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Modulation of sinusoidal and canalicular elimination of bilirubin-glucuronides by rifampicin and other cholestatic drugs in a sandwich culture of rat hepatocytes.

Hepatology research : the official journal of the Japan Society of Hepatology

Lengyel G, Veres Z, Tugyi R, Vereczkey L, Molnár T, Glavinas H, Krajcsi P, Jemnitz K.
PMID: 17760873
Hepatol Res. 2008 Mar;38(3):300-9. doi: 10.1111/j.1872-034X.2007.00255.x. Epub 2007 Aug 29.

AIM: Drug-induced hyperbilirubinemia has been shown to often be derived from modulation of the expression and activity of hepatobiliary transporters. In this study we examined the interactions of some therapeutic agents, which have been shown to cause cholestasis, with...

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Lengyel G, Veres Z, Tugyi R, et al. Modulation of sinusoidal and canalicular elimination of bilirubin-glucuronides by rifampicin and other cholestatic drugs in a sandwich culture of rat hepatocytes. Hepatol Res. 2007;38(3):300-9doi: 10.1111/j.1872-034X.2007.00255.x.
Lengyel, G., Veres, Z., Tugyi, R., Vereczkey, L., Molnár, T., Glavinas, H., Krajcsi, P., & Jemnitz, K. (2008). Modulation of sinusoidal and canalicular elimination of bilirubin-glucuronides by rifampicin and other cholestatic drugs in a sandwich culture of rat hepatocytes. Hepatology research : the official journal of the Japan Society of Hepatology, 38(3), 300-9. https://doi.org/10.1111/j.1872-034X.2007.00255.x
Lengyel, György, et al. "Modulation of sinusoidal and canalicular elimination of bilirubin-glucuronides by rifampicin and other cholestatic drugs in a sandwich culture of rat hepatocytes." Hepatology research : the official journal of the Japan Society of Hepatology vol. 38,3 (2008): 300-9. doi: https://doi.org/10.1111/j.1872-034X.2007.00255.x
Lengyel G, Veres Z, Tugyi R, Vereczkey L, Molnár T, Glavinas H, Krajcsi P, Jemnitz K. Modulation of sinusoidal and canalicular elimination of bilirubin-glucuronides by rifampicin and other cholestatic drugs in a sandwich culture of rat hepatocytes. Hepatol Res. 2008 Mar;38(3):300-9. doi: 10.1111/j.1872-034X.2007.00255.x. Epub 2007 Aug 29. PMID: 17760873.
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Cilia kinases in skeletal development and homeostasis.

Developmental dynamics : an official publication of the American Association of Anatomists

Abraham SP, Nita A, Krejci P, Bosakova M.
PMID: 34582081
Dev Dyn. 2021 Sep 28; doi: 10.1002/dvdy.426. Epub 2021 Sep 28.

Primary cilia are dynamic compartments that regulate multiple aspects of cellular signaling. The production, maintenance, and function of cilia involve more than 1000 genes in mammals, and their mutations disrupt the ciliary signaling which manifests in a plethora of...

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Abraham SP, Nita A, Krejci P, et al. Cilia kinases in skeletal development and homeostasis. Dev Dyn. 2021;doi: 10.1002/dvdy.426.
Abraham, S. P., Nita, A., Krejci, P., & Bosakova, M. (2021). Cilia kinases in skeletal development and homeostasis. Developmental dynamics : an official publication of the American Association of Anatomists, . https://doi.org/10.1002/dvdy.426
Abraham, Sara P, et al. "Cilia kinases in skeletal development and homeostasis." Developmental dynamics : an official publication of the American Association of Anatomists vol. (2021). doi: https://doi.org/10.1002/dvdy.426
Abraham SP, Nita A, Krejci P, Bosakova M. Cilia kinases in skeletal development and homeostasis. Dev Dyn. 2021 Sep 28; doi: 10.1002/dvdy.426. Epub 2021 Sep 28. PMID: 34582081.
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Loss of Sprouty Produces a Ciliopathic Skeletal Phenotype in Mice Through Upregulation of Hedgehog Signaling.

Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research

Hruba E, Kavkova M, Dalecka L, Macholan M, Zikmund T, Varecha M, Bosakova M, Kaiser J, Krejci P, Hovorakova M, Buchtova M.
PMID: 34423857
J Bone Miner Res. 2021 Nov;36(11):2258-2274. doi: 10.1002/jbmr.4427. Epub 2021 Sep 20.

The Sprouty family is a highly conserved group of intracellular modulators of receptor tyrosine kinase (RTK)-signaling pathways, which have been recently linked to primary cilia. Disruptions in the structure and function of primary cilia cause inherited disorders called ciliopathies....

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Hruba E, Kavkova M, Dalecka L, et al. Loss of Sprouty Produces a Ciliopathic Skeletal Phenotype in Mice Through Upregulation of Hedgehog Signaling. J Bone Miner Res. 2021;36(11):2258-2274doi: 10.1002/jbmr.4427.
Hruba, E., Kavkova, M., Dalecka, L., Macholan, M., Zikmund, T., Varecha, M., Bosakova, M., Kaiser, J., Krejci, P., Hovorakova, M., & Buchtova, M. (2021). Loss of Sprouty Produces a Ciliopathic Skeletal Phenotype in Mice Through Upregulation of Hedgehog Signaling. Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research, 36(11), 2258-2274. https://doi.org/10.1002/jbmr.4427
Hruba, Eva, et al. "Loss of Sprouty Produces a Ciliopathic Skeletal Phenotype in Mice Through Upregulation of Hedgehog Signaling." Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research vol. 36,11 (2021): 2258-2274. doi: https://doi.org/10.1002/jbmr.4427
Hruba E, Kavkova M, Dalecka L, Macholan M, Zikmund T, Varecha M, Bosakova M, Kaiser J, Krejci P, Hovorakova M, Buchtova M. Loss of Sprouty Produces a Ciliopathic Skeletal Phenotype in Mice Through Upregulation of Hedgehog Signaling. J Bone Miner Res. 2021 Nov;36(11):2258-2274. doi: 10.1002/jbmr.4427. Epub 2021 Sep 20. PMID: 34423857.
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Age-Related Functional and Expressional Changes in Efflux Pathways at the Blood-Brain Barrier.

Frontiers in aging neuroscience

Erdő F, Krajcsi P.
PMID: 31417399
Front Aging Neurosci. 2019 Jul 30;11:196. doi: 10.3389/fnagi.2019.00196. eCollection 2019.

During the last decade, several articles have reported a relationship between advanced age and changes in the integrity of the blood-brain barrier (BBB). These changes were manifested not only in the morphology and structure of the cerebral microvessels but...

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Erdő F, Krajcsi P. Age-Related Functional and Expressional Changes in Efflux Pathways at the Blood-Brain Barrier. Front Aging Neurosci. 2019;11:196doi: 10.3389/fnagi.2019.00196.
Erdő, F., & Krajcsi, P. (2019). Age-Related Functional and Expressional Changes in Efflux Pathways at the Blood-Brain Barrier. Frontiers in aging neuroscience, 11196. https://doi.org/10.3389/fnagi.2019.00196
Erdő, Franciska, and Krajcsi, Péter. "Age-Related Functional and Expressional Changes in Efflux Pathways at the Blood-Brain Barrier." Frontiers in aging neuroscience vol. 11 (2019): 196. doi: https://doi.org/10.3389/fnagi.2019.00196
Erdő F, Krajcsi P. Age-Related Functional and Expressional Changes in Efflux Pathways at the Blood-Brain Barrier. Front Aging Neurosci. 2019 Jul 30;11:196. doi: 10.3389/fnagi.2019.00196. eCollection 2019. PMID: 31417399; PMCID: PMC6682691.
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The Source of the Symbolic Numerical Distance and Size Effects.

Frontiers in psychology

Krajcsi A, Lengyel G, Kojouharova P.
PMID: 27917139
Front Psychol. 2016 Nov 21;7:1795. doi: 10.3389/fpsyg.2016.01795. eCollection 2016.

Human number understanding is thought to rely on the analog number system (ANS), working according to Weber's law. We propose an alternative account, suggesting that symbolic mathematical knowledge is based on a discrete semantic system (DSS), a representation that...

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Krajcsi A, Lengyel G, Kojouharova P. The Source of the Symbolic Numerical Distance and Size Effects. Front Psychol. 2016;7:1795doi: 10.3389/fpsyg.2016.01795.
Krajcsi, A., Lengyel, G., & Kojouharova, P. (2016). The Source of the Symbolic Numerical Distance and Size Effects. Frontiers in psychology, 71795. https://doi.org/10.3389/fpsyg.2016.01795
Krajcsi, Attila, et al. "The Source of the Symbolic Numerical Distance and Size Effects." Frontiers in psychology vol. 7 (2016): 1795. doi: https://doi.org/10.3389/fpsyg.2016.01795
Krajcsi A, Lengyel G, Kojouharova P. The Source of the Symbolic Numerical Distance and Size Effects. Front Psychol. 2016 Nov 21;7:1795. doi: 10.3389/fpsyg.2016.01795. eCollection 2016. PMID: 27917139; PMCID: PMC5116562.
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Expanding horizons of achondroplasia treatment: current options and future developments.

Osteoarthritis and cartilage

Fafilek B, Bosakova M, Krejci P.
PMID: 34864168
Osteoarthritis Cartilage. 2021 Dec 02; doi: 10.1016/j.joca.2021.11.017. Epub 2021 Dec 02.

Activating mutations in the FGFR3 receptor tyrosine kinase lead to most prevalent form of genetic dwarfism in humans, the achondroplasia. Many features of the complex function of FGFR3 in growing skeleton were characterized, which facilitated identification of therapy targets,...

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Fafilek B, Bosakova M, Krejci P. Expanding horizons of achondroplasia treatment: current options and future developments. Osteoarthritis Cartilage. 2021;doi: 10.1016/j.joca.2021.11.017.
Fafilek, B., Bosakova, M., & Krejci, P. (2021). Expanding horizons of achondroplasia treatment: current options and future developments. Osteoarthritis and cartilage, . https://doi.org/10.1016/j.joca.2021.11.017
Fafilek, Bohumil, et al. "Expanding horizons of achondroplasia treatment: current options and future developments." Osteoarthritis and cartilage vol. (2021). doi: https://doi.org/10.1016/j.joca.2021.11.017
Fafilek B, Bosakova M, Krejci P. Expanding horizons of achondroplasia treatment: current options and future developments. Osteoarthritis Cartilage. 2021 Dec 02; doi: 10.1016/j.joca.2021.11.017. Epub 2021 Dec 02. PMID: 34864168.
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4-PBA treatment improves bone phenotypes in the Aga2 mouse model of osteogenesis imperfecta.

Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research

Duran I, Zieba J, Csukasi F, Martin JH, Wachtell D, Barad M, Dawson B, Fafilek B, Jacobson CM, Ambrose CG, Cohn DH, Krejci P, Lee BH, Krakow D.
PMID: 34997935
J Bone Miner Res. 2022 Jan 08; doi: 10.1002/jbmr.4501. Epub 2022 Jan 08.

Osteogenesis Imperfecta (OI) is a genetically heterogenous disorder most often due to heterozygosity for mutations in the type I procollagen genes, COL1A1 or COL1A2. The disorder is characterized by bone fragility leading to increased fracture incidence and long-bone deformities....

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Duran I, Zieba J, Csukasi F, et al. 4-PBA treatment improves bone phenotypes in the Aga2 mouse model of osteogenesis imperfecta. J Bone Miner Res. 2022;doi: 10.1002/jbmr.4501.
Duran, I., Zieba, J., Csukasi, F., Martin, J. H., Wachtell, D., Barad, M., Dawson, B., Fafilek, B., Jacobson, C. M., Ambrose, C. G., Cohn, D. H., Krejci, P., Lee, B. H., & Krakow, D. (2022). 4-PBA treatment improves bone phenotypes in the Aga2 mouse model of osteogenesis imperfecta. Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research, . https://doi.org/10.1002/jbmr.4501
Duran, Ivan, et al. "4-PBA treatment improves bone phenotypes in the Aga2 mouse model of osteogenesis imperfecta." Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research vol. (2022). doi: https://doi.org/10.1002/jbmr.4501
Duran I, Zieba J, Csukasi F, Martin JH, Wachtell D, Barad M, Dawson B, Fafilek B, Jacobson CM, Ambrose CG, Cohn DH, Krejci P, Lee BH, Krakow D. 4-PBA treatment improves bone phenotypes in the Aga2 mouse model of osteogenesis imperfecta. J Bone Miner Res. 2022 Jan 08; doi: 10.1002/jbmr.4501. Epub 2022 Jan 08. PMID: 34997935.
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Cladribine as a Potential Object of Nucleoside Transporter-Based Drug Interactions.

Clinical pharmacokinetics

Hermann R, Krajcsi P, Fluck M, Seithel-Keuth A, Bytyqi A, Galazka A, Munafo A.
PMID: 34894346
Clin Pharmacokinet. 2021 Dec 11; doi: 10.1007/s40262-021-01089-9. Epub 2021 Dec 11.

Cladribine is a nucleoside analog that is phosphorylated in its target cells (B and T-lymphocytes) to its active triphosphate form (2-chlorodeoxyadenosine triphosphate). Cladribine tablets 10 mg (Mavenclad

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Hermann R, Krajcsi P, Fluck M, et al. Cladribine as a Potential Object of Nucleoside Transporter-Based Drug Interactions. Clin Pharmacokinet. 2021;doi: 10.1007/s40262-021-01089-9.
Hermann, R., Krajcsi, P., Fluck, M., Seithel-Keuth, A., Bytyqi, A., Galazka, A., & Munafo, A. (2021). Cladribine as a Potential Object of Nucleoside Transporter-Based Drug Interactions. Clinical pharmacokinetics, . https://doi.org/10.1007/s40262-021-01089-9
Hermann, Robert, et al. "Cladribine as a Potential Object of Nucleoside Transporter-Based Drug Interactions." Clinical pharmacokinetics vol. (2021). doi: https://doi.org/10.1007/s40262-021-01089-9
Hermann R, Krajcsi P, Fluck M, Seithel-Keuth A, Bytyqi A, Galazka A, Munafo A. Cladribine as a Potential Object of Nucleoside Transporter-Based Drug Interactions. Clin Pharmacokinet. 2021 Dec 11; doi: 10.1007/s40262-021-01089-9. Epub 2021 Dec 11. PMID: 34894346.
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Development of Preschoolers' Understanding of Zero.

Frontiers in psychology

Krajcsi A, Kojouharova P, Lengyel G.
PMID: 34385941
Front Psychol. 2021 Jul 27;12:583734. doi: 10.3389/fpsyg.2021.583734. eCollection 2021.

While knowledge on the development of understanding positive integers is rapidly growing, the development of understanding zero remains not well-understood. Here, we test several components of preschoolers' understanding of zero: Whether they can use empty sets in numerical tasks...

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Krajcsi A, Kojouharova P, Lengyel G. Development of Preschoolers' Understanding of Zero. Front Psychol. 2021;12:583734doi: 10.3389/fpsyg.2021.583734.
Krajcsi, A., Kojouharova, P., & Lengyel, G. (2021). Development of Preschoolers' Understanding of Zero. Frontiers in psychology, 12583734. https://doi.org/10.3389/fpsyg.2021.583734
Krajcsi, Attila, et al. "Development of Preschoolers' Understanding of Zero." Frontiers in psychology vol. 12 (2021): 583734. doi: https://doi.org/10.3389/fpsyg.2021.583734
Krajcsi A, Kojouharova P, Lengyel G. Development of Preschoolers' Understanding of Zero. Front Psychol. 2021 Jul 27;12:583734. doi: 10.3389/fpsyg.2021.583734. eCollection 2021. PMID: 34385941; PMCID: PMC8353124.
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Apoptosis inhibitor 5 (API-5; AAC-11; FIF) is upregulated in human carcinomas in vivo.

Oncology letters

Koci L, Chlebova K, Hyzdalova M, Hofmanova J, Jira M, Kysela P, Kozubik A, Kala Z, Krejci P.
PMID: 22741017
Oncol Lett. 2012 Apr 01;3(4):913-916. doi: 10.3892/ol.2012.593. Epub 2012 Feb 03.

Apoptosis inhibitor 5 (API-5) is a 55 kDa nuclear protein with potent anti-apoptotic signaling in tumor cells in vitro. In this study, we analyzed the expression of the API-5 protein in vivo in a broad spectrum of human carcinomas,...

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Koci L, Chlebova K, Hyzdalova M, et al. Apoptosis inhibitor 5 (API-5; AAC-11; FIF) is upregulated in human carcinomas in vivo. Oncol Lett. 2012;3(4):913-916doi: 10.3892/ol.2012.593.
Koci, L., Chlebova, K., Hyzdalova, M., Hofmanova, J., Jira, M., Kysela, P., Kozubik, A., Kala, Z., & Krejci, P. (2012). Apoptosis inhibitor 5 (API-5; AAC-11; FIF) is upregulated in human carcinomas in vivo. Oncology letters, 3(4), 913-916. https://doi.org/10.3892/ol.2012.593
Koci, Lenka, et al. "Apoptosis inhibitor 5 (API-5; AAC-11; FIF) is upregulated in human carcinomas in vivo." Oncology letters vol. 3,4 (2012): 913-916. doi: https://doi.org/10.3892/ol.2012.593
Koci L, Chlebova K, Hyzdalova M, Hofmanova J, Jira M, Kysela P, Kozubik A, Kala Z, Krejci P. Apoptosis inhibitor 5 (API-5; AAC-11; FIF) is upregulated in human carcinomas in vivo. Oncol Lett. 2012 Apr 01;3(4):913-916. doi: 10.3892/ol.2012.593. Epub 2012 Feb 03. PMID: 22741017; PMCID: PMC3362606.
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The Kühtai data set: 25 years of lysimetric, snow pillow, and meteorological measurements.

Water resources research

Krajči P, Kirnbauer R, Parajka J, Schöber J, Blöschl G.
PMID: 28931957
Water Resour Res. 2017 Jun;53(6):5158-5165. doi: 10.1002/2017WR020445. Epub 2017 Jun 13.

Snow measurements at the Kühtai station in Tirol, Austria, (1920 m.a.s.l.) are described. The data set includes snow water equivalent from a 10 m

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Krajči P, Kirnbauer R, Parajka J, et al. The Kühtai data set: 25 years of lysimetric, snow pillow, and meteorological measurements. Water Resour Res. 2017;53(6):5158-5165doi: 10.1002/2017WR020445.
Krajči, P., Kirnbauer, R., Parajka, J., Schöber, J., & Blöschl, G. (2017). The Kühtai data set: 25 years of lysimetric, snow pillow, and meteorological measurements. Water resources research, 53(6), 5158-5165. https://doi.org/10.1002/2017WR020445
Krajči, P, et al. "The Kühtai data set: 25 years of lysimetric, snow pillow, and meteorological measurements." Water resources research vol. 53,6 (2017): 5158-5165. doi: https://doi.org/10.1002/2017WR020445
Krajči P, Kirnbauer R, Parajka J, Schöber J, Blöschl G. The Kühtai data set: 25 years of lysimetric, snow pillow, and meteorological measurements. Water Resour Res. 2017 Jun;53(6):5158-5165. doi: 10.1002/2017WR020445. Epub 2017 Jun 13. PMID: 28931957; PMCID: PMC5575548.
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Prediction of Drug-Induced Hyperbilirubinemia by In Vitro Testing.

Pharmaceutics

Tátrai P, Krajcsi P.
PMID: 32796590
Pharmaceutics. 2020 Aug 11;12(8). doi: 10.3390/pharmaceutics12080755.

Bilirubin, the end product of heme catabolism, is produced continuously in the body and may reach toxic levels if accumulates in the serum and tissues; therefore, a highly efficient mechanism evolved for its disposition. Normally, unconjugated bilirubin enters hepatocytes...

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Tátrai P, Krajcsi P. Prediction of Drug-Induced Hyperbilirubinemia by In Vitro Testing. Pharmaceutics. 2020;12(8)doi: 10.3390/pharmaceutics12080755.
Tátrai, P., & Krajcsi, P. (2020). Prediction of Drug-Induced Hyperbilirubinemia by In Vitro Testing. Pharmaceutics, 12(8), . https://doi.org/10.3390/pharmaceutics12080755
Tátrai, Péter, and Krajcsi, Péter. "Prediction of Drug-Induced Hyperbilirubinemia by In Vitro Testing." Pharmaceutics vol. 12,8 (2020). doi: https://doi.org/10.3390/pharmaceutics12080755
Tátrai P, Krajcsi P. Prediction of Drug-Induced Hyperbilirubinemia by In Vitro Testing. Pharmaceutics. 2020 Aug 11;12(8). doi: 10.3390/pharmaceutics12080755. PMID: 32796590; PMCID: PMC7465333.
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Showing 1 to 12 of 26 entries