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DNA methylation at birth potentially mediates the association between prenatal lead (Pb) exposure and infant neurodevelopmental outcomes.

Environmental epigenetics

Rygiel CA, Dolinoy DC, Bakulski KM, Aung MT, Perng W, Jones TR, Solano-González M, Hu H, Tellez-Rojo MM, Schnaas L, Marcela E, Peterson KE, Goodrich JM.
PMID: 34141453
Environ Epigenet. 2021 Jun 16;7(1):dvab005. doi: 10.1093/eep/dvab005. eCollection 2021.

Early-life lead (Pb) exposure has been linked to adverse neurodevelopmental outcomes. Recent evidence has indicated a critical role of DNA methylation (DNAm) in cognition, and Pb exposure has also been shown to alter DNAm. However, it is unknown whether...

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Rygiel CA, Dolinoy DC, Bakulski KM, et al. DNA methylation at birth potentially mediates the association between prenatal lead (Pb) exposure and infant neurodevelopmental outcomes. Environ Epigenet. 2021;7(1):dvab005doi: 10.1093/eep/dvab005.
Rygiel, C. A., Dolinoy, D. C., Bakulski, K. M., Aung, M. T., Perng, W., Jones, T. R., Solano-González, M., Hu, H., Tellez-Rojo, M. M., Schnaas, L., Marcela, E., Peterson, K. E., & Goodrich, J. M. (2021). DNA methylation at birth potentially mediates the association between prenatal lead (Pb) exposure and infant neurodevelopmental outcomes. Environmental epigenetics, 7(1), dvab005. https://doi.org/10.1093/eep/dvab005
Rygiel, Christine A, et al. "DNA methylation at birth potentially mediates the association between prenatal lead (Pb) exposure and infant neurodevelopmental outcomes." Environmental epigenetics vol. 7,1 (2021): dvab005. doi: https://doi.org/10.1093/eep/dvab005
Rygiel CA, Dolinoy DC, Bakulski KM, Aung MT, Perng W, Jones TR, Solano-González M, Hu H, Tellez-Rojo MM, Schnaas L, Marcela E, Peterson KE, Goodrich JM. DNA methylation at birth potentially mediates the association between prenatal lead (Pb) exposure and infant neurodevelopmental outcomes. Environ Epigenet. 2021 Jun 16;7(1):dvab005. doi: 10.1093/eep/dvab005. eCollection 2021. PMID: 34141453; PMCID: PMC8206046.
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Prenatal Particulate Matter Exposure Is Associated with Saliva DNA Methylation at Age 15: Applying Cumulative DNA Methylation Scores as an Exposure Biomarker.

Toxics

Bakulski KM, Fisher JD, Dou JF, Gard A, Schneper L, Notterman DA, Ware EB, Mitchell C.
PMID: 34678958
Toxics. 2021 Oct 13;9(10). doi: 10.3390/toxics9100262.

Exposure in utero to particulate matter (PM2.5 and PM10) is associated with maladaptive health outcomes. Although exposure to prenatal PM2.5 and PM10 has cord blood DNA methylation signatures at birth, signature persistence into childhood and saliva cross-tissue applicability has...

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Bakulski KM, Fisher JD, Dou JF, et al. Prenatal Particulate Matter Exposure Is Associated with Saliva DNA Methylation at Age 15: Applying Cumulative DNA Methylation Scores as an Exposure Biomarker. Toxics. 2021;9(10)doi: 10.3390/toxics9100262.
Bakulski, K. M., Fisher, J. D., Dou, J. F., Gard, A., Schneper, L., Notterman, D. A., Ware, E. B., & Mitchell, C. (2021). Prenatal Particulate Matter Exposure Is Associated with Saliva DNA Methylation at Age 15: Applying Cumulative DNA Methylation Scores as an Exposure Biomarker. Toxics, 9(10), . https://doi.org/10.3390/toxics9100262
Bakulski, Kelly M, et al. "Prenatal Particulate Matter Exposure Is Associated with Saliva DNA Methylation at Age 15: Applying Cumulative DNA Methylation Scores as an Exposure Biomarker." Toxics vol. 9,10 (2021). doi: https://doi.org/10.3390/toxics9100262
Bakulski KM, Fisher JD, Dou JF, Gard A, Schneper L, Notterman DA, Ware EB, Mitchell C. Prenatal Particulate Matter Exposure Is Associated with Saliva DNA Methylation at Age 15: Applying Cumulative DNA Methylation Scores as an Exposure Biomarker. Toxics. 2021 Oct 13;9(10). doi: 10.3390/toxics9100262. PMID: 34678958; PMCID: PMC8538839.
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Prenatal Particulate Matter Exposure Is Associated with Saliva DNA Methylation at Age 15: Applying Cumulative DNA Methylation Scores as an Exposure Biomarker.

Toxics

Bakulski KM, Fisher JD, Dou JF, Gard A, Schneper L, Notterman DA, Ware EB, Mitchell C.
PMID: 34678958
Toxics. 2021 Oct 13;9(10). doi: 10.3390/toxics9100262.

Exposure in utero to particulate matter (PM2.5 and PM10) is associated with maladaptive health outcomes. Although exposure to prenatal PM2.5 and PM10 has cord blood DNA methylation signatures at birth, signature persistence into childhood and saliva cross-tissue applicability has...

Cite
Bakulski KM, Fisher JD, Dou JF, et al. Prenatal Particulate Matter Exposure Is Associated with Saliva DNA Methylation at Age 15: Applying Cumulative DNA Methylation Scores as an Exposure Biomarker. Toxics. 2021;9(10)doi: 10.3390/toxics9100262.
Bakulski, K. M., Fisher, J. D., Dou, J. F., Gard, A., Schneper, L., Notterman, D. A., Ware, E. B., & Mitchell, C. (2021). Prenatal Particulate Matter Exposure Is Associated with Saliva DNA Methylation at Age 15: Applying Cumulative DNA Methylation Scores as an Exposure Biomarker. Toxics, 9(10), . https://doi.org/10.3390/toxics9100262
Bakulski, Kelly M, et al. "Prenatal Particulate Matter Exposure Is Associated with Saliva DNA Methylation at Age 15: Applying Cumulative DNA Methylation Scores as an Exposure Biomarker." Toxics vol. 9,10 (2021). doi: https://doi.org/10.3390/toxics9100262
Bakulski KM, Fisher JD, Dou JF, Gard A, Schneper L, Notterman DA, Ware EB, Mitchell C. Prenatal Particulate Matter Exposure Is Associated with Saliva DNA Methylation at Age 15: Applying Cumulative DNA Methylation Scores as an Exposure Biomarker. Toxics. 2021 Oct 13;9(10). doi: 10.3390/toxics9100262. PMID: 34678958; PMCID: PMC8538839.
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High Prediagnosis Inflammation-Related Risk Score Associated with Decreased Ovarian Cancer Survival.

Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology

Brieger KK, Phung MT, Mukherjee B, Bakulski KM, Anton-Culver H, Bandera EV, Bowtell DDL, Cramer DW, DeFazio A, Doherty JA, Fereday S, Fortner RT, Gentry-Maharaj A, Goode EL, Goodman MT, Harris HR, Matsuo K, Menon U, Modugno F, Moysich KB, Qin B, Ramus SJ, Risch HA, Rossing MA, Schildkraut JM, Trabert B, Vierkant RA, Winham SJ, Wentzensen N, Wu AH, Ziogas A, Khoja L, Cho KR, McLean K, Richardson J, Grout B, Chase A, Deurloo CM, Odunsi K, Nelson BH, Brenton JD, Terry KL, Pharoah PDP, Berchuck A, Hanley GE, Webb PM, Pike MC, Pearce CL.
PMID: 34789471
Cancer Epidemiol Biomarkers Prev. 2021 Nov 17; doi: 10.1158/1055-9965.EPI-21-0977. Epub 2021 Nov 17.

BACKGROUND: There is suggestive evidence that inflammation is related to ovarian cancer survival. However, more research is needed to identify inflammation-related factors that are associated with ovarian cancer survival and to determine their combined effects.METHODS: This analysis used pooled...

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Brieger KK, Phung MT, Mukherjee B, et al. High Prediagnosis Inflammation-Related Risk Score Associated with Decreased Ovarian Cancer Survival. Cancer Epidemiol Biomarkers Prev. 2021;doi: 10.1158/1055-9965.EPI-21-0977.
Brieger, K. K., Phung, M. T., Mukherjee, B., Bakulski, K. M., Anton-Culver, H., Bandera, E. V., Bowtell, D. D. L., Cramer, D. W., DeFazio, A., Doherty, J. A., Fereday, S., Fortner, R. T., Gentry-Maharaj, A., Goode, E. L., Goodman, M. T., Harris, H. R., Matsuo, K., Menon, U., Modugno, F., Moysich, K. B., Qin, B., Ramus, S. J., Risch, H. A., Rossing, M. A., Schildkraut, J. M., Trabert, B., Vierkant, R. A., Winham, S. J., Wentzensen, N., Wu, A. H., Ziogas, A., Khoja, L., Cho, K. R., McLean, K., Richardson, J., Grout, B., Chase, A., Deurloo, C. M., Odunsi, K., Nelson, B. H., Brenton, J. D., Terry, K. L., Pharoah, P. D. P., Berchuck, A., Hanley, G. E., Webb, P. M., Pike, M. C., Pearce, C. L. (2021). High Prediagnosis Inflammation-Related Risk Score Associated with Decreased Ovarian Cancer Survival. Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology, . https://doi.org/10.1158/1055-9965.EPI-21-0977
Brieger, Katharine K, et al. "High Prediagnosis Inflammation-Related Risk Score Associated with Decreased Ovarian Cancer Survival." Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology vol. (2021). doi: https://doi.org/10.1158/1055-9965.EPI-21-0977
Brieger KK, Phung MT, Mukherjee B, Bakulski KM, Anton-Culver H, Bandera EV, Bowtell DDL, Cramer DW, DeFazio A, Doherty JA, Fereday S, Fortner RT, Gentry-Maharaj A, Goode EL, Goodman MT, Harris HR, Matsuo K, Menon U, Modugno F, Moysich KB, Qin B, Ramus SJ, Risch HA, Rossing MA, Schildkraut JM, Trabert B, Vierkant RA, Winham SJ, Wentzensen N, Wu AH, Ziogas A, Khoja L, Cho KR, McLean K, Richardson J, Grout B, Chase A, Deurloo CM, Odunsi K, Nelson BH, Brenton JD, Terry KL, Pharoah PDP, Berchuck A, Hanley GE, Webb PM, Pike MC, Pearce CL. High Prediagnosis Inflammation-Related Risk Score Associated with Decreased Ovarian Cancer Survival. Cancer Epidemiol Biomarkers Prev. 2021 Nov 17; doi: 10.1158/1055-9965.EPI-21-0977. Epub 2021 Nov 17. PMID: 34789471.
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Developmental Programming: Prenatal Testosterone Excess on Liver and Muscle Coding and Noncoding RNA in Female Sheep.

Endocrinology

Saadat N, Puttabyatappa M, Elangovan VR, Dou J, Ciarelli JN, Thompson RC, Bakulski KM, Padmanabhan V.
PMID: 34718504
Endocrinology. 2022 Jan 01;163(1). doi: 10.1210/endocr/bqab225.

Prenatal testosterone (T)-treated female sheep manifest peripheral insulin resistance, ectopic lipid accumulation, and insulin signaling disruption in liver and muscle. This study investigated transcriptional changes and transcriptome signature of prenatal T excess-induced hepatic and muscle-specific metabolic disruptions. Genome-wide coding...

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Saadat N, Puttabyatappa M, Elangovan VR, et al. Developmental Programming: Prenatal Testosterone Excess on Liver and Muscle Coding and Noncoding RNA in Female Sheep. Endocrinology. 2022;163(1)doi: 10.1210/endocr/bqab225.
Saadat, N., Puttabyatappa, M., Elangovan, V. R., Dou, J., Ciarelli, J. N., Thompson, R. C., Bakulski, K. M., & Padmanabhan, V. (2022). Developmental Programming: Prenatal Testosterone Excess on Liver and Muscle Coding and Noncoding RNA in Female Sheep. Endocrinology, 163(1), . https://doi.org/10.1210/endocr/bqab225
Saadat, Nadia, et al. "Developmental Programming: Prenatal Testosterone Excess on Liver and Muscle Coding and Noncoding RNA in Female Sheep." Endocrinology vol. 163,1 (2022). doi: https://doi.org/10.1210/endocr/bqab225
Saadat N, Puttabyatappa M, Elangovan VR, Dou J, Ciarelli JN, Thompson RC, Bakulski KM, Padmanabhan V. Developmental Programming: Prenatal Testosterone Excess on Liver and Muscle Coding and Noncoding RNA in Female Sheep. Endocrinology. 2022 Jan 01;163(1). doi: 10.1210/endocr/bqab225. PMID: 34718504; PMCID: PMC8667859.
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High pre-diagnosis inflammation-related risk score associated with decreased ovarian cancer survival.

Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology

Brieger KK, Phung MT, Mukherjee B, Bakulski KM, Anton-Culver H, Bandera EV, Bowtell DDL, Cramer DW, DeFazio A, Doherty JA, Fereday S, Fortner RT, Gentry-Maharaj A, Goode EL, Goodman MT, Harris HR, Matsuo K, Menon U, Modugno F, Moysich KB, Qin B, Ramus SJ, Risch HA, Rossing MA, Schildkraut JM, Trabert B, Vierkant RA, Winham SJ, Wentzensen N, Wu AH, Ziogas A, Khoja L, Cho KR, McLean K, Richardson J, Grout B, Chase A, McKinnon Deurloo C, Odunsi K, Nelson BH, Brenton JD, Terry KL, Pharaoh PD, Berchuck A, Hanley GE, Webb PM, Pike MC, Pearce CL.
PMID: 34789471
Cancer Epidemiol Biomarkers Prev. 2021 Nov 17; doi: 10.1158/1055-9965.EPI-21-0977. Epub 2021 Nov 17.

Background There is suggestive evidence that inflammation is related to ovarian cancer survival. However, more research is needed to identify inflammation-related factors that are associated with ovarian cancer survival and to determine their combined effects. Methods This analysis used...

Cite
Brieger KK, Phung MT, Mukherjee B, et al. High pre-diagnosis inflammation-related risk score associated with decreased ovarian cancer survival. Cancer Epidemiol Biomarkers Prev. 2021;doi: 10.1158/1055-9965.EPI-21-0977.
Brieger, K. K., Phung, M. T., Mukherjee, B., Bakulski, K. M., Anton-Culver, H., Bandera, E. V., Bowtell, D. D. L., Cramer, D. W., DeFazio, A., Doherty, J. A., Fereday, S., Fortner, R. T., Gentry-Maharaj, A., Goode, E. L., Goodman, M. T., Harris, H. R., Matsuo, K., Menon, U., Modugno, F., Moysich, K. B., Qin, B., Ramus, S. J., Risch, H. A., Rossing, M. A., Schildkraut, J. M., Trabert, B., Vierkant, R. A., Winham, S. J., Wentzensen, N., Wu, A. H., Ziogas, A., Khoja, L., Cho, K. R., McLean, K., Richardson, J., Grout, B., Chase, A., McKinnon Deurloo, C., Odunsi, K., Nelson, B. H., Brenton, J. D., Terry, K. L., Pharaoh, P. D., Berchuck, A., Hanley, G. E., Webb, P. M., Pike, M. C., & Pearce, C. L. (2021). High pre-diagnosis inflammation-related risk score associated with decreased ovarian cancer survival. Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology, . https://doi.org/10.1158/1055-9965.EPI-21-0977
Brieger, Katharine K, et al. "High pre-diagnosis inflammation-related risk score associated with decreased ovarian cancer survival." Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology vol. (2021). doi: https://doi.org/10.1158/1055-9965.EPI-21-0977
Brieger KK, Phung MT, Mukherjee B, Bakulski KM, Anton-Culver H, Bandera EV, Bowtell DDL, Cramer DW, DeFazio A, Doherty JA, Fereday S, Fortner RT, Gentry-Maharaj A, Goode EL, Goodman MT, Harris HR, Matsuo K, Menon U, Modugno F, Moysich KB, Qin B, Ramus SJ, Risch HA, Rossing MA, Schildkraut JM, Trabert B, Vierkant RA, Winham SJ, Wentzensen N, Wu AH, Ziogas A, Khoja L, Cho KR, McLean K, Richardson J, Grout B, Chase A, McKinnon Deurloo C, Odunsi K, Nelson BH, Brenton JD, Terry KL, Pharaoh PD, Berchuck A, Hanley GE, Webb PM, Pike MC, Pearce CL. High pre-diagnosis inflammation-related risk score associated with decreased ovarian cancer survival. Cancer Epidemiol Biomarkers Prev. 2021 Nov 17; doi: 10.1158/1055-9965.EPI-21-0977. Epub 2021 Nov 17. PMID: 34789471.
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Autism-Associated DNA Methylation at Birth From Multiple Tissues Is Enriched for Autism Genes in the Early Autism Risk Longitudinal Investigation.

Frontiers in molecular neuroscience

Bakulski KM, Dou JF, Feinberg JI, Aung MT, Ladd-Acosta C, Volk HE, Newschaffer CJ, Croen LA, Hertz-Picciotto I, Levy SE, Landa R, Feinberg AP, Fallin MD.
PMID: 34899183
Front Mol Neurosci. 2021 Nov 25;14:775390. doi: 10.3389/fnmol.2021.775390. eCollection 2021.

No abstract available.

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Bakulski KM, Dou JF, Feinberg JI, et al. Autism-Associated DNA Methylation at Birth From Multiple Tissues Is Enriched for Autism Genes in the Early Autism Risk Longitudinal Investigation. Front Mol Neurosci. 2021;14:775390doi: 10.3389/fnmol.2021.775390.
Bakulski, K. M., Dou, J. F., Feinberg, J. I., Aung, M. T., Ladd-Acosta, C., Volk, H. E., Newschaffer, C. J., Croen, L. A., Hertz-Picciotto, I., Levy, S. E., Landa, R., Feinberg, A. P., & Fallin, M. D. (2021). Autism-Associated DNA Methylation at Birth From Multiple Tissues Is Enriched for Autism Genes in the Early Autism Risk Longitudinal Investigation. Frontiers in molecular neuroscience, 14775390. https://doi.org/10.3389/fnmol.2021.775390
Bakulski, Kelly M, et al. "Autism-Associated DNA Methylation at Birth From Multiple Tissues Is Enriched for Autism Genes in the Early Autism Risk Longitudinal Investigation." Frontiers in molecular neuroscience vol. 14 (2021): 775390. doi: https://doi.org/10.3389/fnmol.2021.775390
Bakulski KM, Dou JF, Feinberg JI, Aung MT, Ladd-Acosta C, Volk HE, Newschaffer CJ, Croen LA, Hertz-Picciotto I, Levy SE, Landa R, Feinberg AP, Fallin MD. Autism-Associated DNA Methylation at Birth From Multiple Tissues Is Enriched for Autism Genes in the Early Autism Risk Longitudinal Investigation. Front Mol Neurosci. 2021 Nov 25;14:775390. doi: 10.3389/fnmol.2021.775390. eCollection 2021. PMID: 34899183; PMCID: PMC8655859.
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Developmental Programming: Prenatal Testosterone Excess on Liver and Muscle Coding and Noncoding RNA in Female Sheep.

Endocrinology

Saadat N, Puttabyatappa M, Elangovan VR, Dou J, Ciarelli JN, Thompson RC, Bakulski KM, Padmanabhan V.
PMID: 34718504
Endocrinology. 2022 Jan 01;163(1). doi: 10.1210/endocr/bqab225.

Prenatal testosterone (T)-treated female sheep manifest peripheral insulin resistance, ectopic lipid accumulation, and insulin signaling disruption in liver and muscle. This study investigated transcriptional changes and transcriptome signature of prenatal T excess-induced hepatic and muscle-specific metabolic disruptions. Genome-wide coding...

Cite
Saadat N, Puttabyatappa M, Elangovan VR, et al. Developmental Programming: Prenatal Testosterone Excess on Liver and Muscle Coding and Noncoding RNA in Female Sheep. Endocrinology. 2022;163(1)doi: 10.1210/endocr/bqab225.
Saadat, N., Puttabyatappa, M., Elangovan, V. R., Dou, J., Ciarelli, J. N., Thompson, R. C., Bakulski, K. M., & Padmanabhan, V. (2022). Developmental Programming: Prenatal Testosterone Excess on Liver and Muscle Coding and Noncoding RNA in Female Sheep. Endocrinology, 163(1), . https://doi.org/10.1210/endocr/bqab225
Saadat, Nadia, et al. "Developmental Programming: Prenatal Testosterone Excess on Liver and Muscle Coding and Noncoding RNA in Female Sheep." Endocrinology vol. 163,1 (2022). doi: https://doi.org/10.1210/endocr/bqab225
Saadat N, Puttabyatappa M, Elangovan VR, Dou J, Ciarelli JN, Thompson RC, Bakulski KM, Padmanabhan V. Developmental Programming: Prenatal Testosterone Excess on Liver and Muscle Coding and Noncoding RNA in Female Sheep. Endocrinology. 2022 Jan 01;163(1). doi: 10.1210/endocr/bqab225. PMID: 34718504.
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The Association of Prenatal Vitamins and Folic Acid Supplement Intake with Odds of Autism Spectrum Disorder in a High-Risk Sibling Cohort, the Early Autism Risk Longitudinal Investigation (EARLI).

Journal of autism and developmental disorders

Brieger KK, Bakulski KM, Pearce CL, Baylin A, Dou JF, Feinberg JI, Croen LA, Hertz-Picciotto I, Newschaffer CJ, Fallin MD, Schmidt RJ.
PMID: 34110557
J Autism Dev Disord. 2021 Jun 10; doi: 10.1007/s10803-021-05110-9. Epub 2021 Jun 10.

We examined maternal prenatal vitamin use or supplemental folic acid intake during month one of pregnancy for association with autism spectrum disorder (ASD) in the Early Autism Risk Longitudinal Investigation, an enriched-risk pregnancy cohort. Total folic acid intake was...

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Brieger KK, Bakulski KM, Pearce CL, et al. The Association of Prenatal Vitamins and Folic Acid Supplement Intake with Odds of Autism Spectrum Disorder in a High-Risk Sibling Cohort, the Early Autism Risk Longitudinal Investigation (EARLI). J Autism Dev Disord. 2021;doi: 10.1007/s10803-021-05110-9.
Brieger, K. K., Bakulski, K. M., Pearce, C. L., Baylin, A., Dou, J. F., Feinberg, J. I., Croen, L. A., Hertz-Picciotto, I., Newschaffer, C. J., Fallin, M. D., & Schmidt, R. J. (2021). The Association of Prenatal Vitamins and Folic Acid Supplement Intake with Odds of Autism Spectrum Disorder in a High-Risk Sibling Cohort, the Early Autism Risk Longitudinal Investigation (EARLI). Journal of autism and developmental disorders, . https://doi.org/10.1007/s10803-021-05110-9
Brieger, Katharine K, et al. "The Association of Prenatal Vitamins and Folic Acid Supplement Intake with Odds of Autism Spectrum Disorder in a High-Risk Sibling Cohort, the Early Autism Risk Longitudinal Investigation (EARLI)." Journal of autism and developmental disorders vol. (2021). doi: https://doi.org/10.1007/s10803-021-05110-9
Brieger KK, Bakulski KM, Pearce CL, Baylin A, Dou JF, Feinberg JI, Croen LA, Hertz-Picciotto I, Newschaffer CJ, Fallin MD, Schmidt RJ. The Association of Prenatal Vitamins and Folic Acid Supplement Intake with Odds of Autism Spectrum Disorder in a High-Risk Sibling Cohort, the Early Autism Risk Longitudinal Investigation (EARLI). J Autism Dev Disord. 2021 Jun 10; doi: 10.1007/s10803-021-05110-9. Epub 2021 Jun 10. PMID: 34110557.
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Mendelian Randomization of Dyslipidemia on Cognitive Impairment Among Older Americans.

Frontiers in neurology

Fu M, Bakulski KM, Higgins C, Ware EB.
PMID: 34248819
Front Neurol. 2021 Jun 23;12:660212. doi: 10.3389/fneur.2021.660212. eCollection 2021.

No abstract available.

Cite
Fu M, Bakulski KM, Higgins C, et al. Mendelian Randomization of Dyslipidemia on Cognitive Impairment Among Older Americans. Front Neurol. 2021;12:660212doi: 10.3389/fneur.2021.660212.
Fu, M., Bakulski, K. M., Higgins, C., & Ware, E. B. (2021). Mendelian Randomization of Dyslipidemia on Cognitive Impairment Among Older Americans. Frontiers in neurology, 12660212. https://doi.org/10.3389/fneur.2021.660212
Fu, Mingzhou, et al. "Mendelian Randomization of Dyslipidemia on Cognitive Impairment Among Older Americans." Frontiers in neurology vol. 12 (2021): 660212. doi: https://doi.org/10.3389/fneur.2021.660212
Fu M, Bakulski KM, Higgins C, Ware EB. Mendelian Randomization of Dyslipidemia on Cognitive Impairment Among Older Americans. Front Neurol. 2021 Jun 23;12:660212. doi: 10.3389/fneur.2021.660212. eCollection 2021. PMID: 34248819; PMCID: PMC8260932.
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Corrigendum to "Associations between maternal plasma measurements of inflammatory markers and urinary levels of phenols and parabens during pregnancy: A repeated measures study" [Sci. Total Environ. 650 (Pt 1) (2019) 1131-1140].

The Science of the total environment

Aung MT, Ferguson KK, Cantonwine DE, Bakulski KM, Mukherjee B, Loch-Caruso R, McElrath TF, Meeker JD.
PMID: 30616924
Sci Total Environ. 2019 Mar 25;658:1640. doi: 10.1016/j.scitotenv.2018.12.338. Epub 2019 Jan 05.

No abstract available.

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Aung MT, Ferguson KK, Cantonwine DE, et al. Corrigendum to "Associations between maternal plasma measurements of inflammatory markers and urinary levels of phenols and parabens during pregnancy: A repeated measures study" [Sci. Total Environ. 650 (Pt 1) (2019) 1131-1140]. Sci Total Environ. 2019;658:1640doi: 10.1016/j.scitotenv.2018.12.338.
Aung, M. T., Ferguson, K. K., Cantonwine, D. E., Bakulski, K. M., Mukherjee, B., Loch-Caruso, R., McElrath, T. F., & Meeker, J. D. (2019). Corrigendum to "Associations between maternal plasma measurements of inflammatory markers and urinary levels of phenols and parabens during pregnancy: A repeated measures study" [Sci. Total Environ. 650 (Pt 1) (2019) 1131-1140]. The Science of the total environment, 6581640. https://doi.org/10.1016/j.scitotenv.2018.12.338
Aung, M T, et al. "Corrigendum to "Associations between maternal plasma measurements of inflammatory markers and urinary levels of phenols and parabens during pregnancy: A repeated measures study" [Sci. Total Environ. 650 (Pt 1) (2019) 1131-1140]." The Science of the total environment vol. 658 (2019): 1640. doi: https://doi.org/10.1016/j.scitotenv.2018.12.338
Aung MT, Ferguson KK, Cantonwine DE, Bakulski KM, Mukherjee B, Loch-Caruso R, McElrath TF, Meeker JD. Corrigendum to "Associations between maternal plasma measurements of inflammatory markers and urinary levels of phenols and parabens during pregnancy: A repeated measures study" [Sci. Total Environ. 650 (Pt 1) (2019) 1131-1140]. Sci Total Environ. 2019 Mar 25;658:1640. doi: 10.1016/j.scitotenv.2018.12.338. Epub 2019 Jan 05. PMID: 30616924.
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Epigenetic Research in Neuropsychiatric Disorders: the "Tissue Issue".

Current behavioral neuroscience reports

Bakulski KM, Halladay A, Hu VW, Mill J, Fallin MD.
PMID: 28093577
Curr Behav Neurosci Rep. 2016 Sep;3(3):264-274. doi: 10.1007/s40473-016-0083-4. Epub 2016 Aug 02.

PURPOSE OF REVIEW: Evidence has linked neuropsychiatric disorders with epigenetic marks as either a biomarker of disease, biomarker of exposure, or mechanism of disease processes. Neuropsychiatric epidemiologic studies using either target brain tissue or surrogate blood tissue each have...

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Bakulski KM, Halladay A, Hu VW, et al. Epigenetic Research in Neuropsychiatric Disorders: the "Tissue Issue". Curr Behav Neurosci Rep. 2016;3(3):264-274doi: 10.1007/s40473-016-0083-4.
Bakulski, K. M., Halladay, A., Hu, V. W., Mill, J., & Fallin, M. D. (2016). Epigenetic Research in Neuropsychiatric Disorders: the "Tissue Issue". Current behavioral neuroscience reports, 3(3), 264-274. https://doi.org/10.1007/s40473-016-0083-4
Bakulski, Kelly M, et al. "Epigenetic Research in Neuropsychiatric Disorders: the "Tissue Issue"." Current behavioral neuroscience reports vol. 3,3 (2016): 264-274. doi: https://doi.org/10.1007/s40473-016-0083-4
Bakulski KM, Halladay A, Hu VW, Mill J, Fallin MD. Epigenetic Research in Neuropsychiatric Disorders: the "Tissue Issue". Curr Behav Neurosci Rep. 2016 Sep;3(3):264-274. doi: 10.1007/s40473-016-0083-4. Epub 2016 Aug 02. PMID: 28093577; PMCID: PMC5235359.
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