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Activities of the Right Temporo-Parieto-Occipital Junction Reflect Spatial Hearing Ability in Cochlear Implant Users.

Frontiers in neuroscience

Schäfer E, Vedoveli AE, Righetti G, Gamerdinger P, Knipper M, Tropitzsch A, Karnath HO, Braun C, Li Hegner Y.
PMID: 33776632
Front Neurosci. 2021 Mar 12;15:613101. doi: 10.3389/fnins.2021.613101. eCollection 2021.

Spatial hearing is critical for us not only to orient ourselves in space, but also to follow a conversation with multiple speakers involved in a complex sound environment. The hearing ability of people who suffered from severe sensorineural hearing...

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Schäfer E, Vedoveli AE, Righetti G, et al. Activities of the Right Temporo-Parieto-Occipital Junction Reflect Spatial Hearing Ability in Cochlear Implant Users. Front Neurosci. 2021;15:613101doi: 10.3389/fnins.2021.613101.
Schäfer, E., Vedoveli, A. E., Righetti, G., Gamerdinger, P., Knipper, M., Tropitzsch, A., Karnath, H. O., Braun, C., & Li Hegner, Y. (2021). Activities of the Right Temporo-Parieto-Occipital Junction Reflect Spatial Hearing Ability in Cochlear Implant Users. Frontiers in neuroscience, 15613101. https://doi.org/10.3389/fnins.2021.613101
Schäfer, Eusebia, et al. "Activities of the Right Temporo-Parieto-Occipital Junction Reflect Spatial Hearing Ability in Cochlear Implant Users." Frontiers in neuroscience vol. 15 (2021): 613101. doi: https://doi.org/10.3389/fnins.2021.613101
Schäfer E, Vedoveli AE, Righetti G, Gamerdinger P, Knipper M, Tropitzsch A, Karnath HO, Braun C, Li Hegner Y. Activities of the Right Temporo-Parieto-Occipital Junction Reflect Spatial Hearing Ability in Cochlear Implant Users. Front Neurosci. 2021 Mar 12;15:613101. doi: 10.3389/fnins.2021.613101. eCollection 2021. PMID: 33776632; PMCID: PMC7994335.
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Aberrant COL11A1 splicing causes prelingual autosomal dominant nonsyndromic hearing loss in the DFNA37 locus.

Human mutation

Rad A, Schade-Mann T, Gamerdinger P, Yanus GA, Schulte B, Müller M, Imyanitov EN, Biskup S, Löwenheim H, Tropitzsch A, Vona B.
PMID: 33169910
Hum Mutat. 2021 Jan;42(1):25-30. doi: 10.1002/humu.24136. Epub 2020 Nov 11.

Alpha-chain collagen molecules encoded by genes that include COL11A1 are essential for skeletal, ocular, and auditory function. COL11A1 variants have been reported in syndromes involving these organ systems. However, a description of the complete clinical spectrum is lacking, as...

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Rad A, Schade-Mann T, Gamerdinger P, et al. Aberrant COL11A1 splicing causes prelingual autosomal dominant nonsyndromic hearing loss in the DFNA37 locus. Hum Mutat. 2020;42(1):25-30doi: 10.1002/humu.24136.
Rad, A., Schade-Mann, T., Gamerdinger, P., Yanus, G. A., Schulte, B., Müller, M., Imyanitov, E. N., Biskup, S., Löwenheim, H., Tropitzsch, A., & Vona, B. (2021). Aberrant COL11A1 splicing causes prelingual autosomal dominant nonsyndromic hearing loss in the DFNA37 locus. Human mutation, 42(1), 25-30. https://doi.org/10.1002/humu.24136
Rad, Aboulfazl, et al. "Aberrant COL11A1 splicing causes prelingual autosomal dominant nonsyndromic hearing loss in the DFNA37 locus." Human mutation vol. 42,1 (2021): 25-30. doi: https://doi.org/10.1002/humu.24136
Rad A, Schade-Mann T, Gamerdinger P, Yanus GA, Schulte B, Müller M, Imyanitov EN, Biskup S, Löwenheim H, Tropitzsch A, Vona B. Aberrant COL11A1 splicing causes prelingual autosomal dominant nonsyndromic hearing loss in the DFNA37 locus. Hum Mutat. 2021 Jan;42(1):25-30. doi: 10.1002/humu.24136. Epub 2020 Nov 11. PMID: 33169910.
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Conformational and functional characterization of artificially conjugated non-canonical ubiquitin dimers.

Scientific reports

Schneider T, Berg A, Ulusoy Z, Gamerdinger M, Peter C, Kovermann M.
PMID: 31882959
Sci Rep. 2019 Dec 27;9(1):19991. doi: 10.1038/s41598-019-56458-z.

Ubiquitylation is an eminent posttranslational modification referring to the covalent attachment of single ubiquitin molecules or polyubiquitin chains to a target protein dictating the fate of such labeled polypeptide chains. Here, we have biochemically produced artificially Lys11-, and Lys27-,...

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Schneider T, Berg A, Ulusoy Z, et al. Conformational and functional characterization of artificially conjugated non-canonical ubiquitin dimers. Sci Rep. 2019;9(1):19991doi: 10.1038/s41598-019-56458-z.
Schneider, T., Berg, A., Ulusoy, Z., Gamerdinger, M., Peter, C., & Kovermann, M. (2019). Conformational and functional characterization of artificially conjugated non-canonical ubiquitin dimers. Scientific reports, 9(1), 19991. https://doi.org/10.1038/s41598-019-56458-z
Schneider, Tobias, et al. "Conformational and functional characterization of artificially conjugated non-canonical ubiquitin dimers." Scientific reports vol. 9,1 (2019): 19991. doi: https://doi.org/10.1038/s41598-019-56458-z
Schneider T, Berg A, Ulusoy Z, Gamerdinger M, Peter C, Kovermann M. Conformational and functional characterization of artificially conjugated non-canonical ubiquitin dimers. Sci Rep. 2019 Dec 27;9(1):19991. doi: 10.1038/s41598-019-56458-z. PMID: 31882959; PMCID: PMC6934565.
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Evaluation of in situ Columns for Characterizing Organic Contaminant Sorption During Transport.

Environmental science & technology

Gamerdinger AP, Van Rees KC, Rao PS, Jessup RE.
PMID: 22165869
Environ Sci Technol. 1994 Mar 01;28(3):376-82. doi: 10.1021/es00052a006.

No abstract available.

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Gamerdinger AP, Van Rees KC, Rao PS, et al. Evaluation of in situ Columns for Characterizing Organic Contaminant Sorption During Transport. Environ Sci Technol. 1994;28(3):376-82doi: 10.1021/es00052a006.
Gamerdinger, A. P., Van Rees, K. C., Rao, P. S., & Jessup, R. E. (1994). Evaluation of in situ Columns for Characterizing Organic Contaminant Sorption During Transport. Environmental science & technology, 28(3), 376-82. https://doi.org/10.1021/es00052a006
Gamerdinger, A P, et al. "Evaluation of in situ Columns for Characterizing Organic Contaminant Sorption During Transport." Environmental science & technology vol. 28,3 (1994): 376-82. doi: https://doi.org/10.1021/es00052a006
Gamerdinger AP, Van Rees KC, Rao PS, Jessup RE. Evaluation of in situ Columns for Characterizing Organic Contaminant Sorption During Transport. Environ Sci Technol. 1994 Mar 01;28(3):376-82. doi: 10.1021/es00052a006. PMID: 22165869.
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Aberrant COL11A1 splicing causes prelingual autosomal dominant nonsyndromic hearing loss in the DFNA37 locus.

Human mutation

Rad A, Schade-Mann T, Gamerdinger P, Yanus GA, Schulte B, Müller M, Imyanitov EN, Biskup S, Löwenheim H, Tropitzsch A, Vona B.
PMID: 33169910
Hum Mutat. 2021 Jan;42(1):25-30. doi: 10.1002/humu.24136. Epub 2020 Nov 11.

Alpha-chain collagen molecules encoded by genes that include COL11A1 are essential for skeletal, ocular, and auditory function. COL11A1 variants have been reported in syndromes involving these organ systems. However, a description of the complete clinical spectrum is lacking, as...

Cite
Rad A, Schade-Mann T, Gamerdinger P, et al. Aberrant COL11A1 splicing causes prelingual autosomal dominant nonsyndromic hearing loss in the DFNA37 locus. Hum Mutat. 2020;42(1):25-30doi: 10.1002/humu.24136.
Rad, A., Schade-Mann, T., Gamerdinger, P., Yanus, G. A., Schulte, B., Müller, M., Imyanitov, E. N., Biskup, S., Löwenheim, H., Tropitzsch, A., & Vona, B. (2021). Aberrant COL11A1 splicing causes prelingual autosomal dominant nonsyndromic hearing loss in the DFNA37 locus. Human mutation, 42(1), 25-30. https://doi.org/10.1002/humu.24136
Rad, Aboulfazl, et al. "Aberrant COL11A1 splicing causes prelingual autosomal dominant nonsyndromic hearing loss in the DFNA37 locus." Human mutation vol. 42,1 (2021): 25-30. doi: https://doi.org/10.1002/humu.24136
Rad A, Schade-Mann T, Gamerdinger P, Yanus GA, Schulte B, Müller M, Imyanitov EN, Biskup S, Löwenheim H, Tropitzsch A, Vona B. Aberrant COL11A1 splicing causes prelingual autosomal dominant nonsyndromic hearing loss in the DFNA37 locus. Hum Mutat. 2021 Jan;42(1):25-30. doi: 10.1002/humu.24136. Epub 2020 Nov 11. PMID: 33169910.
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Macrolides: the plug is out.

Cell

Gamerdinger M, Deuerling E.
PMID: 23101618
Cell. 2012 Oct 26;151(3):469-71. doi: 10.1016/j.cell.2012.10.015.

Macrolide antibiotics are thought to clog up the ribosomal tunnel and thereby block general protein synthesis. By using a combination of elegant in vivo and in vitro approaches, Kannan et al. show that the inhibitory action of these drugs...

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Gamerdinger M, Deuerling E. Macrolides: the plug is out. Cell. 2012;151(3):469-71doi: 10.1016/j.cell.2012.10.015.
Gamerdinger, M., & Deuerling, E. (2012). Macrolides: the plug is out. Cell, 151(3), 469-71. https://doi.org/10.1016/j.cell.2012.10.015
Gamerdinger, Martin, and Deuerling, Elke. "Macrolides: the plug is out." Cell vol. 151,3 (2012): 469-71. doi: https://doi.org/10.1016/j.cell.2012.10.015
Gamerdinger M, Deuerling E. Macrolides: the plug is out. Cell. 2012 Oct 26;151(3):469-71. doi: 10.1016/j.cell.2012.10.015. PMID: 23101618.
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Mechanical properties of human lung cells after mechanostimulation.

Biomedizinische Technik. Biomedical engineering

Gamerdinger K, Schumann S, Smudde E, Wernet F, Guttmann J.
PMID: 24042836
Biomed Tech (Berl). 2013 Aug;58. doi: 10.1515/bmt-2013-4201. Epub 2013 Sep 07.

No abstract available.

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Gamerdinger K, Schumann S, Smudde E, et al. Mechanical properties of human lung cells after mechanostimulation. Biomed Tech (Berl). 2013;58doi: 10.1515/bmt-2013-4201.
Gamerdinger, K., Schumann, S., Smudde, E., Wernet, F., & Guttmann, J. (2013). Mechanical properties of human lung cells after mechanostimulation. Biomedizinische Technik. Biomedical engineering, 58. https://doi.org/10.1515/bmt-2013-4201
Gamerdinger, K, et al. "Mechanical properties of human lung cells after mechanostimulation." Biomedizinische Technik. Biomedical engineering vol. 58 (2013). doi: https://doi.org/10.1515/bmt-2013-4201
Gamerdinger K, Schumann S, Smudde E, Wernet F, Guttmann J. Mechanical properties of human lung cells after mechanostimulation. Biomed Tech (Berl). 2013 Aug;58. doi: 10.1515/bmt-2013-4201. Epub 2013 Sep 07. PMID: 24042836.
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Enantioselective Synthesis of Cyclohepta[.

Organic letters

Häfner M, Sokolenko YM, Gamerdinger P, Stempel E, Gaich T.
PMID: 31497969
Org Lett. 2019 Sep 20;21(18):7370-7374. doi: 10.1021/acs.orglett.9b02687. Epub 2019 Sep 09.

An enantioselective synthesis of functionalized cyclohepta[

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Häfner M, Sokolenko YM, Gamerdinger P, et al. Enantioselective Synthesis of Cyclohepta[. Org Lett. 2019;21(18):7370-7374doi: 10.1021/acs.orglett.9b02687.
Häfner, M., Sokolenko, Y. M., Gamerdinger, P., Stempel, E., & Gaich, T. (2019). Enantioselective Synthesis of Cyclohepta[. Organic letters, 21(18), 7370-7374. https://doi.org/10.1021/acs.orglett.9b02687
Häfner, Maximilian, et al. "Enantioselective Synthesis of Cyclohepta[." Organic letters vol. 21,18 (2019): 7370-7374. doi: https://doi.org/10.1021/acs.orglett.9b02687
Häfner M, Sokolenko YM, Gamerdinger P, Stempel E, Gaich T. Enantioselective Synthesis of Cyclohepta[. Org Lett. 2019 Sep 20;21(18):7370-7374. doi: 10.1021/acs.orglett.9b02687. Epub 2019 Sep 09. PMID: 31497969.
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Parallel evaluation of cell‑based phage display panning strategies: Optimized selection and depletion steps result in AML blast‑binding consensus antibodies.

Molecular medicine reports

Weber T, Pscherer S, Gamerdinger U, Teigler-Schlegel A, Rutz N, Blau W, Rummel M, Gattenlöhner S, Tur MK, Nielsen K.
PMID: 34490477
Mol Med Rep. 2021 Nov;24(5). doi: 10.3892/mmr.2021.12407. Epub 2021 Sep 07.

Phage display technology (PD) is a powerful technique for the generation of tumor‑targeting antibodies. However, there are a number of different selection methods established in different laboratories around the world. Cell‑based PD panning methods using primary tumor cells are...

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Weber T, Pscherer S, Gamerdinger U, et al. Parallel evaluation of cell‑based phage display panning strategies: Optimized selection and depletion steps result in AML blast‑binding consensus antibodies. Mol Med Rep. 2021;24(5)doi: 10.3892/mmr.2021.12407.
Weber, T., Pscherer, S., Gamerdinger, U., Teigler-Schlegel, A., Rutz, N., Blau, W., Rummel, M., Gattenlöhner, S., Tur, M. K., & Nielsen, K. (2021). Parallel evaluation of cell‑based phage display panning strategies: Optimized selection and depletion steps result in AML blast‑binding consensus antibodies. Molecular medicine reports, 24(5), . https://doi.org/10.3892/mmr.2021.12407
Weber, Theresa, et al. "Parallel evaluation of cell‑based phage display panning strategies: Optimized selection and depletion steps result in AML blast‑binding consensus antibodies." Molecular medicine reports vol. 24,5 (2021). doi: https://doi.org/10.3892/mmr.2021.12407
Weber T, Pscherer S, Gamerdinger U, Teigler-Schlegel A, Rutz N, Blau W, Rummel M, Gattenlöhner S, Tur MK, Nielsen K. Parallel evaluation of cell‑based phage display panning strategies: Optimized selection and depletion steps result in AML blast‑binding consensus antibodies. Mol Med Rep. 2021 Nov;24(5). doi: 10.3892/mmr.2021.12407. Epub 2021 Sep 07. PMID: 34490477; PMCID: PMC8430305.
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Diagnostic Yield of Targeted Hearing Loss Gene Panel Sequencing in a Large German Cohort With a Balanced Age Distribution from a Single Diagnostic Center: An Eight-year Study.

Ear and hearing

Tropitzsch A, Schade-Mann T, Gamerdinger P, Dofek S, Schulte B, Schulze M, Battke F, Fehr S, Biskup S, Heyd A, Müller M, Löwenheim H, Vona B, Holderried M.
PMID: 34753855
Ear Hear. 2021 Nov 08; doi: 10.1097/AUD.0000000000001159. Epub 2021 Nov 08.

OBJECTIVES: Hereditary hearing loss exhibits high degrees of genetic and clinical heterogeneity. To elucidate the population-specific and age-related genetic and clinical spectra of hereditary hearing loss, we investigated the sequencing data of causally associated hearing loss genes in a...

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Tropitzsch A, Schade-Mann T, Gamerdinger P, et al. Diagnostic Yield of Targeted Hearing Loss Gene Panel Sequencing in a Large German Cohort With a Balanced Age Distribution from a Single Diagnostic Center: An Eight-year Study. Ear Hear. 2021;doi: 10.1097/AUD.0000000000001159.
Tropitzsch, A., Schade-Mann, T., Gamerdinger, P., Dofek, S., Schulte, B., Schulze, M., Battke, F., Fehr, S., Biskup, S., Heyd, A., Müller, M., Löwenheim, H., Vona, B., & Holderried, M. (2021). Diagnostic Yield of Targeted Hearing Loss Gene Panel Sequencing in a Large German Cohort With a Balanced Age Distribution from a Single Diagnostic Center: An Eight-year Study. Ear and hearing, . https://doi.org/10.1097/AUD.0000000000001159
Tropitzsch, Anke, et al. "Diagnostic Yield of Targeted Hearing Loss Gene Panel Sequencing in a Large German Cohort With a Balanced Age Distribution from a Single Diagnostic Center: An Eight-year Study." Ear and hearing vol. (2021). doi: https://doi.org/10.1097/AUD.0000000000001159
Tropitzsch A, Schade-Mann T, Gamerdinger P, Dofek S, Schulte B, Schulze M, Battke F, Fehr S, Biskup S, Heyd A, Müller M, Löwenheim H, Vona B, Holderried M. Diagnostic Yield of Targeted Hearing Loss Gene Panel Sequencing in a Large German Cohort With a Balanced Age Distribution from a Single Diagnostic Center: An Eight-year Study. Ear Hear. 2021 Nov 08; doi: 10.1097/AUD.0000000000001159. Epub 2021 Nov 08. PMID: 34753855.
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Duplication 3q13.11q23: Longitudinal study in a patient over a period of more than 7 years and refinements of the breakpoints.

Journal of pediatric genetics

Eggermann T, Spengler S, Gamerdinger U, Denecke B, Grimm S, Grimm M, Schubert R, Schwanitz G.
PMID: 27625815
J Pediatr Genet. 2012 Jun;1(2):143-7. doi: 10.3233/PGE-2012-023.

In 2006, we reported the first case with a pure duplication of proximal 3q. In these rare aberrations, detailed clinical and developmental investigations at different ages are required to provide sufficient phenotypic documentation. Clinical and psychological differences were therefore...

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Eggermann T, Spengler S, Gamerdinger U, et al. Duplication 3q13.11q23: Longitudinal study in a patient over a period of more than 7 years and refinements of the breakpoints. J Pediatr Genet. 2012;1(2):143-7doi: 10.3233/PGE-2012-023.
Eggermann, T., Spengler, S., Gamerdinger, U., Denecke, B., Grimm, S., Grimm, M., Schubert, R., & Schwanitz, G. (2012). Duplication 3q13.11q23: Longitudinal study in a patient over a period of more than 7 years and refinements of the breakpoints. Journal of pediatric genetics, 1(2), 143-7. https://doi.org/10.3233/PGE-2012-023
Eggermann, Thomas, et al. "Duplication 3q13.11q23: Longitudinal study in a patient over a period of more than 7 years and refinements of the breakpoints." Journal of pediatric genetics vol. 1,2 (2012): 143-7. doi: https://doi.org/10.3233/PGE-2012-023
Eggermann T, Spengler S, Gamerdinger U, Denecke B, Grimm S, Grimm M, Schubert R, Schwanitz G. Duplication 3q13.11q23: Longitudinal study in a patient over a period of more than 7 years and refinements of the breakpoints. J Pediatr Genet. 2012 Jun;1(2):143-7. doi: 10.3233/PGE-2012-023. PMID: 27625815; PMCID: PMC5020934.
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Showing 1 to 11 of 11 entries