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Cognitive remediation for bipolar patients with objective cognitive impairment: a naturalistic study.

International journal of bipolar disorders

Veeh J, Kopf J, Kittel-Schneider S, Deckert J, Reif A.
PMID: 28168631
Int J Bipolar Disord. 2017 Dec;5(1):8. doi: 10.1186/s40345-017-0079-3. Epub 2017 Apr 13.

BACKGROUND: Many bipolar patients (BP) are affected by cognitive impairments and reduced psychosocial function even after complete remission. In the present naturalistic study, we developed a tailored cognitive remediation program (CR) to evaluate the effect on objective and subjective...

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Veeh J, Kopf J, Kittel-Schneider S, et al. Cognitive remediation for bipolar patients with objective cognitive impairment: a naturalistic study. Int J Bipolar Disord. 2017;5(1):8doi: 10.1186/s40345-017-0079-3.
Veeh, J., Kopf, J., Kittel-Schneider, S., Deckert, J., & Reif, A. (2017). Cognitive remediation for bipolar patients with objective cognitive impairment: a naturalistic study. International journal of bipolar disorders, 5(1), 8. https://doi.org/10.1186/s40345-017-0079-3
Veeh, J, et al. "Cognitive remediation for bipolar patients with objective cognitive impairment: a naturalistic study." International journal of bipolar disorders vol. 5,1 (2017): 8. doi: https://doi.org/10.1186/s40345-017-0079-3
Veeh J, Kopf J, Kittel-Schneider S, Deckert J, Reif A. Cognitive remediation for bipolar patients with objective cognitive impairment: a naturalistic study. Int J Bipolar Disord. 2017 Dec;5(1):8. doi: 10.1186/s40345-017-0079-3. Epub 2017 Apr 13. PMID: 28168631; PMCID: PMC5389951.
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Corrigendum to "Generation of human induced pluripotent stem cell lines (hiPSC) from one bipolar disorder patient carrier of a DGKH risk haplotype and one non-risk-variant-carrier bipolar disorder patient" [Stem Cell Res. 2018 Oct;32:104-109. doi: 10.1016/j.scr.2018.09.008. Epub 2018 Sep 17].

Stem cell research

Palladino VS, Subrata NOC, Geburtig-Chiocchetti A, McNeill R, Hoffmann P, Reif A, Kittel-Schneider S.
PMID: 30898465
Stem Cell Res. 2019 Apr;36:101419. doi: 10.1016/j.scr.2019.101419. Epub 2019 Mar 18.

No abstract available.

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Palladino VS, Subrata NOC, Geburtig-Chiocchetti A, et al. Corrigendum to "Generation of human induced pluripotent stem cell lines (hiPSC) from one bipolar disorder patient carrier of a DGKH risk haplotype and one non-risk-variant-carrier bipolar disorder patient" [Stem Cell Res. 2018 Oct;32:104-109. doi: 10.1016/j.scr.2018.09.008. Epub 2018 Sep 17]. Stem Cell Res. 2019;36:101419doi: 10.1016/j.scr.2019.101419.
Palladino, V. S., Subrata, N. O. C., Geburtig-Chiocchetti, A., McNeill, R., Hoffmann, P., Reif, A., & Kittel-Schneider, S. (2019). Corrigendum to "Generation of human induced pluripotent stem cell lines (hiPSC) from one bipolar disorder patient carrier of a DGKH risk haplotype and one non-risk-variant-carrier bipolar disorder patient" [Stem Cell Res. 2018 Oct;32:104-109. doi: 10.1016/j.scr.2018.09.008. Epub 2018 Sep 17]. Stem cell research, 36101419. https://doi.org/10.1016/j.scr.2019.101419
Palladino, V S, et al. "Corrigendum to "Generation of human induced pluripotent stem cell lines (hiPSC) from one bipolar disorder patient carrier of a DGKH risk haplotype and one non-risk-variant-carrier bipolar disorder patient" [Stem Cell Res. 2018 Oct;32:104-109. doi: 10.1016/j.scr.2018.09.008. Epub 2018 Sep 17]." Stem cell research vol. 36 (2019): 101419. doi: https://doi.org/10.1016/j.scr.2019.101419
Palladino VS, Subrata NOC, Geburtig-Chiocchetti A, McNeill R, Hoffmann P, Reif A, Kittel-Schneider S. Corrigendum to "Generation of human induced pluripotent stem cell lines (hiPSC) from one bipolar disorder patient carrier of a DGKH risk haplotype and one non-risk-variant-carrier bipolar disorder patient" [Stem Cell Res. 2018 Oct;32:104-109. doi: 10.1016/j.scr.2018.09.008. Epub 2018 Sep 17]. Stem Cell Res. 2019 Apr;36:101419. doi: 10.1016/j.scr.2019.101419. Epub 2019 Mar 18. PMID: 30898465.
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Brunner syndrome associated MAOA mutations result in NMDAR hyperfunction and increased network activity in human dopaminergic neurons.

Neurobiology of disease

van Rhijn JR, Shi Y, Bormann M, Mossink B, Frega M, Recaioglu H, Hakobjan M, Klein Gunnewiek T, Schoenmaker C, Palmer E, Faivre L, Kittel-Schneider S, Schubert D, Brunner H, Franke B, Nadif Kasri N.
PMID: 34923109
Neurobiol Dis. 2021 Dec 16;163:105587. doi: 10.1016/j.nbd.2021.105587. Epub 2021 Dec 16.

Monoamine neurotransmitter abundance affects motor control, emotion, and cognitive function and is regulated by monoamine oxidases. Among these, Monoamine oxidase A (MAOA) catalyzes the degradation of dopamine, norepinephrine, and serotonin into their inactive metabolites. Loss-of-function mutations in the X-linked...

Cite
van Rhijn JR, Shi Y, Bormann M, et al. Brunner syndrome associated MAOA mutations result in NMDAR hyperfunction and increased network activity in human dopaminergic neurons. Neurobiol Dis. 2021;163:105587doi: 10.1016/j.nbd.2021.105587.
van Rhijn, J. R., Shi, Y., Bormann, M., Mossink, B., Frega, M., Recaioglu, H., Hakobjan, M., Klein Gunnewiek, T., Schoenmaker, C., Palmer, E., Faivre, L., Kittel-Schneider, S., Schubert, D., Brunner, H., Franke, B., & Nadif Kasri, N. (2021). Brunner syndrome associated MAOA mutations result in NMDAR hyperfunction and increased network activity in human dopaminergic neurons. Neurobiology of disease, 163105587. https://doi.org/10.1016/j.nbd.2021.105587
van Rhijn, Jon-Ruben, et al. "Brunner syndrome associated MAOA mutations result in NMDAR hyperfunction and increased network activity in human dopaminergic neurons." Neurobiology of disease vol. 163 (2021): 105587. doi: https://doi.org/10.1016/j.nbd.2021.105587
van Rhijn JR, Shi Y, Bormann M, Mossink B, Frega M, Recaioglu H, Hakobjan M, Klein Gunnewiek T, Schoenmaker C, Palmer E, Faivre L, Kittel-Schneider S, Schubert D, Brunner H, Franke B, Nadif Kasri N. Brunner syndrome associated MAOA mutations result in NMDAR hyperfunction and increased network activity in human dopaminergic neurons. Neurobiol Dis. 2021 Dec 16;163:105587. doi: 10.1016/j.nbd.2021.105587. Epub 2021 Dec 16. PMID: 34923109.
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C-reactive protein concentration in bipolar disorder: association with genetic variants.

International journal of bipolar disorders

Evers AK, Veeh J, McNeill R, Reif A, Kittel-Schneider S.
PMID: 31788733
Int J Bipolar Disord. 2019 Dec 02;7(1):26. doi: 10.1186/s40345-019-0162-z.

BACKGROUND: Several recent studies have investigated the role of C-reactive protein (CRP) in bipolar disorder (BD), but few studies have directly investigated the interaction between CRP genetic variants and peripheral CRP concentration across different phases of BD. In this...

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Evers AK, Veeh J, McNeill R, et al. C-reactive protein concentration in bipolar disorder: association with genetic variants. Int J Bipolar Disord. 2019;7(1):26doi: 10.1186/s40345-019-0162-z.
Evers, A. K., Veeh, J., McNeill, R., Reif, A., & Kittel-Schneider, S. (2019). C-reactive protein concentration in bipolar disorder: association with genetic variants. International journal of bipolar disorders, 7(1), 26. https://doi.org/10.1186/s40345-019-0162-z
Evers, Ann-Kristin, et al. "C-reactive protein concentration in bipolar disorder: association with genetic variants." International journal of bipolar disorders vol. 7,1 (2019): 26. doi: https://doi.org/10.1186/s40345-019-0162-z
Evers AK, Veeh J, McNeill R, Reif A, Kittel-Schneider S. C-reactive protein concentration in bipolar disorder: association with genetic variants. Int J Bipolar Disord. 2019 Dec 02;7(1):26. doi: 10.1186/s40345-019-0162-z. PMID: 31788733; PMCID: PMC6885457.
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Rationale, Mission and Vision for a National Centre of Affective Disorders in Germany.

Pharmacopsychiatry

Reif A, Baune BT, Deckert J, Juckel G, Kittel-Schneider S, Kircher T, Kornhuber J, Rupprecht R, Bauer M.
PMID: 34921380
Pharmacopsychiatry. 2021 Dec 17; doi: 10.1055/a-1697-5854. Epub 2021 Dec 17.

Affective disorders are common, complex disorders representing one of the major challenges to global health in the 21st century. To mitigate the burden of disease, substantial public health efforts need to be undertaken since research on the causes and...

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Reif A, Baune BT, Deckert J, et al. Rationale, Mission and Vision for a National Centre of Affective Disorders in Germany. Pharmacopsychiatry. 2021;doi: 10.1055/a-1697-5854.
Reif, A., Baune, B. T., Deckert, J., Juckel, G., Kittel-Schneider, S., Kircher, T., Kornhuber, J., Rupprecht, R., & Bauer, M. (2021). Rationale, Mission and Vision for a National Centre of Affective Disorders in Germany. Pharmacopsychiatry, . https://doi.org/10.1055/a-1697-5854
Reif, Andreas, et al. "Rationale, Mission and Vision for a National Centre of Affective Disorders in Germany." Pharmacopsychiatry vol. (2021). doi: https://doi.org/10.1055/a-1697-5854
Reif A, Baune BT, Deckert J, Juckel G, Kittel-Schneider S, Kircher T, Kornhuber J, Rupprecht R, Bauer M. Rationale, Mission and Vision for a National Centre of Affective Disorders in Germany. Pharmacopsychiatry. 2021 Dec 17; doi: 10.1055/a-1697-5854. Epub 2021 Dec 17. PMID: 34921380.
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Brunner syndrome associated MAOA mutations result in NMDAR hyperfunction and increased network activity in human dopaminergic neurons.

Neurobiology of disease

van Rhijn JR, Shi Y, Bormann M, Mossink B, Frega M, Recaioglu H, Hakobjan M, Klein Gunnewiek T, Schoenmaker C, Palmer E, Faivre L, Kittel-Schneider S, Schubert D, Brunner H, Franke B, Nadif Kasri N.
PMID: 34923109
Neurobiol Dis. 2021 Dec 16;163:105587. doi: 10.1016/j.nbd.2021.105587. Epub 2021 Dec 16.

Monoamine neurotransmitter abundance affects motor control, emotion, and cognitive function and is regulated by monoamine oxidases. Among these, Monoamine oxidase A (MAOA) catalyzes the degradation of dopamine, norepinephrine, and serotonin into their inactive metabolites. Loss-of-function mutations in the X-linked...

Cite
van Rhijn JR, Shi Y, Bormann M, et al. Brunner syndrome associated MAOA mutations result in NMDAR hyperfunction and increased network activity in human dopaminergic neurons. Neurobiol Dis. 2021;163:105587doi: 10.1016/j.nbd.2021.105587.
van Rhijn, J. R., Shi, Y., Bormann, M., Mossink, B., Frega, M., Recaioglu, H., Hakobjan, M., Klein Gunnewiek, T., Schoenmaker, C., Palmer, E., Faivre, L., Kittel-Schneider, S., Schubert, D., Brunner, H., Franke, B., & Nadif Kasri, N. (2021). Brunner syndrome associated MAOA mutations result in NMDAR hyperfunction and increased network activity in human dopaminergic neurons. Neurobiology of disease, 163105587. https://doi.org/10.1016/j.nbd.2021.105587
van Rhijn, Jon-Ruben, et al. "Brunner syndrome associated MAOA mutations result in NMDAR hyperfunction and increased network activity in human dopaminergic neurons." Neurobiology of disease vol. 163 (2021): 105587. doi: https://doi.org/10.1016/j.nbd.2021.105587
van Rhijn JR, Shi Y, Bormann M, Mossink B, Frega M, Recaioglu H, Hakobjan M, Klein Gunnewiek T, Schoenmaker C, Palmer E, Faivre L, Kittel-Schneider S, Schubert D, Brunner H, Franke B, Nadif Kasri N. Brunner syndrome associated MAOA mutations result in NMDAR hyperfunction and increased network activity in human dopaminergic neurons. Neurobiol Dis. 2021 Dec 16;163:105587. doi: 10.1016/j.nbd.2021.105587. Epub 2021 Dec 16. PMID: 34923109.
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A pilot study of multilevel analysis of BDNF in paternal and maternal perinatal depression.

Archives of women's mental health

Kittel-Schneider S, Davidova P, Kalok M, Essel C, Ahmed FB, Kingeter Y, Matentzoglu M, Leutritz AL, Kersken K, Koreny C, Weber H, Kollert L, McNeill RV, Reif A, Bahlmann F, Trautmann-Villalba P.
PMID: 34989854
Arch Womens Ment Health. 2022 Jan 06; doi: 10.1007/s00737-021-01197-2. Epub 2022 Jan 06.

Depression in the perinatal period is common in mothers worldwide. Emerging research indicates that fathers are also at risk of developing perinatal depression. However, knowledge regarding biological risk factors and pathophysiological mechanisms of perinatal depression is still scarce, particularly...

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Kittel-Schneider S, Davidova P, Kalok M, et al. A pilot study of multilevel analysis of BDNF in paternal and maternal perinatal depression. Arch Womens Ment Health. 2022;doi: 10.1007/s00737-021-01197-2.
Kittel-Schneider, S., Davidova, P., Kalok, M., Essel, C., Ahmed, F. B., Kingeter, Y., Matentzoglu, M., Leutritz, A. L., Kersken, K., Koreny, C., Weber, H., Kollert, L., McNeill, R. V., Reif, A., Bahlmann, F., & Trautmann-Villalba, P. (2022). A pilot study of multilevel analysis of BDNF in paternal and maternal perinatal depression. Archives of women's mental health, . https://doi.org/10.1007/s00737-021-01197-2
Kittel-Schneider, Sarah, et al. "A pilot study of multilevel analysis of BDNF in paternal and maternal perinatal depression." Archives of women's mental health vol. (2022). doi: https://doi.org/10.1007/s00737-021-01197-2
Kittel-Schneider S, Davidova P, Kalok M, Essel C, Ahmed FB, Kingeter Y, Matentzoglu M, Leutritz AL, Kersken K, Koreny C, Weber H, Kollert L, McNeill RV, Reif A, Bahlmann F, Trautmann-Villalba P. A pilot study of multilevel analysis of BDNF in paternal and maternal perinatal depression. Arch Womens Ment Health. 2022 Jan 06; doi: 10.1007/s00737-021-01197-2. Epub 2022 Jan 06. PMID: 34989854.
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Brunner syndrome associated MAOA mutations result in NMDAR hyperfunction and increased network activity in human dopaminergic neurons.

Neurobiology of disease

van Rhijn JR, Shi Y, Bormann M, Mossink B, Frega M, Recaioglu H, Hakobjan M, Klein Gunnewiek T, Schoenmaker C, Palmer E, Faivre L, Kittel-Schneider S, Schubert D, Brunner H, Franke B, Nadif Kasri N.
PMID: 34923109
Neurobiol Dis. 2021 Dec 16;163:105587. doi: 10.1016/j.nbd.2021.105587. Epub 2021 Dec 16.

Monoamine neurotransmitter abundance affects motor control, emotion, and cognitive function and is regulated by monoamine oxidases. Among these, Monoamine oxidase A (MAOA) catalyzes the degradation of dopamine, norepinephrine, and serotonin into their inactive metabolites. Loss-of-function mutations in the X-linked...

Cite
van Rhijn JR, Shi Y, Bormann M, et al. Brunner syndrome associated MAOA mutations result in NMDAR hyperfunction and increased network activity in human dopaminergic neurons. Neurobiol Dis. 2021;163:105587doi: 10.1016/j.nbd.2021.105587.
van Rhijn, J. R., Shi, Y., Bormann, M., Mossink, B., Frega, M., Recaioglu, H., Hakobjan, M., Klein Gunnewiek, T., Schoenmaker, C., Palmer, E., Faivre, L., Kittel-Schneider, S., Schubert, D., Brunner, H., Franke, B., & Nadif Kasri, N. (2021). Brunner syndrome associated MAOA mutations result in NMDAR hyperfunction and increased network activity in human dopaminergic neurons. Neurobiology of disease, 163105587. https://doi.org/10.1016/j.nbd.2021.105587
van Rhijn, Jon-Ruben, et al. "Brunner syndrome associated MAOA mutations result in NMDAR hyperfunction and increased network activity in human dopaminergic neurons." Neurobiology of disease vol. 163 (2021): 105587. doi: https://doi.org/10.1016/j.nbd.2021.105587
van Rhijn JR, Shi Y, Bormann M, Mossink B, Frega M, Recaioglu H, Hakobjan M, Klein Gunnewiek T, Schoenmaker C, Palmer E, Faivre L, Kittel-Schneider S, Schubert D, Brunner H, Franke B, Nadif Kasri N. Brunner syndrome associated MAOA mutations result in NMDAR hyperfunction and increased network activity in human dopaminergic neurons. Neurobiol Dis. 2021 Dec 16;163:105587. doi: 10.1016/j.nbd.2021.105587. Epub 2021 Dec 16. PMID: 34923109.
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[No title available]

MMW Fortschritte der Medizin

Kittel-Schneider S.
PMID: 34811679
MMW Fortschr Med. 2021 Nov;163:30-31. doi: 10.1007/s15006-021-0564-6.

No abstract available.

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Kittel-Schneider S. Kontrazeption wohl ohne Einfluss auf depressive Symptome. . MMW Fortschr Med. 2021;163:30-31doi: 10.1007/s15006-021-0564-6.
Kittel-Schneider, S. (2021). Kontrazeption wohl ohne Einfluss auf depressive Symptome. . MMW Fortschritte der Medizin, 16330-31. https://doi.org/10.1007/s15006-021-0564-6
Kittel-Schneider, Sarah. "Kontrazeption wohl ohne Einfluss auf depressive Symptome." . MMW Fortschritte der Medizin vol. 163 (2021): 30-31. doi: https://doi.org/10.1007/s15006-021-0564-6
Kittel-Schneider S. Kontrazeption wohl ohne Einfluss auf depressive Symptome. . MMW Fortschr Med. 2021 Nov;163:30-31. doi: 10.1007/s15006-021-0564-6. German. PMID: 34811679.
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Transdiagnostic comparison of visual working memory capacity in bipolar disorder and schizophrenia.

International journal of bipolar disorders

Barnes-Scheufler CV, Passow C, Rösler L, Mayer JS, Oertel V, Kittel-Schneider S, Matura S, Reif A, Bittner RA.
PMID: 33797645
Int J Bipolar Disord. 2021 Apr 02;9(1):12. doi: 10.1186/s40345-020-00217-x.

BACKGROUND: Impaired working memory is a core cognitive deficit in both bipolar disorder and schizophrenia. Its study might yield crucial insights into the underpinnings of both disorders on the cognitive and neurophysiological level. Visual working memory capacity is a...

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Barnes-Scheufler CV, Passow C, Rösler L, et al. Transdiagnostic comparison of visual working memory capacity in bipolar disorder and schizophrenia. Int J Bipolar Disord. 2021;9(1):12doi: 10.1186/s40345-020-00217-x.
Barnes-Scheufler, C. V., Passow, C., Rösler, L., Mayer, J. S., Oertel, V., Kittel-Schneider, S., Matura, S., Reif, A., & Bittner, R. A. (2021). Transdiagnostic comparison of visual working memory capacity in bipolar disorder and schizophrenia. International journal of bipolar disorders, 9(1), 12. https://doi.org/10.1186/s40345-020-00217-x
Barnes-Scheufler, Catherine V, et al. "Transdiagnostic comparison of visual working memory capacity in bipolar disorder and schizophrenia." International journal of bipolar disorders vol. 9,1 (2021): 12. doi: https://doi.org/10.1186/s40345-020-00217-x
Barnes-Scheufler CV, Passow C, Rösler L, Mayer JS, Oertel V, Kittel-Schneider S, Matura S, Reif A, Bittner RA. Transdiagnostic comparison of visual working memory capacity in bipolar disorder and schizophrenia. Int J Bipolar Disord. 2021 Apr 02;9(1):12. doi: 10.1186/s40345-020-00217-x. PMID: 33797645; PMCID: PMC8018920.
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Improving early recognition and intervention in people at increased risk for the development of bipolar disorder: study protocol of a prospective-longitudinal, naturalistic cohort study (Early-BipoLife).

International journal of bipolar disorders

Pfennig A, Leopold K, Martini J, Boehme A, Lambert M, Stamm T, Bermpohl F, Reif A, Kittel-Schneider S, Juckel G, Fallgatter AJ, Kircher T, Jansen A, Pfeiffer S, Berndt C, Rottmann-Wolf M, Sauer C, Ritter P, Correll CU, Bechdolf A, Falkenberg I, Bauer M.
PMID: 32607662
Int J Bipolar Disord. 2020 Jul 01;8(1):22. doi: 10.1186/s40345-020-00183-4.

BACKGROUND: Bipolar disorders (BD) belong to the most severe mental disorders, characterized by an early onset and recurrent, severe episodes or a chronic course with poor psychosocial functioning in a proportion of patients. Many patients with BD experience substantial...

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Pfennig A, Leopold K, Martini J, et al. Improving early recognition and intervention in people at increased risk for the development of bipolar disorder: study protocol of a prospective-longitudinal, naturalistic cohort study (Early-BipoLife). Int J Bipolar Disord. 2020;8(1):22doi: 10.1186/s40345-020-00183-4.
Pfennig, A., Leopold, K., Martini, J., Boehme, A., Lambert, M., Stamm, T., Bermpohl, F., Reif, A., Kittel-Schneider, S., Juckel, G., Fallgatter, A. J., Kircher, T., Jansen, A., Pfeiffer, S., Berndt, C., Rottmann-Wolf, M., Sauer, C., Ritter, P., Correll, C. U., Bechdolf, A., Falkenberg, I., & Bauer, M. (2020). Improving early recognition and intervention in people at increased risk for the development of bipolar disorder: study protocol of a prospective-longitudinal, naturalistic cohort study (Early-BipoLife). International journal of bipolar disorders, 8(1), 22. https://doi.org/10.1186/s40345-020-00183-4
Pfennig, Andrea, et al. "Improving early recognition and intervention in people at increased risk for the development of bipolar disorder: study protocol of a prospective-longitudinal, naturalistic cohort study (Early-BipoLife)." International journal of bipolar disorders vol. 8,1 (2020): 22. doi: https://doi.org/10.1186/s40345-020-00183-4
Pfennig A, Leopold K, Martini J, Boehme A, Lambert M, Stamm T, Bermpohl F, Reif A, Kittel-Schneider S, Juckel G, Fallgatter AJ, Kircher T, Jansen A, Pfeiffer S, Berndt C, Rottmann-Wolf M, Sauer C, Ritter P, Correll CU, Bechdolf A, Falkenberg I, Bauer M. Improving early recognition and intervention in people at increased risk for the development of bipolar disorder: study protocol of a prospective-longitudinal, naturalistic cohort study (Early-BipoLife). Int J Bipolar Disord. 2020 Jul 01;8(1):22. doi: 10.1186/s40345-020-00183-4. PMID: 32607662; PMCID: PMC7326843.
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Analysis of the Influence of microRNAs in Lithium Response in Bipolar Disorder.

Frontiers in psychiatry

Reinbold CS, Forstner AJ, Hecker J, Fullerton JM, Hoffmann P, Hou L, Heilbronner U, Degenhardt F, Adli M, Akiyama K, Akula N, Ardau R, Arias B, Backlund L, Benabarre A, Bengesser S, Bhattacharjee AK, Biernacka JM, Birner A, Marie-Claire C, Cervantes P, Chen GB, Chen HC, Chillotti C, Clark SR, Colom F, Cousins DA, Cruceanu C, Czerski PM, Dayer A, Étain B, Falkai P, Frisén L, Gard S, Garnham JS, Goes FS, Grof P, Gruber O, Hashimoto R, Hauser J, Herms S, Jamain S, Jiménez E, Kahn JP, Kassem L, Kittel-Schneider S, Kliwicki S, König B, Kusumi I, Lackner N, Laje G, Landén M, Lavebratt C, Leboyer M, Leckband SG, López Jaramillo CA, MacQueen G, Manchia M, Martinsson L, Mattheisen M, McCarthy MJ, McElroy SL, Mitjans M, Mondimore FM, Monteleone P, Nievergelt CM, Ösby U, Ozaki N, Perlis RH, Pfennig A, Reich-Erkelenz D, Rouleau GA, Schofield PR, Schubert KO, Schweizer BW, Seemüller F, Severino G, Shekhtman T, Shilling PD, Shimoda K, Simhandl C, Slaney CM, Smoller JW, Squassina A, Stamm TJ, Stopkova P, Tighe SK, Tortorella A, Turecki G, Volkert J, Witt SH, Wright AJ, Young LT, Zandi PP, Potash JB, DePaulo JR, Bauer M, Reininghaus E, Novák T, Aubry JM, Maj M, Baune BT, Mitchell PB, Vieta E, Frye MA, Rybakowski JK, Kuo PH, Kato T, Grigoroiu-Serbanescu M, Reif A, Del Zompo M, Bellivier F, Schalling M, Wray NR, Kelsoe JR, Alda M, McMahon FJ, Schulze TG, Rietschel M, Nöthen MM, Cichon S.
PMID: 29904359
Front Psychiatry. 2018 May 31;9:207. doi: 10.3389/fpsyt.2018.00207. eCollection 2018.

Bipolar disorder (BD) is a common, highly heritable neuropsychiatric disease characterized by recurrent episodes of mania and depression. Lithium is the best-established long-term treatment for BD, even though individual response is highly variable. Evidence suggests that some of this...

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Reinbold CS, Forstner AJ, Hecker J, et al. Analysis of the Influence of microRNAs in Lithium Response in Bipolar Disorder. Front Psychiatry. 2018;9:207doi: 10.3389/fpsyt.2018.00207.
Reinbold, C. S., Forstner, A. J., Hecker, J., Fullerton, J. M., Hoffmann, P., Hou, L., Heilbronner, U., Degenhardt, F., Adli, M., Akiyama, K., Akula, N., Ardau, R., Arias, B., Backlund, L., Benabarre, A., Bengesser, S., Bhattacharjee, A. K., Biernacka, J. M., Birner, A., Marie-Claire, C., Cervantes, P., Chen, G. B., Chen, H. C., Chillotti, C., Clark, S. R., Colom, F., Cousins, D. A., Cruceanu, C., Czerski, P. M., Dayer, A., Étain, B., Falkai, P., Frisén, L., Gard, S., Garnham, J. S., Goes, F. S., Grof, P., Gruber, O., Hashimoto, R., Hauser, J., Herms, S., Jamain, S., Jiménez, E., Kahn, J. P., Kassem, L., Kittel-Schneider, S., Kliwicki, S., König, B., Kusumi, I., Lackner, N., Laje, G., Landén, M., Lavebratt, C., Leboyer, M., Leckband, S. G., López Jaramillo, C. A., MacQueen, G., Manchia, M., Martinsson, L., Mattheisen, M., McCarthy, M. J., McElroy, S. L., Mitjans, M., Mondimore, F. M., Monteleone, P., Nievergelt, C. M., Ösby, U., Ozaki, N., Perlis, R. H., Pfennig, A., Reich-Erkelenz, D., Rouleau, G. A., Schofield, P. R., Schubert, K. O., Schweizer, B. W., Seemüller, F., Severino, G., Shekhtman, T., Shilling, P. D., Shimoda, K., Simhandl, C., Slaney, C. M., Smoller, J. W., Squassina, A., Stamm, T. J., Stopkova, P., Tighe, S. K., Tortorella, A., Turecki, G., Volkert, J., Witt, S. H., Wright, A. J., Young, L. T., Zandi, P. P., Potash, J. B., DePaulo, J. R., Bauer, M., Reininghaus, E., Novák, T., Aubry, J. M., Maj, M., Baune, B. T., Mitchell, P. B., Vieta, E., Frye, M. A., Rybakowski, J. K., Kuo, P. H., Kato, T., Grigoroiu-Serbanescu, M., Reif, A., Del Zompo, M., Bellivier, F., Schalling, M., Wray, N. R., Kelsoe, J. R., Alda, M., McMahon, F. J., Schulze, T. G., Rietschel, M., Nöthen, M. M., & Cichon, S. (2018). Analysis of the Influence of microRNAs in Lithium Response in Bipolar Disorder. Frontiers in psychiatry, 9207. https://doi.org/10.3389/fpsyt.2018.00207
Reinbold, Céline S, et al. "Analysis of the Influence of microRNAs in Lithium Response in Bipolar Disorder." Frontiers in psychiatry vol. 9 (2018): 207. doi: https://doi.org/10.3389/fpsyt.2018.00207
Reinbold CS, Forstner AJ, Hecker J, Fullerton JM, Hoffmann P, Hou L, Heilbronner U, Degenhardt F, Adli M, Akiyama K, Akula N, Ardau R, Arias B, Backlund L, Benabarre A, Bengesser S, Bhattacharjee AK, Biernacka JM, Birner A, Marie-Claire C, Cervantes P, Chen GB, Chen HC, Chillotti C, Clark SR, Colom F, Cousins DA, Cruceanu C, Czerski PM, Dayer A, Étain B, Falkai P, Frisén L, Gard S, Garnham JS, Goes FS, Grof P, Gruber O, Hashimoto R, Hauser J, Herms S, Jamain S, Jiménez E, Kahn JP, Kassem L, Kittel-Schneider S, Kliwicki S, König B, Kusumi I, Lackner N, Laje G, Landén M, Lavebratt C, Leboyer M, Leckband SG, López Jaramillo CA, MacQueen G, Manchia M, Martinsson L, Mattheisen M, McCarthy MJ, McElroy SL, Mitjans M, Mondimore FM, Monteleone P, Nievergelt CM, Ösby U, Ozaki N, Perlis RH, Pfennig A, Reich-Erkelenz D, Rouleau GA, Schofield PR, Schubert KO, Schweizer BW, Seemüller F, Severino G, Shekhtman T, Shilling PD, Shimoda K, Simhandl C, Slaney CM, Smoller JW, Squassina A, Stamm TJ, Stopkova P, Tighe SK, Tortorella A, Turecki G, Volkert J, Witt SH, Wright AJ, Young LT, Zandi PP, Potash JB, DePaulo JR, Bauer M, Reininghaus E, Novák T, Aubry JM, Maj M, Baune BT, Mitchell PB, Vieta E, Frye MA, Rybakowski JK, Kuo PH, Kato T, Grigoroiu-Serbanescu M, Reif A, Del Zompo M, Bellivier F, Schalling M, Wray NR, Kelsoe JR, Alda M, McMahon FJ, Schulze TG, Rietschel M, Nöthen MM, Cichon S. Analysis of the Influence of microRNAs in Lithium Response in Bipolar Disorder. Front Psychiatry. 2018 May 31;9:207. doi: 10.3389/fpsyt.2018.00207. eCollection 2018. PMID: 29904359; PMCID: PMC5991073.
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