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Cytokine measurement in vivo and in vitro.

Methods in molecular medicine

van Deuren M, Brandtzaeg P.
PMID: 21337161
Methods Mol Med. 2001;67:459-86. doi: 10.1385/1-59259-149-3:459.

Cytokines play a pivotal role in both defense against meningococcal infection and the pathophysiology of invasive meningococcal disease. These chemical messengers are crucial cogwheels in the machinery of the innate immune system and are also powerful forces in the...

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van Deuren M, Brandtzaeg P. Cytokine measurement in vivo and in vitro. Methods Mol Med. 2001;67:459-86doi: 10.1385/1-59259-149-3:459.
van Deuren, M., & Brandtzaeg, P. (2001). Cytokine measurement in vivo and in vitro. Methods in molecular medicine, 67459-86. https://doi.org/10.1385/1-59259-149-3:459
van Deuren, M, and Brandtzaeg, P. "Cytokine measurement in vivo and in vitro." Methods in molecular medicine vol. 67 (2001): 459-86. doi: https://doi.org/10.1385/1-59259-149-3:459
van Deuren M, Brandtzaeg P. Cytokine measurement in vivo and in vitro. Methods Mol Med. 2001;67:459-86. doi: 10.1385/1-59259-149-3:459. PMID: 21337161.
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Investigating Monoliths (Vinyl Azlactone-co-Ethylene Dimethacrylate) as a Support for Enzymes and Drugs, for Proteomics and Drug-Target Studies.

Frontiers in chemistry

Olsen C, Skottvoll FS, Brandtzaeg OK, Schnaars C, Rongved P, Lundanes E, Wilson SR.
PMID: 31850321
Front Chem. 2019 Dec 03;7:835. doi: 10.3389/fchem.2019.00835. eCollection 2019.

Prior to mass spectrometry, on-line sample preparation can be beneficial to reduce manual steps, increase speed, and enable analysis of limited sample amounts. For example, bottom-up proteomics sample preparation and analysis can be accelerated by digesting proteins to peptides...

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Olsen C, Skottvoll FS, Brandtzaeg OK, et al. Investigating Monoliths (Vinyl Azlactone-co-Ethylene Dimethacrylate) as a Support for Enzymes and Drugs, for Proteomics and Drug-Target Studies. Front Chem. 2019;7:835doi: 10.3389/fchem.2019.00835.
Olsen, C., Skottvoll, F. S., Brandtzaeg, O. K., Schnaars, C., Rongved, P., Lundanes, E., & Wilson, S. R. (2019). Investigating Monoliths (Vinyl Azlactone-co-Ethylene Dimethacrylate) as a Support for Enzymes and Drugs, for Proteomics and Drug-Target Studies. Frontiers in chemistry, 7835. https://doi.org/10.3389/fchem.2019.00835
Olsen, Christine, et al. "Investigating Monoliths (Vinyl Azlactone-co-Ethylene Dimethacrylate) as a Support for Enzymes and Drugs, for Proteomics and Drug-Target Studies." Frontiers in chemistry vol. 7 (2019): 835. doi: https://doi.org/10.3389/fchem.2019.00835
Olsen C, Skottvoll FS, Brandtzaeg OK, Schnaars C, Rongved P, Lundanes E, Wilson SR. Investigating Monoliths (Vinyl Azlactone-co-Ethylene Dimethacrylate) as a Support for Enzymes and Drugs, for Proteomics and Drug-Target Studies. Front Chem. 2019 Dec 03;7:835. doi: 10.3389/fchem.2019.00835. eCollection 2019. PMID: 31850321; PMCID: PMC6902630.
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HIV is an inappropriate name for the AIDS virus.

Immunology today

Brandtzaeg P.
PMID: 25291487
Immunol Today. 1987;8(1):8. doi: 10.1016/0167-5699(87)90821-8.

No abstract available.

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Brandtzaeg P. HIV is an inappropriate name for the AIDS virus. Immunol Today. 1987;8(1):8doi: 10.1016/0167-5699(87)90821-8.
Brandtzaeg, P. (1987). HIV is an inappropriate name for the AIDS virus. Immunology today, 8(1), 8. https://doi.org/10.1016/0167-5699(87)90821-8
Brandtzaeg, P. "HIV is an inappropriate name for the AIDS virus." Immunology today vol. 8,1 (1987): 8. doi: https://doi.org/10.1016/0167-5699(87)90821-8
Brandtzaeg P. HIV is an inappropriate name for the AIDS virus. Immunol Today. 1987;8(1):8. doi: 10.1016/0167-5699(87)90821-8. PMID: 25291487.
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Secretory IgA: Designed for Anti-Microbial Defense.

Frontiers in immunology

Brandtzaeg P.
PMID: 23964273
Front Immunol. 2013 Aug 06;4:222. doi: 10.3389/fimmu.2013.00222. eCollection 2013.

Prevention of infections by vaccination remains a compelling goal to improve public health. Mucosal vaccines would make immunization procedures easier, be better suited for mass administration, and most efficiently induce immune exclusion - a term coined for non-inflammatory antibody...

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Brandtzaeg P. Secretory IgA: Designed for Anti-Microbial Defense. Front Immunol. 2013;4:222doi: 10.3389/fimmu.2013.00222.
Brandtzaeg, P. (2013). Secretory IgA: Designed for Anti-Microbial Defense. Frontiers in immunology, 4222. https://doi.org/10.3389/fimmu.2013.00222
Brandtzaeg, Per. "Secretory IgA: Designed for Anti-Microbial Defense." Frontiers in immunology vol. 4 (2013): 222. doi: https://doi.org/10.3389/fimmu.2013.00222
Brandtzaeg P. Secretory IgA: Designed for Anti-Microbial Defense. Front Immunol. 2013 Aug 06;4:222. doi: 10.3389/fimmu.2013.00222. eCollection 2013. PMID: 23964273; PMCID: PMC3734371.
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Immunohistochemistry in research and diagnosis of celiac disease.

Methods in molecular medicine

Brandtzaeg P, Jahnsen FL.
PMID: 21374442
Methods Mol Med. 2000;41:203-39. doi: 10.1385/1-59259-082-9:203.

Immunohistochemistry comprises methods used to recognize tissue components as antigens in situ by means of directly or indirectly labeled antibodies, usually (but not always) derived from another species. When applied to cell preparations, the same methods are called immunocytochemistry...

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Brandtzaeg P, Jahnsen FL. Immunohistochemistry in research and diagnosis of celiac disease. Methods Mol Med. 2000;41:203-39doi: 10.1385/1-59259-082-9:203.
Brandtzaeg, P., & Jahnsen, F. L. (2000). Immunohistochemistry in research and diagnosis of celiac disease. Methods in molecular medicine, 41203-39. https://doi.org/10.1385/1-59259-082-9:203
Brandtzaeg, P, and Jahnsen, F L. "Immunohistochemistry in research and diagnosis of celiac disease." Methods in molecular medicine vol. 41 (2000): 203-39. doi: https://doi.org/10.1385/1-59259-082-9:203
Brandtzaeg P, Jahnsen FL. Immunohistochemistry in research and diagnosis of celiac disease. Methods Mol Med. 2000;41:203-39. doi: 10.1385/1-59259-082-9:203. PMID: 21374442.
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Correction: Impact of extensive antibiotic treatment on faecal carriage of antibiotic-resistant enterobacteria in children in a low resistance prevalence setting.

PloS one

Knudsen PK, Brandtzaeg P, Høiby EA, Bohlin J, Samuelsen Ø, Steinbakk M, Abrahamsen TG, Müller F, Gammelsrud KW.
PMID: 29466441
PLoS One. 2018 Feb 21;13(2):e0193439. doi: 10.1371/journal.pone.0193439. eCollection 2018.

[This corrects the article DOI: 10.1371/journal.pone.0187618.].

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Knudsen PK, Brandtzaeg P, Høiby EA, et al. Correction: Impact of extensive antibiotic treatment on faecal carriage of antibiotic-resistant enterobacteria in children in a low resistance prevalence setting. PLoS One. 2018;13(2):e0193439doi: 10.1371/journal.pone.0193439.
Knudsen, P. K., Brandtzaeg, P., Høiby, E. A., Bohlin, J., Samuelsen, �. �., Steinbakk, M., Abrahamsen, T. G., Müller, F., & Gammelsrud, K. W. (2018). Correction: Impact of extensive antibiotic treatment on faecal carriage of antibiotic-resistant enterobacteria in children in a low resistance prevalence setting. PloS one, 13(2), e0193439. https://doi.org/10.1371/journal.pone.0193439
Knudsen, Per Kristian, et al. "Correction: Impact of extensive antibiotic treatment on faecal carriage of antibiotic-resistant enterobacteria in children in a low resistance prevalence setting." PloS one vol. 13,2 (2018): e0193439. doi: https://doi.org/10.1371/journal.pone.0193439
Knudsen PK, Brandtzaeg P, Høiby EA, Bohlin J, Samuelsen Ø, Steinbakk M, Abrahamsen TG, Müller F, Gammelsrud KW. Correction: Impact of extensive antibiotic treatment on faecal carriage of antibiotic-resistant enterobacteria in children in a low resistance prevalence setting. PLoS One. 2018 Feb 21;13(2):e0193439. doi: 10.1371/journal.pone.0193439. eCollection 2018. PMID: 29466441; PMCID: PMC5821385.
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Compartmentalization of TNF and IL-6 in meningitis and septic shock.

Mediators of inflammation

Waage A, Halstensen A, Espevik T, Brandtzæg P.
PMID: 18475498
Mediators Inflamm. 1993;2(1):23-5. doi: 10.1155/S096293519300002X.

We examined the compartmentalization of bioactive tumour necrosis factor (TNF) and interleukin 6 (IL-6) to the subarachnoid space and systemic circulation in patients with meningococcal meningitis and septic shock/bacteraemia. In patients with meningitis, median levels of TNF in 31...

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Waage A, Halstensen A, Espevik T, et al. Compartmentalization of TNF and IL-6 in meningitis and septic shock. Mediators Inflamm. 1993;2(1):23-5doi: 10.1155/S096293519300002X.
Waage, A., Halstensen, A., Espevik, T., & Brandtzæg, P. (1993). Compartmentalization of TNF and IL-6 in meningitis and septic shock. Mediators of inflammation, 2(1), 23-5. https://doi.org/10.1155/S096293519300002X
Waage, A, et al. "Compartmentalization of TNF and IL-6 in meningitis and septic shock." Mediators of inflammation vol. 2,1 (1993): 23-5. doi: https://doi.org/10.1155/S096293519300002X
Waage A, Halstensen A, Espevik T, Brandtzæg P. Compartmentalization of TNF and IL-6 in meningitis and septic shock. Mediators Inflamm. 1993;2(1):23-5. doi: 10.1155/S096293519300002X. PMID: 18475498; PMCID: PMC2365385.
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Session summary .

Pathobiology : journal of immunopathology, molecular and cellular biology

Brandtzaeg P, Wagner H.
PMID: 9693309
Pathobiology. 1998;66(3):114-6. doi: 10.1159/000028005.

No abstract available.

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Brandtzaeg P, Wagner H. Session summary . Pathobiology. 1998;66(3):114-6doi: 10.1159/000028005.
Brandtzaeg, P., & Wagner, H. (1998). Session summary . Pathobiology : journal of immunopathology, molecular and cellular biology, 66(3), 114-6. https://doi.org/10.1159/000028005
Brandtzaeg, and Wagner. "Session summary ." Pathobiology : journal of immunopathology, molecular and cellular biology vol. 66,3 (1998): 114-6. doi: https://doi.org/10.1159/000028005
Brandtzaeg P, Wagner H. Session summary . Pathobiology. 1998;66(3):114-6. doi: 10.1159/000028005. PMID: 9693309.
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Lack of prognostic significance for p53-overexpression and Ki-67-immunoreactivity in oral T1-2 squamous cell carcinomas.

Oncology reports

Lange D, Heyden A, Lie E, Brandtzaeg P, Boysen M, Clausen O.
PMID: 21590131
Oncol Rep. 1997 Jul-Aug;4(4):737-42. doi: 10.3892/or.4.4.737.

Previous investigations on squamous cell carcinomas (SCC) of the head and neck region have failed to reveal a significant correlation between p53-overexpression or Ki-67-immunoreactivity and survival. Contrary to these studies we restricted the evaluation to T1-2 SCC from the...

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Lange D, Heyden A, Lie E, et al. Lack of prognostic significance for p53-overexpression and Ki-67-immunoreactivity in oral T1-2 squamous cell carcinomas. Oncol Rep. 1997;4(4):737-42doi: 10.3892/or.4.4.737.
Lange, D., Heyden, A., Lie, E., Brandtzaeg, P., Boysen, M., & Clausen, O. (1997). Lack of prognostic significance for p53-overexpression and Ki-67-immunoreactivity in oral T1-2 squamous cell carcinomas. Oncology reports, 4(4), 737-42. https://doi.org/10.3892/or.4.4.737
Lange, D, et al. "Lack of prognostic significance for p53-overexpression and Ki-67-immunoreactivity in oral T1-2 squamous cell carcinomas." Oncology reports vol. 4,4 (1997): 737-42. doi: https://doi.org/10.3892/or.4.4.737
Lange D, Heyden A, Lie E, Brandtzaeg P, Boysen M, Clausen O. Lack of prognostic significance for p53-overexpression and Ki-67-immunoreactivity in oral T1-2 squamous cell carcinomas. Oncol Rep. 1997 Jul-Aug;4(4):737-42. doi: 10.3892/or.4.4.737. PMID: 21590131.
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Secretory immunity with special reference to the oral cavity.

Journal of oral microbiology

Brandtzaeg P.
PMID: 23487566
J Oral Microbiol. 2013;5. doi: 10.3402/jom.v5i0.20401. Epub 2013 Mar 11.

The two principal antibody classes present in saliva are secretory IgA (SIgA) and IgG; the former is produced as dimeric IgA by local plasma cells (PCs) in the stroma of salivary glands and is transported through secretory epithelia by...

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Brandtzaeg P. Secretory immunity with special reference to the oral cavity. J Oral Microbiol. 2013;5doi: 10.3402/jom.v5i0.20401.
Brandtzaeg, P. (2013). Secretory immunity with special reference to the oral cavity. Journal of oral microbiology, 5. https://doi.org/10.3402/jom.v5i0.20401
Brandtzaeg, Per. "Secretory immunity with special reference to the oral cavity." Journal of oral microbiology vol. 5 (2013). doi: https://doi.org/10.3402/jom.v5i0.20401
Brandtzaeg P. Secretory immunity with special reference to the oral cavity. J Oral Microbiol. 2013;5. doi: 10.3402/jom.v5i0.20401. Epub 2013 Mar 11. PMID: 23487566; PMCID: PMC3595421.
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Combined inhibition of C5 and CD14 efficiently attenuated the inflammatory response in a porcine model of meningococcal sepsis.

Journal of intensive care

Hellerud BC, Orrem HL, Dybwik K, Pischke SE, Baratt-Due A, Castellheim A, Fure H, Bergseth G, Christiansen D, Nunn MA, Espevik T, Lau C, Brandtzæg P, Nielsen EW, Mollnes TE.
PMID: 28261486
J Intensive Care. 2017 Feb 27;5:21. doi: 10.1186/s40560-017-0217-0. eCollection 2017.

BACKGROUND: Fulminant meningococcal sepsis, characterized by overwhelming innate immune activation, mostly affects young people and causes high mortality. This study aimed to investigate the effect of targeting two key molecules of innate immunity, complement component C5, and co-receptor CD14...

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Hellerud BC, Orrem HL, Dybwik K, et al. Combined inhibition of C5 and CD14 efficiently attenuated the inflammatory response in a porcine model of meningococcal sepsis. J Intensive Care. 2017;5:21doi: 10.1186/s40560-017-0217-0.
Hellerud, B. C., Orrem, H. L., Dybwik, K., Pischke, S. E., Baratt-Due, A., Castellheim, A., Fure, H., Bergseth, G., Christiansen, D., Nunn, M. A., Espevik, T., Lau, C., Brandtzæg, P., Nielsen, E. W., & Mollnes, T. E. (2017). Combined inhibition of C5 and CD14 efficiently attenuated the inflammatory response in a porcine model of meningococcal sepsis. Journal of intensive care, 521. https://doi.org/10.1186/s40560-017-0217-0
Hellerud, Bernt C, et al. "Combined inhibition of C5 and CD14 efficiently attenuated the inflammatory response in a porcine model of meningococcal sepsis." Journal of intensive care vol. 5 (2017): 21. doi: https://doi.org/10.1186/s40560-017-0217-0
Hellerud BC, Orrem HL, Dybwik K, Pischke SE, Baratt-Due A, Castellheim A, Fure H, Bergseth G, Christiansen D, Nunn MA, Espevik T, Lau C, Brandtzæg P, Nielsen EW, Mollnes TE. Combined inhibition of C5 and CD14 efficiently attenuated the inflammatory response in a porcine model of meningococcal sepsis. J Intensive Care. 2017 Feb 27;5:21. doi: 10.1186/s40560-017-0217-0. eCollection 2017. PMID: 28261486; PMCID: PMC5327570.
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Measurements of cytokine mRNA expression by quantitative polymerase chain reaction in studies of celiac disease.

Methods in molecular medicine

Nilsen EM, Johansen FE, Brandtzaeg P.
PMID: 21374441
Methods Mol Med. 2000;41:185-202. doi: 10.1385/1-59259-082-9:185.

Cytokines are known to play a key regulatory role in immune responses. The onset or progression of immunopathology in various diseases is often associated with aberrant production of one or more cytokines. It is therefore of considerable interest to...

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Nilsen EM, Johansen FE, Brandtzaeg P. Measurements of cytokine mRNA expression by quantitative polymerase chain reaction in studies of celiac disease. Methods Mol Med. 2000;41:185-202doi: 10.1385/1-59259-082-9:185.
Nilsen, E. M., Johansen, F. E., & Brandtzaeg, P. (2000). Measurements of cytokine mRNA expression by quantitative polymerase chain reaction in studies of celiac disease. Methods in molecular medicine, 41185-202. https://doi.org/10.1385/1-59259-082-9:185
Nilsen, E M, et al. "Measurements of cytokine mRNA expression by quantitative polymerase chain reaction in studies of celiac disease." Methods in molecular medicine vol. 41 (2000): 185-202. doi: https://doi.org/10.1385/1-59259-082-9:185
Nilsen EM, Johansen FE, Brandtzaeg P. Measurements of cytokine mRNA expression by quantitative polymerase chain reaction in studies of celiac disease. Methods Mol Med. 2000;41:185-202. doi: 10.1385/1-59259-082-9:185. PMID: 21374441.
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Showing 1 to 12 of 17 entries