Advanced Search
Display options
Filter resources
Text Availability
Article type
Publication date
Species
Language
Sex
Age
Showing 1 to 12 of 19 entries
Sorted by: Best Match Show Resources per page
Senolytic elimination of Cox2-expressing senescent cells inhibits the growth of premalignant pancreatic lesions.

Gut

Kolodkin-Gal D, Roitman L, Ovadya Y, Azazmeh N, Assouline B, Schlesinger Y, Kalifa R, Horwitz S, Khalatnik Y, Hochner-Ger A, Imam A, Demma JA, Winter E, Benyamini H, Elgavish S, Khatib AA, Meir K, Atlan K, Pikarsky E, Parnas O, Dor Y, Zamir G, Ben-Porath I, Krizhanovsky V.
PMID: 33649045
Gut. 2022 Feb;71(2):345-355. doi: 10.1136/gutjnl-2020-321112. Epub 2021 Mar 01.

OBJECTIVE: Cellular senescence limits tumourigenesis by blocking the proliferation of premalignant cells. Additionally, however, senescent cells can exert paracrine effects influencing tumour growth. Senescent cells are present in premalignant pancreatic intraepithelial neoplasia (PanIN) lesions, yet their effects on the...

Cite
Kolodkin-Gal D, Roitman L, Ovadya Y, et al. Senolytic elimination of Cox2-expressing senescent cells inhibits the growth of premalignant pancreatic lesions. Gut. 2021;71(2):345-355doi: 10.1136/gutjnl-2020-321112.
Kolodkin-Gal, D., Roitman, L., Ovadya, Y., Azazmeh, N., Assouline, B., Schlesinger, Y., Kalifa, R., Horwitz, S., Khalatnik, Y., Hochner-Ger, A., Imam, A., Demma, J. A., Winter, E., Benyamini, H., Elgavish, S., Khatib, A. A., Meir, K., Atlan, K., Pikarsky, E., Parnas, O., Dor, Y., Zamir, G., Ben-Porath, I., & Krizhanovsky, V. (2022). Senolytic elimination of Cox2-expressing senescent cells inhibits the growth of premalignant pancreatic lesions. Gut, 71(2), 345-355. https://doi.org/10.1136/gutjnl-2020-321112
Kolodkin-Gal, Dror, et al. "Senolytic elimination of Cox2-expressing senescent cells inhibits the growth of premalignant pancreatic lesions." Gut vol. 71,2 (2022): 345-355. doi: https://doi.org/10.1136/gutjnl-2020-321112
Kolodkin-Gal D, Roitman L, Ovadya Y, Azazmeh N, Assouline B, Schlesinger Y, Kalifa R, Horwitz S, Khalatnik Y, Hochner-Ger A, Imam A, Demma JA, Winter E, Benyamini H, Elgavish S, Khatib AA, Meir K, Atlan K, Pikarsky E, Parnas O, Dor Y, Zamir G, Ben-Porath I, Krizhanovsky V. Senolytic elimination of Cox2-expressing senescent cells inhibits the growth of premalignant pancreatic lesions. Gut. 2022 Feb;71(2):345-355. doi: 10.1136/gutjnl-2020-321112. Epub 2021 Mar 01. PMID: 33649045.
Copy Download .nbib Format: NLM
Nonbinary quantification technique accounting for myocardial infarct heterogeneity: Feasibility of applying percent infarct mapping in patients.

Journal of magnetic resonance imaging : JMRI

Mastrodicasa D, Elgavish GA, Schoepf UJ, Suranyi P, van Assen M, Albrecht MH, De Cecco CN, van der Geest RJ, Hardy R, Mantini C, Griffith LP, Ruzsics B, Varga-Szemes A.
PMID: 29446527
J Magn Reson Imaging. 2018 Feb 15; doi: 10.1002/jmri.25973. Epub 2018 Feb 15.

BACKGROUND: Binary threshold-based quantification techniques ignore myocardial infarct (MI) heterogeneity, yielding substantial misquantification of MI.PURPOSE: To assess the technical feasibility of MI quantification using percent infarct mapping (PIM), a prototype nonbinary algorithm, in patients with suspected MI.STUDY TYPE: Prospective...

Cite
Mastrodicasa D, Elgavish GA, Schoepf UJ, et al. Nonbinary quantification technique accounting for myocardial infarct heterogeneity: Feasibility of applying percent infarct mapping in patients. J Magn Reson Imaging. 2018;doi: 10.1002/jmri.25973.
Mastrodicasa, D., Elgavish, G. A., Schoepf, U. J., Suranyi, P., van Assen, M., Albrecht, M. H., De Cecco, C. N., van der Geest, R. J., Hardy, R., Mantini, C., Griffith, L. P., Ruzsics, B., & Varga-Szemes, A. (2018). Nonbinary quantification technique accounting for myocardial infarct heterogeneity: Feasibility of applying percent infarct mapping in patients. Journal of magnetic resonance imaging : JMRI, . https://doi.org/10.1002/jmri.25973
Mastrodicasa, Domenico, et al. "Nonbinary quantification technique accounting for myocardial infarct heterogeneity: Feasibility of applying percent infarct mapping in patients." Journal of magnetic resonance imaging : JMRI vol. (2018). doi: https://doi.org/10.1002/jmri.25973
Mastrodicasa D, Elgavish GA, Schoepf UJ, Suranyi P, van Assen M, Albrecht MH, De Cecco CN, van der Geest RJ, Hardy R, Mantini C, Griffith LP, Ruzsics B, Varga-Szemes A. Nonbinary quantification technique accounting for myocardial infarct heterogeneity: Feasibility of applying percent infarct mapping in patients. J Magn Reson Imaging. 2018 Feb 15; doi: 10.1002/jmri.25973. Epub 2018 Feb 15. PMID: 29446527.
Copy Download .nbib Format: NLM
Senolytic elimination of Cox2-expressing senescent cells inhibits the growth of premalignant pancreatic lesions.

Gut

Kolodkin-Gal D, Roitman L, Ovadya Y, Azazmeh N, Assouline B, Schlesinger Y, Kalifa R, Horwitz S, Khalatnik Y, Hochner-Ger A, Imam A, Demma JA, Winter E, Benyamini H, Elgavish S, Khatib AA, Meir K, Atlan K, Pikarsky E, Parnas O, Dor Y, Zamir G, Ben-Porath I, Krizhanovsky V.
PMID: 33649045
Gut. 2021 Mar 01; doi: 10.1136/gutjnl-2020-321112. Epub 2021 Mar 01.

OBJECTIVE: Cellular senescence limits tumourigenesis by blocking the proliferation of premalignant cells. Additionally, however, senescent cells can exert paracrine effects influencing tumour growth. Senescent cells are present in premalignant pancreatic intraepithelial neoplasia (PanIN) lesions, yet their effects on the...

Cite
Kolodkin-Gal D, Roitman L, Ovadya Y, et al. Senolytic elimination of Cox2-expressing senescent cells inhibits the growth of premalignant pancreatic lesions. Gut. 2021;doi: 10.1136/gutjnl-2020-321112.
Kolodkin-Gal, D., Roitman, L., Ovadya, Y., Azazmeh, N., Assouline, B., Schlesinger, Y., Kalifa, R., Horwitz, S., Khalatnik, Y., Hochner-Ger, A., Imam, A., Demma, J. A., Winter, E., Benyamini, H., Elgavish, S., Khatib, A. A., Meir, K., Atlan, K., Pikarsky, E., Parnas, O., Dor, Y., Zamir, G., Ben-Porath, I., & Krizhanovsky, V. (2021). Senolytic elimination of Cox2-expressing senescent cells inhibits the growth of premalignant pancreatic lesions. Gut, . https://doi.org/10.1136/gutjnl-2020-321112
Kolodkin-Gal, Dror, et al. "Senolytic elimination of Cox2-expressing senescent cells inhibits the growth of premalignant pancreatic lesions." Gut vol. (2021). doi: https://doi.org/10.1136/gutjnl-2020-321112
Kolodkin-Gal D, Roitman L, Ovadya Y, Azazmeh N, Assouline B, Schlesinger Y, Kalifa R, Horwitz S, Khalatnik Y, Hochner-Ger A, Imam A, Demma JA, Winter E, Benyamini H, Elgavish S, Khatib AA, Meir K, Atlan K, Pikarsky E, Parnas O, Dor Y, Zamir G, Ben-Porath I, Krizhanovsky V. Senolytic elimination of Cox2-expressing senescent cells inhibits the growth of premalignant pancreatic lesions. Gut. 2021 Mar 01; doi: 10.1136/gutjnl-2020-321112. Epub 2021 Mar 01. PMID: 33649045.
Copy Download .nbib Format: NLM
Multiple Roles of IL6 in Hepatic Injury, Steatosis, and Senescence Aggregate to Suppress Tumorigenesis.

Cancer research

Shriki A, Lanton T, Sonnenblick A, Levkovitch-Siany O, Eidelshtein D, Abramovitch R, Rosenberg N, Pappo O, Elgavish S, Nevo Y, Safadi R, Peled A, Rose-John S, Galun E, Axelrod JH.
PMID: 34117031
Cancer Res. 2021 Sep 15;81(18):4766-4777. doi: 10.1158/0008-5472.CAN-21-0321. Epub 2021 Jun 11.

Hepatocellular carcinoma (HCC) typically develops on a background of chronic hepatitis for which the proinflammatory cytokine IL6 is conventionally considered a crucial driving factor. Paradoxically, IL6 also acts as a hepatoprotective factor in chronic liver injury. Here we used...

Cite
Shriki A, Lanton T, Sonnenblick A, et al. Multiple Roles of IL6 in Hepatic Injury, Steatosis, and Senescence Aggregate to Suppress Tumorigenesis. Cancer Res. 2021;81(18):4766-4777doi: 10.1158/0008-5472.CAN-21-0321.
Shriki, A., Lanton, T., Sonnenblick, A., Levkovitch-Siany, O., Eidelshtein, D., Abramovitch, R., Rosenberg, N., Pappo, O., Elgavish, S., Nevo, Y., Safadi, R., Peled, A., Rose-John, S., Galun, E., & Axelrod, J. H. (2021). Multiple Roles of IL6 in Hepatic Injury, Steatosis, and Senescence Aggregate to Suppress Tumorigenesis. Cancer research, 81(18), 4766-4777. https://doi.org/10.1158/0008-5472.CAN-21-0321
Shriki, Anat, et al. "Multiple Roles of IL6 in Hepatic Injury, Steatosis, and Senescence Aggregate to Suppress Tumorigenesis." Cancer research vol. 81,18 (2021): 4766-4777. doi: https://doi.org/10.1158/0008-5472.CAN-21-0321
Shriki A, Lanton T, Sonnenblick A, Levkovitch-Siany O, Eidelshtein D, Abramovitch R, Rosenberg N, Pappo O, Elgavish S, Nevo Y, Safadi R, Peled A, Rose-John S, Galun E, Axelrod JH. Multiple Roles of IL6 in Hepatic Injury, Steatosis, and Senescence Aggregate to Suppress Tumorigenesis. Cancer Res. 2021 Sep 15;81(18):4766-4777. doi: 10.1158/0008-5472.CAN-21-0321. Epub 2021 Jun 11. PMID: 34117031.
Copy Download .nbib Format: NLM
MTADV 5-MER peptide suppresses chronic inflammations as well as autoimmune pathologies and unveils a new potential target-Serum Amyloid A.

Journal of autoimmunity

Hemed-Shaked M, Cowman MK, Kim JR, Huang X, Chau E, Ovadia H, Amar KO, Eshkar-Sebban L, Melamed M, Lev LB, Kedar E, Armengol J, Alemany J, Beyth S, Okon E, Kanduc D, Elgavish S, Wallach-Dayan SB, Cohen SJ, Naor D.
PMID: 34390919
J Autoimmun. 2021 Nov;124:102713. doi: 10.1016/j.jaut.2021.102713. Epub 2021 Aug 12.

Despite the existence of potent anti-inflammatory biological drugs e.g., anti-TNF and anti IL-6 receptor antibodies, for treating chronic inflammatory and autoimmune diseases, these are costly and not specific. Cheaper oral available drugs remain an unmet need. Expression of the...

Cite
Hemed-Shaked M, Cowman MK, Kim JR, et al. MTADV 5-MER peptide suppresses chronic inflammations as well as autoimmune pathologies and unveils a new potential target-Serum Amyloid A. J Autoimmun. 2021;124:102713doi: 10.1016/j.jaut.2021.102713.
Hemed-Shaked, M., Cowman, M. K., Kim, J. R., Huang, X., Chau, E., Ovadia, H., Amar, K. O., Eshkar-Sebban, L., Melamed, M., Lev, L. B., Kedar, E., Armengol, J., Alemany, J., Beyth, S., Okon, E., Kanduc, D., Elgavish, S., Wallach-Dayan, S. B., Cohen, S. J., & Naor, D. (2021). MTADV 5-MER peptide suppresses chronic inflammations as well as autoimmune pathologies and unveils a new potential target-Serum Amyloid A. Journal of autoimmunity, 124102713. https://doi.org/10.1016/j.jaut.2021.102713
Hemed-Shaked, Maayan, et al. "MTADV 5-MER peptide suppresses chronic inflammations as well as autoimmune pathologies and unveils a new potential target-Serum Amyloid A." Journal of autoimmunity vol. 124 (2021): 102713. doi: https://doi.org/10.1016/j.jaut.2021.102713
Hemed-Shaked M, Cowman MK, Kim JR, Huang X, Chau E, Ovadia H, Amar KO, Eshkar-Sebban L, Melamed M, Lev LB, Kedar E, Armengol J, Alemany J, Beyth S, Okon E, Kanduc D, Elgavish S, Wallach-Dayan SB, Cohen SJ, Naor D. MTADV 5-MER peptide suppresses chronic inflammations as well as autoimmune pathologies and unveils a new potential target-Serum Amyloid A. J Autoimmun. 2021 Nov;124:102713. doi: 10.1016/j.jaut.2021.102713. Epub 2021 Aug 12. PMID: 34390919.
Copy Download .nbib Format: NLM
Slow Transcription of the 99a/let-7c/125b-2 Cluster Results in Differential MiRNA Expression and Promotes Melanoma Phenotypic Plasticity.

The Journal of investigative dermatology

Sheinboim D, Parikh S, Parikh R, Menuchin A, Shapira G, Kapitansky O, Elkoshi N, Ruppo S, Shaham L, Golan T, Elgavish S, Nevo Y, Bell RE, Malcov-Brog H, Shomron N, Taub JW, Izraeli S, Levy C.
PMID: 34186058
J Invest Dermatol. 2021 Dec;141(12):2944-2956.e6. doi: 10.1016/j.jid.2021.03.036. Epub 2021 Jun 26.

Almost half of the human microRNAs (miRNAs) are encoded in clusters. Although transcribed as a single unit, the levels of individual mature miRNAs often differ. The mechanisms underlying differential biogenesis of clustered miRNAs and the resulting physiological implications are...

Cite
Sheinboim D, Parikh S, Parikh R, et al. Slow Transcription of the 99a/let-7c/125b-2 Cluster Results in Differential MiRNA Expression and Promotes Melanoma Phenotypic Plasticity. J Invest Dermatol. 2021;141(12):2944-2956.e6doi: 10.1016/j.jid.2021.03.036.
Sheinboim, D., Parikh, S., Parikh, R., Menuchin, A., Shapira, G., Kapitansky, O., Elkoshi, N., Ruppo, S., Shaham, L., Golan, T., Elgavish, S., Nevo, Y., Bell, R. E., Malcov-Brog, H., Shomron, N., Taub, J. W., Izraeli, S., & Levy, C. (2021). Slow Transcription of the 99a/let-7c/125b-2 Cluster Results in Differential MiRNA Expression and Promotes Melanoma Phenotypic Plasticity. The Journal of investigative dermatology, 141(12), 2944-2956.e6. https://doi.org/10.1016/j.jid.2021.03.036
Sheinboim, Danna, et al. "Slow Transcription of the 99a/let-7c/125b-2 Cluster Results in Differential MiRNA Expression and Promotes Melanoma Phenotypic Plasticity." The Journal of investigative dermatology vol. 141,12 (2021): 2944-2956.e6. doi: https://doi.org/10.1016/j.jid.2021.03.036
Sheinboim D, Parikh S, Parikh R, Menuchin A, Shapira G, Kapitansky O, Elkoshi N, Ruppo S, Shaham L, Golan T, Elgavish S, Nevo Y, Bell RE, Malcov-Brog H, Shomron N, Taub JW, Izraeli S, Levy C. Slow Transcription of the 99a/let-7c/125b-2 Cluster Results in Differential MiRNA Expression and Promotes Melanoma Phenotypic Plasticity. J Invest Dermatol. 2021 Dec;141(12):2944-2956.e6. doi: 10.1016/j.jid.2021.03.036. Epub 2021 Jun 26. PMID: 34186058.
Copy Download .nbib Format: NLM
Genome Sequence of the Pathogenic Bacterium Vibrio vulnificus Biotype 3.

Genome announcements

Danin-Poleg Y, Elgavish S, Raz N, Efimov V, Kashi Y.
PMID: 23599289
Genome Announc. 2013 Apr 18;1(2):e0013613. doi: 10.1128/genomeA.00136-13.

We report the first genome sequence of the pathogenic Vibrio vulnificus biotype 3. This draft genome sequence of the environmental strain VVyb1(BT3), isolated in Israel, provides a representation of this newly emerged clonal group, which reveals higher similarity to...

Cite
Danin-Poleg Y, Elgavish S, Raz N, et al. Genome Sequence of the Pathogenic Bacterium Vibrio vulnificus Biotype 3. Genome Announc. 2013;1(2):e0013613doi: 10.1128/genomeA.00136-13.
Danin-Poleg, Y., Elgavish, S., Raz, N., Efimov, V., & Kashi, Y. (2013). Genome Sequence of the Pathogenic Bacterium Vibrio vulnificus Biotype 3. Genome announcements, 1(2), e0013613. https://doi.org/10.1128/genomeA.00136-13
Danin-Poleg, Yael, et al. "Genome Sequence of the Pathogenic Bacterium Vibrio vulnificus Biotype 3." Genome announcements vol. 1,2 (2013): e0013613. doi: https://doi.org/10.1128/genomeA.00136-13
Danin-Poleg Y, Elgavish S, Raz N, Efimov V, Kashi Y. Genome Sequence of the Pathogenic Bacterium Vibrio vulnificus Biotype 3. Genome Announc. 2013 Apr 18;1(2):e0013613. doi: 10.1128/genomeA.00136-13. PMID: 23599289; PMCID: PMC3630400.
Copy Download .nbib Format: NLM
Draft Genome Sequence of Lactobacillus johnsonii Strain 16, Isolated from Mice.

Genome announcements

Buhnik-Rosenblau K, Danin-Poleg Y, Elgavish S, Kashi Y.
PMID: 26450724
Genome Announc. 2015 Oct 08;3(5). doi: 10.1128/genomeA.01141-15.

Here, we report the genome sequence of Lactobacillus johnsonii, a member of the gut lactobacilli. This draft genome of L. johnsonii strain 16 isolated from C57BL/6J mice enables the identification of bacterial genes responsible for host-specific gut persistence.

Cite
Buhnik-Rosenblau K, Danin-Poleg Y, Elgavish S, et al. Draft Genome Sequence of Lactobacillus johnsonii Strain 16, Isolated from Mice. Genome Announc. 2015;3(5)doi: 10.1128/genomeA.01141-15.
Buhnik-Rosenblau, K., Danin-Poleg, Y., Elgavish, S., & Kashi, Y. (2015). Draft Genome Sequence of Lactobacillus johnsonii Strain 16, Isolated from Mice. Genome announcements, 3(5), . https://doi.org/10.1128/genomeA.01141-15
Buhnik-Rosenblau, Keren, et al. "Draft Genome Sequence of Lactobacillus johnsonii Strain 16, Isolated from Mice." Genome announcements vol. 3,5 (2015). doi: https://doi.org/10.1128/genomeA.01141-15
Buhnik-Rosenblau K, Danin-Poleg Y, Elgavish S, Kashi Y. Draft Genome Sequence of Lactobacillus johnsonii Strain 16, Isolated from Mice. Genome Announc. 2015 Oct 08;3(5). doi: 10.1128/genomeA.01141-15. PMID: 26450724; PMCID: PMC4599083.
Copy Download .nbib Format: NLM
Correction: Early age decline in DNA repair capacity in the liver: in depth profile of differential gene expression.

Aging

Guedj A, Geiger-Maor A, Benyamini H, Nevo Y, Elgavish S, Galun E, Amsalem H, Rachmilewitz J.
PMID: 28687000
Aging (Albany NY). 2017 May 30;9(6):1640. doi: 10.18632/aging.101242.

No abstract available.

Cite
Guedj A, Geiger-Maor A, Benyamini H, et al. Correction: Early age decline in DNA repair capacity in the liver: in depth profile of differential gene expression. Aging (Albany NY). 2017;9(6):1640doi: 10.18632/aging.101242.
Guedj, A., Geiger-Maor, A., Benyamini, H., Nevo, Y., Elgavish, S., Galun, E., Amsalem, H., & Rachmilewitz, J. (2017). Correction: Early age decline in DNA repair capacity in the liver: in depth profile of differential gene expression. Aging, 9(6), 1640. https://doi.org/10.18632/aging.101242
Guedj, Avital, et al. "Correction: Early age decline in DNA repair capacity in the liver: in depth profile of differential gene expression." Aging vol. 9,6 (2017): 1640. doi: https://doi.org/10.18632/aging.101242
Guedj A, Geiger-Maor A, Benyamini H, Nevo Y, Elgavish S, Galun E, Amsalem H, Rachmilewitz J. Correction: Early age decline in DNA repair capacity in the liver: in depth profile of differential gene expression. Aging (Albany NY). 2017 May 30;9(6):1640. doi: 10.18632/aging.101242. PMID: 28687000; PMCID: PMC5509461.
Copy Download .nbib Format: NLM
The MRI characteristics of the no-flow region are similar in reperfused and non-reperfused myocardial infarcts: an MRI and histopathology study in swine.

European radiology experimental

Elgavish GA, Simor T, van der Geest RJ, Suranyi P, Kiss PP, Lenkey Z, Kirschner R, Wang D, Brott BC, Varga-Szemes A.
PMID: 29708171
Eur Radiol Exp. 2017;1(1):2. doi: 10.1186/s41747-017-0001-x. Epub 2017 Jun 29.

BACKGROUND: The no-flow region (NF) visualised by magnetic resonance imaging (MRI) in myocardial infarction (MI) has been explained as the product of reperfusion-injury-induced microvascular obstruction. However, a similar MRI phenomenon occurs in non-reperfused MI. Accordingly, our purpose was to...

Cite
Elgavish GA, Simor T, van der Geest RJ, et al. The MRI characteristics of the no-flow region are similar in reperfused and non-reperfused myocardial infarcts: an MRI and histopathology study in swine. Eur Radiol Exp. 2017;1(1):2doi: 10.1186/s41747-017-0001-x.
Elgavish, G. A., Simor, T., van der Geest, R. J., Suranyi, P., Kiss, P. P., Lenkey, Z., Kirschner, R., Wang, D., Brott, B. C., & Varga-Szemes, A. (2017). The MRI characteristics of the no-flow region are similar in reperfused and non-reperfused myocardial infarcts: an MRI and histopathology study in swine. European radiology experimental, 1(1), 2. https://doi.org/10.1186/s41747-017-0001-x
Elgavish, Gabriel A, et al. "The MRI characteristics of the no-flow region are similar in reperfused and non-reperfused myocardial infarcts: an MRI and histopathology study in swine." European radiology experimental vol. 1,1 (2017): 2. doi: https://doi.org/10.1186/s41747-017-0001-x
Elgavish GA, Simor T, van der Geest RJ, Suranyi P, Kiss PP, Lenkey Z, Kirschner R, Wang D, Brott BC, Varga-Szemes A. The MRI characteristics of the no-flow region are similar in reperfused and non-reperfused myocardial infarcts: an MRI and histopathology study in swine. Eur Radiol Exp. 2017;1(1):2. doi: 10.1186/s41747-017-0001-x. Epub 2017 Jun 29. PMID: 29708171; PMCID: PMC5909333.
Copy Download .nbib Format: NLM
Myocardial tissue characterization by combining late gadolinium enhancement imaging and percent edema mapping: a novel T2 map-based MRI method in canine myocardial infarction.

European radiology experimental

Suranyi P, Elgavish GA, Schoepf UJ, Ruzsics B, Kiss P, van Assen M, Jacobs BE, Brott BC, Elgavish A, Varga-Szemes A.
PMID: 29708212
Eur Radiol Exp. 2018;2(1):6. doi: 10.1186/s41747-018-0037-6. Epub 2018 Mar 16.

BACKGROUND: Assessing the extent of ischemic and reperfusion-associated myocardial injuries remains challenging with current magnetic resonance imaging (MRI) techniques. Our aim was to develop a tissue characterization mapping (TCM) technique by combining late gadolinium enhancement (LGE) with our novel...

Cite
Suranyi P, Elgavish GA, Schoepf UJ, et al. Myocardial tissue characterization by combining late gadolinium enhancement imaging and percent edema mapping: a novel T2 map-based MRI method in canine myocardial infarction. Eur Radiol Exp. 2018;2(1):6doi: 10.1186/s41747-018-0037-6.
Suranyi, P., Elgavish, G. A., Schoepf, U. J., Ruzsics, B., Kiss, P., van Assen, M., Jacobs, B. E., Brott, B. C., Elgavish, A., & Varga-Szemes, A. (2018). Myocardial tissue characterization by combining late gadolinium enhancement imaging and percent edema mapping: a novel T2 map-based MRI method in canine myocardial infarction. European radiology experimental, 2(1), 6. https://doi.org/10.1186/s41747-018-0037-6
Suranyi, Pal, et al. "Myocardial tissue characterization by combining late gadolinium enhancement imaging and percent edema mapping: a novel T2 map-based MRI method in canine myocardial infarction." European radiology experimental vol. 2,1 (2018): 6. doi: https://doi.org/10.1186/s41747-018-0037-6
Suranyi P, Elgavish GA, Schoepf UJ, Ruzsics B, Kiss P, van Assen M, Jacobs BE, Brott BC, Elgavish A, Varga-Szemes A. Myocardial tissue characterization by combining late gadolinium enhancement imaging and percent edema mapping: a novel T2 map-based MRI method in canine myocardial infarction. Eur Radiol Exp. 2018;2(1):6. doi: 10.1186/s41747-018-0037-6. Epub 2018 Mar 16. PMID: 29708212; PMCID: PMC5909369.
Copy Download .nbib Format: NLM
Melilotus indicus extract induces apoptosis in hepatocellular carcinoma cells via a mechanism involving mitochondria-mediated pathways.

Cytotechnology

Abd El-Hafeez AA, Khalifa HO, Elgawish RA, Shouman SA, Abd El-Twab MH, Kawamoto S.
PMID: 29372465
Cytotechnology. 2018 Apr;70(2):831-842. doi: 10.1007/s10616-018-0195-7. Epub 2018 Jan 25.

Melilotus indicus, is a traditional medicine used as analgesic and emollient. Although Melilotus indicus extract (MIE) has recently been shown to suppress growth of several tumor cell lines, information regarding its antitumor mechanism is completely unknown. Here, we report...

Cite
Abd El-Hafeez AA, Khalifa HO, Elgawish RA, et al. Melilotus indicus extract induces apoptosis in hepatocellular carcinoma cells via a mechanism involving mitochondria-mediated pathways. Cytotechnology. 2018;70(2):831-842doi: 10.1007/s10616-018-0195-7.
Abd El-Hafeez, A. A., Khalifa, H. O., Elgawish, R. A., Shouman, S. A., Abd El-Twab, M. H., & Kawamoto, S. (2018). Melilotus indicus extract induces apoptosis in hepatocellular carcinoma cells via a mechanism involving mitochondria-mediated pathways. Cytotechnology, 70(2), 831-842. https://doi.org/10.1007/s10616-018-0195-7
Abd El-Hafeez, Amer Ali, et al. "Melilotus indicus extract induces apoptosis in hepatocellular carcinoma cells via a mechanism involving mitochondria-mediated pathways." Cytotechnology vol. 70,2 (2018): 831-842. doi: https://doi.org/10.1007/s10616-018-0195-7
Abd El-Hafeez AA, Khalifa HO, Elgawish RA, Shouman SA, Abd El-Twab MH, Kawamoto S. Melilotus indicus extract induces apoptosis in hepatocellular carcinoma cells via a mechanism involving mitochondria-mediated pathways. Cytotechnology. 2018 Apr;70(2):831-842. doi: 10.1007/s10616-018-0195-7. Epub 2018 Jan 25. PMID: 29372465; PMCID: PMC5851975.
Copy Download .nbib Format: NLM
Showing 1 to 12 of 19 entries