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Investigating the potential role of TRPA1 in locomotion and cardiovascular control during hypertension.

Pharmacology research & perspectives

Bodkin JV, Thakore P, Aubdool AA, Liang L, Fernandes ES, Nandi M, Spina D, Clark JE, Aaronson PI, Shattock MJ, Brain SD.
PMID: 25505598
Pharmacol Res Perspect. 2014 Aug;2(4):e00052. doi: 10.1002/prp2.52. Epub 2014 Jun 23.

Radiotelemetry was used to investigate the in vivo cardiovascular and activity phenotype of both TRPA1 (transient receptor potential ankyrin 1) wild-type (WT) and TRPA1 knockout (KO) mice. After baseline recording, experimental hypertension was induced using angiotensin II infusion (1.1...

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Bodkin JV, Thakore P, Aubdool AA, et al. Investigating the potential role of TRPA1 in locomotion and cardiovascular control during hypertension. Pharmacol Res Perspect. 2014;2(4):e00052doi: 10.1002/prp2.52.
Bodkin, J. V., Thakore, P., Aubdool, A. A., Liang, L., Fernandes, E. S., Nandi, M., Spina, D., Clark, J. E., Aaronson, P. I., Shattock, M. J., & Brain, S. D. (2014). Investigating the potential role of TRPA1 in locomotion and cardiovascular control during hypertension. Pharmacology research & perspectives, 2(4), e00052. https://doi.org/10.1002/prp2.52
Bodkin, Jennifer V, et al. "Investigating the potential role of TRPA1 in locomotion and cardiovascular control during hypertension." Pharmacology research & perspectives vol. 2,4 (2014): e00052. doi: https://doi.org/10.1002/prp2.52
Bodkin JV, Thakore P, Aubdool AA, Liang L, Fernandes ES, Nandi M, Spina D, Clark JE, Aaronson PI, Shattock MJ, Brain SD. Investigating the potential role of TRPA1 in locomotion and cardiovascular control during hypertension. Pharmacol Res Perspect. 2014 Aug;2(4):e00052. doi: 10.1002/prp2.52. Epub 2014 Jun 23. PMID: 25505598; PMCID: PMC4186440.
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Occipital seizures and persistent homonymous hemianopia with T2 hypointensity on MRI in nonketotic hyperglycemia.

Epilepsy & behavior case reports

Nissa Z, Siddiqi SA, Abdool SA.
PMID: 27358769
Epilepsy Behav Case Rep. 2016 May 10;6:3-5. doi: 10.1016/j.ebcr.2016.04.008. eCollection 2016.

INTRODUCTION: Nonketotic hyperglycemia (NKH) is known to cause focal motor or secondarily generalized seizures. Occipital seizures in NKH are seldom reported, especially with visual hallucinations and persistent homonymous hemianopia (HH) with characteristic radiological and electroencephalographic (EEG) findings.SUMMARY: Our patient...

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Nissa Z, Siddiqi SA, Abdool SA. Occipital seizures and persistent homonymous hemianopia with T2 hypointensity on MRI in nonketotic hyperglycemia. Epilepsy Behav Case Rep. 2016;6:3-5doi: 10.1016/j.ebcr.2016.04.008.
Nissa, Z., Siddiqi, S. A., & Abdool, S. A. (2016). Occipital seizures and persistent homonymous hemianopia with T2 hypointensity on MRI in nonketotic hyperglycemia. Epilepsy & behavior case reports, 63-5. https://doi.org/10.1016/j.ebcr.2016.04.008
Nissa, Zebun, et al. "Occipital seizures and persistent homonymous hemianopia with T2 hypointensity on MRI in nonketotic hyperglycemia." Epilepsy & behavior case reports vol. 6 (2016): 3-5. doi: https://doi.org/10.1016/j.ebcr.2016.04.008
Nissa Z, Siddiqi SA, Abdool SA. Occipital seizures and persistent homonymous hemianopia with T2 hypointensity on MRI in nonketotic hyperglycemia. Epilepsy Behav Case Rep. 2016 May 10;6:3-5. doi: 10.1016/j.ebcr.2016.04.008. eCollection 2016. PMID: 27358769; PMCID: PMC4916048.
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Multidrug resistance proteins preferentially regulate natriuretic peptide-driven cGMP signalling in the heart and vasculature.

British journal of pharmacology

Grange RMH, Preedy MEJ, Renukanthan A, Dignam JP, Lowe VJ, Moyes AJ, Pérez-Ternero C, Aubdool AA, Baliga RS, Hobbs AJ.
PMID: 34131904
Br J Pharmacol. 2021 Jun 15; doi: 10.1111/bph.15593. Epub 2021 Jun 15.

BACKGROUND AND PURPOSE: cGMP underpins the bioactivity of NO and natriuretic peptides and is key to cardiovascular homeostasis. cGMP-driven responses are terminated primarily by PDEs, but cellular efflux via multidrug resistance proteins (MRPs) might contribute. Herein, the effect of...

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Grange RMH, Preedy MEJ, Renukanthan A, et al. Multidrug resistance proteins preferentially regulate natriuretic peptide-driven cGMP signalling in the heart and vasculature. Br J Pharmacol. 2021;doi: 10.1111/bph.15593.
Grange, R. M. H., Preedy, M. E. J., Renukanthan, A., Dignam, J. P., Lowe, V. J., Moyes, A. J., Pérez-Ternero, C., Aubdool, A. A., Baliga, R. S., & Hobbs, A. J. (2021). Multidrug resistance proteins preferentially regulate natriuretic peptide-driven cGMP signalling in the heart and vasculature. British journal of pharmacology, . https://doi.org/10.1111/bph.15593
Grange, Robert M H, et al. "Multidrug resistance proteins preferentially regulate natriuretic peptide-driven cGMP signalling in the heart and vasculature." British journal of pharmacology vol. (2021). doi: https://doi.org/10.1111/bph.15593
Grange RMH, Preedy MEJ, Renukanthan A, Dignam JP, Lowe VJ, Moyes AJ, Pérez-Ternero C, Aubdool AA, Baliga RS, Hobbs AJ. Multidrug resistance proteins preferentially regulate natriuretic peptide-driven cGMP signalling in the heart and vasculature. Br J Pharmacol. 2021 Jun 15; doi: 10.1111/bph.15593. Epub 2021 Jun 15. PMID: 34131904.
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