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Autoantibody Repertoire in APECED Patients Targets Two Distinct Subgroups of Proteins.

Frontiers in immunology

Fishman D, Kisand K, Hertel C, Rothe M, Remm A, Pihlap M, Adler P, Vilo J, Peet A, Meloni A, Podkrajsek KT, Battelino T, Bruserud Ø, Wolff ASB, Husebye ES, Kluger N, Krohn K, Ranki A, Peterson H, Hayday A, Peterson P.
PMID: 28861084
Front Immunol. 2017 Aug 16;8:976. doi: 10.3389/fimmu.2017.00976. eCollection 2017.

High titer autoantibodies produced by B lymphocytes are clinically important features of many common autoimmune diseases. APECED patients with deficient autoimmune regulator (AIRE) gene collectively display a broad repertoire of high titer autoantibodies, including some which are pathognomonic for...

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Fishman D, Kisand K, Hertel C, et al. Autoantibody Repertoire in APECED Patients Targets Two Distinct Subgroups of Proteins. Front Immunol. 2017;8:976doi: 10.3389/fimmu.2017.00976.
Fishman, D., Kisand, K., Hertel, C., Rothe, M., Remm, A., Pihlap, M., Adler, P., Vilo, J., Peet, A., Meloni, A., Podkrajsek, K. T., Battelino, T., Bruserud, �. �., Wolff, A. S. B., Husebye, E. S., Kluger, N., Krohn, K., Ranki, A., Peterson, H., Hayday, A., & Peterson, P. (2017). Autoantibody Repertoire in APECED Patients Targets Two Distinct Subgroups of Proteins. Frontiers in immunology, 8976. https://doi.org/10.3389/fimmu.2017.00976
Fishman, Dmytro, et al. "Autoantibody Repertoire in APECED Patients Targets Two Distinct Subgroups of Proteins." Frontiers in immunology vol. 8 (2017): 976. doi: https://doi.org/10.3389/fimmu.2017.00976
Fishman D, Kisand K, Hertel C, Rothe M, Remm A, Pihlap M, Adler P, Vilo J, Peet A, Meloni A, Podkrajsek KT, Battelino T, Bruserud Ø, Wolff ASB, Husebye ES, Kluger N, Krohn K, Ranki A, Peterson H, Hayday A, Peterson P. Autoantibody Repertoire in APECED Patients Targets Two Distinct Subgroups of Proteins. Front Immunol. 2017 Aug 16;8:976. doi: 10.3389/fimmu.2017.00976. eCollection 2017. PMID: 28861084; PMCID: PMC5561390.
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Correction: Linkage Analysis in Autoimmune Addison's Disease: NFATC1 as a Potential Novel Susceptibility Locus.

PloS one

Mitchell AL, Bøe Wolff A, MacArthur K, Weaver JU, Vaidya B, Erichsen MM, Darlay R, Husebye ES, Cordell HJ, Pearce SH.
PMID: 26382621
PLoS One. 2015 Sep 18;10(9):e0138844. doi: 10.1371/journal.pone.0138844. eCollection 2015.

No abstract available.

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Mitchell AL, Bøe Wolff A, MacArthur K, et al. Correction: Linkage Analysis in Autoimmune Addison's Disease: NFATC1 as a Potential Novel Susceptibility Locus. PLoS One. 2015;10(9):e0138844doi: 10.1371/journal.pone.0138844.
Mitchell, A. L., Bøe Wolff, A., MacArthur, K., Weaver, J. U., Vaidya, B., Erichsen, M. M., Darlay, R., Husebye, E. S., Cordell, H. J., & Pearce, S. H. (2015). Correction: Linkage Analysis in Autoimmune Addison's Disease: NFATC1 as a Potential Novel Susceptibility Locus. PloS one, 10(9), e0138844. https://doi.org/10.1371/journal.pone.0138844
Mitchell, Anna L, et al. "Correction: Linkage Analysis in Autoimmune Addison's Disease: NFATC1 as a Potential Novel Susceptibility Locus." PloS one vol. 10,9 (2015): e0138844. doi: https://doi.org/10.1371/journal.pone.0138844
Mitchell AL, Bøe Wolff A, MacArthur K, Weaver JU, Vaidya B, Erichsen MM, Darlay R, Husebye ES, Cordell HJ, Pearce SH. Correction: Linkage Analysis in Autoimmune Addison's Disease: NFATC1 as a Potential Novel Susceptibility Locus. PLoS One. 2015 Sep 18;10(9):e0138844. doi: 10.1371/journal.pone.0138844. eCollection 2015. PMID: 26382621; PMCID: PMC4575066.
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Oral microbiota in autoimmune polyendocrine syndrome type 1.

Journal of oral microbiology

Bruserud Ø, Siddiqui H, Marthinussen MC, Chen T, Jonsson R, Oftedal BE, Olsen I, Husebye ES, Wolff AB.
PMID: 29503707
J Oral Microbiol. 2018 Feb 26;10(1):1442986. doi: 10.1080/20002297.2018.1442986. eCollection 2018.

No abstract available.

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Bruserud Ø, Siddiqui H, Marthinussen MC, et al. Oral microbiota in autoimmune polyendocrine syndrome type 1. J Oral Microbiol. 2018;10(1):1442986doi: 10.1080/20002297.2018.1442986.
Bruserud, �. �., Siddiqui, H., Marthinussen, M. C., Chen, T., Jonsson, R., Oftedal, B. E., Olsen, I., Husebye, E. S., & Wolff, A. B. (2018). Oral microbiota in autoimmune polyendocrine syndrome type 1. Journal of oral microbiology, 10(1), 1442986. https://doi.org/10.1080/20002297.2018.1442986
Bruserud, Øyvind, et al. "Oral microbiota in autoimmune polyendocrine syndrome type 1." Journal of oral microbiology vol. 10,1 (2018): 1442986. doi: https://doi.org/10.1080/20002297.2018.1442986
Bruserud Ø, Siddiqui H, Marthinussen MC, Chen T, Jonsson R, Oftedal BE, Olsen I, Husebye ES, Wolff AB. Oral microbiota in autoimmune polyendocrine syndrome type 1. J Oral Microbiol. 2018 Feb 26;10(1):1442986. doi: 10.1080/20002297.2018.1442986. eCollection 2018. PMID: 29503707; PMCID: PMC5827717.
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Potential Transcriptional Biomarkers to Guide Glucocorticoid Replacement in Autoimmune Addison's Disease.

Journal of the Endocrine Society

Sævik ÅB, Wolff AB, Björnsdottir S, Simunkova K, Hynne MS, Dolan DWP, Bratland E, Knappskog PM, Methlie P, Carlsen S, Isaksson M, Bensing S, Kämpe O, Husebye ES, Løvås K, Øksnes M.
PMID: 33553982
J Endocr Soc. 2021 Jan 04;5(3):bvaa202. doi: 10.1210/jendso/bvaa202. eCollection 2021 Mar 01.

BACKGROUND: No reliable biomarkers exist to guide glucocorticoid (GC) replacement treatment in autoimmune Addison's disease (AAD), leading to overtreatment with alarming and persistent side effects or undertreatment, which could be fatal.OBJECTIVE: To explore changes in gene expression following different...

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Sævik ÅB, Wolff AB, Björnsdottir S, et al. Potential Transcriptional Biomarkers to Guide Glucocorticoid Replacement in Autoimmune Addison's Disease. J Endocr Soc. 2021;5(3):bvaa202doi: 10.1210/jendso/bvaa202.
Sævik, �. �. B., Wolff, A. B., Björnsdottir, S., Simunkova, K., Hynne, M. S., Dolan, D. W. P., Bratland, E., Knappskog, P. M., Methlie, P., Carlsen, S., Isaksson, M., Bensing, S., Kämpe, O., Husebye, E. S., Løvås, K., & Øksnes, M. (2021). Potential Transcriptional Biomarkers to Guide Glucocorticoid Replacement in Autoimmune Addison's Disease. Journal of the Endocrine Society, 5(3), bvaa202. https://doi.org/10.1210/jendso/bvaa202
Sævik, Åse Bjorvatn, et al. "Potential Transcriptional Biomarkers to Guide Glucocorticoid Replacement in Autoimmune Addison's Disease." Journal of the Endocrine Society vol. 5,3 (2021): bvaa202. doi: https://doi.org/10.1210/jendso/bvaa202
Sævik ÅB, Wolff AB, Björnsdottir S, Simunkova K, Hynne MS, Dolan DWP, Bratland E, Knappskog PM, Methlie P, Carlsen S, Isaksson M, Bensing S, Kämpe O, Husebye ES, Løvås K, Øksnes M. Potential Transcriptional Biomarkers to Guide Glucocorticoid Replacement in Autoimmune Addison's Disease. J Endocr Soc. 2021 Jan 04;5(3):bvaa202. doi: 10.1210/jendso/bvaa202. eCollection 2021 Mar 01. PMID: 33553982; PMCID: PMC7853175.
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The curious case of a cryptic Cryptosporidium and a missing dendritic cell subset.

Trends in parasitology

Lee D, Huseby ES, Gowthaman U.
PMID: 34953724
Trends Parasitol. 2022 Feb;38(2):101-103. doi: 10.1016/j.pt.2021.12.003. Epub 2021 Dec 22.

Animal models for studying immune responses to Cryptosporidium, a parasite that causes gastrointestinal disease, have been a challenge due to the parasite's poor infectivity in mice. Russler-Germain et al. discovered a 'commensal' strain of Cryptosporidium, capable of stable infection...

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Lee D, Huseby ES, Gowthaman U. The curious case of a cryptic Cryptosporidium and a missing dendritic cell subset. Trends Parasitol. 2021;38(2):101-103doi: 10.1016/j.pt.2021.12.003.
Lee, D., Huseby, E. S., & Gowthaman, U. (2022). The curious case of a cryptic Cryptosporidium and a missing dendritic cell subset. Trends in parasitology, 38(2), 101-103. https://doi.org/10.1016/j.pt.2021.12.003
Lee, Donguk, et al. "The curious case of a cryptic Cryptosporidium and a missing dendritic cell subset." Trends in parasitology vol. 38,2 (2022): 101-103. doi: https://doi.org/10.1016/j.pt.2021.12.003
Lee D, Huseby ES, Gowthaman U. The curious case of a cryptic Cryptosporidium and a missing dendritic cell subset. Trends Parasitol. 2022 Feb;38(2):101-103. doi: 10.1016/j.pt.2021.12.003. Epub 2021 Dec 22. PMID: 34953724.
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Bedtime Salivary Cortisol as a Screening Test for Cushing Syndrome in Children.

Journal of the Endocrine Society

Ueland GÅ, Kellmann R, Jørstad Davidsen M, Viste K, Husebye ES, Almås B, Storr HL, Sagen JV, Mellgren G, Júlíusson PB, Methlie P.
PMID: 33928203
J Endocr Soc. 2021 Mar 04;5(5):bvab033. doi: 10.1210/jendso/bvab033. eCollection 2021 May 01.

BACKGROUND: Diagnosing Cushing syndrome (CS) can be challenging. The 24-hour urine free cortisol (UFC) measurement is considered gold standard. This is a laborious test, dependent on correct urine collection. Late-night salivary cortisol is easier and is used as a...

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Ueland GÅ, Kellmann R, Jørstad Davidsen M, et al. Bedtime Salivary Cortisol as a Screening Test for Cushing Syndrome in Children. J Endocr Soc. 2021;5(5):bvab033doi: 10.1210/jendso/bvab033.
Ueland, G. �. �., Kellmann, R., Jørstad Davidsen, M., Viste, K., Husebye, E. S., Almås, B., Storr, H. L., Sagen, J. V., Mellgren, G., Júlíusson, P. B., & Methlie, P. (2021). Bedtime Salivary Cortisol as a Screening Test for Cushing Syndrome in Children. Journal of the Endocrine Society, 5(5), bvab033. https://doi.org/10.1210/jendso/bvab033
Ueland, Grethe Å, et al. "Bedtime Salivary Cortisol as a Screening Test for Cushing Syndrome in Children." Journal of the Endocrine Society vol. 5,5 (2021): bvab033. doi: https://doi.org/10.1210/jendso/bvab033
Ueland GÅ, Kellmann R, Jørstad Davidsen M, Viste K, Husebye ES, Almås B, Storr HL, Sagen JV, Mellgren G, Júlíusson PB, Methlie P. Bedtime Salivary Cortisol as a Screening Test for Cushing Syndrome in Children. J Endocr Soc. 2021 Mar 04;5(5):bvab033. doi: 10.1210/jendso/bvab033. eCollection 2021 May 01. PMID: 33928203; PMCID: PMC8064046.
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Bedtime Salivary Cortisol as a Screening Test for Cushing Syndrome in Children.

Journal of the Endocrine Society

Ueland GÅ, Kellmann R, Jørstad Davidsen M, Viste K, Husebye ES, Almås B, Storr HL, Sagen JV, Mellgren G, Júlíusson PB, Methlie P.
PMID: 33928203
J Endocr Soc. 2021 Mar 04;5(5):bvab033. doi: 10.1210/jendso/bvab033. eCollection 2021 May 01.

BACKGROUND: Diagnosing Cushing syndrome (CS) can be challenging. The 24-hour urine free cortisol (UFC) measurement is considered gold standard. This is a laborious test, dependent on correct urine collection. Late-night salivary cortisol is easier and is used as a...

Cite
Ueland GÅ, Kellmann R, Jørstad Davidsen M, et al. Bedtime Salivary Cortisol as a Screening Test for Cushing Syndrome in Children. J Endocr Soc. 2021;5(5):bvab033doi: 10.1210/jendso/bvab033.
Ueland, G. �. �., Kellmann, R., Jørstad Davidsen, M., Viste, K., Husebye, E. S., Almås, B., Storr, H. L., Sagen, J. V., Mellgren, G., Júlíusson, P. B., & Methlie, P. (2021). Bedtime Salivary Cortisol as a Screening Test for Cushing Syndrome in Children. Journal of the Endocrine Society, 5(5), bvab033. https://doi.org/10.1210/jendso/bvab033
Ueland, Grethe Å, et al. "Bedtime Salivary Cortisol as a Screening Test for Cushing Syndrome in Children." Journal of the Endocrine Society vol. 5,5 (2021): bvab033. doi: https://doi.org/10.1210/jendso/bvab033
Ueland GÅ, Kellmann R, Jørstad Davidsen M, Viste K, Husebye ES, Almås B, Storr HL, Sagen JV, Mellgren G, Júlíusson PB, Methlie P. Bedtime Salivary Cortisol as a Screening Test for Cushing Syndrome in Children. J Endocr Soc. 2021 Mar 04;5(5):bvab033. doi: 10.1210/jendso/bvab033. eCollection 2021 May 01. PMID: 33928203; PMCID: PMC8064046.
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Potential Transcriptional Biomarkers to Guide Glucocorticoid Replacement in Autoimmune Addison's Disease.

Journal of the Endocrine Society

Sævik ÅB, Wolff AB, Björnsdottir S, Simunkova K, Hynne MS, Dolan DWP, Bratland E, Knappskog PM, Methlie P, Carlsen S, Isaksson M, Bensing S, Kämpe O, Husebye ES, Løvås K, Øksnes M.
PMID: 33553982
J Endocr Soc. 2021 Jan 04;5(3):bvaa202. doi: 10.1210/jendso/bvaa202. eCollection 2021 Mar 01.

BACKGROUND: No reliable biomarkers exist to guide glucocorticoid (GC) replacement treatment in autoimmune Addison's disease (AAD), leading to overtreatment with alarming and persistent side effects or undertreatment, which could be fatal.OBJECTIVE: To explore changes in gene expression following different...

Cite
Sævik ÅB, Wolff AB, Björnsdottir S, et al. Potential Transcriptional Biomarkers to Guide Glucocorticoid Replacement in Autoimmune Addison's Disease. J Endocr Soc. 2021;5(3):bvaa202doi: 10.1210/jendso/bvaa202.
Sævik, �. �. B., Wolff, A. B., Björnsdottir, S., Simunkova, K., Hynne, M. S., Dolan, D. W. P., Bratland, E., Knappskog, P. M., Methlie, P., Carlsen, S., Isaksson, M., Bensing, S., Kämpe, O., Husebye, E. S., Løvås, K., & Øksnes, M. (2021). Potential Transcriptional Biomarkers to Guide Glucocorticoid Replacement in Autoimmune Addison's Disease. Journal of the Endocrine Society, 5(3), bvaa202. https://doi.org/10.1210/jendso/bvaa202
Sævik, Åse Bjorvatn, et al. "Potential Transcriptional Biomarkers to Guide Glucocorticoid Replacement in Autoimmune Addison's Disease." Journal of the Endocrine Society vol. 5,3 (2021): bvaa202. doi: https://doi.org/10.1210/jendso/bvaa202
Sævik ÅB, Wolff AB, Björnsdottir S, Simunkova K, Hynne MS, Dolan DWP, Bratland E, Knappskog PM, Methlie P, Carlsen S, Isaksson M, Bensing S, Kämpe O, Husebye ES, Løvås K, Øksnes M. Potential Transcriptional Biomarkers to Guide Glucocorticoid Replacement in Autoimmune Addison's Disease. J Endocr Soc. 2021 Jan 04;5(3):bvaa202. doi: 10.1210/jendso/bvaa202. eCollection 2021 Mar 01. PMID: 33553982; PMCID: PMC7853175.
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21-Hydroxylase-Specific CD8+ T Cells in Autoimmune Addison's Disease Are Restricted by HLA-A2 and HLA-C7 Molecules.

Frontiers in immunology

Hellesen A, Aslaksen S, Breivik L, Røyrvik EC, Bruserud Ø, Edvardsen K, Brokstad KA, Wolff ASB, Husebye ES, Bratland E.
PMID: 34721410
Front Immunol. 2021 Oct 14;12:742848. doi: 10.3389/fimmu.2021.742848. eCollection 2021.

OBJECTIVES: CD8+ T cells targeting 21-hydroxylase (21OH) are presumed to play a central role in the destruction of adrenocortical cells in autoimmune Addison's disease (AAD). Earlier reports have suggested two immunodominant CD8+ T cell epitopes within 21OH: LLNATIAEV (21OHMETHODS:...

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Hellesen A, Aslaksen S, Breivik L, et al. 21-Hydroxylase-Specific CD8+ T Cells in Autoimmune Addison's Disease Are Restricted by HLA-A2 and HLA-C7 Molecules. Front Immunol. 2021;12:742848doi: 10.3389/fimmu.2021.742848.
Hellesen, A., Aslaksen, S., Breivik, L., Røyrvik, E. C., Bruserud, �. �., Edvardsen, K., Brokstad, K. A., Wolff, A. S. B., Husebye, E. S., & Bratland, E. (2021). 21-Hydroxylase-Specific CD8+ T Cells in Autoimmune Addison's Disease Are Restricted by HLA-A2 and HLA-C7 Molecules. Frontiers in immunology, 12742848. https://doi.org/10.3389/fimmu.2021.742848
Hellesen, Alexander, et al. "21-Hydroxylase-Specific CD8+ T Cells in Autoimmune Addison's Disease Are Restricted by HLA-A2 and HLA-C7 Molecules." Frontiers in immunology vol. 12 (2021): 742848. doi: https://doi.org/10.3389/fimmu.2021.742848
Hellesen A, Aslaksen S, Breivik L, Røyrvik EC, Bruserud Ø, Edvardsen K, Brokstad KA, Wolff ASB, Husebye ES, Bratland E. 21-Hydroxylase-Specific CD8+ T Cells in Autoimmune Addison's Disease Are Restricted by HLA-A2 and HLA-C7 Molecules. Front Immunol. 2021 Oct 14;12:742848. doi: 10.3389/fimmu.2021.742848. eCollection 2021. PMID: 34721410; PMCID: PMC8551825.
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Outcome after nonoperative treatment of stable Lisfranc injuries. A prospective cohort study.

Foot and ankle surgery : official journal of the European Society of Foot and Ankle Surgeons

Stødle AH, Hvaal KH, Brøgger H, Madsen JE, Husebye EE.
PMID: 33832813
Foot Ankle Surg. 2021 Mar 27; doi: 10.1016/j.fas.2021.03.017. Epub 2021 Mar 27.

BACKGROUND: The aim of this study was to evaluate the outcome after nondisplaced and stable Lisfranc injuries.METHODS: 26 patients with injuries to the Lisfranc joint complex detected on CT scans, but without displacement were tested to be stable using...

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Stødle AH, Hvaal KH, Brøgger H, et al. Outcome after nonoperative treatment of stable Lisfranc injuries. A prospective cohort study. Foot Ankle Surg. 2021;doi: 10.1016/j.fas.2021.03.017.
Stødle, A. H., Hvaal, K. H., Brøgger, H., Madsen, J. E., & Husebye, E. E. (2021). Outcome after nonoperative treatment of stable Lisfranc injuries. A prospective cohort study. Foot and ankle surgery : official journal of the European Society of Foot and Ankle Surgeons, . https://doi.org/10.1016/j.fas.2021.03.017
Stødle, Are H, et al. "Outcome after nonoperative treatment of stable Lisfranc injuries. A prospective cohort study." Foot and ankle surgery : official journal of the European Society of Foot and Ankle Surgeons vol. (2021). doi: https://doi.org/10.1016/j.fas.2021.03.017
Stødle AH, Hvaal KH, Brøgger H, Madsen JE, Husebye EE. Outcome after nonoperative treatment of stable Lisfranc injuries. A prospective cohort study. Foot Ankle Surg. 2021 Mar 27; doi: 10.1016/j.fas.2021.03.017. Epub 2021 Mar 27. PMID: 33832813.
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Mechanistic dissection of dominant AIRE mutations in mouse models reveals AIRE autoregulation.

The Journal of experimental medicine

Goldfarb Y, Givony T, Kadouri N, Dobeš J, Peligero-Cruz C, Zalayat I, Damari G, Dassa B, Ben-Dor S, Gruper Y, Oftedal BE, Bratland E, Erichsen MM, Berger A, Avin A, Nevo S, Haljasorg U, Kuperman Y, Ulman A, Haffner-Krausz R, Porat Z, Atasoy U, Leshkowitz D, Husebye ES, Abramson J.
PMID: 34477806
J Exp Med. 2021 Nov 01;218(11). doi: 10.1084/jem.20201076. Epub 2021 Sep 03.

The autoimmune regulator (AIRE) is essential for the establishment of central tolerance and prevention of autoimmunity. Interestingly, different AIRE mutations cause autoimmunity in either recessive or dominant-negative manners. Using engineered mouse models, we establish that some monoallelic mutants, including...

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Goldfarb Y, Givony T, Kadouri N, et al. Mechanistic dissection of dominant AIRE mutations in mouse models reveals AIRE autoregulation. J Exp Med. 2021;218(11)doi: 10.1084/jem.20201076.
Goldfarb, Y., Givony, T., Kadouri, N., Dobeš, J., Peligero-Cruz, C., Zalayat, I., Damari, G., Dassa, B., Ben-Dor, S., Gruper, Y., Oftedal, B. E., Bratland, E., Erichsen, M. M., Berger, A., Avin, A., Nevo, S., Haljasorg, U., Kuperman, Y., Ulman, A., Haffner-Krausz, R., Porat, Z., Atasoy, U., Leshkowitz, D., Husebye, E. S., & Abramson, J. (2021). Mechanistic dissection of dominant AIRE mutations in mouse models reveals AIRE autoregulation. The Journal of experimental medicine, 218(11), . https://doi.org/10.1084/jem.20201076
Goldfarb, Yael, et al. "Mechanistic dissection of dominant AIRE mutations in mouse models reveals AIRE autoregulation." The Journal of experimental medicine vol. 218,11 (2021). doi: https://doi.org/10.1084/jem.20201076
Goldfarb Y, Givony T, Kadouri N, Dobeš J, Peligero-Cruz C, Zalayat I, Damari G, Dassa B, Ben-Dor S, Gruper Y, Oftedal BE, Bratland E, Erichsen MM, Berger A, Avin A, Nevo S, Haljasorg U, Kuperman Y, Ulman A, Haffner-Krausz R, Porat Z, Atasoy U, Leshkowitz D, Husebye ES, Abramson J. Mechanistic dissection of dominant AIRE mutations in mouse models reveals AIRE autoregulation. J Exp Med. 2021 Nov 01;218(11). doi: 10.1084/jem.20201076. Epub 2021 Sep 03. PMID: 34477806; PMCID: PMC8421262.
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Hepcidin and Ferritin Predict Microbial Etiology in Community-Acquired Pneumonia.

Open forum infectious diseases

Oppen K, Ueland T, Siljan WW, Skadberg Ø, Brede C, Lauritzen T, Aukrust P, Steinsvik T, Husebye E, Michelsen AE, Holter JC, Heggelund L.
PMID: 33880390
Open Forum Infect Dis. 2021 Feb 18;8(4):ofab082. doi: 10.1093/ofid/ofab082. eCollection 2021 Apr.

BACKGROUND: Iron is crucial for survival and growth of microbes. Consequently, limiting iron availability is a human antimicrobial defense mechanism. We explored iron and iron-related proteins as potential biomarkers in community-acquired pneumonia and hypothesized that infection-induced changes in these...

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Oppen K, Ueland T, Siljan WW, et al. Hepcidin and Ferritin Predict Microbial Etiology in Community-Acquired Pneumonia. Open Forum Infect Dis. 2021;8(4):ofab082doi: 10.1093/ofid/ofab082.
Oppen, K., Ueland, T., Siljan, W. W., Skadberg, �. �., Brede, C., Lauritzen, T., Aukrust, P., Steinsvik, T., Husebye, E., Michelsen, A. E., Holter, J. C., & Heggelund, L. (2021). Hepcidin and Ferritin Predict Microbial Etiology in Community-Acquired Pneumonia. Open forum infectious diseases, 8(4), ofab082. https://doi.org/10.1093/ofid/ofab082
Oppen, Kjersti, et al. "Hepcidin and Ferritin Predict Microbial Etiology in Community-Acquired Pneumonia." Open forum infectious diseases vol. 8,4 (2021): ofab082. doi: https://doi.org/10.1093/ofid/ofab082
Oppen K, Ueland T, Siljan WW, Skadberg Ø, Brede C, Lauritzen T, Aukrust P, Steinsvik T, Husebye E, Michelsen AE, Holter JC, Heggelund L. Hepcidin and Ferritin Predict Microbial Etiology in Community-Acquired Pneumonia. Open Forum Infect Dis. 2021 Feb 18;8(4):ofab082. doi: 10.1093/ofid/ofab082. eCollection 2021 Apr. PMID: 33880390; PMCID: PMC8043258.
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Showing 1 to 12 of 57 entries