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Synthesis and Crystal and Molecular Structure of (BEDT-TTF)(2)[Pt(S(2)C(4)O(2))(2)]: A Radical-Cation Salt of the Platinum(II) Dithiosquarate Complex.

Inorganic chemistry

Bellitto C, Bonamico M, Fares V, Serino P.
PMID: 11666607
Inorg Chem. 1996 Jun 19;35(13):4070-4072. doi: 10.1021/ic951401w.

No abstract available.

Cite
Bellitto C, Bonamico M, Fares V, et al. Synthesis and Crystal and Molecular Structure of (BEDT-TTF)(2)[Pt(S(2)C(4)O(2))(2)]: A Radical-Cation Salt of the Platinum(II) Dithiosquarate Complex. Inorg Chem. 1996;35(13):4070-4072doi: 10.1021/ic951401w.
Bellitto, C., Bonamico, M., Fares, V., & Serino, P. (1996). Synthesis and Crystal and Molecular Structure of (BEDT-TTF)(2)[Pt(S(2)C(4)O(2))(2)]: A Radical-Cation Salt of the Platinum(II) Dithiosquarate Complex. Inorganic chemistry, 35(13), 4070-4072. https://doi.org/10.1021/ic951401w
Bellitto, C., et al. "Synthesis and Crystal and Molecular Structure of (BEDT-TTF)(2)[Pt(S(2)C(4)O(2))(2)]: A Radical-Cation Salt of the Platinum(II) Dithiosquarate Complex." Inorganic chemistry vol. 35,13 (1996): 4070-4072. doi: https://doi.org/10.1021/ic951401w
Bellitto C, Bonamico M, Fares V, Serino P. Synthesis and Crystal and Molecular Structure of (BEDT-TTF)(2)[Pt(S(2)C(4)O(2))(2)]: A Radical-Cation Salt of the Platinum(II) Dithiosquarate Complex. Inorg Chem. 1996 Jun 19;35(13):4070-4072. doi: 10.1021/ic951401w. PMID: 11666607.
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Synthesis, Magnetism, and X-ray Molecular Structure of the Mixed-Valence Vanadium(IV/V)-Oxygen Cluster [VO(4) subset(V(18)O(45))](9-).

Inorganic chemistry

Suber L, Bonamico M, Fares V.
PMID: 11669819
Inorg Chem. 1997 May 07;36(10):2030-2033. doi: 10.1021/ic960706n.

Within the transition metal oxide systems, vanadium presents a unique chemistry due to its capacity to form a great number of mixed-valence oxo clusters which often have the peculiarity to incorporate species that function, for size, shape, and charge,...

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Suber L, Bonamico M, Fares V. Synthesis, Magnetism, and X-ray Molecular Structure of the Mixed-Valence Vanadium(IV/V)-Oxygen Cluster [VO(4) subset(V(18)O(45))](9-). Inorg Chem. 1997;36(10):2030-2033doi: 10.1021/ic960706n.
Suber, L., Bonamico, M., & Fares, V. (1997). Synthesis, Magnetism, and X-ray Molecular Structure of the Mixed-Valence Vanadium(IV/V)-Oxygen Cluster [VO(4) subset(V(18)O(45))](9-). Inorganic chemistry, 36(10), 2030-2033. https://doi.org/10.1021/ic960706n
Suber, Lorenza, et al. "Synthesis, Magnetism, and X-ray Molecular Structure of the Mixed-Valence Vanadium(IV/V)-Oxygen Cluster [VO(4) subset(V(18)O(45))](9-)." Inorganic chemistry vol. 36,10 (1997): 2030-2033. doi: https://doi.org/10.1021/ic960706n
Suber L, Bonamico M, Fares V. Synthesis, Magnetism, and X-ray Molecular Structure of the Mixed-Valence Vanadium(IV/V)-Oxygen Cluster [VO(4) subset(V(18)O(45))](9-). Inorg Chem. 1997 May 07;36(10):2030-2033. doi: 10.1021/ic960706n. PMID: 11669819.
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RASopathies: Clinical Diagnosis in the First Year of Life.

Molecular syndromology

Digilio MC, Lepri F, Baban A, Dentici ML, Versacci P, Capolino R, Ferese R, De Luca A, Tartaglia M, Marino B, Dallapiccola B.
PMID: 22190897
Mol Syndromol. 2011 Sep;1(6):282-289. doi: 10.1159/000331266. Epub 2011 Sep 14.

Diagnosis within Noonan syndrome and related disorders (RASopathies) still presents a challenge during the first months of life, since most clinical features used to differentiate these conditions become manifest later in childhood. Here, we retrospectively reviewed the clinical records...

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Digilio MC, Lepri F, Baban A, et al. RASopathies: Clinical Diagnosis in the First Year of Life. Mol Syndromol. 2011;1(6):282-289doi: 10.1159/000331266.
Digilio, M. C., Lepri, F., Baban, A., Dentici, M. L., Versacci, P., Capolino, R., Ferese, R., De Luca, A., Tartaglia, M., Marino, B., & Dallapiccola, B. (2011). RASopathies: Clinical Diagnosis in the First Year of Life. Molecular syndromology, 1(6), 282-289. https://doi.org/10.1159/000331266
Digilio, M C, et al. "RASopathies: Clinical Diagnosis in the First Year of Life." Molecular syndromology vol. 1,6 (2011): 282-289. doi: https://doi.org/10.1159/000331266
Digilio MC, Lepri F, Baban A, Dentici ML, Versacci P, Capolino R, Ferese R, De Luca A, Tartaglia M, Marino B, Dallapiccola B. RASopathies: Clinical Diagnosis in the First Year of Life. Mol Syndromol. 2011 Sep;1(6):282-289. doi: 10.1159/000331266. Epub 2011 Sep 14. PMID: 22190897; PMCID: PMC3214957.
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Mitochondrial Serine Protease HTRA2 p.G399S in a Female with Di George Syndrome and Parkinson's Disease.

Parkinson's disease

Gambardella S, Ferese R, Scala S, Carboni S, Biagioni F, Giardina E, Zampatti S, Modugno N, Fabbiano F, Fornai F, Centonze D, Ruggieri S.
PMID: 30034773
Parkinsons Dis. 2018 Jun 21;2018:5651435. doi: 10.1155/2018/5651435. eCollection 2018.

Deletion at 22q11.2 responsible for Di George syndrome (DGs) is a risk factor for early-onset Parkinson's disease (EOPD). To date, all patients reported with 22q11.2 deletions and parkinsonian features are negative for a family history of PD, and possible...

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Gambardella S, Ferese R, Scala S, et al. Mitochondrial Serine Protease HTRA2 p.G399S in a Female with Di George Syndrome and Parkinson's Disease. Parkinsons Dis. 2018;2018:5651435doi: 10.1155/2018/5651435.
Gambardella, S., Ferese, R., Scala, S., Carboni, S., Biagioni, F., Giardina, E., Zampatti, S., Modugno, N., Fabbiano, F., Fornai, F., Centonze, D., & Ruggieri, S. (2018). Mitochondrial Serine Protease HTRA2 p.G399S in a Female with Di George Syndrome and Parkinson's Disease. Parkinson's disease, 20185651435. https://doi.org/10.1155/2018/5651435
Gambardella, Stefano, et al. "Mitochondrial Serine Protease HTRA2 p.G399S in a Female with Di George Syndrome and Parkinson's Disease." Parkinson's disease vol. 2018 (2018): 5651435. doi: https://doi.org/10.1155/2018/5651435
Gambardella S, Ferese R, Scala S, Carboni S, Biagioni F, Giardina E, Zampatti S, Modugno N, Fabbiano F, Fornai F, Centonze D, Ruggieri S. Mitochondrial Serine Protease HTRA2 p.G399S in a Female with Di George Syndrome and Parkinson's Disease. Parkinsons Dis. 2018 Jun 21;2018:5651435. doi: 10.1155/2018/5651435. eCollection 2018. PMID: 30034773; PMCID: PMC6032645.
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A Novel Homozygous Variant in the Fork-Head-Associated Domain of Polynucleotide Kinase Phosphatase in a Patient Affected by Late-Onset Ataxia With Oculomotor Apraxia Type 4.

Frontiers in neurology

Campopiano R, Ferese R, Buttari F, Femiano C, Centonze D, Fornai F, Biagioni F, Chiaravalloti MA, Magnani M, Giardina E, Ruzzo A, Gambardella S.
PMID: 32010037
Front Neurol. 2020 Jan 15;10:1331. doi: 10.3389/fneur.2019.01331. eCollection 2019.

Ataxia with oculomotor apraxia (AOA) is a clinical syndrome featuring a group of genetic diseases including at least four separate autosomal-recessive cerebellar ataxias. All these disorders are due to altered genes involved in DNA repair. AOA type 4 (AOA4)...

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Campopiano R, Ferese R, Buttari F, et al. A Novel Homozygous Variant in the Fork-Head-Associated Domain of Polynucleotide Kinase Phosphatase in a Patient Affected by Late-Onset Ataxia With Oculomotor Apraxia Type 4. Front Neurol. 2020;10:1331doi: 10.3389/fneur.2019.01331.
Campopiano, R., Ferese, R., Buttari, F., Femiano, C., Centonze, D., Fornai, F., Biagioni, F., Chiaravalloti, M. A., Magnani, M., Giardina, E., Ruzzo, A., & Gambardella, S. (2019). A Novel Homozygous Variant in the Fork-Head-Associated Domain of Polynucleotide Kinase Phosphatase in a Patient Affected by Late-Onset Ataxia With Oculomotor Apraxia Type 4. Frontiers in neurology, 101331. https://doi.org/10.3389/fneur.2019.01331
Campopiano, Rosa, et al. "A Novel Homozygous Variant in the Fork-Head-Associated Domain of Polynucleotide Kinase Phosphatase in a Patient Affected by Late-Onset Ataxia With Oculomotor Apraxia Type 4." Frontiers in neurology vol. 10 (2019): 1331. doi: https://doi.org/10.3389/fneur.2019.01331
Campopiano R, Ferese R, Buttari F, Femiano C, Centonze D, Fornai F, Biagioni F, Chiaravalloti MA, Magnani M, Giardina E, Ruzzo A, Gambardella S. A Novel Homozygous Variant in the Fork-Head-Associated Domain of Polynucleotide Kinase Phosphatase in a Patient Affected by Late-Onset Ataxia With Oculomotor Apraxia Type 4. Front Neurol. 2020 Jan 15;10:1331. doi: 10.3389/fneur.2019.01331. eCollection 2019. PMID: 32010037; PMCID: PMC6974581.
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An open-access volume electron microscopy atlas of whole cells and tissues.

Nature

Xu CS, Pang S, Shtengel G, Müller A, Ritter AT, Hoffman HK, Takemura SY, Lu Z, Pasolli HA, Iyer N, Chung J, Bennett D, Weigel AV, Freeman M, van Engelenburg SB, Walther TC, Farese RV, Lippincott-Schwartz J, Mellman I, Solimena M, Hess HF.
PMID: 34616045
Nature. 2021 Nov;599(7883):147-151. doi: 10.1038/s41586-021-03992-4. Epub 2021 Oct 06.

Understanding cellular architecture is essential for understanding biology. Electron microscopy (EM) uniquely visualizes cellular structures with nanometre resolution. However, traditional methods, such as thin-section EM or EM tomography, have limitations in that they visualize only a single slice or...

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Xu CS, Pang S, Shtengel G, et al. An open-access volume electron microscopy atlas of whole cells and tissues. Nature. 2021;599(7883):147-151doi: 10.1038/s41586-021-03992-4.
Xu, C. S., Pang, S., Shtengel, G., Müller, A., Ritter, A. T., Hoffman, H. K., Takemura, S. Y., Lu, Z., Pasolli, H. A., Iyer, N., Chung, J., Bennett, D., Weigel, A. V., Freeman, M., van Engelenburg, S. B., Walther, T. C., Farese, R. V., Lippincott-Schwartz, J., Mellman, I., Solimena, M., & Hess, H. F. (2021). An open-access volume electron microscopy atlas of whole cells and tissues. Nature, 599(7883), 147-151. https://doi.org/10.1038/s41586-021-03992-4
Xu, C Shan, et al. "An open-access volume electron microscopy atlas of whole cells and tissues." Nature vol. 599,7883 (2021): 147-151. doi: https://doi.org/10.1038/s41586-021-03992-4
Xu CS, Pang S, Shtengel G, Müller A, Ritter AT, Hoffman HK, Takemura SY, Lu Z, Pasolli HA, Iyer N, Chung J, Bennett D, Weigel AV, Freeman M, van Engelenburg SB, Walther TC, Farese RV, Lippincott-Schwartz J, Mellman I, Solimena M, Hess HF. An open-access volume electron microscopy atlas of whole cells and tissues. Nature. 2021 Nov;599(7883):147-151. doi: 10.1038/s41586-021-03992-4. Epub 2021 Oct 06. PMID: 34616045.
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Analysis of Genetic and Non-genetic Predictors of Levodopa Induced Dyskinesia in Parkinson's Disease.

Frontiers in pharmacology

Tirozzi A, Modugno N, Palomba NP, Ferese R, Lombardi A, Olivola E, Gialluisi A, Esposito T.
PMID: 33995045
Front Pharmacol. 2021 Apr 29;12:640603. doi: 10.3389/fphar.2021.640603. eCollection 2021.

No abstract available.

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Tirozzi A, Modugno N, Palomba NP, et al. Analysis of Genetic and Non-genetic Predictors of Levodopa Induced Dyskinesia in Parkinson's Disease. Front Pharmacol. 2021;12:640603doi: 10.3389/fphar.2021.640603.
Tirozzi, A., Modugno, N., Palomba, N. P., Ferese, R., Lombardi, A., Olivola, E., Gialluisi, A., & Esposito, T. (2021). Analysis of Genetic and Non-genetic Predictors of Levodopa Induced Dyskinesia in Parkinson's Disease. Frontiers in pharmacology, 12640603. https://doi.org/10.3389/fphar.2021.640603
Tirozzi, Alfonsina, et al. "Analysis of Genetic and Non-genetic Predictors of Levodopa Induced Dyskinesia in Parkinson's Disease." Frontiers in pharmacology vol. 12 (2021): 640603. doi: https://doi.org/10.3389/fphar.2021.640603
Tirozzi A, Modugno N, Palomba NP, Ferese R, Lombardi A, Olivola E, Gialluisi A, Esposito T. Analysis of Genetic and Non-genetic Predictors of Levodopa Induced Dyskinesia in Parkinson's Disease. Front Pharmacol. 2021 Apr 29;12:640603. doi: 10.3389/fphar.2021.640603. eCollection 2021. PMID: 33995045; PMCID: PMC8118664.
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Animal Models: A Non-human Primate and Rodent Animal Model Research Platform, Natural History, and Biomarkers to Predict Clinical Outcome.

Health physics

MacVittie TJ, Farese AM, Kane MA.
PMID: 34546212
Health Phys. 2021 Oct 01;121(4):277-281. doi: 10.1097/HP.0000000000001479.

No abstract available.

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MacVittie TJ, Farese AM, Kane MA. Animal Models: A Non-human Primate and Rodent Animal Model Research Platform, Natural History, and Biomarkers to Predict Clinical Outcome. Health Phys. 2021;121(4):277-281doi: 10.1097/HP.0000000000001479.
MacVittie, T. J., Farese, A. M., & Kane, M. A. (2021). Animal Models: A Non-human Primate and Rodent Animal Model Research Platform, Natural History, and Biomarkers to Predict Clinical Outcome. Health physics, 121(4), 277-281. https://doi.org/10.1097/HP.0000000000001479
MacVittie, Thomas J, et al. "Animal Models: A Non-human Primate and Rodent Animal Model Research Platform, Natural History, and Biomarkers to Predict Clinical Outcome." Health physics vol. 121,4 (2021): 277-281. doi: https://doi.org/10.1097/HP.0000000000001479
MacVittie TJ, Farese AM, Kane MA. Animal Models: A Non-human Primate and Rodent Animal Model Research Platform, Natural History, and Biomarkers to Predict Clinical Outcome. Health Phys. 2021 Oct 01;121(4):277-281. doi: 10.1097/HP.0000000000001479. PMID: 34546212; PMCID: PMC8462056.
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AEOL 10150 Alleviates Radiation-induced Innate Immune Responses in Non-human Primate Lung Tissue.

Health physics

Cui W, Zhang P, Hankey KG, Xiao M, Farese AM, MacVittie TJ.
PMID: 34546215
Health Phys. 2021 Oct 01;121(4):331-344. doi: 10.1097/HP.0000000000001443.

ABSTRACT: To study the molecular and cellular mechanisms of radiation-induced lung injury (RILI) in a non-human primate model, Rhesus macaques were irradiated with lethal doses of radiation to the whole thorax. A subset of the irradiated animals was treated...

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Cui W, Zhang P, Hankey KG, et al. AEOL 10150 Alleviates Radiation-induced Innate Immune Responses in Non-human Primate Lung Tissue. Health Phys. 2021;121(4):331-344doi: 10.1097/HP.0000000000001443.
Cui, W., Zhang, P., Hankey, K. G., Xiao, M., Farese, A. M., & MacVittie, T. J. (2021). AEOL 10150 Alleviates Radiation-induced Innate Immune Responses in Non-human Primate Lung Tissue. Health physics, 121(4), 331-344. https://doi.org/10.1097/HP.0000000000001443
Cui, Wanchang, et al. "AEOL 10150 Alleviates Radiation-induced Innate Immune Responses in Non-human Primate Lung Tissue." Health physics vol. 121,4 (2021): 331-344. doi: https://doi.org/10.1097/HP.0000000000001443
Cui W, Zhang P, Hankey KG, Xiao M, Farese AM, MacVittie TJ. AEOL 10150 Alleviates Radiation-induced Innate Immune Responses in Non-human Primate Lung Tissue. Health Phys. 2021 Oct 01;121(4):331-344. doi: 10.1097/HP.0000000000001443. PMID: 34546215; PMCID: PMC8601036.
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Acute Proteomic Changes in Lung after Radiation: Toward Identifying Initiating Events of Delayed Effects of Acute Radiation Exposure in Non-human Primate after Partial Body Irradiation with Minimal Bone Marrow Sparing.

Health physics

Huang W, Yu J, Liu T, Defnet AE, Zalesak-Kravec S, Farese AM, MacVittie TJ, Kane MA.
PMID: 34546219
Health Phys. 2021 Oct 01;121(4):384-394. doi: 10.1097/HP.0000000000001476.

ABSTRACT: Radiation-induced lung injury is a delayed effect of acute radiation exposure resulting in pulmonary pneumonitis and fibrosis. Molecular mechanisms that lead to radiation-induced lung injury remain incompletely understood. Using a non-human primate model of partial body irradiation with...

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Huang W, Yu J, Liu T, et al. Acute Proteomic Changes in Lung after Radiation: Toward Identifying Initiating Events of Delayed Effects of Acute Radiation Exposure in Non-human Primate after Partial Body Irradiation with Minimal Bone Marrow Sparing. Health Phys. 2021;121(4):384-394doi: 10.1097/HP.0000000000001476.
Huang, W., Yu, J., Liu, T., Defnet, A. E., Zalesak-Kravec, S., Farese, A. M., MacVittie, T. J., & Kane, M. A. (2021). Acute Proteomic Changes in Lung after Radiation: Toward Identifying Initiating Events of Delayed Effects of Acute Radiation Exposure in Non-human Primate after Partial Body Irradiation with Minimal Bone Marrow Sparing. Health physics, 121(4), 384-394. https://doi.org/10.1097/HP.0000000000001476
Huang, Weiliang, et al. "Acute Proteomic Changes in Lung after Radiation: Toward Identifying Initiating Events of Delayed Effects of Acute Radiation Exposure in Non-human Primate after Partial Body Irradiation with Minimal Bone Marrow Sparing." Health physics vol. 121,4 (2021): 384-394. doi: https://doi.org/10.1097/HP.0000000000001476
Huang W, Yu J, Liu T, Defnet AE, Zalesak-Kravec S, Farese AM, MacVittie TJ, Kane MA. Acute Proteomic Changes in Lung after Radiation: Toward Identifying Initiating Events of Delayed Effects of Acute Radiation Exposure in Non-human Primate after Partial Body Irradiation with Minimal Bone Marrow Sparing. Health Phys. 2021 Oct 01;121(4):384-394. doi: 10.1097/HP.0000000000001476. PMID: 34546219; PMCID: PMC8546870.
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Ultra-Performance Liquid Chromatographic and Densitometric Methods for Sensitive Determination of Xipamide and Triamterene in Pure and Pharmaceutical Dosage Forms.

Journal of AOAC International

Fares NV, El Fiky HA, Badawey AM, Abd El Ghany MF.
PMID: 34436582
J AOAC Int. 2021 Aug 26; doi: 10.1093/jaoacint/qsab110. Epub 2021 Aug 26.

BACKGROUND: Validated UPLC method and TLC densitometric method were prescribed for determination of antihypertensive components. Objectives: To establish and validate rapid and accurate Ultra performance liquid chromatographic (UPLC) and TLC densitometric methods for determination of Xipamide and Triamterene in...

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Fares NV, El Fiky HA, Badawey AM, et al. Ultra-Performance Liquid Chromatographic and Densitometric Methods for Sensitive Determination of Xipamide and Triamterene in Pure and Pharmaceutical Dosage Forms. J AOAC Int. 2021;doi: 10.1093/jaoacint/qsab110.
Fares, N. V., El Fiky, H. A., Badawey, A. M., & Abd El Ghany, M. F. (2021). Ultra-Performance Liquid Chromatographic and Densitometric Methods for Sensitive Determination of Xipamide and Triamterene in Pure and Pharmaceutical Dosage Forms. Journal of AOAC International, . https://doi.org/10.1093/jaoacint/qsab110
Fares, N V, et al. "Ultra-Performance Liquid Chromatographic and Densitometric Methods for Sensitive Determination of Xipamide and Triamterene in Pure and Pharmaceutical Dosage Forms." Journal of AOAC International vol. (2021). doi: https://doi.org/10.1093/jaoacint/qsab110
Fares NV, El Fiky HA, Badawey AM, Abd El Ghany MF. Ultra-Performance Liquid Chromatographic and Densitometric Methods for Sensitive Determination of Xipamide and Triamterene in Pure and Pharmaceutical Dosage Forms. J AOAC Int. 2021 Aug 26; doi: 10.1093/jaoacint/qsab110. Epub 2021 Aug 26. PMID: 34436582.
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[Frosted branch angiitis-Rare differential diagnosis of retinal vasculitis].

Der Ophthalmologe : Zeitschrift der Deutschen Ophthalmologischen Gesellschaft

Farese G, Lorenz AT, Bocqué C, Szurman P, Rickmann A.
PMID: 35006309
Ophthalmologe. 2022 Jan 10; doi: 10.1007/s00347-021-01563-6. Epub 2022 Jan 10.

No abstract available.

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Farese G, Lorenz AT, Bocqué C, et al. Frosted-Branch-Angiitis – seltene Differenzialdiagnose einer retinalen Vaskulitis. [Frosted branch angiitis-Rare differential diagnosis of retinal vasculitis]. Ophthalmologe. 2022;doi: 10.1007/s00347-021-01563-6.
Farese, G., Lorenz, A. T., Bocqué, C., Szurman, P., & Rickmann, A. (2022). Frosted-Branch-Angiitis – seltene Differenzialdiagnose einer retinalen Vaskulitis. [Frosted branch angiitis-Rare differential diagnosis of retinal vasculitis]. Der Ophthalmologe : Zeitschrift der Deutschen Ophthalmologischen Gesellschaft, . https://doi.org/10.1007/s00347-021-01563-6
Farese, Gerardo, et al. "Frosted-Branch-Angiitis – seltene Differenzialdiagnose einer retinalen Vaskulitis." [Frosted branch angiitis-Rare differential diagnosis of retinal vasculitis]. Der Ophthalmologe : Zeitschrift der Deutschen Ophthalmologischen Gesellschaft vol. (2022). doi: https://doi.org/10.1007/s00347-021-01563-6
Farese G, Lorenz AT, Bocqué C, Szurman P, Rickmann A. Frosted-Branch-Angiitis – seltene Differenzialdiagnose einer retinalen Vaskulitis. [Frosted branch angiitis-Rare differential diagnosis of retinal vasculitis]. Ophthalmologe. 2022 Jan 10; doi: 10.1007/s00347-021-01563-6. Epub 2022 Jan 10. German. PMID: 35006309.
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