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Determination of strontium in human tooth enamel by flameless atomic-absorption spectrometry.

Talanta

Helsby CA.
PMID: 18962020
Talanta. 1977 Jan;24(1):46-8. doi: 10.1016/0039-9140(77)80184-7.

The determination of strontium in human tooth enamel by flameless atomic-absorption spectrometry was found to be subject to interference from the calcium/phosphate matrix of the enamel. The standard addition method was used to compensate for this interference. Hydrochloric acid...

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Helsby CA. Determination of strontium in human tooth enamel by flameless atomic-absorption spectrometry. Talanta. 1977;24(1):46-8doi: 10.1016/0039-9140(77)80184-7.
Helsby, C. A. (1977). Determination of strontium in human tooth enamel by flameless atomic-absorption spectrometry. Talanta, 24(1), 46-8. https://doi.org/10.1016/0039-9140(77)80184-7
Helsby, C A. "Determination of strontium in human tooth enamel by flameless atomic-absorption spectrometry." Talanta vol. 24,1 (1977): 46-8. doi: https://doi.org/10.1016/0039-9140(77)80184-7
Helsby CA. Determination of strontium in human tooth enamel by flameless atomic-absorption spectrometry. Talanta. 1977 Jan;24(1):46-8. doi: 10.1016/0039-9140(77)80184-7. PMID: 18962020.
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Factors that influence family and parental preferences and decision making for unscheduled paediatric healthcare: a systematic review protocol.

HRB open research

Nicholson E, McDonnell T, Hamza M, Barrett M, Brunsdon C, Bury G, Charlton M, Collins C, Deasy C, De Brun A, Fitzsimons J, Galligan M, Hensey C, Kiernan F, McAuliffe E.
PMID: 32104777
HRB Open Res. 2019 Jun 14;2:11. doi: 10.12688/hrbopenres.12897.2. eCollection 2019.

There is a plethora of factors that dictate where parents and families choose to seek unscheduled healthcare for their child; and the complexity of these decisions can present a challenge for policy makers and healthcare planners as these behaviours...

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Nicholson E, McDonnell T, Hamza M, et al. Factors that influence family and parental preferences and decision making for unscheduled paediatric healthcare: a systematic review protocol. HRB Open Res. 2019;2:11doi: 10.12688/hrbopenres.12897.2.
Nicholson, E., McDonnell, T., Hamza, M., Barrett, M., Brunsdon, C., Bury, G., Charlton, M., Collins, C., Deasy, C., De Brun, A., Fitzsimons, J., Galligan, M., Hensey, C., Kiernan, F., & McAuliffe, E. (2019). Factors that influence family and parental preferences and decision making for unscheduled paediatric healthcare: a systematic review protocol. HRB open research, 211. https://doi.org/10.12688/hrbopenres.12897.2
Nicholson, Emma, et al. "Factors that influence family and parental preferences and decision making for unscheduled paediatric healthcare: a systematic review protocol." HRB open research vol. 2 (2019): 11. doi: https://doi.org/10.12688/hrbopenres.12897.2
Nicholson E, McDonnell T, Hamza M, Barrett M, Brunsdon C, Bury G, Charlton M, Collins C, Deasy C, De Brun A, Fitzsimons J, Galligan M, Hensey C, Kiernan F, McAuliffe E. Factors that influence family and parental preferences and decision making for unscheduled paediatric healthcare: a systematic review protocol. HRB Open Res. 2019 Jun 14;2:11. doi: 10.12688/hrbopenres.12897.2. eCollection 2019. PMID: 32104777; PMCID: PMC7016877.
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CUPID COVID-19: emergency department attendance by paediatric patients during COVID-19 - project protocol.

HRB open research

McDonnell T, McAuliffe E, Barrett M, Conlon C, Cummins F, Deasy C, Hensey C, Martin C, Nicholson E.
PMID: 32666040
HRB Open Res. 2020 Jun 09;3:37. doi: 10.12688/hrbopenres.13066.1. eCollection 2020.

No abstract available.

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McDonnell T, McAuliffe E, Barrett M, et al. CUPID COVID-19: emergency department attendance by paediatric patients during COVID-19 - project protocol. HRB Open Res. 2020;3:37doi: 10.12688/hrbopenres.13066.1.
McDonnell, T., McAuliffe, E., Barrett, M., Conlon, C., Cummins, F., Deasy, C., Hensey, C., Martin, C., & Nicholson, E. (2020). CUPID COVID-19: emergency department attendance by paediatric patients during COVID-19 - project protocol. HRB open research, 337. https://doi.org/10.12688/hrbopenres.13066.1
McDonnell, Thérèse, et al. "CUPID COVID-19: emergency department attendance by paediatric patients during COVID-19 - project protocol." HRB open research vol. 3 (2020): 37. doi: https://doi.org/10.12688/hrbopenres.13066.1
McDonnell T, McAuliffe E, Barrett M, Conlon C, Cummins F, Deasy C, Hensey C, Martin C, Nicholson E. CUPID COVID-19: emergency department attendance by paediatric patients during COVID-19 - project protocol. HRB Open Res. 2020 Jun 09;3:37. doi: 10.12688/hrbopenres.13066.1. eCollection 2020. PMID: 32666040; PMCID: PMC7327719.
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Loss of the maintenance methyltransferase, xDnmt1, induces apoptosis in Xenopus embryos.

The EMBO journal

Stancheva I, Hensey C, Meehan RR.
PMID: 31612522
EMBO J. 2019 Oct 15;38(20):e103221. doi: 10.15252/embj.2019103221.

No abstract available.

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Stancheva I, Hensey C, Meehan RR. Loss of the maintenance methyltransferase, xDnmt1, induces apoptosis in Xenopus embryos. EMBO J. 2019;38(20):e103221doi: 10.15252/embj.2019103221.
Stancheva, I., Hensey, C., & Meehan, R. R. (2019). Loss of the maintenance methyltransferase, xDnmt1, induces apoptosis in Xenopus embryos. The EMBO journal, 38(20), e103221. https://doi.org/10.15252/embj.2019103221
Stancheva, Irina, et al. "Loss of the maintenance methyltransferase, xDnmt1, induces apoptosis in Xenopus embryos." The EMBO journal vol. 38,20 (2019): e103221. doi: https://doi.org/10.15252/embj.2019103221
Stancheva I, Hensey C, Meehan RR. Loss of the maintenance methyltransferase, xDnmt1, induces apoptosis in Xenopus embryos. EMBO J. 2019 Oct 15;38(20):e103221. doi: 10.15252/embj.2019103221. PMID: 31612522; PMCID: PMC6792011.
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CUPID COVID-19: emergency department attendance by paediatric patients during COVID-19 - project protocol.

HRB open research

McDonnell T, McAuliffe E, Barrett M, Conlon C, Cummins F, Deasy C, Hensey C, Martin C, Nicholson E.
PMID: 32666040
HRB Open Res. 2020 Aug 10;3:37. doi: 10.12688/hrbopenres.13066.2. eCollection 2020.

No abstract available.

Cite
McDonnell T, McAuliffe E, Barrett M, et al. CUPID COVID-19: emergency department attendance by paediatric patients during COVID-19 - project protocol. HRB Open Res. 2020;3:37doi: 10.12688/hrbopenres.13066.2.
McDonnell, T., McAuliffe, E., Barrett, M., Conlon, C., Cummins, F., Deasy, C., Hensey, C., Martin, C., & Nicholson, E. (2020). CUPID COVID-19: emergency department attendance by paediatric patients during COVID-19 - project protocol. HRB open research, 337. https://doi.org/10.12688/hrbopenres.13066.2
McDonnell, Thérèse, et al. "CUPID COVID-19: emergency department attendance by paediatric patients during COVID-19 - project protocol." HRB open research vol. 3 (2020): 37. doi: https://doi.org/10.12688/hrbopenres.13066.2
McDonnell T, McAuliffe E, Barrett M, Conlon C, Cummins F, Deasy C, Hensey C, Martin C, Nicholson E. CUPID COVID-19: emergency department attendance by paediatric patients during COVID-19 - project protocol. HRB Open Res. 2020 Aug 10;3:37. doi: 10.12688/hrbopenres.13066.2. eCollection 2020. PMID: 32666040; PMCID: PMC7327719.
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The U-shaped Relationship of Traditional Cardiovascular Risk Factors and Adverse Outcomes in Later Life.

Aging and disease

Abdelhafiz AH, Loo BE, Hensey N, Bailey C, Sinclair A.
PMID: 23251851
Aging Dis. 2012 Dec;3(6):454-64. Epub 2012 Oct 17.

With advancing age traditional cardiovascular risk factors follow a U-shaped relationship with survival outcomes. This relationship has been described as reverse metabolism. The mechanism of such relationship is unclear. However, it appears that malnutrition, inflammation and functional decline are...

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Abdelhafiz AH, Loo BE, Hensey N, et al. The U-shaped Relationship of Traditional Cardiovascular Risk Factors and Adverse Outcomes in Later Life. Aging Dis. 2012;3(6):454-64
Abdelhafiz, A. H., Loo, B. E., Hensey, N., Bailey, C., & Sinclair, A. (2012). The U-shaped Relationship of Traditional Cardiovascular Risk Factors and Adverse Outcomes in Later Life. Aging and disease, 3(6), 454-64.
Abdelhafiz, Ahmed H, et al. "The U-shaped Relationship of Traditional Cardiovascular Risk Factors and Adverse Outcomes in Later Life." Aging and disease vol. 3,6 (2012): 454-64.
Abdelhafiz AH, Loo BE, Hensey N, Bailey C, Sinclair A. The U-shaped Relationship of Traditional Cardiovascular Risk Factors and Adverse Outcomes in Later Life. Aging Dis. 2012 Dec;3(6):454-64. Epub 2012 Oct 17. PMID: 23251851; PMCID: PMC3522512.
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Determination of copper and molybdenum in the hard dental tissues of rats by atomic-absorption spectrophotometry.

Talanta

Helsby CA.
PMID: 18961345
Talanta. 1973 Aug;20(8):779-82. doi: 10.1016/0039-9140(73)80256-5.

The measurement of uptake of copper and molybdenum from the diet into rat's teeth required the use of an accurate but rapid method of analysis. The simultaneous extraction of copper and molybdenum with APDC from the sample, combined with...

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Helsby CA. Determination of copper and molybdenum in the hard dental tissues of rats by atomic-absorption spectrophotometry. Talanta. 1973;20(8):779-82doi: 10.1016/0039-9140(73)80256-5.
Helsby, C. A. (1973). Determination of copper and molybdenum in the hard dental tissues of rats by atomic-absorption spectrophotometry. Talanta, 20(8), 779-82. https://doi.org/10.1016/0039-9140(73)80256-5
Helsby, C A. "Determination of copper and molybdenum in the hard dental tissues of rats by atomic-absorption spectrophotometry." Talanta vol. 20,8 (1973): 779-82. doi: https://doi.org/10.1016/0039-9140(73)80256-5
Helsby CA. Determination of copper and molybdenum in the hard dental tissues of rats by atomic-absorption spectrophotometry. Talanta. 1973 Aug;20(8):779-82. doi: 10.1016/0039-9140(73)80256-5. PMID: 18961345.
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Association between the low-dose irinotecan regimen-induced occurrence of grade 4 neutropenia and genetic variants of .

Oncology letters

Moriya H, Saito K, Helsby N, Sugino S, Yamakage M, Sawaguchi T, Takasaki M, Kato H, Kurosawa N.
PMID: 24932285
Oncol Lett. 2014 Jun;7(6):2035-2040. doi: 10.3892/ol.2014.2046. Epub 2014 Apr 08.

The occurrence of severe neutropenia during treatment with irinotecan (CPT-11) is associated with the

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Moriya H, Saito K, Helsby N, et al. Association between the low-dose irinotecan regimen-induced occurrence of grade 4 neutropenia and genetic variants of . Oncol Lett. 2014;7(6):2035-2040doi: 10.3892/ol.2014.2046.
Moriya, H., Saito, K., Helsby, N., Sugino, S., Yamakage, M., Sawaguchi, T., Takasaki, M., Kato, H., & Kurosawa, N. (2014). Association between the low-dose irinotecan regimen-induced occurrence of grade 4 neutropenia and genetic variants of . Oncology letters, 7(6), 2035-2040. https://doi.org/10.3892/ol.2014.2046
Moriya, Hiroyuki, et al. "Association between the low-dose irinotecan regimen-induced occurrence of grade 4 neutropenia and genetic variants of ." Oncology letters vol. 7,6 (2014): 2035-2040. doi: https://doi.org/10.3892/ol.2014.2046
Moriya H, Saito K, Helsby N, Sugino S, Yamakage M, Sawaguchi T, Takasaki M, Kato H, Kurosawa N. Association between the low-dose irinotecan regimen-induced occurrence of grade 4 neutropenia and genetic variants of . Oncol Lett. 2014 Jun;7(6):2035-2040. doi: 10.3892/ol.2014.2046. Epub 2014 Apr 08. PMID: 24932285; PMCID: PMC4049750.
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Severe 5-Fluorouracil-Associated Gastrointestinal Toxicity Unexplained by Dihydropyrimidine Dehydrogenase Deficiency and Renal Impairment: Should We Be Investigating Other Elimination Pathways to Assess the Risk of 5-Fluorouracil Toxicity?.

European journal of drug metabolism and pharmacokinetics

Jeong SH, Chavani O, Burns K, Porter D, Findlay M, Helsby N.
PMID: 34570362
Eur J Drug Metab Pharmacokinet. 2021 Nov;46(6):817-820. doi: 10.1007/s13318-021-00721-0. Epub 2021 Sep 27.

No abstract available.

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Jeong SH, Chavani O, Burns K, et al. Severe 5-Fluorouracil-Associated Gastrointestinal Toxicity Unexplained by Dihydropyrimidine Dehydrogenase Deficiency and Renal Impairment: Should We Be Investigating Other Elimination Pathways to Assess the Risk of 5-Fluorouracil Toxicity?. Eur J Drug Metab Pharmacokinet. 2021;46(6):817-820doi: 10.1007/s13318-021-00721-0.
Jeong, S. H., Chavani, O., Burns, K., Porter, D., Findlay, M., & Helsby, N. (2021). Severe 5-Fluorouracil-Associated Gastrointestinal Toxicity Unexplained by Dihydropyrimidine Dehydrogenase Deficiency and Renal Impairment: Should We Be Investigating Other Elimination Pathways to Assess the Risk of 5-Fluorouracil Toxicity?. European journal of drug metabolism and pharmacokinetics, 46(6), 817-820. https://doi.org/10.1007/s13318-021-00721-0
Jeong, Soo Hee, et al. "Severe 5-Fluorouracil-Associated Gastrointestinal Toxicity Unexplained by Dihydropyrimidine Dehydrogenase Deficiency and Renal Impairment: Should We Be Investigating Other Elimination Pathways to Assess the Risk of 5-Fluorouracil Toxicity?." European journal of drug metabolism and pharmacokinetics vol. 46,6 (2021): 817-820. doi: https://doi.org/10.1007/s13318-021-00721-0
Jeong SH, Chavani O, Burns K, Porter D, Findlay M, Helsby N. Severe 5-Fluorouracil-Associated Gastrointestinal Toxicity Unexplained by Dihydropyrimidine Dehydrogenase Deficiency and Renal Impairment: Should We Be Investigating Other Elimination Pathways to Assess the Risk of 5-Fluorouracil Toxicity?. Eur J Drug Metab Pharmacokinet. 2021 Nov;46(6):817-820. doi: 10.1007/s13318-021-00721-0. Epub 2021 Sep 27. PMID: 34570362.
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Single-nucleotide polymorphisms and copy number variations of the .

Biomedical reports

Moriya H, Saito K, Helsby N, Hayashi N, Sugino S, Yamakage M, Sawaguchi T, Takasaki M, Takahashi M, Kurosawa N.
PMID: 24649108
Biomed Rep. 2014 Mar;2(2):265-269. doi: 10.3892/br.2013.210. Epub 2013 Dec 06.

FcγRII and FcγRIII are low-affinity Fcγ receptors that are encoded by the

Cite
Moriya H, Saito K, Helsby N, et al. Single-nucleotide polymorphisms and copy number variations of the . Biomed Rep. 2013;2(2):265-269doi: 10.3892/br.2013.210.
Moriya, H., Saito, K., Helsby, N., Hayashi, N., Sugino, S., Yamakage, M., Sawaguchi, T., Takasaki, M., Takahashi, M., & Kurosawa, N. (2014). Single-nucleotide polymorphisms and copy number variations of the . Biomedical reports, 2(2), 265-269. https://doi.org/10.3892/br.2013.210
Moriya, Hiroyuki, et al. "Single-nucleotide polymorphisms and copy number variations of the ." Biomedical reports vol. 2,2 (2014): 265-269. doi: https://doi.org/10.3892/br.2013.210
Moriya H, Saito K, Helsby N, Hayashi N, Sugino S, Yamakage M, Sawaguchi T, Takasaki M, Takahashi M, Kurosawa N. Single-nucleotide polymorphisms and copy number variations of the . Biomed Rep. 2014 Mar;2(2):265-269. doi: 10.3892/br.2013.210. Epub 2013 Dec 06. PMID: 24649108; PMCID: PMC3917743.
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Extracellular Vesicles from 3D Engineered Microtissues Harbor Disease-Related Cargo Absent in EVs from 2D Cultures.

Advanced healthcare materials

Millan C, Prause L, Vallmajo-Martin Q, Hensky N, Eberli D.
PMID: 33890421
Adv Healthc Mater. 2021 Apr 22;e2002067. doi: 10.1002/adhm.202002067. Epub 2021 Apr 22.

Engineered microtissues that recapitulate key properties of the tumor microenvironment can induce clinically relevant cancer phenotypes in vitro. However, their effect on molecular cargo of secreted extracellular vesicles (EVs) has not yet been investigated. Here, the impact of hydrogel-based...

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Millan C, Prause L, Vallmajo-Martin Q, et al. Extracellular Vesicles from 3D Engineered Microtissues Harbor Disease-Related Cargo Absent in EVs from 2D Cultures. Adv Healthc Mater. 2021;e2002067doi: 10.1002/adhm.202002067.
Millan, C., Prause, L., Vallmajo-Martin, Q., Hensky, N., & Eberli, D. (2021). Extracellular Vesicles from 3D Engineered Microtissues Harbor Disease-Related Cargo Absent in EVs from 2D Cultures. Advanced healthcare materials, e2002067. https://doi.org/10.1002/adhm.202002067
Millan, Christopher, et al. "Extracellular Vesicles from 3D Engineered Microtissues Harbor Disease-Related Cargo Absent in EVs from 2D Cultures." Advanced healthcare materials vol. (2021): e2002067. doi: https://doi.org/10.1002/adhm.202002067
Millan C, Prause L, Vallmajo-Martin Q, Hensky N, Eberli D. Extracellular Vesicles from 3D Engineered Microtissues Harbor Disease-Related Cargo Absent in EVs from 2D Cultures. Adv Healthc Mater. 2021 Apr 22;e2002067. doi: 10.1002/adhm.202002067. Epub 2021 Apr 22. PMID: 33890421.
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Cross-Comparison of Exome Analysis, Next-Generation Sequencing of Amplicons, and the iPLEX(®) ADME PGx Panel for Pharmacogenomic Profiling.

Frontiers in pharmacology

Chua EW, Cree SL, Ton KN, Lehnert K, Shepherd P, Helsby N, Kennedy MA.
PMID: 26858644
Front Pharmacol. 2016 Jan 26;7:1. doi: 10.3389/fphar.2016.00001. eCollection 2016.

Whole-exome sequencing (WES) has been widely used for analysis of human genetic diseases, but its value for the pharmacogenomic profiling of individuals is not well studied. Initially, we performed an in-depth evaluation of the accuracy of WES variant calling...

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Chua EW, Cree SL, Ton KN, et al. Cross-Comparison of Exome Analysis, Next-Generation Sequencing of Amplicons, and the iPLEX(®) ADME PGx Panel for Pharmacogenomic Profiling. Front Pharmacol. 2016;7:1doi: 10.3389/fphar.2016.00001.
Chua, E. W., Cree, S. L., Ton, K. N., Lehnert, K., Shepherd, P., Helsby, N., & Kennedy, M. A. (2016). Cross-Comparison of Exome Analysis, Next-Generation Sequencing of Amplicons, and the iPLEX(®) ADME PGx Panel for Pharmacogenomic Profiling. Frontiers in pharmacology, 71. https://doi.org/10.3389/fphar.2016.00001
Chua, Eng Wee, et al. "Cross-Comparison of Exome Analysis, Next-Generation Sequencing of Amplicons, and the iPLEX(®) ADME PGx Panel for Pharmacogenomic Profiling." Frontiers in pharmacology vol. 7 (2016): 1. doi: https://doi.org/10.3389/fphar.2016.00001
Chua EW, Cree SL, Ton KN, Lehnert K, Shepherd P, Helsby N, Kennedy MA. Cross-Comparison of Exome Analysis, Next-Generation Sequencing of Amplicons, and the iPLEX(®) ADME PGx Panel for Pharmacogenomic Profiling. Front Pharmacol. 2016 Jan 26;7:1. doi: 10.3389/fphar.2016.00001. eCollection 2016. PMID: 26858644; PMCID: PMC4726781.
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Showing 1 to 12 of 13 entries