & Schilperoort, M. (2021). Chronobiology and Chronotherapy of Osteoporosis. JBMR plus, 5(10), e10504. https://doi.org/10.1002/jbm4.10504
Winter, Elizabeth M, et al. "Chronobiology and Chronotherapy of Osteoporosis." JBMR plus vol. 5,10 (2021): e10504. doi: https://doi.org/10.1002/jbm4.10504
Winter EM, Kooijman S, Appelman-Dijkstra NM, Meijer OC, Rensen PC, Schilperoort M. Chronobiology and Chronotherapy of Osteoporosis. JBMR Plus. 2021 May 05;5(10):e10504. doi: 10.1002/jbm4.10504. eCollection 2021 Oct. PMID: 34693186; PMCID: PMC8520066.
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Guillain-Barre' Syndrome Following Scrub Typhus: A Rare Case.

Annals of Indian Academy of Neurology

Pandey S, Chaudhari D, Renjen PN, Ahmad K.
PMID: 34447024
Ann Indian Acad Neurol. 2021 May-Jun;24(3):451-452. doi: 10.4103/aian.AIAN_471_20. Epub 2021 Jan 25.

No abstract available.

Cite
Pandey S, Chaudhari D, Renjen PN, et al. Guillain-Barre' Syndrome Following Scrub Typhus: A Rare Case. Ann Indian Acad Neurol. 2021;24(3):451-452doi: 10.4103/aian.AIAN_471_20.
Pandey, S., Chaudhari, D., Renjen, P. N., & Ahmad, K. (2021). Guillain-Barre' Syndrome Following Scrub Typhus: A Rare Case. Annals of Indian Academy of Neurology, 24(3), 451-452. https://doi.org/10.4103/aian.AIAN_471_20
Pandey, Shishir, et al. "Guillain-Barre' Syndrome Following Scrub Typhus: A Rare Case." Annals of Indian Academy of Neurology vol. 24,3 (2021): 451-452. doi: https://doi.org/10.4103/aian.AIAN_471_20
Pandey S, Chaudhari D, Renjen PN, Ahmad K. Guillain-Barre' Syndrome Following Scrub Typhus: A Rare Case. Ann Indian Acad Neurol. 2021 May-Jun;24(3):451-452. doi: 10.4103/aian.AIAN_471_20. Epub 2021 Jan 25. PMID: 34447024; PMCID: PMC8370181.
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