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Improving work functioning and mental health of health care employees using an e-mental health approach to workers' health surveillance: pretest-posttest study.

Safety and health at work

Ketelaar SM, Nieuwenhuijsen K, Bolier L, Smeets O, Sluiter JK.
PMID: 25516815
Saf Health Work. 2014 Dec;5(4):216-21. doi: 10.1016/j.shaw.2014.08.002. Epub 2014 Sep 16.

BACKGROUND: Mental health complaints are quite common in health care employees and can have adverse effects on work functioning. The aim of this study was to evaluate an e-mental health (EMH) approach to workers' health surveillance (WHS) for nurses...

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Ketelaar SM, Nieuwenhuijsen K, Bolier L, et al. Improving work functioning and mental health of health care employees using an e-mental health approach to workers' health surveillance: pretest-posttest study. Saf Health Work. 2014;5(4):216-21doi: 10.1016/j.shaw.2014.08.002.
Ketelaar, S. M., Nieuwenhuijsen, K., Bolier, L., Smeets, O., & Sluiter, J. K. (2014). Improving work functioning and mental health of health care employees using an e-mental health approach to workers' health surveillance: pretest-posttest study. Safety and health at work, 5(4), 216-21. https://doi.org/10.1016/j.shaw.2014.08.002
Ketelaar, Sarah M, et al. "Improving work functioning and mental health of health care employees using an e-mental health approach to workers' health surveillance: pretest-posttest study." Safety and health at work vol. 5,4 (2014): 216-21. doi: https://doi.org/10.1016/j.shaw.2014.08.002
Ketelaar SM, Nieuwenhuijsen K, Bolier L, Smeets O, Sluiter JK. Improving work functioning and mental health of health care employees using an e-mental health approach to workers' health surveillance: pretest-posttest study. Saf Health Work. 2014 Dec;5(4):216-21. doi: 10.1016/j.shaw.2014.08.002. Epub 2014 Sep 16. PMID: 25516815; PMCID: PMC4266797.
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Benign upper extremity tumors: factors associated with operative treatment.

Hand (New York, N.Y.)

Sluijmer HC, Becker SJ, Ring DC.
PMID: 24426934
Hand (N Y). 2013 Sep;8(3):274-81. doi: 10.1007/s11552-013-9518-5.

BACKGROUND: Tumors are common in the upper extremity and are mostly benign and inconsequential. The purpose of this study was to determine factors associated with operative treatment for suspected benign tumors of the upper extremity.METHODS: Treated by three different...

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Sluijmer HC, Becker SJ, Ring DC. Benign upper extremity tumors: factors associated with operative treatment. Hand (N Y). 2013;8(3):274-81doi: 10.1007/s11552-013-9518-5.
Sluijmer, H. C., Becker, S. J., & Ring, D. C. (2013). Benign upper extremity tumors: factors associated with operative treatment. Hand (New York, N.Y.), 8(3), 274-81. https://doi.org/10.1007/s11552-013-9518-5
Sluijmer, Heleen C E, et al. "Benign upper extremity tumors: factors associated with operative treatment." Hand (New York, N.Y.) vol. 8,3 (2013): 274-81. doi: https://doi.org/10.1007/s11552-013-9518-5
Sluijmer HC, Becker SJ, Ring DC. Benign upper extremity tumors: factors associated with operative treatment. Hand (N Y). 2013 Sep;8(3):274-81. doi: 10.1007/s11552-013-9518-5. PMID: 24426934; PMCID: PMC3745237.
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Long-term variability in sugarcane bagasse feedstock compositional methods: sources and magnitude of analytical variability.

Biotechnology for biofuels

Templeton DW, Sluiter JB, Sluiter A, Payne C, Crocker DP, Tao L, Wolfrum E.
PMID: 27777625
Biotechnol Biofuels. 2016 Oct 18;9:223. doi: 10.1186/s13068-016-0621-z. eCollection 2016.

BACKGROUND: In an effort to find economical, carbon-neutral transportation fuels, biomass feedstock compositional analysis methods are used to monitor, compare, and improve biofuel conversion processes. These methods are empirical, and the analytical variability seen in the feedstock compositional data...

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Templeton DW, Sluiter JB, Sluiter A, et al. Long-term variability in sugarcane bagasse feedstock compositional methods: sources and magnitude of analytical variability. Biotechnol Biofuels. 2016;9:223doi: 10.1186/s13068-016-0621-z.
Templeton, D. W., Sluiter, J. B., Sluiter, A., Payne, C., Crocker, D. P., Tao, L., & Wolfrum, E. (2016). Long-term variability in sugarcane bagasse feedstock compositional methods: sources and magnitude of analytical variability. Biotechnology for biofuels, 9223. https://doi.org/10.1186/s13068-016-0621-z
Templeton, David W, et al. "Long-term variability in sugarcane bagasse feedstock compositional methods: sources and magnitude of analytical variability." Biotechnology for biofuels vol. 9 (2016): 223. doi: https://doi.org/10.1186/s13068-016-0621-z
Templeton DW, Sluiter JB, Sluiter A, Payne C, Crocker DP, Tao L, Wolfrum E. Long-term variability in sugarcane bagasse feedstock compositional methods: sources and magnitude of analytical variability. Biotechnol Biofuels. 2016 Oct 18;9:223. doi: 10.1186/s13068-016-0621-z. eCollection 2016. PMID: 27777625; PMCID: PMC5069941.
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Integrative multiomics analysis of human atherosclerosis reveals a serum response factor-driven network associated with intraplaque hemorrhage.

Clinical and translational medicine

Jin H, Goossens P, Juhasz P, Eijgelaar W, Manca M, Karel JMH, Smirnov E, Sikkink CJJM, Mees BME, Waring O, van Kuijk K, Fazzi GE, Gijbels MJJ, Kutmon M, Evelo CTA, Hedin U, Daemen MJAP, Sluimer JC, Matic L, Biessen EAL.
PMID: 34185408
Clin Transl Med. 2021 Jun;11(6):e458. doi: 10.1002/ctm2.458.

BACKGROUND: While single-omics analyses on human atherosclerotic plaque have been very useful to map stage- or disease-related differences in expression, they only partly capture the array of changes in this tissue and suffer from scale-intrinsic limitations. In order to...

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Jin H, Goossens P, Juhasz P, et al. Integrative multiomics analysis of human atherosclerosis reveals a serum response factor-driven network associated with intraplaque hemorrhage. Clin Transl Med. 2021;11(6):e458doi: 10.1002/ctm2.458.
Jin, H., Goossens, P., Juhasz, P., Eijgelaar, W., Manca, M., Karel, J. M. H., Smirnov, E., Sikkink, C. J. J. M., Mees, B. M. E., Waring, O., van Kuijk, K., Fazzi, G. E., Gijbels, M. J. J., Kutmon, M., Evelo, C. T. A., Hedin, U., Daemen, M. J. A. P., Sluimer, J. C., Matic, L., & Biessen, E. A. L. (2021). Integrative multiomics analysis of human atherosclerosis reveals a serum response factor-driven network associated with intraplaque hemorrhage. Clinical and translational medicine, 11(6), e458. https://doi.org/10.1002/ctm2.458
Jin, Han, et al. "Integrative multiomics analysis of human atherosclerosis reveals a serum response factor-driven network associated with intraplaque hemorrhage." Clinical and translational medicine vol. 11,6 (2021): e458. doi: https://doi.org/10.1002/ctm2.458
Jin H, Goossens P, Juhasz P, Eijgelaar W, Manca M, Karel JMH, Smirnov E, Sikkink CJJM, Mees BME, Waring O, van Kuijk K, Fazzi GE, Gijbels MJJ, Kutmon M, Evelo CTA, Hedin U, Daemen MJAP, Sluimer JC, Matic L, Biessen EAL. Integrative multiomics analysis of human atherosclerosis reveals a serum response factor-driven network associated with intraplaque hemorrhage. Clin Transl Med. 2021 Jun;11(6):e458. doi: 10.1002/ctm2.458. PMID: 34185408; PMCID: PMC8236116.
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[No title available]

Circulation research

Vacante F, Rodor J, Lalwani MK, Mahmoud AD, Bennett M, De Pace AL, Miller E, Van Kuijk K, de Bruijn J, Gijbels M, Williams TC, Clark MB, Scanlon JP, Doran AC, Montgomery R, Newby DE, Giacca M, O'Carroll D, Hadoke PWF, Denby L, Sluimer JC, Baker AH.
PMID: 33622045
Circ Res. 2021 Apr 30;128(9):1258-1275. doi: 10.1161/CIRCRESAHA.120.318688. Epub 2021 Feb 24.

[Figure: see text].

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Vacante F, Rodor J, Lalwani MK, et al. . Circ Res. 2021;128(9):1258-1275doi: 10.1161/CIRCRESAHA.120.318688.
Vacante, F., Rodor, J., Lalwani, M. K., Mahmoud, A. D., Bennett, M., De Pace, A. L., Miller, E., Van Kuijk, K., de Bruijn, J., Gijbels, M., Williams, T. C., Clark, M. B., Scanlon, J. P., Doran, A. C., Montgomery, R., Newby, D. E., Giacca, M., O'Carroll, D., Hadoke, P. W. F., Denby, L., Sluimer, J. C., & Baker, A. H. (2021). . Circulation research, 128(9), 1258-1275. https://doi.org/10.1161/CIRCRESAHA.120.318688
Vacante, Francesca, et al. "." Circulation research vol. 128,9 (2021): 1258-1275. doi: https://doi.org/10.1161/CIRCRESAHA.120.318688
Vacante F, Rodor J, Lalwani MK, Mahmoud AD, Bennett M, De Pace AL, Miller E, Van Kuijk K, de Bruijn J, Gijbels M, Williams TC, Clark MB, Scanlon JP, Doran AC, Montgomery R, Newby DE, Giacca M, O'Carroll D, Hadoke PWF, Denby L, Sluimer JC, Baker AH. . Circ Res. 2021 Apr 30;128(9):1258-1275. doi: 10.1161/CIRCRESAHA.120.318688. Epub 2021 Feb 24. PMID: 33622045; PMCID: PMC7610708.
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DEFICIENCY OF MYELOID PHD PROTEINS AGGRAVATES ATHEROGENESIS VIA MACROPHAGE APOPTOSIS AND PARACRINE FIBROTIC SIGNALING: Atherogenic effects of myeloid PHD knockdown.

Cardiovascular research

van Kuijk K, Demandt JAF, Perales-Patón J, Theelen TL, Kuppe C, Marsch E, de Bruijn J, Jin H, Gijbels MJ, Matic L, Mees BME, Reutelingsperger CPM, Hedin U, Biessen EAL, Carmeliet P, Baker AH, Kramann RK, Schurgers LJ, Saez-Rodriguez J, Sluimer JC.
PMID: 33913468
Cardiovasc Res. 2021 Apr 26; doi: 10.1093/cvr/cvab152. Epub 2021 Apr 26.

AIMS: Atherosclerotic plaque hypoxia is detrimental for macrophage function. Prolyl hydroxylases (PHDs) initiate cellular hypoxic responses, possibly influencing macrophage function in plaque hypoxia. Thus, we aimed to elucidate the role of myeloid PHDs in atherosclerosis.METHODS & RESULTS: Myeloid specific...

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van Kuijk K, Demandt JAF, Perales-Patón J, et al. DEFICIENCY OF MYELOID PHD PROTEINS AGGRAVATES ATHEROGENESIS VIA MACROPHAGE APOPTOSIS AND PARACRINE FIBROTIC SIGNALING: Atherogenic effects of myeloid PHD knockdown. Cardiovasc Res. 2021;doi: 10.1093/cvr/cvab152.
van Kuijk, K., Demandt, J. A. F., Perales-Patón, J., Theelen, T. L., Kuppe, C., Marsch, E., de Bruijn, J., Jin, H., Gijbels, M. J., Matic, L., Mees, B. M. E., Reutelingsperger, C. P. M., Hedin, U., Biessen, E. A. L., Carmeliet, P., Baker, A. H., Kramann, R. K., Schurgers, L. J., Saez-Rodriguez, J., & Sluimer, J. C. (2021). DEFICIENCY OF MYELOID PHD PROTEINS AGGRAVATES ATHEROGENESIS VIA MACROPHAGE APOPTOSIS AND PARACRINE FIBROTIC SIGNALING: Atherogenic effects of myeloid PHD knockdown. Cardiovascular research, . https://doi.org/10.1093/cvr/cvab152
van Kuijk, K, et al. "DEFICIENCY OF MYELOID PHD PROTEINS AGGRAVATES ATHEROGENESIS VIA MACROPHAGE APOPTOSIS AND PARACRINE FIBROTIC SIGNALING: Atherogenic effects of myeloid PHD knockdown." Cardiovascular research vol. (2021). doi: https://doi.org/10.1093/cvr/cvab152
van Kuijk K, Demandt JAF, Perales-Patón J, Theelen TL, Kuppe C, Marsch E, de Bruijn J, Jin H, Gijbels MJ, Matic L, Mees BME, Reutelingsperger CPM, Hedin U, Biessen EAL, Carmeliet P, Baker AH, Kramann RK, Schurgers LJ, Saez-Rodriguez J, Sluimer JC. DEFICIENCY OF MYELOID PHD PROTEINS AGGRAVATES ATHEROGENESIS VIA MACROPHAGE APOPTOSIS AND PARACRINE FIBROTIC SIGNALING: Atherogenic effects of myeloid PHD knockdown. Cardiovasc Res. 2021 Apr 26; doi: 10.1093/cvr/cvab152. Epub 2021 Apr 26. PMID: 33913468.
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Showing 37 to 42 of 42 entries