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Showing 1 to 12 of 1574 entries
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[The current status of nursing studies at Sangyo Medical University].

Kango tenbo. The Japanese journal of nursing science

Koga K, Toyozawa E.
PMID: 3853683
Kango Tenbo. 1985 Dec;10(13):1292-5.

No abstract available.

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Koga K, Toyozawa E. [The current status of nursing studies at Sangyo Medical University]. Kango Tenbo. 1985;10(13):1292-5
Koga, K., & Toyozawa, E. (1985). [The current status of nursing studies at Sangyo Medical University]. Kango tenbo. The Japanese journal of nursing science, 10(13), 1292-5.
Koga, K, and Toyozawa, E. "[The current status of nursing studies at Sangyo Medical University]." Kango tenbo. The Japanese journal of nursing science vol. 10,13 (1985): 1292-5.
Koga K, Toyozawa E. [The current status of nursing studies at Sangyo Medical University]. Kango Tenbo. 1985 Dec;10(13):1292-5. Japanese. PMID: 3853683.
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[Theories of magnifying radiography].

Rinsho hoshasen. Clinical radiography

Takahashi S, Sasaki T, Takuma S, Koga S.
PMID: 5204656
Rinsho Hoshasen. 1971 Jan;16(1):1-6.

No abstract available.

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Takahashi S, Sasaki T, Takuma S, et al. [Theories of magnifying radiography]. Rinsho Hoshasen. 1971;16(1):1-6
Takahashi, S., Sasaki, T., Takuma, S., & Koga, S. (1971). [Theories of magnifying radiography]. Rinsho hoshasen. Clinical radiography, 16(1), 1-6.
Takahashi, S, et al. "[Theories of magnifying radiography]." Rinsho hoshasen. Clinical radiography vol. 16,1 (1971): 1-6.
Takahashi S, Sasaki T, Takuma S, Koga S. [Theories of magnifying radiography]. Rinsho Hoshasen. 1971 Jan;16(1):1-6. Japanese. PMID: 5204656.
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Chemical Modification of Cellulose Nanofibers for the Production of Highly Thermal Resistant and Optically Transparent Nanopaper for Paper Devices.

ACS applied materials & interfaces

Yagyu H, Saito T, Isogai A, Koga H, Nogi M.
PMID: 26402324
ACS Appl Mater Interfaces. 2015 Oct 07;7(39):22012-7. doi: 10.1021/acsami.5b06915. Epub 2015 Sep 24.

Optically transparent cellulose nanopaper is one of the best candidate substrates for flexible electronics. Some types of cellulose nanopaper are made of mechanically or chemically modified cellulose nanofibers. Among these, nanopapers produced from chemically modified cellulose nanofibers are the...

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Yagyu H, Saito T, Isogai A, et al. Chemical Modification of Cellulose Nanofibers for the Production of Highly Thermal Resistant and Optically Transparent Nanopaper for Paper Devices. ACS Appl Mater Interfaces. 2015;7(39):22012-7doi: 10.1021/acsami.5b06915.
Yagyu, H., Saito, T., Isogai, A., Koga, H., & Nogi, M. (2015). Chemical Modification of Cellulose Nanofibers for the Production of Highly Thermal Resistant and Optically Transparent Nanopaper for Paper Devices. ACS applied materials & interfaces, 7(39), 22012-7. https://doi.org/10.1021/acsami.5b06915
Yagyu, Hitomi, et al. "Chemical Modification of Cellulose Nanofibers for the Production of Highly Thermal Resistant and Optically Transparent Nanopaper for Paper Devices." ACS applied materials & interfaces vol. 7,39 (2015): 22012-7. doi: https://doi.org/10.1021/acsami.5b06915
Yagyu H, Saito T, Isogai A, Koga H, Nogi M. Chemical Modification of Cellulose Nanofibers for the Production of Highly Thermal Resistant and Optically Transparent Nanopaper for Paper Devices. ACS Appl Mater Interfaces. 2015 Oct 07;7(39):22012-7. doi: 10.1021/acsami.5b06915. Epub 2015 Sep 24. PMID: 26402324.
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Targeting hyaluronan for the treatment of pancreatic ductal adenocarcinoma.

Acta pharmaceutica Sinica. B

Sato N, Cheng XB, Kohi S, Koga A, Hirata K.
PMID: 27006892
Acta Pharm Sin B. 2016 Mar;6(2):101-5. doi: 10.1016/j.apsb.2016.01.002. Epub 2016 Jan 21.

Progression of cancer is often associated with interactions between cancer cells and extracellular matrix (ECM) surrounding them. Increasing evidence has suggested that accumulation of hyaluronan (HA), a major component of ECM, provides a favorable microenvironment for cancer progression. Pancreatic...

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Sato N, Cheng XB, Kohi S, et al. Targeting hyaluronan for the treatment of pancreatic ductal adenocarcinoma. Acta Pharm Sin B. 2016;6(2):101-5doi: 10.1016/j.apsb.2016.01.002.
Sato, N., Cheng, X. B., Kohi, S., Koga, A., & Hirata, K. (2016). Targeting hyaluronan for the treatment of pancreatic ductal adenocarcinoma. Acta pharmaceutica Sinica. B, 6(2), 101-5. https://doi.org/10.1016/j.apsb.2016.01.002
Sato, Norihiro, et al. "Targeting hyaluronan for the treatment of pancreatic ductal adenocarcinoma." Acta pharmaceutica Sinica. B vol. 6,2 (2016): 101-5. doi: https://doi.org/10.1016/j.apsb.2016.01.002
Sato N, Cheng XB, Kohi S, Koga A, Hirata K. Targeting hyaluronan for the treatment of pancreatic ductal adenocarcinoma. Acta Pharm Sin B. 2016 Mar;6(2):101-5. doi: 10.1016/j.apsb.2016.01.002. Epub 2016 Jan 21. PMID: 27006892; PMCID: PMC4788704.
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Acute diffuse peritonitis due to spontaneous rupture of a primary gastrointestinal stromal tumor of the jejunum: A case report.

International journal of surgery case reports

Sato K, Tazawa H, Fujisaki S, Fukuhara S, Imaoka K, Hirata Y, Takahashi M, Fukuda S, Kuga Y, Nishida T, Sakimoto H.
PMID: 28888914
Int J Surg Case Rep. 2017;39:288-292. doi: 10.1016/j.ijscr.2017.08.041. Epub 2017 Aug 23.

INTRODUCTION: Gastrointestinal stromal tumors (GISTs) are the most common mesenchymal tumors of the gastrointestinal tract. Overt peritonitis caused by GIST rupture is very uncommon. Three types of GIST rupture have been described: closed perforation due to abscess (abscess type),...

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Sato K, Tazawa H, Fujisaki S, et al. Acute diffuse peritonitis due to spontaneous rupture of a primary gastrointestinal stromal tumor of the jejunum: A case report. Int J Surg Case Rep. 2017;39:288-292doi: 10.1016/j.ijscr.2017.08.041.
Sato, K., Tazawa, H., Fujisaki, S., Fukuhara, S., Imaoka, K., Hirata, Y., Takahashi, M., Fukuda, S., Kuga, Y., Nishida, T., & Sakimoto, H. (2017). Acute diffuse peritonitis due to spontaneous rupture of a primary gastrointestinal stromal tumor of the jejunum: A case report. International journal of surgery case reports, 39288-292. https://doi.org/10.1016/j.ijscr.2017.08.041
Sato, Koki, et al. "Acute diffuse peritonitis due to spontaneous rupture of a primary gastrointestinal stromal tumor of the jejunum: A case report." International journal of surgery case reports vol. 39 (2017): 288-292. doi: https://doi.org/10.1016/j.ijscr.2017.08.041
Sato K, Tazawa H, Fujisaki S, Fukuhara S, Imaoka K, Hirata Y, Takahashi M, Fukuda S, Kuga Y, Nishida T, Sakimoto H. Acute diffuse peritonitis due to spontaneous rupture of a primary gastrointestinal stromal tumor of the jejunum: A case report. Int J Surg Case Rep. 2017;39:288-292. doi: 10.1016/j.ijscr.2017.08.041. Epub 2017 Aug 23. PMID: 28888914; PMCID: PMC5596258.
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Effects of H.

Physical chemistry chemical physics : PCCP

Koga Y, Miki K, Nishikawa K.
PMID: 28975179
Phys Chem Chem Phys. 2017 Oct 18;19(40):27413-27420. doi: 10.1039/c7cp05519f.

We applied what we call the "1-propanol (1P) probing methodology" on the effects of H

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Koga Y, Miki K, Nishikawa K. Effects of H. Phys Chem Chem Phys. 2017;19(40):27413-27420doi: 10.1039/c7cp05519f.
Koga, Y., Miki, K., & Nishikawa, K. (2017). Effects of H. Physical chemistry chemical physics : PCCP, 19(40), 27413-27420. https://doi.org/10.1039/c7cp05519f
Koga, Yoshikata, et al. "Effects of H." Physical chemistry chemical physics : PCCP vol. 19,40 (2017): 27413-27420. doi: https://doi.org/10.1039/c7cp05519f
Koga Y, Miki K, Nishikawa K. Effects of H. Phys Chem Chem Phys. 2017 Oct 18;19(40):27413-27420. doi: 10.1039/c7cp05519f. PMID: 28975179.
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Pancreatic DCLK1 marks quiescent but oncogenic progenitors: a possible link to neuroendocrine tumors.

Stem cell investigation

Koga H, Ikezono Y, Torimura T.
PMID: 27668244
Stem Cell Investig. 2016 Aug 12;3:37. doi: 10.21037/sci.2016.08.03. eCollection 2016.

No abstract available.

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Koga H, Ikezono Y, Torimura T. Pancreatic DCLK1 marks quiescent but oncogenic progenitors: a possible link to neuroendocrine tumors. Stem Cell Investig. 2016;3:37doi: 10.21037/sci.2016.08.03.
Koga, H., Ikezono, Y., & Torimura, T. (2016). Pancreatic DCLK1 marks quiescent but oncogenic progenitors: a possible link to neuroendocrine tumors. Stem cell investigation, 337. https://doi.org/10.21037/sci.2016.08.03
Koga, Hironori, et al. "Pancreatic DCLK1 marks quiescent but oncogenic progenitors: a possible link to neuroendocrine tumors." Stem cell investigation vol. 3 (2016): 37. doi: https://doi.org/10.21037/sci.2016.08.03
Koga H, Ikezono Y, Torimura T. Pancreatic DCLK1 marks quiescent but oncogenic progenitors: a possible link to neuroendocrine tumors. Stem Cell Investig. 2016 Aug 12;3:37. doi: 10.21037/sci.2016.08.03. eCollection 2016. PMID: 27668244; PMCID: PMC5014849.
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Dielectric Mismatch Mediates Carrier Mobility in Organic-Intercalated Layered TiS2.

Nano letters

Wan C, Kodama Y, Kondo M, Sasai R, Qian X, Gu X, Koga K, Yabuki K, Yang R, Koumoto K.
PMID: 26308495
Nano Lett. 2015 Oct 14;15(10):6302-8. doi: 10.1021/acs.nanolett.5b01013. Epub 2015 Sep 01.

The dielectric constant is a key parameter that determines both optical and electronic properties of materials. It is desirable to tune electronic properties though dielectric engineering approach. Here, we present a systematic approach to tune carrier mobilities of hybrid...

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Wan C, Kodama Y, Kondo M, et al. Dielectric Mismatch Mediates Carrier Mobility in Organic-Intercalated Layered TiS2. Nano Lett. 2015;15(10):6302-8doi: 10.1021/acs.nanolett.5b01013.
Wan, C., Kodama, Y., Kondo, M., Sasai, R., Qian, X., Gu, X., Koga, K., Yabuki, K., Yang, R., & Koumoto, K. (2015). Dielectric Mismatch Mediates Carrier Mobility in Organic-Intercalated Layered TiS2. Nano letters, 15(10), 6302-8. https://doi.org/10.1021/acs.nanolett.5b01013
Wan, Chunlei, et al. "Dielectric Mismatch Mediates Carrier Mobility in Organic-Intercalated Layered TiS2." Nano letters vol. 15,10 (2015): 6302-8. doi: https://doi.org/10.1021/acs.nanolett.5b01013
Wan C, Kodama Y, Kondo M, Sasai R, Qian X, Gu X, Koga K, Yabuki K, Yang R, Koumoto K. Dielectric Mismatch Mediates Carrier Mobility in Organic-Intercalated Layered TiS2. Nano Lett. 2015 Oct 14;15(10):6302-8. doi: 10.1021/acs.nanolett.5b01013. Epub 2015 Sep 01. PMID: 26308495.
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Entrapment of middle cluneal nerves as an unknown cause of low back pain.

World journal of orthopedics

Aota Y.
PMID: 27004164
World J Orthop. 2016 Mar 18;7(3):167-70. doi: 10.5312/wjo.v7.i3.167. eCollection 2016 Mar 18.

Entrapment of middle cluneal nerves induces low back pain and leg symptoms. The middle cluneal nerves can become spontaneously entrapped where this nerve pass under the long posterior sacroiliac ligament. A case of severe low back pain, which was...

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Aota Y. Entrapment of middle cluneal nerves as an unknown cause of low back pain. World J Orthop. 2016;7(3):167-70doi: 10.5312/wjo.v7.i3.167.
Aota, Y. (2016). Entrapment of middle cluneal nerves as an unknown cause of low back pain. World journal of orthopedics, 7(3), 167-70. https://doi.org/10.5312/wjo.v7.i3.167
Aota, Yoichi. "Entrapment of middle cluneal nerves as an unknown cause of low back pain." World journal of orthopedics vol. 7,3 (2016): 167-70. doi: https://doi.org/10.5312/wjo.v7.i3.167
Aota Y. Entrapment of middle cluneal nerves as an unknown cause of low back pain. World J Orthop. 2016 Mar 18;7(3):167-70. doi: 10.5312/wjo.v7.i3.167. eCollection 2016 Mar 18. PMID: 27004164; PMCID: PMC4794535.
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What do cardiologists know about the safety indexes of echocardiography?.

Journal of echocardiography

Koza Y.
PMID: 27276901
J Echocardiogr. 2014 Sep;12(3):127-8. doi: 10.1007/s12574-014-0219-z. Epub 2014 Jul 16.

No abstract available.

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Koza Y. What do cardiologists know about the safety indexes of echocardiography?. J Echocardiogr. 2014;12(3):127-8doi: 10.1007/s12574-014-0219-z.
Koza, Y. (2014). What do cardiologists know about the safety indexes of echocardiography?. Journal of echocardiography, 12(3), 127-8. https://doi.org/10.1007/s12574-014-0219-z
Koza, Yavuzer. "What do cardiologists know about the safety indexes of echocardiography?." Journal of echocardiography vol. 12,3 (2014): 127-8. doi: https://doi.org/10.1007/s12574-014-0219-z
Koza Y. What do cardiologists know about the safety indexes of echocardiography?. J Echocardiogr. 2014 Sep;12(3):127-8. doi: 10.1007/s12574-014-0219-z. Epub 2014 Jul 16. PMID: 27276901.
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Syntheses and Potato Tuber-inducing Activity of Coronafacic Acid Analogues.

Bioscience, biotechnology, and biochemistry

Toshima H, Nara S, Ichihara A, Koda Y, Kikuta Y.
PMID: 27392555
Biosci Biotechnol Biochem. 1998;62(4):681-8. doi: 10.1271/bbb.62.681.

Coronafacic acid (1) is an acid component of coronatine, and has been isolated from several pathovars of Pseudomonas syringae. Syntheses of C6-non- and C6-alkyl-substituted analogues of 1 were accomplished via intramolecular 1,6-conjugate addition as the key step. Among them,...

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Toshima H, Nara S, Ichihara A, et al. Syntheses and Potato Tuber-inducing Activity of Coronafacic Acid Analogues. Biosci Biotechnol Biochem. 1998;62(4):681-8doi: 10.1271/bbb.62.681.
Toshima, H., Nara, S., Ichihara, A., Koda, Y., & Kikuta, Y. (1998). Syntheses and Potato Tuber-inducing Activity of Coronafacic Acid Analogues. Bioscience, biotechnology, and biochemistry, 62(4), 681-8. https://doi.org/10.1271/bbb.62.681
Toshima, H, et al. "Syntheses and Potato Tuber-inducing Activity of Coronafacic Acid Analogues." Bioscience, biotechnology, and biochemistry vol. 62,4 (1998): 681-8. doi: https://doi.org/10.1271/bbb.62.681
Toshima H, Nara S, Ichihara A, Koda Y, Kikuta Y. Syntheses and Potato Tuber-inducing Activity of Coronafacic Acid Analogues. Biosci Biotechnol Biochem. 1998;62(4):681-8. doi: 10.1271/bbb.62.681. PMID: 27392555.
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17(R)-resolvin D1 ameliorates bleomycin-induced pulmonary fibrosis in mice.

Physiological reports

Yatomi M, Hisada T, Ishizuka T, Koga Y, Ono A, Kamide Y, Seki K, Aoki-Saito H, Tsurumaki H, Sunaga N, Kaira K, Dobashi K, Yamada M, Okajima F.
PMID: 26660549
Physiol Rep. 2015 Dec;3(12). doi: 10.14814/phy2.12628.

Idiopathic pulmonary fibrosis (IPF) is a destructive inflammatory disease with limited therapeutic options. Inflammation plays an integral role in the development of pulmonary fibrosis. Unresolved inflammatory responses can lead to substantial tissue injury, chronic inflammation, and fibrosis. The resolvins...

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Yatomi M, Hisada T, Ishizuka T, et al. 17(R)-resolvin D1 ameliorates bleomycin-induced pulmonary fibrosis in mice. Physiol Rep. 2015;3(12)doi: 10.14814/phy2.12628.
Yatomi, M., Hisada, T., Ishizuka, T., Koga, Y., Ono, A., Kamide, Y., Seki, K., Aoki-Saito, H., Tsurumaki, H., Sunaga, N., Kaira, K., Dobashi, K., Yamada, M., & Okajima, F. (2015). 17(R)-resolvin D1 ameliorates bleomycin-induced pulmonary fibrosis in mice. Physiological reports, 3(12), . https://doi.org/10.14814/phy2.12628
Yatomi, Masakiyo, et al. "17(R)-resolvin D1 ameliorates bleomycin-induced pulmonary fibrosis in mice." Physiological reports vol. 3,12 (2015). doi: https://doi.org/10.14814/phy2.12628
Yatomi M, Hisada T, Ishizuka T, Koga Y, Ono A, Kamide Y, Seki K, Aoki-Saito H, Tsurumaki H, Sunaga N, Kaira K, Dobashi K, Yamada M, Okajima F. 17(R)-resolvin D1 ameliorates bleomycin-induced pulmonary fibrosis in mice. Physiol Rep. 2015 Dec;3(12). doi: 10.14814/phy2.12628. PMID: 26660549; PMCID: PMC4760456.
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Showing 1 to 12 of 1574 entries