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Implementation of TERT promoter mutations improve prognostication of the WHO classification in meningioma.

Neuropathology and applied neurobiology

Mirian C, Grell K, Juratli TA, Sahm F, Spiegl-Kreinecker S, Peyre M, Biczok A, Tonn JC, Goutagny S, Bertero L, Maier AD, Jensen LR, Schackert G, Broholm H, Scheie D, Cahill DP, Brastianos PK, Skjøth-Rasmussen J, Fugleholm K, Ziebell M, Munch TN, Kristensen BW, Mathiesen T.
PMID: 34799864
Neuropathol Appl Neurobiol. 2021 Nov 19; doi: 10.1111/nan.12773. Epub 2021 Nov 19.

TERT promoter mutations have been associated with increased risk of recurrence in meningioma cohorts, thus a potential biomarker for aggressive phenotypes. A main purpose of refining tumour classification is better predictions on the patient level. We compiled data from...

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Mirian C, Grell K, Juratli TA, et al. Implementation of TERT promoter mutations improve prognostication of the WHO classification in meningioma. Neuropathol Appl Neurobiol. 2021;doi: 10.1111/nan.12773.
Mirian, C., Grell, K., Juratli, T. A., Sahm, F., Spiegl-Kreinecker, S., Peyre, M., Biczok, A., Tonn, J. C., Goutagny, S., Bertero, L., Maier, A. D., Jensen, L. R., Schackert, G., Broholm, H., Scheie, D., Cahill, D. P., Brastianos, P. K., Skjøth-Rasmussen, J., Fugleholm, K., Ziebell, M., Munch, T. N., Kristensen, B. W., & Mathiesen, T. (2021). Implementation of TERT promoter mutations improve prognostication of the WHO classification in meningioma. Neuropathology and applied neurobiology, . https://doi.org/10.1111/nan.12773
Mirian, Christian, et al. "Implementation of TERT promoter mutations improve prognostication of the WHO classification in meningioma." Neuropathology and applied neurobiology vol. (2021). doi: https://doi.org/10.1111/nan.12773
Mirian C, Grell K, Juratli TA, Sahm F, Spiegl-Kreinecker S, Peyre M, Biczok A, Tonn JC, Goutagny S, Bertero L, Maier AD, Jensen LR, Schackert G, Broholm H, Scheie D, Cahill DP, Brastianos PK, Skjøth-Rasmussen J, Fugleholm K, Ziebell M, Munch TN, Kristensen BW, Mathiesen T. Implementation of TERT promoter mutations improve prognostication of the WHO classification in meningioma. Neuropathol Appl Neurobiol. 2021 Nov 19; doi: 10.1111/nan.12773. Epub 2021 Nov 19. PMID: 34799864.
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Inflammasome Deletion Promotes Anti-tumor NK Cell Function in an IL-1/IL-18 Independent Way in Murine Invasive Breast Cancer.

Frontiers in oncology

Guey B, Bodnar-Wachtel M, Drouillard A, Eberhardt A, Pratviel M, Goutagny N, Bendriss-Vermare N, Puisieux I, Caux C, Walzer T, Petrilli V.
PMID: 33042810
Front Oncol. 2020 Sep 16;10:1683. doi: 10.3389/fonc.2020.01683. eCollection 2020.

Inflammasomes are molecular complexes that trigger an inflammatory response upon detection of pathogens or danger signals. Recent studies suggest that they are also involved in cancer progression. However, their roles during tumorigenesis remain poorly understood and controversial. Here, we...

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Guey B, Bodnar-Wachtel M, Drouillard A, et al. Inflammasome Deletion Promotes Anti-tumor NK Cell Function in an IL-1/IL-18 Independent Way in Murine Invasive Breast Cancer. Front Oncol. 2020;10:1683doi: 10.3389/fonc.2020.01683.
Guey, B., Bodnar-Wachtel, M., Drouillard, A., Eberhardt, A., Pratviel, M., Goutagny, N., Bendriss-Vermare, N., Puisieux, I., Caux, C., Walzer, T., & Petrilli, V. (2020). Inflammasome Deletion Promotes Anti-tumor NK Cell Function in an IL-1/IL-18 Independent Way in Murine Invasive Breast Cancer. Frontiers in oncology, 101683. https://doi.org/10.3389/fonc.2020.01683
Guey, Baptiste, et al. "Inflammasome Deletion Promotes Anti-tumor NK Cell Function in an IL-1/IL-18 Independent Way in Murine Invasive Breast Cancer." Frontiers in oncology vol. 10 (2020): 1683. doi: https://doi.org/10.3389/fonc.2020.01683
Guey B, Bodnar-Wachtel M, Drouillard A, Eberhardt A, Pratviel M, Goutagny N, Bendriss-Vermare N, Puisieux I, Caux C, Walzer T, Petrilli V. Inflammasome Deletion Promotes Anti-tumor NK Cell Function in an IL-1/IL-18 Independent Way in Murine Invasive Breast Cancer. Front Oncol. 2020 Sep 16;10:1683. doi: 10.3389/fonc.2020.01683. eCollection 2020. PMID: 33042810; PMCID: PMC7526436.
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Precocious Alterations of Brain Oscillatory Activity in Alzheimer's Disease: A Window of Opportunity for Early Diagnosis and Treatment.

Frontiers in cellular neuroscience

Hamm V, Héraud C, Cassel JC, Mathis C, Goutagny R.
PMID: 26733816
Front Cell Neurosci. 2015 Dec 21;9:491. doi: 10.3389/fncel.2015.00491. eCollection 2015.

Alzheimer's disease (AD) is the most common form of neurodegenerative dementia accounting for 50-80% of all age-related dementia. This pathology is characterized by the progressive and irreversible alteration of cognitive functions, such as memory, leading inexorably to the loss...

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Hamm V, Héraud C, Cassel JC, et al. Precocious Alterations of Brain Oscillatory Activity in Alzheimer's Disease: A Window of Opportunity for Early Diagnosis and Treatment. Front Cell Neurosci. 2015;9:491doi: 10.3389/fncel.2015.00491.
Hamm, V., Héraud, C., Cassel, J. C., Mathis, C., & Goutagny, R. (2015). Precocious Alterations of Brain Oscillatory Activity in Alzheimer's Disease: A Window of Opportunity for Early Diagnosis and Treatment. Frontiers in cellular neuroscience, 9491. https://doi.org/10.3389/fncel.2015.00491
Hamm, Valentine, et al. "Precocious Alterations of Brain Oscillatory Activity in Alzheimer's Disease: A Window of Opportunity for Early Diagnosis and Treatment." Frontiers in cellular neuroscience vol. 9 (2015): 491. doi: https://doi.org/10.3389/fncel.2015.00491
Hamm V, Héraud C, Cassel JC, Mathis C, Goutagny R. Precocious Alterations of Brain Oscillatory Activity in Alzheimer's Disease: A Window of Opportunity for Early Diagnosis and Treatment. Front Cell Neurosci. 2015 Dec 21;9:491. doi: 10.3389/fncel.2015.00491. eCollection 2015. PMID: 26733816; PMCID: PMC4685112.
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TLR9 re-expression in cancer cells extends the S-phase and stabilizes p16(INK4a) protein expression.

Oncogenesis

Parroche P, Roblot G, Le Calvez-Kelm F, Tout I, Marotel M, Malfroy M, Durand G, McKay J, Ainouze M, Carreira C, Allatif O, Traverse-Glehen A, Mendiola M, Pozo-Kreilinger JJ, Caux C, Tommasino M, Goutagny N, Hasan UA.
PMID: 27454079
Oncogenesis. 2016 Jul 25;5(7):e244. doi: 10.1038/oncsis.2016.49.

Toll-like receptor 9 (TLR9) recognizes bacterial, viral or cell damage-associated DNA, which initiates innate immune responses. We have previously shown that TLR9 expression is downregulated in several viral induced cancers including HPV16-induced cervical neoplasia. Findings supported that downregulation of...

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Parroche P, Roblot G, Le Calvez-Kelm F, et al. TLR9 re-expression in cancer cells extends the S-phase and stabilizes p16(INK4a) protein expression. Oncogenesis. 2016;5(7):e244doi: 10.1038/oncsis.2016.49.
Parroche, P., Roblot, G., Le Calvez-Kelm, F., Tout, I., Marotel, M., Malfroy, M., Durand, G., McKay, J., Ainouze, M., Carreira, C., Allatif, O., Traverse-Glehen, A., Mendiola, M., Pozo-Kreilinger, J. J., Caux, C., Tommasino, M., Goutagny, N., & Hasan, U. A. (2016). TLR9 re-expression in cancer cells extends the S-phase and stabilizes p16(INK4a) protein expression. Oncogenesis, 5(7), e244. https://doi.org/10.1038/oncsis.2016.49
Parroche, P, et al. "TLR9 re-expression in cancer cells extends the S-phase and stabilizes p16(INK4a) protein expression." Oncogenesis vol. 5,7 (2016): e244. doi: https://doi.org/10.1038/oncsis.2016.49
Parroche P, Roblot G, Le Calvez-Kelm F, Tout I, Marotel M, Malfroy M, Durand G, McKay J, Ainouze M, Carreira C, Allatif O, Traverse-Glehen A, Mendiola M, Pozo-Kreilinger JJ, Caux C, Tommasino M, Goutagny N, Hasan UA. TLR9 re-expression in cancer cells extends the S-phase and stabilizes p16(INK4a) protein expression. Oncogenesis. 2016 Jul 25;5(7):e244. doi: 10.1038/oncsis.2016.49. PMID: 27454079; PMCID: PMC4972902.
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Regulation of the Hippocampal Network by VGLUT3-Positive CCK- GABAergic Basket Cells.

Frontiers in cellular neuroscience

Fasano C, Rocchetti J, Pietrajtis K, Zander JF, Manseau F, Sakae DY, Marcus-Sells M, Ramet L, Morel LJ, Carrel D, Dumas S, Bolte S, Bernard V, Vigneault E, Goutagny R, Ahnert-Hilger G, Giros B, Daumas S, Williams S, El Mestikawy S.
PMID: 28559797
Front Cell Neurosci. 2017 May 16;11:140. doi: 10.3389/fncel.2017.00140. eCollection 2017.

Hippocampal interneurons release the inhibitory transmitter GABA to regulate excitation, rhythm generation and synaptic plasticity. A subpopulation of GABAergic basket cells co-expresses the GABA/glycine vesicular transporters (VIAAT) and the atypical type III vesicular glutamate transporter (VGLUT3); therefore, these cells...

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Fasano C, Rocchetti J, Pietrajtis K, et al. Regulation of the Hippocampal Network by VGLUT3-Positive CCK- GABAergic Basket Cells. Front Cell Neurosci. 2017;11:140doi: 10.3389/fncel.2017.00140.
Fasano, C., Rocchetti, J., Pietrajtis, K., Zander, J. F., Manseau, F., Sakae, D. Y., Marcus-Sells, M., Ramet, L., Morel, L. J., Carrel, D., Dumas, S., Bolte, S., Bernard, V., Vigneault, E., Goutagny, R., Ahnert-Hilger, G., Giros, B., Daumas, S., Williams, S., & El Mestikawy, S. (2017). Regulation of the Hippocampal Network by VGLUT3-Positive CCK- GABAergic Basket Cells. Frontiers in cellular neuroscience, 11140. https://doi.org/10.3389/fncel.2017.00140
Fasano, Caroline, et al. "Regulation of the Hippocampal Network by VGLUT3-Positive CCK- GABAergic Basket Cells." Frontiers in cellular neuroscience vol. 11 (2017): 140. doi: https://doi.org/10.3389/fncel.2017.00140
Fasano C, Rocchetti J, Pietrajtis K, Zander JF, Manseau F, Sakae DY, Marcus-Sells M, Ramet L, Morel LJ, Carrel D, Dumas S, Bolte S, Bernard V, Vigneault E, Goutagny R, Ahnert-Hilger G, Giros B, Daumas S, Williams S, El Mestikawy S. Regulation of the Hippocampal Network by VGLUT3-Positive CCK- GABAergic Basket Cells. Front Cell Neurosci. 2017 May 16;11:140. doi: 10.3389/fncel.2017.00140. eCollection 2017. PMID: 28559797; PMCID: PMC5432579.
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Optimized Generation of Functional Neutrophils and Macrophages from Patient-Specific Induced Pluripotent Stem Cells: Ex Vivo Models of X(0)-Linked, AR22(0)- and AR47(0)- Chronic Granulomatous Diseases.

BioResearch open access

Brault J, Goutagny E, Telugu N, Shao K, Baquié M, Satre V, Coutton C, Grunwald D, Brion JP, Barlogis V, Stephan JL, Plantaz D, Hescheler J, Krause KH, Sarić T, Stasia MJ.
PMID: 25469316
Biores Open Access. 2014 Dec 01;3(6):311-26. doi: 10.1089/biores.2014.0045.

Chronic granulomatous disease (CGD) is an inherited orphan disorder caused by mutations in one of the five genes encoding reduced nicotinamide-adenine-dinucleotide-phosphate oxidase subunits, which subsequently lead to impairment in the production of microbicidal reactive oxygen species (ROS). In order...

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Brault J, Goutagny E, Telugu N, et al. Optimized Generation of Functional Neutrophils and Macrophages from Patient-Specific Induced Pluripotent Stem Cells: Ex Vivo Models of X(0)-Linked, AR22(0)- and AR47(0)- Chronic Granulomatous Diseases. Biores Open Access. 2014;3(6):311-26doi: 10.1089/biores.2014.0045.
Brault, J., Goutagny, E., Telugu, N., Shao, K., Baquié, M., Satre, V., Coutton, C., Grunwald, D., Brion, J. P., Barlogis, V., Stephan, J. L., Plantaz, D., Hescheler, J., Krause, K. H., Sarić, T., & Stasia, M. J. (2014). Optimized Generation of Functional Neutrophils and Macrophages from Patient-Specific Induced Pluripotent Stem Cells: Ex Vivo Models of X(0)-Linked, AR22(0)- and AR47(0)- Chronic Granulomatous Diseases. BioResearch open access, 3(6), 311-26. https://doi.org/10.1089/biores.2014.0045
Brault, Julie, et al. "Optimized Generation of Functional Neutrophils and Macrophages from Patient-Specific Induced Pluripotent Stem Cells: Ex Vivo Models of X(0)-Linked, AR22(0)- and AR47(0)- Chronic Granulomatous Diseases." BioResearch open access vol. 3,6 (2014): 311-26. doi: https://doi.org/10.1089/biores.2014.0045
Brault J, Goutagny E, Telugu N, Shao K, Baquié M, Satre V, Coutton C, Grunwald D, Brion JP, Barlogis V, Stephan JL, Plantaz D, Hescheler J, Krause KH, Sarić T, Stasia MJ. Optimized Generation of Functional Neutrophils and Macrophages from Patient-Specific Induced Pluripotent Stem Cells: Ex Vivo Models of X(0)-Linked, AR22(0)- and AR47(0)- Chronic Granulomatous Diseases. Biores Open Access. 2014 Dec 01;3(6):311-26. doi: 10.1089/biores.2014.0045. PMID: 25469316; PMCID: PMC4245876.
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Pattern recognition receptors: an update.

Expert review of clinical immunology

Goutagny N, Fitzgerald KA.
PMID: 20477614
Expert Rev Clin Immunol. 2006 Jul;2(4):569-83. doi: 10.1586/1744666X.2.4.569.

The vertebrate immune system consists of two inter-related components, the innate and adaptive responses, which are required for the resolution of infection. The innate immune response is critical for the immediate protection from infection and for marshalling the B-...

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Goutagny N, Fitzgerald KA. Pattern recognition receptors: an update. Expert Rev Clin Immunol. 2006;2(4):569-83doi: 10.1586/1744666X.2.4.569.
Goutagny, N., & Fitzgerald, K. A. (2006). Pattern recognition receptors: an update. Expert review of clinical immunology, 2(4), 569-83. https://doi.org/10.1586/1744666X.2.4.569
Goutagny, Nadege, and Fitzgerald, Katherine A. "Pattern recognition receptors: an update." Expert review of clinical immunology vol. 2,4 (2006): 569-83. doi: https://doi.org/10.1586/1744666X.2.4.569
Goutagny N, Fitzgerald KA. Pattern recognition receptors: an update. Expert Rev Clin Immunol. 2006 Jul;2(4):569-83. doi: 10.1586/1744666X.2.4.569. PMID: 20477614.
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Hippocampal oscillatory activity in Alzheimer's disease: toward the identification of early biomarkers?.

Aging and disease

Goutagny R, Krantic S.
PMID: 23730529
Aging Dis. 2013 Jan 23;4(3):134-40. Print 2013 Jun.

Alzheimer's disease (AD) develops for a yet unknown period of time and can progress undiagnosed for years before its first clinical manifestation consisting of characteristic cognitive impairments. Current AD treatments offer only a small symptomatic benefit, likely because AD...

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Goutagny R, Krantic S. Hippocampal oscillatory activity in Alzheimer's disease: toward the identification of early biomarkers?. Aging Dis. 2013;4(3):134-40
Goutagny, R., & Krantic, S. (2013). Hippocampal oscillatory activity in Alzheimer's disease: toward the identification of early biomarkers?. Aging and disease, 4(3), 134-40.
Goutagny, Romain, and Krantic, Slavica. "Hippocampal oscillatory activity in Alzheimer's disease: toward the identification of early biomarkers?." Aging and disease vol. 4,3 (2013): 134-40.
Goutagny R, Krantic S. Hippocampal oscillatory activity in Alzheimer's disease: toward the identification of early biomarkers?. Aging Dis. 2013 Jan 23;4(3):134-40. Print 2013 Jun. PMID: 23730529; PMCID: PMC3660123.
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Teenagers and young adults with a past of allogenic hematopoietic stem cell transplantation are at significant risk of chronic kidney disease.

Pediatric nephrology (Berlin, Germany)

da Silva Selistre L, Renard C, Bacchetta J, Goutagny MP, Hu J, Carla de Souza V, Bertrand Y, Dubourg L, Domenech C.
PMID: 34735600
Pediatr Nephrol. 2021 Nov 04; doi: 10.1007/s00467-021-05319-x. Epub 2021 Nov 04.

BACKGROUND: Allogenic hematopoietic stem cell transplantation (aHSCT) remains the treatment of choice for some malignant hemopathies in children, albeit with the risk of long-term consequences, including chronic kidney disease (CKD).METHODS: In our single tertiary referral center, we retrospectively assessed...

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da Silva Selistre L, Renard C, Bacchetta J, et al. Teenagers and young adults with a past of allogenic hematopoietic stem cell transplantation are at significant risk of chronic kidney disease. Pediatr Nephrol. 2021;doi: 10.1007/s00467-021-05319-x.
da Silva Selistre, L., Renard, C., Bacchetta, J., Goutagny, M. P., Hu, J., Carla de Souza, V., Bertrand, Y., Dubourg, L., & Domenech, C. (2021). Teenagers and young adults with a past of allogenic hematopoietic stem cell transplantation are at significant risk of chronic kidney disease. Pediatric nephrology (Berlin, Germany), . https://doi.org/10.1007/s00467-021-05319-x
da Silva Selistre, Luciano, et al. "Teenagers and young adults with a past of allogenic hematopoietic stem cell transplantation are at significant risk of chronic kidney disease." Pediatric nephrology (Berlin, Germany) vol. (2021). doi: https://doi.org/10.1007/s00467-021-05319-x
da Silva Selistre L, Renard C, Bacchetta J, Goutagny MP, Hu J, Carla de Souza V, Bertrand Y, Dubourg L, Domenech C. Teenagers and young adults with a past of allogenic hematopoietic stem cell transplantation are at significant risk of chronic kidney disease. Pediatr Nephrol. 2021 Nov 04; doi: 10.1007/s00467-021-05319-x. Epub 2021 Nov 04. PMID: 34735600.
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