Advanced Search
Display options
Filter resources
Text Availability
Article type
Publication date
Species
Language
Sex
Age
Showing 1 to 12 of 27 entries
Sorted by: Best Match Show Resources per page
Campylobacter concisus pathotypes induce distinct global responses in intestinal epithelial cells.

Scientific reports

Deshpande NP, Wilkins MR, Castaño-Rodríguez N, Bainbridge E, Sodhi N, Riordan SM, Mitchell HM, Kaakoush NO.
PMID: 27677841
Sci Rep. 2016 Sep 28;6:34288. doi: 10.1038/srep34288.

The epithelial response to the opportunistic pathogen Campylobacter concisus is poorly characterised. Here, we assessed the intestinal epithelial responses to two C. concisus strains with different virulence characteristics in Caco-2 cells using RNAseq, and validated a subset of the...

Cite
Deshpande NP, Wilkins MR, Castaño-Rodríguez N, et al. Campylobacter concisus pathotypes induce distinct global responses in intestinal epithelial cells. Sci Rep. 2016;6:34288doi: 10.1038/srep34288.
Deshpande, N. P., Wilkins, M. R., Castaño-Rodríguez, N., Bainbridge, E., Sodhi, N., Riordan, S. M., Mitchell, H. M., & Kaakoush, N. O. (2016). Campylobacter concisus pathotypes induce distinct global responses in intestinal epithelial cells. Scientific reports, 634288. https://doi.org/10.1038/srep34288
Deshpande, Nandan P, et al. "Campylobacter concisus pathotypes induce distinct global responses in intestinal epithelial cells." Scientific reports vol. 6 (2016): 34288. doi: https://doi.org/10.1038/srep34288
Deshpande NP, Wilkins MR, Castaño-Rodríguez N, Bainbridge E, Sodhi N, Riordan SM, Mitchell HM, Kaakoush NO. Campylobacter concisus pathotypes induce distinct global responses in intestinal epithelial cells. Sci Rep. 2016 Sep 28;6:34288. doi: 10.1038/srep34288. PMID: 27677841; PMCID: PMC5039708.
Copy Download .nbib Format: NLM
The prevalence of the duodenal ulcer promoting gene (dupA) in Helicobacter pylori isolates varies by ethnic group and is not universally associated with disease development: a case-control study.

Gut pathogens

Schmidt HM, Andres S, Kaakoush NO, Engstrand L, Eriksson L, Goh KL, Fock KM, Hilmi I, Dhamodaran S, Forman D, Mitchell H.
PMID: 19338650
Gut Pathog. 2009 Mar 11;1(1):5. doi: 10.1186/1757-4749-1-5.

BACKGROUND: The putative H. pylori pathogenicity-associated factor dupA has been associated with IL-8 induction in vitro, and duodenal ulcer (DU) and gastric cancer (GC) development in certain populations, but this association is inconsistent between studies. We aimed to investigate...

Cite
Schmidt HM, Andres S, Kaakoush NO, et al. The prevalence of the duodenal ulcer promoting gene (dupA) in Helicobacter pylori isolates varies by ethnic group and is not universally associated with disease development: a case-control study. Gut Pathog. 2009;1(1):5doi: 10.1186/1757-4749-1-5.
Schmidt, H. M., Andres, S., Kaakoush, N. O., Engstrand, L., Eriksson, L., Goh, K. L., Fock, K. M., Hilmi, I., Dhamodaran, S., Forman, D., & Mitchell, H. (2009). The prevalence of the duodenal ulcer promoting gene (dupA) in Helicobacter pylori isolates varies by ethnic group and is not universally associated with disease development: a case-control study. Gut pathogens, 1(1), 5. https://doi.org/10.1186/1757-4749-1-5
Schmidt, Heather-Marie A, et al. "The prevalence of the duodenal ulcer promoting gene (dupA) in Helicobacter pylori isolates varies by ethnic group and is not universally associated with disease development: a case-control study." Gut pathogens vol. 1,1 (2009): 5. doi: https://doi.org/10.1186/1757-4749-1-5
Schmidt HM, Andres S, Kaakoush NO, Engstrand L, Eriksson L, Goh KL, Fock KM, Hilmi I, Dhamodaran S, Forman D, Mitchell H. The prevalence of the duodenal ulcer promoting gene (dupA) in Helicobacter pylori isolates varies by ethnic group and is not universally associated with disease development: a case-control study. Gut Pathog. 2009 Mar 11;1(1):5. doi: 10.1186/1757-4749-1-5. PMID: 19338650; PMCID: PMC2667403.
Copy Download .nbib Format: NLM
Lyophilised oral faecal microbiota transplantation for ulcerative colitis (LOTUS): a randomised, double-blind, placebo-controlled trial.

The lancet. Gastroenterology & hepatology

Haifer C, Paramsothy S, Kaakoush NO, Saikal A, Ghaly S, Yang T, Luu LDW, Borody TJ, Leong RW.
PMID: 34863330
Lancet Gastroenterol Hepatol. 2022 Feb;7(2):141-151. doi: 10.1016/S2468-1253(21)00400-3. Epub 2021 Dec 02.

BACKGROUND: Faecal microbiota transplantation (FMT) delivered via colonoscopic infusion or enemas have been shown to induce remission in a proportion of patients with active ulcerative colitis. Whether orally administered FMT is effective in ulcerative colitis is unknown. We aimed...

Cite
Haifer C, Paramsothy S, Kaakoush NO, et al. Lyophilised oral faecal microbiota transplantation for ulcerative colitis (LOTUS): a randomised, double-blind, placebo-controlled trial. Lancet Gastroenterol Hepatol. 2021;7(2):141-151doi: 10.1016/S2468-1253(21)00400-3.
Haifer, C., Paramsothy, S., Kaakoush, N. O., Saikal, A., Ghaly, S., Yang, T., Luu, L. D. W., Borody, T. J., & Leong, R. W. (2022). Lyophilised oral faecal microbiota transplantation for ulcerative colitis (LOTUS): a randomised, double-blind, placebo-controlled trial. The lancet. Gastroenterology & hepatology, 7(2), 141-151. https://doi.org/10.1016/S2468-1253(21)00400-3
Haifer, Craig, et al. "Lyophilised oral faecal microbiota transplantation for ulcerative colitis (LOTUS): a randomised, double-blind, placebo-controlled trial." The lancet. Gastroenterology & hepatology vol. 7,2 (2022): 141-151. doi: https://doi.org/10.1016/S2468-1253(21)00400-3
Haifer C, Paramsothy S, Kaakoush NO, Saikal A, Ghaly S, Yang T, Luu LDW, Borody TJ, Leong RW. Lyophilised oral faecal microbiota transplantation for ulcerative colitis (LOTUS): a randomised, double-blind, placebo-controlled trial. Lancet Gastroenterol Hepatol. 2022 Feb;7(2):141-151. doi: 10.1016/S2468-1253(21)00400-3. Epub 2021 Dec 02. PMID: 34863330.
Copy Download .nbib Format: NLM
Genetic variants involved in innate immunity modulate the risk of inflammatory bowel diseases in an understudied Malaysian population.

Journal of gastroenterology and hepatology

Luu LDW, Popple G, Tsang SPW, Vinasco K, Hilmi I, Ng RT, Chew KS, Wong SY, Riordan S, Lee WS, Mitchell HM, Kaakoush NO, Castaño-Rodríguez N.
PMID: 34888949
J Gastroenterol Hepatol. 2021 Dec 09; doi: 10.1111/jgh.15752. Epub 2021 Dec 09.

BACKGROUND AND AIM: Inflammatory bowel diseases (IBD) are chronic gastrointestinal inflammatory conditions comprising two major subtypes: Crohn's disease (CD) and ulcerative colitis (UC). The incidence of IBD is increasing in Asian countries including Malaysia. The aim of this study...

Cite
Luu LDW, Popple G, Tsang SPW, et al. Genetic variants involved in innate immunity modulate the risk of inflammatory bowel diseases in an understudied Malaysian population. J Gastroenterol Hepatol. 2021;doi: 10.1111/jgh.15752.
Luu, L. D. W., Popple, G., Tsang, S. P. W., Vinasco, K., Hilmi, I., Ng, R. T., Chew, K. S., Wong, S. Y., Riordan, S., Lee, W. S., Mitchell, H. M., Kaakoush, N. O., & Castaño-Rodríguez, N. (2021). Genetic variants involved in innate immunity modulate the risk of inflammatory bowel diseases in an understudied Malaysian population. Journal of gastroenterology and hepatology, . https://doi.org/10.1111/jgh.15752
Luu, Laurence Don Wai, et al. "Genetic variants involved in innate immunity modulate the risk of inflammatory bowel diseases in an understudied Malaysian population." Journal of gastroenterology and hepatology vol. (2021). doi: https://doi.org/10.1111/jgh.15752
Luu LDW, Popple G, Tsang SPW, Vinasco K, Hilmi I, Ng RT, Chew KS, Wong SY, Riordan S, Lee WS, Mitchell HM, Kaakoush NO, Castaño-Rodríguez N. Genetic variants involved in innate immunity modulate the risk of inflammatory bowel diseases in an understudied Malaysian population. J Gastroenterol Hepatol. 2021 Dec 09; doi: 10.1111/jgh.15752. Epub 2021 Dec 09. PMID: 34888949.
Copy Download .nbib Format: NLM
Genetic variants involved in innate immunity modulate the risk of inflammatory bowel diseases in an understudied Malaysian population.

Journal of gastroenterology and hepatology

Luu LDW, Popple G, Tsang SPW, Vinasco K, Hilmi I, Ng RT, Chew KS, Wong SY, Riordan S, Lee WS, Mitchell HM, Kaakoush NO, Castaño-Rodríguez N.
PMID: 34888949
J Gastroenterol Hepatol. 2021 Dec 09; doi: 10.1111/jgh.15752. Epub 2021 Dec 09.

BACKGROUND AND AIM: Inflammatory bowel diseases (IBD) are chronic gastrointestinal inflammatory conditions comprising two major subtypes: Crohn's disease (CD) and ulcerative colitis (UC). The incidence of IBD is increasing in Asian countries including Malaysia. The aim of this study...

Cite
Luu LDW, Popple G, Tsang SPW, et al. Genetic variants involved in innate immunity modulate the risk of inflammatory bowel diseases in an understudied Malaysian population. J Gastroenterol Hepatol. 2021;doi: 10.1111/jgh.15752.
Luu, L. D. W., Popple, G., Tsang, S. P. W., Vinasco, K., Hilmi, I., Ng, R. T., Chew, K. S., Wong, S. Y., Riordan, S., Lee, W. S., Mitchell, H. M., Kaakoush, N. O., & Castaño-Rodríguez, N. (2021). Genetic variants involved in innate immunity modulate the risk of inflammatory bowel diseases in an understudied Malaysian population. Journal of gastroenterology and hepatology, . https://doi.org/10.1111/jgh.15752
Luu, Laurence Don Wai, et al. "Genetic variants involved in innate immunity modulate the risk of inflammatory bowel diseases in an understudied Malaysian population." Journal of gastroenterology and hepatology vol. (2021). doi: https://doi.org/10.1111/jgh.15752
Luu LDW, Popple G, Tsang SPW, Vinasco K, Hilmi I, Ng RT, Chew KS, Wong SY, Riordan S, Lee WS, Mitchell HM, Kaakoush NO, Castaño-Rodríguez N. Genetic variants involved in innate immunity modulate the risk of inflammatory bowel diseases in an understudied Malaysian population. J Gastroenterol Hepatol. 2021 Dec 09; doi: 10.1111/jgh.15752. Epub 2021 Dec 09. PMID: 34888949.
Copy Download .nbib Format: NLM
Genome Sequence of Campylobacter showae UNSWCD, Isolated from a Patient with Crohn's Disease.

Genome announcements

Tay AP, Kaakoush NO, Deshpande NP, Chen Z, Mitchell H, Wilkins MR.
PMID: 23409253
Genome Announc. 2013 Jan;1(1). doi: 10.1128/genomeA.00193-12. Epub 2013 Feb 14.

Campylobacter showae UNSWCD was isolated from a patient with Crohn's disease. Here we present a 2.1 Mb draft assembly of its genome.

Cite
Tay AP, Kaakoush NO, Deshpande NP, et al. Genome Sequence of Campylobacter showae UNSWCD, Isolated from a Patient with Crohn's Disease. Genome Announc. 2013;1(1)doi: 10.1128/genomeA.00193-12.
Tay, A. P., Kaakoush, N. O., Deshpande, N. P., Chen, Z., Mitchell, H., & Wilkins, M. R. (2013). Genome Sequence of Campylobacter showae UNSWCD, Isolated from a Patient with Crohn's Disease. Genome announcements, 1(1), . https://doi.org/10.1128/genomeA.00193-12
Tay, Aidan P, et al. "Genome Sequence of Campylobacter showae UNSWCD, Isolated from a Patient with Crohn's Disease." Genome announcements vol. 1,1 (2013). doi: https://doi.org/10.1128/genomeA.00193-12
Tay AP, Kaakoush NO, Deshpande NP, Chen Z, Mitchell H, Wilkins MR. Genome Sequence of Campylobacter showae UNSWCD, Isolated from a Patient with Crohn's Disease. Genome Announc. 2013 Jan;1(1). doi: 10.1128/genomeA.00193-12. Epub 2013 Feb 14. PMID: 23409253; PMCID: PMC3569271.
Copy Download .nbib Format: NLM
Pattern-recognition receptors and gastric cancer.

Frontiers in immunology

Castaño-Rodríguez N, Kaakoush NO, Mitchell HM.
PMID: 25101079
Front Immunol. 2014 Jul 22;5:336. doi: 10.3389/fimmu.2014.00336. eCollection 2014.

Chronic inflammation has been associated with an increased risk of several human malignancies, a classic example being gastric adenocarcinoma (GC). Development of GC is known to result from infection of the gastric mucosa by Helicobacter pylori, which initially induces...

Cite
Castaño-Rodríguez N, Kaakoush NO, Mitchell HM. Pattern-recognition receptors and gastric cancer. Front Immunol. 2014;5:336doi: 10.3389/fimmu.2014.00336.
Castaño-Rodríguez, N., Kaakoush, N. O., & Mitchell, H. M. (2014). Pattern-recognition receptors and gastric cancer. Frontiers in immunology, 5336. https://doi.org/10.3389/fimmu.2014.00336
Castaño-Rodríguez, Natalia, et al. "Pattern-recognition receptors and gastric cancer." Frontiers in immunology vol. 5 (2014): 336. doi: https://doi.org/10.3389/fimmu.2014.00336
Castaño-Rodríguez N, Kaakoush NO, Mitchell HM. Pattern-recognition receptors and gastric cancer. Front Immunol. 2014 Jul 22;5:336. doi: 10.3389/fimmu.2014.00336. eCollection 2014. PMID: 25101079; PMCID: PMC4105827.
Copy Download .nbib Format: NLM
Comparative analyses of Campylobacter concisus strains reveal the genome of the reference strain BAA-1457 is not representative of the species.

Gut pathogens

Kaakoush NO, Deshpande NP, Wilkins MR, Raftery MJ, Janitz K, Mitchell H.
PMID: 21992484
Gut Pathog. 2011 Oct 13;3:15. doi: 10.1186/1757-4749-3-15.

BACKGROUND: Several studies have shown that significant genotypic heterogeneity exists among Campylobacter concisus strains. Recently, the genome of C. concisus UNSWCD, isolated from a patient with Crohn's disease, was sequenced.RESULTS: In this study, comparative analyses were performed between strain...

Cite
Kaakoush NO, Deshpande NP, Wilkins MR, et al. Comparative analyses of Campylobacter concisus strains reveal the genome of the reference strain BAA-1457 is not representative of the species. Gut Pathog. 2011;3:15doi: 10.1186/1757-4749-3-15.
Kaakoush, N. O., Deshpande, N. P., Wilkins, M. R., Raftery, M. J., Janitz, K., & Mitchell, H. (2011). Comparative analyses of Campylobacter concisus strains reveal the genome of the reference strain BAA-1457 is not representative of the species. Gut pathogens, 315. https://doi.org/10.1186/1757-4749-3-15
Kaakoush, Nadeem O, et al. "Comparative analyses of Campylobacter concisus strains reveal the genome of the reference strain BAA-1457 is not representative of the species." Gut pathogens vol. 3 (2011): 15. doi: https://doi.org/10.1186/1757-4749-3-15
Kaakoush NO, Deshpande NP, Wilkins MR, Raftery MJ, Janitz K, Mitchell H. Comparative analyses of Campylobacter concisus strains reveal the genome of the reference strain BAA-1457 is not representative of the species. Gut Pathog. 2011 Oct 13;3:15. doi: 10.1186/1757-4749-3-15. PMID: 21992484; PMCID: PMC3219677.
Copy Download .nbib Format: NLM
Reduction in Gut Microbial Diversity as a Mechanism of Action of Exclusive Enteral Nutrition.

The American journal of gastroenterology

Kaakoush NO, Day AS, Leach ST, Lemberg DA, Mitchell HM.
PMID: 27356827
Am J Gastroenterol. 2016 Jul;111(7):1033. doi: 10.1038/ajg.2016.61.

No abstract available.

Cite
Kaakoush NO, Day AS, Leach ST, et al. Reduction in Gut Microbial Diversity as a Mechanism of Action of Exclusive Enteral Nutrition. Am J Gastroenterol. 2016;111(7):1033doi: 10.1038/ajg.2016.61.
Kaakoush, N. O., Day, A. S., Leach, S. T., Lemberg, D. A., & Mitchell, H. M. (2016). Reduction in Gut Microbial Diversity as a Mechanism of Action of Exclusive Enteral Nutrition. The American journal of gastroenterology, 111(7), 1033. https://doi.org/10.1038/ajg.2016.61
Kaakoush, Nadeem O, et al. "Reduction in Gut Microbial Diversity as a Mechanism of Action of Exclusive Enteral Nutrition." The American journal of gastroenterology vol. 111,7 (2016): 1033. doi: https://doi.org/10.1038/ajg.2016.61
Kaakoush NO, Day AS, Leach ST, Lemberg DA, Mitchell HM. Reduction in Gut Microbial Diversity as a Mechanism of Action of Exclusive Enteral Nutrition. Am J Gastroenterol. 2016 Jul;111(7):1033. doi: 10.1038/ajg.2016.61. PMID: 27356827.
Copy Download .nbib Format: NLM
Response to faecal microbiota transplantation in ulcerative colitis is not sustained long term following induction therapy.

Gut

Haifer C, Saikal A, Paramsothy R, Kaakoush NO, Leong RW, Borody TJ, Kamm MA, Paramsothy S.
PMID: 33303563
Gut. 2021 Nov;70(11):2210-2211. doi: 10.1136/gutjnl-2020-323581. Epub 2020 Dec 10.

No abstract available.

Cite
Haifer C, Saikal A, Paramsothy R, et al. Response to faecal microbiota transplantation in ulcerative colitis is not sustained long term following induction therapy. Gut. 2020;70(11):2210-2211doi: 10.1136/gutjnl-2020-323581.
Haifer, C., Saikal, A., Paramsothy, R., Kaakoush, N. O., Leong, R. W., Borody, T. J., Kamm, M. A., & Paramsothy, S. (2021). Response to faecal microbiota transplantation in ulcerative colitis is not sustained long term following induction therapy. Gut, 70(11), 2210-2211. https://doi.org/10.1136/gutjnl-2020-323581
Haifer, Craig, et al. "Response to faecal microbiota transplantation in ulcerative colitis is not sustained long term following induction therapy." Gut vol. 70,11 (2021): 2210-2211. doi: https://doi.org/10.1136/gutjnl-2020-323581
Haifer C, Saikal A, Paramsothy R, Kaakoush NO, Leong RW, Borody TJ, Kamm MA, Paramsothy S. Response to faecal microbiota transplantation in ulcerative colitis is not sustained long term following induction therapy. Gut. 2021 Nov;70(11):2210-2211. doi: 10.1136/gutjnl-2020-323581. Epub 2020 Dec 10. PMID: 33303563.
Copy Download .nbib Format: NLM
Long-Term Bacterial and Fungal Dynamics following Oral Lyophilized Fecal Microbiota Transplantation in Clostridioides difficile Infection.

mSystems

Haifer C, Paramsothy S, Borody TJ, Clancy A, Leong RW, Kaakoush NO.
PMID: 33531405
mSystems. 2021 Feb 02;6(1). doi: 10.1128/mSystems.00905-20.

Oral lyophilized fecal microbiota transplantation (FMT) is effective in recurrent

Cite
Haifer C, Paramsothy S, Borody TJ, et al. Long-Term Bacterial and Fungal Dynamics following Oral Lyophilized Fecal Microbiota Transplantation in Clostridioides difficile Infection. mSystems. 2021;6(1)doi: 10.1128/mSystems.00905-20.
Haifer, C., Paramsothy, S., Borody, T. J., Clancy, A., Leong, R. W., & Kaakoush, N. O. (2021). Long-Term Bacterial and Fungal Dynamics following Oral Lyophilized Fecal Microbiota Transplantation in Clostridioides difficile Infection. mSystems, 6(1), . https://doi.org/10.1128/mSystems.00905-20
Haifer, Craig, et al. "Long-Term Bacterial and Fungal Dynamics following Oral Lyophilized Fecal Microbiota Transplantation in Clostridioides difficile Infection." mSystems vol. 6,1 (2021). doi: https://doi.org/10.1128/mSystems.00905-20
Haifer C, Paramsothy S, Borody TJ, Clancy A, Leong RW, Kaakoush NO. Long-Term Bacterial and Fungal Dynamics following Oral Lyophilized Fecal Microbiota Transplantation in Clostridioides difficile Infection. mSystems. 2021 Feb 02;6(1). doi: 10.1128/mSystems.00905-20. PMID: 33531405; PMCID: PMC7857531.
Copy Download .nbib Format: NLM
Multiple Genome Sequences of Helicobacter pylori Strains of Diverse Disease and Antibiotic Resistance Backgrounds from Malaysia.

Genome announcements

Rehvathy V, Tan MH, Gunaletchumy SP, Teh X, Wang S, Baybayan P, Singh S, Ashby M, Kaakoush NO, Mitchell HM, Croft LJ, Goh KL, Loke MF, Vadivelu J.
PMID: 24051312
Genome Announc. 2013 Sep 19;1(5). doi: 10.1128/genomeA.00687-13.

Helicobacter pylori causes human gastroduodenal diseases, including chronic gastritis and peptic ulcer disease. It is also a major microbial risk factor for the development of gastric adenocarcinoma and mucosa-associated lymphoid tissue (MALT) lymphoma. Twenty-one strains with different ethnicity, disease,...

Cite
Rehvathy V, Tan MH, Gunaletchumy SP, et al. Multiple Genome Sequences of Helicobacter pylori Strains of Diverse Disease and Antibiotic Resistance Backgrounds from Malaysia. Genome Announc. 2013;1(5)doi: 10.1128/genomeA.00687-13.
Rehvathy, V., Tan, M. H., Gunaletchumy, S. P., Teh, X., Wang, S., Baybayan, P., Singh, S., Ashby, M., Kaakoush, N. O., Mitchell, H. M., Croft, L. J., Goh, K. L., Loke, M. F., & Vadivelu, J. (2013). Multiple Genome Sequences of Helicobacter pylori Strains of Diverse Disease and Antibiotic Resistance Backgrounds from Malaysia. Genome announcements, 1(5), . https://doi.org/10.1128/genomeA.00687-13
Rehvathy, Vellaya, et al. "Multiple Genome Sequences of Helicobacter pylori Strains of Diverse Disease and Antibiotic Resistance Backgrounds from Malaysia." Genome announcements vol. 1,5 (2013). doi: https://doi.org/10.1128/genomeA.00687-13
Rehvathy V, Tan MH, Gunaletchumy SP, Teh X, Wang S, Baybayan P, Singh S, Ashby M, Kaakoush NO, Mitchell HM, Croft LJ, Goh KL, Loke MF, Vadivelu J. Multiple Genome Sequences of Helicobacter pylori Strains of Diverse Disease and Antibiotic Resistance Backgrounds from Malaysia. Genome Announc. 2013 Sep 19;1(5). doi: 10.1128/genomeA.00687-13. PMID: 24051312; PMCID: PMC3778195.
Copy Download .nbib Format: NLM
Showing 1 to 12 of 27 entries