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[Family Hypercholesterolemia and Early Development of Atherosclerosis of the Coronary Arteries in a Patient 34 years].

Kardiologiia

Rozhkova T A, Malyshev P P, Koshechkin V A, Zubareva M Y, Kuharchuk V V.
PMID: 28294851
Kardiologiia. 2015 Mar;55(3):115-120.

No abstract available.

Cite
Rozhkova T A, Malyshev P P, Koshechkin V A, et al. [Family Hypercholesterolemia and Early Development of Atherosclerosis of the Coronary Arteries in a Patient 34 years]. Kardiologiia. 2015;55(3):115-120
Rozhkova T, A., Malyshev P, P., Koshechkin V, A., Zubareva M, Y., & Kuharchuk V, V. (2015). [Family Hypercholesterolemia and Early Development of Atherosclerosis of the Coronary Arteries in a Patient 34 years]. Kardiologiia, 55(3), 115-120.
Rozhkova T, A, et al. "[Family Hypercholesterolemia and Early Development of Atherosclerosis of the Coronary Arteries in a Patient 34 years]." Kardiologiia vol. 55,3 (2015): 115-120.
Rozhkova T A, Malyshev P P, Koshechkin V A, Zubareva M Y, Kuharchuk V V. [Family Hypercholesterolemia and Early Development of Atherosclerosis of the Coronary Arteries in a Patient 34 years]. Kardiologiia. 2015 Mar;55(3):115-120. Russian. PMID: 28294851.
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Information Conversion in Measuring Channels with Optoelectronic Sensors.

Sensors (Basel, Switzerland)

Kukharchuk VV, Pavlov SV, Holodiuk VS, Kryvonosov VE, Skorupski K, Mussabekova A, Karnakova G.
PMID: 35009813
Sensors (Basel). 2021 Dec 30;22(1). doi: 10.3390/s22010271.

The purpose of this work is the authors' attempt to identify the main phases of information transformation in measurement channels on the example of an optical measurement channel with microprocessor control. The authors include such phases: hardware implementation and...

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Kukharchuk VV, Pavlov SV, Holodiuk VS, et al. Information Conversion in Measuring Channels with Optoelectronic Sensors. Sensors (Basel). 2021;22(1)doi: 10.3390/s22010271.
Kukharchuk, V. V., Pavlov, S. V., Holodiuk, V. S., Kryvonosov, V. E., Skorupski, K., Mussabekova, A., & Karnakova, G. (2021). Information Conversion in Measuring Channels with Optoelectronic Sensors. Sensors (Basel, Switzerland), 22(1), . https://doi.org/10.3390/s22010271
Kukharchuk, Vasyl V, et al. "Information Conversion in Measuring Channels with Optoelectronic Sensors." Sensors (Basel, Switzerland) vol. 22,1 (2021). doi: https://doi.org/10.3390/s22010271
Kukharchuk VV, Pavlov SV, Holodiuk VS, Kryvonosov VE, Skorupski K, Mussabekova A, Karnakova G. Information Conversion in Measuring Channels with Optoelectronic Sensors. Sensors (Basel). 2021 Dec 30;22(1). doi: 10.3390/s22010271. PMID: 35009813.
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[The positional specificity of individual triglycerides-substrates for lipase action. The oleic triglycerides of palm oil, palmitic triglycerides of milk, ions of calcium and magnesium.].

Klinicheskaia laboratornaia diagnostika

Titov VN, Aripovsky AV, Samokhodskaya LM, Kotlovsky MV, Yakimenko AV, Kukharchuk VV.
PMID: 31536689
Klin Lab Diagn. 2016;61(12):800-809. doi: 10.18821/0869-2084-2016-61-12-800-809.

The counter-insulin effect of surplus of palmitic fatty acid in food is implemented under: a) formation in vivo of palmitic type of fatty acids metabolism with deficiency of substrate for ATP synthesis and permanent shortage of energy for accomplishment...

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Titov VN, Aripovsky AV, Samokhodskaya LM, et al. [The positional specificity of individual triglycerides-substrates for lipase action. The oleic triglycerides of palm oil, palmitic triglycerides of milk, ions of calcium and magnesium.]. Klin Lab Diagn. 2016;61(12):800-809doi: 10.18821/0869-2084-2016-61-12-800-809.
Titov, V. N., Aripovsky, A. V., Samokhodskaya, L. M., Kotlovsky, M. V., Yakimenko, A. V., & Kukharchuk, V. V. (2016). [The positional specificity of individual triglycerides-substrates for lipase action. The oleic triglycerides of palm oil, palmitic triglycerides of milk, ions of calcium and magnesium.]. Klinicheskaia laboratornaia diagnostika, 61(12), 800-809. https://doi.org/10.18821/0869-2084-2016-61-12-800-809
Titov, V N, et al. "[The positional specificity of individual triglycerides-substrates for lipase action. The oleic triglycerides of palm oil, palmitic triglycerides of milk, ions of calcium and magnesium.]." Klinicheskaia laboratornaia diagnostika vol. 61,12 (2016): 800-809. doi: https://doi.org/10.18821/0869-2084-2016-61-12-800-809
Titov VN, Aripovsky AV, Samokhodskaya LM, Kotlovsky MV, Yakimenko AV, Kukharchuk VV. [The positional specificity of individual triglycerides-substrates for lipase action. The oleic triglycerides of palm oil, palmitic triglycerides of milk, ions of calcium and magnesium.]. Klin Lab Diagn. 2016;61(12):800-809. doi: 10.18821/0869-2084-2016-61-12-800-809. Russian. PMID: 31536689.
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Induced pluripotent stem cell line ICGi036-A generated by reprogramming peripheral blood mononuclear cells from a patient with familial hypercholesterolemia caused due to compound heterozygous p.Ser177Leu/p.Cys352Arg mutations in LDLR.

Stem cell research

Zakharova IS, Shevchenko AI, Tmoyan NA, Elisaphenko EA, Zubkova ES, Sleptcov AA, Nazarenko MS, Ezhov MV, Kukharchuk VV, Parfyonova YV, Zakian SM.
PMID: 34999421
Stem Cell Res. 2022 Jan 03;59:102653. doi: 10.1016/j.scr.2022.102653. Epub 2022 Jan 03.

Familial hypercholesterolemia (FH) is a monogenic disease, leading to atherosclerosis due to a high level of low-density lipoprotein cholesterol. Most cases of the disease are based on pathological variants in the LDLR gene. Hepatocyte-like and endothelial cells derived from...

Cite
Zakharova IS, Shevchenko AI, Tmoyan NA, et al. Induced pluripotent stem cell line ICGi036-A generated by reprogramming peripheral blood mononuclear cells from a patient with familial hypercholesterolemia caused due to compound heterozygous p.Ser177Leu/p.Cys352Arg mutations in LDLR. Stem Cell Res. 2022;59:102653doi: 10.1016/j.scr.2022.102653.
Zakharova, I. S., Shevchenko, A. I., Tmoyan, N. A., Elisaphenko, E. A., Zubkova, E. S., Sleptcov, A. A., Nazarenko, M. S., Ezhov, M. V., Kukharchuk, V. V., Parfyonova, Y. V., & Zakian, S. M. (2022). Induced pluripotent stem cell line ICGi036-A generated by reprogramming peripheral blood mononuclear cells from a patient with familial hypercholesterolemia caused due to compound heterozygous p.Ser177Leu/p.Cys352Arg mutations in LDLR. Stem cell research, 59102653. https://doi.org/10.1016/j.scr.2022.102653
Zakharova, Irina S, et al. "Induced pluripotent stem cell line ICGi036-A generated by reprogramming peripheral blood mononuclear cells from a patient with familial hypercholesterolemia caused due to compound heterozygous p.Ser177Leu/p.Cys352Arg mutations in LDLR." Stem cell research vol. 59 (2022): 102653. doi: https://doi.org/10.1016/j.scr.2022.102653
Zakharova IS, Shevchenko AI, Tmoyan NA, Elisaphenko EA, Zubkova ES, Sleptcov AA, Nazarenko MS, Ezhov MV, Kukharchuk VV, Parfyonova YV, Zakian SM. Induced pluripotent stem cell line ICGi036-A generated by reprogramming peripheral blood mononuclear cells from a patient with familial hypercholesterolemia caused due to compound heterozygous p.Ser177Leu/p.Cys352Arg mutations in LDLR. Stem Cell Res. 2022 Jan 03;59:102653. doi: 10.1016/j.scr.2022.102653. Epub 2022 Jan 03. PMID: 34999421.
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[The individual fatty acids of blood plasma: biological role of substrates, parameters of quantity and quality, diagnostic of atherosclerosis and atheromotosis.].

Klinicheskaia laboratornaia diagnostika

Rozhkova TA, Aripovsky AV, Yarovaya EB, Kaminnaya VI, Kukharchuk VV, Titov VN.
PMID: 30840369
Klin Lab Diagn. 2017;62(11):655-665. doi: 10.18821/0869-2084-2017-62-11-655-665.

The atherosclerosis and atheromotosis are supposed to be, according to phylogenetic theory of general pathology, two etiologically different aphysiological processes, unified by community of pathogenesis. The atherosclerosis is a derangement of biological function of trophology (feeding), biological reaction of...

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Rozhkova TA, Aripovsky AV, Yarovaya EB, et al. [The individual fatty acids of blood plasma: biological role of substrates, parameters of quantity and quality, diagnostic of atherosclerosis and atheromotosis.]. Klin Lab Diagn. 2017;62(11):655-665doi: 10.18821/0869-2084-2017-62-11-655-665.
Rozhkova, T. A., Aripovsky, A. V., Yarovaya, E. B., Kaminnaya, V. I., Kukharchuk, V. V., & Titov, V. N. (2017). [The individual fatty acids of blood plasma: biological role of substrates, parameters of quantity and quality, diagnostic of atherosclerosis and atheromotosis.]. Klinicheskaia laboratornaia diagnostika, 62(11), 655-665. https://doi.org/10.18821/0869-2084-2017-62-11-655-665
Rozhkova, T A, et al. "[The individual fatty acids of blood plasma: biological role of substrates, parameters of quantity and quality, diagnostic of atherosclerosis and atheromotosis.]." Klinicheskaia laboratornaia diagnostika vol. 62,11 (2017): 655-665. doi: https://doi.org/10.18821/0869-2084-2017-62-11-655-665
Rozhkova TA, Aripovsky AV, Yarovaya EB, Kaminnaya VI, Kukharchuk VV, Titov VN. [The individual fatty acids of blood plasma: biological role of substrates, parameters of quantity and quality, diagnostic of atherosclerosis and atheromotosis.]. Klin Lab Diagn. 2017;62(11):655-665. doi: 10.18821/0869-2084-2017-62-11-655-665. Russian. PMID: 30840369.
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Induced pluripotent stem cell line ICGi036-A generated by reprogramming peripheral blood mononuclear cells from a patient with familial hypercholesterolemia caused due to compound heterozygous p.Ser177Leu/p.Cys352Arg mutations in LDLR.

Stem cell research

Zakharova IS, Shevchenko AI, Tmoyan NA, Elisaphenko EA, Zubkova ES, Sleptcov AA, Nazarenko MS, Ezhov MV, Kukharchuk VV, Parfyonova YV, Zakian SM.
PMID: 34999421
Stem Cell Res. 2022 Jan 03;59:102653. doi: 10.1016/j.scr.2022.102653. Epub 2022 Jan 03.

Familial hypercholesterolemia (FH) is a monogenic disease, leading to atherosclerosis due to a high level of low-density lipoprotein cholesterol. Most cases of the disease are based on pathological variants in the LDLR gene. Hepatocyte-like and endothelial cells derived from...

Cite
Zakharova IS, Shevchenko AI, Tmoyan NA, et al. Induced pluripotent stem cell line ICGi036-A generated by reprogramming peripheral blood mononuclear cells from a patient with familial hypercholesterolemia caused due to compound heterozygous p.Ser177Leu/p.Cys352Arg mutations in LDLR. Stem Cell Res. 2022;59:102653doi: 10.1016/j.scr.2022.102653.
Zakharova, I. S., Shevchenko, A. I., Tmoyan, N. A., Elisaphenko, E. A., Zubkova, E. S., Sleptcov, A. A., Nazarenko, M. S., Ezhov, M. V., Kukharchuk, V. V., Parfyonova, Y. V., & Zakian, S. M. (2022). Induced pluripotent stem cell line ICGi036-A generated by reprogramming peripheral blood mononuclear cells from a patient with familial hypercholesterolemia caused due to compound heterozygous p.Ser177Leu/p.Cys352Arg mutations in LDLR. Stem cell research, 59102653. https://doi.org/10.1016/j.scr.2022.102653
Zakharova, Irina S, et al. "Induced pluripotent stem cell line ICGi036-A generated by reprogramming peripheral blood mononuclear cells from a patient with familial hypercholesterolemia caused due to compound heterozygous p.Ser177Leu/p.Cys352Arg mutations in LDLR." Stem cell research vol. 59 (2022): 102653. doi: https://doi.org/10.1016/j.scr.2022.102653
Zakharova IS, Shevchenko AI, Tmoyan NA, Elisaphenko EA, Zubkova ES, Sleptcov AA, Nazarenko MS, Ezhov MV, Kukharchuk VV, Parfyonova YV, Zakian SM. Induced pluripotent stem cell line ICGi036-A generated by reprogramming peripheral blood mononuclear cells from a patient with familial hypercholesterolemia caused due to compound heterozygous p.Ser177Leu/p.Cys352Arg mutations in LDLR. Stem Cell Res. 2022 Jan 03;59:102653. doi: 10.1016/j.scr.2022.102653. Epub 2022 Jan 03. PMID: 34999421.
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[The physical chemical and biological features of triglycerides. The cell absorption of functionally different palmitic+oleic lipoproteins of very low and density and linoleic+linolenic lipoproteins of low density.].

Klinicheskaia laboratornaia diagnostika

Titov VN, Amelyushkina VA, Aripovsky AV, Tibilova OA, Kaba SI, Kukharchuk VV.
PMID: 30821938
Klin Lab Diagn. 2017;62(10):580-592. doi: 10.18821/0869-2084-2017-62-10-580-592.

The earlier insulin-independent low-density lipoproteins and more late insulin-dependent very low-density lipoproteins implement different functions at the stages of phylogenesis. The disorder of biological function of trophology, alteration of fatty acids in triglycerides, prevalence of palmitic very low-density lipoproteins...

Cite
Titov VN, Amelyushkina VA, Aripovsky AV, et al. [The physical chemical and biological features of triglycerides. The cell absorption of functionally different palmitic+oleic lipoproteins of very low and density and linoleic+linolenic lipoproteins of low density.]. Klin Lab Diagn. 2017;62(10):580-592doi: 10.18821/0869-2084-2017-62-10-580-592.
Titov, V. N., Amelyushkina, V. A., Aripovsky, A. V., Tibilova, O. A., Kaba, S. I., & Kukharchuk, V. V. (2017). [The physical chemical and biological features of triglycerides. The cell absorption of functionally different palmitic+oleic lipoproteins of very low and density and linoleic+linolenic lipoproteins of low density.]. Klinicheskaia laboratornaia diagnostika, 62(10), 580-592. https://doi.org/10.18821/0869-2084-2017-62-10-580-592
Titov, V N, et al. "[The physical chemical and biological features of triglycerides. The cell absorption of functionally different palmitic+oleic lipoproteins of very low and density and linoleic+linolenic lipoproteins of low density.]." Klinicheskaia laboratornaia diagnostika vol. 62,10 (2017): 580-592. doi: https://doi.org/10.18821/0869-2084-2017-62-10-580-592
Titov VN, Amelyushkina VA, Aripovsky AV, Tibilova OA, Kaba SI, Kukharchuk VV. [The physical chemical and biological features of triglycerides. The cell absorption of functionally different palmitic+oleic lipoproteins of very low and density and linoleic+linolenic lipoproteins of low density.]. Klin Lab Diagn. 2017;62(10):580-592. doi: 10.18821/0869-2084-2017-62-10-580-592. Russian. PMID: 30821938.
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Induced pluripotent stem cell line ICGi036-A generated by reprogramming peripheral blood mononuclear cells from a patient with familial hypercholesterolemia caused due to compound heterozygous p.Ser177Leu/p.Cys352Arg mutations in LDLR.

Stem cell research

Zakharova IS, Shevchenko AI, Tmoyan NA, Elisaphenko EA, Zubkova ES, Sleptcov AA, Nazarenko MS, Ezhov MV, Kukharchuk VV, Parfyonova YV, Zakian SM.
PMID: 34999421
Stem Cell Res. 2022 Jan 03;59:102653. doi: 10.1016/j.scr.2022.102653. Epub 2022 Jan 03.

Familial hypercholesterolemia (FH) is a monogenic disease, leading to atherosclerosis due to a high level of low-density lipoprotein cholesterol. Most cases of the disease are based on pathological variants in the LDLR gene. Hepatocyte-like and endothelial cells derived from...

Cite
Zakharova IS, Shevchenko AI, Tmoyan NA, et al. Induced pluripotent stem cell line ICGi036-A generated by reprogramming peripheral blood mononuclear cells from a patient with familial hypercholesterolemia caused due to compound heterozygous p.Ser177Leu/p.Cys352Arg mutations in LDLR. Stem Cell Res. 2022;59:102653doi: 10.1016/j.scr.2022.102653.
Zakharova, I. S., Shevchenko, A. I., Tmoyan, N. A., Elisaphenko, E. A., Zubkova, E. S., Sleptcov, A. A., Nazarenko, M. S., Ezhov, M. V., Kukharchuk, V. V., Parfyonova, Y. V., & Zakian, S. M. (2022). Induced pluripotent stem cell line ICGi036-A generated by reprogramming peripheral blood mononuclear cells from a patient with familial hypercholesterolemia caused due to compound heterozygous p.Ser177Leu/p.Cys352Arg mutations in LDLR. Stem cell research, 59102653. https://doi.org/10.1016/j.scr.2022.102653
Zakharova, Irina S, et al. "Induced pluripotent stem cell line ICGi036-A generated by reprogramming peripheral blood mononuclear cells from a patient with familial hypercholesterolemia caused due to compound heterozygous p.Ser177Leu/p.Cys352Arg mutations in LDLR." Stem cell research vol. 59 (2022): 102653. doi: https://doi.org/10.1016/j.scr.2022.102653
Zakharova IS, Shevchenko AI, Tmoyan NA, Elisaphenko EA, Zubkova ES, Sleptcov AA, Nazarenko MS, Ezhov MV, Kukharchuk VV, Parfyonova YV, Zakian SM. Induced pluripotent stem cell line ICGi036-A generated by reprogramming peripheral blood mononuclear cells from a patient with familial hypercholesterolemia caused due to compound heterozygous p.Ser177Leu/p.Cys352Arg mutations in LDLR. Stem Cell Res. 2022 Jan 03;59:102653. doi: 10.1016/j.scr.2022.102653. Epub 2022 Jan 03. PMID: 34999421.
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Carotid atherosclerosis, endothelial disfunction, and arterial stiffness in young and middle-aged men with coronary artery disease.

International journal of vascular medicine

Kozlov S, Balachonova T, Machmudova H, Tripoten M, Andreevskaya M, Rogoza A, Kuharchuk V.
PMID: 22500238
Int J Vasc Med. 2012;2012:950130. doi: 10.1155/2012/950130. Epub 2012 Feb 28.

Background. To assess structural and functional status of the arteries in young and middle-aged men with recently developed CAD. Methods and Results. A total of 78 men aged 28 to 50 years underwent carotid ultrasound, endothelial function, and arterial...

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Kozlov S, Balachonova T, Machmudova H, et al. Carotid atherosclerosis, endothelial disfunction, and arterial stiffness in young and middle-aged men with coronary artery disease. Int J Vasc Med. 2012;2012:950130doi: 10.1155/2012/950130.
Kozlov, S., Balachonova, T., Machmudova, H., Tripoten, M., Andreevskaya, M., Rogoza, A., & Kuharchuk, V. (2012). Carotid atherosclerosis, endothelial disfunction, and arterial stiffness in young and middle-aged men with coronary artery disease. International journal of vascular medicine, 2012950130. https://doi.org/10.1155/2012/950130
Kozlov, Sergey, et al. "Carotid atherosclerosis, endothelial disfunction, and arterial stiffness in young and middle-aged men with coronary artery disease." International journal of vascular medicine vol. 2012 (2012): 950130. doi: https://doi.org/10.1155/2012/950130
Kozlov S, Balachonova T, Machmudova H, Tripoten M, Andreevskaya M, Rogoza A, Kuharchuk V. Carotid atherosclerosis, endothelial disfunction, and arterial stiffness in young and middle-aged men with coronary artery disease. Int J Vasc Med. 2012;2012:950130. doi: 10.1155/2012/950130. Epub 2012 Feb 28. PMID: 22500238; PMCID: PMC3303679.
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[The integrated etiology and separate pathogenesis of atherosclerosis and atheromatosis. The differences of fatty acids transfer in lipoproteins of herbivorous and carnivorous animals.].

Klinicheskaia laboratornaia diagnostika

Titov VN, Kukharchuk VV.
PMID: 31518075
Klin Lab Diagn. 2017;62(4):196-204. doi: 10.18821/0869-2084-2017-62-4-196-204.

According phylogenetic theory of general pathology, overconsumption of meat food by herbivorous animals always results in atherosclerosis and atheromatosis of intima of arteries. The etiological factors of atherosclerosis, atheromatosis in vivo: a) absorption by cells of polyene fatty acids...

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Titov VN, Kukharchuk VV. [The integrated etiology and separate pathogenesis of atherosclerosis and atheromatosis. The differences of fatty acids transfer in lipoproteins of herbivorous and carnivorous animals.]. Klin Lab Diagn. 2017;62(4):196-204doi: 10.18821/0869-2084-2017-62-4-196-204.
Titov, V. N., & Kukharchuk, V. V. (2017). [The integrated etiology and separate pathogenesis of atherosclerosis and atheromatosis. The differences of fatty acids transfer in lipoproteins of herbivorous and carnivorous animals.]. Klinicheskaia laboratornaia diagnostika, 62(4), 196-204. https://doi.org/10.18821/0869-2084-2017-62-4-196-204
Titov, V N, and Kukharchuk, V V. "[The integrated etiology and separate pathogenesis of atherosclerosis and atheromatosis. The differences of fatty acids transfer in lipoproteins of herbivorous and carnivorous animals.]." Klinicheskaia laboratornaia diagnostika vol. 62,4 (2017): 196-204. doi: https://doi.org/10.18821/0869-2084-2017-62-4-196-204
Titov VN, Kukharchuk VV. [The integrated etiology and separate pathogenesis of atherosclerosis and atheromatosis. The differences of fatty acids transfer in lipoproteins of herbivorous and carnivorous animals.]. Klin Lab Diagn. 2017;62(4):196-204. doi: 10.18821/0869-2084-2017-62-4-196-204. Russian. PMID: 31518075.
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[How surplus of palmitic fatty acid in food initiates hypertriglyceridemia, increases cholesterol of low density lipoproteins, triggers atherosclerosis and develops atheromatosis.].

Klinicheskaia laboratornaia diagnostika

Rozhkova TA, Titov VN, Amelyushkina VA, Kuharchuk VV.
PMID: 31505108
Klin Lab Diagn. 2017;62(6):330-338. doi: 10.18821/0869-2084-2017-62-6-330-338.

In phylogenesis, the first transfer of all fatty acids to cells is implemented by high density lipoproteins. Later, unsaturated and polyene fatty acids are transferred to cell by low density lipoproteins. The insulin-depended cells absorb palmitic saturated fatty acid,...

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Rozhkova TA, Titov VN, Amelyushkina VA, et al. [How surplus of palmitic fatty acid in food initiates hypertriglyceridemia, increases cholesterol of low density lipoproteins, triggers atherosclerosis and develops atheromatosis.]. Klin Lab Diagn. 2017;62(6):330-338doi: 10.18821/0869-2084-2017-62-6-330-338.
Rozhkova, T. A., Titov, V. N., Amelyushkina, V. A., & Kuharchuk, V. V. (2017). [How surplus of palmitic fatty acid in food initiates hypertriglyceridemia, increases cholesterol of low density lipoproteins, triggers atherosclerosis and develops atheromatosis.]. Klinicheskaia laboratornaia diagnostika, 62(6), 330-338. https://doi.org/10.18821/0869-2084-2017-62-6-330-338
Rozhkova, T A, et al. "[How surplus of palmitic fatty acid in food initiates hypertriglyceridemia, increases cholesterol of low density lipoproteins, triggers atherosclerosis and develops atheromatosis.]." Klinicheskaia laboratornaia diagnostika vol. 62,6 (2017): 330-338. doi: https://doi.org/10.18821/0869-2084-2017-62-6-330-338
Rozhkova TA, Titov VN, Amelyushkina VA, Kuharchuk VV. [How surplus of palmitic fatty acid in food initiates hypertriglyceridemia, increases cholesterol of low density lipoproteins, triggers atherosclerosis and develops atheromatosis.]. Klin Lab Diagn. 2017;62(6):330-338. doi: 10.18821/0869-2084-2017-62-6-330-338. Russian. PMID: 31505108.
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Induced pluripotent stem cell line ICGi036-A generated by reprogramming peripheral blood mononuclear cells from a patient with familial hypercholesterolemia caused due to compound heterozygous p.Ser177Leu/p.Cys352Arg mutations in LDLR.

Stem cell research

Zakharova IS, Shevchenko AI, Tmoyan NA, Elisaphenko EA, Zubkova ES, Sleptcov AA, Nazarenko MS, Ezhov MV, Kukharchuk VV, Parfyonova YV, Zakian SM.
PMID: 34999421
Stem Cell Res. 2022 Jan 03;59:102653. doi: 10.1016/j.scr.2022.102653. Epub 2022 Jan 03.

Familial hypercholesterolemia (FH) is a monogenic disease, leading to atherosclerosis due to a high level of low-density lipoprotein cholesterol. Most cases of the disease are based on pathological variants in the LDLR gene. Hepatocyte-like and endothelial cells derived from...

Cite
Zakharova IS, Shevchenko AI, Tmoyan NA, et al. Induced pluripotent stem cell line ICGi036-A generated by reprogramming peripheral blood mononuclear cells from a patient with familial hypercholesterolemia caused due to compound heterozygous p.Ser177Leu/p.Cys352Arg mutations in LDLR. Stem Cell Res. 2022;59:102653doi: 10.1016/j.scr.2022.102653.
Zakharova, I. S., Shevchenko, A. I., Tmoyan, N. A., Elisaphenko, E. A., Zubkova, E. S., Sleptcov, A. A., Nazarenko, M. S., Ezhov, M. V., Kukharchuk, V. V., Parfyonova, Y. V., & Zakian, S. M. (2022). Induced pluripotent stem cell line ICGi036-A generated by reprogramming peripheral blood mononuclear cells from a patient with familial hypercholesterolemia caused due to compound heterozygous p.Ser177Leu/p.Cys352Arg mutations in LDLR. Stem cell research, 59102653. https://doi.org/10.1016/j.scr.2022.102653
Zakharova, Irina S, et al. "Induced pluripotent stem cell line ICGi036-A generated by reprogramming peripheral blood mononuclear cells from a patient with familial hypercholesterolemia caused due to compound heterozygous p.Ser177Leu/p.Cys352Arg mutations in LDLR." Stem cell research vol. 59 (2022): 102653. doi: https://doi.org/10.1016/j.scr.2022.102653
Zakharova IS, Shevchenko AI, Tmoyan NA, Elisaphenko EA, Zubkova ES, Sleptcov AA, Nazarenko MS, Ezhov MV, Kukharchuk VV, Parfyonova YV, Zakian SM. Induced pluripotent stem cell line ICGi036-A generated by reprogramming peripheral blood mononuclear cells from a patient with familial hypercholesterolemia caused due to compound heterozygous p.Ser177Leu/p.Cys352Arg mutations in LDLR. Stem Cell Res. 2022 Jan 03;59:102653. doi: 10.1016/j.scr.2022.102653. Epub 2022 Jan 03. PMID: 34999421.
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Showing 1 to 12 of 14 entries