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THE SELECTION BARRIER BETWEEN POPULATIONS SUBJECT TO STABILIZING SELECTION.

Evolution; international journal of organic evolution

Zhivotovsky LA, Christiansen FB.
PMID: 28565090
Evolution. 1995 Jun;49(3):490-501. doi: 10.1111/j.1558-5646.1995.tb02281.x.

The introgression of genes carried by a small group of immigrants is studied. The recipient and the donor populations differ at several autosomal loci subject to weak selection, and two allelic forms of each gene are considered. Fitness variation...

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Zhivotovsky LA, Christiansen FB. THE SELECTION BARRIER BETWEEN POPULATIONS SUBJECT TO STABILIZING SELECTION. Evolution. 1995;49(3):490-501doi: 10.1111/j.1558-5646.1995.tb02281.x.
Zhivotovsky, L. A., & Christiansen, F. B. (1995). THE SELECTION BARRIER BETWEEN POPULATIONS SUBJECT TO STABILIZING SELECTION. Evolution; international journal of organic evolution, 49(3), 490-501. https://doi.org/10.1111/j.1558-5646.1995.tb02281.x
Zhivotovsky, Lev A, and Christiansen, Freddy Bugge. "THE SELECTION BARRIER BETWEEN POPULATIONS SUBJECT TO STABILIZING SELECTION." Evolution; international journal of organic evolution vol. 49,3 (1995): 490-501. doi: https://doi.org/10.1111/j.1558-5646.1995.tb02281.x
Zhivotovsky LA, Christiansen FB. THE SELECTION BARRIER BETWEEN POPULATIONS SUBJECT TO STABILIZING SELECTION. Evolution. 1995 Jun;49(3):490-501. doi: 10.1111/j.1558-5646.1995.tb02281.x. PMID: 28565090.
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Systems biology approaches to develop innovative strategies for lung cancer therapy.

Cell death & disease

Viktorsson K, Lewensohn R, Zhivotovsky B.
PMID: 24874732
Cell Death Dis. 2014 May 29;5:e1260. doi: 10.1038/cddis.2014.28.

Lung cancer (LC) is a number one killer of cancer-related death among men and women worldwide. Major advances have been made in the diagnosis, staging and use of surgery for LC, but systemic chemotherapy and radiotherapy alone or in...

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Viktorsson K, Lewensohn R, Zhivotovsky B. Systems biology approaches to develop innovative strategies for lung cancer therapy. Cell Death Dis. 2014;5:e1260doi: 10.1038/cddis.2014.28.
Viktorsson, K., Lewensohn, R., & Zhivotovsky, B. (2014). Systems biology approaches to develop innovative strategies for lung cancer therapy. Cell death & disease, 5e1260. https://doi.org/10.1038/cddis.2014.28
Viktorsson, K, et al. "Systems biology approaches to develop innovative strategies for lung cancer therapy." Cell death & disease vol. 5 (2014): e1260. doi: https://doi.org/10.1038/cddis.2014.28
Viktorsson K, Lewensohn R, Zhivotovsky B. Systems biology approaches to develop innovative strategies for lung cancer therapy. Cell Death Dis. 2014 May 29;5:e1260. doi: 10.1038/cddis.2014.28. PMID: 24874732; PMCID: PMC4047893.
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Mitochondrial dysfunction is an essential step for killing of non-small cell lung carcinomas resistant to conventional treatment.

Oncogene

Joseph B, Marchetti P, Formstecher P, Kroemer G, Lewensohn R, Zhivotovsky B.
PMID: 28394341
Oncogene. 2017 Aug 17;36(33):4818. doi: 10.1038/onc.2017.78. Epub 2017 Apr 10.

This corrects the article DOI: 10.1038/sj.onc.1205018.

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Joseph B, Marchetti P, Formstecher P, et al. Mitochondrial dysfunction is an essential step for killing of non-small cell lung carcinomas resistant to conventional treatment. Oncogene. 2017;36(33):4818doi: 10.1038/onc.2017.78.
Joseph, B., Marchetti, P., Formstecher, P., Kroemer, G., Lewensohn, R., & Zhivotovsky, B. (2017). Mitochondrial dysfunction is an essential step for killing of non-small cell lung carcinomas resistant to conventional treatment. Oncogene, 36(33), 4818. https://doi.org/10.1038/onc.2017.78
Joseph, B, et al. "Mitochondrial dysfunction is an essential step for killing of non-small cell lung carcinomas resistant to conventional treatment." Oncogene vol. 36,33 (2017): 4818. doi: https://doi.org/10.1038/onc.2017.78
Joseph B, Marchetti P, Formstecher P, Kroemer G, Lewensohn R, Zhivotovsky B. Mitochondrial dysfunction is an essential step for killing of non-small cell lung carcinomas resistant to conventional treatment. Oncogene. 2017 Aug 17;36(33):4818. doi: 10.1038/onc.2017.78. Epub 2017 Apr 10. PMID: 28394341.
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Corrigendum to: "Mitochondrial targeting of α-tocopheryl succinate enhances its pro-apoptotic efficacy: A new paradigm for effective cancer therapy" [Free Radic Biol Med. 50 (2011) 1546-1555].

Free radical biology & medicine

Dong LF, Jameson VJA, Tilly D, Prochazka L, Rohlena J, Valis K, Truksa J, Zobalova R, Mahdavian E, Kluckova K, Stantic M, Stursa J, Freeman R, Witting PK, Norberg E, Goodwin J, Salvatore BA, Novotna J, Turanek J, Ledvina M, Hozak P, Zhivotovsky B, Coster MJ, Ralph SJ, Smith RAJ, Neuzil J.
PMID: 30184722
Free Radic Biol Med. 2013 Dec;65:895-896. doi: 10.1016/j.freeradbiomed.2013.08.164. Epub 2013 Aug 29.

No abstract available.

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Dong LF, Jameson VJA, Tilly D, et al. Corrigendum to: "Mitochondrial targeting of α-tocopheryl succinate enhances its pro-apoptotic efficacy: A new paradigm for effective cancer therapy" [Free Radic Biol Med. 50 (2011) 1546-1555]. Free Radic Biol Med. 2013;65:895-896doi: 10.1016/j.freeradbiomed.2013.08.164.
Dong, L. F., Jameson, V. J. A., Tilly, D., Prochazka, L., Rohlena, J., Valis, K., Truksa, J., Zobalova, R., Mahdavian, E., Kluckova, K., Stantic, M., Stursa, J., Freeman, R., Witting, P. K., Norberg, E., Goodwin, J., Salvatore, B. A., Novotna, J., Turanek, J., Ledvina, M., Hozak, P., Zhivotovsky, B., Coster, M. J., Ralph, S. J., Smith, R. A. J., & Neuzil, J. (2013). Corrigendum to: "Mitochondrial targeting of α-tocopheryl succinate enhances its pro-apoptotic efficacy: A new paradigm for effective cancer therapy" [Free Radic Biol Med. 50 (2011) 1546-1555]. Free radical biology & medicine, 65895-896. https://doi.org/10.1016/j.freeradbiomed.2013.08.164
Dong, Lan-Feng, et al. "Corrigendum to: "Mitochondrial targeting of α-tocopheryl succinate enhances its pro-apoptotic efficacy: A new paradigm for effective cancer therapy" [Free Radic Biol Med. 50 (2011) 1546-1555]." Free radical biology & medicine vol. 65 (2013): 895-896. doi: https://doi.org/10.1016/j.freeradbiomed.2013.08.164
Dong LF, Jameson VJA, Tilly D, Prochazka L, Rohlena J, Valis K, Truksa J, Zobalova R, Mahdavian E, Kluckova K, Stantic M, Stursa J, Freeman R, Witting PK, Norberg E, Goodwin J, Salvatore BA, Novotna J, Turanek J, Ledvina M, Hozak P, Zhivotovsky B, Coster MJ, Ralph SJ, Smith RAJ, Neuzil J. Corrigendum to: "Mitochondrial targeting of α-tocopheryl succinate enhances its pro-apoptotic efficacy: A new paradigm for effective cancer therapy" [Free Radic Biol Med. 50 (2011) 1546-1555]. Free Radic Biol Med. 2013 Dec;65:895-896. doi: 10.1016/j.freeradbiomed.2013.08.164. Epub 2013 Aug 29. PMID: 30184722.
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Mitochondrial Involvement in Migration, Invasion and Metastasis.

Frontiers in cell and developmental biology

Denisenko TV, Gorbunova AS, Zhivotovsky B.
PMID: 31921862
Front Cell Dev Biol. 2019 Dec 20;7:355. doi: 10.3389/fcell.2019.00355. eCollection 2019.

Mitochondria in addition to be a main cellular power station, are involved in the regulation of many physiological processes, such as generation of reactive oxygen species, metabolite production and the maintenance of the intracellular Ca

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Denisenko TV, Gorbunova AS, Zhivotovsky B. Mitochondrial Involvement in Migration, Invasion and Metastasis. Front Cell Dev Biol. 2019;7:355doi: 10.3389/fcell.2019.00355.
Denisenko, T. V., Gorbunova, A. S., & Zhivotovsky, B. (2019). Mitochondrial Involvement in Migration, Invasion and Metastasis. Frontiers in cell and developmental biology, 7355. https://doi.org/10.3389/fcell.2019.00355
Denisenko, Tatiana V, et al. "Mitochondrial Involvement in Migration, Invasion and Metastasis." Frontiers in cell and developmental biology vol. 7 (2019): 355. doi: https://doi.org/10.3389/fcell.2019.00355
Denisenko TV, Gorbunova AS, Zhivotovsky B. Mitochondrial Involvement in Migration, Invasion and Metastasis. Front Cell Dev Biol. 2019 Dec 20;7:355. doi: 10.3389/fcell.2019.00355. eCollection 2019. PMID: 31921862; PMCID: PMC6932960.
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ON THE EVOLUTION OF PHENOTYPIC PLASTICITY IN A SPATIALLY HETEROGENEOUS ENVIRONMENT.

Evolution; international journal of organic evolution

Zhivotovsky LA, Feldman MW, Bergman A.
PMID: 28568922
Evolution. 1996 Apr;50(2):547-558. doi: 10.1111/j.1558-5646.1996.tb03867.x.

A genetic model for the dynamics of a quantitative trait is analyzed in terms of gene frequencies, linkage disequilibria, and environmental effects on the trait. In a randomly mating population, at each generation progeny move to niches where they...

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Zhivotovsky LA, Feldman MW, Bergman A. ON THE EVOLUTION OF PHENOTYPIC PLASTICITY IN A SPATIALLY HETEROGENEOUS ENVIRONMENT. Evolution. 1996;50(2):547-558doi: 10.1111/j.1558-5646.1996.tb03867.x.
Zhivotovsky, L. A., Feldman, M. W., & Bergman, A. (1996). ON THE EVOLUTION OF PHENOTYPIC PLASTICITY IN A SPATIALLY HETEROGENEOUS ENVIRONMENT. Evolution; international journal of organic evolution, 50(2), 547-558. https://doi.org/10.1111/j.1558-5646.1996.tb03867.x
Zhivotovsky, Lev A, et al. "ON THE EVOLUTION OF PHENOTYPIC PLASTICITY IN A SPATIALLY HETEROGENEOUS ENVIRONMENT." Evolution; international journal of organic evolution vol. 50,2 (1996): 547-558. doi: https://doi.org/10.1111/j.1558-5646.1996.tb03867.x
Zhivotovsky LA, Feldman MW, Bergman A. ON THE EVOLUTION OF PHENOTYPIC PLASTICITY IN A SPATIALLY HETEROGENEOUS ENVIRONMENT. Evolution. 1996 Apr;50(2):547-558. doi: 10.1111/j.1558-5646.1996.tb03867.x. PMID: 28568922.
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Correction: 5-Fluorouracil-induced RNA stress engages a TRAIL-DISC-dependent apoptosis axis facilitated by p53.

Oncotarget

Akpinar B, Bracht EV, Reijnders D, Safarikova B, Jelinkova I, Grandien A, Vaculova AH, Zhivotovsky B, Olsson M.
PMID: 28053293
Oncotarget. 2016 Nov 01;7(44):72380. doi: 10.18632/oncotarget.12849.

No abstract available.

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Akpinar B, Bracht EV, Reijnders D, et al. Correction: 5-Fluorouracil-induced RNA stress engages a TRAIL-DISC-dependent apoptosis axis facilitated by p53. Oncotarget. 2016;7(44):72380doi: 10.18632/oncotarget.12849.
Akpinar, B., Bracht, E. V., Reijnders, D., Safarikova, B., Jelinkova, I., Grandien, A., Vaculova, A. H., Zhivotovsky, B., & Olsson, M. (2016). Correction: 5-Fluorouracil-induced RNA stress engages a TRAIL-DISC-dependent apoptosis axis facilitated by p53. Oncotarget, 7(44), 72380. https://doi.org/10.18632/oncotarget.12849
Akpinar, Birce, et al. "Correction: 5-Fluorouracil-induced RNA stress engages a TRAIL-DISC-dependent apoptosis axis facilitated by p53." Oncotarget vol. 7,44 (2016): 72380. doi: https://doi.org/10.18632/oncotarget.12849
Akpinar B, Bracht EV, Reijnders D, Safarikova B, Jelinkova I, Grandien A, Vaculova AH, Zhivotovsky B, Olsson M. Correction: 5-Fluorouracil-induced RNA stress engages a TRAIL-DISC-dependent apoptosis axis facilitated by p53. Oncotarget. 2016 Nov 01;7(44):72380. doi: 10.18632/oncotarget.12849. PMID: 28053293; PMCID: PMC5342169.
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Inorganic mercury modifies Ca2+ signals, triggers apoptosis and potentiates NMDA toxicity in cerebellar granule neurons.

Cell death and differentiation

Rossi AD, Viviani B, Zhivotovsky B, Manzo L, Orrenius S, Vahter M, Nicotera P.
PMID: 16465247
Cell Death Differ. 1997 May;4(4):317-24. doi: 10.1038/sj.cdd.4400244.

Hg2+ (0.1 microM-0.5 microM) modified the Ca2+ signals elicited by either KCl or the glutamate-receptor agonist, N-methyl-D-aspartate (NMDA), in cerebellar granule cells (CGCs). Hg2+ enhanced the intracellular Ca2+ transient elicited by high K+ and prevented a complete recovery of...

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Rossi AD, Viviani B, Zhivotovsky B, et al. Inorganic mercury modifies Ca2+ signals, triggers apoptosis and potentiates NMDA toxicity in cerebellar granule neurons. Cell Death Differ. 1997;4(4):317-24doi: 10.1038/sj.cdd.4400244.
Rossi, A. D., Viviani, B., Zhivotovsky, B., Manzo, L., Orrenius, S., Vahter, M., & Nicotera, P. (1997). Inorganic mercury modifies Ca2+ signals, triggers apoptosis and potentiates NMDA toxicity in cerebellar granule neurons. Cell death and differentiation, 4(4), 317-24. https://doi.org/10.1038/sj.cdd.4400244
Rossi, A D, et al. "Inorganic mercury modifies Ca2+ signals, triggers apoptosis and potentiates NMDA toxicity in cerebellar granule neurons." Cell death and differentiation vol. 4,4 (1997): 317-24. doi: https://doi.org/10.1038/sj.cdd.4400244
Rossi AD, Viviani B, Zhivotovsky B, Manzo L, Orrenius S, Vahter M, Nicotera P. Inorganic mercury modifies Ca2+ signals, triggers apoptosis and potentiates NMDA toxicity in cerebellar granule neurons. Cell Death Differ. 1997 May;4(4):317-24. doi: 10.1038/sj.cdd.4400244. PMID: 16465247.
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PRIMA-1Met induces mitochondrial apoptosis through activation of caspase-2.

Oncogene

Shen J, Vakifahmetoglu H, Stridh H, Zhivotovsky B, Wiman KG.
PMID: 27375019
Oncogene. 2016 Dec 15;35(50):6446. doi: 10.1038/onc.2016.210. Epub 2016 Jul 04.

No abstract available.

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Shen J, Vakifahmetoglu H, Stridh H, et al. PRIMA-1Met induces mitochondrial apoptosis through activation of caspase-2. Oncogene. 2016;35(50):6446doi: 10.1038/onc.2016.210.
Shen, J., Vakifahmetoglu, H., Stridh, H., Zhivotovsky, B., & Wiman, K. G. (2016). PRIMA-1Met induces mitochondrial apoptosis through activation of caspase-2. Oncogene, 35(50), 6446. https://doi.org/10.1038/onc.2016.210
Shen, J, et al. "PRIMA-1Met induces mitochondrial apoptosis through activation of caspase-2." Oncogene vol. 35,50 (2016): 6446. doi: https://doi.org/10.1038/onc.2016.210
Shen J, Vakifahmetoglu H, Stridh H, Zhivotovsky B, Wiman KG. PRIMA-1Met induces mitochondrial apoptosis through activation of caspase-2. Oncogene. 2016 Dec 15;35(50):6446. doi: 10.1038/onc.2016.210. Epub 2016 Jul 04. PMID: 27375019.
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Systems biology: a way to make complex problems more understandable.

Cell death & disease

Lavrik IN, Zhivotovsky B.
PMID: 24874728
Cell Death Dis. 2014 May 29;5:e1256. doi: 10.1038/cddis.2014.195.

No abstract available.

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Lavrik IN, Zhivotovsky B. Systems biology: a way to make complex problems more understandable. Cell Death Dis. 2014;5:e1256doi: 10.1038/cddis.2014.195.
Lavrik, I. N., & Zhivotovsky, B. (2014). Systems biology: a way to make complex problems more understandable. Cell death & disease, 5e1256. https://doi.org/10.1038/cddis.2014.195
Lavrik, I N, and Zhivotovsky, B. "Systems biology: a way to make complex problems more understandable." Cell death & disease vol. 5 (2014): e1256. doi: https://doi.org/10.1038/cddis.2014.195
Lavrik IN, Zhivotovsky B. Systems biology: a way to make complex problems more understandable. Cell Death Dis. 2014 May 29;5:e1256. doi: 10.1038/cddis.2014.195. PMID: 24874728; PMCID: PMC4047879.
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Biodegradable Porous Silicon Nanocontainers as an Effective Drug Carrier for Regulation of the Tumor Cell Death Pathways.

ACS biomaterials science & engineering

Maximchik PV, Tamarov K, Sheval EV, Tolstik E, Kirchberger-Tolstik T, Yang Z, Sivakov V, Zhivotovsky B, Osminkina LA.
PMID: 33405660
ACS Biomater Sci Eng. 2019 Nov 11;5(11):6063-6071. doi: 10.1021/acsbiomaterials.9b01292. Epub 2019 Oct 25.

Nanocontainers based on solid materials have great potential for drug delivery applications. However, since nanocontainer-mediated delivery can alter the drug internalization pathways and metabolism, it is important to find out what are the mechanisms of cancer cell death induced...

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Maximchik PV, Tamarov K, Sheval EV, et al. Biodegradable Porous Silicon Nanocontainers as an Effective Drug Carrier for Regulation of the Tumor Cell Death Pathways. ACS Biomater Sci Eng. 2019;5(11):6063-6071doi: 10.1021/acsbiomaterials.9b01292.
Maximchik, P. V., Tamarov, K., Sheval, E. V., Tolstik, E., Kirchberger-Tolstik, T., Yang, Z., Sivakov, V., Zhivotovsky, B., & Osminkina, L. A. (2019). Biodegradable Porous Silicon Nanocontainers as an Effective Drug Carrier for Regulation of the Tumor Cell Death Pathways. ACS biomaterials science & engineering, 5(11), 6063-6071. https://doi.org/10.1021/acsbiomaterials.9b01292
Maximchik, Polina V, et al. "Biodegradable Porous Silicon Nanocontainers as an Effective Drug Carrier for Regulation of the Tumor Cell Death Pathways." ACS biomaterials science & engineering vol. 5,11 (2019): 6063-6071. doi: https://doi.org/10.1021/acsbiomaterials.9b01292
Maximchik PV, Tamarov K, Sheval EV, Tolstik E, Kirchberger-Tolstik T, Yang Z, Sivakov V, Zhivotovsky B, Osminkina LA. Biodegradable Porous Silicon Nanocontainers as an Effective Drug Carrier for Regulation of the Tumor Cell Death Pathways. ACS Biomater Sci Eng. 2019 Nov 11;5(11):6063-6071. doi: 10.1021/acsbiomaterials.9b01292. Epub 2019 Oct 25. PMID: 33405660.
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Mitochondria-targeted betulinic and ursolic acid derivatives: synthesis and anticancer activity.

MedChemComm

Nedopekina DA, Gubaidullin RR, Odinokov VN, Maximchik PV, Zhivotovsky B, Bel'skii YP, Khazanov VA, Manuylova AV, Gogvadze V, Spivak AY.
PMID: 30108714
Medchemcomm. 2017 Sep 13;8(10):1934-1945. doi: 10.1039/c7md00248c. eCollection 2017 Oct 01.

A series of new betulinic and ursolic acid conjugates with a lipophilic triphenylphosphonium cation, meant to enhance the bioavailability and mitochondriotropic action of natural triterpenes, have been synthesized. The

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Nedopekina DA, Gubaidullin RR, Odinokov VN, et al. Mitochondria-targeted betulinic and ursolic acid derivatives: synthesis and anticancer activity. Medchemcomm. 2017;8(10):1934-1945doi: 10.1039/c7md00248c.
Nedopekina, D. A., Gubaidullin, R. R., Odinokov, V. N., Maximchik, P. V., Zhivotovsky, B., Bel'skii, Y. P., Khazanov, V. A., Manuylova, A. V., Gogvadze, V., & Spivak, A. Y. (2017). Mitochondria-targeted betulinic and ursolic acid derivatives: synthesis and anticancer activity. MedChemComm, 8(10), 1934-1945. https://doi.org/10.1039/c7md00248c
Nedopekina, Darya A, et al. "Mitochondria-targeted betulinic and ursolic acid derivatives: synthesis and anticancer activity." MedChemComm vol. 8,10 (2017): 1934-1945. doi: https://doi.org/10.1039/c7md00248c
Nedopekina DA, Gubaidullin RR, Odinokov VN, Maximchik PV, Zhivotovsky B, Bel'skii YP, Khazanov VA, Manuylova AV, Gogvadze V, Spivak AY. Mitochondria-targeted betulinic and ursolic acid derivatives: synthesis and anticancer activity. Medchemcomm. 2017 Sep 13;8(10):1934-1945. doi: 10.1039/c7md00248c. eCollection 2017 Oct 01. PMID: 30108714; PMCID: PMC6072465.
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Showing 25 to 36 of 50 entries