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A heterochronic genetic change from an EGFR mutation to an ALK rearrangement in a patient with lung adenocarcinoma: a case report.

Journal of thoracic disease

Shiroyama T, Tamiya M, Hayama M, Nishihara T, Nishida T, Tanaka A, Morishita N, Suzuki H, Okamoto N, Kawahara K, Hirashima T.
PMID: 27162697
J Thorac Dis. 2016 May;8(5):E345-8. doi: 10.21037/jtd.2016.03.43.

An 87-year-old man with postoperative recurrent lung adenocarcinoma was treated with gefitinib. At the beginning of treatment, surgically resected archived tumor tissue revealed a deletion in exon 19 of the EGFR gene, but no ALK gene rearrangement. After gefitinib...

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Shiroyama T, Tamiya M, Hayama M, et al. A heterochronic genetic change from an EGFR mutation to an ALK rearrangement in a patient with lung adenocarcinoma: a case report. J Thorac Dis. 2016;8(5):E345-8doi: 10.21037/jtd.2016.03.43.
Shiroyama, T., Tamiya, M., Hayama, M., Nishihara, T., Nishida, T., Tanaka, A., Morishita, N., Suzuki, H., Okamoto, N., Kawahara, K., & Hirashima, T. (2016). A heterochronic genetic change from an EGFR mutation to an ALK rearrangement in a patient with lung adenocarcinoma: a case report. Journal of thoracic disease, 8(5), E345-8. https://doi.org/10.21037/jtd.2016.03.43
Shiroyama, Takayuki, et al. "A heterochronic genetic change from an EGFR mutation to an ALK rearrangement in a patient with lung adenocarcinoma: a case report." Journal of thoracic disease vol. 8,5 (2016): E345-8. doi: https://doi.org/10.21037/jtd.2016.03.43
Shiroyama T, Tamiya M, Hayama M, Nishihara T, Nishida T, Tanaka A, Morishita N, Suzuki H, Okamoto N, Kawahara K, Hirashima T. A heterochronic genetic change from an EGFR mutation to an ALK rearrangement in a patient with lung adenocarcinoma: a case report. J Thorac Dis. 2016 May;8(5):E345-8. doi: 10.21037/jtd.2016.03.43. PMID: 27162697; PMCID: PMC4842787.
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In situ observation of a hydrogel-glass interface during sliding friction.

Soft matter

Yamamoto T, Kurokawa T, Ahmed J, Kamita G, Yashima S, Furukawa Y, Ota Y, Furukawa H, Gong JP.
PMID: 24962139
Soft Matter. 2014 Aug 14;10(30):5589-96. doi: 10.1039/c4sm00338a.

Direct observation of hydrogel contact with a solid surface in water is indispensable for understanding the friction, lubrication, and adhesion of hydrogels under water. However, this is a difficult task since the refractive index of hydrogels is very close...

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Yamamoto T, Kurokawa T, Ahmed J, et al. In situ observation of a hydrogel-glass interface during sliding friction. Soft Matter. 2014;10(30):5589-96doi: 10.1039/c4sm00338a.
Yamamoto, T., Kurokawa, T., Ahmed, J., Kamita, G., Yashima, S., Furukawa, Y., Ota, Y., Furukawa, H., & Gong, J. P. (2014). In situ observation of a hydrogel-glass interface during sliding friction. Soft matter, 10(30), 5589-96. https://doi.org/10.1039/c4sm00338a
Yamamoto, Tetsurou, et al. "In situ observation of a hydrogel-glass interface during sliding friction." Soft matter vol. 10,30 (2014): 5589-96. doi: https://doi.org/10.1039/c4sm00338a
Yamamoto T, Kurokawa T, Ahmed J, Kamita G, Yashima S, Furukawa Y, Ota Y, Furukawa H, Gong JP. In situ observation of a hydrogel-glass interface during sliding friction. Soft Matter. 2014 Aug 14;10(30):5589-96. doi: 10.1039/c4sm00338a. PMID: 24962139.
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Important factors for achieving survival of five years or more in non-small cell lung cancer patients with distant metastasis.

Oncology letters

Hirashima T, Suzuki H, Okamoto N, Morishita N, Yamadori T, Tamiya M, Shiroyama T, Kurata K, Kawase I.
PMID: 24959271
Oncol Lett. 2014 Jul;8(1):327-334. doi: 10.3892/ol.2014.2107. Epub 2014 Apr 30.

In order to examine which factors were important for achieving a ≥5 year survival time in non-small cell lung cancer (NSCLC) patients with distant metastasis, 268 NSCLC patients who received first-line chemotherapy between January 2004 and December 2007 were...

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Hirashima T, Suzuki H, Okamoto N, et al. Important factors for achieving survival of five years or more in non-small cell lung cancer patients with distant metastasis. Oncol Lett. 2014;8(1):327-334doi: 10.3892/ol.2014.2107.
Hirashima, T., Suzuki, H., Okamoto, N., Morishita, N., Yamadori, T., Tamiya, M., Shiroyama, T., Kurata, K., & Kawase, I. (2014). Important factors for achieving survival of five years or more in non-small cell lung cancer patients with distant metastasis. Oncology letters, 8(1), 327-334. https://doi.org/10.3892/ol.2014.2107
Hirashima, Tomonori, et al. "Important factors for achieving survival of five years or more in non-small cell lung cancer patients with distant metastasis." Oncology letters vol. 8,1 (2014): 327-334. doi: https://doi.org/10.3892/ol.2014.2107
Hirashima T, Suzuki H, Okamoto N, Morishita N, Yamadori T, Tamiya M, Shiroyama T, Kurata K, Kawase I. Important factors for achieving survival of five years or more in non-small cell lung cancer patients with distant metastasis. Oncol Lett. 2014 Jul;8(1):327-334. doi: 10.3892/ol.2014.2107. Epub 2014 Apr 30. PMID: 24959271; PMCID: PMC4063572.
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Co-localization of iron binding on silica with p62/sequestosome1 (SQSTM1) in lung granulomas of mice with acute silicosis.

Journal of clinical biochemistry and nutrition

Shimizu Y, Dobashi K, Nagase H, Ohta K, Sano T, Matsuzaki S, Ishii Y, Satoh T, Koka M, Yokoyama A, Ohkubo T, Ishii Y, Kamiya T.
PMID: 25834305
J Clin Biochem Nutr. 2015 Jan;56(1):74-83. doi: 10.3164/jcbn.14-44. Epub 2014 Nov 28.

The cellular mechanisms involved in the development of silicosis have not been fully elucidated. This study aimed to examine influence of silica-induced lung injury on autophagy. Suspensions of crystalline silica particles were administered transnasally to C57BL/6j mice. Immunohistochemical examination...

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Shimizu Y, Dobashi K, Nagase H, et al. Co-localization of iron binding on silica with p62/sequestosome1 (SQSTM1) in lung granulomas of mice with acute silicosis. J Clin Biochem Nutr. 2014;56(1):74-83doi: 10.3164/jcbn.14-44.
Shimizu, Y., Dobashi, K., Nagase, H., Ohta, K., Sano, T., Matsuzaki, S., Ishii, Y., Satoh, T., Koka, M., Yokoyama, A., Ohkubo, T., Ishii, Y., & Kamiya, T. (2015). Co-localization of iron binding on silica with p62/sequestosome1 (SQSTM1) in lung granulomas of mice with acute silicosis. Journal of clinical biochemistry and nutrition, 56(1), 74-83. https://doi.org/10.3164/jcbn.14-44
Shimizu, Yasuo, et al. "Co-localization of iron binding on silica with p62/sequestosome1 (SQSTM1) in lung granulomas of mice with acute silicosis." Journal of clinical biochemistry and nutrition vol. 56,1 (2015): 74-83. doi: https://doi.org/10.3164/jcbn.14-44
Shimizu Y, Dobashi K, Nagase H, Ohta K, Sano T, Matsuzaki S, Ishii Y, Satoh T, Koka M, Yokoyama A, Ohkubo T, Ishii Y, Kamiya T. Co-localization of iron binding on silica with p62/sequestosome1 (SQSTM1) in lung granulomas of mice with acute silicosis. J Clin Biochem Nutr. 2015 Jan;56(1):74-83. doi: 10.3164/jcbn.14-44. Epub 2014 Nov 28. PMID: 25834305; PMCID: PMC4306660.
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The first case of biological therapy discontinuation after a complete remission induced by maintenance therapy with adalimumab for refractory ulcerative colitis.

Journal of clinical medicine research

Tanida S, Mizoshita T, Ozeki K, Tsukamoto H, Mori Y, Kubota E, Kataoka H, Kamiya T, Joh T.
PMID: 25436030
J Clin Med Res. 2015 Feb;7(2):118-21. doi: 10.14740/jocmr1991w. Epub 2014 Nov 19.

A 43-year-old woman, diagnosed with ulcerative colitis (UC) at age of 30, received outpatient treatment with corticosteroids. However, flare-up occurred, and adalimumab (ADA) treatment commenced in July 2009. A complete remission with mucosal healing was achieved by 32 weeks...

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Tanida S, Mizoshita T, Ozeki K, et al. The first case of biological therapy discontinuation after a complete remission induced by maintenance therapy with adalimumab for refractory ulcerative colitis. J Clin Med Res. 2014;7(2):118-21doi: 10.14740/jocmr1991w.
Tanida, S., Mizoshita, T., Ozeki, K., Tsukamoto, H., Mori, Y., Kubota, E., Kataoka, H., Kamiya, T., & Joh, T. (2015). The first case of biological therapy discontinuation after a complete remission induced by maintenance therapy with adalimumab for refractory ulcerative colitis. Journal of clinical medicine research, 7(2), 118-21. https://doi.org/10.14740/jocmr1991w
Tanida, Satoshi, et al. "The first case of biological therapy discontinuation after a complete remission induced by maintenance therapy with adalimumab for refractory ulcerative colitis." Journal of clinical medicine research vol. 7,2 (2015): 118-21. doi: https://doi.org/10.14740/jocmr1991w
Tanida S, Mizoshita T, Ozeki K, Tsukamoto H, Mori Y, Kubota E, Kataoka H, Kamiya T, Joh T. The first case of biological therapy discontinuation after a complete remission induced by maintenance therapy with adalimumab for refractory ulcerative colitis. J Clin Med Res. 2015 Feb;7(2):118-21. doi: 10.14740/jocmr1991w. Epub 2014 Nov 19. PMID: 25436030; PMCID: PMC4245064.
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Anterior and Posterior Corneal Astigmatism after Refractive Lenticule Extraction for Myopic Astigmatism.

Journal of ophthalmology

Kamiya K, Shimizu K, Yamagishi M, Igarashi A, Kobashi H.
PMID: 26097749
J Ophthalmol. 2015;2015:915853. doi: 10.1155/2015/915853. Epub 2015 May 12.

Purpose. To assess the amount and the axis orientation of anterior and posterior corneal astigmatism after refractive lenticule extraction (ReLEx) for myopic astigmatism. Methods. We retrospectively examined 53 eyes of 53 consecutive patients (mean age ± standard deviation, 33.2...

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Kamiya K, Shimizu K, Yamagishi M, et al. Anterior and Posterior Corneal Astigmatism after Refractive Lenticule Extraction for Myopic Astigmatism. J Ophthalmol. 2015;2015:915853doi: 10.1155/2015/915853.
Kamiya, K., Shimizu, K., Yamagishi, M., Igarashi, A., & Kobashi, H. (2015). Anterior and Posterior Corneal Astigmatism after Refractive Lenticule Extraction for Myopic Astigmatism. Journal of ophthalmology, 2015915853. https://doi.org/10.1155/2015/915853
Kamiya, Kazutaka, et al. "Anterior and Posterior Corneal Astigmatism after Refractive Lenticule Extraction for Myopic Astigmatism." Journal of ophthalmology vol. 2015 (2015): 915853. doi: https://doi.org/10.1155/2015/915853
Kamiya K, Shimizu K, Yamagishi M, Igarashi A, Kobashi H. Anterior and Posterior Corneal Astigmatism after Refractive Lenticule Extraction for Myopic Astigmatism. J Ophthalmol. 2015;2015:915853. doi: 10.1155/2015/915853. Epub 2015 May 12. PMID: 26097749; PMCID: PMC4443750.
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Challenges of breeding potato cultivars to grow in various environments and to meet different demands.

Breeding science

Mori K, Asano K, Tamiya S, Nakao T, Mori M.
PMID: 25931976
Breed Sci. 2015 Mar;65(1):3-16. doi: 10.1270/jsbbs.65.3. Epub 2015 Mar 01.

The potato (Solanum tuberosum L.) is cultivated all year round in Japan by using four types of cropping: summer and winter croppings, and double cropping in spring and fall. In each cropping season, growth conditions such as temperature, day...

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Mori K, Asano K, Tamiya S, et al. Challenges of breeding potato cultivars to grow in various environments and to meet different demands. Breed Sci. 2015;65(1):3-16doi: 10.1270/jsbbs.65.3.
Mori, K., Asano, K., Tamiya, S., Nakao, T., & Mori, M. (2015). Challenges of breeding potato cultivars to grow in various environments and to meet different demands. Breeding science, 65(1), 3-16. https://doi.org/10.1270/jsbbs.65.3
Mori, Kazuyuki, et al. "Challenges of breeding potato cultivars to grow in various environments and to meet different demands." Breeding science vol. 65,1 (2015): 3-16. doi: https://doi.org/10.1270/jsbbs.65.3
Mori K, Asano K, Tamiya S, Nakao T, Mori M. Challenges of breeding potato cultivars to grow in various environments and to meet different demands. Breed Sci. 2015 Mar;65(1):3-16. doi: 10.1270/jsbbs.65.3. Epub 2015 Mar 01. PMID: 25931976; PMCID: PMC4374562.
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High glucose impairs the proliferation and increases the apoptosis of endothelial progenitor cells by suppression of Akt.

Journal of diabetes investigation

Nakamura N, Naruse K, Kobayashi Y, Matsuki T, Hamada Y, Nakashima E, Kamiya H, Hata M, Nishikawa T, Enomoto A, Takahashi M, Murohara T, Matsubara T, Oiso Y, Nakamura J.
PMID: 24843496
J Diabetes Investig. 2011 Aug 02;2(4):262-70. doi: 10.1111/j.2040-1124.2010.00093.x.

UNLABELLED: Aims/Introduction:  Endothelial progenitor cells (EPC) play a critical role in adult vasculogenesis and vascular repair. Previous studies have described the dysfunction of EPC in diabetic patients, but the precise mechanism is still unclear. To elucidate the dysfunction of...

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Nakamura N, Naruse K, Kobayashi Y, et al. High glucose impairs the proliferation and increases the apoptosis of endothelial progenitor cells by suppression of Akt. J Diabetes Investig. 2011;2(4):262-70doi: 10.1111/j.2040-1124.2010.00093.x.
Nakamura, N., Naruse, K., Kobayashi, Y., Matsuki, T., Hamada, Y., Nakashima, E., Kamiya, H., Hata, M., Nishikawa, T., Enomoto, A., Takahashi, M., Murohara, T., Matsubara, T., Oiso, Y., & Nakamura, J. (2011). High glucose impairs the proliferation and increases the apoptosis of endothelial progenitor cells by suppression of Akt. Journal of diabetes investigation, 2(4), 262-70. https://doi.org/10.1111/j.2040-1124.2010.00093.x
Nakamura, Nobuhisa, et al. "High glucose impairs the proliferation and increases the apoptosis of endothelial progenitor cells by suppression of Akt." Journal of diabetes investigation vol. 2,4 (2011): 262-70. doi: https://doi.org/10.1111/j.2040-1124.2010.00093.x
Nakamura N, Naruse K, Kobayashi Y, Matsuki T, Hamada Y, Nakashima E, Kamiya H, Hata M, Nishikawa T, Enomoto A, Takahashi M, Murohara T, Matsubara T, Oiso Y, Nakamura J. High glucose impairs the proliferation and increases the apoptosis of endothelial progenitor cells by suppression of Akt. J Diabetes Investig. 2011 Aug 02;2(4):262-70. doi: 10.1111/j.2040-1124.2010.00093.x. PMID: 24843496; PMCID: PMC4014965.
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Ingestion of a moderate high-sucrose diet results in glucose intolerance with reduced liver glucokinase activity and impaired glucagon-like peptide-1 secretion.

Journal of diabetes investigation

Sakamoto E, Seino Y, Fukami A, Mizutani N, Tsunekawa S, Ishikawa K, Ogata H, Uenishi E, Kamiya H, Hamada Y, Sato H, Harada N, Toyoda Y, Miwa I, Nakamura J, Inagaki N, Oiso Y, Ozaki N.
PMID: 24843603
J Diabetes Investig. 2012 Oct 18;3(5):432-40. doi: 10.1111/j.2040-1124.2012.00208.x.

UNLABELLED: Aims/Introduction:  Excessive intake of sucrose can cause severe health issues, such as diabetes mellitus. In animal studies, consumption of a high-sucrose diet (SUC) has been shown to cause obesity, insulin resistance and glucose intolerance. However, several in vivo...

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Sakamoto E, Seino Y, Fukami A, et al. Ingestion of a moderate high-sucrose diet results in glucose intolerance with reduced liver glucokinase activity and impaired glucagon-like peptide-1 secretion. J Diabetes Investig. 2012;3(5):432-40doi: 10.1111/j.2040-1124.2012.00208.x.
Sakamoto, E., Seino, Y., Fukami, A., Mizutani, N., Tsunekawa, S., Ishikawa, K., Ogata, H., Uenishi, E., Kamiya, H., Hamada, Y., Sato, H., Harada, N., Toyoda, Y., Miwa, I., Nakamura, J., Inagaki, N., Oiso, Y., & Ozaki, N. (2012). Ingestion of a moderate high-sucrose diet results in glucose intolerance with reduced liver glucokinase activity and impaired glucagon-like peptide-1 secretion. Journal of diabetes investigation, 3(5), 432-40. https://doi.org/10.1111/j.2040-1124.2012.00208.x
Sakamoto, Eriko, et al. "Ingestion of a moderate high-sucrose diet results in glucose intolerance with reduced liver glucokinase activity and impaired glucagon-like peptide-1 secretion." Journal of diabetes investigation vol. 3,5 (2012): 432-40. doi: https://doi.org/10.1111/j.2040-1124.2012.00208.x
Sakamoto E, Seino Y, Fukami A, Mizutani N, Tsunekawa S, Ishikawa K, Ogata H, Uenishi E, Kamiya H, Hamada Y, Sato H, Harada N, Toyoda Y, Miwa I, Nakamura J, Inagaki N, Oiso Y, Ozaki N. Ingestion of a moderate high-sucrose diet results in glucose intolerance with reduced liver glucokinase activity and impaired glucagon-like peptide-1 secretion. J Diabetes Investig. 2012 Oct 18;3(5):432-40. doi: 10.1111/j.2040-1124.2012.00208.x. PMID: 24843603; PMCID: PMC4019243.
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n-type conversion of SnS by isovalent ion substitution: Geometrical doping as a new doping route.

Scientific reports

Ran FY, Xiao Z, Toda Y, Hiramatsu H, Hosono H, Kamiya T.
PMID: 26020855
Sci Rep. 2015 May 28;5:10428. doi: 10.1038/srep10428.

Tin monosulfide (SnS) is a naturally p-type semiconductor with a layered crystal structure, but no reliable n-type SnS has been obtained by conventional aliovalent ion substitution. In this work, carrier polarity conversion to n-type was achieved by isovalent ion...

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Ran FY, Xiao Z, Toda Y, et al. n-type conversion of SnS by isovalent ion substitution: Geometrical doping as a new doping route. Sci Rep. 2015;5:10428doi: 10.1038/srep10428.
Ran, F. Y., Xiao, Z., Toda, Y., Hiramatsu, H., Hosono, H., & Kamiya, T. (2015). n-type conversion of SnS by isovalent ion substitution: Geometrical doping as a new doping route. Scientific reports, 510428. https://doi.org/10.1038/srep10428
Ran, Fan-Yong, et al. "n-type conversion of SnS by isovalent ion substitution: Geometrical doping as a new doping route." Scientific reports vol. 5 (2015): 10428. doi: https://doi.org/10.1038/srep10428
Ran FY, Xiao Z, Toda Y, Hiramatsu H, Hosono H, Kamiya T. n-type conversion of SnS by isovalent ion substitution: Geometrical doping as a new doping route. Sci Rep. 2015 May 28;5:10428. doi: 10.1038/srep10428. PMID: 26020855; PMCID: PMC4446993.
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Combination of 5-aminolevulinic acid and ferrous ion reduces plasma glucose and hemoglobin A1c levels in Zucker diabetic fatty rats.

FEBS open bio

Hara T, Koda A, Nozawa N, Ota U, Kondo H, Nakagawa H, Kamiya A, Miyashita K, Itoh H, Nakajima M, Tanaka T.
PMID: 27239432
FEBS Open Bio. 2016 Apr 29;6(6):515-28. doi: 10.1002/2211-5463.12048. eCollection 2016 Jun.

Mitochondrial dysfunction is associated with type 2 diabetes mellitus (T2DM). 5-Aminolevulinic acid (ALA), a natural amino acid produced only in the mitochondria, is a precursor of heme. Cytochromes that contain heme play an important role in aerobic energy metabolism....

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Hara T, Koda A, Nozawa N, et al. Combination of 5-aminolevulinic acid and ferrous ion reduces plasma glucose and hemoglobin A1c levels in Zucker diabetic fatty rats. FEBS Open Bio. 2016;6(6):515-28doi: 10.1002/2211-5463.12048.
Hara, T., Koda, A., Nozawa, N., Ota, U., Kondo, H., Nakagawa, H., Kamiya, A., Miyashita, K., Itoh, H., Nakajima, M., & Tanaka, T. (2016). Combination of 5-aminolevulinic acid and ferrous ion reduces plasma glucose and hemoglobin A1c levels in Zucker diabetic fatty rats. FEBS open bio, 6(6), 515-28. https://doi.org/10.1002/2211-5463.12048
Hara, Takeshi, et al. "Combination of 5-aminolevulinic acid and ferrous ion reduces plasma glucose and hemoglobin A1c levels in Zucker diabetic fatty rats." FEBS open bio vol. 6,6 (2016): 515-28. doi: https://doi.org/10.1002/2211-5463.12048
Hara T, Koda A, Nozawa N, Ota U, Kondo H, Nakagawa H, Kamiya A, Miyashita K, Itoh H, Nakajima M, Tanaka T. Combination of 5-aminolevulinic acid and ferrous ion reduces plasma glucose and hemoglobin A1c levels in Zucker diabetic fatty rats. FEBS Open Bio. 2016 Apr 29;6(6):515-28. doi: 10.1002/2211-5463.12048. eCollection 2016 Jun. PMID: 27239432; PMCID: PMC4880722.
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Evaluation of Kampo medicine in the clinical practice guideline for irritable bowel syndrome.

Journal of gastroenterology

Fukudo S, Kaneko H, Akiho H, Inamori M, Endo Y, Okumura T, Kanazawa M, Kamiya T, Sato K, Chiba T, Furuta K, Yamato S, Arakawa T, Fujiyama Y, Azuma T, Fujimoto K, Mine T, Miura S, Kinoshita Y, Watanabe M, Sugano K, Shimosegawa T.
PMID: 26100007
J Gastroenterol. 2015 Jul;50(7):817-8. doi: 10.1007/s00535-015-1074-z. Epub 2015 Jun 23.

No abstract available.

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Fukudo S, Kaneko H, Akiho H, et al. Evaluation of Kampo medicine in the clinical practice guideline for irritable bowel syndrome. J Gastroenterol. 2015;50(7):817-8doi: 10.1007/s00535-015-1074-z.
Fukudo, S., Kaneko, H., Akiho, H., Inamori, M., Endo, Y., Okumura, T., Kanazawa, M., Kamiya, T., Sato, K., Chiba, T., Furuta, K., Yamato, S., Arakawa, T., Fujiyama, Y., Azuma, T., Fujimoto, K., Mine, T., Miura, S., Kinoshita, Y., Watanabe, M., Sugano, K., & Shimosegawa, T. (2015). Evaluation of Kampo medicine in the clinical practice guideline for irritable bowel syndrome. Journal of gastroenterology, 50(7), 817-8. https://doi.org/10.1007/s00535-015-1074-z
Fukudo, Shin, et al. "Evaluation of Kampo medicine in the clinical practice guideline for irritable bowel syndrome." Journal of gastroenterology vol. 50,7 (2015): 817-8. doi: https://doi.org/10.1007/s00535-015-1074-z
Fukudo S, Kaneko H, Akiho H, Inamori M, Endo Y, Okumura T, Kanazawa M, Kamiya T, Sato K, Chiba T, Furuta K, Yamato S, Arakawa T, Fujiyama Y, Azuma T, Fujimoto K, Mine T, Miura S, Kinoshita Y, Watanabe M, Sugano K, Shimosegawa T. Evaluation of Kampo medicine in the clinical practice guideline for irritable bowel syndrome. J Gastroenterol. 2015 Jul;50(7):817-8. doi: 10.1007/s00535-015-1074-z. Epub 2015 Jun 23. PMID: 26100007.
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