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[Survival rate].

Kyobu geka. The Japanese journal of thoracic surgery

Agata T, Kano K.
PMID: 3386082
Kyobu Geka. 1988 Mar;41(3):260-2.

No abstract available.

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Agata T, Kano K. [Survival rate]. Kyobu Geka. 1988;41(3):260-2
Agata, T., & Kano, K. (1988). [Survival rate]. Kyobu geka. The Japanese journal of thoracic surgery, 41(3), 260-2.
Agata, T, and Kano, K. "[Survival rate]." Kyobu geka. The Japanese journal of thoracic surgery vol. 41,3 (1988): 260-2.
Agata T, Kano K. [Survival rate]. Kyobu Geka. 1988 Mar;41(3):260-2. Japanese. PMID: 3386082.
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A catalog of hemizygous variation in 127 22q11 deletion patients.

Human genome variation

Hestand MS, Nowakowska BA, Vergaelen E, Van Houdt J, Dehaspe L, Suhl JA, Del-Favero J, Mortier G, Zackai E, Swillen A, Devriendt K, Gur RE, McDonald-McGinn DM, Warren ST, Emanuel BS, Vermeesch JR.
PMID: 27274857
Hum Genome Var. 2016 Jan 14;3:15065. doi: 10.1038/hgv.2015.65. eCollection 2016.

The 22q11.2 deletion syndrome is the most common microdeletion disorder, with wide phenotypic variability. To investigate variation within the non-deleted allele we performed targeted resequencing of the 22q11.2 region for 127 patients, identifying multiple deletion sizes, including two deletions...

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Hestand MS, Nowakowska BA, Vergaelen E, et al. A catalog of hemizygous variation in 127 22q11 deletion patients. Hum Genome Var. 2016;3:15065doi: 10.1038/hgv.2015.65.
Hestand, M. S., Nowakowska, B. A., Vergaelen, E., Van Houdt, J., Dehaspe, L., Suhl, J. A., Del-Favero, J., Mortier, G., Zackai, E., Swillen, A., Devriendt, K., Gur, R. E., McDonald-McGinn, D. M., Warren, S. T., Emanuel, B. S., & Vermeesch, J. R. (2016). A catalog of hemizygous variation in 127 22q11 deletion patients. Human genome variation, 315065. https://doi.org/10.1038/hgv.2015.65
Hestand, Matthew S, et al. "A catalog of hemizygous variation in 127 22q11 deletion patients." Human genome variation vol. 3 (2016): 15065. doi: https://doi.org/10.1038/hgv.2015.65
Hestand MS, Nowakowska BA, Vergaelen E, Van Houdt J, Dehaspe L, Suhl JA, Del-Favero J, Mortier G, Zackai E, Swillen A, Devriendt K, Gur RE, McDonald-McGinn DM, Warren ST, Emanuel BS, Vermeesch JR. A catalog of hemizygous variation in 127 22q11 deletion patients. Hum Genome Var. 2016 Jan 14;3:15065. doi: 10.1038/hgv.2015.65. eCollection 2016. PMID: 27274857; PMCID: PMC4892188.
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Isolation of microorganisms involved in reduction of crystalline iron(III) oxides in natural environments.

Frontiers in microbiology

Hori T, Aoyagi T, Itoh H, Narihiro T, Oikawa A, Suzuki K, Ogata A, Friedrich MW, Conrad R, Kamagata Y.
PMID: 25999927
Front Microbiol. 2015 May 05;6:386. doi: 10.3389/fmicb.2015.00386. eCollection 2015.

Reduction of crystalline Fe(III) oxides is one of the most important electron sinks for organic compound oxidation in natural environments. Yet the limited number of isolates makes it difficult to understand the physiology and ecological impact of the microorganisms...

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Hori T, Aoyagi T, Itoh H, et al. Isolation of microorganisms involved in reduction of crystalline iron(III) oxides in natural environments. Front Microbiol. 2015;6:386doi: 10.3389/fmicb.2015.00386.
Hori, T., Aoyagi, T., Itoh, H., Narihiro, T., Oikawa, A., Suzuki, K., Ogata, A., Friedrich, M. W., Conrad, R., & Kamagata, Y. (2015). Isolation of microorganisms involved in reduction of crystalline iron(III) oxides in natural environments. Frontiers in microbiology, 6386. https://doi.org/10.3389/fmicb.2015.00386
Hori, Tomoyuki, et al. "Isolation of microorganisms involved in reduction of crystalline iron(III) oxides in natural environments." Frontiers in microbiology vol. 6 (2015): 386. doi: https://doi.org/10.3389/fmicb.2015.00386
Hori T, Aoyagi T, Itoh H, Narihiro T, Oikawa A, Suzuki K, Ogata A, Friedrich MW, Conrad R, Kamagata Y. Isolation of microorganisms involved in reduction of crystalline iron(III) oxides in natural environments. Front Microbiol. 2015 May 05;6:386. doi: 10.3389/fmicb.2015.00386. eCollection 2015. PMID: 25999927; PMCID: PMC4419728.
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A novel baculovirus-derived promoter with high activity in the baculovirus expression system.

PeerJ

Martínez-Solís M, Gómez-Sebastián S, Escribano JM, Jakubowska AK, Herrero S.
PMID: 27375973
PeerJ. 2016 Jun 28;4:e2183. doi: 10.7717/peerj.2183. eCollection 2016.

The baculovirus expression vector system (BEVS) has been widely used to produce a large number of recombinant proteins, and is becoming one of the most powerful, robust, and cost-effective systems for the production of eukaryotic proteins. Nevertheless, as in...

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Martínez-Solís M, Gómez-Sebastián S, Escribano JM, et al. A novel baculovirus-derived promoter with high activity in the baculovirus expression system. PeerJ. 2016;4:e2183doi: 10.7717/peerj.2183.
Martínez-Solís, M., Gómez-Sebastián, S., Escribano, J. M., Jakubowska, A. K., & Herrero, S. (2016). A novel baculovirus-derived promoter with high activity in the baculovirus expression system. PeerJ, 4e2183. https://doi.org/10.7717/peerj.2183
Martínez-Solís, María, et al. "A novel baculovirus-derived promoter with high activity in the baculovirus expression system." PeerJ vol. 4 (2016): e2183. doi: https://doi.org/10.7717/peerj.2183
Martínez-Solís M, Gómez-Sebastián S, Escribano JM, Jakubowska AK, Herrero S. A novel baculovirus-derived promoter with high activity in the baculovirus expression system. PeerJ. 2016 Jun 28;4:e2183. doi: 10.7717/peerj.2183. eCollection 2016. PMID: 27375973; PMCID: PMC4928464.
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The availability of six tracer medicines in private medicine outlets in Uganda.

Journal of pharmaceutical policy and practice

Birabwa C, Murison J, Evans V, Obua C, Agaba A, Waako P, Pollock A.
PMID: 25667747
J Pharm Policy Pract. 2014 Dec 08;7(1):18. doi: 10.1186/s40545-014-0018-y. eCollection 2014.

OBJECTIVES: Many low income countries struggle to provide safe and effective medicines due to poor public health care infrastructure, budgetary constraints, and lack of human resource capacity. Private sector pharmacies and drug shops are used by a majority of...

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Birabwa C, Murison J, Evans V, et al. The availability of six tracer medicines in private medicine outlets in Uganda. J Pharm Policy Pract. 2014;7(1):18doi: 10.1186/s40545-014-0018-y.
Birabwa, C., Murison, J., Evans, V., Obua, C., Agaba, A., Waako, P., & Pollock, A. (2014). The availability of six tracer medicines in private medicine outlets in Uganda. Journal of pharmaceutical policy and practice, 7(1), 18. https://doi.org/10.1186/s40545-014-0018-y
Birabwa, Catherine, et al. "The availability of six tracer medicines in private medicine outlets in Uganda." Journal of pharmaceutical policy and practice vol. 7,1 (2014): 18. doi: https://doi.org/10.1186/s40545-014-0018-y
Birabwa C, Murison J, Evans V, Obua C, Agaba A, Waako P, Pollock A. The availability of six tracer medicines in private medicine outlets in Uganda. J Pharm Policy Pract. 2014 Dec 08;7(1):18. doi: 10.1186/s40545-014-0018-y. eCollection 2014. PMID: 25667747; PMCID: PMC4276013.
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Corrigendum: Arginine-rhamnosylation as new strategy to activate translation elongation factor P.

Nature chemical biology

Lassak J, Keilhauer EC, Fürst M, Wuichet K, Gödeke J, Starosta AL, Chen JM, Søgaard-Andersen L, Rohr J, Wilson DN, Häussler S, Mann M, Jung K.
PMID: 25785429
Nat Chem Biol. 2015 Apr;11(4):299. doi: 10.1038/nchembio0415-299d.

No abstract available.

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Lassak J, Keilhauer EC, Fürst M, et al. Corrigendum: Arginine-rhamnosylation as new strategy to activate translation elongation factor P. Nat Chem Biol. 2015;11(4):299doi: 10.1038/nchembio0415-299d.
Lassak, J., Keilhauer, E. C., Fürst, M., Wuichet, K., Gödeke, J., Starosta, A. L., Chen, J. M., Søgaard-Andersen, L., Rohr, J., Wilson, D. N., Häussler, S., Mann, M., & Jung, K. (2015). Corrigendum: Arginine-rhamnosylation as new strategy to activate translation elongation factor P. Nature chemical biology, 11(4), 299. https://doi.org/10.1038/nchembio0415-299d
Lassak, Jürgen, et al. "Corrigendum: Arginine-rhamnosylation as new strategy to activate translation elongation factor P." Nature chemical biology vol. 11,4 (2015): 299. doi: https://doi.org/10.1038/nchembio0415-299d
Lassak J, Keilhauer EC, Fürst M, Wuichet K, Gödeke J, Starosta AL, Chen JM, Søgaard-Andersen L, Rohr J, Wilson DN, Häussler S, Mann M, Jung K. Corrigendum: Arginine-rhamnosylation as new strategy to activate translation elongation factor P. Nat Chem Biol. 2015 Apr;11(4):299. doi: 10.1038/nchembio0415-299d. PMID: 25785429.
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Inactivation of Sonic Hedgehog Signaling and Polydactyly in Limbs of Hereditary Multiple Malformation, a Novel Type of .

Frontiers in cell and developmental biology

Matsubara Y, Nakano M, Kawamura K, Tsudzuki M, Funahashi JI, Agata K, Matsuda Y, Kuroiwa A, Suzuki T.
PMID: 28083533
Front Cell Dev Biol. 2016 Dec 27;4:149. doi: 10.3389/fcell.2016.00149. eCollection 2016.

Hereditary Multiple Malformation (HMM) is a naturally occurring, autosomal recessive, homozygous lethal mutation found in Japanese quail. Homozygote embryos (

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Matsubara Y, Nakano M, Kawamura K, et al. Inactivation of Sonic Hedgehog Signaling and Polydactyly in Limbs of Hereditary Multiple Malformation, a Novel Type of . Front Cell Dev Biol. 2016;4:149doi: 10.3389/fcell.2016.00149.
Matsubara, Y., Nakano, M., Kawamura, K., Tsudzuki, M., Funahashi, J. I., Agata, K., Matsuda, Y., Kuroiwa, A., & Suzuki, T. (2016). Inactivation of Sonic Hedgehog Signaling and Polydactyly in Limbs of Hereditary Multiple Malformation, a Novel Type of . Frontiers in cell and developmental biology, 4149. https://doi.org/10.3389/fcell.2016.00149
Matsubara, Yoshiyuki, et al. "Inactivation of Sonic Hedgehog Signaling and Polydactyly in Limbs of Hereditary Multiple Malformation, a Novel Type of ." Frontiers in cell and developmental biology vol. 4 (2016): 149. doi: https://doi.org/10.3389/fcell.2016.00149
Matsubara Y, Nakano M, Kawamura K, Tsudzuki M, Funahashi JI, Agata K, Matsuda Y, Kuroiwa A, Suzuki T. Inactivation of Sonic Hedgehog Signaling and Polydactyly in Limbs of Hereditary Multiple Malformation, a Novel Type of . Front Cell Dev Biol. 2016 Dec 27;4:149. doi: 10.3389/fcell.2016.00149. eCollection 2016. PMID: 28083533; PMCID: PMC5187386.
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Mild Electrical Stimulation with Heat Shock Reduces Visceral Adiposity and Improves Metabolic Abnormalities in Subjects with Metabolic Syndrome or Type 2 Diabetes: Randomized Crossover Trials.

EBioMedicine

Kondo T, Ono K, Kitano S, Matsuyama R, Goto R, Suico MA, Kawasaki S, Igata M, Kawashima J, Motoshima H, Matsumura T, Kai H, Araki E.
PMID: 26137510
EBioMedicine. 2014 Nov 11;1(1):80-9. doi: 10.1016/j.ebiom.2014.11.001. eCollection 2014 Nov.

BACKGROUND: The induction of heat shock protein (HSP) 72 by mild electrical stimulation with heat shock (MES + HS), which improves visceral adiposity and insulin resistance in mice, may be beneficial in treating metabolic syndrome (MS) or type 2...

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Kondo T, Ono K, Kitano S, et al. Mild Electrical Stimulation with Heat Shock Reduces Visceral Adiposity and Improves Metabolic Abnormalities in Subjects with Metabolic Syndrome or Type 2 Diabetes: Randomized Crossover Trials. EBioMedicine. 2014;1(1):80-9doi: 10.1016/j.ebiom.2014.11.001.
Kondo, T., Ono, K., Kitano, S., Matsuyama, R., Goto, R., Suico, M. A., Kawasaki, S., Igata, M., Kawashima, J., Motoshima, H., Matsumura, T., Kai, H., & Araki, E. (2014). Mild Electrical Stimulation with Heat Shock Reduces Visceral Adiposity and Improves Metabolic Abnormalities in Subjects with Metabolic Syndrome or Type 2 Diabetes: Randomized Crossover Trials. EBioMedicine, 1(1), 80-9. https://doi.org/10.1016/j.ebiom.2014.11.001
Kondo, Tatsuya, et al. "Mild Electrical Stimulation with Heat Shock Reduces Visceral Adiposity and Improves Metabolic Abnormalities in Subjects with Metabolic Syndrome or Type 2 Diabetes: Randomized Crossover Trials." EBioMedicine vol. 1,1 (2014): 80-9. doi: https://doi.org/10.1016/j.ebiom.2014.11.001
Kondo T, Ono K, Kitano S, Matsuyama R, Goto R, Suico MA, Kawasaki S, Igata M, Kawashima J, Motoshima H, Matsumura T, Kai H, Araki E. Mild Electrical Stimulation with Heat Shock Reduces Visceral Adiposity and Improves Metabolic Abnormalities in Subjects with Metabolic Syndrome or Type 2 Diabetes: Randomized Crossover Trials. EBioMedicine. 2014 Nov 11;1(1):80-9. doi: 10.1016/j.ebiom.2014.11.001. eCollection 2014 Nov. PMID: 26137510; PMCID: PMC4457350.
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Dependence of Temperature-Sensitivity of Poly(N-isopropylacrylamide-co-acrylic acid) Hydrogel Microspheres upon Their Sizes.

Journal of colloid and interface science

Makino K, Agata H, Ohshima H.
PMID: 10998296
J Colloid Interface Sci. 2000 Oct 01;230(1):128-134. doi: 10.1006/jcis.2000.7084.

Monodisperse poly(N-isopropylacrylamide-co-acrylic acid) hydrogel microspheres were prepared by a membrane emulsification method using membranes of pore diameters of 0.33, 0.73, 1.15, and 1.70 µm. The hydrogels were synthesized by polymerization of 3.6 M N-isopropylacrylamide (N-IPAAm or NIPAM) and 0.4...

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Makino K, Agata H, Ohshima H. Dependence of Temperature-Sensitivity of Poly(N-isopropylacrylamide-co-acrylic acid) Hydrogel Microspheres upon Their Sizes. J Colloid Interface Sci. 2000;230(1):128-134doi: 10.1006/jcis.2000.7084.
Makino, K., Agata, H., & Ohshima, H. (2000). Dependence of Temperature-Sensitivity of Poly(N-isopropylacrylamide-co-acrylic acid) Hydrogel Microspheres upon Their Sizes. Journal of colloid and interface science, 230(1), 128-134. https://doi.org/10.1006/jcis.2000.7084
Makino, et al. "Dependence of Temperature-Sensitivity of Poly(N-isopropylacrylamide-co-acrylic acid) Hydrogel Microspheres upon Their Sizes." Journal of colloid and interface science vol. 230,1 (2000): 128-134. doi: https://doi.org/10.1006/jcis.2000.7084
Makino K, Agata H, Ohshima H. Dependence of Temperature-Sensitivity of Poly(N-isopropylacrylamide-co-acrylic acid) Hydrogel Microspheres upon Their Sizes. J Colloid Interface Sci. 2000 Oct 01;230(1):128-134. doi: 10.1006/jcis.2000.7084. PMID: 10998296.
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SERRS. In situ substrate formation and improved detection using microfluidics.

Analytical chemistry

Keir R, Igata E, Arundell M, Smith WE, Graham D, McHugh C, Cooper JM.
PMID: 12033237
Anal Chem. 2002 Apr 01;74(7):1503-8. doi: 10.1021/ac015625+.

Surface-enhanced resonance Raman scattering (SERRS) of a model derivative of TNT was detected using a microflow cell designed within the framework of the lab-on-a-chip concept, using only the analyte and readily available reagents. The SERRS substrate, silver colloid, was...

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Keir R, Igata E, Arundell M, et al. SERRS. In situ substrate formation and improved detection using microfluidics. Anal Chem. 2002;74(7):1503-8doi: 10.1021/ac015625+.
Keir, R., Igata, E., Arundell, M., Smith, W. E., Graham, D., McHugh, C., & Cooper, J. M. (2002). SERRS. In situ substrate formation and improved detection using microfluidics. Analytical chemistry, 74(7), 1503-8. https://doi.org/10.1021/ac015625+
Keir, Ruth, et al. "SERRS. In situ substrate formation and improved detection using microfluidics." Analytical chemistry vol. 74,7 (2002): 1503-8. doi: https://doi.org/10.1021/ac015625+
Keir R, Igata E, Arundell M, Smith WE, Graham D, McHugh C, Cooper JM. SERRS. In situ substrate formation and improved detection using microfluidics. Anal Chem. 2002 Apr 01;74(7):1503-8. doi: 10.1021/ac015625+. PMID: 12033237.
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An experimental study of membrane materials and inner contacting layers for ion-selective K+ electrodes with a stable response and good dynamic range.

Analytical chemistry

Michalska AJ, Appaih-Kusi C, Heng LY, Walkiewicz S, Hall EA.
PMID: 15053668
Anal Chem. 2004 Apr 01;76(7):2031-9. doi: 10.1021/ac0353132.

The goal was to identify formulations for use in valinomycin K(+) ion-selective electrodes that could routinely achieve a detection limit of

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Michalska AJ, Appaih-Kusi C, Heng LY, et al. An experimental study of membrane materials and inner contacting layers for ion-selective K+ electrodes with a stable response and good dynamic range. Anal Chem. 2004;76(7):2031-9doi: 10.1021/ac0353132.
Michalska, A. J., Appaih-Kusi, C., Heng, L. Y., Walkiewicz, S., & Hall, E. A. (2004). An experimental study of membrane materials and inner contacting layers for ion-selective K+ electrodes with a stable response and good dynamic range. Analytical chemistry, 76(7), 2031-9. https://doi.org/10.1021/ac0353132
Michalska, Agata J, et al. "An experimental study of membrane materials and inner contacting layers for ion-selective K+ electrodes with a stable response and good dynamic range." Analytical chemistry vol. 76,7 (2004): 2031-9. doi: https://doi.org/10.1021/ac0353132
Michalska AJ, Appaih-Kusi C, Heng LY, Walkiewicz S, Hall EA. An experimental study of membrane materials and inner contacting layers for ion-selective K+ electrodes with a stable response and good dynamic range. Anal Chem. 2004 Apr 01;76(7):2031-9. doi: 10.1021/ac0353132. PMID: 15053668.
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Increased serum interleukin-18 in a patient with systemic lupus erythematosus and T-cell large granular lymphocytic leukemia.

Modern rheumatology

Ogata A, Kitano M, Fukamizu M, Hamano T, Sano H.
PMID: 17143689
Mod Rheumatol. 2004;14(3):267-70. doi: 10.1007/s10165-004-0306-5.

Interleukin-18 (IL-18) is a potent cofactor for T-helper (Th-1) cell development and inducer of cytotoxic T lymphocytes (CTL), and is reported to contribute to autoimmune diseases. T-cell large granular lymphocyte (T-LGL) leukemia involves the proliferation of autoreactive CTL that...

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Ogata A, Kitano M, Fukamizu M, et al. Increased serum interleukin-18 in a patient with systemic lupus erythematosus and T-cell large granular lymphocytic leukemia. Mod Rheumatol. 2004;14(3):267-70doi: 10.1007/s10165-004-0306-5.
Ogata, A., Kitano, M., Fukamizu, M., Hamano, T., & Sano, H. (2004). Increased serum interleukin-18 in a patient with systemic lupus erythematosus and T-cell large granular lymphocytic leukemia. Modern rheumatology, 14(3), 267-70. https://doi.org/10.1007/s10165-004-0306-5
Ogata, Atsushi, et al. "Increased serum interleukin-18 in a patient with systemic lupus erythematosus and T-cell large granular lymphocytic leukemia." Modern rheumatology vol. 14,3 (2004): 267-70. doi: https://doi.org/10.1007/s10165-004-0306-5
Ogata A, Kitano M, Fukamizu M, Hamano T, Sano H. Increased serum interleukin-18 in a patient with systemic lupus erythematosus and T-cell large granular lymphocytic leukemia. Mod Rheumatol. 2004;14(3):267-70. doi: 10.1007/s10165-004-0306-5. PMID: 17143689.
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