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[The current status and future prospect of the 2-year-nursing course].

[Kango kyoiku] Japanese journal of nurses' education

Iribe H, Asohina S, Maeda K, Goto K, Yamomoto F, Yamashita K, Nobuchika H.
PMID: 6902791
Kango Kyoiku. 1980 Aug;21(8):489-500.

No abstract available.

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Iribe H, Asohina S, Maeda K, et al. [The current status and future prospect of the 2-year-nursing course]. Kango Kyoiku. 1980;21(8):489-500
Iribe, H., Asohina, S., Maeda, K., Goto, K., Yamomoto, F., Yamashita, K., & Nobuchika, H. (1980). [The current status and future prospect of the 2-year-nursing course]. [Kango kyoiku] Japanese journal of nurses' education, 21(8), 489-500.
Iribe, H, et al. "[The current status and future prospect of the 2-year-nursing course]." [Kango kyoiku] Japanese journal of nurses' education vol. 21,8 (1980): 489-500.
Iribe H, Asohina S, Maeda K, Goto K, Yamomoto F, Yamashita K, Nobuchika H. [The current status and future prospect of the 2-year-nursing course]. Kango Kyoiku. 1980 Aug;21(8):489-500. Japanese. PMID: 6902791.
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Gene-Activated Matrix Comprised of Atelocollagen and Plasmid DNA Encoding BMP4 or Runx2 Promotes Rat Cranial Bone Augmentation.

BioResearch open access

Umebayashi M, Sumita Y, Kawai Y, Watanabe S, Asahina I.
PMID: 26309793
Biores Open Access. 2015 Feb 01;4(1):164-74. doi: 10.1089/biores.2014.0057. eCollection 2015.

To date, therapeutic method for in vivo gene delivery has not been established on bone engineering though its potential usefulness has been suggested. For clinical applications, an effective condition should be developed to transfer the genes in vivo without...

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Umebayashi M, Sumita Y, Kawai Y, et al. Gene-Activated Matrix Comprised of Atelocollagen and Plasmid DNA Encoding BMP4 or Runx2 Promotes Rat Cranial Bone Augmentation. Biores Open Access. 2015;4(1):164-74doi: 10.1089/biores.2014.0057.
Umebayashi, M., Sumita, Y., Kawai, Y., Watanabe, S., & Asahina, I. (2015). Gene-Activated Matrix Comprised of Atelocollagen and Plasmid DNA Encoding BMP4 or Runx2 Promotes Rat Cranial Bone Augmentation. BioResearch open access, 4(1), 164-74. https://doi.org/10.1089/biores.2014.0057
Umebayashi, Mayumi, et al. "Gene-Activated Matrix Comprised of Atelocollagen and Plasmid DNA Encoding BMP4 or Runx2 Promotes Rat Cranial Bone Augmentation." BioResearch open access vol. 4,1 (2015): 164-74. doi: https://doi.org/10.1089/biores.2014.0057
Umebayashi M, Sumita Y, Kawai Y, Watanabe S, Asahina I. Gene-Activated Matrix Comprised of Atelocollagen and Plasmid DNA Encoding BMP4 or Runx2 Promotes Rat Cranial Bone Augmentation. Biores Open Access. 2015 Feb 01;4(1):164-74. doi: 10.1089/biores.2014.0057. eCollection 2015. PMID: 26309793; PMCID: PMC4497668.
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Bone morphogenetic protein-4 modulates proliferation and terminal differentiation of fetal hepatic stem/progenitor cells.

Hepatology research : the official journal of the Japan Society of Hepatology

Goto F, Kakinuma S, Miyoshi M, Tsunoda T, Kaneko S, Sato A, Asano Y, Otani S, Azuma S, Nagata H, Kawai-Kitahata F, Murakawa M, Nitta S, Itsui Y, Nakagawa M, Asahina Y, Watanabe M.
PMID: 27670640
Hepatol Res. 2017 Aug;47(9):941-952. doi: 10.1111/hepr.12823. Epub 2016 Oct 31.

Fetal hepatic stem/progenitor cells, called hepatoblasts, play central roles in liver organogenesis; however, molecular mechanisms regulating proliferation and terminal differentiation of such cells have not been completely elucidated. Bone morphogenetic protein-4 (BMP-4) is essential for the development of stem...

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Goto F, Kakinuma S, Miyoshi M, et al. Bone morphogenetic protein-4 modulates proliferation and terminal differentiation of fetal hepatic stem/progenitor cells. Hepatol Res. 2016;47(9):941-952doi: 10.1111/hepr.12823.
Goto, F., Kakinuma, S., Miyoshi, M., Tsunoda, T., Kaneko, S., Sato, A., Asano, Y., Otani, S., Azuma, S., Nagata, H., Kawai-Kitahata, F., Murakawa, M., Nitta, S., Itsui, Y., Nakagawa, M., Asahina, Y., & Watanabe, M. (2017). Bone morphogenetic protein-4 modulates proliferation and terminal differentiation of fetal hepatic stem/progenitor cells. Hepatology research : the official journal of the Japan Society of Hepatology, 47(9), 941-952. https://doi.org/10.1111/hepr.12823
Goto, Fumio, et al. "Bone morphogenetic protein-4 modulates proliferation and terminal differentiation of fetal hepatic stem/progenitor cells." Hepatology research : the official journal of the Japan Society of Hepatology vol. 47,9 (2017): 941-952. doi: https://doi.org/10.1111/hepr.12823
Goto F, Kakinuma S, Miyoshi M, Tsunoda T, Kaneko S, Sato A, Asano Y, Otani S, Azuma S, Nagata H, Kawai-Kitahata F, Murakawa M, Nitta S, Itsui Y, Nakagawa M, Asahina Y, Watanabe M. Bone morphogenetic protein-4 modulates proliferation and terminal differentiation of fetal hepatic stem/progenitor cells. Hepatol Res. 2017 Aug;47(9):941-952. doi: 10.1111/hepr.12823. Epub 2016 Oct 31. PMID: 27670640.
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Fournier's gangrene caused by Listeria monocytogenes as the primary organism.

The Canadian journal of infectious diseases & medical microbiology = Journal canadien des maladies infectieuses et de la microbiologie medicale

Asahata S, Hirai Y, Ainoda Y, Fujita T, Okada Y, Kikuchi K.
PMID: 25798155
Can J Infect Dis Med Microbiol. 2015 Jan-Feb;26(1):44-6. doi: 10.1155/2015/501386.

A 70-year-old man with a history of tongue cancer presented with Fournier's gangrene caused by Listeria monocytogenes serotype 4b. Surgical debridement revealed undiagnosed rectal adenocarcinoma. The patient did not have an apparent dietary or travel history but reported daily...

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Asahata S, Hirai Y, Ainoda Y, et al. Fournier's gangrene caused by Listeria monocytogenes as the primary organism. Can J Infect Dis Med Microbiol. 2015;26(1):44-6doi: 10.1155/2015/501386.
Asahata, S., Hirai, Y., Ainoda, Y., Fujita, T., Okada, Y., & Kikuchi, K. (2015). Fournier's gangrene caused by Listeria monocytogenes as the primary organism. The Canadian journal of infectious diseases & medical microbiology = Journal canadien des maladies infectieuses et de la microbiologie medicale, 26(1), 44-6. https://doi.org/10.1155/2015/501386
Asahata, Sayaka, et al. "Fournier's gangrene caused by Listeria monocytogenes as the primary organism." The Canadian journal of infectious diseases & medical microbiology = Journal canadien des maladies infectieuses et de la microbiologie medicale vol. 26,1 (2015): 44-6. doi: https://doi.org/10.1155/2015/501386
Asahata S, Hirai Y, Ainoda Y, Fujita T, Okada Y, Kikuchi K. Fournier's gangrene caused by Listeria monocytogenes as the primary organism. Can J Infect Dis Med Microbiol. 2015 Jan-Feb;26(1):44-6. doi: 10.1155/2015/501386. PMID: 25798155; PMCID: PMC4353270.
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Griffith Criterion for Nanoscale Stress Singularity in Brittle Silicon.

ACS nano

Sumigawa T, Shimada T, Tanaka S, Unno H, Ozaki N, Ashida S, Kitamura T.
PMID: 28549214
ACS Nano. 2017 Jun 27;11(6):6271-6276. doi: 10.1021/acsnano.7b02493. Epub 2017 Jun 01.

Brittle materials such as silicon fail via the crack nucleation and propagation, which is characterized by the singular stress field formed near the crack tip according to Griffith or fracture mechanics theory. The applicability of these continuum-based theories is,...

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Sumigawa T, Shimada T, Tanaka S, et al. Griffith Criterion for Nanoscale Stress Singularity in Brittle Silicon. ACS Nano. 2017;11(6):6271-6276doi: 10.1021/acsnano.7b02493.
Sumigawa, T., Shimada, T., Tanaka, S., Unno, H., Ozaki, N., Ashida, S., & Kitamura, T. (2017). Griffith Criterion for Nanoscale Stress Singularity in Brittle Silicon. ACS nano, 11(6), 6271-6276. https://doi.org/10.1021/acsnano.7b02493
Sumigawa, Takashi, et al. "Griffith Criterion for Nanoscale Stress Singularity in Brittle Silicon." ACS nano vol. 11,6 (2017): 6271-6276. doi: https://doi.org/10.1021/acsnano.7b02493
Sumigawa T, Shimada T, Tanaka S, Unno H, Ozaki N, Ashida S, Kitamura T. Griffith Criterion for Nanoscale Stress Singularity in Brittle Silicon. ACS Nano. 2017 Jun 27;11(6):6271-6276. doi: 10.1021/acsnano.7b02493. Epub 2017 Jun 01. PMID: 28549214.
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Corrigendum: Amino Acid Polymorphisms in Hepatitis C Virus Core Affect Infectious Virus Production and Major Histocompatibility Complex Class I Molecule Expression.

Scientific reports

Tasaka-Fujita M, Sugiyama N, Kang W, Masaki T, Murayama A, Yamada N, Sugiyama R, Tsukuda S, Watashi K, Asahina Y, Sakamoto N, Wakita T, Shin EC, Kato T.
PMID: 26670698
Sci Rep. 2015 Dec 16;5:17208. doi: 10.1038/srep17208.

No abstract available.

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Tasaka-Fujita M, Sugiyama N, Kang W, et al. Corrigendum: Amino Acid Polymorphisms in Hepatitis C Virus Core Affect Infectious Virus Production and Major Histocompatibility Complex Class I Molecule Expression. Sci Rep. 2015;5:17208doi: 10.1038/srep17208.
Tasaka-Fujita, M., Sugiyama, N., Kang, W., Masaki, T., Murayama, A., Yamada, N., Sugiyama, R., Tsukuda, S., Watashi, K., Asahina, Y., Sakamoto, N., Wakita, T., Shin, E. C., & Kato, T. (2015). Corrigendum: Amino Acid Polymorphisms in Hepatitis C Virus Core Affect Infectious Virus Production and Major Histocompatibility Complex Class I Molecule Expression. Scientific reports, 517208. https://doi.org/10.1038/srep17208
Tasaka-Fujita, Megumi, et al. "Corrigendum: Amino Acid Polymorphisms in Hepatitis C Virus Core Affect Infectious Virus Production and Major Histocompatibility Complex Class I Molecule Expression." Scientific reports vol. 5 (2015): 17208. doi: https://doi.org/10.1038/srep17208
Tasaka-Fujita M, Sugiyama N, Kang W, Masaki T, Murayama A, Yamada N, Sugiyama R, Tsukuda S, Watashi K, Asahina Y, Sakamoto N, Wakita T, Shin EC, Kato T. Corrigendum: Amino Acid Polymorphisms in Hepatitis C Virus Core Affect Infectious Virus Production and Major Histocompatibility Complex Class I Molecule Expression. Sci Rep. 2015 Dec 16;5:17208. doi: 10.1038/srep17208. PMID: 26670698; PMCID: PMC4681186.
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Histopathological and immunohistochemical study in keratocystic odontogenic tumors: Predictive factors of recurrence.

Oncology letters

Naruse T, Yamashita K, Yanamoto S, Rokutanda S, Matsushita Y, Sakamoto Y, Sakamoto H, Ikeda H, Ikeda T, Asahina I, Umeda M.
PMID: 28521454
Oncol Lett. 2017 May;13(5):3487-3493. doi: 10.3892/ol.2017.5905. Epub 2017 Mar 24.

The aim of the present study was to identify the most useful markers for predicting recurrence of keratocystic odontogenic tumors (KCOTs). A total of 65 tumor samples from 63 patients diagnosed with typical parakeratinized cysts and KCOTs between 1992...

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Naruse T, Yamashita K, Yanamoto S, et al. Histopathological and immunohistochemical study in keratocystic odontogenic tumors: Predictive factors of recurrence. Oncol Lett. 2017;13(5):3487-3493doi: 10.3892/ol.2017.5905.
Naruse, T., Yamashita, K., Yanamoto, S., Rokutanda, S., Matsushita, Y., Sakamoto, Y., Sakamoto, H., Ikeda, H., Ikeda, T., Asahina, I., & Umeda, M. (2017). Histopathological and immunohistochemical study in keratocystic odontogenic tumors: Predictive factors of recurrence. Oncology letters, 13(5), 3487-3493. https://doi.org/10.3892/ol.2017.5905
Naruse, Tomofumi, et al. "Histopathological and immunohistochemical study in keratocystic odontogenic tumors: Predictive factors of recurrence." Oncology letters vol. 13,5 (2017): 3487-3493. doi: https://doi.org/10.3892/ol.2017.5905
Naruse T, Yamashita K, Yanamoto S, Rokutanda S, Matsushita Y, Sakamoto Y, Sakamoto H, Ikeda H, Ikeda T, Asahina I, Umeda M. Histopathological and immunohistochemical study in keratocystic odontogenic tumors: Predictive factors of recurrence. Oncol Lett. 2017 May;13(5):3487-3493. doi: 10.3892/ol.2017.5905. Epub 2017 Mar 24. PMID: 28521454; PMCID: PMC5431266.
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Amyloid burden, cortical thickness, and cognitive function in the Wisconsin Registry for Alzheimer's Prevention.

Alzheimer's & dementia (Amsterdam, Netherlands)

Doherty BM, Schultz SA, Oh JM, Koscik RL, Dowling NM, Barnhart TE, Murali D, Gallagher CL, Carlsson CM, Bendlin BB, LaRue A, Hermann BP, Rowley HA, Asthana S, Sager MA, Christian BT, Johnson SC, Okonkwo OC.
PMID: 26161436
Alzheimers Dement (Amst). 2015 Jun 01;1(2):160-169. doi: 10.1016/j.dadm.2015.01.003.

There is a growing interest in understanding how amyloid-β (Aβ) accumulation in preclinical Alzheimer's disease relates to brain morphometric measures and cognition. Existing investigations in this area have been primarily conducted in older cognitively-normal (CN) individuals. Therefore, not much...

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Doherty BM, Schultz SA, Oh JM, et al. Amyloid burden, cortical thickness, and cognitive function in the Wisconsin Registry for Alzheimer's Prevention. Alzheimers Dement (Amst). 2015;1(2):160-169doi: 10.1016/j.dadm.2015.01.003.
Doherty, B. M., Schultz, S. A., Oh, J. M., Koscik, R. L., Dowling, N. M., Barnhart, T. E., Murali, D., Gallagher, C. L., Carlsson, C. M., Bendlin, B. B., LaRue, A., Hermann, B. P., Rowley, H. A., Asthana, S., Sager, M. A., Christian, B. T., Johnson, S. C., & Okonkwo, O. C. (2015). Amyloid burden, cortical thickness, and cognitive function in the Wisconsin Registry for Alzheimer's Prevention. Alzheimer's & dementia (Amsterdam, Netherlands), 1(2), 160-169. https://doi.org/10.1016/j.dadm.2015.01.003
Doherty, Benjamin M, et al. "Amyloid burden, cortical thickness, and cognitive function in the Wisconsin Registry for Alzheimer's Prevention." Alzheimer's & dementia (Amsterdam, Netherlands) vol. 1,2 (2015): 160-169. doi: https://doi.org/10.1016/j.dadm.2015.01.003
Doherty BM, Schultz SA, Oh JM, Koscik RL, Dowling NM, Barnhart TE, Murali D, Gallagher CL, Carlsson CM, Bendlin BB, LaRue A, Hermann BP, Rowley HA, Asthana S, Sager MA, Christian BT, Johnson SC, Okonkwo OC. Amyloid burden, cortical thickness, and cognitive function in the Wisconsin Registry for Alzheimer's Prevention. Alzheimers Dement (Amst). 2015 Jun 01;1(2):160-169. doi: 10.1016/j.dadm.2015.01.003. PMID: 26161436; PMCID: PMC4492165.
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Effects of Batatasin III and Its Analogs on Gibberellic Acid-Dependent α-Amylase Induction in Embryoless Barley Seeds and on Cress Growth.

Bioscience, biotechnology, and biochemistry

Asahina H, Yoshikawa H, Shuto Y.
PMID: 27388847
Biosci Biotechnol Biochem. 1998;62(8):1619-20. doi: 10.1271/bbb.62.1619.

The effects of batatasin III and its analogs on gibberellic acid (GA3)-dependent α-amylase induction in embryoless barley seeds and on cress root-growth were examined. Batatasin III was most effective and caused 68% inhibition of α-amylase induction at 4×10(-4) M,...

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Asahina H, Yoshikawa H, Shuto Y. Effects of Batatasin III and Its Analogs on Gibberellic Acid-Dependent α-Amylase Induction in Embryoless Barley Seeds and on Cress Growth. Biosci Biotechnol Biochem. 1998;62(8):1619-20doi: 10.1271/bbb.62.1619.
Asahina, H., Yoshikawa, H., & Shuto, Y. (1998). Effects of Batatasin III and Its Analogs on Gibberellic Acid-Dependent α-Amylase Induction in Embryoless Barley Seeds and on Cress Growth. Bioscience, biotechnology, and biochemistry, 62(8), 1619-20. https://doi.org/10.1271/bbb.62.1619
Asahina, H, et al. "Effects of Batatasin III and Its Analogs on Gibberellic Acid-Dependent α-Amylase Induction in Embryoless Barley Seeds and on Cress Growth." Bioscience, biotechnology, and biochemistry vol. 62,8 (1998): 1619-20. doi: https://doi.org/10.1271/bbb.62.1619
Asahina H, Yoshikawa H, Shuto Y. Effects of Batatasin III and Its Analogs on Gibberellic Acid-Dependent α-Amylase Induction in Embryoless Barley Seeds and on Cress Growth. Biosci Biotechnol Biochem. 1998;62(8):1619-20. doi: 10.1271/bbb.62.1619. PMID: 27388847.
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Cetuximab for the treatment of locally advanced and recurrent/metastatic oral cancer: An investigation of distant metastasis.

Molecular and clinical oncology

Naruse T, Yanamoto S, Matsushita Y, Sakamoto Y, Morishita K, Ohba S, Shiraishi T, Yamada SI, Asahina I, Umeda M.
PMID: 27446558
Mol Clin Oncol. 2016 Aug;5(2):246-252. doi: 10.3892/mco.2016.928. Epub 2016 Jun 13.

The aim of this retrospective study was to assess the efficacy and safety of cetuximab therapy for patients with locally advanced (LA) and recurrent/metastatic (R/M) oral squamous cell carcinoma (OSCC), with a specific focus on distant metastases (DMs). Data...

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Naruse T, Yanamoto S, Matsushita Y, et al. Cetuximab for the treatment of locally advanced and recurrent/metastatic oral cancer: An investigation of distant metastasis. Mol Clin Oncol. 2016;5(2):246-252doi: 10.3892/mco.2016.928.
Naruse, T., Yanamoto, S., Matsushita, Y., Sakamoto, Y., Morishita, K., Ohba, S., Shiraishi, T., Yamada, S. I., Asahina, I., & Umeda, M. (2016). Cetuximab for the treatment of locally advanced and recurrent/metastatic oral cancer: An investigation of distant metastasis. Molecular and clinical oncology, 5(2), 246-252. https://doi.org/10.3892/mco.2016.928
Naruse, Tomofumi, et al. "Cetuximab for the treatment of locally advanced and recurrent/metastatic oral cancer: An investigation of distant metastasis." Molecular and clinical oncology vol. 5,2 (2016): 246-252. doi: https://doi.org/10.3892/mco.2016.928
Naruse T, Yanamoto S, Matsushita Y, Sakamoto Y, Morishita K, Ohba S, Shiraishi T, Yamada SI, Asahina I, Umeda M. Cetuximab for the treatment of locally advanced and recurrent/metastatic oral cancer: An investigation of distant metastasis. Mol Clin Oncol. 2016 Aug;5(2):246-252. doi: 10.3892/mco.2016.928. Epub 2016 Jun 13. PMID: 27446558; PMCID: PMC4950138.
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A Comprehensive Profile of ChIP-Seq-Based Olig2 Target Genes in Motor Neuron Progenitor Cells Suggests the Possible Involvement of Olig2 in the Pathogenesis of Amyotrophic Lateral Sclerosis.

Journal of central nervous system disease

Satoh J, Asahina N, Kitano S, Kino Y.
PMID: 26023283
J Cent Nerv Syst Dis. 2015 May 18;7:1-14. doi: 10.4137/JCNSD.S23210. eCollection 2015.

BACKGROUND: Amyotrophic lateral sclerosis (ALS) is an intractable neurodegenerative disease that primarily affects motor neurons in the cerebral cortex and the spinal cord. Recent evidence indicates that dysfunction of oligodendrocytes is implicated in the pathogenesis of ALS. The basic...

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Satoh J, Asahina N, Kitano S, et al. A Comprehensive Profile of ChIP-Seq-Based Olig2 Target Genes in Motor Neuron Progenitor Cells Suggests the Possible Involvement of Olig2 in the Pathogenesis of Amyotrophic Lateral Sclerosis. J Cent Nerv Syst Dis. 2015;7:1-14doi: 10.4137/JCNSD.S23210.
Satoh, J., Asahina, N., Kitano, S., & Kino, Y. (2015). A Comprehensive Profile of ChIP-Seq-Based Olig2 Target Genes in Motor Neuron Progenitor Cells Suggests the Possible Involvement of Olig2 in the Pathogenesis of Amyotrophic Lateral Sclerosis. Journal of central nervous system disease, 71-14. https://doi.org/10.4137/JCNSD.S23210
Satoh, Jun-Ichi, et al. "A Comprehensive Profile of ChIP-Seq-Based Olig2 Target Genes in Motor Neuron Progenitor Cells Suggests the Possible Involvement of Olig2 in the Pathogenesis of Amyotrophic Lateral Sclerosis." Journal of central nervous system disease vol. 7 (2015): 1-14. doi: https://doi.org/10.4137/JCNSD.S23210
Satoh J, Asahina N, Kitano S, Kino Y. A Comprehensive Profile of ChIP-Seq-Based Olig2 Target Genes in Motor Neuron Progenitor Cells Suggests the Possible Involvement of Olig2 in the Pathogenesis of Amyotrophic Lateral Sclerosis. J Cent Nerv Syst Dis. 2015 May 18;7:1-14. doi: 10.4137/JCNSD.S23210. eCollection 2015. PMID: 26023283; PMCID: PMC4437538.
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[Studies on sleep. I. EEG in natural sleep].

Nihon seirigaku zasshi. Journal of the Physiological Society of Japan

ASAHINA K, MATSUI R.
PMID: 13684639
Nihon Seirigaku Zasshi. 1961 Feb;23:76-92.

No abstract available.

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ASAHINA K, MATSUI R. [Studies on sleep. I. EEG in natural sleep]. Nihon Seirigaku Zasshi. 1961;23:76-92
ASAHINA, K., & MATSUI, R. (1961). [Studies on sleep. I. EEG in natural sleep]. Nihon seirigaku zasshi. Journal of the Physiological Society of Japan, 2376-92.
ASAHINA, K, and MATSUI, R. "[Studies on sleep. I. EEG in natural sleep]." Nihon seirigaku zasshi. Journal of the Physiological Society of Japan vol. 23 (1961): 76-92.
ASAHINA K, MATSUI R. [Studies on sleep. I. EEG in natural sleep]. Nihon Seirigaku Zasshi. 1961 Feb;23:76-92. Japanese. PMID: 13684639.
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