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GRIM-19: A Double-edged Sword that Regulates Anti-Tumor and Innate Immune Responses.

Translational oncogenomics

Nallar SC, Kalakonda S, Sun P, Kalvakolanu DV.
PMID: 21566745
Transl Oncogenomics. 2008 Mar 17;3:67-79.

Gene associated with retinoid-interferon-β-induced mortality (GRIM)-19, was originally identified as a critical regulatory protein necessary for Interferon-β-Retinoic acid-induced cell death. Overexpression of GRIM-19 activates cell death and its suppression or inactivation promotes cell growth. GRIM-19 targets multiple proteins/pathways for...

Cite
Nallar SC, Kalakonda S, Sun P, et al. GRIM-19: A Double-edged Sword that Regulates Anti-Tumor and Innate Immune Responses. Transl Oncogenomics. 2008;3:67-79
Nallar, S. C., Kalakonda, S., Sun, P., & Kalvakolanu, D. V. (2008). GRIM-19: A Double-edged Sword that Regulates Anti-Tumor and Innate Immune Responses. Translational oncogenomics, 367-79.
Nallar, Shreeram C, et al. "GRIM-19: A Double-edged Sword that Regulates Anti-Tumor and Innate Immune Responses." Translational oncogenomics vol. 3 (2008): 67-79.
Nallar SC, Kalakonda S, Sun P, Kalvakolanu DV. GRIM-19: A Double-edged Sword that Regulates Anti-Tumor and Innate Immune Responses. Transl Oncogenomics. 2008 Mar 17;3:67-79. PMID: 21566745; PMCID: PMC3022361.
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Corrigendum to "RGFP966, a histone deacetylase 3 inhibitor, promotes glioma stem cell differentiation by blocking TGF-β signaling via SMAD7" [Biochem. Pharmacol. 180 (2020) 114118].

Biochemical pharmacology

Liang H, Wang Q, Wang D, Zheng H, Kalvakolanu DV, Lu H, Wen N, Chen X, Xu L, Ren J, Guo B, Zhang L.
PMID: 33039815
Biochem Pharmacol. 2020 Dec;182:114223. doi: 10.1016/j.bcp.2020.114223. Epub 2020 Oct 08.

No abstract available.

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Liang H, Wang Q, Wang D, et al. Corrigendum to "RGFP966, a histone deacetylase 3 inhibitor, promotes glioma stem cell differentiation by blocking TGF-β signaling via SMAD7" [Biochem. Pharmacol. 180 (2020) 114118]. Biochem Pharmacol. 2020;182:114223doi: 10.1016/j.bcp.2020.114223.
Liang, H., Wang, Q., Wang, D., Zheng, H., Kalvakolanu, D. V., Lu, H., Wen, N., Chen, X., Xu, L., Ren, J., Guo, B., & Zhang, L. (2020). Corrigendum to "RGFP966, a histone deacetylase 3 inhibitor, promotes glioma stem cell differentiation by blocking TGF-β signaling via SMAD7" [Biochem. Pharmacol. 180 (2020) 114118]. Biochemical pharmacology, 182114223. https://doi.org/10.1016/j.bcp.2020.114223
Liang, Hang, et al. "Corrigendum to "RGFP966, a histone deacetylase 3 inhibitor, promotes glioma stem cell differentiation by blocking TGF-β signaling via SMAD7" [Biochem. Pharmacol. 180 (2020) 114118]." Biochemical pharmacology vol. 182 (2020): 114223. doi: https://doi.org/10.1016/j.bcp.2020.114223
Liang H, Wang Q, Wang D, Zheng H, Kalvakolanu DV, Lu H, Wen N, Chen X, Xu L, Ren J, Guo B, Zhang L. Corrigendum to "RGFP966, a histone deacetylase 3 inhibitor, promotes glioma stem cell differentiation by blocking TGF-β signaling via SMAD7" [Biochem. Pharmacol. 180 (2020) 114118]. Biochem Pharmacol. 2020 Dec;182:114223. doi: 10.1016/j.bcp.2020.114223. Epub 2020 Oct 08. PMID: 33039815.
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Tunicamycin induced endoplasmic reticulum stress promotes apoptosis of prostate cancer cells by activating mTORC1.

Oncotarget

Guha P, Kaptan E, Gade P, Kalvakolanu DV, Ahmed H.
PMID: 28978108
Oncotarget. 2017 Jul 15;8(40):68191-68207. doi: 10.18632/oncotarget.19277. eCollection 2017 Sep 15.

Studies suggest that tunicamycin may work as a therapeutic drug to cancer cells by inducing stress in the endoplasmic reticulum (ER) through unfolded protein response (UPR) and thereby promoting apoptosis. However, mechanisms of the prolonged activation of the UPR...

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Guha P, Kaptan E, Gade P, et al. Tunicamycin induced endoplasmic reticulum stress promotes apoptosis of prostate cancer cells by activating mTORC1. Oncotarget. 2017;8(40):68191-68207doi: 10.18632/oncotarget.19277.
Guha, P., Kaptan, E., Gade, P., Kalvakolanu, D. V., & Ahmed, H. (2017). Tunicamycin induced endoplasmic reticulum stress promotes apoptosis of prostate cancer cells by activating mTORC1. Oncotarget, 8(40), 68191-68207. https://doi.org/10.18632/oncotarget.19277
Guha, Prasun, et al. "Tunicamycin induced endoplasmic reticulum stress promotes apoptosis of prostate cancer cells by activating mTORC1." Oncotarget vol. 8,40 (2017): 68191-68207. doi: https://doi.org/10.18632/oncotarget.19277
Guha P, Kaptan E, Gade P, Kalvakolanu DV, Ahmed H. Tunicamycin induced endoplasmic reticulum stress promotes apoptosis of prostate cancer cells by activating mTORC1. Oncotarget. 2017 Jul 15;8(40):68191-68207. doi: 10.18632/oncotarget.19277. eCollection 2017 Sep 15. PMID: 28978108; PMCID: PMC5620248.
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Retraction: Identification and characterization of GRIM-1, a cell-death-associated gene product.

Journal of cell science

Hofmann ER, Nallar SC, Lin L, D'Cunha J, Lindner DJ, Weihua X, Kalvakolanu DV.
PMID: 34817589
J Cell Sci. 2021 Nov 15;134(22). doi: 10.1242/jcs.259484. Epub 2021 Nov 24.

No abstract available.

Cite
Hofmann ER, Nallar SC, Lin L, et al. Retraction: Identification and characterization of GRIM-1, a cell-death-associated gene product. J Cell Sci. 2021;134(22)doi: 10.1242/jcs.259484.
Hofmann, E. R., Nallar, S. C., Lin, L., D'Cunha, J., Lindner, D. J., Weihua, X., & Kalvakolanu, D. V. (2021). Retraction: Identification and characterization of GRIM-1, a cell-death-associated gene product. Journal of cell science, 134(22), . https://doi.org/10.1242/jcs.259484
Hofmann, Edward R, et al. "Retraction: Identification and characterization of GRIM-1, a cell-death-associated gene product." Journal of cell science vol. 134,22 (2021). doi: https://doi.org/10.1242/jcs.259484
Hofmann ER, Nallar SC, Lin L, D'Cunha J, Lindner DJ, Weihua X, Kalvakolanu DV. Retraction: Identification and characterization of GRIM-1, a cell-death-associated gene product. J Cell Sci. 2021 Nov 15;134(22). doi: 10.1242/jcs.259484. Epub 2021 Nov 24. PMID: 34817589.
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