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Showing 1 to 12 of 22 entries
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Pathway analysis of differentially expressed genes in patients with acute aortic dissection.

Biomarker insights

Mohamed SA, Sievers HH, Hanke T, Richardt D, Schmidtke C, Charitos EI, Belge G, Bullerdiek J.
PMID: 19652764
Biomark Insights. 2009 May 06;4:81-90. doi: 10.4137/bmi.s2530.

BACKGROUND: Acute aortic dissection (AAD) is a life-threatening condition with high mortality and a relatively unclarified pathophysiological mechanism. Although differentially expressed genes in AAD have been recognized, interactions between these genes remain poorly defined. This study was conducted to...

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Mohamed SA, Sievers HH, Hanke T, et al. Pathway analysis of differentially expressed genes in patients with acute aortic dissection. Biomark Insights. 2009;4:81-90doi: 10.4137/bmi.s2530.
Mohamed, S. A., Sievers, H. H., Hanke, T., Richardt, D., Schmidtke, C., Charitos, E. I., Belge, G., & Bullerdiek, J. (2009). Pathway analysis of differentially expressed genes in patients with acute aortic dissection. Biomarker insights, 481-90. https://doi.org/10.4137/bmi.s2530
Mohamed, Salah A, et al. "Pathway analysis of differentially expressed genes in patients with acute aortic dissection." Biomarker insights vol. 4 (2009): 81-90. doi: https://doi.org/10.4137/bmi.s2530
Mohamed SA, Sievers HH, Hanke T, Richardt D, Schmidtke C, Charitos EI, Belge G, Bullerdiek J. Pathway analysis of differentially expressed genes in patients with acute aortic dissection. Biomark Insights. 2009 May 06;4:81-90. doi: 10.4137/bmi.s2530. PMID: 19652764; PMCID: PMC2716678.
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Co-transfection of plasmid DNA and laser-generated gold nanoparticles does not disturb the bioactivity of GFP-HMGB1 fusion protein.

Journal of nanobiotechnology

Petersen S, Soller JT, Wagner S, Richter A, Bullerdiek J, Nolte I, Barcikowski S, Murua Escobar H.
PMID: 19852831
J Nanobiotechnology. 2009 Oct 24;7:6. doi: 10.1186/1477-3155-7-6.

Ultrashort pulsed laser ablation in liquids represents a powerful tool for the generation of pure gold nanoparticles (AuNPs) avoiding chemical precursors and thereby making them especially interesting for biomedical applications. However, because of their electron accepting properties, laser-generated AuNPs...

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Petersen S, Soller JT, Wagner S, et al. Co-transfection of plasmid DNA and laser-generated gold nanoparticles does not disturb the bioactivity of GFP-HMGB1 fusion protein. J Nanobiotechnology. 2009;7:6doi: 10.1186/1477-3155-7-6.
Petersen, S., Soller, J. T., Wagner, S., Richter, A., Bullerdiek, J., Nolte, I., Barcikowski, S., & Murua Escobar, H. (2009). Co-transfection of plasmid DNA and laser-generated gold nanoparticles does not disturb the bioactivity of GFP-HMGB1 fusion protein. Journal of nanobiotechnology, 76. https://doi.org/10.1186/1477-3155-7-6
Petersen, Svea, et al. "Co-transfection of plasmid DNA and laser-generated gold nanoparticles does not disturb the bioactivity of GFP-HMGB1 fusion protein." Journal of nanobiotechnology vol. 7 (2009): 6. doi: https://doi.org/10.1186/1477-3155-7-6
Petersen S, Soller JT, Wagner S, Richter A, Bullerdiek J, Nolte I, Barcikowski S, Murua Escobar H. Co-transfection of plasmid DNA and laser-generated gold nanoparticles does not disturb the bioactivity of GFP-HMGB1 fusion protein. J Nanobiotechnology. 2009 Oct 24;7:6. doi: 10.1186/1477-3155-7-6. PMID: 19852831; PMCID: PMC2775017.
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Decrease in thyroid adenoma associated (THADA) expression is a marker of dedifferentiation of thyroid tissue.

BMC clinical pathology

Kloth L, Belge G, Burchardt K, Loeschke S, Wosniok W, Fu X, Nimzyk R, Mohamed SA, Drieschner N, Rippe V, Bullerdiek J.
PMID: 22050638
BMC Clin Pathol. 2011 Nov 04;11:13. doi: 10.1186/1472-6890-11-13.

BACKGROUND: Thyroid adenoma associated (THADA) has been identified as the target gene affected by chromosome 2p21 translocations in thyroid adenomas, but the role of THADA in the thyroid is still elusive. The aim of this study was to quantify...

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Kloth L, Belge G, Burchardt K, et al. Decrease in thyroid adenoma associated (THADA) expression is a marker of dedifferentiation of thyroid tissue. BMC Clin Pathol. 2011;11:13doi: 10.1186/1472-6890-11-13.
Kloth, L., Belge, G., Burchardt, K., Loeschke, S., Wosniok, W., Fu, X., Nimzyk, R., Mohamed, S. A., Drieschner, N., Rippe, V., & Bullerdiek, J. (2011). Decrease in thyroid adenoma associated (THADA) expression is a marker of dedifferentiation of thyroid tissue. BMC clinical pathology, 1113. https://doi.org/10.1186/1472-6890-11-13
Kloth, Lars, et al. "Decrease in thyroid adenoma associated (THADA) expression is a marker of dedifferentiation of thyroid tissue." BMC clinical pathology vol. 11 (2011): 13. doi: https://doi.org/10.1186/1472-6890-11-13
Kloth L, Belge G, Burchardt K, Loeschke S, Wosniok W, Fu X, Nimzyk R, Mohamed SA, Drieschner N, Rippe V, Bullerdiek J. Decrease in thyroid adenoma associated (THADA) expression is a marker of dedifferentiation of thyroid tissue. BMC Clin Pathol. 2011 Nov 04;11:13. doi: 10.1186/1472-6890-11-13. PMID: 22050638; PMCID: PMC3229435.
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Chromosomal assignment of canine THADA gene to CFA 10q25.

Molecular cytogenetics

Soller JT, Beuing C, Murua Escobar H, Winkler S, Reimann-Berg N, Drieschner N, Dolf G, Schelling C, Nolte I, Bullerdiek J.
PMID: 18522714
Mol Cytogenet. 2008 Jun 03;1:11. doi: 10.1186/1755-8166-1-11.

BACKGROUND: Chromosomal translocations affecting the chromosome 2p21 cluster in a 450 kb breakpoint region are frequently observed in human benign thyroid adenomas. THADA (thyroid adenoma associated) was identified as the affected gene within this breakpoint region. In contrast to...

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Soller JT, Beuing C, Murua Escobar H, et al. Chromosomal assignment of canine THADA gene to CFA 10q25. Mol Cytogenet. 2008;1:11doi: 10.1186/1755-8166-1-11.
Soller, J. T., Beuing, C., Murua Escobar, H., Winkler, S., Reimann-Berg, N., Drieschner, N., Dolf, G., Schelling, C., Nolte, I., & Bullerdiek, J. (2008). Chromosomal assignment of canine THADA gene to CFA 10q25. Molecular cytogenetics, 111. https://doi.org/10.1186/1755-8166-1-11
Soller, Jan T, et al. "Chromosomal assignment of canine THADA gene to CFA 10q25." Molecular cytogenetics vol. 1 (2008): 11. doi: https://doi.org/10.1186/1755-8166-1-11
Soller JT, Beuing C, Murua Escobar H, Winkler S, Reimann-Berg N, Drieschner N, Dolf G, Schelling C, Nolte I, Bullerdiek J. Chromosomal assignment of canine THADA gene to CFA 10q25. Mol Cytogenet. 2008 Jun 03;1:11. doi: 10.1186/1755-8166-1-11. PMID: 18522714; PMCID: PMC2430699.
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Generation of an iPSC line (AKOSi004-A) from fibroblasts of a female adult NPC1 patient, carrying the compound heterozygous mutation p.Val1023Serfs*15/p.Gly992Arg and of an iPSC line (AKOSi005-A) from a female adult control individual.

Stem cell research

Völkner C, Liedtke M, Petters J, Lukas J, Escobar HM, Knuebel G, Bullerdiek J, Holzmann C, Hermann A, Frech MJ.
PMID: 33360098
Stem Cell Res. 2020 Dec 15;50:102127. doi: 10.1016/j.scr.2020.102127. Epub 2020 Dec 15.

Niemann-Pick disease Type C (NPC) is a rare progressive neurodegenerative disorder with an incidence of 1:120,000 caused by mutations in the NPC1 or NPC2 gene leading to a massive cholesterol accumulation. Here, we describe the generation of induced pluripotent...

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Völkner C, Liedtke M, Petters J, et al. Generation of an iPSC line (AKOSi004-A) from fibroblasts of a female adult NPC1 patient, carrying the compound heterozygous mutation p.Val1023Serfs*15/p.Gly992Arg and of an iPSC line (AKOSi005-A) from a female adult control individual. Stem Cell Res. 2020;50:102127doi: 10.1016/j.scr.2020.102127.
Völkner, C., Liedtke, M., Petters, J., Lukas, J., Escobar, H. M., Knuebel, G., Bullerdiek, J., Holzmann, C., Hermann, A., & Frech, M. J. (2020). Generation of an iPSC line (AKOSi004-A) from fibroblasts of a female adult NPC1 patient, carrying the compound heterozygous mutation p.Val1023Serfs*15/p.Gly992Arg and of an iPSC line (AKOSi005-A) from a female adult control individual. Stem cell research, 50102127. https://doi.org/10.1016/j.scr.2020.102127
Völkner, Christin, et al. "Generation of an iPSC line (AKOSi004-A) from fibroblasts of a female adult NPC1 patient, carrying the compound heterozygous mutation p.Val1023Serfs*15/p.Gly992Arg and of an iPSC line (AKOSi005-A) from a female adult control individual." Stem cell research vol. 50 (2020): 102127. doi: https://doi.org/10.1016/j.scr.2020.102127
Völkner C, Liedtke M, Petters J, Lukas J, Escobar HM, Knuebel G, Bullerdiek J, Holzmann C, Hermann A, Frech MJ. Generation of an iPSC line (AKOSi004-A) from fibroblasts of a female adult NPC1 patient, carrying the compound heterozygous mutation p.Val1023Serfs*15/p.Gly992Arg and of an iPSC line (AKOSi005-A) from a female adult control individual. Stem Cell Res. 2020 Dec 15;50:102127. doi: 10.1016/j.scr.2020.102127. Epub 2020 Dec 15. PMID: 33360098.
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The mitotic spindle: linking teratogenic effects of Zika virus with human genetics?.

Molecular cytogenetics

Bullerdiek J, Dotzauer A, Bauer I.
PMID: 27099632
Mol Cytogenet. 2016 Apr 19;9:32. doi: 10.1186/s13039-016-0240-1. eCollection 2016.

BACKGROUND: Recently, an association between Zika virus infection and microcephaly/ocular findings was found to be reasonable e.g. because of the demonstration that the virus was found in the brain of a fetus after presumed maternal infection. Although there is...

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Bullerdiek J, Dotzauer A, Bauer I. The mitotic spindle: linking teratogenic effects of Zika virus with human genetics?. Mol Cytogenet. 2016;9:32doi: 10.1186/s13039-016-0240-1.
Bullerdiek, J., Dotzauer, A., & Bauer, I. (2016). The mitotic spindle: linking teratogenic effects of Zika virus with human genetics?. Molecular cytogenetics, 932. https://doi.org/10.1186/s13039-016-0240-1
Bullerdiek, Joern, et al. "The mitotic spindle: linking teratogenic effects of Zika virus with human genetics?." Molecular cytogenetics vol. 9 (2016): 32. doi: https://doi.org/10.1186/s13039-016-0240-1
Bullerdiek J, Dotzauer A, Bauer I. The mitotic spindle: linking teratogenic effects of Zika virus with human genetics?. Mol Cytogenet. 2016 Apr 19;9:32. doi: 10.1186/s13039-016-0240-1. eCollection 2016. PMID: 27099632; PMCID: PMC4837584.
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HMGA2 expression in white adipose tissue linking cellular senescence with diabetes.

Genes & nutrition

Markowski DN, Thies HW, Gottlieb A, Wenk H, Wischnewsky M, Bullerdiek J.
PMID: 23881689
Genes Nutr. 2013 Sep;8(5):449-56. doi: 10.1007/s12263-013-0354-6. Epub 2013 Jul 24.

There is a clear link between overweight, gain of white adipose tissue, and diabetes type 2 (T2D). The molecular mechanism of the gain of adipose tissue is linked with the expression of high mobility group protein AT-hook 2 (HMGA2),...

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Markowski DN, Thies HW, Gottlieb A, et al. HMGA2 expression in white adipose tissue linking cellular senescence with diabetes. Genes Nutr. 2013;8(5):449-56doi: 10.1007/s12263-013-0354-6.
Markowski, D. N., Thies, H. W., Gottlieb, A., Wenk, H., Wischnewsky, M., & Bullerdiek, J. (2013). HMGA2 expression in white adipose tissue linking cellular senescence with diabetes. Genes & nutrition, 8(5), 449-56. https://doi.org/10.1007/s12263-013-0354-6
Markowski, Dominique Nadine, et al. "HMGA2 expression in white adipose tissue linking cellular senescence with diabetes." Genes & nutrition vol. 8,5 (2013): 449-56. doi: https://doi.org/10.1007/s12263-013-0354-6
Markowski DN, Thies HW, Gottlieb A, Wenk H, Wischnewsky M, Bullerdiek J. HMGA2 expression in white adipose tissue linking cellular senescence with diabetes. Genes Nutr. 2013 Sep;8(5):449-56. doi: 10.1007/s12263-013-0354-6. Epub 2013 Jul 24. PMID: 23881689; PMCID: PMC3755135.
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Gross genetic alterations and genetic heterogeneity in a periductal stromal tumor of the breast.

Molecular cytogenetics

Holzmann C, Helmke B, Bullerdiek J.
PMID: 33292379
Mol Cytogenet. 2020 Nov 23;13(1):49. doi: 10.1186/s13039-020-00516-z.

BACKGROUND: Periductal stromal tumors of the breast are exceedingly rare biphasic breast tumors with close morphological relationship to phyllodes tumors. So far, results of genetic analyses on these tumors have not been reported.CASE PRESENTATION: A 50 year old female...

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Holzmann C, Helmke B, Bullerdiek J. Gross genetic alterations and genetic heterogeneity in a periductal stromal tumor of the breast. Mol Cytogenet. 2020;13(1):49doi: 10.1186/s13039-020-00516-z.
Holzmann, C., Helmke, B., & Bullerdiek, J. (2020). Gross genetic alterations and genetic heterogeneity in a periductal stromal tumor of the breast. Molecular cytogenetics, 13(1), 49. https://doi.org/10.1186/s13039-020-00516-z
Holzmann, Carsten, et al. "Gross genetic alterations and genetic heterogeneity in a periductal stromal tumor of the breast." Molecular cytogenetics vol. 13,1 (2020): 49. doi: https://doi.org/10.1186/s13039-020-00516-z
Holzmann C, Helmke B, Bullerdiek J. Gross genetic alterations and genetic heterogeneity in a periductal stromal tumor of the breast. Mol Cytogenet. 2020 Nov 23;13(1):49. doi: 10.1186/s13039-020-00516-z. PMID: 33292379; PMCID: PMC7686689.
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Quadricuspid aortic valve in a patient with Turner syndrome.

Experimental and clinical cardiology

Mohamed SA, Misfeld M, Hanke T, Belge G, Bullerdiek J, Sievers HH.
PMID: 18650999
Exp Clin Cardiol. 2007;12(3):161-2.

A quadricuspid aortic valve is an uncommon congenital anomaly that is often associated with other cardiac disorders. Most reported cases of quadricuspid aortic valves are detected incidentally during necropsy or aortic valve replacement and, therefore, the potential clinical course...

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Mohamed SA, Misfeld M, Hanke T, et al. Quadricuspid aortic valve in a patient with Turner syndrome. Exp Clin Cardiol. 2007;12(3):161-2
Mohamed, S. A., Misfeld, M., Hanke, T., Belge, G., Bullerdiek, J., & Sievers, H. H. (2007). Quadricuspid aortic valve in a patient with Turner syndrome. Experimental and clinical cardiology, 12(3), 161-2.
Mohamed, Salah A, et al. "Quadricuspid aortic valve in a patient with Turner syndrome." Experimental and clinical cardiology vol. 12,3 (2007): 161-2.
Mohamed SA, Misfeld M, Hanke T, Belge G, Bullerdiek J, Sievers HH. Quadricuspid aortic valve in a patient with Turner syndrome. Exp Clin Cardiol. 2007;12(3):161-2. PMID: 18650999; PMCID: PMC2323752.
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Exosome-delivered microRNAs of "chromosome 19 microRNA cluster" as immunomodulators in pregnancy and tumorigenesis.

Molecular cytogenetics

Bullerdiek J, Flor I.
PMID: 22559272
Mol Cytogenet. 2012 Jul 02;5(1):27. doi: 10.1186/1755-8166-5-27.

BACKGROUND: Structural rearrangements of chromosomal band 19q13 are a non-random cytogenetic abnormality in thyroid adenomas and adenomatous goiters and lead to an expression of miRNAs of the chromosome 19 microRNA cluster C19MC. Normally, expression of these miRNAs is silenced...

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Bullerdiek J, Flor I. Exosome-delivered microRNAs of "chromosome 19 microRNA cluster" as immunomodulators in pregnancy and tumorigenesis. Mol Cytogenet. 2012;5(1):27doi: 10.1186/1755-8166-5-27.
Bullerdiek, J., & Flor, I. (2012). Exosome-delivered microRNAs of "chromosome 19 microRNA cluster" as immunomodulators in pregnancy and tumorigenesis. Molecular cytogenetics, 5(1), 27. https://doi.org/10.1186/1755-8166-5-27
Bullerdiek, Jörn, and Flor, Inga. "Exosome-delivered microRNAs of "chromosome 19 microRNA cluster" as immunomodulators in pregnancy and tumorigenesis." Molecular cytogenetics vol. 5,1 (2012): 27. doi: https://doi.org/10.1186/1755-8166-5-27
Bullerdiek J, Flor I. Exosome-delivered microRNAs of "chromosome 19 microRNA cluster" as immunomodulators in pregnancy and tumorigenesis. Mol Cytogenet. 2012 Jul 02;5(1):27. doi: 10.1186/1755-8166-5-27. PMID: 22559272; PMCID: PMC3388007.
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Locally different endothelial nitric oxide synthase protein levels in ascending aortic aneurysms of bicuspid and tricuspid aortic valve.

Cardiology research and practice

Mohamed SA, Radtke A, Saraei R, Bullerdiek J, Sorani H, Nimzyk R, Karluss A, Sievers HH, Belge G.
PMID: 22745920
Cardiol Res Pract. 2012;2012:165957. doi: 10.1155/2012/165957. Epub 2012 Jun 18.

Aims. Dysregulated expression of the endothelial nitric oxide synthase (eNOS) is observed in aortic aneurysms associated with bicuspid aortic valve (BAV). We determined eNOS protein levels in various areas in ascending aortic aneurysms. Methods and Results. Aneurysmal specimens were...

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Mohamed SA, Radtke A, Saraei R, et al. Locally different endothelial nitric oxide synthase protein levels in ascending aortic aneurysms of bicuspid and tricuspid aortic valve. Cardiol Res Pract. 2012;2012:165957doi: 10.1155/2012/165957.
Mohamed, S. A., Radtke, A., Saraei, R., Bullerdiek, J., Sorani, H., Nimzyk, R., Karluss, A., Sievers, H. H., & Belge, G. (2012). Locally different endothelial nitric oxide synthase protein levels in ascending aortic aneurysms of bicuspid and tricuspid aortic valve. Cardiology research and practice, 2012165957. https://doi.org/10.1155/2012/165957
Mohamed, Salah A, et al. "Locally different endothelial nitric oxide synthase protein levels in ascending aortic aneurysms of bicuspid and tricuspid aortic valve." Cardiology research and practice vol. 2012 (2012): 165957. doi: https://doi.org/10.1155/2012/165957
Mohamed SA, Radtke A, Saraei R, Bullerdiek J, Sorani H, Nimzyk R, Karluss A, Sievers HH, Belge G. Locally different endothelial nitric oxide synthase protein levels in ascending aortic aneurysms of bicuspid and tricuspid aortic valve. Cardiol Res Pract. 2012;2012:165957. doi: 10.1155/2012/165957. Epub 2012 Jun 18. PMID: 22745920; PMCID: PMC3382953.
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Genome-wide acquired uniparental disomy as well as chromosomal gains and losses in an uterine epithelioid leiomyoma.

Molecular cytogenetics

Holzmann C, Markowski DN, Koczan D, Helmke BM, Bullerdiek J.
PMID: 24593849
Mol Cytogenet. 2014 Mar 03;7(1):19. doi: 10.1186/1755-8166-7-19.

BACKGROUND: Epitheloid leiomyoma is a rare subtype of benign smooth muscle tumors.RESULTS: Herein, we present the results of classical cytogenetics, MED12 mutation analysis, and copy number variation array evaluation in one such case. Whereas cytogenetic did not show evidence...

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Holzmann C, Markowski DN, Koczan D, et al. Genome-wide acquired uniparental disomy as well as chromosomal gains and losses in an uterine epithelioid leiomyoma. Mol Cytogenet. 2014;7(1):19doi: 10.1186/1755-8166-7-19.
Holzmann, C., Markowski, D. N., Koczan, D., Helmke, B. M., & Bullerdiek, J. (2014). Genome-wide acquired uniparental disomy as well as chromosomal gains and losses in an uterine epithelioid leiomyoma. Molecular cytogenetics, 7(1), 19. https://doi.org/10.1186/1755-8166-7-19
Holzmann, Carsten, et al. "Genome-wide acquired uniparental disomy as well as chromosomal gains and losses in an uterine epithelioid leiomyoma." Molecular cytogenetics vol. 7,1 (2014): 19. doi: https://doi.org/10.1186/1755-8166-7-19
Holzmann C, Markowski DN, Koczan D, Helmke BM, Bullerdiek J. Genome-wide acquired uniparental disomy as well as chromosomal gains and losses in an uterine epithelioid leiomyoma. Mol Cytogenet. 2014 Mar 03;7(1):19. doi: 10.1186/1755-8166-7-19. PMID: 24593849; PMCID: PMC3996012.
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Showing 1 to 12 of 22 entries