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Differential Cytotoxicity of Bacillus anthracis and Bacillus cereus Culture Filtrates.

Journal of bacteriology

Bonventre PF.
PMID: 16562033
J Bacteriol. 1965 Jul;90(1):284-5. doi: 10.1128/jb.90.1.284-285.1965.

No abstract available.

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Bonventre PF. Differential Cytotoxicity of Bacillus anthracis and Bacillus cereus Culture Filtrates. J Bacteriol. 1965;90(1):284-5doi: 10.1128/jb.90.1.284-285.1965.
Bonventre, P. F. (1965). Differential Cytotoxicity of Bacillus anthracis and Bacillus cereus Culture Filtrates. Journal of bacteriology, 90(1), 284-5. https://doi.org/10.1128/jb.90.1.284-285.1965
Bonventre, P F. "Differential Cytotoxicity of Bacillus anthracis and Bacillus cereus Culture Filtrates." Journal of bacteriology vol. 90,1 (1965): 284-5. doi: https://doi.org/10.1128/jb.90.1.284-285.1965
Bonventre PF. Differential Cytotoxicity of Bacillus anthracis and Bacillus cereus Culture Filtrates. J Bacteriol. 1965 Jul;90(1):284-5. doi: 10.1128/jb.90.1.284-285.1965. PMID: 16562033; PMCID: PMC315628.
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Differential Roles of Cysteinyl Cathepsins in TGF-β Signaling and Tissue Fibrosis.

iScience

Zhang X, Zhou Y, Yu X, Huang Q, Fang W, Li J, Bonventre JV, Sukhova GK, Libby P, Shi GP.
PMID: 31446224
iScience. 2019 Sep 27;19:607-622. doi: 10.1016/j.isci.2019.08.014. Epub 2019 Aug 09.

Transforming growth factor beta (TGF-β) signaling contributes to tissue fibrosis. Here we demonstrate that TGF-β enhances CatS and CatK expression but reduces CatB and CatL expression in mouse kidney tubular epithelial cells (TECs). CatS- and CatK deficiency reduces TEC...

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Zhang X, Zhou Y, Yu X, et al. Differential Roles of Cysteinyl Cathepsins in TGF-β Signaling and Tissue Fibrosis. iScience. 2019;19:607-622doi: 10.1016/j.isci.2019.08.014.
Zhang, X., Zhou, Y., Yu, X., Huang, Q., Fang, W., Li, J., Bonventre, J. V., Sukhova, G. K., Libby, P., & Shi, G. P. (2019). Differential Roles of Cysteinyl Cathepsins in TGF-β Signaling and Tissue Fibrosis. iScience, 19607-622. https://doi.org/10.1016/j.isci.2019.08.014
Zhang, Xian, et al. "Differential Roles of Cysteinyl Cathepsins in TGF-β Signaling and Tissue Fibrosis." iScience vol. 19 (2019): 607-622. doi: https://doi.org/10.1016/j.isci.2019.08.014
Zhang X, Zhou Y, Yu X, Huang Q, Fang W, Li J, Bonventre JV, Sukhova GK, Libby P, Shi GP. Differential Roles of Cysteinyl Cathepsins in TGF-β Signaling and Tissue Fibrosis. iScience. 2019 Sep 27;19:607-622. doi: 10.1016/j.isci.2019.08.014. Epub 2019 Aug 09. PMID: 31446224; PMCID: PMC6715892.
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Variability in CKD Biomarker Studies: Soluble Urokinase Plasminogen Activator Receptor (suPAR) and Kidney Disease Progression in the Chronic Kidney Disease in Children (CKiD) Study.

Kidney medicine

Abraham AG, Xu Y, Roem JL, Greenberg JH, Weidemann DK, Sabbisetti VS, Bonventre JV, Denburg M, Warady BA, Furth SL.
PMID: 34693253
Kidney Med. 2021 Jun 17;3(5):712-721.e1. doi: 10.1016/j.xkme.2021.04.007. eCollection 2021.

RATIONALE & OBJECTIVE: Biomarker studies are important for generating mechanistic insight and providing clinically useful predictors of chronic kidney disease (CKD) progression. However, variability across studies can often muddy the evidence waters. Here we evaluated real-world variability in biomarker...

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Abraham AG, Xu Y, Roem JL, et al. Variability in CKD Biomarker Studies: Soluble Urokinase Plasminogen Activator Receptor (suPAR) and Kidney Disease Progression in the Chronic Kidney Disease in Children (CKiD) Study. Kidney Med. 2021;3(5):712-721.e1doi: 10.1016/j.xkme.2021.04.007.
Abraham, A. G., Xu, Y., Roem, J. L., Greenberg, J. H., Weidemann, D. K., Sabbisetti, V. S., Bonventre, J. V., Denburg, M., Warady, B. A., & Furth, S. L. (2021). Variability in CKD Biomarker Studies: Soluble Urokinase Plasminogen Activator Receptor (suPAR) and Kidney Disease Progression in the Chronic Kidney Disease in Children (CKiD) Study. Kidney medicine, 3(5), 712-721.e1. https://doi.org/10.1016/j.xkme.2021.04.007
Abraham, Alison G, et al. "Variability in CKD Biomarker Studies: Soluble Urokinase Plasminogen Activator Receptor (suPAR) and Kidney Disease Progression in the Chronic Kidney Disease in Children (CKiD) Study." Kidney medicine vol. 3,5 (2021): 712-721.e1. doi: https://doi.org/10.1016/j.xkme.2021.04.007
Abraham AG, Xu Y, Roem JL, Greenberg JH, Weidemann DK, Sabbisetti VS, Bonventre JV, Denburg M, Warady BA, Furth SL. Variability in CKD Biomarker Studies: Soluble Urokinase Plasminogen Activator Receptor (suPAR) and Kidney Disease Progression in the Chronic Kidney Disease in Children (CKiD) Study. Kidney Med. 2021 Jun 17;3(5):712-721.e1. doi: 10.1016/j.xkme.2021.04.007. eCollection 2021. PMID: 34693253; PMCID: PMC8515077.
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From Bench to the Clinic: The Path to Translation of Nanotechnology-Enabled mRNA SARS-CoV-2 Vaccines.

Nano-micro letters

Lopez-Cantu DO, Wang X, Carrasco-Magallanes H, Afewerki S, Zhang X, Bonventre JV, Ruiz-Esparza GU.
PMID: 34981278
Nanomicro Lett. 2022 Jan 03;14(1):41. doi: 10.1007/s40820-021-00771-8.

During the last decades, the use of nanotechnology in medicine has effectively been translated to the design of drug delivery systems, nanostructured tissues, diagnostic platforms, and novel nanomaterials against several human diseases and infectious pathogens. Nanotechnology-enabled vaccines have been...

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Lopez-Cantu DO, Wang X, Carrasco-Magallanes H, et al. From Bench to the Clinic: The Path to Translation of Nanotechnology-Enabled mRNA SARS-CoV-2 Vaccines. Nanomicro Lett. 2022;14(1):41doi: 10.1007/s40820-021-00771-8.
Lopez-Cantu, D. O., Wang, X., Carrasco-Magallanes, H., Afewerki, S., Zhang, X., Bonventre, J. V., & Ruiz-Esparza, G. U. (2022). From Bench to the Clinic: The Path to Translation of Nanotechnology-Enabled mRNA SARS-CoV-2 Vaccines. Nano-micro letters, 14(1), 41. https://doi.org/10.1007/s40820-021-00771-8
Lopez-Cantu, Diana O, et al. "From Bench to the Clinic: The Path to Translation of Nanotechnology-Enabled mRNA SARS-CoV-2 Vaccines." Nano-micro letters vol. 14,1 (2022): 41. doi: https://doi.org/10.1007/s40820-021-00771-8
Lopez-Cantu DO, Wang X, Carrasco-Magallanes H, Afewerki S, Zhang X, Bonventre JV, Ruiz-Esparza GU. From Bench to the Clinic: The Path to Translation of Nanotechnology-Enabled mRNA SARS-CoV-2 Vaccines. Nanomicro Lett. 2022 Jan 03;14(1):41. doi: 10.1007/s40820-021-00771-8. PMID: 34981278; PMCID: PMC8722410.
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Plasma Kidney Injury Molecule 1 in CKD: Findings From the Boston Kidney Biopsy Cohort and CRIC Studies.

American journal of kidney diseases : the official journal of the National Kidney Foundation

Schmidt IM, Srivastava A, Sabbisetti V, McMahon GM, He J, Chen J, Kusek JW, Taliercio J, Ricardo AC, Hsu CY, Kimmel PL, Liu KD, Mifflin TE, Nelson RG, Vasan RS, Xie D, Zhang X, Palsson R, Stillman IE, Rennke HG, Feldman HI, Bonventre JV, Waikar SS.
PMID: 34175376
Am J Kidney Dis. 2021 Jun 25; doi: 10.1053/j.ajkd.2021.05.013. Epub 2021 Jun 25.

RATIONALE & OBJECTIVE: Plasma kidney injury molecule 1 (KIM-1) is a sensitive marker of proximal tubule injury, but its association with risks of adverse clinical outcomes across a spectrum of kidney diseases is unknown.STUDY DESIGN: Prospective, observational cohort study.SETTING...

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Schmidt IM, Srivastava A, Sabbisetti V, et al. Plasma Kidney Injury Molecule 1 in CKD: Findings From the Boston Kidney Biopsy Cohort and CRIC Studies. Am J Kidney Dis. 2021;doi: 10.1053/j.ajkd.2021.05.013.
Schmidt, I. M., Srivastava, A., Sabbisetti, V., McMahon, G. M., He, J., Chen, J., Kusek, J. W., Taliercio, J., Ricardo, A. C., Hsu, C. Y., Kimmel, P. L., Liu, K. D., Mifflin, T. E., Nelson, R. G., Vasan, R. S., Xie, D., Zhang, X., Palsson, R., Stillman, I. E., Rennke, H. G., Feldman, H. I., Bonventre, J. V., Waikar, S. S. (2021). Plasma Kidney Injury Molecule 1 in CKD: Findings From the Boston Kidney Biopsy Cohort and CRIC Studies. American journal of kidney diseases : the official journal of the National Kidney Foundation, . https://doi.org/10.1053/j.ajkd.2021.05.013
Schmidt, Insa M, et al. "Plasma Kidney Injury Molecule 1 in CKD: Findings From the Boston Kidney Biopsy Cohort and CRIC Studies." American journal of kidney diseases : the official journal of the National Kidney Foundation vol. (2021). doi: https://doi.org/10.1053/j.ajkd.2021.05.013
Schmidt IM, Srivastava A, Sabbisetti V, McMahon GM, He J, Chen J, Kusek JW, Taliercio J, Ricardo AC, Hsu CY, Kimmel PL, Liu KD, Mifflin TE, Nelson RG, Vasan RS, Xie D, Zhang X, Palsson R, Stillman IE, Rennke HG, Feldman HI, Bonventre JV, Waikar SS. Plasma Kidney Injury Molecule 1 in CKD: Findings From the Boston Kidney Biopsy Cohort and CRIC Studies. Am J Kidney Dis. 2021 Jun 25; doi: 10.1053/j.ajkd.2021.05.013. Epub 2021 Jun 25. PMID: 34175376; PMCID: PMC8709877.
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Therapeutic silencing of SMOC2 prevents kidney function loss in mouse model of chronic kidney disease.

iScience

Xin C, Lei J, Wang Q, Yin Y, Yang X, Moran Guerrero JA, Sabbisetti V, Sun X, Vaidya VS, Bonventre JV.
PMID: 34703992
iScience. 2021 Sep 29;24(10):103193. doi: 10.1016/j.isci.2021.103193. eCollection 2021 Oct 22.

Chronic kidney disease (CKD) is associated with substantial morbidity and mortality. We developed a mouse model that mimics human CKD with inflammation, extracellular matrix deposition, tubulointerstitial fibrosis, increased proteinuria, and associated reduction in glomerular filtration rate over time. Using...

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Xin C, Lei J, Wang Q, et al. Therapeutic silencing of SMOC2 prevents kidney function loss in mouse model of chronic kidney disease. iScience. 2021;24(10):103193doi: 10.1016/j.isci.2021.103193.
Xin, C., Lei, J., Wang, Q., Yin, Y., Yang, X., Moran Guerrero, J. A., Sabbisetti, V., Sun, X., Vaidya, V. S., & Bonventre, J. V. (2021). Therapeutic silencing of SMOC2 prevents kidney function loss in mouse model of chronic kidney disease. iScience, 24(10), 103193. https://doi.org/10.1016/j.isci.2021.103193
Xin, Cuiyan, et al. "Therapeutic silencing of SMOC2 prevents kidney function loss in mouse model of chronic kidney disease." iScience vol. 24,10 (2021): 103193. doi: https://doi.org/10.1016/j.isci.2021.103193
Xin C, Lei J, Wang Q, Yin Y, Yang X, Moran Guerrero JA, Sabbisetti V, Sun X, Vaidya VS, Bonventre JV. Therapeutic silencing of SMOC2 prevents kidney function loss in mouse model of chronic kidney disease. iScience. 2021 Sep 29;24(10):103193. doi: 10.1016/j.isci.2021.103193. eCollection 2021 Oct 22. PMID: 34703992; PMCID: PMC8524153.
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6β-Hydroxytestosterone Promotes Angiotensin II-Induced Hypertension via Enhanced Cytosolic Phospholipase A.

Hypertension (Dallas, Tex. : 1979)

Singh P, Song CY, Dutta SR, Pingili A, Shin JS, Gonzalez FJ, Bonventre JV, Malik KU.
PMID: 34420370
Hypertension. 2021 Sep;78(4):1053-1066. doi: 10.1161/HYPERTENSIONAHA.121.17927. Epub 2021 Aug 23.

[Figure: see text].

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Singh P, Song CY, Dutta SR, et al. 6β-Hydroxytestosterone Promotes Angiotensin II-Induced Hypertension via Enhanced Cytosolic Phospholipase A. Hypertension. 2021;78(4):1053-1066doi: 10.1161/HYPERTENSIONAHA.121.17927.
Singh, P., Song, C. Y., Dutta, S. R., Pingili, A., Shin, J. S., Gonzalez, F. J., Bonventre, J. V., & Malik, K. U. (2021). 6β-Hydroxytestosterone Promotes Angiotensin II-Induced Hypertension via Enhanced Cytosolic Phospholipase A. Hypertension (Dallas, Tex. : 1979), 78(4), 1053-1066. https://doi.org/10.1161/HYPERTENSIONAHA.121.17927
Singh, Purnima, et al. "6β-Hydroxytestosterone Promotes Angiotensin II-Induced Hypertension via Enhanced Cytosolic Phospholipase A." Hypertension (Dallas, Tex. : 1979) vol. 78,4 (2021): 1053-1066. doi: https://doi.org/10.1161/HYPERTENSIONAHA.121.17927
Singh P, Song CY, Dutta SR, Pingili A, Shin JS, Gonzalez FJ, Bonventre JV, Malik KU. 6β-Hydroxytestosterone Promotes Angiotensin II-Induced Hypertension via Enhanced Cytosolic Phospholipase A. Hypertension. 2021 Sep;78(4):1053-1066. doi: 10.1161/HYPERTENSIONAHA.121.17927. Epub 2021 Aug 23. PMID: 34420370; PMCID: PMC8415516.
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KIM-1/TIM-1 is a Receptor for SARS-CoV-2 in Lung and Kidney.

medRxiv : the preprint server for health sciences

Ichimura T, Mori Y, Aschauer P, Padmanabha Das KM, Padera RF, Weins A, Nasr ML, Bonventre JV.
PMID: 32995803
medRxiv. 2020 Sep 18; doi: 10.1101/2020.09.16.20190694.

SARS-CoV-2 precipitates respiratory distress by infection of airway epithelial cells and is often accompanied by acute kidney injury. We report that Kidney Injury Molecule-1/T cell immunoglobulin mucin domain 1 (KIM-1/TIM-1) is expressed in lung and kidney epithelial cells in...

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Ichimura T, Mori Y, Aschauer P, et al. KIM-1/TIM-1 is a Receptor for SARS-CoV-2 in Lung and Kidney. medRxiv. 2020;doi: 10.1101/2020.09.16.20190694.
Ichimura, T., Mori, Y., Aschauer, P., Padmanabha Das, K. M., Padera, R. F., Weins, A., Nasr, M. L., & Bonventre, J. V. (2020). KIM-1/TIM-1 is a Receptor for SARS-CoV-2 in Lung and Kidney. medRxiv : the preprint server for health sciences, . https://doi.org/10.1101/2020.09.16.20190694
Ichimura, Takaharu, et al. "KIM-1/TIM-1 is a Receptor for SARS-CoV-2 in Lung and Kidney." medRxiv : the preprint server for health sciences vol. (2020). doi: https://doi.org/10.1101/2020.09.16.20190694
Ichimura T, Mori Y, Aschauer P, Padmanabha Das KM, Padera RF, Weins A, Nasr ML, Bonventre JV. KIM-1/TIM-1 is a Receptor for SARS-CoV-2 in Lung and Kidney. medRxiv. 2020 Sep 18; doi: 10.1101/2020.09.16.20190694. PMID: 32995803; PMCID: PMC7523142.
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Ataxia Telangiectasia and Rad3-Related Activation by DNA Damage Mitigates Maladaptive Repair after Acute Kidney Injury.

Nephron

Bonventre JV.
PMID: 34649246
Nephron. 2021 Oct 14;1-4. doi: 10.1159/000519447. Epub 2021 Oct 14.

DNA damage is an important consequence of injury to the proximal tubule. The proximal tubule cell responds to this damage by mounting a DNA damage response (DDR). Two protein kinases, ataxia-telangiectasia mutated (ATM) or ataxia telangiectasia and Rad3-related (ATR),...

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Bonventre JV. Ataxia Telangiectasia and Rad3-Related Activation by DNA Damage Mitigates Maladaptive Repair after Acute Kidney Injury. Nephron. 2021;1-4doi: 10.1159/000519447.
Bonventre, J. V. (2021). Ataxia Telangiectasia and Rad3-Related Activation by DNA Damage Mitigates Maladaptive Repair after Acute Kidney Injury. Nephron, 1-4. https://doi.org/10.1159/000519447
Bonventre, Joseph V. "Ataxia Telangiectasia and Rad3-Related Activation by DNA Damage Mitigates Maladaptive Repair after Acute Kidney Injury." Nephron vol. (2021): 1-4. doi: https://doi.org/10.1159/000519447
Bonventre JV. Ataxia Telangiectasia and Rad3-Related Activation by DNA Damage Mitigates Maladaptive Repair after Acute Kidney Injury. Nephron. 2021 Oct 14;1-4. doi: 10.1159/000519447. Epub 2021 Oct 14. PMID: 34649246.
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Author Correction: Blockade of IL-22 signaling reverses erythroid dysfunction in stress-induced anemias.

Nature immunology

Raundhal M, Ghosh S, Myers SA, Cuoco MS, Singer M, Carr SA, Waikar SS, Bonventre JV, Ritz J, Stone RM, Steensma DP, Regev A, Glimcher LH.
PMID: 34522041
Nat Immunol. 2021 Nov;22(11):1465. doi: 10.1038/s41590-021-01031-y.

No abstract available.

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Raundhal M, Ghosh S, Myers SA, et al. Author Correction: Blockade of IL-22 signaling reverses erythroid dysfunction in stress-induced anemias. Nat Immunol. 2021;22(11):1465doi: 10.1038/s41590-021-01031-y.
Raundhal, M., Ghosh, S., Myers, S. A., Cuoco, M. S., Singer, M., Carr, S. A., Waikar, S. S., Bonventre, J. V., Ritz, J., Stone, R. M., Steensma, D. P., Regev, A., & Glimcher, L. H. (2021). Author Correction: Blockade of IL-22 signaling reverses erythroid dysfunction in stress-induced anemias. Nature immunology, 22(11), 1465. https://doi.org/10.1038/s41590-021-01031-y
Raundhal, Mahesh, et al. "Author Correction: Blockade of IL-22 signaling reverses erythroid dysfunction in stress-induced anemias." Nature immunology vol. 22,11 (2021): 1465. doi: https://doi.org/10.1038/s41590-021-01031-y
Raundhal M, Ghosh S, Myers SA, Cuoco MS, Singer M, Carr SA, Waikar SS, Bonventre JV, Ritz J, Stone RM, Steensma DP, Regev A, Glimcher LH. Author Correction: Blockade of IL-22 signaling reverses erythroid dysfunction in stress-induced anemias. Nat Immunol. 2021 Nov;22(11):1465. doi: 10.1038/s41590-021-01031-y. PMID: 34522041.
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Reply.

The Journal of pediatrics

Yonker LM, Neilan AM, Bartsch Y, Patel AB, Regan J, Arya P, Gootkind E, Park G, Hardcastle M, St John A, Appleman L, Chiu ML, Fialkowski A, De la Flor D, Lima R, Bordt EA, Yockey LJ, D'Avino P, Fischinger S, Shui JE, Lerou PH, Bonventre JV, Yu XG, Ryan ET, Bassett IV, Irimia D, Edlow AG, Alter G, Li JZ, Fasano A.
PMID: 32956696
J Pediatr. 2021 Jan;228:320-323. doi: 10.1016/j.jpeds.2020.09.035. Epub 2020 Sep 18.

No abstract available.

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Yonker LM, Neilan AM, Bartsch Y, et al. Reply. J Pediatr. 2020;228:320-323doi: 10.1016/j.jpeds.2020.09.035.
Yonker, L. M., Neilan, A. M., Bartsch, Y., Patel, A. B., Regan, J., Arya, P., Gootkind, E., Park, G., Hardcastle, M., St John, A., Appleman, L., Chiu, M. L., Fialkowski, A., De la Flor, D., Lima, R., Bordt, E. A., Yockey, L. J., D'Avino, P., Fischinger, S., Shui, J. E., Lerou, P. H., Bonventre, J. V., Yu, X. G., Ryan, E. T., Bassett, I. V., Irimia, D., Edlow, A. G., Alter, G., Li, J. Z., & Fasano, A. (2021). Reply. The Journal of pediatrics, 228320-323. https://doi.org/10.1016/j.jpeds.2020.09.035
Yonker, Lael M, et al. "Reply." The Journal of pediatrics vol. 228 (2021): 320-323. doi: https://doi.org/10.1016/j.jpeds.2020.09.035
Yonker LM, Neilan AM, Bartsch Y, Patel AB, Regan J, Arya P, Gootkind E, Park G, Hardcastle M, St John A, Appleman L, Chiu ML, Fialkowski A, De la Flor D, Lima R, Bordt EA, Yockey LJ, D'Avino P, Fischinger S, Shui JE, Lerou PH, Bonventre JV, Yu XG, Ryan ET, Bassett IV, Irimia D, Edlow AG, Alter G, Li JZ, Fasano A. Reply. J Pediatr. 2021 Jan;228:320-323. doi: 10.1016/j.jpeds.2020.09.035. Epub 2020 Sep 18. PMID: 32956696; PMCID: PMC7500434.
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Molecularly Imprinted Polymer Nanogels for Protein Recognition: Direct Proof of Specific Binding Sites by Solution STD and WaterLOGSY NMR Spectroscopies.

Angewandte Chemie (International ed. in English)

Mier A, Maffucci I, Merlier F, Prost E, Montagna V, Ruiz-Esparza GU, Bonventre JV, Dhal PK, Tse Sum Bui B, Sakhaii P, Haupt K.
PMID: 34296498
Angew Chem Int Ed Engl. 2021 Sep 13;60(38):20849-20857. doi: 10.1002/anie.202106507. Epub 2021 Aug 24.

Molecularly imprinted polymers (MIPs) are tailor-made synthetic antibodies possessing specific binding cavities designed for a target molecule. Currently, MIPs for protein targets are synthesized by imprinting a short surface-exposed fragment of the protein, called epitope or antigenic determinant. However,...

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Mier A, Maffucci I, Merlier F, et al. Molecularly Imprinted Polymer Nanogels for Protein Recognition: Direct Proof of Specific Binding Sites by Solution STD and WaterLOGSY NMR Spectroscopies. Angew Chem Int Ed Engl. 2021;60(38):20849-20857doi: 10.1002/anie.202106507.
Mier, A., Maffucci, I., Merlier, F., Prost, E., Montagna, V., Ruiz-Esparza, G. U., Bonventre, J. V., Dhal, P. K., Tse Sum Bui, B., Sakhaii, P., & Haupt, K. (2021). Molecularly Imprinted Polymer Nanogels for Protein Recognition: Direct Proof of Specific Binding Sites by Solution STD and WaterLOGSY NMR Spectroscopies. Angewandte Chemie (International ed. in English), 60(38), 20849-20857. https://doi.org/10.1002/anie.202106507
Mier, Alejandra, et al. "Molecularly Imprinted Polymer Nanogels for Protein Recognition: Direct Proof of Specific Binding Sites by Solution STD and WaterLOGSY NMR Spectroscopies." Angewandte Chemie (International ed. in English) vol. 60,38 (2021): 20849-20857. doi: https://doi.org/10.1002/anie.202106507
Mier A, Maffucci I, Merlier F, Prost E, Montagna V, Ruiz-Esparza GU, Bonventre JV, Dhal PK, Tse Sum Bui B, Sakhaii P, Haupt K. Molecularly Imprinted Polymer Nanogels for Protein Recognition: Direct Proof of Specific Binding Sites by Solution STD and WaterLOGSY NMR Spectroscopies. Angew Chem Int Ed Engl. 2021 Sep 13;60(38):20849-20857. doi: 10.1002/anie.202106507. Epub 2021 Aug 24. PMID: 34296498; PMCID: PMC8562893.
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