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Showing 1 to 8 of 8 entries
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Combined Plasma and Cerebrospinal Fluid Signature for the Prediction of Midterm Progression From Mild Cognitive Impairment to Alzheimer Disease.

JAMA neurology

Lehallier B, Essioux L, Gayan J, Alexandridis R, Nikolcheva T, Wyss-Coray T, Britschgi M.
PMID: 26659895
JAMA Neurol. 2016 Feb;73(2):203-212. doi: 10.1001/jamaneurol.2015.3135. Epub 2015 Dec 14.

IMPORTANCE: A reliable method of detecting Alzheimer disease (AD) in its prodromal state is needed for patient stratification in clinical trials or for personalizing existing or potential upcoming therapies. Current cerebrospinal fluid (CSF)- or imaging-based single biomarkers for AD...

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Lehallier B, Essioux L, Gayan J, et al. Combined Plasma and Cerebrospinal Fluid Signature for the Prediction of Midterm Progression From Mild Cognitive Impairment to Alzheimer Disease. JAMA Neurol. 2015;73(2):203-212doi: 10.1001/jamaneurol.2015.3135.
Lehallier, B., Essioux, L., Gayan, J., Alexandridis, R., Nikolcheva, T., Wyss-Coray, T., Britschgi, M. (2016). Combined Plasma and Cerebrospinal Fluid Signature for the Prediction of Midterm Progression From Mild Cognitive Impairment to Alzheimer Disease. JAMA neurology, 73(2), 203-212. https://doi.org/10.1001/jamaneurol.2015.3135
Lehallier, Benoit, et al. "Combined Plasma and Cerebrospinal Fluid Signature for the Prediction of Midterm Progression From Mild Cognitive Impairment to Alzheimer Disease." JAMA neurology vol. 73,2 (2016): 203-212. doi: https://doi.org/10.1001/jamaneurol.2015.3135
Lehallier B, Essioux L, Gayan J, Alexandridis R, Nikolcheva T, Wyss-Coray T, Britschgi M. Combined Plasma and Cerebrospinal Fluid Signature for the Prediction of Midterm Progression From Mild Cognitive Impairment to Alzheimer Disease. JAMA Neurol. 2016 Feb;73(2):203-212. doi: 10.1001/jamaneurol.2015.3135. Epub 2015 Dec 14. PMID: 26659895; PMCID: PMC5214993.
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Drug Target Identification and Validation: Global Pharmaceutical Industry Experts on Challenges, Best Strategies, Innovative Precompetitive Collaboration Concepts, and Future Areas of Industry Precompetitive Research and Development.

Therapeutic innovation & regulatory science

Bergauer T, Ruppert T, Essioux L, Spleiss O.
PMID: 30231745
Ther Innov Regul Sci. 2016 Nov;50(6):769-776. doi: 10.1177/2168479016651298. Epub 2016 Jul 10.

Focused interviews were conducted with global pharmaceutical company representatives in order to derive a consistent view on drug target identification/validation challenges, collaborative strategies, and future developments in a precompetitive space. Analysis revealed translation into clinical utility as a major...

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Bergauer T, Ruppert T, Essioux L, et al. Drug Target Identification and Validation: Global Pharmaceutical Industry Experts on Challenges, Best Strategies, Innovative Precompetitive Collaboration Concepts, and Future Areas of Industry Precompetitive Research and Development. Ther Innov Regul Sci. 2016;50(6):769-776doi: 10.1177/2168479016651298.
Bergauer, T., Ruppert, T., Essioux, L., & Spleiss, O. (2016). Drug Target Identification and Validation: Global Pharmaceutical Industry Experts on Challenges, Best Strategies, Innovative Precompetitive Collaboration Concepts, and Future Areas of Industry Precompetitive Research and Development. Therapeutic innovation & regulatory science, 50(6), 769-776. https://doi.org/10.1177/2168479016651298
Bergauer, Tobias, et al. "Drug Target Identification and Validation: Global Pharmaceutical Industry Experts on Challenges, Best Strategies, Innovative Precompetitive Collaboration Concepts, and Future Areas of Industry Precompetitive Research and Development." Therapeutic innovation & regulatory science vol. 50,6 (2016): 769-776. doi: https://doi.org/10.1177/2168479016651298
Bergauer T, Ruppert T, Essioux L, Spleiss O. Drug Target Identification and Validation: Global Pharmaceutical Industry Experts on Challenges, Best Strategies, Innovative Precompetitive Collaboration Concepts, and Future Areas of Industry Precompetitive Research and Development. Ther Innov Regul Sci. 2016 Nov;50(6):769-776. doi: 10.1177/2168479016651298. Epub 2016 Jul 10. PMID: 30231745.
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A high-pressure flow through test vessel for neutron imaging and neutron diffraction-based strain measurement of geological materials.

The Review of scientific instruments

Carmichael JR, Polsky Y, An K, Anovitz LM, Bingham P, Dessieux L, Ekici K, Frost M, Pemberton S.
PMID: 32872895
Rev Sci Instrum. 2020 Aug 01;91(8):084502. doi: 10.1063/5.0013433.

Neutron scattering and neutron imaging have emerged as powerful methods for experimentally investigating material deformation and fluid flow in the interior of otherwise inaccessible or opaque structures. This paper describes the design and provides example uses of a pressure...

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Carmichael JR, Polsky Y, An K, et al. A high-pressure flow through test vessel for neutron imaging and neutron diffraction-based strain measurement of geological materials. Rev Sci Instrum. 2020;91(8):084502doi: 10.1063/5.0013433.
Carmichael, J. R., Polsky, Y., An, K., Anovitz, L. M., Bingham, P., Dessieux, L., Ekici, K., Frost, M., & Pemberton, S. (2020). A high-pressure flow through test vessel for neutron imaging and neutron diffraction-based strain measurement of geological materials. The Review of scientific instruments, 91(8), 084502. https://doi.org/10.1063/5.0013433
Carmichael, J R, et al. "A high-pressure flow through test vessel for neutron imaging and neutron diffraction-based strain measurement of geological materials." The Review of scientific instruments vol. 91,8 (2020): 084502. doi: https://doi.org/10.1063/5.0013433
Carmichael JR, Polsky Y, An K, Anovitz LM, Bingham P, Dessieux L, Ekici K, Frost M, Pemberton S. A high-pressure flow through test vessel for neutron imaging and neutron diffraction-based strain measurement of geological materials. Rev Sci Instrum. 2020 Aug 01;91(8):084502. doi: 10.1063/5.0013433. PMID: 32872895.
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Linkage analyses of 3 French families to Loci on chromosome-2p and chromosome-3p predisposing to hereditary nonpolyposis colon-cancer.

International journal of oncology

Bernardgallon D, Ralliere C, Essioux L, Gosse S, Cure H, Bay J, Schraub S, Bonaitipellie C, Sobol H, Bignon Y.
PMID: 21556592
Int J Oncol. 1995 Mar;6(3):699-703. doi: 10.3892/ijo.6.3.699.

Hereditary, non-polyposis colon cancer (HNPCC) is caused by mutations in different loci. One gene causing HNPCC was mapped to chromosome 2p and recently a tight linkage between a polymorphic marker on the chromosome 3p and the disease locus has...

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Bernardgallon D, Ralliere C, Essioux L, et al. Linkage analyses of 3 French families to Loci on chromosome-2p and chromosome-3p predisposing to hereditary nonpolyposis colon-cancer. Int J Oncol. 1995;6(3):699-703doi: 10.3892/ijo.6.3.699.
Bernardgallon, D., Ralliere, C., Essioux, L., Gosse, S., Cure, H., Bay, J., Schraub, S., Bonaitipellie, C., Sobol, H., & Bignon, Y. (1995). Linkage analyses of 3 French families to Loci on chromosome-2p and chromosome-3p predisposing to hereditary nonpolyposis colon-cancer. International journal of oncology, 6(3), 699-703. https://doi.org/10.3892/ijo.6.3.699
Bernardgallon, D, et al. "Linkage analyses of 3 French families to Loci on chromosome-2p and chromosome-3p predisposing to hereditary nonpolyposis colon-cancer." International journal of oncology vol. 6,3 (1995): 699-703. doi: https://doi.org/10.3892/ijo.6.3.699
Bernardgallon D, Ralliere C, Essioux L, Gosse S, Cure H, Bay J, Schraub S, Bonaitipellie C, Sobol H, Bignon Y. Linkage analyses of 3 French families to Loci on chromosome-2p and chromosome-3p predisposing to hereditary nonpolyposis colon-cancer. Int J Oncol. 1995 Mar;6(3):699-703. doi: 10.3892/ijo.6.3.699. PMID: 21556592.
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Improved Acquisition and Reconstruction for Wavelength-Resolved Neutron Tomography.

Journal of imaging

Venkatakrishnan S, Zhang Y, Dessieux L, Hoffmann C, Bingham P, Bilheux H.
PMID: 34460581
J Imaging. 2021 Jan 15;7(1). doi: 10.3390/jimaging7010010.

Wavelength-resolved neutron tomography (WRNT) is an emerging technique for characterizing samples relevant to the materials sciences in 3D. WRNT studies can be carried out at beam lines in spallation neutron or reactor-based user facilities. Because of the limited availability...

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Venkatakrishnan S, Zhang Y, Dessieux L, et al. Improved Acquisition and Reconstruction for Wavelength-Resolved Neutron Tomography. J Imaging. 2021;7(1)doi: 10.3390/jimaging7010010.
Venkatakrishnan, S., Zhang, Y., Dessieux, L., Hoffmann, C., Bingham, P., & Bilheux, H. (2021). Improved Acquisition and Reconstruction for Wavelength-Resolved Neutron Tomography. Journal of imaging, 7(1), . https://doi.org/10.3390/jimaging7010010
Venkatakrishnan, Singanallur, et al. "Improved Acquisition and Reconstruction for Wavelength-Resolved Neutron Tomography." Journal of imaging vol. 7,1 (2021). doi: https://doi.org/10.3390/jimaging7010010
Venkatakrishnan S, Zhang Y, Dessieux L, Hoffmann C, Bingham P, Bilheux H. Improved Acquisition and Reconstruction for Wavelength-Resolved Neutron Tomography. J Imaging. 2021 Jan 15;7(1). doi: 10.3390/jimaging7010010. PMID: 34460581; PMCID: PMC8321247.
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Combining expression of GPC3 in tumors and CD16 on NK cells from peripheral blood to identify patients responding to codrituzumab.

Oncotarget

Chen G, Chen YC, Reis B, Belousov A, Jukofsky L, Rossin C, Muehlig A, Xu C, Essioux L, Ohtomo T, Di Laurenzio L, Puig O, Lee R.
PMID: 29535817
Oncotarget. 2018 Jan 02;9(12):10436-10444. doi: 10.18632/oncotarget.23830. eCollection 2018 Feb 13.

BACKGROUND: Codrituzumab, a monoclonal antibody targeting an oncofetal protein glypican-3 (GPC3) expressed on cell surface of hepatocellular carcinoma (HCC) induces antibody-dependent cellular cytotoxicity (ADCC) and inhibits tumor growth in preclinical studies. Based on this mechanism, tumor GPC3 expression and...

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Chen G, Chen YC, Reis B, et al. Combining expression of GPC3 in tumors and CD16 on NK cells from peripheral blood to identify patients responding to codrituzumab. Oncotarget. 2018;9(12):10436-10444doi: 10.18632/oncotarget.23830.
Chen, G., Chen, Y. C., Reis, B., Belousov, A., Jukofsky, L., Rossin, C., Muehlig, A., Xu, C., Essioux, L., Ohtomo, T., Di Laurenzio, L., Puig, O., & Lee, R. (2018). Combining expression of GPC3 in tumors and CD16 on NK cells from peripheral blood to identify patients responding to codrituzumab. Oncotarget, 9(12), 10436-10444. https://doi.org/10.18632/oncotarget.23830
Chen, Gong, et al. "Combining expression of GPC3 in tumors and CD16 on NK cells from peripheral blood to identify patients responding to codrituzumab." Oncotarget vol. 9,12 (2018): 10436-10444. doi: https://doi.org/10.18632/oncotarget.23830
Chen G, Chen YC, Reis B, Belousov A, Jukofsky L, Rossin C, Muehlig A, Xu C, Essioux L, Ohtomo T, Di Laurenzio L, Puig O, Lee R. Combining expression of GPC3 in tumors and CD16 on NK cells from peripheral blood to identify patients responding to codrituzumab. Oncotarget. 2018 Jan 02;9(12):10436-10444. doi: 10.18632/oncotarget.23830. eCollection 2018 Feb 13. PMID: 29535817; PMCID: PMC5828203.
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Analyses of linkage to 17q11-q23 in 3 French hereditary nonpolyposis colon-cancer families.

International journal of oncology

Bernardgallon D, Gosse S, Essioux L, Laurentpuig P, Ralliere C, Cure H, Bay J, Schraub S, Bonaitipellie C, Sobol H, Bignon Y.
PMID: 21556591
Int J Oncol. 1995 Mar;6(3):693-7.

Hereditary non-polyposis colorectal cancer (HNPCC) is an autosomal dominant disease, accounting for approximately 6% of colorectal cancers. We performed linkage analyses with the aim of proving or excluding the existence of a susceptibility locus on 17q. Three HNPCC families...

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Bernardgallon D, Gosse S, Essioux L, et al. Analyses of linkage to 17q11-q23 in 3 French hereditary nonpolyposis colon-cancer families. Int J Oncol. 1995;6(3):693-7
Bernardgallon, D., Gosse, S., Essioux, L., Laurentpuig, P., Ralliere, C., Cure, H., Bay, J., Schraub, S., Bonaitipellie, C., Sobol, H., & Bignon, Y. (1995). Analyses of linkage to 17q11-q23 in 3 French hereditary nonpolyposis colon-cancer families. International journal of oncology, 6(3), 693-7.
Bernardgallon, D, et al. "Analyses of linkage to 17q11-q23 in 3 French hereditary nonpolyposis colon-cancer families." International journal of oncology vol. 6,3 (1995): 693-7.
Bernardgallon D, Gosse S, Essioux L, Laurentpuig P, Ralliere C, Cure H, Bay J, Schraub S, Bonaitipellie C, Sobol H, Bignon Y. Analyses of linkage to 17q11-q23 in 3 French hereditary nonpolyposis colon-cancer families. Int J Oncol. 1995 Mar;6(3):693-7. PMID: 21556591.
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The effect of population structure on the relationship between heterosis and heterozygosity at marker loci.

TAG. Theoretical and applied genetics. Theoretische und angewandte Genetik

Charcosset A, Essioux L.
PMID: 24177851
Theor Appl Genet. 1994 Oct;89(2):336-43. doi: 10.1007/BF00225164.

The relationship between heterozygosity at neutral marker loci and heterosis of F1 hybrids is investigated using a theoretical model. Results emphasize that linkage disequilibrium between the markers and the loci implicated in heterosis [quantitative trait loci (QTLs) that exhibit...

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Charcosset A, Essioux L. The effect of population structure on the relationship between heterosis and heterozygosity at marker loci. Theor Appl Genet. 1994;89(2):336-43doi: 10.1007/BF00225164.
Charcosset, A., & Essioux, L. (1994). The effect of population structure on the relationship between heterosis and heterozygosity at marker loci. TAG. Theoretical and applied genetics. Theoretische und angewandte Genetik, 89(2), 336-43. https://doi.org/10.1007/BF00225164
Charcosset, A, and Essioux, L. "The effect of population structure on the relationship between heterosis and heterozygosity at marker loci." TAG. Theoretical and applied genetics. Theoretische und angewandte Genetik vol. 89,2 (1994): 336-43. doi: https://doi.org/10.1007/BF00225164
Charcosset A, Essioux L. The effect of population structure on the relationship between heterosis and heterozygosity at marker loci. Theor Appl Genet. 1994 Oct;89(2):336-43. doi: 10.1007/BF00225164. PMID: 24177851.
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Showing 1 to 8 of 8 entries