Advanced Search
Display options
Filter resources
Text Availability
Article type
Publication date
Species
Language
Sex
Age
Showing 1 to 12 of 38 entries
Sorted by: Best Match Show Resources per page
High mobility group box 1: a novel mediator of Th2-type response-induced airway inflammation of acute allergic asthma.

Journal of thoracic disease

Ma L, Zeng J, Mo B, Wang C, Huang J, Sun Y, Yu Y, Liu S.
PMID: 26623095
J Thorac Dis. 2015 Oct;7(10):1732-41. doi: 10.3978/j.issn.2072-1439.2015.10.18.

BACKGROUND: High mobility group box 1 (HMGB1) is an inflammatory mediator involved into the advanced stage of systemic inflammatory response syndrome (SIRS), and is over-expressed in bacterial sepsis and hemorrhagic shock. Recently, it has been found that the HMGB1...

Cite
Ma L, Zeng J, Mo B, et al. High mobility group box 1: a novel mediator of Th2-type response-induced airway inflammation of acute allergic asthma. J Thorac Dis. 2015;7(10):1732-41doi: 10.3978/j.issn.2072-1439.2015.10.18.
Ma, L., Zeng, J., Mo, B., Wang, C., Huang, J., Sun, Y., Yu, Y., & Liu, S. (2015). High mobility group box 1: a novel mediator of Th2-type response-induced airway inflammation of acute allergic asthma. Journal of thoracic disease, 7(10), 1732-41. https://doi.org/10.3978/j.issn.2072-1439.2015.10.18
Ma, Libing, et al. "High mobility group box 1: a novel mediator of Th2-type response-induced airway inflammation of acute allergic asthma." Journal of thoracic disease vol. 7,10 (2015): 1732-41. doi: https://doi.org/10.3978/j.issn.2072-1439.2015.10.18
Ma L, Zeng J, Mo B, Wang C, Huang J, Sun Y, Yu Y, Liu S. High mobility group box 1: a novel mediator of Th2-type response-induced airway inflammation of acute allergic asthma. J Thorac Dis. 2015 Oct;7(10):1732-41. doi: 10.3978/j.issn.2072-1439.2015.10.18. PMID: 26623095; PMCID: PMC4635276.
Copy Download .nbib Format: NLM
Observation of muon neutrino disappearance with the MINOS detectors in the NuMI neutrino beam.

Physical review letters

Michael DG, Adamson P, Alexopoulos T, Allison WW, Alner GJ, Anderson K, Andreopoulos C, Andrews M, Andrews R, Arms KE, Armstrong R, Arroyo C, Auty DJ, Avvakumov S, Ayres DS, Baller B, Barish B, Barker MA, Barnes PD, Barr G, Barrett WL, Beall E, Becker BR, Belias A, Bergfeld T, Bernstein RH, Bhattacharya D, Bishai M, Blake A, Bocean V, Bock B, Bock GJ, Boehm J, Boehnlein DJ, Bogert D, Border PM, Bower C, Boyd S, Buckley-Geer E, Bungau C, Byon-Wagner A, Cabrera A, Chapman JD, Chase TR, Cherdack D, Chernichenko SK, Childress S, Choudhary BC, Cobb JH, Cossairt JD, Courant H, Crane DA, Culling AJ, Dawson JW, de Jong JK, DeMuth DM, De Santo A, Dierckxsens M, Diwan MV, Dorman M, Drake G, Drakoulakos D, Ducar R, Durkin T, Erwin AR, Escobar CO, Evans JJ, Fackler OD, Falk Harris E, Feldman GJ, Felt N, Fields TH, Ford R, Frohne MV, Gallagher HR, Gebhard M, Giurgiu GA, Godley A, Gogos J, Goodman MC, Gornushkin Y, Gouffon P, Gran R, Grashorn E, Grossman N, Grudzinski JJ, Grzelak K, Guarino V, Habig A, Halsall R, Hanson J, Harris D, Harris PG, Hartnell J, Hartouni EP, Hatcher R, Heller K, Hill N, Ho Y, Holin A, Howcroft C, Hylen J, Ignatenko M, Indurthy D, Irwin GM, Ishitsuka M, Jaffe DE, James C, Jenner L, Jensen D, Joffe-Minor T, Kafka T, Kang HJ, Kasahara SM, Kilmer J, Kim H, Kim MS, Koizumi G, Kopp S, Kordosky M, Koskinen DJ, Kostin M, Kotelnikov SK, Krakauer DA, Kreymer A, Kumaratunga S, Ladran AS, Lang K, Laughton C, Lebedev A, Lee R, Lee WY, Libkind MA, Ling J, Liu J, Litchfield PJ, Litchfield RP, Longley NP, Lucas P, Luebke W, Madani S, Maher E, Makeev V, Mann WA, Marchionni A, Marino AD, Marshak ML, Marshall JS, Mayer N, McDonald J, McGowan AM, Meier JR, Merzon GI, Messier MD, Milburn RH, Miller JL, Miller WH, Mishra SR, Mislivec A, Miyagawa PS, Moore CD, Morfín J, Morse R, Mualem L, Mufson S, Murgia S, Murtagh MJ, Musser J, Naples D, Nelson C, Nelson JK, Newman HB, Nezrick F, Nichol RJ, Nicholls TC, Ochoa-Ricoux JP, Oliver J, Oliver WP, Onuchin VA, Osiecki T, Ospanov R, Paley J, Paolone V, Para A, Patzak T, Pavlović Z, Pearce GF, Pearson N, Peck CW, Perry C, Peterson EA, Petyt DA, Ping H, Piteira R, Pittam R, Pla-Dalmau A, Plunkett RK, Price LE, Proga M, Pushka DR, Rahman D, Rameika RA, Raufer TM, Read AL, Rebel B, Reichenbacher J, Reyna DE, Rosenfeld C, Rubin HA, Ruddick K, Ryabov VA, Saakyan R, Sanchez MC, Saoulidou N, Schneps J, Schoessow PV, Schreiner P, Schwienhorst R, Semenov VK, Seun SM, Shanahan P, Shield PD, Smart W, Smirnitsky V, Smith C, Smith PN, Sousa A, Speakman B, Stamoulis P, Stefanik A, Sullivan P, Swan JM, Symes PA, Tagg N, Talaga RL, Terekhov A, Tetteh-Lartey E, Thomas J, Thompson J, Thomson MA, Thron JL, Tinti G, Trendler R, Trevor J, Trostin I, Tsarev VA, Tzanakos G, Urheim J, Vahle P, Vakili M, Vaziri K, Velissaris C, Verebryusov V, Viren B, Wai L, Ward CP, Ward DR, Watabe M, Weber A, Webb RC, Wehmann A, West N, White C, White RF, Wojcicki SG, Wright DM, Wu QK, Yan WG, Yang T, Yumiceva FX, Yun JC, Zheng H, Zois M, Zwaska R.
PMID: 17155614
Phys Rev Lett. 2006 Nov 10;97(19):191801. doi: 10.1103/PhysRevLett.97.191801. Epub 2006 Nov 08.

This Letter reports results from the MINOS experiment based on its initial exposure to neutrinos from the Fermilab NuMI beam. The rates and energy spectra of charged current nu(mu) interactions are compared in two detectors located along the beam...

Cite
Michael DG, Adamson P, Alexopoulos T, et al. Observation of muon neutrino disappearance with the MINOS detectors in the NuMI neutrino beam. Phys Rev Lett. 2006;97(19):191801doi: 10.1103/PhysRevLett.97.191801.
Michael, D. G., Adamson, P., Alexopoulos, T., Allison, W. W., Alner, G. J., Anderson, K., Andreopoulos, C., Andrews, M., Andrews, R., Arms, K. E., Armstrong, R., Arroyo, C., Auty, D. J., Avvakumov, S., Ayres, D. S., Baller, B., Barish, B., Barker, M. A., Barnes, P. D., Barr, G., Barrett, W. L., Beall, E., Becker, B. R., Belias, A., Bergfeld, T., Bernstein, R. H., Bhattacharya, D., Bishai, M., Blake, A., Bocean, V., Bock, B., Bock, G. J., Boehm, J., Boehnlein, D. J., Bogert, D., Border, P. M., Bower, C., Boyd, S., Buckley-Geer, E., Bungau, C., Byon-Wagner, A., Cabrera, A., Chapman, J. D., Chase, T. R., Cherdack, D., Chernichenko, S. K., Childress, S., Choudhary, B. C., Cobb, J. H., Cossairt, J. D., Courant, H., Crane, D. A., Culling, A. J., Dawson, J. W., de Jong, J. K., DeMuth, D. M., De Santo, A., Dierckxsens, M., Diwan, M. V., Dorman, M., Drake, G., Drakoulakos, D., Ducar, R., Durkin, T., Erwin, A. R., Escobar, C. O., Evans, J. J., Fackler, O. D., Falk Harris, E., Feldman, G. J., Felt, N., Fields, T. H., Ford, R., Frohne, M. V., Gallagher, H. R., Gebhard, M., Giurgiu, G. A., Godley, A., Gogos, J., Goodman, M. C., Gornushkin, Y., Gouffon, P., Gran, R., Grashorn, E., Grossman, N., Grudzinski, J. J., Grzelak, K., Guarino, V., Habig, A., Halsall, R., Hanson, J., Harris, D., Harris, P. G., Hartnell, J., Hartouni, E. P., Hatcher, R., Heller, K., Hill, N., Ho, Y., Holin, A., Howcroft, C., Hylen, J., Ignatenko, M., Indurthy, D., Irwin, G. M., Ishitsuka, M., Jaffe, D. E., James, C., Jenner, L., Jensen, D., Joffe-Minor, T., Kafka, T., Kang, H. J., Kasahara, S. M., Kilmer, J., Kim, H., Kim, M. S., Koizumi, G., Kopp, S., Kordosky, M., Koskinen, D. J., Kostin, M., Kotelnikov, S. K., Krakauer, D. A., Kreymer, A., Kumaratunga, S., Ladran, A. S., Lang, K., Laughton, C., Lebedev, A., Lee, R., Lee, W. Y., Libkind, M. A., Ling, J., Liu, J., Litchfield, P. J., Litchfield, R. P., Longley, N. P., Lucas, P., Luebke, W., Madani, S., Maher, E., Makeev, V., Mann, W. A., Marchionni, A., Marino, A. D., Marshak, M. L., Marshall, J. S., Mayer, N., McDonald, J., McGowan, A. M., Meier, J. R., Merzon, G. I., Messier, M. D., Milburn, R. H., Miller, J. L., Miller, W. H., Mishra, S. R., Mislivec, A., Miyagawa, P. S., Moore, C. D., Morfín, J., Morse, R., Mualem, L., Mufson, S., Murgia, S., Murtagh, M. J., Musser, J., Naples, D., Nelson, C., Nelson, J. K., Newman, H. B., Nezrick, F., Nichol, R. J., Nicholls, T. C., Ochoa-Ricoux, J. P., Oliver, J., Oliver, W. P., Onuchin, V. A., Osiecki, T., Ospanov, R., Paley, J., Paolone, V., Para, A., Patzak, T., Pavlović, Z., Pearce, G. F., Pearson, N., Peck, C. W., Perry, C., Peterson, E. A., Petyt, D. A., Ping, H., Piteira, R., Pittam, R., Pla-Dalmau, A., Plunkett, R. K., Price, L. E., Proga, M., Pushka, D. R., Rahman, D., Rameika, R. A., Raufer, T. M., Read, A. L., Rebel, B., Reichenbacher, J., Reyna, D. E., Rosenfeld, C., Rubin, H. A., Ruddick, K., Ryabov, V. A., Saakyan, R., Sanchez, M. C., Saoulidou, N., Schneps, J., Schoessow, P. V., Schreiner, P., Schwienhorst, R., Semenov, V. K., Seun, S. M., Shanahan, P., Shield, P. D., Smart, W., Smirnitsky, V., Smith, C., Smith, P. N., Sousa, A., Speakman, B., Stamoulis, P., Stefanik, A., Sullivan, P., Swan, J. M., Symes, P. A., Tagg, N., Talaga, R. L., Terekhov, A., Tetteh-Lartey, E., Thomas, J., Thompson, J., Thomson, M. A., Thron, J. L., Tinti, G., Trendler, R., Trevor, J., Trostin, I., Tsarev, V. A., Tzanakos, G., Urheim, J., Vahle, P., Vakili, M., Vaziri, K., Velissaris, C., Verebryusov, V., Viren, B., Wai, L., Ward, C. P., Ward, D. R., Watabe, M., Weber, A., Webb, R. C., Wehmann, A., West, N., White, C., White, R. F., Wojcicki, S. G., Wright, D. M., Wu, Q. K., Yan, W. G., Yang, T., Yumiceva, F. X., Yun, J. C., Zheng, H., Zois, M., Zwaska, R. (2006). Observation of muon neutrino disappearance with the MINOS detectors in the NuMI neutrino beam. Physical review letters, 97(19), 191801. https://doi.org/10.1103/PhysRevLett.97.191801
Michael, D G, et al. "Observation of muon neutrino disappearance with the MINOS detectors in the NuMI neutrino beam." Physical review letters vol. 97,19 (2006): 191801. doi: https://doi.org/10.1103/PhysRevLett.97.191801
Michael DG, Adamson P, Alexopoulos T, Allison WW, Alner GJ, Anderson K, Andreopoulos C, Andrews M, Andrews R, Arms KE, Armstrong R, Arroyo C, Auty DJ, Avvakumov S, Ayres DS, Baller B, Barish B, Barker MA, Barnes PD, Barr G, Barrett WL, Beall E, Becker BR, Belias A, Bergfeld T, Bernstein RH, Bhattacharya D, Bishai M, Blake A, Bocean V, Bock B, Bock GJ, Boehm J, Boehnlein DJ, Bogert D, Border PM, Bower C, Boyd S, Buckley-Geer E, Bungau C, Byon-Wagner A, Cabrera A, Chapman JD, Chase TR, Cherdack D, Chernichenko SK, Childress S, Choudhary BC, Cobb JH, Cossairt JD, Courant H, Crane DA, Culling AJ, Dawson JW, de Jong JK, DeMuth DM, De Santo A, Dierckxsens M, Diwan MV, Dorman M, Drake G, Drakoulakos D, Ducar R, Durkin T, Erwin AR, Escobar CO, Evans JJ, Fackler OD, Falk Harris E, Feldman GJ, Felt N, Fields TH, Ford R, Frohne MV, Gallagher HR, Gebhard M, Giurgiu GA, Godley A, Gogos J, Goodman MC, Gornushkin Y, Gouffon P, Gran R, Grashorn E, Grossman N, Grudzinski JJ, Grzelak K, Guarino V, Habig A, Halsall R, Hanson J, Harris D, Harris PG, Hartnell J, Hartouni EP, Hatcher R, Heller K, Hill N, Ho Y, Holin A, Howcroft C, Hylen J, Ignatenko M, Indurthy D, Irwin GM, Ishitsuka M, Jaffe DE, James C, Jenner L, Jensen D, Joffe-Minor T, Kafka T, Kang HJ, Kasahara SM, Kilmer J, Kim H, Kim MS, Koizumi G, Kopp S, Kordosky M, Koskinen DJ, Kostin M, Kotelnikov SK, Krakauer DA, Kreymer A, Kumaratunga S, Ladran AS, Lang K, Laughton C, Lebedev A, Lee R, Lee WY, Libkind MA, Ling J, Liu J, Litchfield PJ, Litchfield RP, Longley NP, Lucas P, Luebke W, Madani S, Maher E, Makeev V, Mann WA, Marchionni A, Marino AD, Marshak ML, Marshall JS, Mayer N, McDonald J, McGowan AM, Meier JR, Merzon GI, Messier MD, Milburn RH, Miller JL, Miller WH, Mishra SR, Mislivec A, Miyagawa PS, Moore CD, Morfín J, Morse R, Mualem L, Mufson S, Murgia S, Murtagh MJ, Musser J, Naples D, Nelson C, Nelson JK, Newman HB, Nezrick F, Nichol RJ, Nicholls TC, Ochoa-Ricoux JP, Oliver J, Oliver WP, Onuchin VA, Osiecki T, Ospanov R, Paley J, Paolone V, Para A, Patzak T, Pavlović Z, Pearce GF, Pearson N, Peck CW, Perry C, Peterson EA, Petyt DA, Ping H, Piteira R, Pittam R, Pla-Dalmau A, Plunkett RK, Price LE, Proga M, Pushka DR, Rahman D, Rameika RA, Raufer TM, Read AL, Rebel B, Reichenbacher J, Reyna DE, Rosenfeld C, Rubin HA, Ruddick K, Ryabov VA, Saakyan R, Sanchez MC, Saoulidou N, Schneps J, Schoessow PV, Schreiner P, Schwienhorst R, Semenov VK, Seun SM, Shanahan P, Shield PD, Smart W, Smirnitsky V, Smith C, Smith PN, Sousa A, Speakman B, Stamoulis P, Stefanik A, Sullivan P, Swan JM, Symes PA, Tagg N, Talaga RL, Terekhov A, Tetteh-Lartey E, Thomas J, Thompson J, Thomson MA, Thron JL, Tinti G, Trendler R, Trevor J, Trostin I, Tsarev VA, Tzanakos G, Urheim J, Vahle P, Vakili M, Vaziri K, Velissaris C, Verebryusov V, Viren B, Wai L, Ward CP, Ward DR, Watabe M, Weber A, Webb RC, Wehmann A, West N, White C, White RF, Wojcicki SG, Wright DM, Wu QK, Yan WG, Yang T, Yumiceva FX, Yun JC, Zheng H, Zois M, Zwaska R. Observation of muon neutrino disappearance with the MINOS detectors in the NuMI neutrino beam. Phys Rev Lett. 2006 Nov 10;97(19):191801. doi: 10.1103/PhysRevLett.97.191801. Epub 2006 Nov 08. PMID: 17155614.
Copy Download .nbib Format: NLM
COVID-19 vaccination in Chinese children: a cross-sectional study on the cognition, psychological anxiety state and the willingness toward vaccination.

Human vaccines & immunotherapeutics

Yang J, Zhang T, Qi W, Zhang X, Jia M, Leng Z, Wang Q, Yang Y, Yang W, Ma L, Feng L, Wang C.
PMID: 34324407
Hum Vaccin Immunother. 2021 Jul 29;1-7. doi: 10.1080/21645515.2021.1949950. Epub 2021 Jul 29.

It is important to understand the cognition, willingness, and psychological anxiety state of Chinese guardians toward COVID-19 vaccination for their children to predict the future vaccination rate and to help the design of policies that aim to expand the...

Cite
Yang J, Zhang T, Qi W, et al. COVID-19 vaccination in Chinese children: a cross-sectional study on the cognition, psychological anxiety state and the willingness toward vaccination. Hum Vaccin Immunother. 2021;1-7doi: 10.1080/21645515.2021.1949950.
Yang, J., Zhang, T., Qi, W., Zhang, X., Jia, M., Leng, Z., Wang, Q., Yang, Y., Yang, W., Ma, L., Feng, L., & Wang, C. (2021). COVID-19 vaccination in Chinese children: a cross-sectional study on the cognition, psychological anxiety state and the willingness toward vaccination. Human vaccines & immunotherapeutics, 1-7. https://doi.org/10.1080/21645515.2021.1949950
Yang, Jin, et al. "COVID-19 vaccination in Chinese children: a cross-sectional study on the cognition, psychological anxiety state and the willingness toward vaccination." Human vaccines & immunotherapeutics vol. (2021): 1-7. doi: https://doi.org/10.1080/21645515.2021.1949950
Yang J, Zhang T, Qi W, Zhang X, Jia M, Leng Z, Wang Q, Yang Y, Yang W, Ma L, Feng L, Wang C. COVID-19 vaccination in Chinese children: a cross-sectional study on the cognition, psychological anxiety state and the willingness toward vaccination. Hum Vaccin Immunother. 2021 Jul 29;1-7. doi: 10.1080/21645515.2021.1949950. Epub 2021 Jul 29. PMID: 34324407.
Copy Download .nbib Format: NLM
[Non-conventional yeasts as tools for innovation and differentiation in brewing].

Revista Argentina de microbiologia

Burini JA, Eizaguirre JI, Loviso C, Libkind D.
PMID: 33674169
Rev Argent Microbiol. 2021 Oct-Dec;53(4):359-377. doi: 10.1016/j.ram.2021.01.003. Epub 2021 Mar 03.

Yeasts play a crucial role in brewing. During fermentation, besides ethanol and carbon dioxide, yeasts produce a considerable number of organic compounds, which are essential for beer flavor. In particular, Saccharomyces cerevisiae and Saccharomyces pastorianus are traditionally used in...

Cite
Burini JA, Eizaguirre JI, Loviso C, et al. Levaduras no convencionales como herramientas de innovación y diferenciación en la producción de cerveza. [Non-conventional yeasts as tools for innovation and differentiation in brewing]. Rev Argent Microbiol. 2021;53(4):359-377doi: 10.1016/j.ram.2021.01.003.
Burini, J. A., Eizaguirre, J. I., Loviso, C., & Libkind, D. (2021). Levaduras no convencionales como herramientas de innovación y diferenciación en la producción de cerveza. [Non-conventional yeasts as tools for innovation and differentiation in brewing]. Revista Argentina de microbiologia, 53(4), 359-377. https://doi.org/10.1016/j.ram.2021.01.003
Burini, Julieta Amalia, et al. "Levaduras no convencionales como herramientas de innovación y diferenciación en la producción de cerveza." [Non-conventional yeasts as tools for innovation and differentiation in brewing]. Revista Argentina de microbiologia vol. 53,4 (2021): 359-377. doi: https://doi.org/10.1016/j.ram.2021.01.003
Burini JA, Eizaguirre JI, Loviso C, Libkind D. Levaduras no convencionales como herramientas de innovación y diferenciación en la producción de cerveza. [Non-conventional yeasts as tools for innovation and differentiation in brewing]. Rev Argent Microbiol. 2021 Oct-Dec;53(4):359-377. doi: 10.1016/j.ram.2021.01.003. Epub 2021 Mar 03. Spanish. PMID: 33674169.
Copy Download .nbib Format: NLM
The protective effects of Omarigliptin against Lipopolysaccharide (LPS)- induced inflammatory response and expression of mucin 5AC (MUC5AC) in human bronchial epithelial cells.

Molecular immunology

Ma L, Chang E, Ruan X, Zhang B, Tang F, Zhang J.
PMID: 34871838
Mol Immunol. 2021 Dec 03;141:108-115. doi: 10.1016/j.molimm.2021.11.013. Epub 2021 Dec 03.

The epidemic of chronic inflammatory lung diseases such as asthma, bronchitis, and chronic obstructive pulmonary disease (COPD) has become a global public health problem. Oxidative stress, inflammation, and overproduction of airway mucus play critical roles in the progression of...

Cite
Ma L, Chang E, Ruan X, et al. The protective effects of Omarigliptin against Lipopolysaccharide (LPS)- induced inflammatory response and expression of mucin 5AC (MUC5AC) in human bronchial epithelial cells. Mol Immunol. 2021;141:108-115doi: 10.1016/j.molimm.2021.11.013.
Ma, L., Chang, E., Ruan, X., Zhang, B., Tang, F., & Zhang, J. (2021). The protective effects of Omarigliptin against Lipopolysaccharide (LPS)- induced inflammatory response and expression of mucin 5AC (MUC5AC) in human bronchial epithelial cells. Molecular immunology, 141108-115. https://doi.org/10.1016/j.molimm.2021.11.013
Ma, Libin, et al. "The protective effects of Omarigliptin against Lipopolysaccharide (LPS)- induced inflammatory response and expression of mucin 5AC (MUC5AC) in human bronchial epithelial cells." Molecular immunology vol. 141 (2021): 108-115. doi: https://doi.org/10.1016/j.molimm.2021.11.013
Ma L, Chang E, Ruan X, Zhang B, Tang F, Zhang J. The protective effects of Omarigliptin against Lipopolysaccharide (LPS)- induced inflammatory response and expression of mucin 5AC (MUC5AC) in human bronchial epithelial cells. Mol Immunol. 2021 Dec 03;141:108-115. doi: 10.1016/j.molimm.2021.11.013. Epub 2021 Dec 03. PMID: 34871838.
Copy Download .nbib Format: NLM
The protective effects of Omarigliptin against Lipopolysaccharide (LPS)- induced inflammatory response and expression of mucin 5AC (MUC5AC) in human bronchial epithelial cells.

Molecular immunology

Ma L, Chang E, Ruan X, Zhang B, Tang F, Zhang J.
PMID: 34871838
Mol Immunol. 2021 Dec 03;141:108-115. doi: 10.1016/j.molimm.2021.11.013. Epub 2021 Dec 03.

The epidemic of chronic inflammatory lung diseases such as asthma, bronchitis, and chronic obstructive pulmonary disease (COPD) has become a global public health problem. Oxidative stress, inflammation, and overproduction of airway mucus play critical roles in the progression of...

Cite
Ma L, Chang E, Ruan X, et al. The protective effects of Omarigliptin against Lipopolysaccharide (LPS)- induced inflammatory response and expression of mucin 5AC (MUC5AC) in human bronchial epithelial cells. Mol Immunol. 2021;141:108-115doi: 10.1016/j.molimm.2021.11.013.
Ma, L., Chang, E., Ruan, X., Zhang, B., Tang, F., & Zhang, J. (2021). The protective effects of Omarigliptin against Lipopolysaccharide (LPS)- induced inflammatory response and expression of mucin 5AC (MUC5AC) in human bronchial epithelial cells. Molecular immunology, 141108-115. https://doi.org/10.1016/j.molimm.2021.11.013
Ma, Libin, et al. "The protective effects of Omarigliptin against Lipopolysaccharide (LPS)- induced inflammatory response and expression of mucin 5AC (MUC5AC) in human bronchial epithelial cells." Molecular immunology vol. 141 (2021): 108-115. doi: https://doi.org/10.1016/j.molimm.2021.11.013
Ma L, Chang E, Ruan X, Zhang B, Tang F, Zhang J. The protective effects of Omarigliptin against Lipopolysaccharide (LPS)- induced inflammatory response and expression of mucin 5AC (MUC5AC) in human bronchial epithelial cells. Mol Immunol. 2021 Dec 03;141:108-115. doi: 10.1016/j.molimm.2021.11.013. Epub 2021 Dec 03. PMID: 34871838.
Copy Download .nbib Format: NLM
Osthole Attenuates Macrophage Activation in Experimental Asthma by Inhibitingthe NF-ĸB/MIF Signaling Pathway.

Frontiers in pharmacology

Li R, Song P, Tang G, Wei J, Rao L, Ma L, Jiang M, Huang J, Xu Q, Wu J, Lv Q, Yao D, Xiao B, Huang H, Lei L, Feng J, Mo B.
PMID: 33828480
Front Pharmacol. 2021 Mar 22;12:572463. doi: 10.3389/fphar.2021.572463. eCollection 2021.

Inhibition of activated macrophages is an alternative therapeutic strategy for asthma. We investigated whether a coumarin compound, osthole, isolated from

Cite
Li R, Song P, Tang G, et al. Osthole Attenuates Macrophage Activation in Experimental Asthma by Inhibitingthe NF-ĸB/MIF Signaling Pathway. Front Pharmacol. 2021;12:572463doi: 10.3389/fphar.2021.572463.
Li, R., Song, P., Tang, G., Wei, J., Rao, L., Ma, L., Jiang, M., Huang, J., Xu, Q., Wu, J., Lv, Q., Yao, D., Xiao, B., Huang, H., Lei, L., Feng, J., & Mo, B. (2021). Osthole Attenuates Macrophage Activation in Experimental Asthma by Inhibitingthe NF-ĸB/MIF Signaling Pathway. Frontiers in pharmacology, 12572463. https://doi.org/10.3389/fphar.2021.572463
Li, Ruyi, et al. "Osthole Attenuates Macrophage Activation in Experimental Asthma by Inhibitingthe NF-ĸB/MIF Signaling Pathway." Frontiers in pharmacology vol. 12 (2021): 572463. doi: https://doi.org/10.3389/fphar.2021.572463
Li R, Song P, Tang G, Wei J, Rao L, Ma L, Jiang M, Huang J, Xu Q, Wu J, Lv Q, Yao D, Xiao B, Huang H, Lei L, Feng J, Mo B. Osthole Attenuates Macrophage Activation in Experimental Asthma by Inhibitingthe NF-ĸB/MIF Signaling Pathway. Front Pharmacol. 2021 Mar 22;12:572463. doi: 10.3389/fphar.2021.572463. eCollection 2021. PMID: 33828480; PMCID: PMC8020258.
Copy Download .nbib Format: NLM
The protective effects of Omarigliptin against Lipopolysaccharide (LPS)- induced inflammatory response and expression of mucin 5AC (MUC5AC) in human bronchial epithelial cells.

Molecular immunology

Ma L, Chang E, Ruan X, Zhang B, Tang F, Zhang J.
PMID: 34871838
Mol Immunol. 2021 Dec 03;141:108-115. doi: 10.1016/j.molimm.2021.11.013. Epub 2021 Dec 03.

The epidemic of chronic inflammatory lung diseases such as asthma, bronchitis, and chronic obstructive pulmonary disease (COPD) has become a global public health problem. Oxidative stress, inflammation, and overproduction of airway mucus play critical roles in the progression of...

Cite
Ma L, Chang E, Ruan X, et al. The protective effects of Omarigliptin against Lipopolysaccharide (LPS)- induced inflammatory response and expression of mucin 5AC (MUC5AC) in human bronchial epithelial cells. Mol Immunol. 2021;141:108-115doi: 10.1016/j.molimm.2021.11.013.
Ma, L., Chang, E., Ruan, X., Zhang, B., Tang, F., & Zhang, J. (2021). The protective effects of Omarigliptin against Lipopolysaccharide (LPS)- induced inflammatory response and expression of mucin 5AC (MUC5AC) in human bronchial epithelial cells. Molecular immunology, 141108-115. https://doi.org/10.1016/j.molimm.2021.11.013
Ma, Libin, et al. "The protective effects of Omarigliptin against Lipopolysaccharide (LPS)- induced inflammatory response and expression of mucin 5AC (MUC5AC) in human bronchial epithelial cells." Molecular immunology vol. 141 (2021): 108-115. doi: https://doi.org/10.1016/j.molimm.2021.11.013
Ma L, Chang E, Ruan X, Zhang B, Tang F, Zhang J. The protective effects of Omarigliptin against Lipopolysaccharide (LPS)- induced inflammatory response and expression of mucin 5AC (MUC5AC) in human bronchial epithelial cells. Mol Immunol. 2021 Dec 03;141:108-115. doi: 10.1016/j.molimm.2021.11.013. Epub 2021 Dec 03. PMID: 34871838.
Copy Download .nbib Format: NLM
Emodin inhibits the proliferation and invasion of bladder cancer cells via down-regulating Notch1.

International journal of clinical and experimental pathology

Ma L, Chen K, Jiang K, Deng G, Jiang P, Shao J, Yu Z.
PMID: 31966818
Int J Clin Exp Pathol. 2017 Sep 01;10(9):9452-9459. eCollection 2017.

Emodin exhibits anti-proliferative effects in numerous cancer cell lines via different mechanisms. The present study aimed to study the effects and underlying molecular mechanisms of emodin on bladder cancer cells. We treated two bladder cancer cell lines, T24 and...

Cite
Ma L, Chen K, Jiang K, et al. Emodin inhibits the proliferation and invasion of bladder cancer cells via down-regulating Notch1. Int J Clin Exp Pathol. 2017;10(9):9452-9459
Ma, L., Chen, K., Jiang, K., Deng, G., Jiang, P., Shao, J., & Yu, Z. (2017). Emodin inhibits the proliferation and invasion of bladder cancer cells via down-regulating Notch1. International journal of clinical and experimental pathology, 10(9), 9452-9459.
Ma, Libin, et al. "Emodin inhibits the proliferation and invasion of bladder cancer cells via down-regulating Notch1." International journal of clinical and experimental pathology vol. 10,9 (2017): 9452-9459.
Ma L, Chen K, Jiang K, Deng G, Jiang P, Shao J, Yu Z. Emodin inhibits the proliferation and invasion of bladder cancer cells via down-regulating Notch1. Int J Clin Exp Pathol. 2017 Sep 01;10(9):9452-9459. eCollection 2017. PMID: 31966818; PMCID: PMC6965949.
Copy Download .nbib Format: NLM
The protective effects of Omarigliptin against Lipopolysaccharide (LPS)- induced inflammatory response and expression of mucin 5AC (MUC5AC) in human bronchial epithelial cells.

Molecular immunology

Ma L, Chang E, Ruan X, Zhang B, Tang F, Zhang J.
PMID: 34871838
Mol Immunol. 2021 Dec 03;141:108-115. doi: 10.1016/j.molimm.2021.11.013. Epub 2021 Dec 03.

The epidemic of chronic inflammatory lung diseases such as asthma, bronchitis, and chronic obstructive pulmonary disease (COPD) has become a global public health problem. Oxidative stress, inflammation, and overproduction of airway mucus play critical roles in the progression of...

Cite
Ma L, Chang E, Ruan X, et al. The protective effects of Omarigliptin against Lipopolysaccharide (LPS)- induced inflammatory response and expression of mucin 5AC (MUC5AC) in human bronchial epithelial cells. Mol Immunol. 2021;141:108-115doi: 10.1016/j.molimm.2021.11.013.
Ma, L., Chang, E., Ruan, X., Zhang, B., Tang, F., & Zhang, J. (2021). The protective effects of Omarigliptin against Lipopolysaccharide (LPS)- induced inflammatory response and expression of mucin 5AC (MUC5AC) in human bronchial epithelial cells. Molecular immunology, 141108-115. https://doi.org/10.1016/j.molimm.2021.11.013
Ma, Libin, et al. "The protective effects of Omarigliptin against Lipopolysaccharide (LPS)- induced inflammatory response and expression of mucin 5AC (MUC5AC) in human bronchial epithelial cells." Molecular immunology vol. 141 (2021): 108-115. doi: https://doi.org/10.1016/j.molimm.2021.11.013
Ma L, Chang E, Ruan X, Zhang B, Tang F, Zhang J. The protective effects of Omarigliptin against Lipopolysaccharide (LPS)- induced inflammatory response and expression of mucin 5AC (MUC5AC) in human bronchial epithelial cells. Mol Immunol. 2021 Dec 03;141:108-115. doi: 10.1016/j.molimm.2021.11.013. Epub 2021 Dec 03. PMID: 34871838.
Copy Download .nbib Format: NLM
The protective effects of Omarigliptin against Lipopolysaccharide (LPS)- induced inflammatory response and expression of mucin 5AC (MUC5AC) in human bronchial epithelial cells.

Molecular immunology

Ma L, Chang E, Ruan X, Zhang B, Tang F, Zhang J.
PMID: 34871838
Mol Immunol. 2021 Dec 03;141:108-115. doi: 10.1016/j.molimm.2021.11.013. Epub 2021 Dec 03.

The epidemic of chronic inflammatory lung diseases such as asthma, bronchitis, and chronic obstructive pulmonary disease (COPD) has become a global public health problem. Oxidative stress, inflammation, and overproduction of airway mucus play critical roles in the progression of...

Cite
Ma L, Chang E, Ruan X, et al. The protective effects of Omarigliptin against Lipopolysaccharide (LPS)- induced inflammatory response and expression of mucin 5AC (MUC5AC) in human bronchial epithelial cells. Mol Immunol. 2021;141:108-115doi: 10.1016/j.molimm.2021.11.013.
Ma, L., Chang, E., Ruan, X., Zhang, B., Tang, F., & Zhang, J. (2021). The protective effects of Omarigliptin against Lipopolysaccharide (LPS)- induced inflammatory response and expression of mucin 5AC (MUC5AC) in human bronchial epithelial cells. Molecular immunology, 141108-115. https://doi.org/10.1016/j.molimm.2021.11.013
Ma, Libin, et al. "The protective effects of Omarigliptin against Lipopolysaccharide (LPS)- induced inflammatory response and expression of mucin 5AC (MUC5AC) in human bronchial epithelial cells." Molecular immunology vol. 141 (2021): 108-115. doi: https://doi.org/10.1016/j.molimm.2021.11.013
Ma L, Chang E, Ruan X, Zhang B, Tang F, Zhang J. The protective effects of Omarigliptin against Lipopolysaccharide (LPS)- induced inflammatory response and expression of mucin 5AC (MUC5AC) in human bronchial epithelial cells. Mol Immunol. 2021 Dec 03;141:108-115. doi: 10.1016/j.molimm.2021.11.013. Epub 2021 Dec 03. PMID: 34871838.
Copy Download .nbib Format: NLM
C-EBPβ mediates in cigarette/IL-17A-induced bronchial epithelial-mesenchymal transition in COPD mice.

BMC pulmonary medicine

Chu S, Ma L, Wu Y, Zhao X, Xiao B, Pan Q.
PMID: 34794427
BMC Pulm Med. 2021 Nov 18;21(1):376. doi: 10.1186/s12890-021-01738-6.

BACKGROUND: Cigarettes smoking and IL-17A contribute to chronic obstructive pulmonary disease (COPD), and have synergistical effect on bronchial epithelial cell proliferation. CCAAT/enhancer-binding protein β (C-EBPβ) could be induced by IL-17A and is up-regulated in COPD. We explored the effect...

Cite
Chu S, Ma L, Wu Y, et al. C-EBPβ mediates in cigarette/IL-17A-induced bronchial epithelial-mesenchymal transition in COPD mice. BMC Pulm Med. 2021;21(1):376doi: 10.1186/s12890-021-01738-6.
Chu, S., Ma, L., Wu, Y., Zhao, X., Xiao, B., & Pan, Q. (2021). C-EBPβ mediates in cigarette/IL-17A-induced bronchial epithelial-mesenchymal transition in COPD mice. BMC pulmonary medicine, 21(1), 376. https://doi.org/10.1186/s12890-021-01738-6
Chu, Shuyuan, et al. "C-EBPβ mediates in cigarette/IL-17A-induced bronchial epithelial-mesenchymal transition in COPD mice." BMC pulmonary medicine vol. 21,1 (2021): 376. doi: https://doi.org/10.1186/s12890-021-01738-6
Chu S, Ma L, Wu Y, Zhao X, Xiao B, Pan Q. C-EBPβ mediates in cigarette/IL-17A-induced bronchial epithelial-mesenchymal transition in COPD mice. BMC Pulm Med. 2021 Nov 18;21(1):376. doi: 10.1186/s12890-021-01738-6. PMID: 34794427; PMCID: PMC8603568.
Copy Download .nbib Format: NLM
Showing 1 to 12 of 38 entries