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Potential of bone marrow stromal cells in applications for neuro-degenerative, neuro-traumatic and muscle degenerative diseases.

Current neuropharmacology

Dezawa M, Ishikawa H, Hoshino M, Itokazu Y, Nabeshima Y.
PMID: 18369401
Curr Neuropharmacol. 2005 Oct;3(4):257-66. doi: 10.2174/157015905774322507.

Cell transplantation is a promising strategy for the treatment of neurodegenerative and muscle degenerative diseases. Many kinds of cells, including embryonic stem cells and tissue stem cells, have been considered as candidates for transplantation therapy. Bone marrow stromal cells...

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Dezawa M, Ishikawa H, Hoshino M, et al. Potential of bone marrow stromal cells in applications for neuro-degenerative, neuro-traumatic and muscle degenerative diseases. Curr Neuropharmacol. 2005;3(4):257-66doi: 10.2174/157015905774322507.
Dezawa, M., Ishikawa, H., Hoshino, M., Itokazu, Y., & Nabeshima, Y. (2005). Potential of bone marrow stromal cells in applications for neuro-degenerative, neuro-traumatic and muscle degenerative diseases. Current neuropharmacology, 3(4), 257-66. https://doi.org/10.2174/157015905774322507
Dezawa, Mari, et al. "Potential of bone marrow stromal cells in applications for neuro-degenerative, neuro-traumatic and muscle degenerative diseases." Current neuropharmacology vol. 3,4 (2005): 257-66. doi: https://doi.org/10.2174/157015905774322507
Dezawa M, Ishikawa H, Hoshino M, Itokazu Y, Nabeshima Y. Potential of bone marrow stromal cells in applications for neuro-degenerative, neuro-traumatic and muscle degenerative diseases. Curr Neuropharmacol. 2005 Oct;3(4):257-66. doi: 10.2174/157015905774322507. PMID: 18369401; PMCID: PMC2268998.
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Characteristics of the joint position sense in children with developmental dyslexia.

Journal of physical therapy science

Kimura K, Itokazu M, Otabe N.
PMID: 33814710
J Phys Ther Sci. 2021 Mar;33(3):236-240. doi: 10.1589/jpts.33.236. Epub 2021 Mar 17.

[Purpose] Developmental dyslexia is a disorder in which reading and writing of characters is difficult. The present study investigated age-dependent joint position sense of the forearm and wrist and whether children with developmental dyslexia have less joint position sense...

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Kimura K, Itokazu M, Otabe N. Characteristics of the joint position sense in children with developmental dyslexia. J Phys Ther Sci. 2021;33(3):236-240doi: 10.1589/jpts.33.236.
Kimura, K., Itokazu, M., Otabe, N. (2021). Characteristics of the joint position sense in children with developmental dyslexia. Journal of physical therapy science, 33(3), 236-240. https://doi.org/10.1589/jpts.33.236
Kimura, Kazuya, et al. "Characteristics of the joint position sense in children with developmental dyslexia." Journal of physical therapy science vol. 33,3 (2021): 236-240. doi: https://doi.org/10.1589/jpts.33.236
Kimura K, Itokazu M, Otabe N. Characteristics of the joint position sense in children with developmental dyslexia. J Phys Ther Sci. 2021 Mar;33(3):236-240. doi: 10.1589/jpts.33.236. Epub 2021 Mar 17. PMID: 33814710; PMCID: PMC8012183.
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Interleukin-17A regulates ependymal cell proliferation and functional recovery after spinal cord injury in mice.

Cell death & disease

Miyajima H, Itokazu T, Tanabe S, Yamashita T.
PMID: 34344859
Cell Death Dis. 2021 Aug 03;12(8):766. doi: 10.1038/s41419-021-04064-1.

Ependymal cells have been suggested to act as neural stem cells and exert beneficial effects after spinal cord injury (SCI). However, the molecular mechanism underlying ependymal cell regulation after SCI remains unknown. To examine the possible effect of IL-17A...

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Miyajima H, Itokazu T, Tanabe S, et al. Interleukin-17A regulates ependymal cell proliferation and functional recovery after spinal cord injury in mice. Cell Death Dis. 2021;12(8):766doi: 10.1038/s41419-021-04064-1.
Miyajima, H., Itokazu, T., Tanabe, S., & Yamashita, T. (2021). Interleukin-17A regulates ependymal cell proliferation and functional recovery after spinal cord injury in mice. Cell death & disease, 12(8), 766. https://doi.org/10.1038/s41419-021-04064-1
Miyajima, Hisao, et al. "Interleukin-17A regulates ependymal cell proliferation and functional recovery after spinal cord injury in mice." Cell death & disease vol. 12,8 (2021): 766. doi: https://doi.org/10.1038/s41419-021-04064-1
Miyajima H, Itokazu T, Tanabe S, Yamashita T. Interleukin-17A regulates ependymal cell proliferation and functional recovery after spinal cord injury in mice. Cell Death Dis. 2021 Aug 03;12(8):766. doi: 10.1038/s41419-021-04064-1. PMID: 34344859; PMCID: PMC8333070.
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Pharmacologically induced N-methyl-D-aspartate receptor hypofunction impairs goal-directed food seeking in rats.

Neuropsychopharmacology reports

Ozawa T, Itokazu T, Ichitani Y, Yamada K.
PMID: 34542935
Neuropsychopharmacol Rep. 2021 Dec;41(4):526-531. doi: 10.1002/npr2.12209. Epub 2021 Sep 20.

AIM: Acute N-methyl-D-aspartate (NMDA) receptor antagonism is an important pharmacological animal model of schizophrenia. In previous studies, schizophrenia patients show impaired goal-directed behavior in an outcome-specific devaluation procedure. In this study, we investigated whether the rat model of the...

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Ozawa T, Itokazu T, Ichitani Y, et al. Pharmacologically induced N-methyl-D-aspartate receptor hypofunction impairs goal-directed food seeking in rats. Neuropsychopharmacol Rep. 2021;41(4):526-531doi: 10.1002/npr2.12209.
Ozawa, T., Itokazu, T., Ichitani, Y., & Yamada, K. (2021). Pharmacologically induced N-methyl-D-aspartate receptor hypofunction impairs goal-directed food seeking in rats. Neuropsychopharmacology reports, 41(4), 526-531. https://doi.org/10.1002/npr2.12209
Ozawa, Takaaki, et al. "Pharmacologically induced N-methyl-D-aspartate receptor hypofunction impairs goal-directed food seeking in rats." Neuropsychopharmacology reports vol. 41,4 (2021): 526-531. doi: https://doi.org/10.1002/npr2.12209
Ozawa T, Itokazu T, Ichitani Y, Yamada K. Pharmacologically induced N-methyl-D-aspartate receptor hypofunction impairs goal-directed food seeking in rats. Neuropsychopharmacol Rep. 2021 Dec;41(4):526-531. doi: 10.1002/npr2.12209. Epub 2021 Sep 20. PMID: 34542935; PMCID: PMC8698676.
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Haplotype Explorer: an infection cluster visualization tool for spatiotemporal dissection of the COVID-19 pandemic.

G3 (Bethesda, Md.)

Kawano-Sugaya T, Yatsu K, Sekizuka T, Itokawa K, Hashino M, Tanaka R, Kuroda M.
PMID: 33892501
G3 (Bethesda). 2021 Aug 07;11(8). doi: 10.1093/g3journal/jkab126.

The worldwide eruption of coronavirus disease 2019 (COVID-19) that began in Wuhan, China in late 2019 reached 10 million cases by late June 2020. In order to understand the epidemiological landscape of the COVID-19 pandemic, many studies have attempted...

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Kawano-Sugaya T, Yatsu K, Sekizuka T, et al. Haplotype Explorer: an infection cluster visualization tool for spatiotemporal dissection of the COVID-19 pandemic. G3 (Bethesda). 2021;11(8)doi: 10.1093/g3journal/jkab126.
Kawano-Sugaya, T., Yatsu, K., Sekizuka, T., Itokawa, K., Hashino, M., Tanaka, R., & Kuroda, M. (2021). Haplotype Explorer: an infection cluster visualization tool for spatiotemporal dissection of the COVID-19 pandemic. G3 (Bethesda, Md.), 11(8), . https://doi.org/10.1093/g3journal/jkab126
Kawano-Sugaya, Tetsuro, et al. "Haplotype Explorer: an infection cluster visualization tool for spatiotemporal dissection of the COVID-19 pandemic." G3 (Bethesda, Md.) vol. 11,8 (2021). doi: https://doi.org/10.1093/g3journal/jkab126
Kawano-Sugaya T, Yatsu K, Sekizuka T, Itokawa K, Hashino M, Tanaka R, Kuroda M. Haplotype Explorer: an infection cluster visualization tool for spatiotemporal dissection of the COVID-19 pandemic. G3 (Bethesda). 2021 Aug 07;11(8). doi: 10.1093/g3journal/jkab126. PMID: 33892501; PMCID: PMC8135534.
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Effect of ankle-foot orthosis with a built-in spring on muscle activity during the sit-to-stand movement in healthy individuals.

Journal of physical therapy science

Itokazu M.
PMID: 30936647
J Phys Ther Sci. 2019 Mar;31(3):287-290. doi: 10.1589/jpts.31.287. Epub 2019 Mar 19.

[Purpose] This study aimed to evaluate the influence of a newly developed ankle-foot orthosis with a built-in spring on the activity of lower limb muscles during the sit-to-stand movement. [Participants and Methods] This cross-sectional study recruited 20 male volunteers....

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Itokazu M. Effect of ankle-foot orthosis with a built-in spring on muscle activity during the sit-to-stand movement in healthy individuals. J Phys Ther Sci. 2019;31(3):287-290doi: 10.1589/jpts.31.287.
Itokazu, M. (2019). Effect of ankle-foot orthosis with a built-in spring on muscle activity during the sit-to-stand movement in healthy individuals. Journal of physical therapy science, 31(3), 287-290. https://doi.org/10.1589/jpts.31.287
Itokazu, Masafumi. "Effect of ankle-foot orthosis with a built-in spring on muscle activity during the sit-to-stand movement in healthy individuals." Journal of physical therapy science vol. 31,3 (2019): 287-290. doi: https://doi.org/10.1589/jpts.31.287
Itokazu M. Effect of ankle-foot orthosis with a built-in spring on muscle activity during the sit-to-stand movement in healthy individuals. J Phys Ther Sci. 2019 Mar;31(3):287-290. doi: 10.1589/jpts.31.287. Epub 2019 Mar 19. PMID: 30936647; PMCID: PMC6428643.
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Transplantation of Scaffold-Free Cartilage-Like Cell-Sheets Made from Human Bone Marrow Mesenchymal Stem Cells for Cartilage Repair: A Preclinical Study.

Cartilage

Itokazu M, Wakitani S, Mera H, Tamamura Y, Sato Y, Takagi M, Nakamura H.
PMID: 27688844
Cartilage. 2016 Oct;7(4):361-72. doi: 10.1177/1947603515627342. Epub 2016 Feb 09.

OBJECTIVE: The object of this study was to determine culture conditions that create stable scaffold-free cartilage-like cell-sheets from human bone marrow-derived mesenchymal stem cells (hBMSCs) and to assess their effects after transplantation into osteochondral defects in nude rats.DESIGN: (Experiment...

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Itokazu M, Wakitani S, Mera H, et al. Transplantation of Scaffold-Free Cartilage-Like Cell-Sheets Made from Human Bone Marrow Mesenchymal Stem Cells for Cartilage Repair: A Preclinical Study. Cartilage. 2016;7(4):361-72doi: 10.1177/1947603515627342.
Itokazu, M., Wakitani, S., Mera, H., Tamamura, Y., Sato, Y., Takagi, M., & Nakamura, H. (2016). Transplantation of Scaffold-Free Cartilage-Like Cell-Sheets Made from Human Bone Marrow Mesenchymal Stem Cells for Cartilage Repair: A Preclinical Study. Cartilage, 7(4), 361-72. https://doi.org/10.1177/1947603515627342
Itokazu, Maki, et al. "Transplantation of Scaffold-Free Cartilage-Like Cell-Sheets Made from Human Bone Marrow Mesenchymal Stem Cells for Cartilage Repair: A Preclinical Study." Cartilage vol. 7,4 (2016): 361-72. doi: https://doi.org/10.1177/1947603515627342
Itokazu M, Wakitani S, Mera H, Tamamura Y, Sato Y, Takagi M, Nakamura H. Transplantation of Scaffold-Free Cartilage-Like Cell-Sheets Made from Human Bone Marrow Mesenchymal Stem Cells for Cartilage Repair: A Preclinical Study. Cartilage. 2016 Oct;7(4):361-72. doi: 10.1177/1947603515627342. Epub 2016 Feb 09. PMID: 27688844; PMCID: PMC5029565.
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Arid5a Promotes Immune Evasion by Augmenting Tryptophan Metabolism and Chemokine Expression.

Cancer immunology research

Parajuli G, Tekguc M, Wing JB, Hashimoto A, Okuzaki D, Hirata T, Sasaki A, Itokazu T, Handa H, Sugino H, Nishikawa Y, Metwally H, Kodama Y, Tanaka S, Sabe H, Yamashita T, Sakaguchi S, Kishimoto T, Hashimoto S.
PMID: 34006522
Cancer Immunol Res. 2021 Aug;9(8):862-876. doi: 10.1158/2326-6066.CIR-21-0014. Epub 2021 May 18.

The acquisition of mesenchymal traits leads to immune evasion in various cancers, but the underlying molecular mechanisms remain unclear. In this study, we found that the expression levels of AT-rich interaction domain-containing protein 5a (Arid5a), an RNA-binding protein, were...

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Parajuli G, Tekguc M, Wing JB, et al. Arid5a Promotes Immune Evasion by Augmenting Tryptophan Metabolism and Chemokine Expression. Cancer Immunol Res. 2021;9(8):862-876doi: 10.1158/2326-6066.CIR-21-0014.
Parajuli, G., Tekguc, M., Wing, J. B., Hashimoto, A., Okuzaki, D., Hirata, T., Sasaki, A., Itokazu, T., Handa, H., Sugino, H., Nishikawa, Y., Metwally, H., Kodama, Y., Tanaka, S., Sabe, H., Yamashita, T., Sakaguchi, S., Kishimoto, T., & Hashimoto, S. (2021). Arid5a Promotes Immune Evasion by Augmenting Tryptophan Metabolism and Chemokine Expression. Cancer immunology research, 9(8), 862-876. https://doi.org/10.1158/2326-6066.CIR-21-0014
Parajuli, Gyanu, et al. "Arid5a Promotes Immune Evasion by Augmenting Tryptophan Metabolism and Chemokine Expression." Cancer immunology research vol. 9,8 (2021): 862-876. doi: https://doi.org/10.1158/2326-6066.CIR-21-0014
Parajuli G, Tekguc M, Wing JB, Hashimoto A, Okuzaki D, Hirata T, Sasaki A, Itokazu T, Handa H, Sugino H, Nishikawa Y, Metwally H, Kodama Y, Tanaka S, Sabe H, Yamashita T, Sakaguchi S, Kishimoto T, Hashimoto S. Arid5a Promotes Immune Evasion by Augmenting Tryptophan Metabolism and Chemokine Expression. Cancer Immunol Res. 2021 Aug;9(8):862-876. doi: 10.1158/2326-6066.CIR-21-0014. Epub 2021 May 18. PMID: 34006522.
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Kushiyaki-related .

Clinical case reports

Kurihara M, Nasu M, Itokazu D, Tokuda Y.
PMID: 34026150
Clin Case Rep. 2021 May 19;9(5):e04108. doi: 10.1002/ccr3.4108. eCollection 2021 May.

No abstract available.

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Kurihara M, Nasu M, Itokazu D, et al. Kushiyaki-related . Clin Case Rep. 2021;9(5):e04108doi: 10.1002/ccr3.4108.
Kurihara, M., Nasu, M., Itokazu, D., & Tokuda, Y. (2021). Kushiyaki-related . Clinical case reports, 9(5), e04108. https://doi.org/10.1002/ccr3.4108
Kurihara, Masaru, et al. "Kushiyaki-related ." Clinical case reports vol. 9,5 (2021): e04108. doi: https://doi.org/10.1002/ccr3.4108
Kurihara M, Nasu M, Itokazu D, Tokuda Y. Kushiyaki-related . Clin Case Rep. 2021 May 19;9(5):e04108. doi: 10.1002/ccr3.4108. eCollection 2021 May. PMID: 34026150; PMCID: PMC8133079.
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Pharmacologically induced N-methyl-D-aspartate receptor hypofunction impairs goal-directed food seeking in rats.

Neuropsychopharmacology reports

Ozawa T, Itokazu T, Ichitani Y, Yamada K.
PMID: 34542935
Neuropsychopharmacol Rep. 2021 Dec;41(4):526-531. doi: 10.1002/npr2.12209. Epub 2021 Sep 20.

AIM: Acute N-methyl-D-aspartate (NMDA) receptor antagonism is an important pharmacological animal model of schizophrenia. In previous studies, schizophrenia patients show impaired goal-directed behavior in an outcome-specific devaluation procedure. In this study, we investigated whether the rat model of the...

Cite
Ozawa T, Itokazu T, Ichitani Y, et al. Pharmacologically induced N-methyl-D-aspartate receptor hypofunction impairs goal-directed food seeking in rats. Neuropsychopharmacol Rep. 2021;41(4):526-531doi: 10.1002/npr2.12209.
Ozawa, T., Itokazu, T., Ichitani, Y., & Yamada, K. (2021). Pharmacologically induced N-methyl-D-aspartate receptor hypofunction impairs goal-directed food seeking in rats. Neuropsychopharmacology reports, 41(4), 526-531. https://doi.org/10.1002/npr2.12209
Ozawa, Takaaki, et al. "Pharmacologically induced N-methyl-D-aspartate receptor hypofunction impairs goal-directed food seeking in rats." Neuropsychopharmacology reports vol. 41,4 (2021): 526-531. doi: https://doi.org/10.1002/npr2.12209
Ozawa T, Itokazu T, Ichitani Y, Yamada K. Pharmacologically induced N-methyl-D-aspartate receptor hypofunction impairs goal-directed food seeking in rats. Neuropsychopharmacol Rep. 2021 Dec;41(4):526-531. doi: 10.1002/npr2.12209. Epub 2021 Sep 20. PMID: 34542935.
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Intranasal infusion of GD3 and GM1 gangliosides downregulates alpha-synuclein and controls tyrosine hydroxylase gene in a PD model mouse.

Molecular therapy : the journal of the American Society of Gene Therapy

Itokazu Y, Fuchigami T, Morgan JC, Yu RK.
PMID: 34111562
Mol Ther. 2021 Oct 06;29(10):3059-3071. doi: 10.1016/j.ymthe.2021.06.005. Epub 2021 Jun 08.

Parkinson's disease (PD) is characterized by Lewy bodies (composed predominantly of alpha-synuclein [aSyn]) and loss of pigmented midbrain dopaminergic neurons comprising the nigrostriatal pathway. Most PD patients show significant deficiency of gangliosides, including GM1, in the brain, and GM1...

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Itokazu Y, Fuchigami T, Morgan JC, et al. Intranasal infusion of GD3 and GM1 gangliosides downregulates alpha-synuclein and controls tyrosine hydroxylase gene in a PD model mouse. Mol Ther. 2021;29(10):3059-3071doi: 10.1016/j.ymthe.2021.06.005.
Itokazu, Y., Fuchigami, T., Morgan, J. C., & Yu, R. K. (2021). Intranasal infusion of GD3 and GM1 gangliosides downregulates alpha-synuclein and controls tyrosine hydroxylase gene in a PD model mouse. Molecular therapy : the journal of the American Society of Gene Therapy, 29(10), 3059-3071. https://doi.org/10.1016/j.ymthe.2021.06.005
Itokazu, Yutaka, et al. "Intranasal infusion of GD3 and GM1 gangliosides downregulates alpha-synuclein and controls tyrosine hydroxylase gene in a PD model mouse." Molecular therapy : the journal of the American Society of Gene Therapy vol. 29,10 (2021): 3059-3071. doi: https://doi.org/10.1016/j.ymthe.2021.06.005
Itokazu Y, Fuchigami T, Morgan JC, Yu RK. Intranasal infusion of GD3 and GM1 gangliosides downregulates alpha-synuclein and controls tyrosine hydroxylase gene in a PD model mouse. Mol Ther. 2021 Oct 06;29(10):3059-3071. doi: 10.1016/j.ymthe.2021.06.005. Epub 2021 Jun 08. PMID: 34111562; PMCID: PMC8531229.
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Complement cascade functions during brain development and neurodegeneration.

The FEBS journal

Fatoba O, Itokazu T, Yamashita T.
PMID: 33599083
FEBS J. 2021 Feb 18; doi: 10.1111/febs.15772. Epub 2021 Feb 18.

The complement system, an essential tightly regulated innate immune system, is a key regulator of normal central nervous system (CNS) development and function. However, aberrant complement component expression and activation in the brain may culminate into marked neuroinflammatory response,...

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Fatoba O, Itokazu T, Yamashita T. Complement cascade functions during brain development and neurodegeneration. FEBS J. 2021;doi: 10.1111/febs.15772.
Fatoba, O., Itokazu, T., & Yamashita, T. (2021). Complement cascade functions during brain development and neurodegeneration. The FEBS journal, . https://doi.org/10.1111/febs.15772
Fatoba, Oluwaseun, et al. "Complement cascade functions during brain development and neurodegeneration." The FEBS journal vol. (2021). doi: https://doi.org/10.1111/febs.15772
Fatoba O, Itokazu T, Yamashita T. Complement cascade functions during brain development and neurodegeneration. FEBS J. 2021 Feb 18; doi: 10.1111/febs.15772. Epub 2021 Feb 18. PMID: 33599083.
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Showing 1 to 12 of 31 entries