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Associations of polyunsaturated Fatty Acid intake with bone mineral density in postmenopausal women.

Journal of osteoporosis

Harris M, Farrell V, Houtkooper L, Going S, Lohman T, Doppelt Y, Schunn CD.
PMID: 25785226
J Osteoporos. 2015;2015:737521. doi: 10.1155/2015/737521. Epub 2015 Feb 17.

A secondary analysis of cross-sectional data was analyzed from 6 cohorts (Fall 1995-Fall 1997) of postmenopausal women (n = 266; 56.6 ± 4.7 years) participating in the Bone Estrogen Strength Training (BEST) study (a 12-month, block-randomized, clinical trial). Bone...

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Harris M, Farrell V, Houtkooper L, et al. Associations of polyunsaturated Fatty Acid intake with bone mineral density in postmenopausal women. J Osteoporos. 2015;2015:737521doi: 10.1155/2015/737521.
Harris, M., Farrell, V., Houtkooper, L., Going, S., Lohman, T., Doppelt, Y., & Schunn, C. D. (2015). Associations of polyunsaturated Fatty Acid intake with bone mineral density in postmenopausal women. Journal of osteoporosis, 2015737521. https://doi.org/10.1155/2015/737521
Harris, Margaret, et al. "Associations of polyunsaturated Fatty Acid intake with bone mineral density in postmenopausal women." Journal of osteoporosis vol. 2015 (2015): 737521. doi: https://doi.org/10.1155/2015/737521
Harris M, Farrell V, Houtkooper L, Going S, Lohman T, Doppelt Y, Schunn CD. Associations of polyunsaturated Fatty Acid intake with bone mineral density in postmenopausal women. J Osteoporos. 2015;2015:737521. doi: 10.1155/2015/737521. Epub 2015 Feb 17. PMID: 25785226; PMCID: PMC4346685.
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The Physical, Chemical and Physiological Limits of Life.

Life (Basel, Switzerland)

Schulze-Makuch D, Schulze-Makuch A, Houtkooper JM.
PMID: 26193325
Life (Basel). 2015 Jul 17;5(3):1472-86. doi: 10.3390/life5031472.

Life on Earth displays an incredible diversity in form and function, which allows it to survive not only physical extremes, but also periods of time when it is exposed to non-habitable conditions. Extreme physiological adaptations to bridge non-habitable conditions...

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Schulze-Makuch D, Schulze-Makuch A, Houtkooper JM. The Physical, Chemical and Physiological Limits of Life. Life (Basel). 2015;5(3):1472-86doi: 10.3390/life5031472.
Schulze-Makuch, D., Schulze-Makuch, A., & Houtkooper, J. M. (2015). The Physical, Chemical and Physiological Limits of Life. Life (Basel, Switzerland), 5(3), 1472-86. https://doi.org/10.3390/life5031472
Schulze-Makuch, Dirk, et al. "The Physical, Chemical and Physiological Limits of Life." Life (Basel, Switzerland) vol. 5,3 (2015): 1472-86. doi: https://doi.org/10.3390/life5031472
Schulze-Makuch D, Schulze-Makuch A, Houtkooper JM. The Physical, Chemical and Physiological Limits of Life. Life (Basel). 2015 Jul 17;5(3):1472-86. doi: 10.3390/life5031472. PMID: 26193325; PMCID: PMC4598648.
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Variations in exhaled nitric oxide concentration after three types of dives.

Diving and hyperbaric medicine

van Ooij PJ, Houtkooper A, van Hulst R.
PMID: 23111832
Diving Hyperb Med. 2010 Mar;40(1):4-7.

INTRODUCTION: An increase in exhaled nitric oxide concentration (FENO) occurs during an exacerbation of chronic obstructive lung disease or other inflammatory processes of the airway. Raised FENO levels are also observed during normobaric, mild hyperoxic exposures, whereas after hyperbaric...

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van Ooij PJ, Houtkooper A, van Hulst R. Variations in exhaled nitric oxide concentration after three types of dives. Diving Hyperb Med. 2010;40(1):4-7
van Ooij, P. J., Houtkooper, A., & van Hulst, R. (2010). Variations in exhaled nitric oxide concentration after three types of dives. Diving and hyperbaric medicine, 40(1), 4-7.
van Ooij, Pieter-Jan, et al. "Variations in exhaled nitric oxide concentration after three types of dives." Diving and hyperbaric medicine vol. 40,1 (2010): 4-7.
van Ooij PJ, Houtkooper A, van Hulst R. Variations in exhaled nitric oxide concentration after three types of dives. Diving Hyperb Med. 2010 Mar;40(1):4-7. PMID: 23111832.
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Mitochondrial ubiquinone-mediated longevity is marked by reduced cytoplasmic mRNA translation.

Life science alliance

Molenaars M, Janssens GE, Santermans T, Lezzerini M, Jelier R, MacInnes AW, Houtkooper RH.
PMID: 30198021
Life Sci Alliance. 2018 Aug 31;1(5). doi: 10.26508/lsa.201800082.

Mutations in the

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Molenaars M, Janssens GE, Santermans T, et al. Mitochondrial ubiquinone-mediated longevity is marked by reduced cytoplasmic mRNA translation. Life Sci Alliance. 2018;1(5)doi: 10.26508/lsa.201800082.
Molenaars, M., Janssens, G. E., Santermans, T., Lezzerini, M., Jelier, R., MacInnes, A. W., & Houtkooper, R. H. (2018). Mitochondrial ubiquinone-mediated longevity is marked by reduced cytoplasmic mRNA translation. Life science alliance, 1(5), . https://doi.org/10.26508/lsa.201800082
Molenaars, Marte, et al. "Mitochondrial ubiquinone-mediated longevity is marked by reduced cytoplasmic mRNA translation." Life science alliance vol. 1,5 (2018). doi: https://doi.org/10.26508/lsa.201800082
Molenaars M, Janssens GE, Santermans T, Lezzerini M, Jelier R, MacInnes AW, Houtkooper RH. Mitochondrial ubiquinone-mediated longevity is marked by reduced cytoplasmic mRNA translation. Life Sci Alliance. 2018 Aug 31;1(5). doi: 10.26508/lsa.201800082. PMID: 30198021; PMCID: PMC6126614.
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NAD+-Precursor Supplementation With L-Tryptophan, Nicotinic Acid, and Nicotinamide Does Not Affect Mitochondrial Function or Skeletal Muscle Function in Physically Compromised Older Adults.

The Journal of nutrition

Connell NJ, Grevendonk L, Fealy CE, Moonen-Kornips E, Bruls YMH, Schrauwen-Hinderling VB, de Vogel J, Hageman R, Geurts J, Zapata-Perez R, Houtkooper RH, Havekes B, Hoeks J, Schrauwen P.
PMID: 34191033
J Nutr. 2021 Oct 01;151(10):2917-2931. doi: 10.1093/jn/nxab193.

BACKGROUND: Boosting NAD+ via supplementation with niacin equivalents has been proposed as a potential modality capable of promoting healthy aging and negating age-dependent declines of skeletal muscle mass and function.OBJECTIVES: We investigated the efficacy of NAD+-precursor supplementation (tryptophan, nicotinic...

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Connell NJ, Grevendonk L, Fealy CE, et al. NAD+-Precursor Supplementation With L-Tryptophan, Nicotinic Acid, and Nicotinamide Does Not Affect Mitochondrial Function or Skeletal Muscle Function in Physically Compromised Older Adults. J Nutr. 2021;151(10):2917-2931doi: 10.1093/jn/nxab193.
Connell, N. J., Grevendonk, L., Fealy, C. E., Moonen-Kornips, E., Bruls, Y. M. H., Schrauwen-Hinderling, V. B., de Vogel, J., Hageman, R., Geurts, J., Zapata-Perez, R., Houtkooper, R. H., Havekes, B., Hoeks, J., & Schrauwen, P. (2021). NAD+-Precursor Supplementation With L-Tryptophan, Nicotinic Acid, and Nicotinamide Does Not Affect Mitochondrial Function or Skeletal Muscle Function in Physically Compromised Older Adults. The Journal of nutrition, 151(10), 2917-2931. https://doi.org/10.1093/jn/nxab193
Connell, N J, et al. "NAD+-Precursor Supplementation With L-Tryptophan, Nicotinic Acid, and Nicotinamide Does Not Affect Mitochondrial Function or Skeletal Muscle Function in Physically Compromised Older Adults." The Journal of nutrition vol. 151,10 (2021): 2917-2931. doi: https://doi.org/10.1093/jn/nxab193
Connell NJ, Grevendonk L, Fealy CE, Moonen-Kornips E, Bruls YMH, Schrauwen-Hinderling VB, de Vogel J, Hageman R, Geurts J, Zapata-Perez R, Houtkooper RH, Havekes B, Hoeks J, Schrauwen P. NAD+-Precursor Supplementation With L-Tryptophan, Nicotinic Acid, and Nicotinamide Does Not Affect Mitochondrial Function or Skeletal Muscle Function in Physically Compromised Older Adults. J Nutr. 2021 Oct 01;151(10):2917-2931. doi: 10.1093/jn/nxab193. PMID: 34191033; PMCID: PMC8485915.
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Mitochondrial Fatty Acid Oxidation Disorders: Laboratory Diagnosis, Pathogenesis, and the Complicated Route to Treatment.

Journal of lipid and atherosclerosis

Wanders RJA, Visser G, Ferdinandusse S, Vaz FM, Houtkooper RH.
PMID: 33024728
J Lipid Atheroscler. 2020 Sep;9(3):313-333. doi: 10.12997/jla.2020.9.3.313. Epub 2020 Sep 22.

Mitochondrial fatty acid (FA) oxidation deficiencies represent a genetically heterogeneous group of diseases in humans caused by defects in mitochondrial FA beta-oxidation (mFAO). A general characteristic of all mFAO disorders is hypoketotic hypoglycemia resulting from the enhanced reliance on...

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Wanders RJA, Visser G, Ferdinandusse S, et al. Mitochondrial Fatty Acid Oxidation Disorders: Laboratory Diagnosis, Pathogenesis, and the Complicated Route to Treatment. J Lipid Atheroscler. 2020;9(3):313-333doi: 10.12997/jla.2020.9.3.313.
Wanders, R. J. A., Visser, G., Ferdinandusse, S., Vaz, F. M., & Houtkooper, R. H. (2020). Mitochondrial Fatty Acid Oxidation Disorders: Laboratory Diagnosis, Pathogenesis, and the Complicated Route to Treatment. Journal of lipid and atherosclerosis, 9(3), 313-333. https://doi.org/10.12997/jla.2020.9.3.313
Wanders, Ronald J A, et al. "Mitochondrial Fatty Acid Oxidation Disorders: Laboratory Diagnosis, Pathogenesis, and the Complicated Route to Treatment." Journal of lipid and atherosclerosis vol. 9,3 (2020): 313-333. doi: https://doi.org/10.12997/jla.2020.9.3.313
Wanders RJA, Visser G, Ferdinandusse S, Vaz FM, Houtkooper RH. Mitochondrial Fatty Acid Oxidation Disorders: Laboratory Diagnosis, Pathogenesis, and the Complicated Route to Treatment. J Lipid Atheroscler. 2020 Sep;9(3):313-333. doi: 10.12997/jla.2020.9.3.313. Epub 2020 Sep 22. PMID: 33024728; PMCID: PMC7521971.
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An improved functional assay in blood spot to diagnose Barth syndrome using the monolysocardiolipin/cardiolipin ratio.

Journal of inherited metabolic disease

Vaz FM, van Lenthe H, Vervaart MAT, Stet FS, Klinkspoor JH, Vernon HJ, Goorden SMI, Houtkooper RH, Kulik W, Wanders RJA.
PMID: 34382226
J Inherit Metab Dis. 2021 Aug 11; doi: 10.1002/jimd.12425. Epub 2021 Aug 11.

Barth syndrome is an X-linked disorder characterized by cardiomyopathy, skeletal myopathy, and neutropenia, caused by deleterious variants in TAFAZZIN. This gene encodes a phospholipid-lysophospholipid transacylase that is required for the remodeling of the mitochondrial phospholipid cardiolipin (CL). Biochemically, individuals...

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Vaz FM, van Lenthe H, Vervaart MAT, et al. An improved functional assay in blood spot to diagnose Barth syndrome using the monolysocardiolipin/cardiolipin ratio. J Inherit Metab Dis. 2021;doi: 10.1002/jimd.12425.
Vaz, F. M., van Lenthe, H., Vervaart, M. A. T., Stet, F. S., Klinkspoor, J. H., Vernon, H. J., Goorden, S. M. I., Houtkooper, R. H., Kulik, W., & Wanders, R. J. A. (2021). An improved functional assay in blood spot to diagnose Barth syndrome using the monolysocardiolipin/cardiolipin ratio. Journal of inherited metabolic disease, . https://doi.org/10.1002/jimd.12425
Vaz, Frédéric M, et al. "An improved functional assay in blood spot to diagnose Barth syndrome using the monolysocardiolipin/cardiolipin ratio." Journal of inherited metabolic disease vol. (2021). doi: https://doi.org/10.1002/jimd.12425
Vaz FM, van Lenthe H, Vervaart MAT, Stet FS, Klinkspoor JH, Vernon HJ, Goorden SMI, Houtkooper RH, Kulik W, Wanders RJA. An improved functional assay in blood spot to diagnose Barth syndrome using the monolysocardiolipin/cardiolipin ratio. J Inherit Metab Dis. 2021 Aug 11; doi: 10.1002/jimd.12425. Epub 2021 Aug 11. PMID: 34382226.
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Microbial stimulation of different Toll-like receptor signalling pathways induces diverse metabolic programmes in human monocytes.

Nature microbiology

Lachmandas E, Boutens L, Ratter JM, Hijmans A, Hooiveld GJ, Joosten LA, Rodenburg RJ, Fransen JA, Houtkooper RH, van Crevel R, Netea MG, Stienstra R.
PMID: 27991883
Nat Microbiol. 2016 Dec 19;2:16246. doi: 10.1038/nmicrobiol.2016.246.

Microbial stimuli such as lipopolysaccharide (LPS) induce robust metabolic rewiring in immune cells known as the Warburg effect. It is unknown whether this increase in glycolysis and decrease in oxidative phosphorylation (OXPHOS) is a general characteristic of monocytes that...

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Lachmandas E, Boutens L, Ratter JM, et al. Microbial stimulation of different Toll-like receptor signalling pathways induces diverse metabolic programmes in human monocytes. Nat Microbiol. 2016;2:16246doi: 10.1038/nmicrobiol.2016.246.
Lachmandas, E., Boutens, L., Ratter, J. M., Hijmans, A., Hooiveld, G. J., Joosten, L. A., Rodenburg, R. J., Fransen, J. A., Houtkooper, R. H., van Crevel, R., Netea, M. G., & Stienstra, R. (2016). Microbial stimulation of different Toll-like receptor signalling pathways induces diverse metabolic programmes in human monocytes. Nature microbiology, 216246. https://doi.org/10.1038/nmicrobiol.2016.246
Lachmandas, Ekta, et al. "Microbial stimulation of different Toll-like receptor signalling pathways induces diverse metabolic programmes in human monocytes." Nature microbiology vol. 2 (2016): 16246. doi: https://doi.org/10.1038/nmicrobiol.2016.246
Lachmandas E, Boutens L, Ratter JM, Hijmans A, Hooiveld GJ, Joosten LA, Rodenburg RJ, Fransen JA, Houtkooper RH, van Crevel R, Netea MG, Stienstra R. Microbial stimulation of different Toll-like receptor signalling pathways induces diverse metabolic programmes in human monocytes. Nat Microbiol. 2016 Dec 19;2:16246. doi: 10.1038/nmicrobiol.2016.246. PMID: 27991883.
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Sitting less elicits metabolic responses similar to exercise and enhances insulin sensitivity in postmenopausal women.

Diabetologia

Remie CME, Janssens GE, Bilet L, van Weeghel M, Duvivier BMFM, de Wit VHW, Connell NJ, Jörgensen JA, Schomakers BV, Schrauwen-Hinderling VB, Hoeks J, Hesselink MKC, Phielix E, Houtkooper RH, Schrauwen P.
PMID: 34510226
Diabetologia. 2021 Dec;64(12):2817-2828. doi: 10.1007/s00125-021-05558-5. Epub 2021 Sep 12.

AIMS/HYPOTHESIS: In our current society sedentary behaviour predominates in most people and is associated with the risk of developing type 2 diabetes. It has been suggested that replacing sitting time by standing and walking could be beneficial for individuals...

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Remie CME, Janssens GE, Bilet L, et al. Sitting less elicits metabolic responses similar to exercise and enhances insulin sensitivity in postmenopausal women. Diabetologia. 2021;64(12):2817-2828doi: 10.1007/s00125-021-05558-5.
Remie, C. M. E., Janssens, G. E., Bilet, L., van Weeghel, M., Duvivier, B. M. F. M., de Wit, V. H. W., Connell, N. J., Jörgensen, J. A., Schomakers, B. V., Schrauwen-Hinderling, V. B., Hoeks, J., Hesselink, M. K. C., Phielix, E., Houtkooper, R. H., & Schrauwen, P. (2021). Sitting less elicits metabolic responses similar to exercise and enhances insulin sensitivity in postmenopausal women. Diabetologia, 64(12), 2817-2828. https://doi.org/10.1007/s00125-021-05558-5
Remie, Carlijn M E, et al. "Sitting less elicits metabolic responses similar to exercise and enhances insulin sensitivity in postmenopausal women." Diabetologia vol. 64,12 (2021): 2817-2828. doi: https://doi.org/10.1007/s00125-021-05558-5
Remie CME, Janssens GE, Bilet L, van Weeghel M, Duvivier BMFM, de Wit VHW, Connell NJ, Jörgensen JA, Schomakers BV, Schrauwen-Hinderling VB, Hoeks J, Hesselink MKC, Phielix E, Houtkooper RH, Schrauwen P. Sitting less elicits metabolic responses similar to exercise and enhances insulin sensitivity in postmenopausal women. Diabetologia. 2021 Dec;64(12):2817-2828. doi: 10.1007/s00125-021-05558-5. Epub 2021 Sep 12. PMID: 34510226; PMCID: PMC8435176.
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Aging selectively dampens oscillation of lipid abundance in white and brown adipose tissue.

Scientific reports

Held NM, Buijink MR, Elfrink HL, Kooijman S, Janssens GE, Luyf ACM, Pras-Raves ML, Vaz FM, Michel S, Houtkooper RH, van Weeghel M.
PMID: 33723320
Sci Rep. 2021 Mar 15;11(1):5932. doi: 10.1038/s41598-021-85455-4.

Lipid metabolism is under the control of the circadian system and circadian dysregulation has been linked to obesity and dyslipidemia. These factors and outcomes have also been associated to, or affected by, the process of aging. Here, we investigated...

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Held NM, Buijink MR, Elfrink HL, et al. Aging selectively dampens oscillation of lipid abundance in white and brown adipose tissue. Sci Rep. 2021;11(1):5932doi: 10.1038/s41598-021-85455-4.
Held, N. M., Buijink, M. R., Elfrink, H. L., Kooijman, S., Janssens, G. E., Luyf, A. C. M., Pras-Raves, M. L., Vaz, F. M., Michel, S., Houtkooper, R. H., & van Weeghel, M. (2021). Aging selectively dampens oscillation of lipid abundance in white and brown adipose tissue. Scientific reports, 11(1), 5932. https://doi.org/10.1038/s41598-021-85455-4
Held, Ntsiki M, et al. "Aging selectively dampens oscillation of lipid abundance in white and brown adipose tissue." Scientific reports vol. 11,1 (2021): 5932. doi: https://doi.org/10.1038/s41598-021-85455-4
Held NM, Buijink MR, Elfrink HL, Kooijman S, Janssens GE, Luyf ACM, Pras-Raves ML, Vaz FM, Michel S, Houtkooper RH, van Weeghel M. Aging selectively dampens oscillation of lipid abundance in white and brown adipose tissue. Sci Rep. 2021 Mar 15;11(1):5932. doi: 10.1038/s41598-021-85455-4. PMID: 33723320; PMCID: PMC7961067.
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A review of treatment modalities in gyrate atrophy of the choroid and retina (GACR).

Molecular genetics and metabolism

Balfoort BM, Buijs MJN, Ten Asbroek ALMA, Bergen AAB, Boon CJF, Ferreira EA, Houtkooper RH, Wagenmakers MAEM, Wanders RJA, Waterham HR, Timmer C, van Karnebeek CD, Brands MM.
PMID: 34340878
Mol Genet Metab. 2021 Sep-Oct;134(1):96-116. doi: 10.1016/j.ymgme.2021.07.010. Epub 2021 Jul 26.

Gyrate atrophy of the choroid and retina (GACR) is a rare inborn error of amino acid metabolism caused by bi-allelic variations in OAT. GACR is characterised by vision decline in early life eventually leading to complete blindness, and high...

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Balfoort BM, Buijs MJN, Ten Asbroek ALMA, et al. A review of treatment modalities in gyrate atrophy of the choroid and retina (GACR). Mol Genet Metab. 2021;134(1):96-116doi: 10.1016/j.ymgme.2021.07.010.
Balfoort, B. M., Buijs, M. J. N., Ten Asbroek, A. L. M. A., Bergen, A. A. B., Boon, C. J. F., Ferreira, E. A., Houtkooper, R. H., Wagenmakers, M. A. E. M., Wanders, R. J. A., Waterham, H. R., Timmer, C., van Karnebeek, C. D., & Brands, M. M. (2021). A review of treatment modalities in gyrate atrophy of the choroid and retina (GACR). Molecular genetics and metabolism, 134(1), 96-116. https://doi.org/10.1016/j.ymgme.2021.07.010
Balfoort, Berith M, et al. "A review of treatment modalities in gyrate atrophy of the choroid and retina (GACR)." Molecular genetics and metabolism vol. 134,1 (2021): 96-116. doi: https://doi.org/10.1016/j.ymgme.2021.07.010
Balfoort BM, Buijs MJN, Ten Asbroek ALMA, Bergen AAB, Boon CJF, Ferreira EA, Houtkooper RH, Wagenmakers MAEM, Wanders RJA, Waterham HR, Timmer C, van Karnebeek CD, Brands MM. A review of treatment modalities in gyrate atrophy of the choroid and retina (GACR). Mol Genet Metab. 2021 Sep-Oct;134(1):96-116. doi: 10.1016/j.ymgme.2021.07.010. Epub 2021 Jul 26. PMID: 34340878.
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Time-restricted feeding during the inactive phase abolishes the daily rhythm in mitochondrial respiration in rat skeletal muscle.

FASEB journal : official publication of the Federation of American Societies for Experimental Biology

de Goede P, Wüst RCI, Schomakers BV, Denis S, Vaz FM, Pras-Raves ML, van Weeghel M, Yi CX, Kalsbeek A, Houtkooper RH.
PMID: 35032416
FASEB J. 2022 Feb;36(2):e22133. doi: 10.1096/fj.202100707R.

Shift-workers show an increased incidence of type 2 diabetes mellitus (T2DM). A possible mechanism is the disruption of the circadian timing of glucose homeostasis. Skeletal muscle mitochondrial function is modulated by the molecular clock. We used time-restricted feeding (TRF)...

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de Goede P, Wüst RCI, Schomakers BV, et al. Time-restricted feeding during the inactive phase abolishes the daily rhythm in mitochondrial respiration in rat skeletal muscle. FASEB J. 2022;36(2):e22133doi: 10.1096/fj.202100707R.
de Goede, P., Wüst, R. C. I., Schomakers, B. V., Denis, S., Vaz, F. M., Pras-Raves, M. L., van Weeghel, M., Yi, C. X., Kalsbeek, A., & Houtkooper, R. H. (2022). Time-restricted feeding during the inactive phase abolishes the daily rhythm in mitochondrial respiration in rat skeletal muscle. FASEB journal : official publication of the Federation of American Societies for Experimental Biology, 36(2), e22133. https://doi.org/10.1096/fj.202100707R
de Goede, Paul, et al. "Time-restricted feeding during the inactive phase abolishes the daily rhythm in mitochondrial respiration in rat skeletal muscle." FASEB journal : official publication of the Federation of American Societies for Experimental Biology vol. 36,2 (2022): e22133. doi: https://doi.org/10.1096/fj.202100707R
de Goede P, Wüst RCI, Schomakers BV, Denis S, Vaz FM, Pras-Raves ML, van Weeghel M, Yi CX, Kalsbeek A, Houtkooper RH. Time-restricted feeding during the inactive phase abolishes the daily rhythm in mitochondrial respiration in rat skeletal muscle. FASEB J. 2022 Feb;36(2):e22133. doi: 10.1096/fj.202100707R. PMID: 35032416.
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Showing 1 to 12 of 28 entries