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Transparency in publishing.

Journal of travel medicine

Behrens RH, Björkman A, Bryceson A, Corachan M, Freedman D, Hill DR, Jelinek T, Rombo L.
PMID: 15541233
J Travel Med. 2004 Jul-Aug;11(4):263; discussion 264.

No abstract available.

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Behrens RH, Björkman A, Bryceson A, et al. Transparency in publishing. J Travel Med. 2004;11(4):263; discussion 264
Behrens, R. H., Björkman, A., Bryceson, A., Corachan, M., Freedman, D., Hill, D. R., Jelinek, T., & Rombo, L. (2004). Transparency in publishing. Journal of travel medicine, 11(4), 263; discussion 264.
Behrens, Ron H, et al. "Transparency in publishing." Journal of travel medicine vol. 11,4 (2004): 263; discussion 264.
Behrens RH, Björkman A, Bryceson A, Corachan M, Freedman D, Hill DR, Jelinek T, Rombo L. Transparency in publishing. J Travel Med. 2004 Jul-Aug;11(4):263; discussion 264. PMID: 15541233.
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Epigenetic regulation of NK cell differentiation and effector functions.

Frontiers in immunology

Cichocki F, Miller JS, Anderson SK, Bryceson YT.
PMID: 23450696
Front Immunol. 2013 Feb 28;4:55. doi: 10.3389/fimmu.2013.00055. eCollection 2013.

Upon maturation, natural killer (NK) cells acquire effector functions and regulatory receptors. New insights suggest a considerable functional heterogeneity and dynamic regulation of receptor expression in mature human NK cell subsets based on different developmental axes. Such processes include...

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Cichocki F, Miller JS, Anderson SK, et al. Epigenetic regulation of NK cell differentiation and effector functions. Front Immunol. 2013;4:55doi: 10.3389/fimmu.2013.00055.
Cichocki, F., Miller, J. S., Anderson, S. K., & Bryceson, Y. T. (2013). Epigenetic regulation of NK cell differentiation and effector functions. Frontiers in immunology, 455. https://doi.org/10.3389/fimmu.2013.00055
Cichocki, Frank, et al. "Epigenetic regulation of NK cell differentiation and effector functions." Frontiers in immunology vol. 4 (2013): 55. doi: https://doi.org/10.3389/fimmu.2013.00055
Cichocki F, Miller JS, Anderson SK, Bryceson YT. Epigenetic regulation of NK cell differentiation and effector functions. Front Immunol. 2013 Feb 28;4:55. doi: 10.3389/fimmu.2013.00055. eCollection 2013. PMID: 23450696; PMCID: PMC3584244.
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Harnessing features of adaptive NK cells to generate iPSC-derived NK cells for enhanced immunotherapy.

Cell stem cell

Woan KV, Kim H, Bjordahl R, Davis ZB, Gaidarova S, Goulding J, Hancock B, Mahmood S, Abujarour R, Wang H, Tuininga K, Zhang B, Wu CY, Kodal B, Khaw M, Bendzick L, Rogers P, Ge MQ, Bonello G, Meza M, Felices M, Huffman J, Dailey T, Lee TT, Walcheck B, Malmberg KJ, Blazar BR, Bryceson YT, Valamehr B, Miller JS, Cichocki F.
PMID: 34525347
Cell Stem Cell. 2021 Dec 02;28(12):2062-2075.e5. doi: 10.1016/j.stem.2021.08.013. Epub 2021 Sep 14.

Select subsets of immune effector cells have the greatest propensity to mediate antitumor responses. However, procuring these subsets is challenging, and cell-based immunotherapy is hampered by limited effector-cell persistence and lack of on-demand availability. To address these limitations, we...

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Woan KV, Kim H, Bjordahl R, et al. Harnessing features of adaptive NK cells to generate iPSC-derived NK cells for enhanced immunotherapy. Cell Stem Cell. 2021;28(12):2062-2075.e5doi: 10.1016/j.stem.2021.08.013.
Woan, K. V., Kim, H., Bjordahl, R., Davis, Z. B., Gaidarova, S., Goulding, J., Hancock, B., Mahmood, S., Abujarour, R., Wang, H., Tuininga, K., Zhang, B., Wu, C. Y., Kodal, B., Khaw, M., Bendzick, L., Rogers, P., Ge, M. Q., Bonello, G., Meza, M., Felices, M., Huffman, J., Dailey, T., Lee, T. T., Walcheck, B., Malmberg, K. J., Blazar, B. R., Bryceson, Y. T., Valamehr, B., Miller, J. S., & Cichocki, F. (2021). Harnessing features of adaptive NK cells to generate iPSC-derived NK cells for enhanced immunotherapy. Cell stem cell, 28(12), 2062-2075.e5. https://doi.org/10.1016/j.stem.2021.08.013
Woan, Karrune V, et al. "Harnessing features of adaptive NK cells to generate iPSC-derived NK cells for enhanced immunotherapy." Cell stem cell vol. 28,12 (2021): 2062-2075.e5. doi: https://doi.org/10.1016/j.stem.2021.08.013
Woan KV, Kim H, Bjordahl R, Davis ZB, Gaidarova S, Goulding J, Hancock B, Mahmood S, Abujarour R, Wang H, Tuininga K, Zhang B, Wu CY, Kodal B, Khaw M, Bendzick L, Rogers P, Ge MQ, Bonello G, Meza M, Felices M, Huffman J, Dailey T, Lee TT, Walcheck B, Malmberg KJ, Blazar BR, Bryceson YT, Valamehr B, Miller JS, Cichocki F. Harnessing features of adaptive NK cells to generate iPSC-derived NK cells for enhanced immunotherapy. Cell Stem Cell. 2021 Dec 02;28(12):2062-2075.e5. doi: 10.1016/j.stem.2021.08.013. Epub 2021 Sep 14. PMID: 34525347; PMCID: PMC8642276.
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Rubella vaccine-induced granulomas are a novel phenotype with incomplete penetrance of genetic defects in cytotoxicity.

The Journal of allergy and clinical immunology

Groß M, Speckmann C, May A, Gajardo-Carrasco T, Wustrau K, Maier SL, Panning M, Huzly D, Agaimy A, Bryceson YT, Choo S, Chow CW, Dückers G, Fasth A, Fraitag S, Gräwe K, Haxelmans S, Holzinger D, Hudowenz O, Hübschen JM, Khurana C, Kienle K, Klifa R, Korn K, Kutzner H, Lämmermann T, Ledig S, Lipsker D, Meeths M, Naumann-Bartsch N, Rascon J, Schänzer A, Seidl M, Tesi B, Vauloup-Fellous C, Vollmer-Kary B, Warnatz K, Wehr C, Neven B, Vargas P, Sepulveda FE, Lehmberg K, Schmitt-Graeff A, Ehl S.
PMID: 34033843
J Allergy Clin Immunol. 2022 Jan;149(1):388-399.e4. doi: 10.1016/j.jaci.2021.05.007. Epub 2021 May 24.

BACKGROUND: Rubella virus-induced granulomas have been described in patients with various inborn errors of immunity. Most defects impair T-cell immunity, suggesting a critical role of T cells in rubella elimination. However, the molecular mechanism of virus control remains elusive.OBJECTIVE:...

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Groß M, Speckmann C, May A, et al. Rubella vaccine-induced granulomas are a novel phenotype with incomplete penetrance of genetic defects in cytotoxicity. J Allergy Clin Immunol. 2021;149(1):388-399.e4doi: 10.1016/j.jaci.2021.05.007.
Groß, M., Speckmann, C., May, A., Gajardo-Carrasco, T., Wustrau, K., Maier, S. L., Panning, M., Huzly, D., Agaimy, A., Bryceson, Y. T., Choo, S., Chow, C. W., Dückers, G., Fasth, A., Fraitag, S., Gräwe, K., Haxelmans, S., Holzinger, D., Hudowenz, O., Hübschen, J. M., Khurana, C., Kienle, K., Klifa, R., Korn, K., Kutzner, H., Lämmermann, T., Ledig, S., Lipsker, D., Meeths, M., Naumann-Bartsch, N., Rascon, J., Schänzer, A., Seidl, M., Tesi, B., Vauloup-Fellous, C., Vollmer-Kary, B., Warnatz, K., Wehr, C., Neven, B., Vargas, P., Sepulveda, F. E., Lehmberg, K., Schmitt-Graeff, A., & Ehl, S. (2022). Rubella vaccine-induced granulomas are a novel phenotype with incomplete penetrance of genetic defects in cytotoxicity. The Journal of allergy and clinical immunology, 149(1), 388-399.e4. https://doi.org/10.1016/j.jaci.2021.05.007
Groß, Miriam, et al. "Rubella vaccine-induced granulomas are a novel phenotype with incomplete penetrance of genetic defects in cytotoxicity." The Journal of allergy and clinical immunology vol. 149,1 (2022): 388-399.e4. doi: https://doi.org/10.1016/j.jaci.2021.05.007
Groß M, Speckmann C, May A, Gajardo-Carrasco T, Wustrau K, Maier SL, Panning M, Huzly D, Agaimy A, Bryceson YT, Choo S, Chow CW, Dückers G, Fasth A, Fraitag S, Gräwe K, Haxelmans S, Holzinger D, Hudowenz O, Hübschen JM, Khurana C, Kienle K, Klifa R, Korn K, Kutzner H, Lämmermann T, Ledig S, Lipsker D, Meeths M, Naumann-Bartsch N, Rascon J, Schänzer A, Seidl M, Tesi B, Vauloup-Fellous C, Vollmer-Kary B, Warnatz K, Wehr C, Neven B, Vargas P, Sepulveda FE, Lehmberg K, Schmitt-Graeff A, Ehl S. Rubella vaccine-induced granulomas are a novel phenotype with incomplete penetrance of genetic defects in cytotoxicity. J Allergy Clin Immunol. 2022 Jan;149(1):388-399.e4. doi: 10.1016/j.jaci.2021.05.007. Epub 2021 May 24. PMID: 34033843.
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Lymphocyte cytotoxicity: tug-of-war on microtubules.

Blood

Wood SM, Bryceson YT.
PMID: 22538494
Blood. 2012 Apr 26;119(17):3873-5. doi: 10.1182/blood-2012-02-410381.

Lymphocyte cytotoxicity is essential in immune defense. In this issue of Blood, Kurowska and colleagues define a Rab27a/Slp3/kinesin-1 complex that facilitates anterograde microtubule transport of lytic granules, representing a critical step in lymphocyte granule exocytosis and cytotoxicity.

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Wood SM, Bryceson YT. Lymphocyte cytotoxicity: tug-of-war on microtubules. Blood. 2012;119(17):3873-5doi: 10.1182/blood-2012-02-410381.
Wood, S. M., & Bryceson, Y. T. (2012). Lymphocyte cytotoxicity: tug-of-war on microtubules. Blood, 119(17), 3873-5. https://doi.org/10.1182/blood-2012-02-410381
Wood, Stephanie M, and Bryceson, Yenan T. "Lymphocyte cytotoxicity: tug-of-war on microtubules." Blood vol. 119,17 (2012): 3873-5. doi: https://doi.org/10.1182/blood-2012-02-410381
Wood SM, Bryceson YT. Lymphocyte cytotoxicity: tug-of-war on microtubules. Blood. 2012 Apr 26;119(17):3873-5. doi: 10.1182/blood-2012-02-410381. PMID: 22538494.
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The transcription factor Bcl11b promotes both canonical and adaptive NK cell differentiation.

Science immunology

Holmes TD, Pandey RV, Helm EY, Schlums H, Han H, Campbell TM, Drashansky TT, Chiang S, Wu CY, Tao C, Shoukier M, Tolosa E, Von Hardenberg S, Sun M, Klemann C, Marsh RA, Lau CM, Lin Y, Sun JC, Månsson R, Cichocki F, Avram D, Bryceson YT.
PMID: 33712472
Sci Immunol. 2021 Mar 12;6(57). doi: 10.1126/sciimmunol.abc9801.

Epigenetic landscapes can provide insight into regulation of gene expression and cellular diversity. Here, we examined the transcriptional and epigenetic profiles of seven human blood natural killer (NK) cell populations, including adaptive NK cells. The

Cite
Holmes TD, Pandey RV, Helm EY, et al. The transcription factor Bcl11b promotes both canonical and adaptive NK cell differentiation. Sci Immunol. 2021;6(57)doi: 10.1126/sciimmunol.abc9801.
Holmes, T. D., Pandey, R. V., Helm, E. Y., Schlums, H., Han, H., Campbell, T. M., Drashansky, T. T., Chiang, S., Wu, C. Y., Tao, C., Shoukier, M., Tolosa, E., Von Hardenberg, S., Sun, M., Klemann, C., Marsh, R. A., Lau, C. M., Lin, Y., Sun, J. C., Månsson, R., Cichocki, F., Avram, D., & Bryceson, Y. T. (2021). The transcription factor Bcl11b promotes both canonical and adaptive NK cell differentiation. Science immunology, 6(57), . https://doi.org/10.1126/sciimmunol.abc9801
Holmes, Tim D, et al. "The transcription factor Bcl11b promotes both canonical and adaptive NK cell differentiation." Science immunology vol. 6,57 (2021). doi: https://doi.org/10.1126/sciimmunol.abc9801
Holmes TD, Pandey RV, Helm EY, Schlums H, Han H, Campbell TM, Drashansky TT, Chiang S, Wu CY, Tao C, Shoukier M, Tolosa E, Von Hardenberg S, Sun M, Klemann C, Marsh RA, Lau CM, Lin Y, Sun JC, Månsson R, Cichocki F, Avram D, Bryceson YT. The transcription factor Bcl11b promotes both canonical and adaptive NK cell differentiation. Sci Immunol. 2021 Mar 12;6(57). doi: 10.1126/sciimmunol.abc9801. PMID: 33712472; PMCID: PMC8274449.
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Adult-Onset Ataxia With Neuropathy and White Matter Abnormalities Due to a Novel .

Neurology. Genetics

Paucar M, Tesi B, Eshtad S, Eriksson C, Hashim F, Nilsson D, Pourhamidi K, Hellström-Lindberg E, Bryceson YT, Svenningsson P.
PMID: 34722875
Neurol Genet. 2021 Oct 28;7(6):e628. doi: 10.1212/NXG.0000000000000628. eCollection 2021 Dec.

No abstract available.

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Paucar M, Tesi B, Eshtad S, et al. Adult-Onset Ataxia With Neuropathy and White Matter Abnormalities Due to a Novel . Neurol Genet. 2021;7(6):e628doi: 10.1212/NXG.0000000000000628.
Paucar, M., Tesi, B., Eshtad, S., Eriksson, C., Hashim, F., Nilsson, D., Pourhamidi, K., Hellström-Lindberg, E., Bryceson, Y. T., & Svenningsson, P. (2021). Adult-Onset Ataxia With Neuropathy and White Matter Abnormalities Due to a Novel . Neurology. Genetics, 7(6), e628. https://doi.org/10.1212/NXG.0000000000000628
Paucar, Martin, et al. "Adult-Onset Ataxia With Neuropathy and White Matter Abnormalities Due to a Novel ." Neurology. Genetics vol. 7,6 (2021): e628. doi: https://doi.org/10.1212/NXG.0000000000000628
Paucar M, Tesi B, Eshtad S, Eriksson C, Hashim F, Nilsson D, Pourhamidi K, Hellström-Lindberg E, Bryceson YT, Svenningsson P. Adult-Onset Ataxia With Neuropathy and White Matter Abnormalities Due to a Novel . Neurol Genet. 2021 Oct 28;7(6):e628. doi: 10.1212/NXG.0000000000000628. eCollection 2021 Dec. PMID: 34722875; PMCID: PMC8554712.
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Unperturbed Cytotoxic Lymphocyte Phenotype and Function in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome Patients.

Frontiers in immunology

Theorell J, Bileviciute-Ljungar I, Tesi B, Schlums H, Johnsgaard MS, Asadi-Azarbaijani B, Bolle Strand E, Bryceson YT.
PMID: 28694809
Front Immunol. 2017 Jun 26;8:723. doi: 10.3389/fimmu.2017.00723. eCollection 2017.

Myalgic encephalomyelitis or chronic fatigue syndrome (ME/CFS) is a debilitating disorder linked to diverse intracellular infections as well as physiological stress. Cytotoxic lymphocytes combat intracellular infections. Their function is attenuated by stress. Despite numerous studies, the role of cytotoxic...

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Theorell J, Bileviciute-Ljungar I, Tesi B, et al. Unperturbed Cytotoxic Lymphocyte Phenotype and Function in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome Patients. Front Immunol. 2017;8:723doi: 10.3389/fimmu.2017.00723.
Theorell, J., Bileviciute-Ljungar, I., Tesi, B., Schlums, H., Johnsgaard, M. S., Asadi-Azarbaijani, B., Bolle Strand, E., & Bryceson, Y. T. (2017). Unperturbed Cytotoxic Lymphocyte Phenotype and Function in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome Patients. Frontiers in immunology, 8723. https://doi.org/10.3389/fimmu.2017.00723
Theorell, Jakob, et al. "Unperturbed Cytotoxic Lymphocyte Phenotype and Function in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome Patients." Frontiers in immunology vol. 8 (2017): 723. doi: https://doi.org/10.3389/fimmu.2017.00723
Theorell J, Bileviciute-Ljungar I, Tesi B, Schlums H, Johnsgaard MS, Asadi-Azarbaijani B, Bolle Strand E, Bryceson YT. Unperturbed Cytotoxic Lymphocyte Phenotype and Function in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome Patients. Front Immunol. 2017 Jun 26;8:723. doi: 10.3389/fimmu.2017.00723. eCollection 2017. PMID: 28694809; PMCID: PMC5483846.
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Practical progress and new drugs for changing patterns of leishmaniasis.

Parasitology today (Personal ed.)

Olliaro PL, Bryceson AD.
PMID: 15463794
Parasitol Today. 1993 Sep;9(9):323-8. doi: 10.1016/0169-4758(93)90231-4.

The problems surrounding leishmaniasis are changing. An increase in travel, the Indian and Sudanese epidemics of visceral leishmaniasis, parasite resistance to antimony and the emergence of AIDS-related leishmaniasis have all increased the urgency for new drugs, and led to...

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Olliaro PL, Bryceson AD. Practical progress and new drugs for changing patterns of leishmaniasis. Parasitol Today. 1993;9(9):323-8doi: 10.1016/0169-4758(93)90231-4.
Olliaro, P. L., & Bryceson, A. D. (1993). Practical progress and new drugs for changing patterns of leishmaniasis. Parasitology today (Personal ed.), 9(9), 323-8. https://doi.org/10.1016/0169-4758(93)90231-4
Olliaro, P L, and Bryceson, A D. "Practical progress and new drugs for changing patterns of leishmaniasis." Parasitology today (Personal ed.) vol. 9,9 (1993): 323-8. doi: https://doi.org/10.1016/0169-4758(93)90231-4
Olliaro PL, Bryceson AD. Practical progress and new drugs for changing patterns of leishmaniasis. Parasitol Today. 1993 Sep;9(9):323-8. doi: 10.1016/0169-4758(93)90231-4. PMID: 15463794.
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Differences in Granule Morphology yet Equally Impaired Exocytosis among Cytotoxic T Cells and NK Cells from Chediak-Higashi Syndrome Patients.

Frontiers in immunology

Chiang SCC, Wood SM, Tesi B, Akar HH, Al-Herz W, Ammann S, Belen FB, Caliskan U, Kaya Z, Lehmberg K, Patiroglu T, Tokgoz H, Ünüvar A, Introne WJ, Henter JI, Nordenskjöld M, Ljunggren HG, Meeths M, Ehl S, Krzewski K, Bryceson YT.
PMID: 28458669
Front Immunol. 2017 Apr 18;8:426. doi: 10.3389/fimmu.2017.00426. eCollection 2017.

Chediak-Higashi syndrome (CHS) is caused by autosomal recessive mutations in

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Chiang SCC, Wood SM, Tesi B, et al. Differences in Granule Morphology yet Equally Impaired Exocytosis among Cytotoxic T Cells and NK Cells from Chediak-Higashi Syndrome Patients. Front Immunol. 2017;8:426doi: 10.3389/fimmu.2017.00426.
Chiang, S. C. C., Wood, S. M., Tesi, B., Akar, H. H., Al-Herz, W., Ammann, S., Belen, F. B., Caliskan, U., Kaya, Z., Lehmberg, K., Patiroglu, T., Tokgoz, H., Ünüvar, A., Introne, W. J., Henter, J. I., Nordenskjöld, M., Ljunggren, H. G., Meeths, M., Ehl, S., Krzewski, K., & Bryceson, Y. T. (2017). Differences in Granule Morphology yet Equally Impaired Exocytosis among Cytotoxic T Cells and NK Cells from Chediak-Higashi Syndrome Patients. Frontiers in immunology, 8426. https://doi.org/10.3389/fimmu.2017.00426
Chiang, Samuel C C, et al. "Differences in Granule Morphology yet Equally Impaired Exocytosis among Cytotoxic T Cells and NK Cells from Chediak-Higashi Syndrome Patients." Frontiers in immunology vol. 8 (2017): 426. doi: https://doi.org/10.3389/fimmu.2017.00426
Chiang SCC, Wood SM, Tesi B, Akar HH, Al-Herz W, Ammann S, Belen FB, Caliskan U, Kaya Z, Lehmberg K, Patiroglu T, Tokgoz H, Ünüvar A, Introne WJ, Henter JI, Nordenskjöld M, Ljunggren HG, Meeths M, Ehl S, Krzewski K, Bryceson YT. Differences in Granule Morphology yet Equally Impaired Exocytosis among Cytotoxic T Cells and NK Cells from Chediak-Higashi Syndrome Patients. Front Immunol. 2017 Apr 18;8:426. doi: 10.3389/fimmu.2017.00426. eCollection 2017. PMID: 28458669; PMCID: PMC5394158.
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The transcription factor Bcl11b promotes both canonical and adaptive NK cell differentiation.

Science immunology

Holmes TD, Pandey RV, Helm EY, Schlums H, Han H, Campbell TM, Drashansky TT, Chiang S, Wu CY, Tao C, Shoukier M, Tolosa E, Von Hardenberg S, Sun M, Klemann C, Marsh RA, Lau CM, Lin Y, Sun JC, Månsson R, Cichocki F, Avram D, Bryceson YT.
PMID: 33712472
Sci Immunol. 2021 Mar 12;6(57). doi: 10.1126/sciimmunol.abc9801.

Epigenetic landscapes can provide insight into regulation of gene expression and cellular diversity. Here, we examined the transcriptional and epigenetic profiles of seven human blood natural killer (NK) cell populations, including adaptive NK cells. The

Cite
Holmes TD, Pandey RV, Helm EY, et al. The transcription factor Bcl11b promotes both canonical and adaptive NK cell differentiation. Sci Immunol. 2021;6(57)doi: 10.1126/sciimmunol.abc9801.
Holmes, T. D., Pandey, R. V., Helm, E. Y., Schlums, H., Han, H., Campbell, T. M., Drashansky, T. T., Chiang, S., Wu, C. Y., Tao, C., Shoukier, M., Tolosa, E., Von Hardenberg, S., Sun, M., Klemann, C., Marsh, R. A., Lau, C. M., Lin, Y., Sun, J. C., Månsson, R., Cichocki, F., Avram, D., & Bryceson, Y. T. (2021). The transcription factor Bcl11b promotes both canonical and adaptive NK cell differentiation. Science immunology, 6(57), . https://doi.org/10.1126/sciimmunol.abc9801
Holmes, Tim D, et al. "The transcription factor Bcl11b promotes both canonical and adaptive NK cell differentiation." Science immunology vol. 6,57 (2021). doi: https://doi.org/10.1126/sciimmunol.abc9801
Holmes TD, Pandey RV, Helm EY, Schlums H, Han H, Campbell TM, Drashansky TT, Chiang S, Wu CY, Tao C, Shoukier M, Tolosa E, Von Hardenberg S, Sun M, Klemann C, Marsh RA, Lau CM, Lin Y, Sun JC, Månsson R, Cichocki F, Avram D, Bryceson YT. The transcription factor Bcl11b promotes both canonical and adaptive NK cell differentiation. Sci Immunol. 2021 Mar 12;6(57). doi: 10.1126/sciimmunol.abc9801. PMID: 33712472; PMCID: PMC8274449.
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Rubella vaccine-induced granulomas are a novel phenotype with incomplete penetrance of genetic defects in cytotoxicity.

The Journal of allergy and clinical immunology

Groß M, Speckmann C, May A, Gajardo-Carrasco T, Wustrau K, Maier SL, Panning M, Huzly D, Agaimy A, Bryceson YT, Choo S, Chow CW, Dückers G, Fasth A, Fraitag S, Gräwe K, Haxelmans S, Holzinger D, Hudowenz O, Hübschen JM, Khurana C, Kienle K, Klifa R, Korn K, Kutzner H, Lämmermann T, Ledig S, Lipsker D, Meeths M, Naumann-Bartsch N, Rascon J, Schänzer A, Seidl M, Tesi B, Vauloup-Fellous C, Vollmer-Kary B, Warnatz K, Wehr C, Neven B, Vargas P, Sepulveda FE, Lehmberg K, Schmitt-Graeff A, Ehl S.
PMID: 34033843
J Allergy Clin Immunol. 2021 May 24; doi: 10.1016/j.jaci.2021.05.007. Epub 2021 May 24.

BACKGROUND: Rubella virus-induced granulomas have been described in patients with various inborn errors of immunity. Most defects impair T-cell immunity, suggesting a critical role of T cells in rubella elimination. However, the molecular mechanism of virus control remains elusive.OBJECTIVE:...

Cite
Groß M, Speckmann C, May A, et al. Rubella vaccine-induced granulomas are a novel phenotype with incomplete penetrance of genetic defects in cytotoxicity. J Allergy Clin Immunol. 2021;doi: 10.1016/j.jaci.2021.05.007.
Groß, M., Speckmann, C., May, A., Gajardo-Carrasco, T., Wustrau, K., Maier, S. L., Panning, M., Huzly, D., Agaimy, A., Bryceson, Y. T., Choo, S., Chow, C. W., Dückers, G., Fasth, A., Fraitag, S., Gräwe, K., Haxelmans, S., Holzinger, D., Hudowenz, O., Hübschen, J. M., Khurana, C., Kienle, K., Klifa, R., Korn, K., Kutzner, H., Lämmermann, T., Ledig, S., Lipsker, D., Meeths, M., Naumann-Bartsch, N., Rascon, J., Schänzer, A., Seidl, M., Tesi, B., Vauloup-Fellous, C., Vollmer-Kary, B., Warnatz, K., Wehr, C., Neven, B., Vargas, P., Sepulveda, F. E., Lehmberg, K., Schmitt-Graeff, A., & Ehl, S. (2021). Rubella vaccine-induced granulomas are a novel phenotype with incomplete penetrance of genetic defects in cytotoxicity. The Journal of allergy and clinical immunology, . https://doi.org/10.1016/j.jaci.2021.05.007
Groß, Miriam, et al. "Rubella vaccine-induced granulomas are a novel phenotype with incomplete penetrance of genetic defects in cytotoxicity." The Journal of allergy and clinical immunology vol. (2021). doi: https://doi.org/10.1016/j.jaci.2021.05.007
Groß M, Speckmann C, May A, Gajardo-Carrasco T, Wustrau K, Maier SL, Panning M, Huzly D, Agaimy A, Bryceson YT, Choo S, Chow CW, Dückers G, Fasth A, Fraitag S, Gräwe K, Haxelmans S, Holzinger D, Hudowenz O, Hübschen JM, Khurana C, Kienle K, Klifa R, Korn K, Kutzner H, Lämmermann T, Ledig S, Lipsker D, Meeths M, Naumann-Bartsch N, Rascon J, Schänzer A, Seidl M, Tesi B, Vauloup-Fellous C, Vollmer-Kary B, Warnatz K, Wehr C, Neven B, Vargas P, Sepulveda FE, Lehmberg K, Schmitt-Graeff A, Ehl S. Rubella vaccine-induced granulomas are a novel phenotype with incomplete penetrance of genetic defects in cytotoxicity. J Allergy Clin Immunol. 2021 May 24; doi: 10.1016/j.jaci.2021.05.007. Epub 2021 May 24. PMID: 34033843.
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