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Showing 1 to 11 of 11 entries
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Applications of multi-nuclear magnetic resonance spectroscopy at 7T.

World journal of radiology

Stephenson MC, Gunner F, Napolitano A, Greenhaff PL, Macdonald IA, Saeed N, Vennart W, Francis ST, Morris PG.
PMID: 21532871
World J Radiol. 2011 Apr 28;3(4):105-13. doi: 10.4329/wjr.v3.i4.105.

AIM: To discuss the advantages of ultra-high field (7T) for (1)H and (13)C magnetic resonance spectroscopy (MRS) studies of metabolism.METHODS: Measurements of brain metabolites were made at both 3 and 7T using (1)H MRS. Measurements of glycogen and lipids...

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Stephenson MC, Gunner F, Napolitano A, et al. Applications of multi-nuclear magnetic resonance spectroscopy at 7T. World J Radiol. 2011;3(4):105-13doi: 10.4329/wjr.v3.i4.105.
Stephenson, M. C., Gunner, F., Napolitano, A., Greenhaff, P. L., Macdonald, I. A., Saeed, N., Vennart, W., Francis, S. T., & Morris, P. G. (2011). Applications of multi-nuclear magnetic resonance spectroscopy at 7T. World journal of radiology, 3(4), 105-13. https://doi.org/10.4329/wjr.v3.i4.105
Stephenson, Mary C, et al. "Applications of multi-nuclear magnetic resonance spectroscopy at 7T." World journal of radiology vol. 3,4 (2011): 105-13. doi: https://doi.org/10.4329/wjr.v3.i4.105
Stephenson MC, Gunner F, Napolitano A, Greenhaff PL, Macdonald IA, Saeed N, Vennart W, Francis ST, Morris PG. Applications of multi-nuclear magnetic resonance spectroscopy at 7T. World J Radiol. 2011 Apr 28;3(4):105-13. doi: 10.4329/wjr.v3.i4.105. PMID: 21532871; PMCID: PMC3084434.
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Potential role for pyruvate kinase M2 in the regulation of murine cardiac glycolytic flux during in vivo chronic hypoxia.

Bioscience reports

Handzlik MK, Tooth DJ, Constantin-Teodosiu D, Greenhaff PL, Cole MA.
PMID: 33973628
Biosci Rep. 2021 Jun 25;41(6). doi: 10.1042/BSR20203170.

Carbohydrate metabolism in heart failure shares similarities to that following hypoxic exposure, and is thought to maintain energy homoeostasis in the face of reduced O2 availability. As part of these in vivo adaptations during sustained hypoxia, the heart up-regulates...

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Handzlik MK, Tooth DJ, Constantin-Teodosiu D, et al. Potential role for pyruvate kinase M2 in the regulation of murine cardiac glycolytic flux during in vivo chronic hypoxia. Biosci Rep. 2021;41(6)doi: 10.1042/BSR20203170.
Handzlik, M. K., Tooth, D. J., Constantin-Teodosiu, D., Greenhaff, P. L., & Cole, M. A. (2021). Potential role for pyruvate kinase M2 in the regulation of murine cardiac glycolytic flux during in vivo chronic hypoxia. Bioscience reports, 41(6), . https://doi.org/10.1042/BSR20203170
Handzlik, Michal K, et al. "Potential role for pyruvate kinase M2 in the regulation of murine cardiac glycolytic flux during in vivo chronic hypoxia." Bioscience reports vol. 41,6 (2021). doi: https://doi.org/10.1042/BSR20203170
Handzlik MK, Tooth DJ, Constantin-Teodosiu D, Greenhaff PL, Cole MA. Potential role for pyruvate kinase M2 in the regulation of murine cardiac glycolytic flux during in vivo chronic hypoxia. Biosci Rep. 2021 Jun 25;41(6). doi: 10.1042/BSR20203170. PMID: 33973628; PMCID: PMC8173528.
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PPARα, δ and FOXO1 Gene Silencing Overturns Palmitate-Induced Inhibition of Pyruvate Oxidation Differentially in C2C12 Myotubes.

Biology

Chien HC, Constantin D, Greenhaff PL, Constantin-Teodosiu D.
PMID: 34827089
Biology (Basel). 2021 Oct 25;10(11). doi: 10.3390/biology10111098.

The molecular mechanisms by which free fatty acids (FFA) inhibit muscle glucose oxidation is still elusive. We recently showed that C2C12 myotubes treated with palmitate (PAL) presented with greater protein expression levels of PDK4 and transcription factors

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Chien HC, Constantin D, Greenhaff PL, et al. PPARα, δ and FOXO1 Gene Silencing Overturns Palmitate-Induced Inhibition of Pyruvate Oxidation Differentially in C2C12 Myotubes. Biology (Basel). 2021;10(11)doi: 10.3390/biology10111098.
Chien, H. C., Constantin, D., Greenhaff, P. L., & Constantin-Teodosiu, D. (2021). PPARα, δ and FOXO1 Gene Silencing Overturns Palmitate-Induced Inhibition of Pyruvate Oxidation Differentially in C2C12 Myotubes. Biology, 10(11), . https://doi.org/10.3390/biology10111098
Chien, Hung-Che, et al. "PPARα, δ and FOXO1 Gene Silencing Overturns Palmitate-Induced Inhibition of Pyruvate Oxidation Differentially in C2C12 Myotubes." Biology vol. 10,11 (2021). doi: https://doi.org/10.3390/biology10111098
Chien HC, Constantin D, Greenhaff PL, Constantin-Teodosiu D. PPARα, δ and FOXO1 Gene Silencing Overturns Palmitate-Induced Inhibition of Pyruvate Oxidation Differentially in C2C12 Myotubes. Biology (Basel). 2021 Oct 25;10(11). doi: 10.3390/biology10111098. PMID: 34827089; PMCID: PMC8614693.
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Antecubital Vein Cannula Position Impacts Assessment of Forearm Glucose Uptake During an Oral Glucose Challenge in Healthy Volunteers.

Experimental and clinical endocrinology & diabetes : official journal, German Society of Endocrinology [and] German Diabetes Association

Bétry C, Nixon AV, Greenhaff PL, Simpson EJ.
PMID: 33096578
Exp Clin Endocrinol Diabetes. 2020 Oct 23; doi: 10.1055/a-1275-4038. Epub 2020 Oct 23.

INTRODUCTION: Skeletal muscle is a major site for whole-body glucose disposal, and determination of skeletal muscle glucose uptake is an important metabolic measurement, particularly in research focussed on interventions that impact muscle insulin sensitivity. Calculating arterial-venous difference in blood...

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Bétry C, Nixon AV, Greenhaff PL, et al. Antecubital Vein Cannula Position Impacts Assessment of Forearm Glucose Uptake During an Oral Glucose Challenge in Healthy Volunteers. Exp Clin Endocrinol Diabetes. 2020;doi: 10.1055/a-1275-4038.
Bétry, C., Nixon, A. V., Greenhaff, P. L., & Simpson, E. J. (2020). Antecubital Vein Cannula Position Impacts Assessment of Forearm Glucose Uptake During an Oral Glucose Challenge in Healthy Volunteers. Experimental and clinical endocrinology & diabetes : official journal, German Society of Endocrinology [and] German Diabetes Association, . https://doi.org/10.1055/a-1275-4038
Bétry, Cécile, et al. "Antecubital Vein Cannula Position Impacts Assessment of Forearm Glucose Uptake During an Oral Glucose Challenge in Healthy Volunteers." Experimental and clinical endocrinology & diabetes : official journal, German Society of Endocrinology [and] German Diabetes Association vol. (2020). doi: https://doi.org/10.1055/a-1275-4038
Bétry C, Nixon AV, Greenhaff PL, Simpson EJ. Antecubital Vein Cannula Position Impacts Assessment of Forearm Glucose Uptake During an Oral Glucose Challenge in Healthy Volunteers. Exp Clin Endocrinol Diabetes. 2020 Oct 23; doi: 10.1055/a-1275-4038. Epub 2020 Oct 23. PMID: 33096578.
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Degenerin channel activation causes caspase-mediated protein degradation and mitochondrial dysfunction in adult C. elegans muscle.

Journal of cachexia, sarcopenia and muscle

Gaffney CJ, Shephard F, Chu J, Baillie DL, Rose A, Constantin-Teodosiu D, Greenhaff PL, Szewczyk NJ.
PMID: 27493871
J Cachexia Sarcopenia Muscle. 2016 May;7(2):181-92. doi: 10.1002/jcsm.12040. Epub 2015 Jun 04.

BACKGROUND: Declines in skeletal muscle structure and function are found in various clinical populations, but the intramuscular proteolytic pathways that govern declines in these individuals remain relatively poorly understood. The nematode Caenorhabditis elegans has been developed into a model...

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Gaffney CJ, Shephard F, Chu J, et al. Degenerin channel activation causes caspase-mediated protein degradation and mitochondrial dysfunction in adult C. elegans muscle. J Cachexia Sarcopenia Muscle. 2015;7(2):181-92doi: 10.1002/jcsm.12040.
Gaffney, C. J., Shephard, F., Chu, J., Baillie, D. L., Rose, A., Constantin-Teodosiu, D., Greenhaff, P. L., & Szewczyk, N. J. (2016). Degenerin channel activation causes caspase-mediated protein degradation and mitochondrial dysfunction in adult C. elegans muscle. Journal of cachexia, sarcopenia and muscle, 7(2), 181-92. https://doi.org/10.1002/jcsm.12040
Gaffney, Christopher J, et al. "Degenerin channel activation causes caspase-mediated protein degradation and mitochondrial dysfunction in adult C. elegans muscle." Journal of cachexia, sarcopenia and muscle vol. 7,2 (2016): 181-92. doi: https://doi.org/10.1002/jcsm.12040
Gaffney CJ, Shephard F, Chu J, Baillie DL, Rose A, Constantin-Teodosiu D, Greenhaff PL, Szewczyk NJ. Degenerin channel activation causes caspase-mediated protein degradation and mitochondrial dysfunction in adult C. elegans muscle. J Cachexia Sarcopenia Muscle. 2016 May;7(2):181-92. doi: 10.1002/jcsm.12040. Epub 2015 Jun 04. PMID: 27493871; PMCID: PMC4864282.
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Metabolomics as an Important Tool for Determining the Mechanisms of Human Skeletal Muscle Deconditioning.

International journal of molecular sciences

Alldritt I, Greenhaff PL, Wilkinson DJ.
PMID: 34948370
Int J Mol Sci. 2021 Dec 17;22(24). doi: 10.3390/ijms222413575.

Muscle deconditioning impairs both locomotor function and metabolic health, and is associated with reduced quality life and increased mortality rates. Despite an appreciation of the existence of phenomena such as muscle anabolic resistance, mitophagy, and insulin resistance with age...

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Alldritt I, Greenhaff PL, Wilkinson DJ. Metabolomics as an Important Tool for Determining the Mechanisms of Human Skeletal Muscle Deconditioning. Int J Mol Sci. 2021;22(24)doi: 10.3390/ijms222413575.
Alldritt, I., Greenhaff, P. L., & Wilkinson, D. J. (2021). Metabolomics as an Important Tool for Determining the Mechanisms of Human Skeletal Muscle Deconditioning. International journal of molecular sciences, 22(24), . https://doi.org/10.3390/ijms222413575
Alldritt, Isabelle, et al. "Metabolomics as an Important Tool for Determining the Mechanisms of Human Skeletal Muscle Deconditioning." International journal of molecular sciences vol. 22,24 (2021). doi: https://doi.org/10.3390/ijms222413575
Alldritt I, Greenhaff PL, Wilkinson DJ. Metabolomics as an Important Tool for Determining the Mechanisms of Human Skeletal Muscle Deconditioning. Int J Mol Sci. 2021 Dec 17;22(24). doi: 10.3390/ijms222413575. PMID: 34948370.
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Metabolomics as an Important Tool for Determining the Mechanisms of Human Skeletal Muscle Deconditioning.

International journal of molecular sciences

Alldritt I, Greenhaff PL, Wilkinson DJ.
PMID: 34948370
Int J Mol Sci. 2021 Dec 17;22(24). doi: 10.3390/ijms222413575.

Muscle deconditioning impairs both locomotor function and metabolic health, and is associated with reduced quality life and increased mortality rates. Despite an appreciation of the existence of phenomena such as muscle anabolic resistance, mitophagy, and insulin resistance with age...

Cite
Alldritt I, Greenhaff PL, Wilkinson DJ. Metabolomics as an Important Tool for Determining the Mechanisms of Human Skeletal Muscle Deconditioning. Int J Mol Sci. 2021;22(24)doi: 10.3390/ijms222413575.
Alldritt, I., Greenhaff, P. L., & Wilkinson, D. J. (2021). Metabolomics as an Important Tool for Determining the Mechanisms of Human Skeletal Muscle Deconditioning. International journal of molecular sciences, 22(24), . https://doi.org/10.3390/ijms222413575
Alldritt, Isabelle, et al. "Metabolomics as an Important Tool for Determining the Mechanisms of Human Skeletal Muscle Deconditioning." International journal of molecular sciences vol. 22,24 (2021). doi: https://doi.org/10.3390/ijms222413575
Alldritt I, Greenhaff PL, Wilkinson DJ. Metabolomics as an Important Tool for Determining the Mechanisms of Human Skeletal Muscle Deconditioning. Int J Mol Sci. 2021 Dec 17;22(24). doi: 10.3390/ijms222413575. PMID: 34948370; PMCID: PMC8706620.
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Human Skeletal Muscle Disuse Atrophy: Effects on Muscle Protein Synthesis, Breakdown, and Insulin Resistance-A Qualitative Review.

Frontiers in physiology

Rudrappa SS, Wilkinson DJ, Greenhaff PL, Smith K, Idris I, Atherton PJ.
PMID: 27610086
Front Physiol. 2016 Aug 25;7:361. doi: 10.3389/fphys.2016.00361. eCollection 2016.

The ever increasing burden of an aging population and pandemic of metabolic syndrome worldwide demands further understanding of the modifiable risk factors in reducing disability and morbidity associated with these conditions. Disuse skeletal muscle atrophy (sometimes referred to as...

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Rudrappa SS, Wilkinson DJ, Greenhaff PL, et al. Human Skeletal Muscle Disuse Atrophy: Effects on Muscle Protein Synthesis, Breakdown, and Insulin Resistance-A Qualitative Review. Front Physiol. 2016;7:361doi: 10.3389/fphys.2016.00361.
Rudrappa, S. S., Wilkinson, D. J., Greenhaff, P. L., Smith, K., Idris, I., & Atherton, P. J. (2016). Human Skeletal Muscle Disuse Atrophy: Effects on Muscle Protein Synthesis, Breakdown, and Insulin Resistance-A Qualitative Review. Frontiers in physiology, 7361. https://doi.org/10.3389/fphys.2016.00361
Rudrappa, Supreeth S, et al. "Human Skeletal Muscle Disuse Atrophy: Effects on Muscle Protein Synthesis, Breakdown, and Insulin Resistance-A Qualitative Review." Frontiers in physiology vol. 7 (2016): 361. doi: https://doi.org/10.3389/fphys.2016.00361
Rudrappa SS, Wilkinson DJ, Greenhaff PL, Smith K, Idris I, Atherton PJ. Human Skeletal Muscle Disuse Atrophy: Effects on Muscle Protein Synthesis, Breakdown, and Insulin Resistance-A Qualitative Review. Front Physiol. 2016 Aug 25;7:361. doi: 10.3389/fphys.2016.00361. eCollection 2016. PMID: 27610086; PMCID: PMC4997013.
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London 2012: the biomedical basis of elite performance.

The Journal of physiology

Greenhaff P.
PMID: 22399821
J Physiol. 2012 Mar 01;590(5):1047. doi: 10.1113/jphysiol.2012.229617.

No abstract available.

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Greenhaff P. London 2012: the biomedical basis of elite performance. J Physiol. 2012;590(5):1047doi: 10.1113/jphysiol.2012.229617.
Greenhaff, P. (2012). London 2012: the biomedical basis of elite performance. The Journal of physiology, 590(5), 1047. https://doi.org/10.1113/jphysiol.2012.229617
Greenhaff, Paul. "London 2012: the biomedical basis of elite performance." The Journal of physiology vol. 590,5 (2012): 1047. doi: https://doi.org/10.1113/jphysiol.2012.229617
Greenhaff P. London 2012: the biomedical basis of elite performance. J Physiol. 2012 Mar 01;590(5):1047. doi: 10.1113/jphysiol.2012.229617. PMID: 22399821; PMCID: PMC3381812.
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Whole-body & muscle responses to aerobic exercise training and withdrawal in ageing & COPD.

The European respiratory journal

Latimer LE, Constantin-Teodosiu D, Popat B, Constantin D, Houchen-Wolloff L, Bolton CE, Steiner MC, Greenhaff PL, Harrison JR, Bunford N, Evans SW.
PMID: 34588196
Eur Respir J. 2021 Sep 29; doi: 10.1183/13993003.01507-2021. Epub 2021 Sep 29.

COPD patients exhibit lower peak oxygen consumption (V̇O

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Latimer LE, Constantin-Teodosiu D, Popat B, et al. Whole-body & muscle responses to aerobic exercise training and withdrawal in ageing & COPD. Eur Respir J. 2021;doi: 10.1183/13993003.01507-2021.
Latimer, L. E., Constantin-Teodosiu, D., Popat, B., Constantin, D., Houchen-Wolloff, L., Bolton, C. E., Steiner, M. C., Greenhaff, P. L., Harrison, J. R., Bunford, N., & Evans, S. W. (2021). Whole-body & muscle responses to aerobic exercise training and withdrawal in ageing & COPD. The European respiratory journal, . https://doi.org/10.1183/13993003.01507-2021
Latimer, Lorna E, et al. "Whole-body & muscle responses to aerobic exercise training and withdrawal in ageing & COPD." The European respiratory journal vol. (2021). doi: https://doi.org/10.1183/13993003.01507-2021
Latimer LE, Constantin-Teodosiu D, Popat B, Constantin D, Houchen-Wolloff L, Bolton CE, Steiner MC, Greenhaff PL, Harrison JR, Bunford N, Evans SW. Whole-body & muscle responses to aerobic exercise training and withdrawal in ageing & COPD. Eur Respir J. 2021 Sep 29; doi: 10.1183/13993003.01507-2021. Epub 2021 Sep 29. PMID: 34588196.
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Antecubital Vein Cannula Position Impacts Assessment of Forearm Glucose Uptake During an Oral Glucose Challenge in Healthy Volunteers.

Experimental and clinical endocrinology & diabetes : official journal, German Society of Endocrinology [and] German Diabetes Association

Bétry C, Nixon AV, Greenhaff PL, Simpson EJ.
PMID: 33096578
Exp Clin Endocrinol Diabetes. 2022 Jan;130(1):49-54. doi: 10.1055/a-1275-4038. Epub 2020 Oct 23.

INTRODUCTION: Skeletal muscle is a major site for whole-body glucose disposal, and determination of skeletal muscle glucose uptake is an important metabolic measurement, particularly in research focussed on interventions that impact muscle insulin sensitivity. Calculating arterial-venous difference in blood...

Cite
Bétry C, Nixon AV, Greenhaff PL, et al. Antecubital Vein Cannula Position Impacts Assessment of Forearm Glucose Uptake During an Oral Glucose Challenge in Healthy Volunteers. Exp Clin Endocrinol Diabetes. 2020;130(1):49-54doi: 10.1055/a-1275-4038.
Bétry, C., Nixon, A. V., Greenhaff, P. L., & Simpson, E. J. (2022). Antecubital Vein Cannula Position Impacts Assessment of Forearm Glucose Uptake During an Oral Glucose Challenge in Healthy Volunteers. Experimental and clinical endocrinology & diabetes : official journal, German Society of Endocrinology [and] German Diabetes Association, 130(1), 49-54. https://doi.org/10.1055/a-1275-4038
Bétry, Cécile, et al. "Antecubital Vein Cannula Position Impacts Assessment of Forearm Glucose Uptake During an Oral Glucose Challenge in Healthy Volunteers." Experimental and clinical endocrinology & diabetes : official journal, German Society of Endocrinology [and] German Diabetes Association vol. 130,1 (2022): 49-54. doi: https://doi.org/10.1055/a-1275-4038
Bétry C, Nixon AV, Greenhaff PL, Simpson EJ. Antecubital Vein Cannula Position Impacts Assessment of Forearm Glucose Uptake During an Oral Glucose Challenge in Healthy Volunteers. Exp Clin Endocrinol Diabetes. 2022 Jan;130(1):49-54. doi: 10.1055/a-1275-4038. Epub 2020 Oct 23. PMID: 33096578.
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Showing 1 to 11 of 11 entries