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Showing 1 to 12 of 48 entries
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Peripheral arterial occlusive disease during ponatinib therapy after failure of imatinib: a case report.

Journal of clinical pharmacy and therapeutics

Tournaire G, Despas F, Huguet F, Montastruc JL, Bondon-Guitton E.
PMID: 27009771
J Clin Pharm Ther. 2016 Jun;41(3):360-361. doi: 10.1111/jcpt.12383. Epub 2016 Mar 23.

WHAT IS KNOWN AND OBJECTIVE: Peripheral vascular adverse events have been reported with ponatinib treatment in chronic myeloid leukaemia (CML) after failure of dasatinib or nilotinib. We here report peripheral arterial occlusive disease (PAOD) in a patient who had...

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Tournaire G, Despas F, Huguet F, et al. Peripheral arterial occlusive disease during ponatinib therapy after failure of imatinib: a case report. J Clin Pharm Ther. 2016;41(3):360-361doi: 10.1111/jcpt.12383.
Tournaire, G., Despas, F., Huguet, F., Montastruc, J. L., & Bondon-Guitton, E. (2016). Peripheral arterial occlusive disease during ponatinib therapy after failure of imatinib: a case report. Journal of clinical pharmacy and therapeutics, 41(3), 360-361. https://doi.org/10.1111/jcpt.12383
Tournaire, G, et al. "Peripheral arterial occlusive disease during ponatinib therapy after failure of imatinib: a case report." Journal of clinical pharmacy and therapeutics vol. 41,3 (2016): 360-361. doi: https://doi.org/10.1111/jcpt.12383
Tournaire G, Despas F, Huguet F, Montastruc JL, Bondon-Guitton E. Peripheral arterial occlusive disease during ponatinib therapy after failure of imatinib: a case report. J Clin Pharm Ther. 2016 Jun;41(3):360-361. doi: 10.1111/jcpt.12383. Epub 2016 Mar 23. PMID: 27009771.
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How much can an intermediate state influence competing reactive pathways?.

The Journal of chemical physics

Despa F, Berry RS.
PMID: 15267386
J Chem Phys. 2004 Mar 15;120(11):5164-8. doi: 10.1063/1.1647058.

A molecule undergoing reaction may form a short-lived intermediate. Under certain conditions, the rate at which the reaction proceeds toward the product state via the intermediate may exceed that of a simple, direct path. The competition of two alternative...

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Despa F, Berry RS. How much can an intermediate state influence competing reactive pathways?. J Chem Phys. 2004;120(11):5164-8doi: 10.1063/1.1647058.
Despa, F., & Berry, R. S. (2004). How much can an intermediate state influence competing reactive pathways?. The Journal of chemical physics, 120(11), 5164-8. https://doi.org/10.1063/1.1647058
Despa, Florin, and Berry, R Stephen. "How much can an intermediate state influence competing reactive pathways?." The Journal of chemical physics vol. 120,11 (2004): 5164-8. doi: https://doi.org/10.1063/1.1647058
Despa F, Berry RS. How much can an intermediate state influence competing reactive pathways?. J Chem Phys. 2004 Mar 15;120(11):5164-8. doi: 10.1063/1.1647058. PMID: 15267386.
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[Competency-based nursing training].

Soins; la revue de reference infirmiere

Arfe E, Despiau F, Huot M, Bombail M.
PMID: 23115920
Soins. 2012 Sep;(768):19-21.

The application of the new nursing training reference framework requires the active participation of professionals in the field, notably with assessing students' skills during a work placement. The skills approach represents a paradigm shift for caregivers, one which requires...

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Arfe E, Despiau F, Huot M, et al. L'approche par compétences dans la formation des infirmiers. [Competency-based nursing training]. Soins. 2012;(768):19-21
Arfe, E., Despiau, F., Huot, M., & Bombail, M. (2012). L'approche par compétences dans la formation des infirmiers. [Competency-based nursing training]. Soins; la revue de reference infirmiere, (768), 19-21.
Arfe, Emmanuelle, et al. "L'approche par compétences dans la formation des infirmiers." [Competency-based nursing training]. Soins; la revue de reference infirmiere vol. ,768 (2012): 19-21.
Arfe E, Despiau F, Huot M, Bombail M. L'approche par compétences dans la formation des infirmiers. [Competency-based nursing training]. Soins. 2012 Sep;(768):19-21. French. PMID: 23115920.
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Direct evidence of sympathetic hyperactivity in patients with vasospastic angina.

American journal of cardiovascular disease

Boudou N, Despas F, Rothem JV, Lairez O, Elbaz M, Vaccaro A, Lebrin M, Pathak A, Carrié D.
PMID: 28695054
Am J Cardiovasc Dis. 2017 Jun 15;7(3):83-88. eCollection 2017.

BACKGROUND: The autonomic nervous system is reported to be involved in the pathogenesis of vasospastic angina (VSA). Studies based on heart rate variability analysis have shown conflicting results with both a reduction and an enhancement of sympathetic nervous system...

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Boudou N, Despas F, Rothem JV, et al. Direct evidence of sympathetic hyperactivity in patients with vasospastic angina. Am J Cardiovasc Dis. 2017;7(3):83-88
Boudou, N., Despas, F., Rothem, J. V., Lairez, O., Elbaz, M., Vaccaro, A., Lebrin, M., Pathak, A., & Carrié, D. (2017). Direct evidence of sympathetic hyperactivity in patients with vasospastic angina. American journal of cardiovascular disease, 7(3), 83-88.
Boudou, Nicolas, et al. "Direct evidence of sympathetic hyperactivity in patients with vasospastic angina." American journal of cardiovascular disease vol. 7,3 (2017): 83-88.
Boudou N, Despas F, Rothem JV, Lairez O, Elbaz M, Vaccaro A, Lebrin M, Pathak A, Carrié D. Direct evidence of sympathetic hyperactivity in patients with vasospastic angina. Am J Cardiovasc Dis. 2017 Jun 15;7(3):83-88. eCollection 2017. PMID: 28695054; PMCID: PMC5498819.
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Silver nanoparticle embedded copper oxide as an efficient core-shell for the catalytic reduction of 4-nitrophenol and antibacterial activity improvement.

Dalton transactions (Cambridge, England : 2003)

Bouazizi N, Vieillard J, Thebault P, Desriac F, Clamens T, Bargougui R, Couvrat N, Thoumire O, Brun N, Ladam G, Morin S, Mofaddel N, Lesouhaitier O, Azzouz A, Le Derf F.
PMID: 29946586
Dalton Trans. 2018 Jul 10;47(27):9143-9155. doi: 10.1039/c8dt02154f.

A facile and eco-friendly method was developed to prepare a microporous CuO@Ag0 core-shell with high catalytic and antibacterial activities. Scanning and transmission electron microscopy revealed a preponderance of nearly spherical 50 nm particles with slight structure compaction. Comparison of...

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Bouazizi N, Vieillard J, Thebault P, et al. Silver nanoparticle embedded copper oxide as an efficient core-shell for the catalytic reduction of 4-nitrophenol and antibacterial activity improvement. Dalton Trans. 2018;47(27):9143-9155doi: 10.1039/c8dt02154f.
Bouazizi, N., Vieillard, J., Thebault, P., Desriac, F., Clamens, T., Bargougui, R., Couvrat, N., Thoumire, O., Brun, N., Ladam, G., Morin, S., Mofaddel, N., Lesouhaitier, O., Azzouz, A., & Le Derf, F. (2018). Silver nanoparticle embedded copper oxide as an efficient core-shell for the catalytic reduction of 4-nitrophenol and antibacterial activity improvement. Dalton transactions (Cambridge, England : 2003), 47(27), 9143-9155. https://doi.org/10.1039/c8dt02154f
Bouazizi, N, et al. "Silver nanoparticle embedded copper oxide as an efficient core-shell for the catalytic reduction of 4-nitrophenol and antibacterial activity improvement." Dalton transactions (Cambridge, England : 2003) vol. 47,27 (2018): 9143-9155. doi: https://doi.org/10.1039/c8dt02154f
Bouazizi N, Vieillard J, Thebault P, Desriac F, Clamens T, Bargougui R, Couvrat N, Thoumire O, Brun N, Ladam G, Morin S, Mofaddel N, Lesouhaitier O, Azzouz A, Le Derf F. Silver nanoparticle embedded copper oxide as an efficient core-shell for the catalytic reduction of 4-nitrophenol and antibacterial activity improvement. Dalton Trans. 2018 Jul 10;47(27):9143-9155. doi: 10.1039/c8dt02154f. PMID: 29946586.
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Contributing Factors to Diabetic Brain Injury and Cognitive Decline.

Diabetes & metabolism journal

Verma N, Despa F.
PMID: 31694078
Diabetes Metab J. 2019 Oct;43(5):560-567. doi: 10.4093/dmj.2019.0153.

The link of diabetes with co-occurring disorders in the brain involves complex and multifactorial pathways. Genetically engineered rodents that express familial Alzheimer's disease-associated mutant forms of amyloid precursor protein and presenilin 1 (

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Verma N, Despa F. Contributing Factors to Diabetic Brain Injury and Cognitive Decline. Diabetes Metab J. 2019;43(5):560-567doi: 10.4093/dmj.2019.0153.
Verma, N., & Despa, F. (2019). Contributing Factors to Diabetic Brain Injury and Cognitive Decline. Diabetes & metabolism journal, 43(5), 560-567. https://doi.org/10.4093/dmj.2019.0153
Verma, Nirmal, and Despa, Florin. "Contributing Factors to Diabetic Brain Injury and Cognitive Decline." Diabetes & metabolism journal vol. 43,5 (2019): 560-567. doi: https://doi.org/10.4093/dmj.2019.0153
Verma N, Despa F. Contributing Factors to Diabetic Brain Injury and Cognitive Decline. Diabetes Metab J. 2019 Oct;43(5):560-567. doi: 10.4093/dmj.2019.0153. PMID: 31694078; PMCID: PMC6834839.
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The association of circulating amylin with β-amyloid in familial Alzheimer's disease.

Alzheimer's & dementia (New York, N. Y.)

Ly H, Verma N, Sharma S, Kotiya D, Despa S, Abner EL, Nelson PT, Jicha GA, Wilcock DM, Goldstein LB, Guerreiro R, Brás J, Hanson AJ, Craft S, Murray AJ, Biessels GJ, Troakes C, Zetterberg H, Hardy J, Lashley T, Aesg, Despa F.
PMID: 33521236
Alzheimers Dement (N Y). 2021 Jan 20;7(1):e12130. doi: 10.1002/trc2.12130. eCollection 2021.

INTRODUCTION: This study assessed the hypothesis that circulating human amylin (amyloid-forming) cross-seeds with amyloid beta (Aβ) in early Alzheimer's disease (AD).METHODS: Evidence of amylin-AD pathology interaction was tested in brains of 31 familial AD mutation carriers and 20 cognitively...

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Ly H, Verma N, Sharma S, et al. The association of circulating amylin with β-amyloid in familial Alzheimer's disease. Alzheimers Dement (N Y). 2021;7(1):e12130doi: 10.1002/trc2.12130.
Ly, H., Verma, N., Sharma, S., Kotiya, D., Despa, S., Abner, E. L., Nelson, P. T., Jicha, G. A., Wilcock, D. M., Goldstein, L. B., Guerreiro, R., Brás, J., Hanson, A. J., Craft, S., Murray, A. J., Biessels, G. J., Troakes, C., Zetterberg, H., Hardy, J., Lashley, T., Aesg, & Despa, F. (2021). The association of circulating amylin with β-amyloid in familial Alzheimer's disease. Alzheimer's & dementia (New York, N. Y.), 7(1), e12130. https://doi.org/10.1002/trc2.12130
Ly, Han, et al. "The association of circulating amylin with β-amyloid in familial Alzheimer's disease." Alzheimer's & dementia (New York, N. Y.) vol. 7,1 (2021): e12130. doi: https://doi.org/10.1002/trc2.12130
Ly H, Verma N, Sharma S, Kotiya D, Despa S, Abner EL, Nelson PT, Jicha GA, Wilcock DM, Goldstein LB, Guerreiro R, Brás J, Hanson AJ, Craft S, Murray AJ, Biessels GJ, Troakes C, Zetterberg H, Hardy J, Lashley T, Aesg, Despa F. The association of circulating amylin with β-amyloid in familial Alzheimer's disease. Alzheimers Dement (N Y). 2021 Jan 20;7(1):e12130. doi: 10.1002/trc2.12130. eCollection 2021. PMID: 33521236; PMCID: PMC7816817.
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Diabetes-related Amylin Dyshomeostasis: a Contributing Factor to Cerebrovascular Pathology and Dementia.

Journal of lipid and atherosclerosis

Ly H, Despa F.
PMID: 32821704
J Lipid Atheroscler. 2019 Sep;8(2):144-151. doi: 10.12997/jla.2019.8.2.144. Epub 2019 Jul 09.

Type 2 diabetes (T2D) increases the risk for cerebrovascular disease (CVD) and dementia. The underlying molecular mechanisms remain elusive, which hampers the development of treatment or/and effective prevention strategies. Recent studies suggest that dyshomeostasis of amylin, a satiety hormone...

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Ly H, Despa F. Diabetes-related Amylin Dyshomeostasis: a Contributing Factor to Cerebrovascular Pathology and Dementia. J Lipid Atheroscler. 2019;8(2):144-151doi: 10.12997/jla.2019.8.2.144.
Ly, H., & Despa, F. (2019). Diabetes-related Amylin Dyshomeostasis: a Contributing Factor to Cerebrovascular Pathology and Dementia. Journal of lipid and atherosclerosis, 8(2), 144-151. https://doi.org/10.12997/jla.2019.8.2.144
Ly, Han, and Despa, Florin. "Diabetes-related Amylin Dyshomeostasis: a Contributing Factor to Cerebrovascular Pathology and Dementia." Journal of lipid and atherosclerosis vol. 8,2 (2019): 144-151. doi: https://doi.org/10.12997/jla.2019.8.2.144
Ly H, Despa F. Diabetes-related Amylin Dyshomeostasis: a Contributing Factor to Cerebrovascular Pathology and Dementia. J Lipid Atheroscler. 2019 Sep;8(2):144-151. doi: 10.12997/jla.2019.8.2.144. Epub 2019 Jul 09. PMID: 32821704; PMCID: PMC7379112.
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Anti-Biofilm Activity of a Low Weight Proteinaceous Molecule from the Marine Bacterium .

Microorganisms

Doghri I, Portier E, Desriac F, Zhao JM, Bazire A, Dufour A, Rochette V, Sablé S, Lanneluc I.
PMID: 32854286
Microorganisms. 2020 Aug 25;8(9). doi: 10.3390/microorganisms8091295.

No abstract available.

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Doghri I, Portier E, Desriac F, et al. Anti-Biofilm Activity of a Low Weight Proteinaceous Molecule from the Marine Bacterium . Microorganisms. 2020;8(9)doi: 10.3390/microorganisms8091295.
Doghri, I., Portier, E., Desriac, F., Zhao, J. M., Bazire, A., Dufour, A., Rochette, V., Sablé, S., & Lanneluc, I. (2020). Anti-Biofilm Activity of a Low Weight Proteinaceous Molecule from the Marine Bacterium . Microorganisms, 8(9), . https://doi.org/10.3390/microorganisms8091295
Doghri, Ibtissem, et al. "Anti-Biofilm Activity of a Low Weight Proteinaceous Molecule from the Marine Bacterium ." Microorganisms vol. 8,9 (2020). doi: https://doi.org/10.3390/microorganisms8091295
Doghri I, Portier E, Desriac F, Zhao JM, Bazire A, Dufour A, Rochette V, Sablé S, Lanneluc I. Anti-Biofilm Activity of a Low Weight Proteinaceous Molecule from the Marine Bacterium . Microorganisms. 2020 Aug 25;8(9). doi: 10.3390/microorganisms8091295. PMID: 32854286; PMCID: PMC7563690.
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Pharmacology and pharmacovigilance of protein kinase inhibitors.

Therapie

Khouri C, Mahé J, Caquelin L, Locher C, Despas F.
PMID: 34895753
Therapie. 2021 Nov 25; doi: 10.1016/j.therap.2021.11.004. Epub 2021 Nov 25.

Protein kinase inhibitors experienced their advent in the 2000s. Their market introduction made it possible to constitute a class of targeted therapies administered orally. This name was chosen to mark a break with conventional chemotherapy drugs, but it is...

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Khouri C, Mahé J, Caquelin L, et al. Pharmacology and pharmacovigilance of protein kinase inhibitors. Therapie. 2021;doi: 10.1016/j.therap.2021.11.004.
Khouri, C., Mahé, J., Caquelin, L., Locher, C., & Despas, F. (2021). Pharmacology and pharmacovigilance of protein kinase inhibitors. Therapie, . https://doi.org/10.1016/j.therap.2021.11.004
Khouri, Charles, et al. "Pharmacology and pharmacovigilance of protein kinase inhibitors." Therapie vol. (2021). doi: https://doi.org/10.1016/j.therap.2021.11.004
Khouri C, Mahé J, Caquelin L, Locher C, Despas F. Pharmacology and pharmacovigilance of protein kinase inhibitors. Therapie. 2021 Nov 25; doi: 10.1016/j.therap.2021.11.004. Epub 2021 Nov 25. PMID: 34895753.
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Fitness Costs Predict Emotional, Moral, and Attitudinal Inbreeding Aversion.

Frontiers in psychology

Lespiau F, Kaminski G.
PMID: 27933026
Front Psychol. 2016 Nov 24;7:1860. doi: 10.3389/fpsyg.2016.01860. eCollection 2016.

In terms of sexual intercourse, the very last people we think about are our kin. Imagining inbreeding intercourse, whether it involves our closest kin or not, induces aversion in most people who invoke inbreeding depression problems or cultural considerations....

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Lespiau F, Kaminski G. Fitness Costs Predict Emotional, Moral, and Attitudinal Inbreeding Aversion. Front Psychol. 2016;7:1860doi: 10.3389/fpsyg.2016.01860.
Lespiau, F., & Kaminski, G. (2016). Fitness Costs Predict Emotional, Moral, and Attitudinal Inbreeding Aversion. Frontiers in psychology, 71860. https://doi.org/10.3389/fpsyg.2016.01860
Lespiau, Florence, and Kaminski, Gwenaël. "Fitness Costs Predict Emotional, Moral, and Attitudinal Inbreeding Aversion." Frontiers in psychology vol. 7 (2016): 1860. doi: https://doi.org/10.3389/fpsyg.2016.01860
Lespiau F, Kaminski G. Fitness Costs Predict Emotional, Moral, and Attitudinal Inbreeding Aversion. Front Psychol. 2016 Nov 24;7:1860. doi: 10.3389/fpsyg.2016.01860. eCollection 2016. PMID: 27933026; PMCID: PMC5121126.
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Intracranial Pressure Is a Determinant of Sympathetic Activity.

Frontiers in physiology

Schmidt EA, Despas F, Pavy-Le Traon A, Czosnyka Z, Pickard JD, Rahmouni K, Pathak A, Senard JM.
PMID: 29472865
Front Physiol. 2018 Feb 08;9:11. doi: 10.3389/fphys.2018.00011. eCollection 2018.

Intracranial pressure (ICP) is the pressure within the

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Schmidt EA, Despas F, Pavy-Le Traon A, et al. Intracranial Pressure Is a Determinant of Sympathetic Activity. Front Physiol. 2018;9:11doi: 10.3389/fphys.2018.00011.
Schmidt, E. A., Despas, F., Pavy-Le Traon, A., Czosnyka, Z., Pickard, J. D., Rahmouni, K., Pathak, A., & Senard, J. M. (2018). Intracranial Pressure Is a Determinant of Sympathetic Activity. Frontiers in physiology, 911. https://doi.org/10.3389/fphys.2018.00011
Schmidt, Eric A, et al. "Intracranial Pressure Is a Determinant of Sympathetic Activity." Frontiers in physiology vol. 9 (2018): 11. doi: https://doi.org/10.3389/fphys.2018.00011
Schmidt EA, Despas F, Pavy-Le Traon A, Czosnyka Z, Pickard JD, Rahmouni K, Pathak A, Senard JM. Intracranial Pressure Is a Determinant of Sympathetic Activity. Front Physiol. 2018 Feb 08;9:11. doi: 10.3389/fphys.2018.00011. eCollection 2018. PMID: 29472865; PMCID: PMC5809772.
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Showing 1 to 12 of 48 entries