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Diet rich in Docosahexaenoic Acid/Eicosapentaenoic Acid robustly ameliorates hepatic steatosis and insulin resistance in seipin deficient lipodystrophy mice.

Nutrition & metabolism

Xu P, Wang H, Kayoumu A, Wang M, Huang W, Liu G.
PMID: 26690553
Nutr Metab (Lond). 2015 Dec 18;12:58. doi: 10.1186/s12986-015-0054-x. eCollection 2015.

BACKGROUND: N-3 polyunsaturated fatty acids (n-3 PUFAs), in particular eicosapentaenoic acid (EPA) and docosahexaenoic acid (DHA), have been shown to effectively improve hepatic steatosis and insulin resistance caused by obesity. Lipodystrophy could also develop insulin resistance and hepatic steatosis....

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Xu P, Wang H, Kayoumu A, et al. Diet rich in Docosahexaenoic Acid/Eicosapentaenoic Acid robustly ameliorates hepatic steatosis and insulin resistance in seipin deficient lipodystrophy mice. Nutr Metab (Lond). 2015;12:58doi: 10.1186/s12986-015-0054-x.
Xu, P., Wang, H., Kayoumu, A., Wang, M., Huang, W., & Liu, G. (2015). Diet rich in Docosahexaenoic Acid/Eicosapentaenoic Acid robustly ameliorates hepatic steatosis and insulin resistance in seipin deficient lipodystrophy mice. Nutrition & metabolism, 1258. https://doi.org/10.1186/s12986-015-0054-x
Xu, Pengfei, et al. "Diet rich in Docosahexaenoic Acid/Eicosapentaenoic Acid robustly ameliorates hepatic steatosis and insulin resistance in seipin deficient lipodystrophy mice." Nutrition & metabolism vol. 12 (2015): 58. doi: https://doi.org/10.1186/s12986-015-0054-x
Xu P, Wang H, Kayoumu A, Wang M, Huang W, Liu G. Diet rich in Docosahexaenoic Acid/Eicosapentaenoic Acid robustly ameliorates hepatic steatosis and insulin resistance in seipin deficient lipodystrophy mice. Nutr Metab (Lond). 2015 Dec 18;12:58. doi: 10.1186/s12986-015-0054-x. eCollection 2015. PMID: 26690553; PMCID: PMC4683947.
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Dexmedetomidine as an additive to local anesthetics compared with intravenous dexmedetomidine in peribulbar block for cataract surgery.

Saudi journal of anaesthesia

Abdelhamid AM, Mahmoud A, Abdelhaq MM, Yasin HM, Bayoumi A.
PMID: 26952175
Saudi J Anaesth. 2016 Jan-Mar;10(1):50-4. doi: 10.4103/1658-354X.169475.

BACKGROUND: No studies compared parenteral dexmedetomidine with its use as an adjuvant to ophthalmic block. We compared between adding dexmedetomidine to bupivacaine in peribulbar block and intravenous (IV) dexmedetomidine during peribulbar block for cataract surgery.MATERIALS AND METHODS: A prospective,...

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Abdelhamid AM, Mahmoud A, Abdelhaq MM, et al. Dexmedetomidine as an additive to local anesthetics compared with intravenous dexmedetomidine in peribulbar block for cataract surgery. Saudi J Anaesth. 2016;10(1):50-4doi: 10.4103/1658-354X.169475.
Abdelhamid, A. M., Mahmoud, A., Abdelhaq, M. M., Yasin, H. M., & Bayoumi, A. (2016). Dexmedetomidine as an additive to local anesthetics compared with intravenous dexmedetomidine in peribulbar block for cataract surgery. Saudi journal of anaesthesia, 10(1), 50-4. https://doi.org/10.4103/1658-354X.169475
Abdelhamid, A M, et al. "Dexmedetomidine as an additive to local anesthetics compared with intravenous dexmedetomidine in peribulbar block for cataract surgery." Saudi journal of anaesthesia vol. 10,1 (2016): 50-4. doi: https://doi.org/10.4103/1658-354X.169475
Abdelhamid AM, Mahmoud A, Abdelhaq MM, Yasin HM, Bayoumi A. Dexmedetomidine as an additive to local anesthetics compared with intravenous dexmedetomidine in peribulbar block for cataract surgery. Saudi J Anaesth. 2016 Jan-Mar;10(1):50-4. doi: 10.4103/1658-354X.169475. PMID: 26952175; PMCID: PMC4760042.
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Copy number variation and expression of exportin-4 associates with severity of fibrosis in metabolic associated fatty liver disease.

EBioMedicine

Metwally M, Bayoumi A, Khan A, Adams LA, Aller R, García-Monzón C, Arias-Loste MT, Bugianesi E, Miele L, Anna A, Latchoumanin O, Han S, Alenizi S, Sharkawy RE, Elattar A, Gallego-Durán R, Fischer J, Berg T, Liddle C, Romero-Gomez M, George J, Eslam M.
PMID: 34388518
EBioMedicine. 2021 Aug;70:103521. doi: 10.1016/j.ebiom.2021.103521. Epub 2021 Aug 11.

BACKGROUND: Liver fibrosis risk is a heritable trait, the outcome of which is the net deposition of extracellular matrix by hepatic stellate cell-derived myofibroblasts. Whereas nucleotide sequence variations have been extensively studied in liver fibrosis, the role of copy...

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Metwally M, Bayoumi A, Khan A, et al. Copy number variation and expression of exportin-4 associates with severity of fibrosis in metabolic associated fatty liver disease. EBioMedicine. 2021;70:103521doi: 10.1016/j.ebiom.2021.103521.
Metwally, M., Bayoumi, A., Khan, A., Adams, L. A., Aller, R., García-Monzón, C., Arias-Loste, M. T., Bugianesi, E., Miele, L., Anna, A., Latchoumanin, O., Han, S., Alenizi, S., Sharkawy, R. E., Elattar, A., Gallego-Durán, R., Fischer, J., Berg, T., Liddle, C., Romero-Gomez, M., George, J., & Eslam, M. (2021). Copy number variation and expression of exportin-4 associates with severity of fibrosis in metabolic associated fatty liver disease. EBioMedicine, 70103521. https://doi.org/10.1016/j.ebiom.2021.103521
Metwally, Mayada, et al. "Copy number variation and expression of exportin-4 associates with severity of fibrosis in metabolic associated fatty liver disease." EBioMedicine vol. 70 (2021): 103521. doi: https://doi.org/10.1016/j.ebiom.2021.103521
Metwally M, Bayoumi A, Khan A, Adams LA, Aller R, García-Monzón C, Arias-Loste MT, Bugianesi E, Miele L, Anna A, Latchoumanin O, Han S, Alenizi S, Sharkawy RE, Elattar A, Gallego-Durán R, Fischer J, Berg T, Liddle C, Romero-Gomez M, George J, Eslam M. Copy number variation and expression of exportin-4 associates with severity of fibrosis in metabolic associated fatty liver disease. EBioMedicine. 2021 Aug;70:103521. doi: 10.1016/j.ebiom.2021.103521. Epub 2021 Aug 11. PMID: 34388518; PMCID: PMC8365315.
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[No title available]

CMAJ : Canadian Medical Association journal = journal de l'Association medicale canadienne

Bayoumi A, Laupacis A.
PMID: 34844946
CMAJ. 2021 Nov 29;193(47):E1826-E1827. doi: 10.1503/cmaj.211617-f.

No abstract available.

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Bayoumi A, Laupacis A. Des critères d’assurance désuets empêchent le remboursement de soins fondés sur des données probantes. . CMAJ. 2021;193(47):E1826-E1827doi: 10.1503/cmaj.211617-f.
Bayoumi, A., & Laupacis, A. (2021). Des critères d’assurance désuets empêchent le remboursement de soins fondés sur des données probantes. . CMAJ : Canadian Medical Association journal = journal de l'Association medicale canadienne, 193(47), E1826-E1827. https://doi.org/10.1503/cmaj.211617-f
Bayoumi, Ahmed, and Laupacis, Andreas. "Des critères d’assurance désuets empêchent le remboursement de soins fondés sur des données probantes." . CMAJ : Canadian Medical Association journal = journal de l'Association medicale canadienne vol. 193,47 (2021): E1826-E1827. doi: https://doi.org/10.1503/cmaj.211617-f
Bayoumi A, Laupacis A. Des critères d’assurance désuets empêchent le remboursement de soins fondés sur des données probantes. . CMAJ. 2021 Nov 29;193(47):E1826-E1827. doi: 10.1503/cmaj.211617-f. French. PMID: 34844946; PMCID: PMC8654894.
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Spontaneous type 1 pattern, ventricular arrhythmias and sudden cardiac death in Brugada Syndrome: an updated systematic review and meta-analysis.

Journal of geriatric cardiology : JGC

Bayoumy A, Gong MQ, Christien Li KH, Wong SH, Wu WK, Li GP, Bazoukis G, Letsas KP, Wong WT, Xia YL, Liu T, Tse G.
PMID: 29238365
J Geriatr Cardiol. 2017 Oct;14(10):639-643. doi: 10.11909/j.issn.1671-5411.2017.10.010.

No abstract available.

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Bayoumy A, Gong MQ, Christien Li KH, et al. Spontaneous type 1 pattern, ventricular arrhythmias and sudden cardiac death in Brugada Syndrome: an updated systematic review and meta-analysis. J Geriatr Cardiol. 2017;14(10):639-643doi: 10.11909/j.issn.1671-5411.2017.10.010.
Bayoumy, A., Gong, M. Q., Christien Li, K. H., Wong, S. H., Wu, W. K., Li, G. P., Bazoukis, G., Letsas, K. P., Wong, W. T., Xia, Y. L., Liu, T., Tse, G. (2017). Spontaneous type 1 pattern, ventricular arrhythmias and sudden cardiac death in Brugada Syndrome: an updated systematic review and meta-analysis. Journal of geriatric cardiology : JGC, 14(10), 639-643. https://doi.org/10.11909/j.issn.1671-5411.2017.10.010
Bayoumy, Ahmed, et al. "Spontaneous type 1 pattern, ventricular arrhythmias and sudden cardiac death in Brugada Syndrome: an updated systematic review and meta-analysis." Journal of geriatric cardiology : JGC vol. 14,10 (2017): 639-643. doi: https://doi.org/10.11909/j.issn.1671-5411.2017.10.010
Bayoumy A, Gong MQ, Christien Li KH, Wong SH, Wu WK, Li GP, Bazoukis G, Letsas KP, Wong WT, Xia YL, Liu T, Tse G. Spontaneous type 1 pattern, ventricular arrhythmias and sudden cardiac death in Brugada Syndrome: an updated systematic review and meta-analysis. J Geriatr Cardiol. 2017 Oct;14(10):639-643. doi: 10.11909/j.issn.1671-5411.2017.10.010. PMID: 29238365; PMCID: PMC5721199.
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Validation of the PILL-5: A 5-Item Patient Reported Outcome Measure for Pill Dysphagia.

Frontiers in surgery

Nativ-Zeltzer N, Bayoumi A, Mandin VP, Kaufman M, Seeni I, Kuhn MA, Belafsky PC.
PMID: 31396520
Front Surg. 2019 Jul 24;6:43. doi: 10.3389/fsurg.2019.00043. eCollection 2019.

No abstract available.

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Nativ-Zeltzer N, Bayoumi A, Mandin VP, et al. Validation of the PILL-5: A 5-Item Patient Reported Outcome Measure for Pill Dysphagia. Front Surg. 2019;6:43doi: 10.3389/fsurg.2019.00043.
Nativ-Zeltzer, N., Bayoumi, A., Mandin, V. P., Kaufman, M., Seeni, I., Kuhn, M. A., & Belafsky, P. C. (2019). Validation of the PILL-5: A 5-Item Patient Reported Outcome Measure for Pill Dysphagia. Frontiers in surgery, 643. https://doi.org/10.3389/fsurg.2019.00043
Nativ-Zeltzer, Nogah, et al. "Validation of the PILL-5: A 5-Item Patient Reported Outcome Measure for Pill Dysphagia." Frontiers in surgery vol. 6 (2019): 43. doi: https://doi.org/10.3389/fsurg.2019.00043
Nativ-Zeltzer N, Bayoumi A, Mandin VP, Kaufman M, Seeni I, Kuhn MA, Belafsky PC. Validation of the PILL-5: A 5-Item Patient Reported Outcome Measure for Pill Dysphagia. Front Surg. 2019 Jul 24;6:43. doi: 10.3389/fsurg.2019.00043. eCollection 2019. PMID: 31396520; PMCID: PMC6667828.
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Protective effect of L-carnitine and coenzyme Q10 on CCl₄-induced liver injury in rats.

Scientia pharmaceutica

Ali SA, Faddah L, Abdel-Baky A, Bayoumi A.
PMID: 21179323
Sci Pharm. 2010 Oct-Dec;78(4):881-96. doi: 10.3797/scipharm.1006-02. Epub 2010 Aug 23.

This study provides an information about the mechanisms of liver injury induced by CCl(4), and determines the influence of administration of L-carnitine or/and CoQ10 as prophylactic agents against CCl(4) deteriorative effect. The study was carried out on 80 adult...

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Ali SA, Faddah L, Abdel-Baky A, et al. Protective effect of L-carnitine and coenzyme Q10 on CCl₄-induced liver injury in rats. Sci Pharm. 2010;78(4):881-96doi: 10.3797/scipharm.1006-02.
Ali, S. A., Faddah, L., Abdel-Baky, A., & Bayoumi, A. (2010). Protective effect of L-carnitine and coenzyme Q10 on CCl₄-induced liver injury in rats. Scientia pharmaceutica, 78(4), 881-96. https://doi.org/10.3797/scipharm.1006-02
Ali, Sanaa Ahmed, et al. "Protective effect of L-carnitine and coenzyme Q10 on CCl₄-induced liver injury in rats." Scientia pharmaceutica vol. 78,4 (2010): 881-96. doi: https://doi.org/10.3797/scipharm.1006-02
Ali SA, Faddah L, Abdel-Baky A, Bayoumi A. Protective effect of L-carnitine and coenzyme Q10 on CCl₄-induced liver injury in rats. Sci Pharm. 2010 Oct-Dec;78(4):881-96. doi: 10.3797/scipharm.1006-02. Epub 2010 Aug 23. PMID: 21179323; PMCID: PMC3007612.
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Maternal inheritance of severe hypertriglyceridemia impairs glucose metabolism in offspring.

Journal of biomedical research

Ma YH, Yu C, Kayoumu A, Guo X, Ji Z, Liu G.
PMID: 25859267
J Biomed Res. 2015 Apr;29(2):125-31. doi: 10.7555/JBR.29.20140139. Epub 2015 Mar 13.

Maternally inherited familial hypercholesterolemia (FH) impairs glucose metabolism and increases cardiovascular risks in the offspring to a greater degree than paternal inherited FH. However, it remains unknown whether hypertriglyceridemia affects glucose metabolism via inheritance. In this study, we sought...

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Ma YH, Yu C, Kayoumu A, et al. Maternal inheritance of severe hypertriglyceridemia impairs glucose metabolism in offspring. J Biomed Res. 2015;29(2):125-31doi: 10.7555/JBR.29.20140139.
Ma, Y. H., Yu, C., Kayoumu, A., Guo, X., Ji, Z., & Liu, G. (2015). Maternal inheritance of severe hypertriglyceridemia impairs glucose metabolism in offspring. Journal of biomedical research, 29(2), 125-31. https://doi.org/10.7555/JBR.29.20140139
Ma, Ya-Hong, et al. "Maternal inheritance of severe hypertriglyceridemia impairs glucose metabolism in offspring." Journal of biomedical research vol. 29,2 (2015): 125-31. doi: https://doi.org/10.7555/JBR.29.20140139
Ma YH, Yu C, Kayoumu A, Guo X, Ji Z, Liu G. Maternal inheritance of severe hypertriglyceridemia impairs glucose metabolism in offspring. J Biomed Res. 2015 Apr;29(2):125-31. doi: 10.7555/JBR.29.20140139. Epub 2015 Mar 13. PMID: 25859267; PMCID: PMC4389112.
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Case Series of Perforated Keratomycosis after Laser-Assisted In Situ Keratomileusis.

Case reports in ophthalmological medicine

Eleiwa T, Ozcan E, Abdelrahman S, Solyman O, Elhusseiny AM, Youssef G, Bayoumy A.
PMID: 33014490
Case Rep Ophthalmol Med. 2020 Sep 15;2020:7237903. doi: 10.1155/2020/7237903. eCollection 2020.

BACKGROUND: Fungal keratitis is an extremely rare complication of laser vision correction resulting in poor visual outcomes. Amniotic membrane transplantation should be kept in mind in eyes with corneal perforation prior to penetrating keratoplasty.AIM: To assess the outcomes of...

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Eleiwa T, Ozcan E, Abdelrahman S, et al. Case Series of Perforated Keratomycosis after Laser-Assisted In Situ Keratomileusis. Case Rep Ophthalmol Med. 2020;2020:7237903doi: 10.1155/2020/7237903.
Eleiwa, T., Ozcan, E., Abdelrahman, S., Solyman, O., Elhusseiny, A. M., Youssef, G., & Bayoumy, A. (2020). Case Series of Perforated Keratomycosis after Laser-Assisted In Situ Keratomileusis. Case reports in ophthalmological medicine, 20207237903. https://doi.org/10.1155/2020/7237903
Eleiwa, Taher, et al. "Case Series of Perforated Keratomycosis after Laser-Assisted In Situ Keratomileusis." Case reports in ophthalmological medicine vol. 2020 (2020): 7237903. doi: https://doi.org/10.1155/2020/7237903
Eleiwa T, Ozcan E, Abdelrahman S, Solyman O, Elhusseiny AM, Youssef G, Bayoumy A. Case Series of Perforated Keratomycosis after Laser-Assisted In Situ Keratomileusis. Case Rep Ophthalmol Med. 2020 Sep 15;2020:7237903. doi: 10.1155/2020/7237903. eCollection 2020. PMID: 33014490; PMCID: PMC7512097.
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Guillain Barre Syndrome as a Complication of COVID-19: A Systematic Review.

The Canadian journal of neurological sciences. Le journal canadien des sciences neurologiques

Aladawi M, Elfil M, Abu-Esheh B, Abu Jazar D, Armouti A, Bayoumi A, Piccione E.
PMID: 33949300
Can J Neurol Sci. 2022 Jan;49(1):38-48. doi: 10.1017/cjn.2021.102. Epub 2021 May 05.

BACKGROUND: In January 2020, the first case of Guillain Barre syndrome (GBS) due to COVID-19 was documented in China. GBS is known to be postinfectious following several types of infections. Although causality can only be proven through large epidemiological...

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Aladawi M, Elfil M, Abu-Esheh B, et al. Guillain Barre Syndrome as a Complication of COVID-19: A Systematic Review. Can J Neurol Sci. 2021;49(1):38-48doi: 10.1017/cjn.2021.102.
Aladawi, M., Elfil, M., Abu-Esheh, B., Abu Jazar, D., Armouti, A., Bayoumi, A., & Piccione, E. (2022). Guillain Barre Syndrome as a Complication of COVID-19: A Systematic Review. The Canadian journal of neurological sciences. Le journal canadien des sciences neurologiques, 49(1), 38-48. https://doi.org/10.1017/cjn.2021.102
Aladawi, Mohammad, et al. "Guillain Barre Syndrome as a Complication of COVID-19: A Systematic Review." The Canadian journal of neurological sciences. Le journal canadien des sciences neurologiques vol. 49,1 (2022): 38-48. doi: https://doi.org/10.1017/cjn.2021.102
Aladawi M, Elfil M, Abu-Esheh B, Abu Jazar D, Armouti A, Bayoumi A, Piccione E. Guillain Barre Syndrome as a Complication of COVID-19: A Systematic Review. Can J Neurol Sci. 2022 Jan;49(1):38-48. doi: 10.1017/cjn.2021.102. Epub 2021 May 05. PMID: 33949300; PMCID: PMC8267336.
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Volitional control of the upper esophageal sphincter with high-resolution manometry driven biofeedback.

Laryngoscope investigative otolaryngology

Nativ-Zeltzer N, Belafsky PC, Bayoumi A, Kuhn MA.
PMID: 31024999
Laryngoscope Investig Otolaryngol. 2019 Feb 28;4(2):264-268. doi: 10.1002/lio2.255. eCollection 2019 Apr.

INTRODUCTION: Dysfunction of the upper esophageal sphincter (UES) is associated with swallow dysfunction and globus pharyngeus. Although volitional augmentation of the UES has been previously documented, the ability of individuals to control UES pressure with high-resolution manometry (HRM) driven...

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Nativ-Zeltzer N, Belafsky PC, Bayoumi A, et al. Volitional control of the upper esophageal sphincter with high-resolution manometry driven biofeedback. Laryngoscope Investig Otolaryngol. 2019;4(2):264-268doi: 10.1002/lio2.255.
Nativ-Zeltzer, N., Belafsky, P. C., Bayoumi, A., & Kuhn, M. A. (2019). Volitional control of the upper esophageal sphincter with high-resolution manometry driven biofeedback. Laryngoscope investigative otolaryngology, 4(2), 264-268. https://doi.org/10.1002/lio2.255
Nativ-Zeltzer, Nogah, et al. "Volitional control of the upper esophageal sphincter with high-resolution manometry driven biofeedback." Laryngoscope investigative otolaryngology vol. 4,2 (2019): 264-268. doi: https://doi.org/10.1002/lio2.255
Nativ-Zeltzer N, Belafsky PC, Bayoumi A, Kuhn MA. Volitional control of the upper esophageal sphincter with high-resolution manometry driven biofeedback. Laryngoscope Investig Otolaryngol. 2019 Feb 28;4(2):264-268. doi: 10.1002/lio2.255. eCollection 2019 Apr. PMID: 31024999; PMCID: PMC6476264.
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Guillain Barre Syndrome as a Complication of COVID-19: A Systematic Review.

The Canadian journal of neurological sciences. Le journal canadien des sciences neurologiques

Aladawi M, Elfil M, Abu-Esheh B, Abu Jazar D, Armouti A, Bayoumi A, Piccione E.
PMID: 33949300
Can J Neurol Sci. 2022 Jan;49(1):38-48. doi: 10.1017/cjn.2021.102. Epub 2021 May 05.

BACKGROUND: In January 2020, the first case of Guillain Barre syndrome (GBS) due to COVID-19 was documented in China. GBS is known to be postinfectious following several types of infections. Although causality can only be proven through large epidemiological...

Cite
Aladawi M, Elfil M, Abu-Esheh B, et al. Guillain Barre Syndrome as a Complication of COVID-19: A Systematic Review. Can J Neurol Sci. 2021;49(1):38-48doi: 10.1017/cjn.2021.102.
Aladawi, M., Elfil, M., Abu-Esheh, B., Abu Jazar, D., Armouti, A., Bayoumi, A., & Piccione, E. (2022). Guillain Barre Syndrome as a Complication of COVID-19: A Systematic Review. The Canadian journal of neurological sciences. Le journal canadien des sciences neurologiques, 49(1), 38-48. https://doi.org/10.1017/cjn.2021.102
Aladawi, Mohammad, et al. "Guillain Barre Syndrome as a Complication of COVID-19: A Systematic Review." The Canadian journal of neurological sciences. Le journal canadien des sciences neurologiques vol. 49,1 (2022): 38-48. doi: https://doi.org/10.1017/cjn.2021.102
Aladawi M, Elfil M, Abu-Esheh B, Abu Jazar D, Armouti A, Bayoumi A, Piccione E. Guillain Barre Syndrome as a Complication of COVID-19: A Systematic Review. Can J Neurol Sci. 2022 Jan;49(1):38-48. doi: 10.1017/cjn.2021.102. Epub 2021 May 05. PMID: 33949300; PMCID: PMC8267336.
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