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Showing 1 to 11 of 11 entries
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Mast Cell: A Multi-Functional Master Cell.

Frontiers in immunology

Krystel-Whittemore M, Dileepan KN, Wood JG.
PMID: 26779180
Front Immunol. 2016 Jan 06;6:620. doi: 10.3389/fimmu.2015.00620. eCollection 2015.

Mast cells are immune cells of the myeloid lineage and are present in connective tissues throughout the body. The activation and degranulation of mast cells significantly modulates many aspects of physiological and pathological conditions in various settings. With respect...

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Krystel-Whittemore M, Dileepan KN, Wood JG. Mast Cell: A Multi-Functional Master Cell. Front Immunol. 2016;6:620doi: 10.3389/fimmu.2015.00620.
Krystel-Whittemore, M., Dileepan, K. N., & Wood, J. G. (2015). Mast Cell: A Multi-Functional Master Cell. Frontiers in immunology, 6620. https://doi.org/10.3389/fimmu.2015.00620
Krystel-Whittemore, Melissa, et al. "Mast Cell: A Multi-Functional Master Cell." Frontiers in immunology vol. 6 (2015): 620. doi: https://doi.org/10.3389/fimmu.2015.00620
Krystel-Whittemore M, Dileepan KN, Wood JG. Mast Cell: A Multi-Functional Master Cell. Front Immunol. 2016 Jan 06;6:620. doi: 10.3389/fimmu.2015.00620. eCollection 2015. PMID: 26779180; PMCID: PMC4701915.
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Cigarette Smoke Amplifies Inflammatory Response and Atherosclerosis Progression Through Activation of the H1R-TLR2/4-COX2 Axis.

Frontiers in immunology

Barua RS, Sharma M, Dileepan KN.
PMID: 26617606
Front Immunol. 2015 Nov 09;6:572. doi: 10.3389/fimmu.2015.00572. eCollection 2015.

Emerging evidence suggests that infection and persistent inflammation are key players in the pathogenesis of atherosclerotic cardiovascular disease (CVD). Although it is well established that cigarette smoke (CS) promotes atherosclerotic CVD, very little is known about the potential impact...

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Barua RS, Sharma M, Dileepan KN. Cigarette Smoke Amplifies Inflammatory Response and Atherosclerosis Progression Through Activation of the H1R-TLR2/4-COX2 Axis. Front Immunol. 2015;6:572doi: 10.3389/fimmu.2015.00572.
Barua, R. S., Sharma, M., & Dileepan, K. N. (2015). Cigarette Smoke Amplifies Inflammatory Response and Atherosclerosis Progression Through Activation of the H1R-TLR2/4-COX2 Axis. Frontiers in immunology, 6572. https://doi.org/10.3389/fimmu.2015.00572
Barua, Rajat S, et al. "Cigarette Smoke Amplifies Inflammatory Response and Atherosclerosis Progression Through Activation of the H1R-TLR2/4-COX2 Axis." Frontiers in immunology vol. 6 (2015): 572. doi: https://doi.org/10.3389/fimmu.2015.00572
Barua RS, Sharma M, Dileepan KN. Cigarette Smoke Amplifies Inflammatory Response and Atherosclerosis Progression Through Activation of the H1R-TLR2/4-COX2 Axis. Front Immunol. 2015 Nov 09;6:572. doi: 10.3389/fimmu.2015.00572. eCollection 2015. PMID: 26617606; PMCID: PMC4638143.
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Altered CD38/Cyclic ADP-Ribose Signaling Contributes to the Asthmatic Phenotype.

Journal of allergy

Jude JA, Dileepan M, Panettieri RA, Walseth TF, Kannan MS.
PMID: 23213344
J Allergy (Cairo). 2012;2012:289468. doi: 10.1155/2012/289468. Epub 2012 Nov 20.

CD38 is a transmembrane glycoprotein expressed in airway smooth muscle cells. The enzymatic activity of CD38 generates cyclic ADP-ribose from β-NAD. Cyclic ADP-ribose mobilizes intracellular calcium during activation of airway smooth muscle cells by G-protein-coupled receptors through activation of...

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Jude JA, Dileepan M, Panettieri RA, et al. Altered CD38/Cyclic ADP-Ribose Signaling Contributes to the Asthmatic Phenotype. J Allergy (Cairo). 2012;2012:289468doi: 10.1155/2012/289468.
Jude, J. A., Dileepan, M., Panettieri, R. A., Walseth, T. F., & Kannan, M. S. (2012). Altered CD38/Cyclic ADP-Ribose Signaling Contributes to the Asthmatic Phenotype. Journal of allergy, 2012289468. https://doi.org/10.1155/2012/289468
Jude, Joseph A, et al. "Altered CD38/Cyclic ADP-Ribose Signaling Contributes to the Asthmatic Phenotype." Journal of allergy vol. 2012 (2012): 289468. doi: https://doi.org/10.1155/2012/289468
Jude JA, Dileepan M, Panettieri RA, Walseth TF, Kannan MS. Altered CD38/Cyclic ADP-Ribose Signaling Contributes to the Asthmatic Phenotype. J Allergy (Cairo). 2012;2012:289468. doi: 10.1155/2012/289468. Epub 2012 Nov 20. PMID: 23213344; PMCID: PMC3508580.
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Exploration of RNA-Seq data to identify a potential pathogen of the leaf-mining moth, .

Data in brief

Etebari K, Taylor DBJ, Rahman MM, Dhileepan K, Furlong MJ, Asgari S.
PMID: 34977297
Data Brief. 2021 Dec 11;40:107708. doi: 10.1016/j.dib.2021.107708. eCollection 2022 Feb.

The leaf-mining moth,

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Etebari K, Taylor DBJ, Rahman MM, et al. Exploration of RNA-Seq data to identify a potential pathogen of the leaf-mining moth, . Data Brief. 2021;40:107708doi: 10.1016/j.dib.2021.107708.
Etebari, K., Taylor, D. B. J., Rahman, M. M., Dhileepan, K., Furlong, M. J., & Asgari, S. (2022). Exploration of RNA-Seq data to identify a potential pathogen of the leaf-mining moth, . Data in brief, 40107708. https://doi.org/10.1016/j.dib.2021.107708
Etebari, Kayvan, et al. "Exploration of RNA-Seq data to identify a potential pathogen of the leaf-mining moth, ." Data in brief vol. 40 (2022): 107708. doi: https://doi.org/10.1016/j.dib.2021.107708
Etebari K, Taylor DBJ, Rahman MM, Dhileepan K, Furlong MJ, Asgari S. Exploration of RNA-Seq data to identify a potential pathogen of the leaf-mining moth, . Data Brief. 2021 Dec 11;40:107708. doi: 10.1016/j.dib.2021.107708. eCollection 2022 Feb. PMID: 34977297; PMCID: PMC8685975.
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Effect Of Dual sEH/COX-2 Inhibition on Allergen-Induced Airway Inflammation.

Frontiers in pharmacology

Dileepan M, Rastle-Simpson S, Greenberg Y, Wijesinghe DS, Kumar NG, Yang J, Hwang SH, Hammock BD, Sriramarao P, Rao SP.
PMID: 31611798
Front Pharmacol. 2019 Sep 27;10:1118. doi: 10.3389/fphar.2019.01118. eCollection 2019.

Arachidonic acid metabolites resulting from the cyclooxygenase (COX), lipoxygenase, and cytochrome P450 oxidase enzymatic pathways play pro- and anti-inflammatory roles in allergic airway inflammation (AAI) and asthma. Expression of COX-2 and soluble epoxide hydrolase (sEH) are elevated in allergic...

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Dileepan M, Rastle-Simpson S, Greenberg Y, et al. Effect Of Dual sEH/COX-2 Inhibition on Allergen-Induced Airway Inflammation. Front Pharmacol. 2019;10:1118doi: 10.3389/fphar.2019.01118.
Dileepan, M., Rastle-Simpson, S., Greenberg, Y., Wijesinghe, D. S., Kumar, N. G., Yang, J., Hwang, S. H., Hammock, B. D., Sriramarao, P., & Rao, S. P. (2019). Effect Of Dual sEH/COX-2 Inhibition on Allergen-Induced Airway Inflammation. Frontiers in pharmacology, 101118. https://doi.org/10.3389/fphar.2019.01118
Dileepan, Mythili, et al. "Effect Of Dual sEH/COX-2 Inhibition on Allergen-Induced Airway Inflammation." Frontiers in pharmacology vol. 10 (2019): 1118. doi: https://doi.org/10.3389/fphar.2019.01118
Dileepan M, Rastle-Simpson S, Greenberg Y, Wijesinghe DS, Kumar NG, Yang J, Hwang SH, Hammock BD, Sriramarao P, Rao SP. Effect Of Dual sEH/COX-2 Inhibition on Allergen-Induced Airway Inflammation. Front Pharmacol. 2019 Sep 27;10:1118. doi: 10.3389/fphar.2019.01118. eCollection 2019. PMID: 31611798; PMCID: PMC6777353.
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Phagocytosis of mast cell granules results in decreased macrophage superoxide production.

Mediators of inflammation

Shah BA, Li Y, Stechschulte DJ, Dileepan KN.
PMID: 18475672
Mediators Inflamm. 1995;4(6):406-12. doi: 10.1155/S0962935195000652.

The mechanism by which phagocytosed mast cell granules (MCGs) inhibit macrophage superoxide production has not been defined. In this study, rat peritoneal macrophages were co-incubated with either isolated intact MCGs or MCG-sonicate, and their respiratory burst capacity and morphology...

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Shah BA, Li Y, Stechschulte DJ, et al. Phagocytosis of mast cell granules results in decreased macrophage superoxide production. Mediators Inflamm. 1995;4(6):406-12doi: 10.1155/S0962935195000652.
Shah, B. A., Li, Y., Stechschulte, D. J., & Dileepan, K. N. (1995). Phagocytosis of mast cell granules results in decreased macrophage superoxide production. Mediators of inflammation, 4(6), 406-12. https://doi.org/10.1155/S0962935195000652
Shah, B A, et al. "Phagocytosis of mast cell granules results in decreased macrophage superoxide production." Mediators of inflammation vol. 4,6 (1995): 406-12. doi: https://doi.org/10.1155/S0962935195000652
Shah BA, Li Y, Stechschulte DJ, Dileepan KN. Phagocytosis of mast cell granules results in decreased macrophage superoxide production. Mediators Inflamm. 1995;4(6):406-12. doi: 10.1155/S0962935195000652. PMID: 18475672; PMCID: PMC2365671.
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Protective Role of Mast Cells in Primary Systemic Vasculitis: A Perspective.

Frontiers in immunology

Springer JM, Raveendran VV, Gierer SA, Maz M, Dileepan KN.
PMID: 28878769
Front Immunol. 2017 Aug 23;8:990. doi: 10.3389/fimmu.2017.00990. eCollection 2017.

Mast cells are important cells of the immune system. Although traditionally considered as key players in allergic and hypersensitivity reactions, emerging evidence suggests that mast cells have many complex roles in vascular disease. These include regulation of vasodilation, angiogenesis,...

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Springer JM, Raveendran VV, Gierer SA, et al. Protective Role of Mast Cells in Primary Systemic Vasculitis: A Perspective. Front Immunol. 2017;8:990doi: 10.3389/fimmu.2017.00990.
Springer, J. M., Raveendran, V. V., Gierer, S. A., Maz, M., & Dileepan, K. N. (2017). Protective Role of Mast Cells in Primary Systemic Vasculitis: A Perspective. Frontiers in immunology, 8990. https://doi.org/10.3389/fimmu.2017.00990
Springer, Jason M, et al. "Protective Role of Mast Cells in Primary Systemic Vasculitis: A Perspective." Frontiers in immunology vol. 8 (2017): 990. doi: https://doi.org/10.3389/fimmu.2017.00990
Springer JM, Raveendran VV, Gierer SA, Maz M, Dileepan KN. Protective Role of Mast Cells in Primary Systemic Vasculitis: A Perspective. Front Immunol. 2017 Aug 23;8:990. doi: 10.3389/fimmu.2017.00990. eCollection 2017. PMID: 28878769; PMCID: PMC5572344.
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A New Paediatric Diabetes Knowledge Test - M-WIKAD Development and Factor Analysis.

European endocrinology

Tsai S, Patton S, DeLurgio S, Williams DD, Dileepan K, Karmazin A, Storm M, Clements M.
PMID: 31244902
Eur Endocrinol. 2019 Apr;15(1):1-5. doi: 10.17925/EE.2019.15.1.1. Epub 2019 Apr 12.

No abstract available.

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Tsai S, Patton S, DeLurgio S, et al. A New Paediatric Diabetes Knowledge Test - M-WIKAD Development and Factor Analysis. Eur Endocrinol. 2019;15(1):1-5doi: 10.17925/EE.2019.15.1.1.
Tsai, S., Patton, S., DeLurgio, S., Williams, D. D., Dileepan, K., Karmazin, A., Storm, M., & Clements, M. (2019). A New Paediatric Diabetes Knowledge Test - M-WIKAD Development and Factor Analysis. European endocrinology, 15(1), 1-5. https://doi.org/10.17925/EE.2019.15.1.1
Tsai, Sarah, et al. "A New Paediatric Diabetes Knowledge Test - M-WIKAD Development and Factor Analysis." European endocrinology vol. 15,1 (2019): 1-5. doi: https://doi.org/10.17925/EE.2019.15.1.1
Tsai S, Patton S, DeLurgio S, Williams DD, Dileepan K, Karmazin A, Storm M, Clements M. A New Paediatric Diabetes Knowledge Test - M-WIKAD Development and Factor Analysis. Eur Endocrinol. 2019 Apr;15(1):1-5. doi: 10.17925/EE.2019.15.1.1. Epub 2019 Apr 12. PMID: 31244902; PMCID: PMC6587900.
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Exploration of RNA-Seq data to identify a potential pathogen of the leaf-mining moth, .

Data in brief

Etebari K, Taylor DBJ, Rahman MM, Dhileepan K, Furlong MJ, Asgari S.
PMID: 34977297
Data Brief. 2021 Dec 11;40:107708. doi: 10.1016/j.dib.2021.107708. eCollection 2022 Feb.

The leaf-mining moth,

Cite
Etebari K, Taylor DBJ, Rahman MM, et al. Exploration of RNA-Seq data to identify a potential pathogen of the leaf-mining moth, . Data Brief. 2021;40:107708doi: 10.1016/j.dib.2021.107708.
Etebari, K., Taylor, D. B. J., Rahman, M. M., Dhileepan, K., Furlong, M. J., & Asgari, S. (2022). Exploration of RNA-Seq data to identify a potential pathogen of the leaf-mining moth, . Data in brief, 40107708. https://doi.org/10.1016/j.dib.2021.107708
Etebari, Kayvan, et al. "Exploration of RNA-Seq data to identify a potential pathogen of the leaf-mining moth, ." Data in brief vol. 40 (2022): 107708. doi: https://doi.org/10.1016/j.dib.2021.107708
Etebari K, Taylor DBJ, Rahman MM, Dhileepan K, Furlong MJ, Asgari S. Exploration of RNA-Seq data to identify a potential pathogen of the leaf-mining moth, . Data Brief. 2021 Dec 11;40:107708. doi: 10.1016/j.dib.2021.107708. eCollection 2022 Feb. PMID: 34977297; PMCID: PMC8685975.
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Biology, Ecology and Management of the Invasive Navua Sedge (.

Plants (Basel, Switzerland)

Shi B, Osunkoya OO, Chadha A, Florentine SK, Dhileepan K.
PMID: 34579384
Plants (Basel). 2021 Sep 07;10(9). doi: 10.3390/plants10091851.

Navua sedge (

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Shi B, Osunkoya OO, Chadha A, et al. Biology, Ecology and Management of the Invasive Navua Sedge (. Plants (Basel). 2021;10(9)doi: 10.3390/plants10091851.
Shi, B., Osunkoya, O. O., Chadha, A., Florentine, S. K., & Dhileepan, K. (2021). Biology, Ecology and Management of the Invasive Navua Sedge (. Plants (Basel, Switzerland), 10(9), . https://doi.org/10.3390/plants10091851
Shi, Boyang, et al. "Biology, Ecology and Management of the Invasive Navua Sedge (." Plants (Basel, Switzerland) vol. 10,9 (2021). doi: https://doi.org/10.3390/plants10091851
Shi B, Osunkoya OO, Chadha A, Florentine SK, Dhileepan K. Biology, Ecology and Management of the Invasive Navua Sedge (. Plants (Basel). 2021 Sep 07;10(9). doi: 10.3390/plants10091851. PMID: 34579384; PMCID: PMC8465260.
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Recombinant SARS-CoV-2 Nucleocapsid Protein: Expression, Purification, and Its Biochemical Characterization and Utility in Serological Assay Development to Assess Immunological Responses to SARS-CoV-2 Infection.

Pathogens (Basel, Switzerland)

Di D, Dileepan M, Ahmed S, Liang Y, Ly H.
PMID: 34451501
Pathogens. 2021 Aug 16;10(8). doi: 10.3390/pathogens10081039.

The SARS-CoV-2 nucleocapsid protein (N) binds a single-stranded viral RNA genome to form a helical ribonucleoprotein complex that is packaged into virion particles. N is relatively conserved among coronaviruses and consists of the N-terminal domain (NTD) and C-terminal domain...

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Di D, Dileepan M, Ahmed S, et al. Recombinant SARS-CoV-2 Nucleocapsid Protein: Expression, Purification, and Its Biochemical Characterization and Utility in Serological Assay Development to Assess Immunological Responses to SARS-CoV-2 Infection. Pathogens. 2021;10(8)doi: 10.3390/pathogens10081039.
Di, D., Dileepan, M., Ahmed, S., Liang, Y., & Ly, H. (2021). Recombinant SARS-CoV-2 Nucleocapsid Protein: Expression, Purification, and Its Biochemical Characterization and Utility in Serological Assay Development to Assess Immunological Responses to SARS-CoV-2 Infection. Pathogens (Basel, Switzerland), 10(8), . https://doi.org/10.3390/pathogens10081039
Di, Da, et al. "Recombinant SARS-CoV-2 Nucleocapsid Protein: Expression, Purification, and Its Biochemical Characterization and Utility in Serological Assay Development to Assess Immunological Responses to SARS-CoV-2 Infection." Pathogens (Basel, Switzerland) vol. 10,8 (2021). doi: https://doi.org/10.3390/pathogens10081039
Di D, Dileepan M, Ahmed S, Liang Y, Ly H. Recombinant SARS-CoV-2 Nucleocapsid Protein: Expression, Purification, and Its Biochemical Characterization and Utility in Serological Assay Development to Assess Immunological Responses to SARS-CoV-2 Infection. Pathogens. 2021 Aug 16;10(8). doi: 10.3390/pathogens10081039. PMID: 34451501; PMCID: PMC8402198.
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Showing 1 to 11 of 11 entries