Advanced Search
Display options
Filter resources
Text Availability
Article type
Publication date
Species
Language
Sex
Age
Showing 1 to 12 of 79 entries
Sorted by: Best Match Show Resources per page
Ectopic Pituitary Adenomas Presenting as Sphenoid or Clival Lesions: Case Series and Management Recommendations.

Journal of neurological surgery. Part B, Skull base

Tajudeen BA, Kuan EC, Adappa ND, Han JK, Chandra RK, Palmer JN, Kennedy DW, Wang MB, Suh JD.
PMID: 28321374
J Neurol Surg B Skull Base. 2017 Apr;78(2):120-124. doi: 10.1055/s-0036-1592081. Epub 2016 Sep 09.

No abstract available.

Cite
Tajudeen BA, Kuan EC, Adappa ND, et al. Ectopic Pituitary Adenomas Presenting as Sphenoid or Clival Lesions: Case Series and Management Recommendations. J Neurol Surg B Skull Base. 2016;78(2):120-124doi: 10.1055/s-0036-1592081.
Tajudeen, B. A., Kuan, E. C., Adappa, N. D., Han, J. K., Chandra, R. K., Palmer, J. N., Kennedy, D. W., Wang, M. B., & Suh, J. D. (2017). Ectopic Pituitary Adenomas Presenting as Sphenoid or Clival Lesions: Case Series and Management Recommendations. Journal of neurological surgery. Part B, Skull base, 78(2), 120-124. https://doi.org/10.1055/s-0036-1592081
Tajudeen, Bobby A, et al. "Ectopic Pituitary Adenomas Presenting as Sphenoid or Clival Lesions: Case Series and Management Recommendations." Journal of neurological surgery. Part B, Skull base vol. 78,2 (2017): 120-124. doi: https://doi.org/10.1055/s-0036-1592081
Tajudeen BA, Kuan EC, Adappa ND, Han JK, Chandra RK, Palmer JN, Kennedy DW, Wang MB, Suh JD. Ectopic Pituitary Adenomas Presenting as Sphenoid or Clival Lesions: Case Series and Management Recommendations. J Neurol Surg B Skull Base. 2017 Apr;78(2):120-124. doi: 10.1055/s-0036-1592081. Epub 2016 Sep 09. PMID: 28321374; PMCID: PMC5357230.
Copy Download .nbib Format: NLM
Metastatic Breast Cancer with Extensive Osseous Metastasis Presenting with Symptomatic Immune Thrombocytopenic Purpura and Anemia: A Case Report and Review of the Literature.

Case reports in oncology

Niu J, Goldin T, Markman M, Kundranda MN.
PMID: 26120311
Case Rep Oncol. 2015 Jun 05;8(2):256-63. doi: 10.1159/000431213. eCollection 2015.

BACKGROUND: Immune thrombocytopenic purpura (ITP) is a rare acquired bleeding disorder with an estimated incidence of 1 in 10,000 people in the general population. The association of ITP with breast cancer is an even rarer entity with very limited...

Cite
Niu J, Goldin T, Markman M, et al. Metastatic Breast Cancer with Extensive Osseous Metastasis Presenting with Symptomatic Immune Thrombocytopenic Purpura and Anemia: A Case Report and Review of the Literature. Case Rep Oncol. 2015;8(2):256-63doi: 10.1159/000431213.
Niu, J., Goldin, T., Markman, M., & Kundranda, M. N. (2015). Metastatic Breast Cancer with Extensive Osseous Metastasis Presenting with Symptomatic Immune Thrombocytopenic Purpura and Anemia: A Case Report and Review of the Literature. Case reports in oncology, 8(2), 256-63. https://doi.org/10.1159/000431213
Niu, Jiaxin, et al. "Metastatic Breast Cancer with Extensive Osseous Metastasis Presenting with Symptomatic Immune Thrombocytopenic Purpura and Anemia: A Case Report and Review of the Literature." Case reports in oncology vol. 8,2 (2015): 256-63. doi: https://doi.org/10.1159/000431213
Niu J, Goldin T, Markman M, Kundranda MN. Metastatic Breast Cancer with Extensive Osseous Metastasis Presenting with Symptomatic Immune Thrombocytopenic Purpura and Anemia: A Case Report and Review of the Literature. Case Rep Oncol. 2015 Jun 05;8(2):256-63. doi: 10.1159/000431213. eCollection 2015. PMID: 26120311; PMCID: PMC4478340.
Copy Download .nbib Format: NLM
Incidence and clinical significance of ESR1 mutations in heavily pretreated metastatic breast cancer patients.

OncoTargets and therapy

Niu J, Andres G, Kramer K, Kundranda MN, Alvarez RH, Klimant E, Parikh AR, Tan B, Staren ED, Markman M.
PMID: 26648736
Onco Targets Ther. 2015 Nov 11;8:3323-8. doi: 10.2147/OTT.S92443. eCollection 2015.

BACKGROUND: ESR1 mutation has recently emerged as one of the important mechanisms involved in endocrine resistance. The incidence and clinical implication of ESR1 mutation has not been well evaluated in heavily pretreated breast cancer patients.METHODS: We conducted a retrospective...

Cite
Niu J, Andres G, Kramer K, et al. Incidence and clinical significance of ESR1 mutations in heavily pretreated metastatic breast cancer patients. Onco Targets Ther. 2015;8:3323-8doi: 10.2147/OTT.S92443.
Niu, J., Andres, G., Kramer, K., Kundranda, M. N., Alvarez, R. H., Klimant, E., Parikh, A. R., Tan, B., Staren, E. D., & Markman, M. (2015). Incidence and clinical significance of ESR1 mutations in heavily pretreated metastatic breast cancer patients. OncoTargets and therapy, 83323-8. https://doi.org/10.2147/OTT.S92443
Niu, Jiaxin, et al. "Incidence and clinical significance of ESR1 mutations in heavily pretreated metastatic breast cancer patients." OncoTargets and therapy vol. 8 (2015): 3323-8. doi: https://doi.org/10.2147/OTT.S92443
Niu J, Andres G, Kramer K, Kundranda MN, Alvarez RH, Klimant E, Parikh AR, Tan B, Staren ED, Markman M. Incidence and clinical significance of ESR1 mutations in heavily pretreated metastatic breast cancer patients. Onco Targets Ther. 2015 Nov 11;8:3323-8. doi: 10.2147/OTT.S92443. eCollection 2015. PMID: 26648736; PMCID: PMC4648593.
Copy Download .nbib Format: NLM
Intra-operative haemodynamic volatility in a patient undergoing retroperitoneal cyst excision.

Indian journal of anaesthesia

Tantry TP, Shenoy SP, Shetty P, Adappa KK.
PMID: 22701212
Indian J Anaesth. 2012 Mar;56(2):175-8. doi: 10.4103/0019-5049.96341.

Excision of a suspected retroperitoneal, duodenal duplication cyst was performed in a pre-operatively normotensive patient under combined epidural and general anaesthesia. Intraoperatively, the cystic tumour was discovered to be a retroperitoneal mass, free from duodenal or adrenal origin. Development...

Cite
Tantry TP, Shenoy SP, Shetty P, et al. Intra-operative haemodynamic volatility in a patient undergoing retroperitoneal cyst excision. Indian J Anaesth. 2012;56(2):175-8doi: 10.4103/0019-5049.96341.
Tantry, T. P., Shenoy, S. P., Shetty, P., & Adappa, K. K. (2012). Intra-operative haemodynamic volatility in a patient undergoing retroperitoneal cyst excision. Indian journal of anaesthesia, 56(2), 175-8. https://doi.org/10.4103/0019-5049.96341
Tantry, Thrivikrama Padur, et al. "Intra-operative haemodynamic volatility in a patient undergoing retroperitoneal cyst excision." Indian journal of anaesthesia vol. 56,2 (2012): 175-8. doi: https://doi.org/10.4103/0019-5049.96341
Tantry TP, Shenoy SP, Shetty P, Adappa KK. Intra-operative haemodynamic volatility in a patient undergoing retroperitoneal cyst excision. Indian J Anaesth. 2012 Mar;56(2):175-8. doi: 10.4103/0019-5049.96341. PMID: 22701212; PMCID: PMC3371496.
Copy Download .nbib Format: NLM
A catecholamine-secreting skull base sinonasal paraganglioma presenting with labile hypertension in a patient with previously undiagnosed genetic mutation.

Journal of neurological surgery reports

Hahn S, Palmer JN, Adappa ND.
PMID: 23946921
J Neurol Surg Rep. 2012 Oct;73(1):19-24. doi: 10.1055/s-0032-1301408. Epub 2012 Feb 17.

Sinonasal paragangliomas are very uncommon neuroendocrine tumors that can present as skull base lesions. Functional paragangliomas are exceedingly rare. They can be associated with genetic mutations that have been associated with increased risk of head and neck paragangliomas. We...

Cite
Hahn S, Palmer JN, Adappa ND. A catecholamine-secreting skull base sinonasal paraganglioma presenting with labile hypertension in a patient with previously undiagnosed genetic mutation. J Neurol Surg Rep. 2012;73(1):19-24doi: 10.1055/s-0032-1301408.
Hahn, S., Palmer, J. N., & Adappa, N. D. (2012). A catecholamine-secreting skull base sinonasal paraganglioma presenting with labile hypertension in a patient with previously undiagnosed genetic mutation. Journal of neurological surgery reports, 73(1), 19-24. https://doi.org/10.1055/s-0032-1301408
Hahn, Samuel, et al. "A catecholamine-secreting skull base sinonasal paraganglioma presenting with labile hypertension in a patient with previously undiagnosed genetic mutation." Journal of neurological surgery reports vol. 73,1 (2012): 19-24. doi: https://doi.org/10.1055/s-0032-1301408
Hahn S, Palmer JN, Adappa ND. A catecholamine-secreting skull base sinonasal paraganglioma presenting with labile hypertension in a patient with previously undiagnosed genetic mutation. J Neurol Surg Rep. 2012 Oct;73(1):19-24. doi: 10.1055/s-0032-1301408. Epub 2012 Feb 17. PMID: 23946921; PMCID: PMC3658654.
Copy Download .nbib Format: NLM
Tramadol-induced respiratory depression in a morbidly obese patient with normal renal function.

Indian journal of anaesthesia

Tantry TP, Kadam D, Shetty P, Adappa KK.
PMID: 21808420
Indian J Anaesth. 2011 May;55(3):318-20. doi: 10.4103/0019-5049.82667.

No abstract available.

Cite
Tantry TP, Kadam D, Shetty P, et al. Tramadol-induced respiratory depression in a morbidly obese patient with normal renal function. Indian J Anaesth. 2011;55(3):318-20doi: 10.4103/0019-5049.82667.
Tantry, T. P., Kadam, D., Shetty, P., & Adappa, K. K. (2011). Tramadol-induced respiratory depression in a morbidly obese patient with normal renal function. Indian journal of anaesthesia, 55(3), 318-20. https://doi.org/10.4103/0019-5049.82667
Tantry, Thrivikrama P, et al. "Tramadol-induced respiratory depression in a morbidly obese patient with normal renal function." Indian journal of anaesthesia vol. 55,3 (2011): 318-20. doi: https://doi.org/10.4103/0019-5049.82667
Tantry TP, Kadam D, Shetty P, Adappa KK. Tramadol-induced respiratory depression in a morbidly obese patient with normal renal function. Indian J Anaesth. 2011 May;55(3):318-20. doi: 10.4103/0019-5049.82667. PMID: 21808420; PMCID: PMC3141172.
Copy Download .nbib Format: NLM
Solitary chemosensory cells producing interleukin-25 and group-2 innate lymphoid cells are enriched in chronic rhinosinusitis with nasal polyps.

International forum of allergy & rhinology

Patel NN, Kohanski MA, Maina IW, Triantafillou V, Workman AD, Tong CCL, Kuan EC, Bosso JV, Adappa ND, Palmer JN, Herbert DR, Cohen NA.
PMID: 29742315
Int Forum Allergy Rhinol. 2018 May 09; doi: 10.1002/alr.22142. Epub 2018 May 09.

BACKGROUND: Chronic rhinosinusitis with nasal polyps (CRSwNP) is commonly characterized by type-2 inflammation. It is established that group-2 innate lymphoid cells (ILC2s) are a subset of immune cells important in orchestrating mucosal type-2 response. IL-25 is an epithelial-derived cytokine...

Cite
Patel NN, Kohanski MA, Maina IW, et al. Solitary chemosensory cells producing interleukin-25 and group-2 innate lymphoid cells are enriched in chronic rhinosinusitis with nasal polyps. Int Forum Allergy Rhinol. 2018;doi: 10.1002/alr.22142.
Patel, N. N., Kohanski, M. A., Maina, I. W., Triantafillou, V., Workman, A. D., Tong, C. C. L., Kuan, E. C., Bosso, J. V., Adappa, N. D., Palmer, J. N., Herbert, D. R., & Cohen, N. A. (2018). Solitary chemosensory cells producing interleukin-25 and group-2 innate lymphoid cells are enriched in chronic rhinosinusitis with nasal polyps. International forum of allergy & rhinology, . https://doi.org/10.1002/alr.22142
Patel, Neil N, et al. "Solitary chemosensory cells producing interleukin-25 and group-2 innate lymphoid cells are enriched in chronic rhinosinusitis with nasal polyps." International forum of allergy & rhinology vol. (2018). doi: https://doi.org/10.1002/alr.22142
Patel NN, Kohanski MA, Maina IW, Triantafillou V, Workman AD, Tong CCL, Kuan EC, Bosso JV, Adappa ND, Palmer JN, Herbert DR, Cohen NA. Solitary chemosensory cells producing interleukin-25 and group-2 innate lymphoid cells are enriched in chronic rhinosinusitis with nasal polyps. Int Forum Allergy Rhinol. 2018 May 09; doi: 10.1002/alr.22142. Epub 2018 May 09. PMID: 29742315; PMCID: PMC6226383.
Copy Download .nbib Format: NLM
Intrasinus penetration of a silastic malar implant, which resulted in chronic sinusitis: A case report and literature review.

Allergy & rhinology (Providence, R.I.)

Kim LY, Schwartz JS, Tajudeen BA, Adappa ND, Palmer JN.
PMID: 28381326
Allergy Rhinol (Providence). 2017 Mar 01;8(1):37-39. doi: 10.2500/ar.2017.8.0189.

BACKGROUND: Malar augmentation is a common cosmetic procedure utilizing silastic materials. We describe an uncommon complication of a silastic implant eroding into the anterior maxillary sinus wall resulting in chronic rhinosinusitis (CRS).METHODS: A literature review is presented describing the...

Cite
Kim LY, Schwartz JS, Tajudeen BA, et al. Intrasinus penetration of a silastic malar implant, which resulted in chronic sinusitis: A case report and literature review. Allergy Rhinol (Providence). 2017;8(1):37-39doi: 10.2500/ar.2017.8.0189.
Kim, L. Y., Schwartz, J. S., Tajudeen, B. A., Adappa, N. D., & Palmer, J. N. (2017). Intrasinus penetration of a silastic malar implant, which resulted in chronic sinusitis: A case report and literature review. Allergy & rhinology (Providence, R.I.), 8(1), 37-39. https://doi.org/10.2500/ar.2017.8.0189
Kim, Luke Y, et al. "Intrasinus penetration of a silastic malar implant, which resulted in chronic sinusitis: A case report and literature review." Allergy & rhinology (Providence, R.I.) vol. 8,1 (2017): 37-39. doi: https://doi.org/10.2500/ar.2017.8.0189
Kim LY, Schwartz JS, Tajudeen BA, Adappa ND, Palmer JN. Intrasinus penetration of a silastic malar implant, which resulted in chronic sinusitis: A case report and literature review. Allergy Rhinol (Providence). 2017 Mar 01;8(1):37-39. doi: 10.2500/ar.2017.8.0189. PMID: 28381326; PMCID: PMC5380451.
Copy Download .nbib Format: NLM
The bitter end: T2R bitter receptor agonists elevate nuclear calcium and induce apoptosis in non-ciliated airway epithelial cells.

Cell calcium

McMahon DB, Kuek LE, Johnson ME, Johnson PO, Horn RLJ, Carey RM, Adappa ND, Palmer JN, Lee RJ.
PMID: 34839223
Cell Calcium. 2021 Nov 08;101:102499. doi: 10.1016/j.ceca.2021.102499. Epub 2021 Nov 08.

Bitter taste receptors (T2Rs) localize to airway motile cilia and initiate innate immune responses in retaliation to bacterial quorum sensing molecules. Activation of cilia T2Rs leads to calcium-driven NO production that increases cilia beating and directly kills bacteria. Several...

Cite
McMahon DB, Kuek LE, Johnson ME, et al. The bitter end: T2R bitter receptor agonists elevate nuclear calcium and induce apoptosis in non-ciliated airway epithelial cells. Cell Calcium. 2021;101:102499doi: 10.1016/j.ceca.2021.102499.
McMahon, D. B., Kuek, L. E., Johnson, M. E., Johnson, P. O., Horn, R. L. J., Carey, R. M., Adappa, N. D., Palmer, J. N., & Lee, R. J. (2021). The bitter end: T2R bitter receptor agonists elevate nuclear calcium and induce apoptosis in non-ciliated airway epithelial cells. Cell calcium, 101102499. https://doi.org/10.1016/j.ceca.2021.102499
McMahon, Derek B, et al. "The bitter end: T2R bitter receptor agonists elevate nuclear calcium and induce apoptosis in non-ciliated airway epithelial cells." Cell calcium vol. 101 (2021): 102499. doi: https://doi.org/10.1016/j.ceca.2021.102499
McMahon DB, Kuek LE, Johnson ME, Johnson PO, Horn RLJ, Carey RM, Adappa ND, Palmer JN, Lee RJ. The bitter end: T2R bitter receptor agonists elevate nuclear calcium and induce apoptosis in non-ciliated airway epithelial cells. Cell Calcium. 2021 Nov 08;101:102499. doi: 10.1016/j.ceca.2021.102499. Epub 2021 Nov 08. PMID: 34839223.
Copy Download .nbib Format: NLM
The bitter end: T2R bitter receptor agonists elevate nuclear calcium and induce apoptosis in non-ciliated airway epithelial cells.

Cell calcium

McMahon DB, Kuek LE, Johnson ME, Johnson PO, Horn RLJ, Carey RM, Adappa ND, Palmer JN, Lee RJ.
PMID: 34839223
Cell Calcium. 2021 Nov 08;101:102499. doi: 10.1016/j.ceca.2021.102499. Epub 2021 Nov 08.

Bitter taste receptors (T2Rs) localize to airway motile cilia and initiate innate immune responses in retaliation to bacterial quorum sensing molecules. Activation of cilia T2Rs leads to calcium-driven NO production that increases cilia beating and directly kills bacteria. Several...

Cite
McMahon DB, Kuek LE, Johnson ME, et al. The bitter end: T2R bitter receptor agonists elevate nuclear calcium and induce apoptosis in non-ciliated airway epithelial cells. Cell Calcium. 2021;101:102499doi: 10.1016/j.ceca.2021.102499.
McMahon, D. B., Kuek, L. E., Johnson, M. E., Johnson, P. O., Horn, R. L. J., Carey, R. M., Adappa, N. D., Palmer, J. N., & Lee, R. J. (2021). The bitter end: T2R bitter receptor agonists elevate nuclear calcium and induce apoptosis in non-ciliated airway epithelial cells. Cell calcium, 101102499. https://doi.org/10.1016/j.ceca.2021.102499
McMahon, Derek B, et al. "The bitter end: T2R bitter receptor agonists elevate nuclear calcium and induce apoptosis in non-ciliated airway epithelial cells." Cell calcium vol. 101 (2021): 102499. doi: https://doi.org/10.1016/j.ceca.2021.102499
McMahon DB, Kuek LE, Johnson ME, Johnson PO, Horn RLJ, Carey RM, Adappa ND, Palmer JN, Lee RJ. The bitter end: T2R bitter receptor agonists elevate nuclear calcium and induce apoptosis in non-ciliated airway epithelial cells. Cell Calcium. 2021 Nov 08;101:102499. doi: 10.1016/j.ceca.2021.102499. Epub 2021 Nov 08. PMID: 34839223.
Copy Download .nbib Format: NLM
The bitter end: T2R bitter receptor agonists elevate nuclear calcium and induce apoptosis in non-ciliated airway epithelial cells.

Cell calcium

McMahon DB, Kuek LE, Johnson ME, Johnson PO, Horn RLJ, Carey RM, Adappa ND, Palmer JN, Lee RJ.
PMID: 34839223
Cell Calcium. 2021 Nov 08;101:102499. doi: 10.1016/j.ceca.2021.102499. Epub 2021 Nov 08.

Bitter taste receptors (T2Rs) localize to airway motile cilia and initiate innate immune responses in retaliation to bacterial quorum sensing molecules. Activation of cilia T2Rs leads to calcium-driven NO production that increases cilia beating and directly kills bacteria. Several...

Cite
McMahon DB, Kuek LE, Johnson ME, et al. The bitter end: T2R bitter receptor agonists elevate nuclear calcium and induce apoptosis in non-ciliated airway epithelial cells. Cell Calcium. 2021;101:102499doi: 10.1016/j.ceca.2021.102499.
McMahon, D. B., Kuek, L. E., Johnson, M. E., Johnson, P. O., Horn, R. L. J., Carey, R. M., Adappa, N. D., Palmer, J. N., & Lee, R. J. (2021). The bitter end: T2R bitter receptor agonists elevate nuclear calcium and induce apoptosis in non-ciliated airway epithelial cells. Cell calcium, 101102499. https://doi.org/10.1016/j.ceca.2021.102499
McMahon, Derek B, et al. "The bitter end: T2R bitter receptor agonists elevate nuclear calcium and induce apoptosis in non-ciliated airway epithelial cells." Cell calcium vol. 101 (2021): 102499. doi: https://doi.org/10.1016/j.ceca.2021.102499
McMahon DB, Kuek LE, Johnson ME, Johnson PO, Horn RLJ, Carey RM, Adappa ND, Palmer JN, Lee RJ. The bitter end: T2R bitter receptor agonists elevate nuclear calcium and induce apoptosis in non-ciliated airway epithelial cells. Cell Calcium. 2021 Nov 08;101:102499. doi: 10.1016/j.ceca.2021.102499. Epub 2021 Nov 08. PMID: 34839223.
Copy Download .nbib Format: NLM
The bitter end: T2R bitter receptor agonists elevate nuclear calcium and induce apoptosis in non-ciliated airway epithelial cells.

Cell calcium

McMahon DB, Kuek LE, Johnson ME, Johnson PO, Horn RLJ, Carey RM, Adappa ND, Palmer JN, Lee RJ.
PMID: 34839223
Cell Calcium. 2021 Nov 08;101:102499. doi: 10.1016/j.ceca.2021.102499. Epub 2021 Nov 08.

Bitter taste receptors (T2Rs) localize to airway motile cilia and initiate innate immune responses in retaliation to bacterial quorum sensing molecules. Activation of cilia T2Rs leads to calcium-driven NO production that increases cilia beating and directly kills bacteria. Several...

Cite
McMahon DB, Kuek LE, Johnson ME, et al. The bitter end: T2R bitter receptor agonists elevate nuclear calcium and induce apoptosis in non-ciliated airway epithelial cells. Cell Calcium. 2021;101:102499doi: 10.1016/j.ceca.2021.102499.
McMahon, D. B., Kuek, L. E., Johnson, M. E., Johnson, P. O., Horn, R. L. J., Carey, R. M., Adappa, N. D., Palmer, J. N., & Lee, R. J. (2021). The bitter end: T2R bitter receptor agonists elevate nuclear calcium and induce apoptosis in non-ciliated airway epithelial cells. Cell calcium, 101102499. https://doi.org/10.1016/j.ceca.2021.102499
McMahon, Derek B, et al. "The bitter end: T2R bitter receptor agonists elevate nuclear calcium and induce apoptosis in non-ciliated airway epithelial cells." Cell calcium vol. 101 (2021): 102499. doi: https://doi.org/10.1016/j.ceca.2021.102499
McMahon DB, Kuek LE, Johnson ME, Johnson PO, Horn RLJ, Carey RM, Adappa ND, Palmer JN, Lee RJ. The bitter end: T2R bitter receptor agonists elevate nuclear calcium and induce apoptosis in non-ciliated airway epithelial cells. Cell Calcium. 2021 Nov 08;101:102499. doi: 10.1016/j.ceca.2021.102499. Epub 2021 Nov 08. PMID: 34839223.
Copy Download .nbib Format: NLM
Showing 1 to 12 of 79 entries