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Robert Bentley Todd, Saint John's House, and the origins of the modern trained nurse.

Bulletin of the history of medicine

Helmstadter C.
PMID: 8329858
Bull Hist Med. 1993;67(2):282-319.

No abstract available.

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Helmstadter C. Robert Bentley Todd, Saint John's House, and the origins of the modern trained nurse. Bull Hist Med. 1993;67(2):282-319
Helmstadter, C. (1993). Robert Bentley Todd, Saint John's House, and the origins of the modern trained nurse. Bulletin of the history of medicine, 67(2), 282-319.
Helmstadter, C. "Robert Bentley Todd, Saint John's House, and the origins of the modern trained nurse." Bulletin of the history of medicine vol. 67,2 (1993): 282-319.
Helmstadter C. Robert Bentley Todd, Saint John's House, and the origins of the modern trained nurse. Bull Hist Med. 1993;67(2):282-319. PMID: 8329858.
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Self-Reported Mood Changes following 6 Months of Vagus Nerve Stimulation in Epilepsy Patients.

Epilepsy & behavior : E&B

Hoppe C, Helmstaedter C, Scherrmann J, Elger CE.
PMID: 12609210
Epilepsy Behav. 2001 Aug;2(4):335-42. doi: 10.1006/ebeh.2001.0194.

Vagus nerve stimulation (VNS) for treatment of drug-resistant epileptic seizures has been reported to have additional positive mood effects as obtained by psychiatric ratings. To avoid rater bias effects, this study used self-report questionnaires and examined changes in self-reported...

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Hoppe C, Helmstaedter C, Scherrmann J, et al. Self-Reported Mood Changes following 6 Months of Vagus Nerve Stimulation in Epilepsy Patients. Epilepsy Behav. 2001;2(4):335-42doi: 10.1006/ebeh.2001.0194.
Hoppe, C., Helmstaedter, C., Scherrmann, J., & Elger, C. E. (2001). Self-Reported Mood Changes following 6 Months of Vagus Nerve Stimulation in Epilepsy Patients. Epilepsy & behavior : E&B, 2(4), 335-42. https://doi.org/10.1006/ebeh.2001.0194
Hoppe, C, et al. "Self-Reported Mood Changes following 6 Months of Vagus Nerve Stimulation in Epilepsy Patients." Epilepsy & behavior : E&B vol. 2,4 (2001): 335-42. doi: https://doi.org/10.1006/ebeh.2001.0194
Hoppe C, Helmstaedter C, Scherrmann J, Elger CE. Self-Reported Mood Changes following 6 Months of Vagus Nerve Stimulation in Epilepsy Patients. Epilepsy Behav. 2001 Aug;2(4):335-42. doi: 10.1006/ebeh.2001.0194. PMID: 12609210.
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Shifting boundaries: religion, medicine, nursing and domestic service in mid-nineteenth-century Britain.

Nursing inquiry

Helmstadter C.
PMID: 19453358
Nurs Inq. 2009 Jun;16(2):133-43. doi: 10.1111/j.1440-1800.2009.00460.x.

The boundaries between medicine, religion, nursing and domestic service were fluid in mid-nineteenth-century England. The traditional religious understanding of illness conflicted with the newer understanding of anatomically based disease, the Anglican sisters were drawing a line between professional nursing...

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Helmstadter C. Shifting boundaries: religion, medicine, nursing and domestic service in mid-nineteenth-century Britain. Nurs Inq. 2009;16(2):133-43doi: 10.1111/j.1440-1800.2009.00460.x.
Helmstadter, C. (2009). Shifting boundaries: religion, medicine, nursing and domestic service in mid-nineteenth-century Britain. Nursing inquiry, 16(2), 133-43. https://doi.org/10.1111/j.1440-1800.2009.00460.x
Helmstadter, Carol. "Shifting boundaries: religion, medicine, nursing and domestic service in mid-nineteenth-century Britain." Nursing inquiry vol. 16,2 (2009): 133-43. doi: https://doi.org/10.1111/j.1440-1800.2009.00460.x
Helmstadter C. Shifting boundaries: religion, medicine, nursing and domestic service in mid-nineteenth-century Britain. Nurs Inq. 2009 Jun;16(2):133-43. doi: 10.1111/j.1440-1800.2009.00460.x. PMID: 19453358.
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Treating a GAD65 Antibody-Associated Limbic Encephalitis with Basiliximab: A Case Study.

Frontiers in neurology

Widman G, Golombeck K, Hautzel H, Gross CC, Quesada CM, Witt JA, Rota-Kops E, Ermert J, Greschus S, Surges R, Helmstaedter C, Wiendl H, Melzer N, Elger CE.
PMID: 26284025
Front Neurol. 2015 Aug 03;6:167. doi: 10.3389/fneur.2015.00167. eCollection 2015.

BACKGROUND: Antibodies (ABs) against the 65-kDa isoform of the intracellular enzyme glutamate decarboxylase (GAD65) have been found in limbic encephalitis (LE) and other neurological conditions. The direct significance of anti-GAD65-ABs for epilepsy is unclear. However, in histological preparations from...

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Widman G, Golombeck K, Hautzel H, et al. Treating a GAD65 Antibody-Associated Limbic Encephalitis with Basiliximab: A Case Study. Front Neurol. 2015;6:167doi: 10.3389/fneur.2015.00167.
Widman, G., Golombeck, K., Hautzel, H., Gross, C. C., Quesada, C. M., Witt, J. A., Rota-Kops, E., Ermert, J., Greschus, S., Surges, R., Helmstaedter, C., Wiendl, H., Melzer, N., & Elger, C. E. (2015). Treating a GAD65 Antibody-Associated Limbic Encephalitis with Basiliximab: A Case Study. Frontiers in neurology, 6167. https://doi.org/10.3389/fneur.2015.00167
Widman, Guido, et al. "Treating a GAD65 Antibody-Associated Limbic Encephalitis with Basiliximab: A Case Study." Frontiers in neurology vol. 6 (2015): 167. doi: https://doi.org/10.3389/fneur.2015.00167
Widman G, Golombeck K, Hautzel H, Gross CC, Quesada CM, Witt JA, Rota-Kops E, Ermert J, Greschus S, Surges R, Helmstaedter C, Wiendl H, Melzer N, Elger CE. Treating a GAD65 Antibody-Associated Limbic Encephalitis with Basiliximab: A Case Study. Front Neurol. 2015 Aug 03;6:167. doi: 10.3389/fneur.2015.00167. eCollection 2015. PMID: 26284025; PMCID: PMC4522569.
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Using the .

Frontiers in pediatrics

Helmstädter M, Huber TB, Hermle T.
PMID: 29270398
Front Pediatr. 2017 Dec 07;5:262. doi: 10.3389/fped.2017.00262. eCollection 2017.

Glomerular disorders are a major cause of end-stage renal disease and effective therapies are often lacking. Nephrocytes are considered to be part of the

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Helmstädter M, Huber TB, Hermle T. Using the . Front Pediatr. 2017;5:262doi: 10.3389/fped.2017.00262.
Helmstädter, M., Huber, T. B., & Hermle, T. (2017). Using the . Frontiers in pediatrics, 5262. https://doi.org/10.3389/fped.2017.00262
Helmstädter, Martin, et al. "Using the ." Frontiers in pediatrics vol. 5 (2017): 262. doi: https://doi.org/10.3389/fped.2017.00262
Helmstädter M, Huber TB, Hermle T. Using the . Front Pediatr. 2017 Dec 07;5:262. doi: 10.3389/fped.2017.00262. eCollection 2017. PMID: 29270398; PMCID: PMC5725439.
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Neuropsychological features of mind wandering in left-, right- and extra temporal lobe epilepsy.

Seizure

Luelsberg F, Krakau S, Chaieb L, Witt JA, von Wrede R, Fell J, Helmstaedter C.
PMID: 34998088
Seizure. 2021 Dec 31;95:50-55. doi: 10.1016/j.seizure.2021.12.013. Epub 2021 Dec 31.

PURPOSE: Mind wandering, i.e. mental time-travelling and imagery unrelated to the current situation has recently been related to mesial temporal lobe (memory) function. In this regard we evaluated as to whether parameters of mind wandering are related to material...

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Luelsberg F, Krakau S, Chaieb L, et al. Neuropsychological features of mind wandering in left-, right- and extra temporal lobe epilepsy. Seizure. 2021;95:50-55doi: 10.1016/j.seizure.2021.12.013.
Luelsberg, F., Krakau, S., Chaieb, L., Witt, J. A., von Wrede, R., Fell, J., & Helmstaedter, C. (2021). Neuropsychological features of mind wandering in left-, right- and extra temporal lobe epilepsy. Seizure, 9550-55. https://doi.org/10.1016/j.seizure.2021.12.013
Luelsberg, Fabienne, et al. "Neuropsychological features of mind wandering in left-, right- and extra temporal lobe epilepsy." Seizure vol. 95 (2021): 50-55. doi: https://doi.org/10.1016/j.seizure.2021.12.013
Luelsberg F, Krakau S, Chaieb L, Witt JA, von Wrede R, Fell J, Helmstaedter C. Neuropsychological features of mind wandering in left-, right- and extra temporal lobe epilepsy. Seizure. 2021 Dec 31;95:50-55. doi: 10.1016/j.seizure.2021.12.013. Epub 2021 Dec 31. PMID: 34998088.
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EPB41L5 controls podocyte extracellular matrix assembly by adhesome-dependent force transmission.

Cell reports

Maier JI, Rogg M, Helmstädter M, Sammarco A, Schilling O, Sabass B, Miner JH, Dengjel J, Walz G, Werner M, Huber TB, Schell C.
PMID: 33761352
Cell Rep. 2021 Mar 23;34(12):108883. doi: 10.1016/j.celrep.2021.108883.

The integrity of the kidney filtration barrier essentially relies on the balanced interplay of podocytes and the glomerular basement membrane (GBM). Here, we show by analysis of in vitro and in vivo models that a loss of the podocyte-specific...

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Maier JI, Rogg M, Helmstädter M, et al. EPB41L5 controls podocyte extracellular matrix assembly by adhesome-dependent force transmission. Cell Rep. 2021;34(12):108883doi: 10.1016/j.celrep.2021.108883.
Maier, J. I., Rogg, M., Helmstädter, M., Sammarco, A., Schilling, O., Sabass, B., Miner, J. H., Dengjel, J., Walz, G., Werner, M., Huber, T. B., & Schell, C. (2021). EPB41L5 controls podocyte extracellular matrix assembly by adhesome-dependent force transmission. Cell reports, 34(12), 108883. https://doi.org/10.1016/j.celrep.2021.108883
Maier, Jasmin I, et al. "EPB41L5 controls podocyte extracellular matrix assembly by adhesome-dependent force transmission." Cell reports vol. 34,12 (2021): 108883. doi: https://doi.org/10.1016/j.celrep.2021.108883
Maier JI, Rogg M, Helmstädter M, Sammarco A, Schilling O, Sabass B, Miner JH, Dengjel J, Walz G, Werner M, Huber TB, Schell C. EPB41L5 controls podocyte extracellular matrix assembly by adhesome-dependent force transmission. Cell Rep. 2021 Mar 23;34(12):108883. doi: 10.1016/j.celrep.2021.108883. PMID: 33761352.
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Archiv der Pharmazie-200 years.

Archiv der Pharmazie

Friedrich C, Helmstädter A.
PMID: 34738657
Arch Pharm (Weinheim). 2022 Jan;355(1):e2100392. doi: 10.1002/ardp.202100392. Epub 2021 Nov 04.

No abstract available.

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Friedrich C, Helmstädter A. Archiv der Pharmazie-200 years. Arch Pharm (Weinheim). 2021;355(1):e2100392doi: 10.1002/ardp.202100392.
Friedrich, C., & Helmstädter, A. (2022). Archiv der Pharmazie-200 years. Archiv der Pharmazie, 355(1), e2100392. https://doi.org/10.1002/ardp.202100392
Friedrich, Christoph, and Helmstädter, Axel. "Archiv der Pharmazie-200 years." Archiv der Pharmazie vol. 355,1 (2022): e2100392. doi: https://doi.org/10.1002/ardp.202100392
Friedrich C, Helmstädter A. Archiv der Pharmazie-200 years. Arch Pharm (Weinheim). 2022 Jan;355(1):e2100392. doi: 10.1002/ardp.202100392. Epub 2021 Nov 04. PMID: 34738657.
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Structure-Based Design of Dual Partial Peroxisome Proliferator-Activated Receptor γ Agonists/Soluble Epoxide Hydrolase Inhibitors.

Journal of medicinal chemistry

Lillich FF, Willems S, Ni X, Kilu W, Borkowsky C, Brodsky M, Kramer JS, Brunst S, Hernandez-Olmos V, Heering J, Schierle S, Kestner RI, Mayser FM, Helmstädter M, Göbel T, Weizel L, Namgaladze D, Kaiser A, Steinhilber D, Pfeilschifter W, Kahnt AS, Proschak A, Chaikuad A, Knapp S, Merk D, Proschak E.
PMID: 34818007
J Med Chem. 2021 Dec 09;64(23):17259-17276. doi: 10.1021/acs.jmedchem.1c01331. Epub 2021 Nov 24.

Polypharmaceutical regimens often impair treatment of patients with metabolic syndrome (MetS), a complex disease cluster, including obesity, hypertension, heart disease, and type II diabetes. Simultaneous targeting of soluble epoxide hydrolase (sEH) and peroxisome proliferator-activated receptor γ (PPARγ) synergistically counteracted...

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Lillich FF, Willems S, Ni X, et al. Structure-Based Design of Dual Partial Peroxisome Proliferator-Activated Receptor γ Agonists/Soluble Epoxide Hydrolase Inhibitors. J Med Chem. 2021;64(23):17259-17276doi: 10.1021/acs.jmedchem.1c01331.
Lillich, F. F., Willems, S., Ni, X., Kilu, W., Borkowsky, C., Brodsky, M., Kramer, J. S., Brunst, S., Hernandez-Olmos, V., Heering, J., Schierle, S., Kestner, R. I., Mayser, F. M., Helmstädter, M., Göbel, T., Weizel, L., Namgaladze, D., Kaiser, A., Steinhilber, D., Pfeilschifter, W., Kahnt, A. S., Proschak, A., Chaikuad, A., Knapp, S., Merk, D., & Proschak, E. (2021). Structure-Based Design of Dual Partial Peroxisome Proliferator-Activated Receptor γ Agonists/Soluble Epoxide Hydrolase Inhibitors. Journal of medicinal chemistry, 64(23), 17259-17276. https://doi.org/10.1021/acs.jmedchem.1c01331
Lillich, Felix F, et al. "Structure-Based Design of Dual Partial Peroxisome Proliferator-Activated Receptor γ Agonists/Soluble Epoxide Hydrolase Inhibitors." Journal of medicinal chemistry vol. 64,23 (2021): 17259-17276. doi: https://doi.org/10.1021/acs.jmedchem.1c01331
Lillich FF, Willems S, Ni X, Kilu W, Borkowsky C, Brodsky M, Kramer JS, Brunst S, Hernandez-Olmos V, Heering J, Schierle S, Kestner RI, Mayser FM, Helmstädter M, Göbel T, Weizel L, Namgaladze D, Kaiser A, Steinhilber D, Pfeilschifter W, Kahnt AS, Proschak A, Chaikuad A, Knapp S, Merk D, Proschak E. Structure-Based Design of Dual Partial Peroxisome Proliferator-Activated Receptor γ Agonists/Soluble Epoxide Hydrolase Inhibitors. J Med Chem. 2021 Dec 09;64(23):17259-17276. doi: 10.1021/acs.jmedchem.1c01331. Epub 2021 Nov 24. PMID: 34818007.
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Scaffold polarity proteins Par3A and Par3B share redundant functions while Par3B acts independent of atypical protein kinase C/Par6 in podocytes to maintain the kidney filtration barrier.

Kidney international

Koehler S, Odenthal J, Ludwig V, Jess DU, Höhne M, Jüngst C, Grawe F, Helmstädter M, Janku JL, Bergmann C, Hoyer PF, Hagmann HHH, Walz G, Bloch W, Niessen C, Schermer B, Wodarz A, Denholm B, Benzing T, Iden S, Brinkkoetter PT.
PMID: 34929254
Kidney Int. 2021 Dec 17; doi: 10.1016/j.kint.2021.11.030. Epub 2021 Dec 17.

Glomerular diseases are a major cause for chronic kidney disorders. In most cases podocyte injury is causative for disease development. Cytoskeletal rearrangements and morphological changes are hallmark features of podocyte injury and result in dedifferentiation and loss of podocytes....

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Koehler S, Odenthal J, Ludwig V, et al. Scaffold polarity proteins Par3A and Par3B share redundant functions while Par3B acts independent of atypical protein kinase C/Par6 in podocytes to maintain the kidney filtration barrier. Kidney Int. 2021;doi: 10.1016/j.kint.2021.11.030.
Koehler, S., Odenthal, J., Ludwig, V., Jess, D. U., Höhne, M., Jüngst, C., Grawe, F., Helmstädter, M., Janku, J. L., Bergmann, C., Hoyer, P. F., Hagmann, H. H. H., Walz, G., Bloch, W., Niessen, C., Schermer, B., Wodarz, A., Denholm, B., Benzing, T., Iden, S., & Brinkkoetter, P. T. (2021). Scaffold polarity proteins Par3A and Par3B share redundant functions while Par3B acts independent of atypical protein kinase C/Par6 in podocytes to maintain the kidney filtration barrier. Kidney international, . https://doi.org/10.1016/j.kint.2021.11.030
Koehler, Sybille, et al. "Scaffold polarity proteins Par3A and Par3B share redundant functions while Par3B acts independent of atypical protein kinase C/Par6 in podocytes to maintain the kidney filtration barrier." Kidney international vol. (2021). doi: https://doi.org/10.1016/j.kint.2021.11.030
Koehler S, Odenthal J, Ludwig V, Jess DU, Höhne M, Jüngst C, Grawe F, Helmstädter M, Janku JL, Bergmann C, Hoyer PF, Hagmann HHH, Walz G, Bloch W, Niessen C, Schermer B, Wodarz A, Denholm B, Benzing T, Iden S, Brinkkoetter PT. Scaffold polarity proteins Par3A and Par3B share redundant functions while Par3B acts independent of atypical protein kinase C/Par6 in podocytes to maintain the kidney filtration barrier. Kidney Int. 2021 Dec 17; doi: 10.1016/j.kint.2021.11.030. Epub 2021 Dec 17. PMID: 34929254.
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The impact of perampanel on cognition: A systematic review of studies employing standardized tests in patients with epilepsy.

Seizure

Witt JA, Helmstaedter C.
PMID: 34890976
Seizure. 2021 Dec 04;94:107-111. doi: 10.1016/j.seizure.2021.12.001. Epub 2021 Dec 04.

This systematic review was conducted to reveal the cognitive effects of perampanel (PER) as assessed by objective standardized neuropsychological measures in patients with epilepsy. A systematic literature search was performed in PubMed. In addition we cross-checked a list of...

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Witt JA, Helmstaedter C. The impact of perampanel on cognition: A systematic review of studies employing standardized tests in patients with epilepsy. Seizure. 2021;94:107-111doi: 10.1016/j.seizure.2021.12.001.
Witt, J. A., & Helmstaedter, C. (2021). The impact of perampanel on cognition: A systematic review of studies employing standardized tests in patients with epilepsy. Seizure, 94107-111. https://doi.org/10.1016/j.seizure.2021.12.001
Witt, Juri-Alexander, and Helmstaedter, Christoph. "The impact of perampanel on cognition: A systematic review of studies employing standardized tests in patients with epilepsy." Seizure vol. 94 (2021): 107-111. doi: https://doi.org/10.1016/j.seizure.2021.12.001
Witt JA, Helmstaedter C. The impact of perampanel on cognition: A systematic review of studies employing standardized tests in patients with epilepsy. Seizure. 2021 Dec 04;94:107-111. doi: 10.1016/j.seizure.2021.12.001. Epub 2021 Dec 04. PMID: 34890976.
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Angiotensin II Induces Oxidative Stress and Endothelial Dysfunction in Mouse Ophthalmic Arteries via Involvement of AT1 Receptors and NOX2.

Antioxidants (Basel, Switzerland)

Birk M, Baum E, Zadeh JK, Manicam C, Pfeiffer N, Patzak A, Helmstädter J, Steven S, Kuntic M, Daiber A, Gericke A.
PMID: 34439486
Antioxidants (Basel). 2021 Aug 02;10(8). doi: 10.3390/antiox10081238.

Angiotensin II (Ang II) has been implicated in the pathophysiology of various age-dependent ocular diseases. The purpose of this study was to test the hypothesis that Ang II induces endothelial dysfunction in mouse ophthalmic arteries and to identify the...

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Birk M, Baum E, Zadeh JK, et al. Angiotensin II Induces Oxidative Stress and Endothelial Dysfunction in Mouse Ophthalmic Arteries via Involvement of AT1 Receptors and NOX2. Antioxidants (Basel). 2021;10(8)doi: 10.3390/antiox10081238.
Birk, M., Baum, E., Zadeh, J. K., Manicam, C., Pfeiffer, N., Patzak, A., Helmstädter, J., Steven, S., Kuntic, M., Daiber, A., & Gericke, A. (2021). Angiotensin II Induces Oxidative Stress and Endothelial Dysfunction in Mouse Ophthalmic Arteries via Involvement of AT1 Receptors and NOX2. Antioxidants (Basel, Switzerland), 10(8), . https://doi.org/10.3390/antiox10081238
Birk, Michael, et al. "Angiotensin II Induces Oxidative Stress and Endothelial Dysfunction in Mouse Ophthalmic Arteries via Involvement of AT1 Receptors and NOX2." Antioxidants (Basel, Switzerland) vol. 10,8 (2021). doi: https://doi.org/10.3390/antiox10081238
Birk M, Baum E, Zadeh JK, Manicam C, Pfeiffer N, Patzak A, Helmstädter J, Steven S, Kuntic M, Daiber A, Gericke A. Angiotensin II Induces Oxidative Stress and Endothelial Dysfunction in Mouse Ophthalmic Arteries via Involvement of AT1 Receptors and NOX2. Antioxidants (Basel). 2021 Aug 02;10(8). doi: 10.3390/antiox10081238. PMID: 34439486; PMCID: PMC8389243.
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