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Author Correction: Metformin reverses established lung fibrosis in a bleomycin model.

Nature medicine

Rangarajan S, Bone NB, Zmijewska AA, Jiang S, Park DW, Bernard K, Locy ML, Ravi S, Deshane J, Mannon RB, Abraham E, Darley-Usmar V, Thannickal VJ, Zmijewski JW.
PMID: 30104770
Nat Med. 2018 Oct;24(10):1627. doi: 10.1038/s41591-018-0170-z.

In the version of this article originally published, a grant was omitted from the Acknowledgements section. The following sentence should have been included: "R.B.M. was supported by a Department of Veterans Affairs Merit Award (5I01BX003272)." The error has been...

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Rangarajan S, Bone NB, Zmijewska AA, et al. Author Correction: Metformin reverses established lung fibrosis in a bleomycin model. Nat Med. 2018;24(10):1627doi: 10.1038/s41591-018-0170-z.
Rangarajan, S., Bone, N. B., Zmijewska, A. A., Jiang, S., Park, D. W., Bernard, K., Locy, M. L., Ravi, S., Deshane, J., Mannon, R. B., Abraham, E., Darley-Usmar, V., Thannickal, V. J., & Zmijewski, J. W. (2018). Author Correction: Metformin reverses established lung fibrosis in a bleomycin model. Nature medicine, 24(10), 1627. https://doi.org/10.1038/s41591-018-0170-z
Rangarajan, Sunad, et al. "Author Correction: Metformin reverses established lung fibrosis in a bleomycin model." Nature medicine vol. 24,10 (2018): 1627. doi: https://doi.org/10.1038/s41591-018-0170-z
Rangarajan S, Bone NB, Zmijewska AA, Jiang S, Park DW, Bernard K, Locy ML, Ravi S, Deshane J, Mannon RB, Abraham E, Darley-Usmar V, Thannickal VJ, Zmijewski JW. Author Correction: Metformin reverses established lung fibrosis in a bleomycin model. Nat Med. 2018 Oct;24(10):1627. doi: 10.1038/s41591-018-0170-z. PMID: 30104770.
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Metabolic alterations mediated by STAT3 promotes drug persistence in CML.

Leukemia

Patel SB, Nemkov T, Stefanoni D, Benavides GA, Bassal MA, Crown BL, Matkins VR, Camacho V, Kuznetsova V, Hoang AT, Tenen DE, Wolock SL, Park J, Ying L, Yue Z, Chen JY, Yang H, Tenen DG, Ferrell PB, Lu R, Darley-Usmar V, D'Alessandro A, Bhatia R, Welner RS.
PMID: 34120146
Leukemia. 2021 Dec;35(12):3371-3382. doi: 10.1038/s41375-021-01315-0. Epub 2021 Jun 12.

Leukemic stem cells (LSCs) can acquire non-mutational resistance following drug treatment leading to therapeutic failure and relapse. However, oncogene-independent mechanisms of drug persistence in LSCs are incompletely understood, which is the primary focus of this study. We integrated proteomics,...

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Patel SB, Nemkov T, Stefanoni D, et al. Metabolic alterations mediated by STAT3 promotes drug persistence in CML. Leukemia. 2021;35(12):3371-3382doi: 10.1038/s41375-021-01315-0.
Patel, S. B., Nemkov, T., Stefanoni, D., Benavides, G. A., Bassal, M. A., Crown, B. L., Matkins, V. R., Camacho, V., Kuznetsova, V., Hoang, A. T., Tenen, D. E., Wolock, S. L., Park, J., Ying, L., Yue, Z., Chen, J. Y., Yang, H., Tenen, D. G., Ferrell, P. B., Lu, R., Darley-Usmar, V., D'Alessandro, A., Bhatia, R., & Welner, R. S. (2021). Metabolic alterations mediated by STAT3 promotes drug persistence in CML. Leukemia, 35(12), 3371-3382. https://doi.org/10.1038/s41375-021-01315-0
Patel, Sweta B, et al. "Metabolic alterations mediated by STAT3 promotes drug persistence in CML." Leukemia vol. 35,12 (2021): 3371-3382. doi: https://doi.org/10.1038/s41375-021-01315-0
Patel SB, Nemkov T, Stefanoni D, Benavides GA, Bassal MA, Crown BL, Matkins VR, Camacho V, Kuznetsova V, Hoang AT, Tenen DE, Wolock SL, Park J, Ying L, Yue Z, Chen JY, Yang H, Tenen DG, Ferrell PB, Lu R, Darley-Usmar V, D'Alessandro A, Bhatia R, Welner RS. Metabolic alterations mediated by STAT3 promotes drug persistence in CML. Leukemia. 2021 Dec;35(12):3371-3382. doi: 10.1038/s41375-021-01315-0. Epub 2021 Jun 12. PMID: 34120146; PMCID: PMC8632690.
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Fasting drives the metabolic, molecular and geroprotective effects of a calorie-restricted diet in mice.

Nature metabolism

Pak HH, Haws SA, Green CL, Koller M, Lavarias MT, Richardson NE, Yang SE, Dumas SN, Sonsalla M, Bray L, Johnson M, Barnes S, Darley-Usmar V, Zhang J, Yen CE, Denu JM, Lamming DW.
PMID: 34663973
Nat Metab. 2021 Oct;3(10):1327-1341. doi: 10.1038/s42255-021-00466-9. Epub 2021 Oct 18.

Calorie restriction (CR) promotes healthy ageing in diverse species. Recently, it has been shown that fasting for a portion of each day has metabolic benefits and promotes lifespan. These findings complicate the interpretation of rodent CR studies, in which...

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Pak HH, Haws SA, Green CL, et al. Fasting drives the metabolic, molecular and geroprotective effects of a calorie-restricted diet in mice. Nat Metab. 2021;3(10):1327-1341doi: 10.1038/s42255-021-00466-9.
Pak, H. H., Haws, S. A., Green, C. L., Koller, M., Lavarias, M. T., Richardson, N. E., Yang, S. E., Dumas, S. N., Sonsalla, M., Bray, L., Johnson, M., Barnes, S., Darley-Usmar, V., Zhang, J., Yen, C. E., Denu, J. M., & Lamming, D. W. (2021). Fasting drives the metabolic, molecular and geroprotective effects of a calorie-restricted diet in mice. Nature metabolism, 3(10), 1327-1341. https://doi.org/10.1038/s42255-021-00466-9
Pak, Heidi H, et al. "Fasting drives the metabolic, molecular and geroprotective effects of a calorie-restricted diet in mice." Nature metabolism vol. 3,10 (2021): 1327-1341. doi: https://doi.org/10.1038/s42255-021-00466-9
Pak HH, Haws SA, Green CL, Koller M, Lavarias MT, Richardson NE, Yang SE, Dumas SN, Sonsalla M, Bray L, Johnson M, Barnes S, Darley-Usmar V, Zhang J, Yen CE, Denu JM, Lamming DW. Fasting drives the metabolic, molecular and geroprotective effects of a calorie-restricted diet in mice. Nat Metab. 2021 Oct;3(10):1327-1341. doi: 10.1038/s42255-021-00466-9. Epub 2021 Oct 18. PMID: 34663973; PMCID: PMC8544824.
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Mitochondrially targeted compounds and their impact on cellular bioenergetics.

Redox biology

Reily C, Mitchell T, Chacko BK, Benavides G, Murphy MP, Darley-Usmar V.
PMID: 23667828
Redox Biol. 2013;1(1):86-93. doi: 10.1016/j.redox.2012.11.009.

Mitochondria are recognized as critical sites of localized injury in a number of chronic pathologies which has led to the development of organelle directed therapeutics. One of the approaches employed to target molecules to the mitochondrion is to conjugate...

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Reily C, Mitchell T, Chacko BK, et al. Mitochondrially targeted compounds and their impact on cellular bioenergetics. Redox Biol. 2013;1(1):86-93doi: 10.1016/j.redox.2012.11.009.
Reily, C., Mitchell, T., Chacko, B. K., Benavides, G., Murphy, M. P., & Darley-Usmar, V. (2013). Mitochondrially targeted compounds and their impact on cellular bioenergetics. Redox biology, 1(1), 86-93. https://doi.org/10.1016/j.redox.2012.11.009
Reily, Colin, et al. "Mitochondrially targeted compounds and their impact on cellular bioenergetics." Redox biology vol. 1,1 (2013): 86-93. doi: https://doi.org/10.1016/j.redox.2012.11.009
Reily C, Mitchell T, Chacko BK, Benavides G, Murphy MP, Darley-Usmar V. Mitochondrially targeted compounds and their impact on cellular bioenergetics. Redox Biol. 2013;1(1):86-93. doi: 10.1016/j.redox.2012.11.009. PMID: 23667828; PMCID: PMC3647698.
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The Identification of a Novel Calcium-Dependent Link Between NAD.

Frontiers in molecular biosciences

Zou L, Collins HE, Young ME, Zhang J, Wende AR, Darley-Usmar VM, Chatham JC.
PMID: 34950703
Front Mol Biosci. 2021 Dec 07;8:780865. doi: 10.3389/fmolb.2021.780865. eCollection 2021.

The modification of proteins by O-linked β-

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Zou L, Collins HE, Young ME, et al. The Identification of a Novel Calcium-Dependent Link Between NAD. Front Mol Biosci. 2021;8:780865doi: 10.3389/fmolb.2021.780865.
Zou, L., Collins, H. E., Young, M. E., Zhang, J., Wende, A. R., Darley-Usmar, V. M., & Chatham, J. C. (2021). The Identification of a Novel Calcium-Dependent Link Between NAD. Frontiers in molecular biosciences, 8780865. https://doi.org/10.3389/fmolb.2021.780865
Zou, Luyun, et al. "The Identification of a Novel Calcium-Dependent Link Between NAD." Frontiers in molecular biosciences vol. 8 (2021): 780865. doi: https://doi.org/10.3389/fmolb.2021.780865
Zou L, Collins HE, Young ME, Zhang J, Wende AR, Darley-Usmar VM, Chatham JC. The Identification of a Novel Calcium-Dependent Link Between NAD. Front Mol Biosci. 2021 Dec 07;8:780865. doi: 10.3389/fmolb.2021.780865. eCollection 2021. PMID: 34950703; PMCID: PMC8691773.
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ZKSCAN3 in severe bacterial lung infection and sepsis-induced immunosuppression.

Laboratory investigation; a journal of technical methods and pathology

Ouyang X, Becker E, Bone NB, Johnson MS, Craver J, Zong WX, Darley-Usmar VM, Zmijewski JW, Zhang J.
PMID: 34504306
Lab Invest. 2021 Nov;101(11):1467-1474. doi: 10.1038/s41374-021-00660-z. Epub 2021 Sep 09.

The mortality rates among patients who initially survive sepsis are, in part, associated with a high risk of secondary lung infections and respiratory failure. Given that phagolysosomes are important for intracellular killing of pathogenic microbes, we investigated how severe...

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Ouyang X, Becker E, Bone NB, et al. ZKSCAN3 in severe bacterial lung infection and sepsis-induced immunosuppression. Lab Invest. 2021;101(11):1467-1474doi: 10.1038/s41374-021-00660-z.
Ouyang, X., Becker, E., Bone, N. B., Johnson, M. S., Craver, J., Zong, W. X., Darley-Usmar, V. M., Zmijewski, J. W., & Zhang, J. (2021). ZKSCAN3 in severe bacterial lung infection and sepsis-induced immunosuppression. Laboratory investigation; a journal of technical methods and pathology, 101(11), 1467-1474. https://doi.org/10.1038/s41374-021-00660-z
Ouyang, Xiaosen, et al. "ZKSCAN3 in severe bacterial lung infection and sepsis-induced immunosuppression." Laboratory investigation; a journal of technical methods and pathology vol. 101,11 (2021): 1467-1474. doi: https://doi.org/10.1038/s41374-021-00660-z
Ouyang X, Becker E, Bone NB, Johnson MS, Craver J, Zong WX, Darley-Usmar VM, Zmijewski JW, Zhang J. ZKSCAN3 in severe bacterial lung infection and sepsis-induced immunosuppression. Lab Invest. 2021 Nov;101(11):1467-1474. doi: 10.1038/s41374-021-00660-z. Epub 2021 Sep 09. PMID: 34504306.
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Hedgehog Signaling Regulates Metabolism and Polarization of Mammary Tumor-Associated Macrophages.

Cancer research

Hinshaw DC, Hanna A, Lama-Sherpa T, Metge B, Kammerud SC, Benavides GA, Kumar A, Alsheikh HA, Mota M, Chen D, Ballinger SW, Rathmell JC, Ponnazhagan S, Darley-Usmar V, Samant RS, Shevde LA.
PMID: 34289986
Cancer Res. 2021 Nov 01;81(21):5425-5437. doi: 10.1158/0008-5472.CAN-20-1723. Epub 2021 Jul 21.

Elevated infiltration of immunosuppressive alternatively polarized (M2) macrophages is associated with poor prognosis in patients with cancer. The tumor microenvironment remarkably orchestrates molecular mechanisms that program these macrophages. Here we identify a novel role for oncogenic Hedgehog (Hh) signaling...

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Hinshaw DC, Hanna A, Lama-Sherpa T, et al. Hedgehog Signaling Regulates Metabolism and Polarization of Mammary Tumor-Associated Macrophages. Cancer Res. 2021;81(21):5425-5437doi: 10.1158/0008-5472.CAN-20-1723.
Hinshaw, D. C., Hanna, A., Lama-Sherpa, T., Metge, B., Kammerud, S. C., Benavides, G. A., Kumar, A., Alsheikh, H. A., Mota, M., Chen, D., Ballinger, S. W., Rathmell, J. C., Ponnazhagan, S., Darley-Usmar, V., Samant, R. S., & Shevde, L. A. (2021). Hedgehog Signaling Regulates Metabolism and Polarization of Mammary Tumor-Associated Macrophages. Cancer research, 81(21), 5425-5437. https://doi.org/10.1158/0008-5472.CAN-20-1723
Hinshaw, Dominique C, et al. "Hedgehog Signaling Regulates Metabolism and Polarization of Mammary Tumor-Associated Macrophages." Cancer research vol. 81,21 (2021): 5425-5437. doi: https://doi.org/10.1158/0008-5472.CAN-20-1723
Hinshaw DC, Hanna A, Lama-Sherpa T, Metge B, Kammerud SC, Benavides GA, Kumar A, Alsheikh HA, Mota M, Chen D, Ballinger SW, Rathmell JC, Ponnazhagan S, Darley-Usmar V, Samant RS, Shevde LA. Hedgehog Signaling Regulates Metabolism and Polarization of Mammary Tumor-Associated Macrophages. Cancer Res. 2021 Nov 01;81(21):5425-5437. doi: 10.1158/0008-5472.CAN-20-1723. Epub 2021 Jul 21. PMID: 34289986; PMCID: PMC8563376.
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A role for GLUT3 in glioblastoma cell invasion that is not recapitulated by GLUT1.

Cell adhesion & migration

Libby CJ, Gc S, Benavides GA, Fisher JL, Williford SE, Zhang S, Tran AN, Gordon ER, Jones AB, Tuy K, Flavahan W, Gordillo J, Long A, Cooper SJ, Lasseigne BN, Augelli-Szafran CE, Darley-Usmar V, Hjelmeland AB.
PMID: 33843470
Cell Adh Migr. 2021 Dec;15(1):101-115. doi: 10.1080/19336918.2021.1903684.

The multifaceted roles of metabolism in invasion have been investigated across many cancers. The brain tumor glioblastoma (GBM) is a highly invasive and metabolically plastic tumor with an inevitable recurrence. The neuronal glucose transporter 3 (GLUT3) was previously reported...

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Libby CJ, Gc S, Benavides GA, et al. A role for GLUT3 in glioblastoma cell invasion that is not recapitulated by GLUT1. Cell Adh Migr. 2021;15(1):101-115doi: 10.1080/19336918.2021.1903684.
Libby, C. J., Gc, S., Benavides, G. A., Fisher, J. L., Williford, S. E., Zhang, S., Tran, A. N., Gordon, E. R., Jones, A. B., Tuy, K., Flavahan, W., Gordillo, J., Long, A., Cooper, S. J., Lasseigne, B. N., Augelli-Szafran, C. E., Darley-Usmar, V., & Hjelmeland, A. B. (2021). A role for GLUT3 in glioblastoma cell invasion that is not recapitulated by GLUT1. Cell adhesion & migration, 15(1), 101-115. https://doi.org/10.1080/19336918.2021.1903684
Libby, Catherine J, et al. "A role for GLUT3 in glioblastoma cell invasion that is not recapitulated by GLUT1." Cell adhesion & migration vol. 15,1 (2021): 101-115. doi: https://doi.org/10.1080/19336918.2021.1903684
Libby CJ, Gc S, Benavides GA, Fisher JL, Williford SE, Zhang S, Tran AN, Gordon ER, Jones AB, Tuy K, Flavahan W, Gordillo J, Long A, Cooper SJ, Lasseigne BN, Augelli-Szafran CE, Darley-Usmar V, Hjelmeland AB. A role for GLUT3 in glioblastoma cell invasion that is not recapitulated by GLUT1. Cell Adh Migr. 2021 Dec;15(1):101-115. doi: 10.1080/19336918.2021.1903684. PMID: 33843470; PMCID: PMC8043167.
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The Role of Autophagy, Mitophagy and Lysosomal Functions in Modulating Bioenergetics and Survival in the Context of Redox and Proteotoxic Damage: Implications for Neurodegenerative Diseases.

Aging and disease

Redmann M, Darley-Usmar V, Zhang J.
PMID: 27114848
Aging Dis. 2016 Mar 15;7(2):150-62. doi: 10.14336/AD.2015.0820. eCollection 2016 Mar.

Redox and proteotoxic stress contributes to age-dependent accumulation of dysfunctional mitochondria and protein aggregates, and is associated with neurodegeneration. The free radical theory of aging inspired many studies using reactive species scavengers such as alpha-tocopherol, ascorbate and coenzyme Q...

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Redmann M, Darley-Usmar V, Zhang J. The Role of Autophagy, Mitophagy and Lysosomal Functions in Modulating Bioenergetics and Survival in the Context of Redox and Proteotoxic Damage: Implications for Neurodegenerative Diseases. Aging Dis. 2016;7(2):150-62doi: 10.14336/AD.2015.0820.
Redmann, M., Darley-Usmar, V., & Zhang, J. (2016). The Role of Autophagy, Mitophagy and Lysosomal Functions in Modulating Bioenergetics and Survival in the Context of Redox and Proteotoxic Damage: Implications for Neurodegenerative Diseases. Aging and disease, 7(2), 150-62. https://doi.org/10.14336/AD.2015.0820
Redmann, Matthew, et al. "The Role of Autophagy, Mitophagy and Lysosomal Functions in Modulating Bioenergetics and Survival in the Context of Redox and Proteotoxic Damage: Implications for Neurodegenerative Diseases." Aging and disease vol. 7,2 (2016): 150-62. doi: https://doi.org/10.14336/AD.2015.0820
Redmann M, Darley-Usmar V, Zhang J. The Role of Autophagy, Mitophagy and Lysosomal Functions in Modulating Bioenergetics and Survival in the Context of Redox and Proteotoxic Damage: Implications for Neurodegenerative Diseases. Aging Dis. 2016 Mar 15;7(2):150-62. doi: 10.14336/AD.2015.0820. eCollection 2016 Mar. PMID: 27114848; PMCID: PMC4809607.
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Antioxidant properties of phytoestrogens.

Journal of medicinal food

Patel RP, Crawford J, Boersma B, Barnes S, Darley-Usmar VM.
PMID: 19281370
J Med Food. 1999;2(3):163-6. doi: 10.1089/jmf.1999.2.163.

The oxidation of lipids is an autocatalytic process consisting of a number of well-defined interrelated chemical reactions. Its importance has long been recognized in the food and polymer industry, and recent advances in the understanding of vascular diseases have...

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Patel RP, Crawford J, Boersma B, et al. Antioxidant properties of phytoestrogens. J Med Food. 1999;2(3):163-6doi: 10.1089/jmf.1999.2.163.
Patel, R. P., Crawford, J., Boersma, B., Barnes, S., & Darley-Usmar, V. M. (1999). Antioxidant properties of phytoestrogens. Journal of medicinal food, 2(3), 163-6. https://doi.org/10.1089/jmf.1999.2.163
Patel, R P, et al. "Antioxidant properties of phytoestrogens." Journal of medicinal food vol. 2,3 (1999): 163-6. doi: https://doi.org/10.1089/jmf.1999.2.163
Patel RP, Crawford J, Boersma B, Barnes S, Darley-Usmar VM. Antioxidant properties of phytoestrogens. J Med Food. 1999;2(3):163-6. doi: 10.1089/jmf.1999.2.163. PMID: 19281370.
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Gender and cardiovascular disease recent insights.

Trends in cardiovascular medicine

White CR, Darley-Usmar V, Oparil S.
PMID: 21235871
Trends Cardiovasc Med. 1997 Apr;7(3):94-100. doi: 10.1016/S1050-1738(97)00009-1.

Cardiovascular disease is rare in premenopausal women compared with men in similar age groups. After menopause, however, the gender difference in cardiovascular disease diminishes, and there is an increased incidence of coronary risk and events in women. Although a...

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White CR, Darley-Usmar V, Oparil S. Gender and cardiovascular disease recent insights. Trends Cardiovasc Med. 1997;7(3):94-100doi: 10.1016/S1050-1738(97)00009-1.
White, C. R., Darley-Usmar, V., & Oparil, S. (1997). Gender and cardiovascular disease recent insights. Trends in cardiovascular medicine, 7(3), 94-100. https://doi.org/10.1016/S1050-1738(97)00009-1
White, C R, et al. "Gender and cardiovascular disease recent insights." Trends in cardiovascular medicine vol. 7,3 (1997): 94-100. doi: https://doi.org/10.1016/S1050-1738(97)00009-1
White CR, Darley-Usmar V, Oparil S. Gender and cardiovascular disease recent insights. Trends Cardiovasc Med. 1997 Apr;7(3):94-100. doi: 10.1016/S1050-1738(97)00009-1. PMID: 21235871.
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Aging and energetics' 'Top 40' future research opportunities 2010-2013.

F1000Research

Allison DB, Antoine LH, Ballinger SW, Bamman MM, Biga P, Darley-Usmar VM, Fisher G, Gohlke JM, Halade GV, Hartman JL, Hunter GR, Messina JL, Nagy TR, Plaisance EP, Powell ML, Roth KA, Sandel MW, Schwartz TS, Smith DL, Sweatt JD, Tollefsbol TO, Watts SA, Yang Y, Zhang J, Austad SN.
PMID: 25324965
F1000Res. 2014 Sep 12;3:219. doi: 10.12688/f1000research.5212.1. eCollection 2014.

BACKGROUND: As part of a coordinated effort to expand our research activity at the interface of Aging and Energetics a team of investigators at The University of Alabama at Birmingham systematically assayed and catalogued the top research priorities identified...

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Allison DB, Antoine LH, Ballinger SW, et al. Aging and energetics' 'Top 40' future research opportunities 2010-2013. F1000Res. 2014;3:219doi: 10.12688/f1000research.5212.1.
Allison, D. B., Antoine, L. H., Ballinger, S. W., Bamman, M. M., Biga, P., Darley-Usmar, V. M., Fisher, G., Gohlke, J. M., Halade, G. V., Hartman, J. L., Hunter, G. R., Messina, J. L., Nagy, T. R., Plaisance, E. P., Powell, M. L., Roth, K. A., Sandel, M. W., Schwartz, T. S., Smith, D. L., Sweatt, J. D., Tollefsbol, T. O., Watts, S. A., Yang, Y., Zhang, J., & Austad, S. N. (2014). Aging and energetics' 'Top 40' future research opportunities 2010-2013. F1000Research, 3219. https://doi.org/10.12688/f1000research.5212.1
Allison, David B, et al. "Aging and energetics' 'Top 40' future research opportunities 2010-2013." F1000Research vol. 3 (2014): 219. doi: https://doi.org/10.12688/f1000research.5212.1
Allison DB, Antoine LH, Ballinger SW, Bamman MM, Biga P, Darley-Usmar VM, Fisher G, Gohlke JM, Halade GV, Hartman JL, Hunter GR, Messina JL, Nagy TR, Plaisance EP, Powell ML, Roth KA, Sandel MW, Schwartz TS, Smith DL, Sweatt JD, Tollefsbol TO, Watts SA, Yang Y, Zhang J, Austad SN. Aging and energetics' 'Top 40' future research opportunities 2010-2013. F1000Res. 2014 Sep 12;3:219. doi: 10.12688/f1000research.5212.1. eCollection 2014. PMID: 25324965; PMCID: PMC4197746.
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