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Erratum to: Splicing factors differentially expressed in psoriasis alter mRNA maturation of disease-associated EDA+ fibronectin.

Molecular and cellular biochemistry

Szlavicz E, Szabo K, Groma G, Bata-Csorgo Z, Pagani F, Kemeny L, Szell M.
PMID: 28770468
Mol Cell Biochem. 2017 Dec;436(1):201. doi: 10.1007/s11010-017-3105-y.

No abstract available.

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Szlavicz E, Szabo K, Groma G, et al. Erratum to: Splicing factors differentially expressed in psoriasis alter mRNA maturation of disease-associated EDA+ fibronectin. Mol Cell Biochem. 2017;436(1):201doi: 10.1007/s11010-017-3105-y.
Szlavicz, E., Szabo, K., Groma, G., Bata-Csorgo, Z., Pagani, F., Kemeny, L., & Szell, M. (2017). Erratum to: Splicing factors differentially expressed in psoriasis alter mRNA maturation of disease-associated EDA+ fibronectin. Molecular and cellular biochemistry, 436(1), 201. https://doi.org/10.1007/s11010-017-3105-y
Szlavicz, E, et al. "Erratum to: Splicing factors differentially expressed in psoriasis alter mRNA maturation of disease-associated EDA+ fibronectin." Molecular and cellular biochemistry vol. 436,1 (2017): 201. doi: https://doi.org/10.1007/s11010-017-3105-y
Szlavicz E, Szabo K, Groma G, Bata-Csorgo Z, Pagani F, Kemeny L, Szell M. Erratum to: Splicing factors differentially expressed in psoriasis alter mRNA maturation of disease-associated EDA+ fibronectin. Mol Cell Biochem. 2017 Dec;436(1):201. doi: 10.1007/s11010-017-3105-y. PMID: 28770468.
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Treatment of pemphigus vulgaris and foliaceus with efgartigimod, a neonatal Fc receptor inhibitor: a phase II multicentre, open-label feasibility trial.

The British journal of dermatology

Goebeler M, Bata-Csörgő Z, De Simone C, Didona B, Remenyik E, Reznichenko N, Stoevesandt J, Ward ES, Parys W, de Haard H, Dupuy P, Verheesen P, Schmidt E, Joly P.
PMID: 34608631
Br J Dermatol. 2021 Oct 05; doi: 10.1111/bjd.20782. Epub 2021 Oct 05.

BACKGROUND: Pemphigus vulgaris and pemphigus foliaceus are potentially life-threatening autoimmune disorders triggered by IgG autoantibodies against mucosal and epidermal desmogleins. There is an unmet need for fast-acting drugs that enable patients to achieve early sustained remission with reduced corticosteroid...

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Goebeler M, Bata-Csörgő Z, De Simone C, et al. Treatment of pemphigus vulgaris and foliaceus with efgartigimod, a neonatal Fc receptor inhibitor: a phase II multicentre, open-label feasibility trial. Br J Dermatol. 2021;doi: 10.1111/bjd.20782.
Goebeler, M., Bata-Csörgő, Z., De Simone, C., Didona, B., Remenyik, E., Reznichenko, N., Stoevesandt, J., Ward, E. S., Parys, W., de Haard, H., Dupuy, P., Verheesen, P., Schmidt, E., Joly, P. (2021). Treatment of pemphigus vulgaris and foliaceus with efgartigimod, a neonatal Fc receptor inhibitor: a phase II multicentre, open-label feasibility trial. The British journal of dermatology, . https://doi.org/10.1111/bjd.20782
Goebeler, M, et al. "Treatment of pemphigus vulgaris and foliaceus with efgartigimod, a neonatal Fc receptor inhibitor: a phase II multicentre, open-label feasibility trial." The British journal of dermatology vol. (2021). doi: https://doi.org/10.1111/bjd.20782
Goebeler M, Bata-Csörgő Z, De Simone C, Didona B, Remenyik E, Reznichenko N, Stoevesandt J, Ward ES, Parys W, de Haard H, Dupuy P, Verheesen P, Schmidt E, Joly P. Treatment of pemphigus vulgaris and foliaceus with efgartigimod, a neonatal Fc receptor inhibitor: a phase II multicentre, open-label feasibility trial. Br J Dermatol. 2021 Oct 05; doi: 10.1111/bjd.20782. Epub 2021 Oct 05. PMID: 34608631.
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Exosomal long non-coding RNAs as biomarkers in human diseases.

EJIFCC

Kelemen E, Danis J, Göblös A, Bata-Csörgő Z, Széll M.
PMID: 31263395
EJIFCC. 2019 Jun 24;30(2):224-236. eCollection 2019 Jun.

The intensive study of extracellular vesicles was started about a decade ago revealing alterations of their amount and content to several cellular stimuli, highly depending on the releasing cell type. Exosomes, a type of extracellular vesicles, are released by...

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Kelemen E, Danis J, Göblös A, et al. Exosomal long non-coding RNAs as biomarkers in human diseases. EJIFCC. 2019;30(2):224-236
Kelemen, E., Danis, J., Göblös, A., Bata-Csörgő, Z., & Széll, M. (2019). Exosomal long non-coding RNAs as biomarkers in human diseases. EJIFCC, 30(2), 224-236.
Kelemen, Evelyn, et al. "Exosomal long non-coding RNAs as biomarkers in human diseases." EJIFCC vol. 30,2 (2019): 224-236.
Kelemen E, Danis J, Göblös A, Bata-Csörgő Z, Széll M. Exosomal long non-coding RNAs as biomarkers in human diseases. EJIFCC. 2019 Jun 24;30(2):224-236. eCollection 2019 Jun. PMID: 31263395; PMCID: PMC6599197.
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Amoxicillin rash in patients with infectious mononucleosis: evidence of true drug sensitization.

Allergy, asthma, and clinical immunology : official journal of the Canadian Society of Allergy and Clinical Immunology

Ónodi-Nagy K, Kinyó Á, Meszes A, Garaczi E, Kemény L, Bata-Csörgő Z.
PMID: 25784943
Allergy Asthma Clin Immunol. 2015 Jan 09;11(1):1. doi: 10.1186/1710-1492-11-1. eCollection 2015.

BACKGROUND: It hasn't been clearly understood yet whether sensitization to antibiotics, the virus itself or transient loss of drug tolerance due to the virus, is responsible for the development of maculopapular exanthems following amoxicillin intake in patients with infectious...

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Ónodi-Nagy K, Kinyó Á, Meszes A, et al. Amoxicillin rash in patients with infectious mononucleosis: evidence of true drug sensitization. Allergy Asthma Clin Immunol. 2015;11(1):1doi: 10.1186/1710-1492-11-1.
Ónodi-Nagy, K., Kinyó, �. �., Meszes, A., Garaczi, E., Kemény, L., & Bata-Csörgő, Z. (2015). Amoxicillin rash in patients with infectious mononucleosis: evidence of true drug sensitization. Allergy, asthma, and clinical immunology : official journal of the Canadian Society of Allergy and Clinical Immunology, 11(1), 1. https://doi.org/10.1186/1710-1492-11-1
Ónodi-Nagy, Katinka, et al. "Amoxicillin rash in patients with infectious mononucleosis: evidence of true drug sensitization." Allergy, asthma, and clinical immunology : official journal of the Canadian Society of Allergy and Clinical Immunology vol. 11,1 (2015): 1. doi: https://doi.org/10.1186/1710-1492-11-1
Ónodi-Nagy K, Kinyó Á, Meszes A, Garaczi E, Kemény L, Bata-Csörgő Z. Amoxicillin rash in patients with infectious mononucleosis: evidence of true drug sensitization. Allergy Asthma Clin Immunol. 2015 Jan 09;11(1):1. doi: 10.1186/1710-1492-11-1. eCollection 2015. PMID: 25784943; PMCID: PMC4362637.
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Phenotypic plasticity of melanocytes derived from human adult skin.

Pigment cell & melanoma research

Vidács DL, Veréb Z, Bozó R, Flink LB, Polyánka H, Németh IB, Póliska S, Papp BT, Manczinger M, Gáspár R, Mirdamadi S, Kemény L, Bata-Csörgő Z.
PMID: 34467641
Pigment Cell Melanoma Res. 2022 Jan;35(1):38-51. doi: 10.1111/pcmr.13012. Epub 2021 Aug 31.

We previously described a novel in vitro culture technique for dedifferentiated human adult skin melanocytes. Melanocytes cultured in a defined, cholera toxin and PMA free medium became bipolar, unpigmented, and highly proliferative. Furthermore, TRP-1 and c-Kit expression disappeared and...

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Vidács DL, Veréb Z, Bozó R, et al. Phenotypic plasticity of melanocytes derived from human adult skin. Pigment Cell Melanoma Res. 2021;35(1):38-51doi: 10.1111/pcmr.13012.
Vidács, D. L., Veréb, Z., Bozó, R., Flink, L. B., Polyánka, H., Németh, I. B., Póliska, S., Papp, B. T., Manczinger, M., Gáspár, R., Mirdamadi, S., Kemény, L., & Bata-Csörgő, Z. (2022). Phenotypic plasticity of melanocytes derived from human adult skin. Pigment cell & melanoma research, 35(1), 38-51. https://doi.org/10.1111/pcmr.13012
Vidács, Dániel László, et al. "Phenotypic plasticity of melanocytes derived from human adult skin." Pigment cell & melanoma research vol. 35,1 (2022): 38-51. doi: https://doi.org/10.1111/pcmr.13012
Vidács DL, Veréb Z, Bozó R, Flink LB, Polyánka H, Németh IB, Póliska S, Papp BT, Manczinger M, Gáspár R, Mirdamadi S, Kemény L, Bata-Csörgő Z. Phenotypic plasticity of melanocytes derived from human adult skin. Pigment Cell Melanoma Res. 2022 Jan;35(1):38-51. doi: 10.1111/pcmr.13012. Epub 2021 Aug 31. PMID: 34467641.
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Stress-Related Regulation Is Abnormal in the Psoriatic Uninvolved Skin.

Life (Basel, Switzerland)

Bozó R, Danis J, Flink LB, Vidács DL, Kemény L, Bata-Csörgő Z.
PMID: 34201431
Life (Basel). 2021 Jun 23;11(7). doi: 10.3390/life11070599.

Keratinocyte stress-response of the uninvolved psoriatic epidermis is known to be altered compared to healthy cells. Therefore, we aimed to reveal potential mechanisms underlying this alteration. We compared the expression of annotated cell-stress-related proteins between uninvolved psoriatic and healthy...

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Bozó R, Danis J, Flink LB, et al. Stress-Related Regulation Is Abnormal in the Psoriatic Uninvolved Skin. Life (Basel). 2021;11(7)doi: 10.3390/life11070599.
Bozó, R., Danis, J., Flink, L. B., Vidács, D. L., Kemény, L., & Bata-Csörgő, Z. (2021). Stress-Related Regulation Is Abnormal in the Psoriatic Uninvolved Skin. Life (Basel, Switzerland), 11(7), . https://doi.org/10.3390/life11070599
Bozó, Renáta, et al. "Stress-Related Regulation Is Abnormal in the Psoriatic Uninvolved Skin." Life (Basel, Switzerland) vol. 11,7 (2021). doi: https://doi.org/10.3390/life11070599
Bozó R, Danis J, Flink LB, Vidács DL, Kemény L, Bata-Csörgő Z. Stress-Related Regulation Is Abnormal in the Psoriatic Uninvolved Skin. Life (Basel). 2021 Jun 23;11(7). doi: 10.3390/life11070599. PMID: 34201431; PMCID: PMC8303303.
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The Psoriatic Nonlesional Skin: A Battlefield between Susceptibility and Protective Factors.

The Journal of investigative dermatology

Kelemen E, Bozó R, Groma G, Bata-Csörgő Z, Kemény L, Danis J, Széll M.
PMID: 34216605
J Invest Dermatol. 2021 Dec;141(12):2785-2790. doi: 10.1016/j.jid.2021.05.020. Epub 2021 Jul 01.

In the last two decades, large-scale gene-expression studies on psoriatic skin samples revealed that even though nonlesional skin is macroscopically identical to healthy skin, it harbors several molecular differences. Originally, these molecular differences were thought to represent susceptibility factors...

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Kelemen E, Bozó R, Groma G, et al. The Psoriatic Nonlesional Skin: A Battlefield between Susceptibility and Protective Factors. J Invest Dermatol. 2021;141(12):2785-2790doi: 10.1016/j.jid.2021.05.020.
Kelemen, E., Bozó, R., Groma, G., Bata-Csörgő, Z., Kemény, L., Danis, J., & Széll, M. (2021). The Psoriatic Nonlesional Skin: A Battlefield between Susceptibility and Protective Factors. The Journal of investigative dermatology, 141(12), 2785-2790. https://doi.org/10.1016/j.jid.2021.05.020
Kelemen, Evelyn, et al. "The Psoriatic Nonlesional Skin: A Battlefield between Susceptibility and Protective Factors." The Journal of investigative dermatology vol. 141,12 (2021): 2785-2790. doi: https://doi.org/10.1016/j.jid.2021.05.020
Kelemen E, Bozó R, Groma G, Bata-Csörgő Z, Kemény L, Danis J, Széll M. The Psoriatic Nonlesional Skin: A Battlefield between Susceptibility and Protective Factors. J Invest Dermatol. 2021 Dec;141(12):2785-2790. doi: 10.1016/j.jid.2021.05.020. Epub 2021 Jul 01. PMID: 34216605.
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Could basement membrane alterations, resembling micro-wounds at the dermo-epidermal junction in psoriatic non-lesional skin, make the skin susceptible to lesion formation?.

Experimental dermatology

Bozó R, Flink LB, Belső N, Gubán B, Széll M, Kemény L, Bata-Csörgő Z.
PMID: 33348435
Exp Dermatol. 2021 Jun;30(6):765-772. doi: 10.1111/exd.14267. Epub 2021 Jan 16.

Current data suggest that tissue microenvironment control immune functions. Therefore, understanding the tissue environment in which immune activation occurs will enhance our capability to interfere with abnormal immune pathology. Here, we argue that studying the constitutively abnormal functions of...

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Bozó R, Flink LB, Belső N, et al. Could basement membrane alterations, resembling micro-wounds at the dermo-epidermal junction in psoriatic non-lesional skin, make the skin susceptible to lesion formation?. Exp Dermatol. 2021;30(6):765-772doi: 10.1111/exd.14267.
Bozó, R., Flink, L. B., Belső, N., Gubán, B., Széll, M., Kemény, L., & Bata-Csörgő, Z. (2021). Could basement membrane alterations, resembling micro-wounds at the dermo-epidermal junction in psoriatic non-lesional skin, make the skin susceptible to lesion formation?. Experimental dermatology, 30(6), 765-772. https://doi.org/10.1111/exd.14267
Bozó, Renáta, et al. "Could basement membrane alterations, resembling micro-wounds at the dermo-epidermal junction in psoriatic non-lesional skin, make the skin susceptible to lesion formation?." Experimental dermatology vol. 30,6 (2021): 765-772. doi: https://doi.org/10.1111/exd.14267
Bozó R, Flink LB, Belső N, Gubán B, Széll M, Kemény L, Bata-Csörgő Z. Could basement membrane alterations, resembling micro-wounds at the dermo-epidermal junction in psoriatic non-lesional skin, make the skin susceptible to lesion formation?. Exp Dermatol. 2021 Jun;30(6):765-772. doi: 10.1111/exd.14267. Epub 2021 Jan 16. PMID: 33348435.
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Psoriasis-Associated Inflammatory Conditions Induce IL-23 mRNA Expression in Normal Human Epidermal Keratinocytes.

International journal of molecular sciences

Kelemen E, Ádám É, Sági SM, Göblös A, Kemény L, Bata-Csörgő Z, Széll M, Danis J.
PMID: 35008970
Int J Mol Sci. 2022 Jan 04;23(1). doi: 10.3390/ijms23010540.

Psoriasis is a multifactorial, chronic inflammatory skin disease, the development of which is affected by both genetic and environmental factors. Cytosolic nucleic acid fragments, recognized as pathogen- and danger-associated molecular patterns, are highly abundant in psoriatic skin. It is...

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Kelemen E, Ádám É, Sági SM, et al. Psoriasis-Associated Inflammatory Conditions Induce IL-23 mRNA Expression in Normal Human Epidermal Keratinocytes. Int J Mol Sci. 2022;23(1)doi: 10.3390/ijms23010540.
Kelemen, E., Ádám, �. �., Sági, S. M., Göblös, A., Kemény, L., Bata-Csörgő, Z., Széll, M., & Danis, J. (2022). Psoriasis-Associated Inflammatory Conditions Induce IL-23 mRNA Expression in Normal Human Epidermal Keratinocytes. International journal of molecular sciences, 23(1), . https://doi.org/10.3390/ijms23010540
Kelemen, Evelyn, et al. "Psoriasis-Associated Inflammatory Conditions Induce IL-23 mRNA Expression in Normal Human Epidermal Keratinocytes." International journal of molecular sciences vol. 23,1 (2022). doi: https://doi.org/10.3390/ijms23010540
Kelemen E, Ádám É, Sági SM, Göblös A, Kemény L, Bata-Csörgő Z, Széll M, Danis J. Psoriasis-Associated Inflammatory Conditions Induce IL-23 mRNA Expression in Normal Human Epidermal Keratinocytes. Int J Mol Sci. 2022 Jan 04;23(1). doi: 10.3390/ijms23010540. PMID: 35008970; PMCID: PMC8745281.
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PRINS Non-Coding RNA Regulates Nucleic Acid-Induced Innate Immune Responses of Human Keratinocytes.

Frontiers in immunology

Danis J, Göblös A, Bata-Csörgő Z, Kemény L, Széll M.
PMID: 28900430
Front Immunol. 2017 Aug 29;8:1053. doi: 10.3389/fimmu.2017.01053. eCollection 2017.

Cytosolic DNA fragments are recognized as pathogen- and danger-associated molecular patterns that induce a cascade of innate immune responses. Moreover, excessive cytosolic DNA can enhance chronic inflammation, predominantly by activating inflammasomes, and thereby contributing to the pathogenesis of chronic...

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Danis J, Göblös A, Bata-Csörgő Z, et al. PRINS Non-Coding RNA Regulates Nucleic Acid-Induced Innate Immune Responses of Human Keratinocytes. Front Immunol. 2017;8:1053doi: 10.3389/fimmu.2017.01053.
Danis, J., Göblös, A., Bata-Csörgő, Z., Kemény, L., & Széll, M. (2017). PRINS Non-Coding RNA Regulates Nucleic Acid-Induced Innate Immune Responses of Human Keratinocytes. Frontiers in immunology, 81053. https://doi.org/10.3389/fimmu.2017.01053
Danis, Judit, et al. "PRINS Non-Coding RNA Regulates Nucleic Acid-Induced Innate Immune Responses of Human Keratinocytes." Frontiers in immunology vol. 8 (2017): 1053. doi: https://doi.org/10.3389/fimmu.2017.01053
Danis J, Göblös A, Bata-Csörgő Z, Kemény L, Széll M. PRINS Non-Coding RNA Regulates Nucleic Acid-Induced Innate Immune Responses of Human Keratinocytes. Front Immunol. 2017 Aug 29;8:1053. doi: 10.3389/fimmu.2017.01053. eCollection 2017. PMID: 28900430; PMCID: PMC5581800.
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Nuclear Factor κB Activation in a Type V Pityriasis Rubra Pilaris Patient Harboring Multiple .

Frontiers in immunology

Danis J, Göblös A, Gál B, Sulák A, Farkas K, Török D, Varga E, Korom I, Kemény L, Széll M, Bata-Csörgö Z, Nagy N.
PMID: 30018619
Front Immunol. 2018 Jul 03;9:1564. doi: 10.3389/fimmu.2018.01564. eCollection 2018.

Pityriasis rubra pilaris (PRP) is a rare papulosquamous skin disorder, which is phenotypically related to psoriasis. Some familial PRP cases show autosomal dominant inheritance due to

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Danis J, Göblös A, Gál B, et al. Nuclear Factor κB Activation in a Type V Pityriasis Rubra Pilaris Patient Harboring Multiple . Front Immunol. 2018;9:1564doi: 10.3389/fimmu.2018.01564.
Danis, J., Göblös, A., Gál, B., Sulák, A., Farkas, K., Török, D., Varga, E., Korom, I., Kemény, L., Széll, M., Bata-Csörgö, Z., & Nagy, N. (2018). Nuclear Factor κB Activation in a Type V Pityriasis Rubra Pilaris Patient Harboring Multiple . Frontiers in immunology, 91564. https://doi.org/10.3389/fimmu.2018.01564
Danis, Judit, et al. "Nuclear Factor κB Activation in a Type V Pityriasis Rubra Pilaris Patient Harboring Multiple ." Frontiers in immunology vol. 9 (2018): 1564. doi: https://doi.org/10.3389/fimmu.2018.01564
Danis J, Göblös A, Gál B, Sulák A, Farkas K, Török D, Varga E, Korom I, Kemény L, Széll M, Bata-Csörgö Z, Nagy N. Nuclear Factor κB Activation in a Type V Pityriasis Rubra Pilaris Patient Harboring Multiple . Front Immunol. 2018 Jul 03;9:1564. doi: 10.3389/fimmu.2018.01564. eCollection 2018. PMID: 30018619; PMCID: PMC6037727.
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Could basement membrane alterations, resembling micro-wounds at the dermo-epidermal junction in psoriatic non-lesional skin, make the skin susceptible to lesion formation?.

Experimental dermatology

Bozó R, Flink LB, Belső N, Gubán B, Széll M, Kemény L, Bata-Csörgő Z.
PMID: 33348435
Exp Dermatol. 2021 Jun;30(6):765-772. doi: 10.1111/exd.14267. Epub 2021 Jan 16.

Current data suggest that tissue microenvironment control immune functions. Therefore, understanding the tissue environment in which immune activation occurs will enhance our capability to interfere with abnormal immune pathology. Here, we argue that studying the constitutively abnormal functions of...

Cite
Bozó R, Flink LB, Belső N, et al. Could basement membrane alterations, resembling micro-wounds at the dermo-epidermal junction in psoriatic non-lesional skin, make the skin susceptible to lesion formation?. Exp Dermatol. 2021;30(6):765-772doi: 10.1111/exd.14267.
Bozó, R., Flink, L. B., Belső, N., Gubán, B., Széll, M., Kemény, L., & Bata-Csörgő, Z. (2021). Could basement membrane alterations, resembling micro-wounds at the dermo-epidermal junction in psoriatic non-lesional skin, make the skin susceptible to lesion formation?. Experimental dermatology, 30(6), 765-772. https://doi.org/10.1111/exd.14267
Bozó, Renáta, et al. "Could basement membrane alterations, resembling micro-wounds at the dermo-epidermal junction in psoriatic non-lesional skin, make the skin susceptible to lesion formation?." Experimental dermatology vol. 30,6 (2021): 765-772. doi: https://doi.org/10.1111/exd.14267
Bozó R, Flink LB, Belső N, Gubán B, Széll M, Kemény L, Bata-Csörgő Z. Could basement membrane alterations, resembling micro-wounds at the dermo-epidermal junction in psoriatic non-lesional skin, make the skin susceptible to lesion formation?. Exp Dermatol. 2021 Jun;30(6):765-772. doi: 10.1111/exd.14267. Epub 2021 Jan 16. PMID: 33348435.
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