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Thyrotoxicosis-induced vasospastic angina.

Revista espanola de cardiologia

Romero-Rodríguez N, Luisa Cabeza Letrán MA, Gil Ortega MV, Ballesteros Pradas S.
PMID: 23047048
Rev Esp Cardiol. 2008 Dec;61(12):1355-6. doi: 10.1016/S0300-8932(08)75747-6. Epub 2009 Apr 22.

No abstract available.

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Romero-Rodríguez N, Luisa Cabeza Letrán MA, Gil Ortega MV, et al. Thyrotoxicosis-induced vasospastic angina. Rev Esp Cardiol. 2009;61(12):1355-6doi: 10.1016/S0300-8932(08)75747-6.
Romero-Rodríguez, N., Luisa Cabeza Letrán, M. A., Gil Ortega, M. V., & Ballesteros Pradas, S. (2008). Thyrotoxicosis-induced vasospastic angina. Revista espanola de cardiologia, 61(12), 1355-6. https://doi.org/10.1016/S0300-8932(08)75747-6
Romero-Rodríguez, Nieves, et al. "Thyrotoxicosis-induced vasospastic angina." Revista espanola de cardiologia vol. 61,12 (2008): 1355-6. doi: https://doi.org/10.1016/S0300-8932(08)75747-6
Romero-Rodríguez N, Luisa Cabeza Letrán MA, Gil Ortega MV, Ballesteros Pradas S. Thyrotoxicosis-induced vasospastic angina. Rev Esp Cardiol. 2008 Dec;61(12):1355-6. doi: 10.1016/S0300-8932(08)75747-6. Epub 2009 Apr 22. Spanish. PMID: 23047048.
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Response to ECG, May 2018.

Revista espanola de cardiologia (English ed.)

Gil-Ortega I, Garrido-Corro B, Castillo-Moreno JA.
PMID: 29803239
Rev Esp Cardiol (Engl Ed). 2018 Jun;71(6):489. doi: 10.1016/j.rec.2017.11.033.

No abstract available.

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Gil-Ortega I, Garrido-Corro B, Castillo-Moreno JA. Response to ECG, May 2018. Rev Esp Cardiol (Engl Ed). 2018;71(6):489doi: 10.1016/j.rec.2017.11.033.
Gil-Ortega, I., Garrido-Corro, B., & Castillo-Moreno, J. A. (2018). Response to ECG, May 2018. Revista espanola de cardiologia (English ed.), 71(6), 489. https://doi.org/10.1016/j.rec.2017.11.033
Gil-Ortega, Ignacio, et al. "Response to ECG, May 2018." Revista espanola de cardiologia (English ed.) vol. 71,6 (2018): 489. doi: https://doi.org/10.1016/j.rec.2017.11.033
Gil-Ortega I, Garrido-Corro B, Castillo-Moreno JA. Response to ECG, May 2018. Rev Esp Cardiol (Engl Ed). 2018 Jun;71(6):489. doi: 10.1016/j.rec.2017.11.033. Spanish. PMID: 29803239.
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Dabigatran, the oral anticoagulant of choice at discharge in patients with non-valvular atrial fibrillation and COVID-19 infection: the ANIBAL protocol.

Drugs in context

Iturbe-Hernandez T, García de Guadiana Romualdo L, Gil Ortega I, Martínez Francés A, Meca Birlanga O, Cerezo-Manchado JJ.
PMID: 33014097
Drugs Context. 2020 Sep 18;9. doi: 10.7573/dic.2020-8-3. eCollection 2020.

Atrial fibrillation is a frequent complication among patients with severe coronavirus disease-2019 (COVID-19) infection. Both direct and indirect mechanisms through COVID-19 have been described to explain this relationship. COVID-19 infection increases the risk of developing both arterial and venous...

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Iturbe-Hernandez T, García de Guadiana Romualdo L, Gil Ortega I, et al. Dabigatran, the oral anticoagulant of choice at discharge in patients with non-valvular atrial fibrillation and COVID-19 infection: the ANIBAL protocol. Drugs Context. 2020;9doi: 10.7573/dic.2020-8-3.
Iturbe-Hernandez, T., García de Guadiana Romualdo, L., Gil Ortega, I., Martínez Francés, A., Meca Birlanga, O., & Cerezo-Manchado, J. J. (2020). Dabigatran, the oral anticoagulant of choice at discharge in patients with non-valvular atrial fibrillation and COVID-19 infection: the ANIBAL protocol. Drugs in context, 9. https://doi.org/10.7573/dic.2020-8-3
Iturbe-Hernandez, Teodoro, et al. "Dabigatran, the oral anticoagulant of choice at discharge in patients with non-valvular atrial fibrillation and COVID-19 infection: the ANIBAL protocol." Drugs in context vol. 9 (2020). doi: https://doi.org/10.7573/dic.2020-8-3
Iturbe-Hernandez T, García de Guadiana Romualdo L, Gil Ortega I, Martínez Francés A, Meca Birlanga O, Cerezo-Manchado JJ. Dabigatran, the oral anticoagulant of choice at discharge in patients with non-valvular atrial fibrillation and COVID-19 infection: the ANIBAL protocol. Drugs Context. 2020 Sep 18;9. doi: 10.7573/dic.2020-8-3. eCollection 2020. PMID: 33014097; PMCID: PMC7505118.
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Role of inflammation and endothelial dysfunction in the pathogenesis of cardiac syndrome X.

Future cardiology

Gil-Ortega I, Marzoa Rivas R, Ríos Vázquez R, Kaski JC.
PMID: 19804133
Future Cardiol. 2006 Jan;2(1):63-73. doi: 10.2217/14796678.2.1.63.

Chest pain with normal coronary arteriograms represents a major diagnostic and therapeutic challenge to contemporary cardiology. Cardiac syndrome X (CSX), defined as typical angina-like chest pain, a positive response to exercise stress testing and normal coronary arteriograms, encompasses patients...

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Gil-Ortega I, Marzoa Rivas R, Ríos Vázquez R, et al. Role of inflammation and endothelial dysfunction in the pathogenesis of cardiac syndrome X. Future Cardiol. 2006;2(1):63-73doi: 10.2217/14796678.2.1.63.
Gil-Ortega, I., Marzoa Rivas, R., Ríos Vázquez, R., & Kaski, J. C. (2006). Role of inflammation and endothelial dysfunction in the pathogenesis of cardiac syndrome X. Future cardiology, 2(1), 63-73. https://doi.org/10.2217/14796678.2.1.63
Gil-Ortega, Ignacio, et al. "Role of inflammation and endothelial dysfunction in the pathogenesis of cardiac syndrome X." Future cardiology vol. 2,1 (2006): 63-73. doi: https://doi.org/10.2217/14796678.2.1.63
Gil-Ortega I, Marzoa Rivas R, Ríos Vázquez R, Kaski JC. Role of inflammation and endothelial dysfunction in the pathogenesis of cardiac syndrome X. Future Cardiol. 2006 Jan;2(1):63-73. doi: 10.2217/14796678.2.1.63. PMID: 19804133.
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Mechanisms of perivascular adipose tissue dysfunction in obesity.

International journal of endocrinology

Fernández-Alfonso MS, Gil-Ortega M, García-Prieto CF, Aranguez I, Ruiz-Gayo M, Somoza B.
PMID: 24307898
Int J Endocrinol. 2013;2013:402053. doi: 10.1155/2013/402053. Epub 2013 Nov 07.

Most blood vessels are surrounded by adipose tissue. Similarly to the adventitia, perivascular adipose tissue (PVAT) was considered only as a passive structural support for the vasculature, and it was routinely removed for isolated blood vessel studies. In 1991,...

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Fernández-Alfonso MS, Gil-Ortega M, García-Prieto CF, et al. Mechanisms of perivascular adipose tissue dysfunction in obesity. Int J Endocrinol. 2013;2013:402053doi: 10.1155/2013/402053.
Fernández-Alfonso, M. S., Gil-Ortega, M., García-Prieto, C. F., Aranguez, I., Ruiz-Gayo, M., & Somoza, B. (2013). Mechanisms of perivascular adipose tissue dysfunction in obesity. International journal of endocrinology, 2013402053. https://doi.org/10.1155/2013/402053
Fernández-Alfonso, Maria S, et al. "Mechanisms of perivascular adipose tissue dysfunction in obesity." International journal of endocrinology vol. 2013 (2013): 402053. doi: https://doi.org/10.1155/2013/402053
Fernández-Alfonso MS, Gil-Ortega M, García-Prieto CF, Aranguez I, Ruiz-Gayo M, Somoza B. Mechanisms of perivascular adipose tissue dysfunction in obesity. Int J Endocrinol. 2013;2013:402053. doi: 10.1155/2013/402053. Epub 2013 Nov 07. PMID: 24307898; PMCID: PMC3838835.
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AT2R stimulation with C21 prevents arterial stiffening and endothelial dysfunction in the abdominal aorta from mice fed a high-fat diet.

Clinical science (London, England : 1979)

González-Blázquez R, Alcalá M, Cárdenas-Rebollo JM, Viana M, Steckelings UM, Boisvert WA, Unger T, Fernández-Alfonso MS, Somoza B, Gil-Ortega M.
PMID: 34854902
Clin Sci (Lond). 2021 Dec 22;135(24):2763-2780. doi: 10.1042/CS20210971.

The aim of the present study was to evaluate the effect of Compound 21 (C21), a selective AT2R agonist, on the prevention of endothelial dysfunction, extracellular matrix (ECM) remodeling and arterial stiffness associated with diet-induced obesity (DIO). Five-week-old male...

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González-Blázquez R, Alcalá M, Cárdenas-Rebollo JM, et al. AT2R stimulation with C21 prevents arterial stiffening and endothelial dysfunction in the abdominal aorta from mice fed a high-fat diet. Clin Sci (Lond). 2021;135(24):2763-2780doi: 10.1042/CS20210971.
González-Blázquez, R., Alcalá, M., Cárdenas-Rebollo, J. M., Viana, M., Steckelings, U. M., Boisvert, W. A., Unger, T., Fernández-Alfonso, M. S., Somoza, B., & Gil-Ortega, M. (2021). AT2R stimulation with C21 prevents arterial stiffening and endothelial dysfunction in the abdominal aorta from mice fed a high-fat diet. Clinical science (London, England : 1979), 135(24), 2763-2780. https://doi.org/10.1042/CS20210971
González-Blázquez, Raquel, et al. "AT2R stimulation with C21 prevents arterial stiffening and endothelial dysfunction in the abdominal aorta from mice fed a high-fat diet." Clinical science (London, England : 1979) vol. 135,24 (2021): 2763-2780. doi: https://doi.org/10.1042/CS20210971
González-Blázquez R, Alcalá M, Cárdenas-Rebollo JM, Viana M, Steckelings UM, Boisvert WA, Unger T, Fernández-Alfonso MS, Somoza B, Gil-Ortega M. AT2R stimulation with C21 prevents arterial stiffening and endothelial dysfunction in the abdominal aorta from mice fed a high-fat diet. Clin Sci (Lond). 2021 Dec 22;135(24):2763-2780. doi: 10.1042/CS20210971. PMID: 34854902.
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Anticontractile Effect of Perivascular Adipose Tissue and Leptin are Reduced in Hypertension.

Frontiers in pharmacology

Gálvez-Prieto B, Somoza B, Gil-Ortega M, García-Prieto CF, de Las Heras AI, González MC, Arribas S, Aranguez I, Bolbrinker J, Kreutz R, Ruiz-Gayo M, Fernández-Alfonso MS.
PMID: 22679436
Front Pharmacol. 2012 Jun 05;3:103. doi: 10.3389/fphar.2012.00103. eCollection 2012.

Leptin causes vasodilatation both by endothelium-dependent and -independent mechanisms. Leptin is synthesized by perivascular adipose tissue (PVAT). The hypothesis of this study is that a decrease of leptin production in PVAT of spontaneously hypertensive rats (SHR) might contribute to...

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Gálvez-Prieto B, Somoza B, Gil-Ortega M, et al. Anticontractile Effect of Perivascular Adipose Tissue and Leptin are Reduced in Hypertension. Front Pharmacol. 2012;3:103doi: 10.3389/fphar.2012.00103.
Gálvez-Prieto, B., Somoza, B., Gil-Ortega, M., García-Prieto, C. F., de Las Heras, A. I., González, M. C., Arribas, S., Aranguez, I., Bolbrinker, J., Kreutz, R., Ruiz-Gayo, M., & Fernández-Alfonso, M. S. (2012). Anticontractile Effect of Perivascular Adipose Tissue and Leptin are Reduced in Hypertension. Frontiers in pharmacology, 3103. https://doi.org/10.3389/fphar.2012.00103
Gálvez-Prieto, Beatriz, et al. "Anticontractile Effect of Perivascular Adipose Tissue and Leptin are Reduced in Hypertension." Frontiers in pharmacology vol. 3 (2012): 103. doi: https://doi.org/10.3389/fphar.2012.00103
Gálvez-Prieto B, Somoza B, Gil-Ortega M, García-Prieto CF, de Las Heras AI, González MC, Arribas S, Aranguez I, Bolbrinker J, Kreutz R, Ruiz-Gayo M, Fernández-Alfonso MS. Anticontractile Effect of Perivascular Adipose Tissue and Leptin are Reduced in Hypertension. Front Pharmacol. 2012 Jun 05;3:103. doi: 10.3389/fphar.2012.00103. eCollection 2012. PMID: 22679436; PMCID: PMC3367267.
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[Not Available].

Revista de calidad asistencial : organo de la Sociedad Espanola de Calidad Asistencial

Martín Castro C, Gil Ortega M, Gómez Jiménez FJ, Macías Rodríguez F, Cabrera Iboleón J.
PMID: 23040038
Rev Calid Asist. 2008 Jan;23(1):7-10. doi: 10.1016/S1134-282X(08)70460-3. Epub 2008 Oct 11.

OBJECTIVE: To determine the occult mortality in the extra-hospital setting.PATIENTS AND METHOD: Retrospective study included patients who required health care using the Andalusian Emergency Services from January 2002 to June 2002 and who remained at home or were transferred...

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Martín Castro C, Gil Ortega M, Gómez Jiménez FJ, et al. Mortalidad oculta en el ámbito extrahospitalario. [Not Available]. Rev Calid Asist. 2008;23(1):7-10doi: 10.1016/S1134-282X(08)70460-3.
Martín Castro, C., Gil Ortega, M., Gómez Jiménez, F. J., Macías Rodríguez, F., & Cabrera Iboleón, J. (2008). Mortalidad oculta en el ámbito extrahospitalario. [Not Available]. Revista de calidad asistencial : organo de la Sociedad Espanola de Calidad Asistencial, 23(1), 7-10. https://doi.org/10.1016/S1134-282X(08)70460-3
Martín Castro, Carmen, et al. "Mortalidad oculta en el ámbito extrahospitalario." [Not Available]. Revista de calidad asistencial : organo de la Sociedad Espanola de Calidad Asistencial vol. 23,1 (2008): 7-10. doi: https://doi.org/10.1016/S1134-282X(08)70460-3
Martín Castro C, Gil Ortega M, Gómez Jiménez FJ, Macías Rodríguez F, Cabrera Iboleón J. Mortalidad oculta en el ámbito extrahospitalario. [Not Available]. Rev Calid Asist. 2008 Jan;23(1):7-10. doi: 10.1016/S1134-282X(08)70460-3. Epub 2008 Oct 11. Spanish. PMID: 23040038.
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An unique case suffering from repetitive syncope episodes due to ictal asystole.

Journal of cardiology cases

Gil-Ortega I, Garrido-Corro B, Gil-Ortega M, Fortuna-Alcaraz ML.
PMID: 30532856
J Cardiol Cases. 2011 Apr 11;3(3):e149-e153. doi: 10.1016/j.jccase.2011.02.007. eCollection 2011 Jun.

We present the case of a patient with syncope with repetition over 12 years, with a clinical profile not clearly related with a cardiogenic origin, who was studied by several medical specialties without any accurate diagnosis. After subcutaneous loop...

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Gil-Ortega I, Garrido-Corro B, Gil-Ortega M, et al. An unique case suffering from repetitive syncope episodes due to ictal asystole. J Cardiol Cases. 2011;3(3):e149-e153doi: 10.1016/j.jccase.2011.02.007.
Gil-Ortega, I., Garrido-Corro, B., Gil-Ortega, M., & Fortuna-Alcaraz, M. L. (2011). An unique case suffering from repetitive syncope episodes due to ictal asystole. Journal of cardiology cases, 3(3), e149-e153. https://doi.org/10.1016/j.jccase.2011.02.007
Gil-Ortega, Ignacio, et al. "An unique case suffering from repetitive syncope episodes due to ictal asystole." Journal of cardiology cases vol. 3,3 (2011): e149-e153. doi: https://doi.org/10.1016/j.jccase.2011.02.007
Gil-Ortega I, Garrido-Corro B, Gil-Ortega M, Fortuna-Alcaraz ML. An unique case suffering from repetitive syncope episodes due to ictal asystole. J Cardiol Cases. 2011 Apr 11;3(3):e149-e153. doi: 10.1016/j.jccase.2011.02.007. eCollection 2011 Jun. PMID: 30532856; PMCID: PMC6265108.
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Development of RAPD and SCAR markers linked to the Pvr4 locus for resistance to PVY in pepper ( Capsicum annuum L.).

TAG. Theoretical and applied genetics. Theoretische und angewandte Genetik

Arnedo-Andrés S, Gil-Ortega R, Luis-Arteaga M, Hormaza I.
PMID: 12582935
Theor Appl Genet. 2002 Nov;105(6):1067-1074. doi: 10.1007/s00122-002-1058-2. Epub 2002 Sep 21.

Potato Virus Y (PVY) is the only potyvirus infecting pepper ( Capsicum annuum L.) in Europe. Currently, the development of pepper varieties resistant to PVY seems to be the most-efficient method to control PVY damage. Among the sources of...

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Arnedo-Andrés S, Gil-Ortega R, Luis-Arteaga M, et al. Development of RAPD and SCAR markers linked to the Pvr4 locus for resistance to PVY in pepper ( Capsicum annuum L.). Theor Appl Genet. 2002;105(6):1067-1074doi: 10.1007/s00122-002-1058-2.
Arnedo-Andrés, S., Gil-Ortega, R., Luis-Arteaga, M., & Hormaza, I. (2002). Development of RAPD and SCAR markers linked to the Pvr4 locus for resistance to PVY in pepper ( Capsicum annuum L.). TAG. Theoretical and applied genetics. Theoretische und angewandte Genetik, 105(6), 1067-1074. https://doi.org/10.1007/s00122-002-1058-2
Arnedo-Andrés, S., et al. "Development of RAPD and SCAR markers linked to the Pvr4 locus for resistance to PVY in pepper ( Capsicum annuum L.)." TAG. Theoretical and applied genetics. Theoretische und angewandte Genetik vol. 105,6 (2002): 1067-1074. doi: https://doi.org/10.1007/s00122-002-1058-2
Arnedo-Andrés S, Gil-Ortega R, Luis-Arteaga M, Hormaza I. Development of RAPD and SCAR markers linked to the Pvr4 locus for resistance to PVY in pepper ( Capsicum annuum L.). Theor Appl Genet. 2002 Nov;105(6):1067-1074. doi: 10.1007/s00122-002-1058-2. Epub 2002 Sep 21. PMID: 12582935.
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Remodeling of energy metabolism and absence of electrophysiological changes in the heart of obese hyperleptinemic mice. New insights into the pleiotropic role of leptin.

Frontiers in endocrinology

Guzmán-Ruiz R, Gómez-Hurtado N, Gil-Ortega M, Somoza B, González MC, Aránguez I, Martín-Ramos M, González-Martín C, Delgado C, Fernández-Alfonso M, Ruiz-Gayo M.
PMID: 24298268
Front Endocrinol (Lausanne). 2013 Nov 15;4:175. doi: 10.3389/fendo.2013.00175. eCollection 2013.

Dietary treatment with high-fat diets (HFD) triggers diabetes and hyperleptinemia, concomitantly with a partial state of leptin resistance that affects hepatic and adipose tissue but not the heart. In this context, characterized by widespread steatosis, cardiac lipid content remains...

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Guzmán-Ruiz R, Gómez-Hurtado N, Gil-Ortega M, et al. Remodeling of energy metabolism and absence of electrophysiological changes in the heart of obese hyperleptinemic mice. New insights into the pleiotropic role of leptin. Front Endocrinol (Lausanne). 2013;4:175doi: 10.3389/fendo.2013.00175.
Guzmán-Ruiz, R., Gómez-Hurtado, N., Gil-Ortega, M., Somoza, B., González, M. C., Aránguez, I., Martín-Ramos, M., González-Martín, C., Delgado, C., Fernández-Alfonso, M., & Ruiz-Gayo, M. (2013). Remodeling of energy metabolism and absence of electrophysiological changes in the heart of obese hyperleptinemic mice. New insights into the pleiotropic role of leptin. Frontiers in endocrinology, 4175. https://doi.org/10.3389/fendo.2013.00175
Guzmán-Ruiz, Rocío, et al. "Remodeling of energy metabolism and absence of electrophysiological changes in the heart of obese hyperleptinemic mice. New insights into the pleiotropic role of leptin." Frontiers in endocrinology vol. 4 (2013): 175. doi: https://doi.org/10.3389/fendo.2013.00175
Guzmán-Ruiz R, Gómez-Hurtado N, Gil-Ortega M, Somoza B, González MC, Aránguez I, Martín-Ramos M, González-Martín C, Delgado C, Fernández-Alfonso M, Ruiz-Gayo M. Remodeling of energy metabolism and absence of electrophysiological changes in the heart of obese hyperleptinemic mice. New insights into the pleiotropic role of leptin. Front Endocrinol (Lausanne). 2013 Nov 15;4:175. doi: 10.3389/fendo.2013.00175. eCollection 2013. PMID: 24298268; PMCID: PMC3828673.
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Finerenone Attenuates Endothelial Dysfunction and Albuminuria in a Chronic Kidney Disease Model by a Reduction in Oxidative Stress.

Frontiers in pharmacology

González-Blázquez R, Somoza B, Gil-Ortega M, Martín Ramos M, Ramiro-Cortijo D, Vega-Martín E, Schulz A, Ruilope LM, Kolkhof P, Kreutz R, Fernández-Alfonso MS.
PMID: 30356804
Front Pharmacol. 2018 Oct 09;9:1131. doi: 10.3389/fphar.2018.01131. eCollection 2018.

Albuminuria is an early marker of renovascular damage associated to an increase in oxidative stress. The Munich Wistar Frömter (MWF) rat is a model of chronic kidney disease (CKD), which exhibits endothelial dysfunction associated to low nitric oxide availability....

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González-Blázquez R, Somoza B, Gil-Ortega M, et al. Finerenone Attenuates Endothelial Dysfunction and Albuminuria in a Chronic Kidney Disease Model by a Reduction in Oxidative Stress. Front Pharmacol. 2018;9:1131doi: 10.3389/fphar.2018.01131.
González-Blázquez, R., Somoza, B., Gil-Ortega, M., Martín Ramos, M., Ramiro-Cortijo, D., Vega-Martín, E., Schulz, A., Ruilope, L. M., Kolkhof, P., Kreutz, R., & Fernández-Alfonso, M. S. (2018). Finerenone Attenuates Endothelial Dysfunction and Albuminuria in a Chronic Kidney Disease Model by a Reduction in Oxidative Stress. Frontiers in pharmacology, 91131. https://doi.org/10.3389/fphar.2018.01131
González-Blázquez, Raquel, et al. "Finerenone Attenuates Endothelial Dysfunction and Albuminuria in a Chronic Kidney Disease Model by a Reduction in Oxidative Stress." Frontiers in pharmacology vol. 9 (2018): 1131. doi: https://doi.org/10.3389/fphar.2018.01131
González-Blázquez R, Somoza B, Gil-Ortega M, Martín Ramos M, Ramiro-Cortijo D, Vega-Martín E, Schulz A, Ruilope LM, Kolkhof P, Kreutz R, Fernández-Alfonso MS. Finerenone Attenuates Endothelial Dysfunction and Albuminuria in a Chronic Kidney Disease Model by a Reduction in Oxidative Stress. Front Pharmacol. 2018 Oct 09;9:1131. doi: 10.3389/fphar.2018.01131. eCollection 2018. PMID: 30356804; PMCID: PMC6189469.
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