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The growth cones of Aplysia sensory neurons: Modulation by serotonin of action potential duration and single potassium channel currents.

Proceedings of the National Academy of Sciences of the United States of America

Belardetti F, Schacher S, Kandel ER, Siegelbaum SA.
PMID: 16593764
Proc Natl Acad Sci U S A. 1986 Sep;83(18):7094-8. doi: 10.1073/pnas.83.18.7094.

Serotonin (5-HT) closes a specific K channel ("S") in the cell body of Aplysia sensory neurons, resulting in a slow excitatory postsynaptic potential and spike broadening. To determine whether the S channel is present and can be modulated in...

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Belardetti F, Schacher S, Kandel ER, et al. The growth cones of Aplysia sensory neurons: Modulation by serotonin of action potential duration and single potassium channel currents. Proc Natl Acad Sci U S A. 1986;83(18):7094-8doi: 10.1073/pnas.83.18.7094.
Belardetti, F., Schacher, S., Kandel, E. R., & Siegelbaum, S. A. (1986). The growth cones of Aplysia sensory neurons: Modulation by serotonin of action potential duration and single potassium channel currents. Proceedings of the National Academy of Sciences of the United States of America, 83(18), 7094-8. https://doi.org/10.1073/pnas.83.18.7094
Belardetti, F, et al. "The growth cones of Aplysia sensory neurons: Modulation by serotonin of action potential duration and single potassium channel currents." Proceedings of the National Academy of Sciences of the United States of America vol. 83,18 (1986): 7094-8. doi: https://doi.org/10.1073/pnas.83.18.7094
Belardetti F, Schacher S, Kandel ER, Siegelbaum SA. The growth cones of Aplysia sensory neurons: Modulation by serotonin of action potential duration and single potassium channel currents. Proc Natl Acad Sci U S A. 1986 Sep;83(18):7094-8. doi: 10.1073/pnas.83.18.7094. PMID: 16593764; PMCID: PMC386659.
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The Neurobiology of Fear Generalization.

Frontiers in behavioral neuroscience

Asok A, Kandel ER, Rayman JB.
PMID: 30697153
Front Behav Neurosci. 2019 Jan 15;12:329. doi: 10.3389/fnbeh.2018.00329. eCollection 2018.

The generalization of fear memories is an adaptive neurobiological process that promotes survival in complex and dynamic environments. When confronted with a potential threat, an animal must select an appropriate defensive response based on previous experiences that are not...

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Asok A, Kandel ER, Rayman JB. The Neurobiology of Fear Generalization. Front Behav Neurosci. 2019;12:329doi: 10.3389/fnbeh.2018.00329.
Asok, A., Kandel, E. R., & Rayman, J. B. (2018). The Neurobiology of Fear Generalization. Frontiers in behavioral neuroscience, 12329. https://doi.org/10.3389/fnbeh.2018.00329
Asok, Arun, et al. "The Neurobiology of Fear Generalization." Frontiers in behavioral neuroscience vol. 12 (2018): 329. doi: https://doi.org/10.3389/fnbeh.2018.00329
Asok A, Kandel ER, Rayman JB. The Neurobiology of Fear Generalization. Front Behav Neurosci. 2019 Jan 15;12:329. doi: 10.3389/fnbeh.2018.00329. eCollection 2018. PMID: 30697153; PMCID: PMC6340999.
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Correction: Replication Study: BET bromodomain inhibition as a therapeutic strategy to target c-Myc.

eLife

Aird F, Kandela I, Mantis C.
PMID: 29309031
Elife. 2018 Jan 08;7. doi: 10.7554/eLife.34572.

No abstract available.

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Aird F, Kandela I, Mantis C. Correction: Replication Study: BET bromodomain inhibition as a therapeutic strategy to target c-Myc. Elife. 2018;7doi: 10.7554/eLife.34572.
Aird, F., Kandela, I., Mantis, C. (2018). Correction: Replication Study: BET bromodomain inhibition as a therapeutic strategy to target c-Myc. eLife, 7. https://doi.org/10.7554/eLife.34572
Aird, Fraser, et al. "Correction: Replication Study: BET bromodomain inhibition as a therapeutic strategy to target c-Myc." eLife vol. 7 (2018). doi: https://doi.org/10.7554/eLife.34572
Aird F, Kandela I, Mantis C. Correction: Replication Study: BET bromodomain inhibition as a therapeutic strategy to target c-Myc. Elife. 2018 Jan 08;7. doi: 10.7554/eLife.34572. PMID: 29309031; PMCID: PMC5758113.
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Cytoplasmic Polyadenylation Element Binding Proteins CPEB1 and CPEB3 Regulate the Translation of FosB and Are Required for Maintaining Addiction-Like Behaviors Induced by Cocaine.

Frontiers in cellular neuroscience

Drisaldi B, Colnaghi L, Levine A, Huang Y, Snyder AM, Metzger DJ, Theis M, Kandel DB, Kandel ER, Fioriti L.
PMID: 32742260
Front Cell Neurosci. 2020 Jul 09;14:207. doi: 10.3389/fncel.2020.00207. eCollection 2020.

A recurrent and devastating feature of addiction to a drug of abuse is its persistence, which is mediated by maladaptive long-term memories of the highly pleasurable experience initially associated with the consumption of the drug. We have recently found...

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Drisaldi B, Colnaghi L, Levine A, et al. Cytoplasmic Polyadenylation Element Binding Proteins CPEB1 and CPEB3 Regulate the Translation of FosB and Are Required for Maintaining Addiction-Like Behaviors Induced by Cocaine. Front Cell Neurosci. 2020;14:207doi: 10.3389/fncel.2020.00207.
Drisaldi, B., Colnaghi, L., Levine, A., Huang, Y., Snyder, A. M., Metzger, D. J., Theis, M., Kandel, D. B., Kandel, E. R., & Fioriti, L. (2020). Cytoplasmic Polyadenylation Element Binding Proteins CPEB1 and CPEB3 Regulate the Translation of FosB and Are Required for Maintaining Addiction-Like Behaviors Induced by Cocaine. Frontiers in cellular neuroscience, 14207. https://doi.org/10.3389/fncel.2020.00207
Drisaldi, Bettina, et al. "Cytoplasmic Polyadenylation Element Binding Proteins CPEB1 and CPEB3 Regulate the Translation of FosB and Are Required for Maintaining Addiction-Like Behaviors Induced by Cocaine." Frontiers in cellular neuroscience vol. 14 (2020): 207. doi: https://doi.org/10.3389/fncel.2020.00207
Drisaldi B, Colnaghi L, Levine A, Huang Y, Snyder AM, Metzger DJ, Theis M, Kandel DB, Kandel ER, Fioriti L. Cytoplasmic Polyadenylation Element Binding Proteins CPEB1 and CPEB3 Regulate the Translation of FosB and Are Required for Maintaining Addiction-Like Behaviors Induced by Cocaine. Front Cell Neurosci. 2020 Jul 09;14:207. doi: 10.3389/fncel.2020.00207. eCollection 2020. PMID: 32742260; PMCID: PMC7365288.
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Photocurable bioresorbable adhesives as functional interfaces between flexible bioelectronic devices and soft biological tissues.

Nature materials

Yang Q, Wei T, Yin RT, Wu M, Xu Y, Koo J, Choi YS, Xie Z, Chen SW, Kandela I, Yao S, Deng Y, Avila R, Liu TL, Bai W, Yang Y, Han M, Zhang Q, Haney CR, Benjamin Lee K, Aras K, Wang T, Seo MH, Luan H, Lee SM, Brikha A, Ghoreishi-Haack N, Tran L, Stepien I, Aird F, Waters EA, Yu X, Banks A, Trachiotis GD, Torkelson JM, Huang Y, Kozorovitskiy Y, Efimov IR, Rogers JA.
PMID: 34326506
Nat Mater. 2021 Nov;20(11):1559-1570. doi: 10.1038/s41563-021-01051-x. Epub 2021 Jul 29.

Flexible electronic/optoelectronic systems that can intimately integrate onto the surfaces of vital organ systems have the potential to offer revolutionary diagnostic and therapeutic capabilities relevant to a wide spectrum of diseases and disorders. The critical interfaces between such technologies...

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Yang Q, Wei T, Yin RT, et al. Photocurable bioresorbable adhesives as functional interfaces between flexible bioelectronic devices and soft biological tissues. Nat Mater. 2021;20(11):1559-1570doi: 10.1038/s41563-021-01051-x.
Yang, Q., Wei, T., Yin, R. T., Wu, M., Xu, Y., Koo, J., Choi, Y. S., Xie, Z., Chen, S. W., Kandela, I., Yao, S., Deng, Y., Avila, R., Liu, T. L., Bai, W., Yang, Y., Han, M., Zhang, Q., Haney, C. R., Benjamin Lee, K., Aras, K., Wang, T., Seo, M. H., Luan, H., Lee, S. M., Brikha, A., Ghoreishi-Haack, N., Tran, L., Stepien, I., Aird, F., Waters, E. A., Yu, X., Banks, A., Trachiotis, G. D., Torkelson, J. M., Huang, Y., Kozorovitskiy, Y., Efimov, I. R., & Rogers, J. A. (2021). Photocurable bioresorbable adhesives as functional interfaces between flexible bioelectronic devices and soft biological tissues. Nature materials, 20(11), 1559-1570. https://doi.org/10.1038/s41563-021-01051-x
Yang, Quansan, et al. "Photocurable bioresorbable adhesives as functional interfaces between flexible bioelectronic devices and soft biological tissues." Nature materials vol. 20,11 (2021): 1559-1570. doi: https://doi.org/10.1038/s41563-021-01051-x
Yang Q, Wei T, Yin RT, Wu M, Xu Y, Koo J, Choi YS, Xie Z, Chen SW, Kandela I, Yao S, Deng Y, Avila R, Liu TL, Bai W, Yang Y, Han M, Zhang Q, Haney CR, Benjamin Lee K, Aras K, Wang T, Seo MH, Luan H, Lee SM, Brikha A, Ghoreishi-Haack N, Tran L, Stepien I, Aird F, Waters EA, Yu X, Banks A, Trachiotis GD, Torkelson JM, Huang Y, Kozorovitskiy Y, Efimov IR, Rogers JA. Photocurable bioresorbable adhesives as functional interfaces between flexible bioelectronic devices and soft biological tissues. Nat Mater. 2021 Nov;20(11):1559-1570. doi: 10.1038/s41563-021-01051-x. Epub 2021 Jul 29. PMID: 34326506; PMCID: PMC8551016.
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Comparative Study of First Order Optimizers for Image Classification Using Convolutional Neural Networks on Histopathology Images.

Journal of imaging

Kandel I, Castelli M, Popovič A.
PMID: 34460749
J Imaging. 2020 Sep 08;6(9). doi: 10.3390/jimaging6090092.

The classification of histopathology images requires an experienced physician with years of experience to classify the histopathology images accurately. In this study, an algorithm was developed to assist physicians in classifying histopathology images; the algorithm receives the histopathology image...

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Kandel I, Castelli M, Popovič A. Comparative Study of First Order Optimizers for Image Classification Using Convolutional Neural Networks on Histopathology Images. J Imaging. 2020;6(9)doi: 10.3390/jimaging6090092.
Kandel, I., Castelli, M., & Popovič, A. (2020). Comparative Study of First Order Optimizers for Image Classification Using Convolutional Neural Networks on Histopathology Images. Journal of imaging, 6(9), . https://doi.org/10.3390/jimaging6090092
Kandel, Ibrahem, et al. "Comparative Study of First Order Optimizers for Image Classification Using Convolutional Neural Networks on Histopathology Images." Journal of imaging vol. 6,9 (2020). doi: https://doi.org/10.3390/jimaging6090092
Kandel I, Castelli M, Popovič A. Comparative Study of First Order Optimizers for Image Classification Using Convolutional Neural Networks on Histopathology Images. J Imaging. 2020 Sep 08;6(9). doi: 10.3390/jimaging6090092. PMID: 34460749; PMCID: PMC8321140.
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Enkephalin release from VIP interneurons in the hippocampal CA2/3a region mediates heterosynaptic plasticity and social memory.

Molecular psychiatry

Leroy F, de Solis CA, Boyle LM, Bock T, Lofaro OM, Buss EW, Asok A, Kandel ER, Siegelbaum SA.
PMID: 33990774
Mol Psychiatry. 2021 May 14; doi: 10.1038/s41380-021-01124-y. Epub 2021 May 14.

The hippocampus contains a diverse array of inhibitory interneurons that gate information flow through local cortico-hippocampal circuits to regulate memory storage. Although most studies of interneurons have focused on their role in fast synaptic inhibition mediated by GABA release,...

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Leroy F, de Solis CA, Boyle LM, et al. Enkephalin release from VIP interneurons in the hippocampal CA2/3a region mediates heterosynaptic plasticity and social memory. Mol Psychiatry. 2021;doi: 10.1038/s41380-021-01124-y.
Leroy, F., de Solis, C. A., Boyle, L. M., Bock, T., Lofaro, O. M., Buss, E. W., Asok, A., Kandel, E. R., & Siegelbaum, S. A. (2021). Enkephalin release from VIP interneurons in the hippocampal CA2/3a region mediates heterosynaptic plasticity and social memory. Molecular psychiatry, . https://doi.org/10.1038/s41380-021-01124-y
Leroy, Felix, et al. "Enkephalin release from VIP interneurons in the hippocampal CA2/3a region mediates heterosynaptic plasticity and social memory." Molecular psychiatry vol. (2021). doi: https://doi.org/10.1038/s41380-021-01124-y
Leroy F, de Solis CA, Boyle LM, Bock T, Lofaro OM, Buss EW, Asok A, Kandel ER, Siegelbaum SA. Enkephalin release from VIP interneurons in the hippocampal CA2/3a region mediates heterosynaptic plasticity and social memory. Mol Psychiatry. 2021 May 14; doi: 10.1038/s41380-021-01124-y. Epub 2021 May 14. PMID: 33990774; PMCID: PMC8590711.
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Deep brain stimulation of the nucleus accumbens shell attenuates cocaine withdrawal but increases cocaine self-administration, cocaine-induced locomotor activity, and GluR1/GluA1 in the central nucleus of the amygdala in male cocaine-dependent rats.

Brain stimulation

Kallupi M, Kononoff J, Melas PA, Qvist JS, de Guglielmo G, Kandel ER, George O.
PMID: 34742997
Brain Stimul. 2021 Nov 03;15(1):13-22. doi: 10.1016/j.brs.2021.11.003. Epub 2021 Nov 03.

BACKGROUND: Cocaine addiction is a major public health problem. Despite decades of intense research, no effective treatments are available. Both preclinical and clinical studies strongly suggest that deep brain stimulation of the nucleus accumbens (NAcc) is a viable target...

Cite
Kallupi M, Kononoff J, Melas PA, et al. Deep brain stimulation of the nucleus accumbens shell attenuates cocaine withdrawal but increases cocaine self-administration, cocaine-induced locomotor activity, and GluR1/GluA1 in the central nucleus of the amygdala in male cocaine-dependent rats. Brain Stimul. 2021;15(1):13-22doi: 10.1016/j.brs.2021.11.003.
Kallupi, M., Kononoff, J., Melas, P. A., Qvist, J. S., de Guglielmo, G., Kandel, E. R., & George, O. (2021). Deep brain stimulation of the nucleus accumbens shell attenuates cocaine withdrawal but increases cocaine self-administration, cocaine-induced locomotor activity, and GluR1/GluA1 in the central nucleus of the amygdala in male cocaine-dependent rats. Brain stimulation, 15(1), 13-22. https://doi.org/10.1016/j.brs.2021.11.003
Kallupi, Marsida, et al. "Deep brain stimulation of the nucleus accumbens shell attenuates cocaine withdrawal but increases cocaine self-administration, cocaine-induced locomotor activity, and GluR1/GluA1 in the central nucleus of the amygdala in male cocaine-dependent rats." Brain stimulation vol. 15,1 (2021): 13-22. doi: https://doi.org/10.1016/j.brs.2021.11.003
Kallupi M, Kononoff J, Melas PA, Qvist JS, de Guglielmo G, Kandel ER, George O. Deep brain stimulation of the nucleus accumbens shell attenuates cocaine withdrawal but increases cocaine self-administration, cocaine-induced locomotor activity, and GluR1/GluA1 in the central nucleus of the amygdala in male cocaine-dependent rats. Brain Stimul. 2021 Nov 03;15(1):13-22. doi: 10.1016/j.brs.2021.11.003. Epub 2021 Nov 03. PMID: 34742997.
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Deep brain stimulation of the nucleus accumbens shell attenuates cocaine withdrawal but increases cocaine self-administration, cocaine-induced locomotor activity, and GluR1/GluA1 in the central nucleus of the amygdala in male cocaine-dependent rats.

Brain stimulation

Kallupi M, Kononoff J, Melas PA, Qvist JS, de Guglielmo G, Kandel ER, George O.
PMID: 34742997
Brain Stimul. 2021 Nov 03;15(1):13-22. doi: 10.1016/j.brs.2021.11.003. Epub 2021 Nov 03.

BACKGROUND: Cocaine addiction is a major public health problem. Despite decades of intense research, no effective treatments are available. Both preclinical and clinical studies strongly suggest that deep brain stimulation of the nucleus accumbens (NAcc) is a viable target...

Cite
Kallupi M, Kononoff J, Melas PA, et al. Deep brain stimulation of the nucleus accumbens shell attenuates cocaine withdrawal but increases cocaine self-administration, cocaine-induced locomotor activity, and GluR1/GluA1 in the central nucleus of the amygdala in male cocaine-dependent rats. Brain Stimul. 2021;15(1):13-22doi: 10.1016/j.brs.2021.11.003.
Kallupi, M., Kononoff, J., Melas, P. A., Qvist, J. S., de Guglielmo, G., Kandel, E. R., & George, O. (2021). Deep brain stimulation of the nucleus accumbens shell attenuates cocaine withdrawal but increases cocaine self-administration, cocaine-induced locomotor activity, and GluR1/GluA1 in the central nucleus of the amygdala in male cocaine-dependent rats. Brain stimulation, 15(1), 13-22. https://doi.org/10.1016/j.brs.2021.11.003
Kallupi, Marsida, et al. "Deep brain stimulation of the nucleus accumbens shell attenuates cocaine withdrawal but increases cocaine self-administration, cocaine-induced locomotor activity, and GluR1/GluA1 in the central nucleus of the amygdala in male cocaine-dependent rats." Brain stimulation vol. 15,1 (2021): 13-22. doi: https://doi.org/10.1016/j.brs.2021.11.003
Kallupi M, Kononoff J, Melas PA, Qvist JS, de Guglielmo G, Kandel ER, George O. Deep brain stimulation of the nucleus accumbens shell attenuates cocaine withdrawal but increases cocaine self-administration, cocaine-induced locomotor activity, and GluR1/GluA1 in the central nucleus of the amygdala in male cocaine-dependent rats. Brain Stimul. 2021 Nov 03;15(1):13-22. doi: 10.1016/j.brs.2021.11.003. Epub 2021 Nov 03. PMID: 34742997.
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Deep brain stimulation of the nucleus accumbens shell attenuates cocaine withdrawal but increases cocaine self-administration, cocaine-induced locomotor activity, and GluR1/GluA1 in the central nucleus of the amygdala in male cocaine-dependent rats.

Brain stimulation

Kallupi M, Kononoff J, Melas PA, Qvist JS, de Guglielmo G, Kandel ER, George O.
PMID: 34742997
Brain Stimul. 2021 Nov 03;15(1):13-22. doi: 10.1016/j.brs.2021.11.003. Epub 2021 Nov 03.

BACKGROUND: Cocaine addiction is a major public health problem. Despite decades of intense research, no effective treatments are available. Both preclinical and clinical studies strongly suggest that deep brain stimulation of the nucleus accumbens (NAcc) is a viable target...

Cite
Kallupi M, Kononoff J, Melas PA, et al. Deep brain stimulation of the nucleus accumbens shell attenuates cocaine withdrawal but increases cocaine self-administration, cocaine-induced locomotor activity, and GluR1/GluA1 in the central nucleus of the amygdala in male cocaine-dependent rats. Brain Stimul. 2021;15(1):13-22doi: 10.1016/j.brs.2021.11.003.
Kallupi, M., Kononoff, J., Melas, P. A., Qvist, J. S., de Guglielmo, G., Kandel, E. R., & George, O. (2021). Deep brain stimulation of the nucleus accumbens shell attenuates cocaine withdrawal but increases cocaine self-administration, cocaine-induced locomotor activity, and GluR1/GluA1 in the central nucleus of the amygdala in male cocaine-dependent rats. Brain stimulation, 15(1), 13-22. https://doi.org/10.1016/j.brs.2021.11.003
Kallupi, Marsida, et al. "Deep brain stimulation of the nucleus accumbens shell attenuates cocaine withdrawal but increases cocaine self-administration, cocaine-induced locomotor activity, and GluR1/GluA1 in the central nucleus of the amygdala in male cocaine-dependent rats." Brain stimulation vol. 15,1 (2021): 13-22. doi: https://doi.org/10.1016/j.brs.2021.11.003
Kallupi M, Kononoff J, Melas PA, Qvist JS, de Guglielmo G, Kandel ER, George O. Deep brain stimulation of the nucleus accumbens shell attenuates cocaine withdrawal but increases cocaine self-administration, cocaine-induced locomotor activity, and GluR1/GluA1 in the central nucleus of the amygdala in male cocaine-dependent rats. Brain Stimul. 2021 Nov 03;15(1):13-22. doi: 10.1016/j.brs.2021.11.003. Epub 2021 Nov 03. PMID: 34742997.
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Diphasic postsynaptic potential: a chemical synapse capable of mediating conjoint excitation and inhibition.

Science (New York, N.Y.)

Gardner D, Kandel ER.
PMID: 17778169
Science. 1972 May 12;176(4035):675-8. doi: 10.1126/science.176.4035.675.

Two identified interneurons in each buccal ganglion of Aplysia can mediate conjoined excitation and inhibition to a single follower cell. A single presynaptic action potential in one of these interneurons produces a diphasic, depolarizing-hyperpolarizing synaptic potential apparently as a...

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Gardner D, Kandel ER. Diphasic postsynaptic potential: a chemical synapse capable of mediating conjoint excitation and inhibition. Science. 1972;176(4035):675-8doi: 10.1126/science.176.4035.675.
Gardner, D., & Kandel, E. R. (1972). Diphasic postsynaptic potential: a chemical synapse capable of mediating conjoint excitation and inhibition. Science (New York, N.Y.), 176(4035), 675-8. https://doi.org/10.1126/science.176.4035.675
Gardner, D, and Kandel, E R. "Diphasic postsynaptic potential: a chemical synapse capable of mediating conjoint excitation and inhibition." Science (New York, N.Y.) vol. 176,4035 (1972): 675-8. doi: https://doi.org/10.1126/science.176.4035.675
Gardner D, Kandel ER. Diphasic postsynaptic potential: a chemical synapse capable of mediating conjoint excitation and inhibition. Science. 1972 May 12;176(4035):675-8. doi: 10.1126/science.176.4035.675. PMID: 17778169.
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Orbitofrontal cortex mediates the differential impact of signaled-reward probability on discrimination accuracy.

Frontiers in neuroscience

Ward RD, Winiger V, Kandel ER, Balsam PD, Simpson EH.
PMID: 26157358
Front Neurosci. 2015 Jun 23;9:230. doi: 10.3389/fnins.2015.00230. eCollection 2015.

Orbitofrontal cortex (OFC) function is critical to decision making and behavior based on the value of expected outcomes. While some of the roles the OFC plays in value computations and behavior have been identified, the role of the OFC...

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Ward RD, Winiger V, Kandel ER, et al. Orbitofrontal cortex mediates the differential impact of signaled-reward probability on discrimination accuracy. Front Neurosci. 2015;9:230doi: 10.3389/fnins.2015.00230.
Ward, R. D., Winiger, V., Kandel, E. R., Balsam, P. D., & Simpson, E. H. (2015). Orbitofrontal cortex mediates the differential impact of signaled-reward probability on discrimination accuracy. Frontiers in neuroscience, 9230. https://doi.org/10.3389/fnins.2015.00230
Ward, Ryan D, et al. "Orbitofrontal cortex mediates the differential impact of signaled-reward probability on discrimination accuracy." Frontiers in neuroscience vol. 9 (2015): 230. doi: https://doi.org/10.3389/fnins.2015.00230
Ward RD, Winiger V, Kandel ER, Balsam PD, Simpson EH. Orbitofrontal cortex mediates the differential impact of signaled-reward probability on discrimination accuracy. Front Neurosci. 2015 Jun 23;9:230. doi: 10.3389/fnins.2015.00230. eCollection 2015. PMID: 26157358; PMCID: PMC4477136.
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