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Showing 1 to 12 of 17 entries
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[Neuroleptic malignant syndrome and atypical antipsychotics: two case reports].

Psychiatrike = Psychiatriki

Andreou C, Bozikas V, Koumenis G, Garyfallos G, Giouzepas I, Fokas K.
PMID: 22466631
Psychiatriki. 2007 Jul;18(3):273-6.

Neuroleptic malignant syndrome (NMS) constitutes the most serious adverse effect of antipsychotic medications. Although it was initially described as a complication of conventional neuroleptics, atypical antipsychotic agents are also capable of inducing the syndrome. It has been suggested that...

Cite
Andreou C, Bozikas V, Koumenis G, et al. [Neuroleptic malignant syndrome and atypical antipsychotics: two case reports]. Psychiatriki. 2007;18(3):273-6
Andreou, C., Bozikas, V., Koumenis, G., Garyfallos, G., Giouzepas, I., & Fokas, K. (2007). [Neuroleptic malignant syndrome and atypical antipsychotics: two case reports]. Psychiatrike = Psychiatriki, 18(3), 273-6.
Andreou, C, et al. "[Neuroleptic malignant syndrome and atypical antipsychotics: two case reports]." Psychiatrike = Psychiatriki vol. 18,3 (2007): 273-6.
Andreou C, Bozikas V, Koumenis G, Garyfallos G, Giouzepas I, Fokas K. [Neuroleptic malignant syndrome and atypical antipsychotics: two case reports]. Psychiatriki. 2007 Jul;18(3):273-6. Greek, Modern. PMID: 22466631.
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Loss of p19Arf Promotes Fibroblast Survival During Leucine Deprivation.

Biology open

Roby KC, Lieberman A, Kim BJ, Rodríguez NZ, Posimo JM, Tsang T, Verginadis II, Puré E, Brady DC, Koumenis C, Ryeom S.
PMID: 34994382
Biol Open. 2022 Jan 07; doi: 10.1242/bio.058728. Epub 2022 Jan 07.

Fibroblasts are quiescent and tumor suppressive in nature but become activated in wound healing and cancer. The response of fibroblasts to cellular stress has not been extensively investigated however the p53 tumor suppressor has been shown to be activated...

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Roby KC, Lieberman A, Kim BJ, et al. Loss of p19Arf Promotes Fibroblast Survival During Leucine Deprivation. Biol Open. 2022;doi: 10.1242/bio.058728.
Roby, K. C., Lieberman, A., Kim, B. J., Rodríguez, N. Z., Posimo, J. M., Tsang, T., Verginadis, I. I., Puré, E., Brady, D. C., Koumenis, C., & Ryeom, S. (2022). Loss of p19Arf Promotes Fibroblast Survival During Leucine Deprivation. Biology open, . https://doi.org/10.1242/bio.058728
Roby, Kerry C, et al. "Loss of p19Arf Promotes Fibroblast Survival During Leucine Deprivation." Biology open vol. (2022). doi: https://doi.org/10.1242/bio.058728
Roby KC, Lieberman A, Kim BJ, Rodríguez NZ, Posimo JM, Tsang T, Verginadis II, Puré E, Brady DC, Koumenis C, Ryeom S. Loss of p19Arf Promotes Fibroblast Survival During Leucine Deprivation. Biol Open. 2022 Jan 07; doi: 10.1242/bio.058728. Epub 2022 Jan 07. PMID: 34994382.
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Molecular relaxation dynamics and self-association of dexamethasone sodium phosphate solutions.

Chemicke zvesti

Kouderis C, Siafarika P, Kalampounias AG.
PMID: 34305275
Chem Zvesti. 2021 Jul 19;1-11. doi: 10.1007/s11696-021-01787-5. Epub 2021 Jul 19.

Detailed concentration-dependent measurements of sound absorption and velocity have been performed in dexamethasone sodium phosphate (DSP) aqueous solutions in the MHz frequency range. A single well-resolved relaxation process dominates the experimental acoustic spectra following a Debye-type distribution function. The...

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Kouderis C, Siafarika P, Kalampounias AG. Molecular relaxation dynamics and self-association of dexamethasone sodium phosphate solutions. Chem Zvesti. 2021;1-11doi: 10.1007/s11696-021-01787-5.
Kouderis, C., Siafarika, P., & Kalampounias, A. G. (2021). Molecular relaxation dynamics and self-association of dexamethasone sodium phosphate solutions. Chemicke zvesti, 1-11. https://doi.org/10.1007/s11696-021-01787-5
Kouderis, C, et al. "Molecular relaxation dynamics and self-association of dexamethasone sodium phosphate solutions." Chemicke zvesti vol. (2021): 1-11. doi: https://doi.org/10.1007/s11696-021-01787-5
Kouderis C, Siafarika P, Kalampounias AG. Molecular relaxation dynamics and self-association of dexamethasone sodium phosphate solutions. Chem Zvesti. 2021 Jul 19;1-11. doi: 10.1007/s11696-021-01787-5. Epub 2021 Jul 19. PMID: 34305275; PMCID: PMC8287118.
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Activation of p38α stress-activated protein kinase drives the formation of the pre-metastatic niche in the lungs.

Nature cancer

Gui J, Zahedi F, Ortiz A, Cho C, Katlinski KV, Alicea-Torres K, Li J, Todd L, Zhang H, Beiting DP, Sander C, Kirkwood JM, Snow BE, Wakeham AC, Mak TW, Diehl JA, Koumenis C, Ryeom SW, Stanger BZ, Puré E, Gabrilovich DI, Fuchs SY.
PMID: 34124690
Nat Cancer. 2020 Jun;1(6):603-619. doi: 10.1038/s43018-020-0064-0. Epub 2020 May 25.

Primary tumor-derived factors (TDFs) act upon normal cells to generate a pre-metastatic niche, which promotes colonization of target organs by disseminated malignant cells. Here we report that TDFs-induced activation of the p38α kinase in lung fibroblasts plays a critical...

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Gui J, Zahedi F, Ortiz A, et al. Activation of p38α stress-activated protein kinase drives the formation of the pre-metastatic niche in the lungs. Nat Cancer. 2020;1(6):603-619doi: 10.1038/s43018-020-0064-0.
Gui, J., Zahedi, F., Ortiz, A., Cho, C., Katlinski, K. V., Alicea-Torres, K., Li, J., Todd, L., Zhang, H., Beiting, D. P., Sander, C., Kirkwood, J. M., Snow, B. E., Wakeham, A. C., Mak, T. W., Diehl, J. A., Koumenis, C., Ryeom, S. W., Stanger, B. Z., Puré, E., Gabrilovich, D. I., & Fuchs, S. Y. (2020). Activation of p38α stress-activated protein kinase drives the formation of the pre-metastatic niche in the lungs. Nature cancer, 1(6), 603-619. https://doi.org/10.1038/s43018-020-0064-0
Gui, Jun, et al. "Activation of p38α stress-activated protein kinase drives the formation of the pre-metastatic niche in the lungs." Nature cancer vol. 1,6 (2020): 603-619. doi: https://doi.org/10.1038/s43018-020-0064-0
Gui J, Zahedi F, Ortiz A, Cho C, Katlinski KV, Alicea-Torres K, Li J, Todd L, Zhang H, Beiting DP, Sander C, Kirkwood JM, Snow BE, Wakeham AC, Mak TW, Diehl JA, Koumenis C, Ryeom SW, Stanger BZ, Puré E, Gabrilovich DI, Fuchs SY. Activation of p38α stress-activated protein kinase drives the formation of the pre-metastatic niche in the lungs. Nat Cancer. 2020 Jun;1(6):603-619. doi: 10.1038/s43018-020-0064-0. Epub 2020 May 25. PMID: 34124690; PMCID: PMC8194112.
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A Monte Carlo Determination of Dose and Range Uncertainties for Preclinical Studies with a Proton Beam.

Cancers

Bongrand A, Koumeir C, Villoing D, Guertin A, Haddad F, Métivier V, Poirier F, Potiron V, Servagent N, Supiot S, Delpon G, Chiavassa S.
PMID: 33920758
Cancers (Basel). 2021 Apr 15;13(8). doi: 10.3390/cancers13081889.

Proton therapy (PRT) is an irradiation technique that aims at limiting normal tissue damage while maintaining the tumor response. To study its specificities, the ARRONAX cyclotron is currently developing a preclinical structure compatible with biological experiments. A prerequisite is...

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Bongrand A, Koumeir C, Villoing D, et al. A Monte Carlo Determination of Dose and Range Uncertainties for Preclinical Studies with a Proton Beam. Cancers (Basel). 2021;13(8)doi: 10.3390/cancers13081889.
Bongrand, A., Koumeir, C., Villoing, D., Guertin, A., Haddad, F., Métivier, V., Poirier, F., Potiron, V., Servagent, N., Supiot, S., Delpon, G., & Chiavassa, S. (2021). A Monte Carlo Determination of Dose and Range Uncertainties for Preclinical Studies with a Proton Beam. Cancers, 13(8), . https://doi.org/10.3390/cancers13081889
Bongrand, Arthur, et al. "A Monte Carlo Determination of Dose and Range Uncertainties for Preclinical Studies with a Proton Beam." Cancers vol. 13,8 (2021). doi: https://doi.org/10.3390/cancers13081889
Bongrand A, Koumeir C, Villoing D, Guertin A, Haddad F, Métivier V, Poirier F, Potiron V, Servagent N, Supiot S, Delpon G, Chiavassa S. A Monte Carlo Determination of Dose and Range Uncertainties for Preclinical Studies with a Proton Beam. Cancers (Basel). 2021 Apr 15;13(8). doi: 10.3390/cancers13081889. PMID: 33920758; PMCID: PMC8071150.
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A Novel Mouse Model of Radiation-Induced Cardiac Injury Reveals Biological and Radiological Biomarkers of Cardiac Dysfunction with Potential Clinical Relevance.

Clinical cancer research : an official journal of the American Association for Cancer Research

Dreyfuss AD, Goia D, Shoniyozov K, Shewale SV, Velalopoulou A, Mazzoni S, Avgousti H, Metzler SD, Bravo PE, Feigenberg SJ, Ky B, Verginadis II, Koumenis C.
PMID: 33542079
Clin Cancer Res. 2021 Apr 15;27(8):2266-2276. doi: 10.1158/1078-0432.CCR-20-3882. Epub 2021 Feb 04.

PURPOSE: Radiation-induced cardiotoxicity is a significant concern in thoracic oncology patients. However, the basis for this disease pathology is not well characterized. We developed a novel mouse model of radiation-induced cardiotoxicity to investigate pathophysiologic mechanisms and identify clinically targetable...

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Dreyfuss AD, Goia D, Shoniyozov K, et al. A Novel Mouse Model of Radiation-Induced Cardiac Injury Reveals Biological and Radiological Biomarkers of Cardiac Dysfunction with Potential Clinical Relevance. Clin Cancer Res. 2021;27(8):2266-2276doi: 10.1158/1078-0432.CCR-20-3882.
Dreyfuss, A. D., Goia, D., Shoniyozov, K., Shewale, S. V., Velalopoulou, A., Mazzoni, S., Avgousti, H., Metzler, S. D., Bravo, P. E., Feigenberg, S. J., Ky, B., Verginadis, I. I., & Koumenis, C. (2021). A Novel Mouse Model of Radiation-Induced Cardiac Injury Reveals Biological and Radiological Biomarkers of Cardiac Dysfunction with Potential Clinical Relevance. Clinical cancer research : an official journal of the American Association for Cancer Research, 27(8), 2266-2276. https://doi.org/10.1158/1078-0432.CCR-20-3882
Dreyfuss, Alexandra D, et al. "A Novel Mouse Model of Radiation-Induced Cardiac Injury Reveals Biological and Radiological Biomarkers of Cardiac Dysfunction with Potential Clinical Relevance." Clinical cancer research : an official journal of the American Association for Cancer Research vol. 27,8 (2021): 2266-2276. doi: https://doi.org/10.1158/1078-0432.CCR-20-3882
Dreyfuss AD, Goia D, Shoniyozov K, Shewale SV, Velalopoulou A, Mazzoni S, Avgousti H, Metzler SD, Bravo PE, Feigenberg SJ, Ky B, Verginadis II, Koumenis C. A Novel Mouse Model of Radiation-Induced Cardiac Injury Reveals Biological and Radiological Biomarkers of Cardiac Dysfunction with Potential Clinical Relevance. Clin Cancer Res. 2021 Apr 15;27(8):2266-2276. doi: 10.1158/1078-0432.CCR-20-3882. Epub 2021 Feb 04. PMID: 33542079.
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Combination of vasculature targeting, hypofractionated radiotherapy, and immune checkpoint inhibitor elicits potent antitumor immune response and blocks tumor progression.

Journal for immunotherapy of cancer

Pierini S, Mishra A, Perales-Linares R, Uribe-Herranz M, Beghi S, Giglio A, Pustylnikov S, Costabile F, Rafail S, Amici A, Facciponte JG, Koumenis C, Facciabene A.
PMID: 33563772
J Immunother Cancer. 2021 Feb;9(2). doi: 10.1136/jitc-2020-001636.

BACKGROUND: Tumor endothelial marker 1 (TEM1) is a protein expressed in the tumor-associated endothelium and/or stroma of various types of cancer. We previously demonstrated that immunization with a plasmid-DNA vaccine targeting TEM1 reduced tumor progression in three murine cancer...

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Pierini S, Mishra A, Perales-Linares R, et al. Combination of vasculature targeting, hypofractionated radiotherapy, and immune checkpoint inhibitor elicits potent antitumor immune response and blocks tumor progression. J Immunother Cancer. 2021;9(2)doi: 10.1136/jitc-2020-001636.
Pierini, S., Mishra, A., Perales-Linares, R., Uribe-Herranz, M., Beghi, S., Giglio, A., Pustylnikov, S., Costabile, F., Rafail, S., Amici, A., Facciponte, J. G., Koumenis, C., & Facciabene, A. (2021). Combination of vasculature targeting, hypofractionated radiotherapy, and immune checkpoint inhibitor elicits potent antitumor immune response and blocks tumor progression. Journal for immunotherapy of cancer, 9(2), . https://doi.org/10.1136/jitc-2020-001636
Pierini, Stefano, et al. "Combination of vasculature targeting, hypofractionated radiotherapy, and immune checkpoint inhibitor elicits potent antitumor immune response and blocks tumor progression." Journal for immunotherapy of cancer vol. 9,2 (2021). doi: https://doi.org/10.1136/jitc-2020-001636
Pierini S, Mishra A, Perales-Linares R, Uribe-Herranz M, Beghi S, Giglio A, Pustylnikov S, Costabile F, Rafail S, Amici A, Facciponte JG, Koumenis C, Facciabene A. Combination of vasculature targeting, hypofractionated radiotherapy, and immune checkpoint inhibitor elicits potent antitumor immune response and blocks tumor progression. J Immunother Cancer. 2021 Feb;9(2). doi: 10.1136/jitc-2020-001636. PMID: 33563772; PMCID: PMC7875275.
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Prevalence of BRCA1 and BRCA2 mutations in unselected breast cancer patients from Greece.

Hereditary cancer in clinical practice

Koumpis C, Dimitrakakis C, Antsaklis A, Royer R, Zhang S, Narod SA, Kotsopoulos J.
PMID: 22085629
Hered Cancer Clin Pract. 2011 Nov 15;9:10. doi: 10.1186/1897-4287-9-10.

BACKGROUND: Inheritance of a mutation in either BRCA1 or BRCA2 accounts for approximately 5% of all breast cancer cases, but varies by country. Investigations into the contribution of BRCA mutations to breast cancer incidence in Greece have been, for...

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Koumpis C, Dimitrakakis C, Antsaklis A, et al. Prevalence of BRCA1 and BRCA2 mutations in unselected breast cancer patients from Greece. Hered Cancer Clin Pract. 2011;9:10doi: 10.1186/1897-4287-9-10.
Koumpis, C., Dimitrakakis, C., Antsaklis, A., Royer, R., Zhang, S., Narod, S. A., & Kotsopoulos, J. (2011). Prevalence of BRCA1 and BRCA2 mutations in unselected breast cancer patients from Greece. Hereditary cancer in clinical practice, 910. https://doi.org/10.1186/1897-4287-9-10
Koumpis, Chrissovaladis, et al. "Prevalence of BRCA1 and BRCA2 mutations in unselected breast cancer patients from Greece." Hereditary cancer in clinical practice vol. 9 (2011): 10. doi: https://doi.org/10.1186/1897-4287-9-10
Koumpis C, Dimitrakakis C, Antsaklis A, Royer R, Zhang S, Narod SA, Kotsopoulos J. Prevalence of BRCA1 and BRCA2 mutations in unselected breast cancer patients from Greece. Hered Cancer Clin Pract. 2011 Nov 15;9:10. doi: 10.1186/1897-4287-9-10. PMID: 22085629; PMCID: PMC3240809.
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[Neuroleptic malignant syndrome and atypical antipsychotics: two case reports].

Psychiatrike = Psychiatriki

Andreou C, Bozikas V, Koumenis G, Garyfallos G, Giouzepas I, Fokas K.
PMID: 22466631
Psychiatriki. 2007 Jul;18(3):273-6.

Neuroleptic malignant syndrome (NMS) constitutes the most serious adverse effect of antipsychotic medications. Although it was initially described as a complication of conventional neuroleptics, atypical antipsychotic agents are also capable of inducing the syndrome. It has been suggested that...

Cite
Andreou C, Bozikas V, Koumenis G, et al. [Neuroleptic malignant syndrome and atypical antipsychotics: two case reports]. Psychiatriki. 2007;18(3):273-6
Andreou, C., Bozikas, V., Koumenis, G., Garyfallos, G., Giouzepas, I., & Fokas, K. (2007). [Neuroleptic malignant syndrome and atypical antipsychotics: two case reports]. Psychiatrike = Psychiatriki, 18(3), 273-6.
Andreou, C, et al. "[Neuroleptic malignant syndrome and atypical antipsychotics: two case reports]." Psychiatrike = Psychiatriki vol. 18,3 (2007): 273-6.
Andreou C, Bozikas V, Koumenis G, Garyfallos G, Giouzepas I, Fokas K. [Neuroleptic malignant syndrome and atypical antipsychotics: two case reports]. Psychiatriki. 2007 Jul;18(3):273-6. Greek, Modern. PMID: 22466631.
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Author Correction: ATF4 couples MYC-dependent translational activity to bioenergetic demands during tumour progression.

Nature cell biology

Tameire F, Verginadis II, Leli NM, Polte C, Conn CS, Ojha R, Salinas CS, Chinga F, Monroy AM, Fu W, Wang P, Kossenkov A, Ye J, Amaravadi RK, Ignatova Z, Fuchs SY, Diehl JA, Ruggero D, Koumenis C.
PMID: 31316187
Nat Cell Biol. 2019 Aug;21(8):1052. doi: 10.1038/s41556-019-0370-x.

An amendment to this paper has been published and can be accessed via a link at the top of the paper.

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Tameire F, Verginadis II, Leli NM, et al. Author Correction: ATF4 couples MYC-dependent translational activity to bioenergetic demands during tumour progression. Nat Cell Biol. 2019;21(8):1052doi: 10.1038/s41556-019-0370-x.
Tameire, F., Verginadis, I. I., Leli, N. M., Polte, C., Conn, C. S., Ojha, R., Salinas, C. S., Chinga, F., Monroy, A. M., Fu, W., Wang, P., Kossenkov, A., Ye, J., Amaravadi, R. K., Ignatova, Z., Fuchs, S. Y., Diehl, J. A., Ruggero, D., & Koumenis, C. (2019). Author Correction: ATF4 couples MYC-dependent translational activity to bioenergetic demands during tumour progression. Nature cell biology, 21(8), 1052. https://doi.org/10.1038/s41556-019-0370-x
Tameire, Feven, et al. "Author Correction: ATF4 couples MYC-dependent translational activity to bioenergetic demands during tumour progression." Nature cell biology vol. 21,8 (2019): 1052. doi: https://doi.org/10.1038/s41556-019-0370-x
Tameire F, Verginadis II, Leli NM, Polte C, Conn CS, Ojha R, Salinas CS, Chinga F, Monroy AM, Fu W, Wang P, Kossenkov A, Ye J, Amaravadi RK, Ignatova Z, Fuchs SY, Diehl JA, Ruggero D, Koumenis C. Author Correction: ATF4 couples MYC-dependent translational activity to bioenergetic demands during tumour progression. Nat Cell Biol. 2019 Aug;21(8):1052. doi: 10.1038/s41556-019-0370-x. PMID: 31316187.
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Proton-transfer in 1,1,3,3 tetramethyl guanidine by means of ultrasonic relaxation and Raman spectroscopies and molecular orbital calculations.

Spectrochimica acta. Part A, Molecular and biomolecular spectroscopy

Tsigoias S, Kouderis C, Mylona-Kosmas A, Boghosian S, Kalampounias AG.
PMID: 31865106
Spectrochim Acta A Mol Biomol Spectrosc. 2020 Mar 15;229:117958. doi: 10.1016/j.saa.2019.117958. Epub 2019 Dec 15.

In this work, we report on the structure and dynamics of the 1,1,3,3 tetramethyl guanidine (TMG) aqueous solutions in a wide concentration and temperature range by combining vibrational and ultrasonic spectroscopies. The experimental Raman spectra have been compared with...

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Tsigoias S, Kouderis C, Mylona-Kosmas A, et al. Proton-transfer in 1,1,3,3 tetramethyl guanidine by means of ultrasonic relaxation and Raman spectroscopies and molecular orbital calculations. Spectrochim Acta A Mol Biomol Spectrosc. 2019;229:117958doi: 10.1016/j.saa.2019.117958.
Tsigoias, S., Kouderis, C., Mylona-Kosmas, A., Boghosian, S., & Kalampounias, A. G. (2020). Proton-transfer in 1,1,3,3 tetramethyl guanidine by means of ultrasonic relaxation and Raman spectroscopies and molecular orbital calculations. Spectrochimica acta. Part A, Molecular and biomolecular spectroscopy, 229117958. https://doi.org/10.1016/j.saa.2019.117958
Tsigoias, S, et al. "Proton-transfer in 1,1,3,3 tetramethyl guanidine by means of ultrasonic relaxation and Raman spectroscopies and molecular orbital calculations." Spectrochimica acta. Part A, Molecular and biomolecular spectroscopy vol. 229 (2020): 117958. doi: https://doi.org/10.1016/j.saa.2019.117958
Tsigoias S, Kouderis C, Mylona-Kosmas A, Boghosian S, Kalampounias AG. Proton-transfer in 1,1,3,3 tetramethyl guanidine by means of ultrasonic relaxation and Raman spectroscopies and molecular orbital calculations. Spectrochim Acta A Mol Biomol Spectrosc. 2020 Mar 15;229:117958. doi: 10.1016/j.saa.2019.117958. Epub 2019 Dec 15. PMID: 31865106.
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FLASH Proton Radiotherapy Spares Normal Epithelial and Mesenchymal Tissues While Preserving Sarcoma Response.

Cancer research

Velalopoulou A, Karagounis IV, Cramer GM, Kim MM, Skoufos G, Goia D, Hagan S, Verginadis II, Shoniyozov K, Chiango J, Cerullo M, Varner K, Yao L, Qin L, Hatzigeorgiou AG, Minn AJ, Putt M, Lanza M, Assenmacher CA, Radaelli E, Huck J, Diffenderfer E, Dong L, Metz J, Koumenis C, Cengel KA, Maity A, Busch TM.
PMID: 34321243
Cancer Res. 2021 Sep 15;81(18):4808-4821. doi: 10.1158/0008-5472.CAN-21-1500. Epub 2021 Jul 28.

In studies of electron and proton radiotherapy, ultrahigh dose rates of FLASH radiotherapy appear to produce fewer toxicities than standard dose rates while maintaining local tumor control. FLASH-proton radiotherapy (F-PRT) brings the spatial advantages of PRT to FLASH dose...

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Velalopoulou A, Karagounis IV, Cramer GM, et al. FLASH Proton Radiotherapy Spares Normal Epithelial and Mesenchymal Tissues While Preserving Sarcoma Response. Cancer Res. 2021;81(18):4808-4821doi: 10.1158/0008-5472.CAN-21-1500.
Velalopoulou, A., Karagounis, I. V., Cramer, G. M., Kim, M. M., Skoufos, G., Goia, D., Hagan, S., Verginadis, I. I., Shoniyozov, K., Chiango, J., Cerullo, M., Varner, K., Yao, L., Qin, L., Hatzigeorgiou, A. G., Minn, A. J., Putt, M., Lanza, M., Assenmacher, C. A., Radaelli, E., Huck, J., Diffenderfer, E., Dong, L., Metz, J., Koumenis, C., Cengel, K. A., Maity, A., & Busch, T. M. (2021). FLASH Proton Radiotherapy Spares Normal Epithelial and Mesenchymal Tissues While Preserving Sarcoma Response. Cancer research, 81(18), 4808-4821. https://doi.org/10.1158/0008-5472.CAN-21-1500
Velalopoulou, Anastasia, et al. "FLASH Proton Radiotherapy Spares Normal Epithelial and Mesenchymal Tissues While Preserving Sarcoma Response." Cancer research vol. 81,18 (2021): 4808-4821. doi: https://doi.org/10.1158/0008-5472.CAN-21-1500
Velalopoulou A, Karagounis IV, Cramer GM, Kim MM, Skoufos G, Goia D, Hagan S, Verginadis II, Shoniyozov K, Chiango J, Cerullo M, Varner K, Yao L, Qin L, Hatzigeorgiou AG, Minn AJ, Putt M, Lanza M, Assenmacher CA, Radaelli E, Huck J, Diffenderfer E, Dong L, Metz J, Koumenis C, Cengel KA, Maity A, Busch TM. FLASH Proton Radiotherapy Spares Normal Epithelial and Mesenchymal Tissues While Preserving Sarcoma Response. Cancer Res. 2021 Sep 15;81(18):4808-4821. doi: 10.1158/0008-5472.CAN-21-1500. Epub 2021 Jul 28. PMID: 34321243; PMCID: PMC8715480.
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Showing 1 to 12 of 17 entries