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[Patients are pleased to be answer questions].

Lakartidningen

Ehrenpreis J.
PMID: 8531520
Lakartidningen. 1995 Dec 06;92(49):4674.

No abstract available.

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Ehrenpreis J. Patienten blir glad att man frågar. [Patients are pleased to be answer questions]. Lakartidningen. 1995;92(49):4674
Ehrenpreis, J. (1995). Patienten blir glad att man frågar. [Patients are pleased to be answer questions]. Lakartidningen, 92(49), 4674.
Ehrenpreis, J. "Patienten blir glad att man frågar." [Patients are pleased to be answer questions]. Lakartidningen vol. 92,49 (1995): 4674.
Ehrenpreis J. Patienten blir glad att man frågar. [Patients are pleased to be answer questions]. Lakartidningen. 1995 Dec 06;92(49):4674. Swedish. PMID: 8531520.
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Mitotic death: a mechanism of survival? A review.

Cancer cell international

Erenpreisa J, Cragg MS.
PMID: 11983025
Cancer Cell Int. 2001 Nov 23;1(1):1. doi: 10.1186/1475-2867-1-1.

Mitotic death is a delayed response of p53 mutant tumours that are resistant to genotoxic damage. Questions surround why this response is so delayed and how its mechanisms serve a survival function. After uncoupling apoptosis from G1 and S...

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Erenpreisa J, Cragg MS. Mitotic death: a mechanism of survival? A review. Cancer Cell Int. 2001;1(1):1doi: 10.1186/1475-2867-1-1.
Erenpreisa, J., & Cragg, M. S. (2001). Mitotic death: a mechanism of survival? A review. Cancer cell international, 1(1), 1. https://doi.org/10.1186/1475-2867-1-1
Erenpreisa, Jekaterina, and Cragg, M S. "Mitotic death: a mechanism of survival? A review." Cancer cell international vol. 1,1 (2001): 1. doi: https://doi.org/10.1186/1475-2867-1-1
Erenpreisa J, Cragg MS. Mitotic death: a mechanism of survival? A review. Cancer Cell Int. 2001 Nov 23;1(1):1. doi: 10.1186/1475-2867-1-1. PMID: 11983025; PMCID: PMC101225.
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A New Baltic Population-Specific Human Genetic Marker in the PMCA4 Gene.

Human heredity

Stavusis J, Inashkina I, Lace B, Pelnena D, Limborska S, Khrunin A, Kucinskas V, Krumina A, Piekuse L, Zorn B, Fodina V, Punab M, Erenpreiss J.
PMID: 29131013
Hum Hered. 2016;82(3):140-146. doi: 10.1159/000481434. Epub 2017 Nov 02.

OBJECTIVES: The PMCA gene family consists of 4 genes and at least 21 splice variants; among these, the Ca2+ ATPase 4 (PMCA4) gene encodes a plasma membrane protein abundantly expressed in several tissues, including the kidney, heart, and sperm....

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Stavusis J, Inashkina I, Lace B, et al. A New Baltic Population-Specific Human Genetic Marker in the PMCA4 Gene. Hum Hered. 2017;82(3):140-146doi: 10.1159/000481434.
Stavusis, J., Inashkina, I., Lace, B., Pelnena, D., Limborska, S., Khrunin, A., Kucinskas, V., Krumina, A., Piekuse, L., Zorn, B., Fodina, V., Punab, M., & Erenpreiss, J. (2016). A New Baltic Population-Specific Human Genetic Marker in the PMCA4 Gene. Human heredity, 82(3), 140-146. https://doi.org/10.1159/000481434
Stavusis, Janis, et al. "A New Baltic Population-Specific Human Genetic Marker in the PMCA4 Gene." Human heredity vol. 82,3 (2016): 140-146. doi: https://doi.org/10.1159/000481434
Stavusis J, Inashkina I, Lace B, Pelnena D, Limborska S, Khrunin A, Kucinskas V, Krumina A, Piekuse L, Zorn B, Fodina V, Punab M, Erenpreiss J. A New Baltic Population-Specific Human Genetic Marker in the PMCA4 Gene. Hum Hered. 2016;82(3):140-146. doi: 10.1159/000481434. Epub 2017 Nov 02. PMID: 29131013.
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Evaluation of embryo aneuploidy (PGT-A) and endometrial receptivity (ERA) testing in patients with recurrent implantation failure in ICSI cycles.

Gynecological endocrinology : the official journal of the International Society of Gynecological Endocrinology

Fodina V, Dudorova A, Erenpreiss J.
PMID: 34937515
Gynecol Endocrinol. 2021;37:17-20. doi: 10.1080/09513590.2021.2006466.

OBJECTIVE: The objective of a study was to assess the ability of the pre-implantation genetic testing of embryos for aneuploidy (PGT-A) and Endometrial receptivity array (ERA)-alone or in combination to improve the clinical outcomes in intracytoplasmic sperm injection (ICSI)...

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Fodina V, Dudorova A, Erenpreiss J. Evaluation of embryo aneuploidy (PGT-A) and endometrial receptivity (ERA) testing in patients with recurrent implantation failure in ICSI cycles. Gynecol Endocrinol. 2021;37:17-20doi: 10.1080/09513590.2021.2006466.
Fodina, V., Dudorova, A., & Erenpreiss, J. (2021). Evaluation of embryo aneuploidy (PGT-A) and endometrial receptivity (ERA) testing in patients with recurrent implantation failure in ICSI cycles. Gynecological endocrinology : the official journal of the International Society of Gynecological Endocrinology, 3717-20. https://doi.org/10.1080/09513590.2021.2006466
Fodina, Violeta, et al. "Evaluation of embryo aneuploidy (PGT-A) and endometrial receptivity (ERA) testing in patients with recurrent implantation failure in ICSI cycles." Gynecological endocrinology : the official journal of the International Society of Gynecological Endocrinology vol. 37 (2021): 17-20. doi: https://doi.org/10.1080/09513590.2021.2006466
Fodina V, Dudorova A, Erenpreiss J. Evaluation of embryo aneuploidy (PGT-A) and endometrial receptivity (ERA) testing in patients with recurrent implantation failure in ICSI cycles. Gynecol Endocrinol. 2021;37:17-20. doi: 10.1080/09513590.2021.2006466. PMID: 34937515.
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Case of Inherited Partial AZFa Deletion without Impact on Male Fertility.

Case reports in genetics

Alksere B, Berzina D, Dudorova A, Conka U, Andersone S, Pimane E, Krasucka S, Blumberga A, Dzalbs A, Grinfelde I, Vedmedovska N, Fodina V, Erenpreiss J.
PMID: 31781421
Case Rep Genet. 2019 Oct 31;2019:3802613. doi: 10.1155/2019/3802613. eCollection 2019.

Male factor infertility accounts for 40-50% of all infertility cases. Deletions of one or more AZF region parts in chromosome Y are one of the most common genetic causes of male infertility. Usually full or partial AZF deletions, including...

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Alksere B, Berzina D, Dudorova A, et al. Case of Inherited Partial AZFa Deletion without Impact on Male Fertility. Case Rep Genet. 2019;2019:3802613doi: 10.1155/2019/3802613.
Alksere, B., Berzina, D., Dudorova, A., Conka, U., Andersone, S., Pimane, E., Krasucka, S., Blumberga, A., Dzalbs, A., Grinfelde, I., Vedmedovska, N., Fodina, V., & Erenpreiss, J. (2019). Case of Inherited Partial AZFa Deletion without Impact on Male Fertility. Case reports in genetics, 20193802613. https://doi.org/10.1155/2019/3802613
Alksere, Baiba, et al. "Case of Inherited Partial AZFa Deletion without Impact on Male Fertility." Case reports in genetics vol. 2019 (2019): 3802613. doi: https://doi.org/10.1155/2019/3802613
Alksere B, Berzina D, Dudorova A, Conka U, Andersone S, Pimane E, Krasucka S, Blumberga A, Dzalbs A, Grinfelde I, Vedmedovska N, Fodina V, Erenpreiss J. Case of Inherited Partial AZFa Deletion without Impact on Male Fertility. Case Rep Genet. 2019 Oct 31;2019:3802613. doi: 10.1155/2019/3802613. eCollection 2019. PMID: 31781421; PMCID: PMC6874946.
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Erratum to: Disentangling the aneuploidy and senescence paradoxes: a study of triploid breast cancers non-responsive to neoadjuvant therapy.

Histochemistry and cell biology

Gerashchenko BI, Salmina K, Eglitis J, Huna A, Grjunberga V, Erenpreisa J.
PMID: 26922948
Histochem Cell Biol. 2016 Apr;145(4):509. doi: 10.1007/s00418-016-1423-x.

No abstract available.

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Gerashchenko BI, Salmina K, Eglitis J, et al. Erratum to: Disentangling the aneuploidy and senescence paradoxes: a study of triploid breast cancers non-responsive to neoadjuvant therapy. Histochem Cell Biol. 2016;145(4):509doi: 10.1007/s00418-016-1423-x.
Gerashchenko, B. I., Salmina, K., Eglitis, J., Huna, A., Grjunberga, V., & Erenpreisa, J. (2016). Erratum to: Disentangling the aneuploidy and senescence paradoxes: a study of triploid breast cancers non-responsive to neoadjuvant therapy. Histochemistry and cell biology, 145(4), 509. https://doi.org/10.1007/s00418-016-1423-x
Gerashchenko, B I, et al. "Erratum to: Disentangling the aneuploidy and senescence paradoxes: a study of triploid breast cancers non-responsive to neoadjuvant therapy." Histochemistry and cell biology vol. 145,4 (2016): 509. doi: https://doi.org/10.1007/s00418-016-1423-x
Gerashchenko BI, Salmina K, Eglitis J, Huna A, Grjunberga V, Erenpreisa J. Erratum to: Disentangling the aneuploidy and senescence paradoxes: a study of triploid breast cancers non-responsive to neoadjuvant therapy. Histochem Cell Biol. 2016 Apr;145(4):509. doi: 10.1007/s00418-016-1423-x. PMID: 26922948.
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The "virgin birth", polyploidy, and the origin of cancer.

Oncoscience

Erenpreisa J, Salmina K, Huna A, Jackson TR, Vazquez-Martin A, Cragg MS.
PMID: 25821840
Oncoscience. 2014 Dec 17;2(1):3-14. doi: 10.18632/oncoscience.108. eCollection 2015.

Recently, it has become clear that the complexity of cancer biology cannot fully be explained by somatic mutation and clonal selection. Meanwhile, data have accumulated on how cancer stem cells or stemloids bestow immortality on tumour cells and how...

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Erenpreisa J, Salmina K, Huna A, et al. The "virgin birth", polyploidy, and the origin of cancer. Oncoscience. 2014;2(1):3-14doi: 10.18632/oncoscience.108.
Erenpreisa, J., Salmina, K., Huna, A., Jackson, T. R., Vazquez-Martin, A., & Cragg, M. S. (2015). The "virgin birth", polyploidy, and the origin of cancer. Oncoscience, 2(1), 3-14. https://doi.org/10.18632/oncoscience.108
Erenpreisa, Jekaterina, et al. "The "virgin birth", polyploidy, and the origin of cancer." Oncoscience vol. 2,1 (2015): 3-14. doi: https://doi.org/10.18632/oncoscience.108
Erenpreisa J, Salmina K, Huna A, Jackson TR, Vazquez-Martin A, Cragg MS. The "virgin birth", polyploidy, and the origin of cancer. Oncoscience. 2014 Dec 17;2(1):3-14. doi: 10.18632/oncoscience.108. eCollection 2015. PMID: 25821840; PMCID: PMC4341460.
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A novel .

Molecular genetics and metabolism reports

Alksere B, Kornejeva L, Grinfelde I, Dzalbs A, Enkure D, Conka U, Andersone S, Blumberga A, Nikitina-Zake L, Kangare L, Radovica-Spalvina I, Vasiljeva I, Gailite L, Erenpreiss J, Fodina V.
PMID: 34584847
Mol Genet Metab Rep. 2021 Sep 20;29:100796. doi: 10.1016/j.ymgmr.2021.100796. eCollection 2021 Dec.

Hereditary ectodermal dysplasias are a complex group of inherited disorders characterised by abnormalities in two or more ectodermal derivatives (skin, nails, sweat glands,

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Alksere B, Kornejeva L, Grinfelde I, et al. A novel . Mol Genet Metab Rep. 2021;29:100796doi: 10.1016/j.ymgmr.2021.100796.
Alksere, B., Kornejeva, L., Grinfelde, I., Dzalbs, A., Enkure, D., Conka, U., Andersone, S., Blumberga, A., Nikitina-Zake, L., Kangare, L., Radovica-Spalvina, I., Vasiljeva, I., Gailite, L., Erenpreiss, J., & Fodina, V. (2021). A novel . Molecular genetics and metabolism reports, 29100796. https://doi.org/10.1016/j.ymgmr.2021.100796
Alksere, Baiba, et al. "A novel ." Molecular genetics and metabolism reports vol. 29 (2021): 100796. doi: https://doi.org/10.1016/j.ymgmr.2021.100796
Alksere B, Kornejeva L, Grinfelde I, Dzalbs A, Enkure D, Conka U, Andersone S, Blumberga A, Nikitina-Zake L, Kangare L, Radovica-Spalvina I, Vasiljeva I, Gailite L, Erenpreiss J, Fodina V. A novel . Mol Genet Metab Rep. 2021 Sep 20;29:100796. doi: 10.1016/j.ymgmr.2021.100796. eCollection 2021 Dec. PMID: 34584847; PMCID: PMC8453221.
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Self-Renewal Signalling in Presenescent Tetraploid IMR90 Cells.

Journal of aging research

Huna A, Salmina K, Jascenko E, Duburs G, Inashkina I, Erenpreisa J.
PMID: 21629737
J Aging Res. 2011;2011:103253. doi: 10.4061/2011/103253. Epub 2011 May 11.

Endopolyploidy and genomic instability are shared features of both stress-induced cellular senescence and malignant growth. Here, we examined these facets in the widely used normal human fibroblast model of senescence, IMR90. At the presenescence stage, a small (2-7%) proportion...

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Huna A, Salmina K, Jascenko E, et al. Self-Renewal Signalling in Presenescent Tetraploid IMR90 Cells. J Aging Res. 2011;2011:103253doi: 10.4061/2011/103253.
Huna, A., Salmina, K., Jascenko, E., Duburs, G., Inashkina, I., & Erenpreisa, J. (2011). Self-Renewal Signalling in Presenescent Tetraploid IMR90 Cells. Journal of aging research, 2011103253. https://doi.org/10.4061/2011/103253
Huna, Anda, et al. "Self-Renewal Signalling in Presenescent Tetraploid IMR90 Cells." Journal of aging research vol. 2011 (2011): 103253. doi: https://doi.org/10.4061/2011/103253
Huna A, Salmina K, Jascenko E, Duburs G, Inashkina I, Erenpreisa J. Self-Renewal Signalling in Presenescent Tetraploid IMR90 Cells. J Aging Res. 2011;2011:103253. doi: 10.4061/2011/103253. Epub 2011 May 11. PMID: 21629737; PMCID: PMC3100543.
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Cancer/testis antigens and gametogenesis: a review and "brain-storming" session.

Cancer cell international

Kalejs M, Erenpreisa J.
PMID: 15715909
Cancer Cell Int. 2005 Feb 16;5(1):4. doi: 10.1186/1475-2867-5-4.

Genes expressed both in normal testis and in malignancies (Cancer/ Testis associated genes - CTA) have become the most extensively studied antigen group in the field of tumour immunology. Despite this, many fundamentally important questions remain unanswered: what is...

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Kalejs M, Erenpreisa J. Cancer/testis antigens and gametogenesis: a review and "brain-storming" session. Cancer Cell Int. 2005;5(1):4doi: 10.1186/1475-2867-5-4.
Kalejs, M., & Erenpreisa, J. (2005). Cancer/testis antigens and gametogenesis: a review and "brain-storming" session. Cancer cell international, 5(1), 4. https://doi.org/10.1186/1475-2867-5-4
Kalejs, Martins, and Erenpreisa, Jekaterina. "Cancer/testis antigens and gametogenesis: a review and "brain-storming" session." Cancer cell international vol. 5,1 (2005): 4. doi: https://doi.org/10.1186/1475-2867-5-4
Kalejs M, Erenpreisa J. Cancer/testis antigens and gametogenesis: a review and "brain-storming" session. Cancer Cell Int. 2005 Feb 16;5(1):4. doi: 10.1186/1475-2867-5-4. PMID: 15715909; PMCID: PMC552320.
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Three steps to the immortality of cancer cells: senescence, polyploidy and self-renewal.

Cancer cell international

Erenpreisa J, Cragg MS.
PMID: 24025698
Cancer Cell Int. 2013 Sep 11;13(1):92. doi: 10.1186/1475-2867-13-92.

Metastatic cancer is rarely cured by current DNA damaging treatments, apparently due to the development of resistance. However, recent data indicates that tumour cells can elicit the opposing processes of senescence and stemness in response to these treatments, the...

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Erenpreisa J, Cragg MS. Three steps to the immortality of cancer cells: senescence, polyploidy and self-renewal. Cancer Cell Int. 2013;13(1):92doi: 10.1186/1475-2867-13-92.
Erenpreisa, J., & Cragg, M. S. (2013). Three steps to the immortality of cancer cells: senescence, polyploidy and self-renewal. Cancer cell international, 13(1), 92. https://doi.org/10.1186/1475-2867-13-92
Erenpreisa, Jekaterina, and Cragg, Mark S. "Three steps to the immortality of cancer cells: senescence, polyploidy and self-renewal." Cancer cell international vol. 13,1 (2013): 92. doi: https://doi.org/10.1186/1475-2867-13-92
Erenpreisa J, Cragg MS. Three steps to the immortality of cancer cells: senescence, polyploidy and self-renewal. Cancer Cell Int. 2013 Sep 11;13(1):92. doi: 10.1186/1475-2867-13-92. PMID: 24025698; PMCID: PMC4015969.
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Idiopathic Infertility as a Feature of Genome Instability.

Life (Basel, Switzerland)

Puzuka A, Alksere B, Gailite L, Erenpreiss J.
PMID: 34209597
Life (Basel). 2021 Jun 29;11(7). doi: 10.3390/life11070628.

Genome instability may play a role in severe cases of male infertility, with disrupted spermatogenesis being just one manifestation of decreased general health and increased morbidity. Here, we review the data on the association of male infertility with genetic,...

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Puzuka A, Alksere B, Gailite L, et al. Idiopathic Infertility as a Feature of Genome Instability. Life (Basel). 2021;11(7)doi: 10.3390/life11070628.
Puzuka, A., Alksere, B., Gailite, L., & Erenpreiss, J. (2021). Idiopathic Infertility as a Feature of Genome Instability. Life (Basel, Switzerland), 11(7), . https://doi.org/10.3390/life11070628
Puzuka, Agrita, et al. "Idiopathic Infertility as a Feature of Genome Instability." Life (Basel, Switzerland) vol. 11,7 (2021). doi: https://doi.org/10.3390/life11070628
Puzuka A, Alksere B, Gailite L, Erenpreiss J. Idiopathic Infertility as a Feature of Genome Instability. Life (Basel). 2021 Jun 29;11(7). doi: 10.3390/life11070628. PMID: 34209597; PMCID: PMC8307193.
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