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Mechanical ventilation preserves diaphragm mitochondrial function in a rat sepsis model.

Intensive care medicine experimental

Eyenga P, Roussel D, Rey B, Ndille P, Teulier L, Eyenga F, Romestaing C, Morel J, Gueguen-Chaignon V, Sheu SS.
PMID: 33825987
Intensive Care Med Exp. 2021 Apr 07;9(1):19. doi: 10.1186/s40635-021-00384-w.

BACKGROUND: To describe the effect of mechanical ventilation on diaphragm mitochondrial oxygen consumption, ATP production, reactive oxygen species (ROS) generation, and cytochrome c oxidase activity and content, and their relationship to diaphragm strength in an experimental model of sepsis.METHODS:...

Cite
Eyenga P, Roussel D, Rey B, et al. Mechanical ventilation preserves diaphragm mitochondrial function in a rat sepsis model. Intensive Care Med Exp. 2021;9(1):19doi: 10.1186/s40635-021-00384-w.
Eyenga, P., Roussel, D., Rey, B., Ndille, P., Teulier, L., Eyenga, F., Romestaing, C., Morel, J., Gueguen-Chaignon, V., & Sheu, S. S. (2021). Mechanical ventilation preserves diaphragm mitochondrial function in a rat sepsis model. Intensive care medicine experimental, 9(1), 19. https://doi.org/10.1186/s40635-021-00384-w
Eyenga, P, et al. "Mechanical ventilation preserves diaphragm mitochondrial function in a rat sepsis model." Intensive care medicine experimental vol. 9,1 (2021): 19. doi: https://doi.org/10.1186/s40635-021-00384-w
Eyenga P, Roussel D, Rey B, Ndille P, Teulier L, Eyenga F, Romestaing C, Morel J, Gueguen-Chaignon V, Sheu SS. Mechanical ventilation preserves diaphragm mitochondrial function in a rat sepsis model. Intensive Care Med Exp. 2021 Apr 07;9(1):19. doi: 10.1186/s40635-021-00384-w. PMID: 33825987; PMCID: PMC8025065.
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Time course of liver mitochondrial function and intrinsic changes in oxidative phosphorylation in a rat model of sepsis.

Intensive care medicine experimental

Eyenga P, Roussel D, Morel J, Rey B, Romestaing C, Gueguen-Chaignon V, Sheu SS, Viale JP.
PMID: 30187255
Intensive Care Med Exp. 2018 Sep 05;6(1):31. doi: 10.1186/s40635-018-0197-y.

BACKGROUND: Tissue ATP depletion and oxidative stress have been associated with the severe outcomes of septic shock. One of the compensatory mechanisms to alleviate the sepsis-induced mitochondrial dysfunction could be the increase in oxidative phosphorylation efficiency (ATP/O). We propose...

Cite
Eyenga P, Roussel D, Morel J, et al. Time course of liver mitochondrial function and intrinsic changes in oxidative phosphorylation in a rat model of sepsis. Intensive Care Med Exp. 2018;6(1):31doi: 10.1186/s40635-018-0197-y.
Eyenga, P., Roussel, D., Morel, J., Rey, B., Romestaing, C., Gueguen-Chaignon, V., Sheu, S. S., & Viale, J. P. (2018). Time course of liver mitochondrial function and intrinsic changes in oxidative phosphorylation in a rat model of sepsis. Intensive care medicine experimental, 6(1), 31. https://doi.org/10.1186/s40635-018-0197-y
Eyenga, Pierre, et al. "Time course of liver mitochondrial function and intrinsic changes in oxidative phosphorylation in a rat model of sepsis." Intensive care medicine experimental vol. 6,1 (2018): 31. doi: https://doi.org/10.1186/s40635-018-0197-y
Eyenga P, Roussel D, Morel J, Rey B, Romestaing C, Gueguen-Chaignon V, Sheu SS, Viale JP. Time course of liver mitochondrial function and intrinsic changes in oxidative phosphorylation in a rat model of sepsis. Intensive Care Med Exp. 2018 Sep 05;6(1):31. doi: 10.1186/s40635-018-0197-y. PMID: 30187255; PMCID: PMC6125261.
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Showing 1 to 2 of 2 entries