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Showing 1 to 7 of 7 entries
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Genetic Depletion of Adipocyte Creatine Metabolism Inhibits Diet-Induced Thermogenesis and Drives Obesity.

Cell metabolism

Kazak L, Chouchani ET, Lu GZ, Jedrychowski MP, Bare CJ, Mina AI, Kumari M, Zhang S, Vuckovic I, Laznik-Bogoslavski D, Dzeja P, Banks AS, Rosen ED, Spiegelman BM.
PMID: 28978428
Cell Metab. 2017 Oct 03;26(4):693. doi: 10.1016/j.cmet.2017.09.007.

No abstract available.

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Kazak L, Chouchani ET, Lu GZ, et al. Genetic Depletion of Adipocyte Creatine Metabolism Inhibits Diet-Induced Thermogenesis and Drives Obesity. Cell Metab. 2017;26(4):693doi: 10.1016/j.cmet.2017.09.007.
Kazak, L., Chouchani, E. T., Lu, G. Z., Jedrychowski, M. P., Bare, C. J., Mina, A. I., Kumari, M., Zhang, S., Vuckovic, I., Laznik-Bogoslavski, D., Dzeja, P., Banks, A. S., Rosen, E. D., & Spiegelman, B. M. (2017). Genetic Depletion of Adipocyte Creatine Metabolism Inhibits Diet-Induced Thermogenesis and Drives Obesity. Cell metabolism, 26(4), 693. https://doi.org/10.1016/j.cmet.2017.09.007
Kazak, Lawrence, et al. "Genetic Depletion of Adipocyte Creatine Metabolism Inhibits Diet-Induced Thermogenesis and Drives Obesity." Cell metabolism vol. 26,4 (2017): 693. doi: https://doi.org/10.1016/j.cmet.2017.09.007
Kazak L, Chouchani ET, Lu GZ, Jedrychowski MP, Bare CJ, Mina AI, Kumari M, Zhang S, Vuckovic I, Laznik-Bogoslavski D, Dzeja P, Banks AS, Rosen ED, Spiegelman BM. Genetic Depletion of Adipocyte Creatine Metabolism Inhibits Diet-Induced Thermogenesis and Drives Obesity. Cell Metab. 2017 Oct 03;26(4):693. doi: 10.1016/j.cmet.2017.09.007. PMID: 28978428; PMCID: PMC5638127.
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Corrigendum to "Modulation of mitochondrial complex I activity averts cognitive decline in multiple animal models of familial Alzheimer's disease" [EBioMedicine 2 (2015) 294-305].

EBioMedicine

Zhang L, Zhang S, Maezawa I, Trushin S, Minhas P, Pinto M, Jin LW, Prasain K, Nguyen TDT, Yamazaki Y, Kanekiyo T, Bu G, Gateno B, Chang KO, Nath KA, Nemutlu E, Dzeja P, Pang YP, Hua DH, Trushina E.
PMID: 30956174
EBioMedicine. 2019 Apr;42:532. doi: 10.1016/j.ebiom.2019.03.062. Epub 2019 Apr 04.

No abstract available.

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Zhang L, Zhang S, Maezawa I, et al. Corrigendum to "Modulation of mitochondrial complex I activity averts cognitive decline in multiple animal models of familial Alzheimer's disease" [EBioMedicine 2 (2015) 294-305]. EBioMedicine. 2019;42:532doi: 10.1016/j.ebiom.2019.03.062.
Zhang, L., Zhang, S., Maezawa, I., Trushin, S., Minhas, P., Pinto, M., Jin, L. W., Prasain, K., Nguyen, T. D. T., Yamazaki, Y., Kanekiyo, T., Bu, G., Gateno, B., Chang, K. O., Nath, K. A., Nemutlu, E., Dzeja, P., Pang, Y. P., Hua, D. H., & Trushina, E. (2019). Corrigendum to "Modulation of mitochondrial complex I activity averts cognitive decline in multiple animal models of familial Alzheimer's disease" [EBioMedicine 2 (2015) 294-305]. EBioMedicine, 42532. https://doi.org/10.1016/j.ebiom.2019.03.062
Zhang, Liang, et al. "Corrigendum to "Modulation of mitochondrial complex I activity averts cognitive decline in multiple animal models of familial Alzheimer's disease" [EBioMedicine 2 (2015) 294-305]." EBioMedicine vol. 42 (2019): 532. doi: https://doi.org/10.1016/j.ebiom.2019.03.062
Zhang L, Zhang S, Maezawa I, Trushin S, Minhas P, Pinto M, Jin LW, Prasain K, Nguyen TDT, Yamazaki Y, Kanekiyo T, Bu G, Gateno B, Chang KO, Nath KA, Nemutlu E, Dzeja P, Pang YP, Hua DH, Trushina E. Corrigendum to "Modulation of mitochondrial complex I activity averts cognitive decline in multiple animal models of familial Alzheimer's disease" [EBioMedicine 2 (2015) 294-305]. EBioMedicine. 2019 Apr;42:532. doi: 10.1016/j.ebiom.2019.03.062. Epub 2019 Apr 04. PMID: 30956174; PMCID: PMC6491797.
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Adenylate Kinase and Metabolic Signaling in Cancer Cells.

Frontiers in oncology

Klepinin A, Zhang S, Klepinina L, Rebane-Klemm E, Terzic A, Kaambre T, Dzeja P.
PMID: 32509571
Front Oncol. 2020 May 19;10:660. doi: 10.3389/fonc.2020.00660. eCollection 2020.

A hallmark of cancer cells is the ability to rewire their bioenergetics and metabolic signaling circuits to fuel their uncontrolled proliferation and metastasis. Adenylate kinase (AK) is the critical enzyme in the metabolic monitoring of cellular adenine nucleotide homeostasis....

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Klepinin A, Zhang S, Klepinina L, et al. Adenylate Kinase and Metabolic Signaling in Cancer Cells. Front Oncol. 2020;10:660doi: 10.3389/fonc.2020.00660.
Klepinin, A., Zhang, S., Klepinina, L., Rebane-Klemm, E., Terzic, A., Kaambre, T., & Dzeja, P. (2020). Adenylate Kinase and Metabolic Signaling in Cancer Cells. Frontiers in oncology, 10660. https://doi.org/10.3389/fonc.2020.00660
Klepinin, Aleksandr, et al. "Adenylate Kinase and Metabolic Signaling in Cancer Cells." Frontiers in oncology vol. 10 (2020): 660. doi: https://doi.org/10.3389/fonc.2020.00660
Klepinin A, Zhang S, Klepinina L, Rebane-Klemm E, Terzic A, Kaambre T, Dzeja P. Adenylate Kinase and Metabolic Signaling in Cancer Cells. Front Oncol. 2020 May 19;10:660. doi: 10.3389/fonc.2020.00660. eCollection 2020. PMID: 32509571; PMCID: PMC7248387.
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Metabolic assessment of a novel chronic myelogenous leukemic cell line and an imatinib resistant subline by H NMR spectroscopy.

Metabolomics : Official journal of the Metabolomic Society

Dewar BJ, Keshari K, Jeffries R, Dzeja P, Graves LM, Macdonald JM.
PMID: 20676217
Metabolomics. 2010 Sep;6(3):439-450. doi: 10.1007/s11306-010-0204-0. Epub 2010 Mar 23.

The goal of this study was to examine metabolic differences between a novel chronic myelogenous leukemic (CML) cell line, MyL, and a sub-clone, MyL-R, which displays enhanced resistance to the targeted Bcr-Abl tyrosine kinase inhibitor imatinib. (1)H nuclear magnetic...

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Dewar BJ, Keshari K, Jeffries R, et al. Metabolic assessment of a novel chronic myelogenous leukemic cell line and an imatinib resistant subline by H NMR spectroscopy. Metabolomics. 2010;6(3):439-450doi: 10.1007/s11306-010-0204-0.
Dewar, B. J., Keshari, K., Jeffries, R., Dzeja, P., Graves, L. M., & Macdonald, J. M. (2010). Metabolic assessment of a novel chronic myelogenous leukemic cell line and an imatinib resistant subline by H NMR spectroscopy. Metabolomics : Official journal of the Metabolomic Society, 6(3), 439-450. https://doi.org/10.1007/s11306-010-0204-0
Dewar, Brian J, et al. "Metabolic assessment of a novel chronic myelogenous leukemic cell line and an imatinib resistant subline by H NMR spectroscopy." Metabolomics : Official journal of the Metabolomic Society vol. 6,3 (2010): 439-450. doi: https://doi.org/10.1007/s11306-010-0204-0
Dewar BJ, Keshari K, Jeffries R, Dzeja P, Graves LM, Macdonald JM. Metabolic assessment of a novel chronic myelogenous leukemic cell line and an imatinib resistant subline by H NMR spectroscopy. Metabolomics. 2010 Sep;6(3):439-450. doi: 10.1007/s11306-010-0204-0. Epub 2010 Mar 23. PMID: 20676217; PMCID: PMC2899017.
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Modulation of mitochondrial complex I activity averts cognitive decline in multiple animal models of familial Alzheimer's Disease.

EBioMedicine

Zhang L, Zhang S, Maezawa I, Trushin S, Minhas P, Pinto M, Jin LW, Prasain K, Nguyen TD, Yamazaki Y, Kanekiyo T, Bu G, Gateno B, Chang KO, Nath KA, Nemutlu E, Dzeja P, Pang YP, Hua DH, Trushina E.
PMID: 26086035
EBioMedicine. 2015 Apr 01;2(4):294-305. doi: 10.1016/j.ebiom.2015.03.009.

Development of therapeutic strategies to prevent Alzheimer's Disease (AD) is of great importance. We show that mild inhibition of mitochondrial complex I with small molecule CP2 reduces levels of amyloid beta and phospho-Tau and averts cognitive decline in three...

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Zhang L, Zhang S, Maezawa I, et al. Modulation of mitochondrial complex I activity averts cognitive decline in multiple animal models of familial Alzheimer's Disease. EBioMedicine. 2015;2(4):294-305doi: 10.1016/j.ebiom.2015.03.009.
Zhang, L., Zhang, S., Maezawa, I., Trushin, S., Minhas, P., Pinto, M., Jin, L. W., Prasain, K., Nguyen, T. D., Yamazaki, Y., Kanekiyo, T., Bu, G., Gateno, B., Chang, K. O., Nath, K. A., Nemutlu, E., Dzeja, P., Pang, Y. P., Hua, D. H., & Trushina, E. (2015). Modulation of mitochondrial complex I activity averts cognitive decline in multiple animal models of familial Alzheimer's Disease. EBioMedicine, 2(4), 294-305. https://doi.org/10.1016/j.ebiom.2015.03.009
Zhang, Liang, et al. "Modulation of mitochondrial complex I activity averts cognitive decline in multiple animal models of familial Alzheimer's Disease." EBioMedicine vol. 2,4 (2015): 294-305. doi: https://doi.org/10.1016/j.ebiom.2015.03.009
Zhang L, Zhang S, Maezawa I, Trushin S, Minhas P, Pinto M, Jin LW, Prasain K, Nguyen TD, Yamazaki Y, Kanekiyo T, Bu G, Gateno B, Chang KO, Nath KA, Nemutlu E, Dzeja P, Pang YP, Hua DH, Trushina E. Modulation of mitochondrial complex I activity averts cognitive decline in multiple animal models of familial Alzheimer's Disease. EBioMedicine. 2015 Apr 01;2(4):294-305. doi: 10.1016/j.ebiom.2015.03.009. PMID: 26086035; PMCID: PMC4465115.
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Profile of Circulatory Metabolites in a Relapsing-remitting Animal Model of Multiple Sclerosis using Global Metabolomics.

Journal of clinical & cellular immunology

Mangalam A, Poisson L, Nemutlu E, Datta I, Denic A, Dzeja P, Rodriguez M, Rattan R, Giri S.
PMID: 24273690
J Clin Cell Immunol. 2013 Jun 30;4. doi: 10.4172/2155-9899.1000150.

Multiple sclerosis (MS) is a chronic inflammatory and demyelinating disease of the CNS. Although, MS is well characterized in terms of the role played by immune cells, cytokines and CNS pathology, nothing is known about the metabolic alterations that...

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Mangalam A, Poisson L, Nemutlu E, et al. Profile of Circulatory Metabolites in a Relapsing-remitting Animal Model of Multiple Sclerosis using Global Metabolomics. J Clin Cell Immunol. 2013;4doi: 10.4172/2155-9899.1000150.
Mangalam, A., Poisson, L., Nemutlu, E., Datta, I., Denic, A., Dzeja, P., Rodriguez, M., Rattan, R., & Giri, S. (2013). Profile of Circulatory Metabolites in a Relapsing-remitting Animal Model of Multiple Sclerosis using Global Metabolomics. Journal of clinical & cellular immunology, 4. https://doi.org/10.4172/2155-9899.1000150
Mangalam, Ak, et al. "Profile of Circulatory Metabolites in a Relapsing-remitting Animal Model of Multiple Sclerosis using Global Metabolomics." Journal of clinical & cellular immunology vol. 4 (2013). doi: https://doi.org/10.4172/2155-9899.1000150
Mangalam A, Poisson L, Nemutlu E, Datta I, Denic A, Dzeja P, Rodriguez M, Rattan R, Giri S. Profile of Circulatory Metabolites in a Relapsing-remitting Animal Model of Multiple Sclerosis using Global Metabolomics. J Clin Cell Immunol. 2013 Jun 30;4. doi: 10.4172/2155-9899.1000150. PMID: 24273690; PMCID: PMC3837296.
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Effects of histamine application and water-immersion stress on gizzard erosion and fattening of broiler chicks.

Avian pathology : journal of the W.V.P.A

Dzaja P, Grabazrevic Z, Peric J, Mazija H, Prukner-Radovcic E, Bratulic M, Zubcic D, Ragland WL.
PMID: 18645863
Avian Pathol. 1996 Jun;25(2):359-67. doi: 10.1080/03079459608419146.

Three groups of Hybro broiler chickens, of which one group were controls, a second treated intragastrically with histamine, and a third stressed by immersion in water, were used in the experiment. Serum activities of aspartate and alanine aminotransferase and...

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Dzaja P, Grabazrevic Z, Peric J, et al. Effects of histamine application and water-immersion stress on gizzard erosion and fattening of broiler chicks. Avian Pathol. 1996;25(2):359-67doi: 10.1080/03079459608419146.
Dzaja, P., Grabazrevic, Z., Peric, J., Mazija, H., Prukner-Radovcic, E., Bratulic, M., Zubcic, D., & Ragland, W. L. (1996). Effects of histamine application and water-immersion stress on gizzard erosion and fattening of broiler chicks. Avian pathology : journal of the W.V.P.A, 25(2), 359-67. https://doi.org/10.1080/03079459608419146
Dzaja, P, et al. "Effects of histamine application and water-immersion stress on gizzard erosion and fattening of broiler chicks." Avian pathology : journal of the W.V.P.A vol. 25,2 (1996): 359-67. doi: https://doi.org/10.1080/03079459608419146
Dzaja P, Grabazrevic Z, Peric J, Mazija H, Prukner-Radovcic E, Bratulic M, Zubcic D, Ragland WL. Effects of histamine application and water-immersion stress on gizzard erosion and fattening of broiler chicks. Avian Pathol. 1996 Jun;25(2):359-67. doi: 10.1080/03079459608419146. PMID: 18645863.
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Showing 1 to 7 of 7 entries