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Showing 1 to 12 of 17 entries
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Craniofacial resection for cancer.

The Western journal of medicine

Makielski K.
PMID: 18750570
West J Med. 1989 Apr;150(4):454.

No abstract available.

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Makielski K. Craniofacial resection for cancer. West J Med. 1989;150(4):454
Makielski, K. (1989). Craniofacial resection for cancer. The Western journal of medicine, 150(4), 454.
Makielski, K. "Craniofacial resection for cancer." The Western journal of medicine vol. 150,4 (1989): 454.
Makielski K. Craniofacial resection for cancer. West J Med. 1989 Apr;150(4):454. PMID: 18750570; PMCID: PMC1026589.
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Expression defect of the rare variant/Brugada mutation R1512W depends upon the SCN5A splice variant background and can be rescued by mexiletine and the common polymorphism H558R.

Channels (Austin, Tex.)

Hu RM, Song EJ, Tester DJ, Deschenes I, Ackerman MJ, Makielski JC, Tan BH.
PMID: 33535892
Channels (Austin). 2021 Dec;15(1):253-261. doi: 10.1080/19336950.2021.1875645.

No abstract available.

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Hu RM, Song EJ, Tester DJ, et al. Expression defect of the rare variant/Brugada mutation R1512W depends upon the SCN5A splice variant background and can be rescued by mexiletine and the common polymorphism H558R. Channels (Austin). 2021;15(1):253-261doi: 10.1080/19336950.2021.1875645.
Hu, R. M., Song, E. J., Tester, D. J., Deschenes, I., Ackerman, M. J., Makielski, J. C., & Tan, B. H. (2021). Expression defect of the rare variant/Brugada mutation R1512W depends upon the SCN5A splice variant background and can be rescued by mexiletine and the common polymorphism H558R. Channels (Austin, Tex.), 15(1), 253-261. https://doi.org/10.1080/19336950.2021.1875645
Hu, Rou-Mu, et al. "Expression defect of the rare variant/Brugada mutation R1512W depends upon the SCN5A splice variant background and can be rescued by mexiletine and the common polymorphism H558R." Channels (Austin, Tex.) vol. 15,1 (2021): 253-261. doi: https://doi.org/10.1080/19336950.2021.1875645
Hu RM, Song EJ, Tester DJ, Deschenes I, Ackerman MJ, Makielski JC, Tan BH. Expression defect of the rare variant/Brugada mutation R1512W depends upon the SCN5A splice variant background and can be rescued by mexiletine and the common polymorphism H558R. Channels (Austin). 2021 Dec;15(1):253-261. doi: 10.1080/19336950.2021.1875645. PMID: 33535892; PMCID: PMC7872018.
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Deletion of Sulfonylurea Receptor 2 in the Adult Myocardium Enhances Cardiac Glucose Uptake and Is Cardioprotective.

JACC. Basic to translational science

Aubert G, Barefield DY, Demonbreun AR, Ramratnam M, Fallon KS, Warner JL, Rossi AE, Hadhazy M, Makielski JC, McNally EM.
PMID: 31061927
JACC Basic Transl Sci. 2019 Apr 29;4(2):251-268. doi: 10.1016/j.jacbts.2018.11.012. eCollection 2019 Apr.

The adult myocardium relies on oxidative metabolism. In ischemic myocardium, such as the embryonic heart, glycolysis contributes more prominently as a fuel source. The sulfonylurea receptor 2 (SUR2) was previously implicated in the normal myocardial transition from glycolytic to...

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Aubert G, Barefield DY, Demonbreun AR, et al. Deletion of Sulfonylurea Receptor 2 in the Adult Myocardium Enhances Cardiac Glucose Uptake and Is Cardioprotective. JACC Basic Transl Sci. 2019;4(2):251-268doi: 10.1016/j.jacbts.2018.11.012.
Aubert, G., Barefield, D. Y., Demonbreun, A. R., Ramratnam, M., Fallon, K. S., Warner, J. L., Rossi, A. E., Hadhazy, M., Makielski, J. C., & McNally, E. M. (2019). Deletion of Sulfonylurea Receptor 2 in the Adult Myocardium Enhances Cardiac Glucose Uptake and Is Cardioprotective. JACC. Basic to translational science, 4(2), 251-268. https://doi.org/10.1016/j.jacbts.2018.11.012
Aubert, Gregory, et al. "Deletion of Sulfonylurea Receptor 2 in the Adult Myocardium Enhances Cardiac Glucose Uptake and Is Cardioprotective." JACC. Basic to translational science vol. 4,2 (2019): 251-268. doi: https://doi.org/10.1016/j.jacbts.2018.11.012
Aubert G, Barefield DY, Demonbreun AR, Ramratnam M, Fallon KS, Warner JL, Rossi AE, Hadhazy M, Makielski JC, McNally EM. Deletion of Sulfonylurea Receptor 2 in the Adult Myocardium Enhances Cardiac Glucose Uptake and Is Cardioprotective. JACC Basic Transl Sci. 2019 Apr 29;4(2):251-268. doi: 10.1016/j.jacbts.2018.11.012. eCollection 2019 Apr. PMID: 31061927; PMCID: PMC6488756.
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A method to generate the surface cell layer of the 3D virtual shoot apex from apical initials.

Plant methods

Kucypera K, Lipowczan M, Piekarska-Stachowiak A, Nakielski J.
PMID: 29238397
Plant Methods. 2017 Dec 11;13:110. doi: 10.1186/s13007-017-0262-7. eCollection 2017.

BACKGROUND: The development of cell pattern in the surface cell layer of the shoot apex can be investigated in vivo by use of a time-lapse confocal images, showing naked meristem in 3D in successive times. However, how this layer...

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Kucypera K, Lipowczan M, Piekarska-Stachowiak A, et al. A method to generate the surface cell layer of the 3D virtual shoot apex from apical initials. Plant Methods. 2017;13:110doi: 10.1186/s13007-017-0262-7.
Kucypera, K., Lipowczan, M., Piekarska-Stachowiak, A., & Nakielski, J. (2017). A method to generate the surface cell layer of the 3D virtual shoot apex from apical initials. Plant methods, 13110. https://doi.org/10.1186/s13007-017-0262-7
Kucypera, Krzysztof, et al. "A method to generate the surface cell layer of the 3D virtual shoot apex from apical initials." Plant methods vol. 13 (2017): 110. doi: https://doi.org/10.1186/s13007-017-0262-7
Kucypera K, Lipowczan M, Piekarska-Stachowiak A, Nakielski J. A method to generate the surface cell layer of the 3D virtual shoot apex from apical initials. Plant Methods. 2017 Dec 11;13:110. doi: 10.1186/s13007-017-0262-7. eCollection 2017. PMID: 29238397; PMCID: PMC5725887.
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Isolation of Cancer-Derived Exosomes Using a Variety of Magnetic Nanostructures: From Fe.

Nanomaterials (Basel, Switzerland)

Nemati Z, Zamani Kouhpanji MR, Zhou F, Das R, Makielski K, Um J, Phan MH, Muela A, Fdez-Gubieda ML, Franklin RR, Stadler BJH, Modiano JF, Alonso J.
PMID: 32854239
Nanomaterials (Basel). 2020 Aug 25;10(9). doi: 10.3390/nano10091662.

Isolating and analyzing tumor-derived exosomes (TEX) can provide important information about the state of a tumor, facilitating early diagnosis and prognosis. Since current isolation methods are mostly laborious and expensive, we propose herein a fast and cost-effective method based...

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Nemati Z, Zamani Kouhpanji MR, Zhou F, et al. Isolation of Cancer-Derived Exosomes Using a Variety of Magnetic Nanostructures: From Fe. Nanomaterials (Basel). 2020;10(9)doi: 10.3390/nano10091662.
Nemati, Z., Zamani Kouhpanji, M. R., Zhou, F., Das, R., Makielski, K., Um, J., Phan, M. H., Muela, A., Fdez-Gubieda, M. L., Franklin, R. R., Stadler, B. J. H., Modiano, J. F., & Alonso, J. (2020). Isolation of Cancer-Derived Exosomes Using a Variety of Magnetic Nanostructures: From Fe. Nanomaterials (Basel, Switzerland), 10(9), . https://doi.org/10.3390/nano10091662
Nemati, Zohreh, et al. "Isolation of Cancer-Derived Exosomes Using a Variety of Magnetic Nanostructures: From Fe." Nanomaterials (Basel, Switzerland) vol. 10,9 (2020). doi: https://doi.org/10.3390/nano10091662
Nemati Z, Zamani Kouhpanji MR, Zhou F, Das R, Makielski K, Um J, Phan MH, Muela A, Fdez-Gubieda ML, Franklin RR, Stadler BJH, Modiano JF, Alonso J. Isolation of Cancer-Derived Exosomes Using a Variety of Magnetic Nanostructures: From Fe. Nanomaterials (Basel). 2020 Aug 25;10(9). doi: 10.3390/nano10091662. PMID: 32854239; PMCID: PMC7558559.
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LQTS-associated mutation A257G in α1-syntrophin interacts with the intragenic variant P74L to modify its biophysical phenotype.

Cardiogenetics

Cheng J, Norstrand DW, Medeiros-Domingo A, Tester DJ, Valdivia CR, Tan BH, Vatta M, Makielski JC, Ackerman MJ.
PMID: 24319568
Cardiogenetics. 2011 Oct 25;1(1). doi: 10.4081/cardiogenetics.2011.e13.

The

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Cheng J, Norstrand DW, Medeiros-Domingo A, et al. LQTS-associated mutation A257G in α1-syntrophin interacts with the intragenic variant P74L to modify its biophysical phenotype. Cardiogenetics. 2011;1(1)doi: 10.4081/cardiogenetics.2011.e13.
Cheng, J., Norstrand, D. W., Medeiros-Domingo, A., Tester, D. J., Valdivia, C. R., Tan, B. H., Vatta, M., Makielski, J. C., & Ackerman, M. J. (2011). LQTS-associated mutation A257G in α1-syntrophin interacts with the intragenic variant P74L to modify its biophysical phenotype. Cardiogenetics, 1(1), . https://doi.org/10.4081/cardiogenetics.2011.e13
Cheng, Jianding, et al. "LQTS-associated mutation A257G in α1-syntrophin interacts with the intragenic variant P74L to modify its biophysical phenotype." Cardiogenetics vol. 1,1 (2011). doi: https://doi.org/10.4081/cardiogenetics.2011.e13
Cheng J, Norstrand DW, Medeiros-Domingo A, Tester DJ, Valdivia CR, Tan BH, Vatta M, Makielski JC, Ackerman MJ. LQTS-associated mutation A257G in α1-syntrophin interacts with the intragenic variant P74L to modify its biophysical phenotype. Cardiogenetics. 2011 Oct 25;1(1). doi: 10.4081/cardiogenetics.2011.e13. PMID: 24319568; PMCID: PMC3852898.
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ΔNp63α promotes Epstein-Barr virus latency in undifferentiated epithelial cells.

PLoS pathogens

Van Sciver N, Ohashi M, Nawandar DM, Pauly NP, Lee D, Makielski KR, Bristol JA, Tsao SW, Lambert PF, Johannsen EC, Kenney SC.
PMID: 34748616
PLoS Pathog. 2021 Nov 08;17(11):e1010045. doi: 10.1371/journal.ppat.1010045. eCollection 2021 Nov.

Epstein-Barr virus (EBV) is a human herpesvirus that causes infectious mononucleosis and contributes to both B-cell and epithelial-cell malignancies. EBV-infected epithelial cell tumors, including nasopharyngeal carcinoma (NPC), are largely composed of latently infected cells, but the mechanism(s) maintaining viral...

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Van Sciver N, Ohashi M, Nawandar DM, et al. ΔNp63α promotes Epstein-Barr virus latency in undifferentiated epithelial cells. PLoS Pathog. 2021;17(11):e1010045doi: 10.1371/journal.ppat.1010045.
Van Sciver, N., Ohashi, M., Nawandar, D. M., Pauly, N. P., Lee, D., Makielski, K. R., Bristol, J. A., Tsao, S. W., Lambert, P. F., Johannsen, E. C., & Kenney, S. C. (2021). ΔNp63α promotes Epstein-Barr virus latency in undifferentiated epithelial cells. PLoS pathogens, 17(11), e1010045. https://doi.org/10.1371/journal.ppat.1010045
Van Sciver, Nicholas, et al. "ΔNp63α promotes Epstein-Barr virus latency in undifferentiated epithelial cells." PLoS pathogens vol. 17,11 (2021): e1010045. doi: https://doi.org/10.1371/journal.ppat.1010045
Van Sciver N, Ohashi M, Nawandar DM, Pauly NP, Lee D, Makielski KR, Bristol JA, Tsao SW, Lambert PF, Johannsen EC, Kenney SC. ΔNp63α promotes Epstein-Barr virus latency in undifferentiated epithelial cells. PLoS Pathog. 2021 Nov 08;17(11):e1010045. doi: 10.1371/journal.ppat.1010045. eCollection 2021 Nov. PMID: 34748616; PMCID: PMC8601603.
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Development of an exosomal gene signature to detect residual disease in dogs with osteosarcoma using a novel xenograft platform and machine learning.

Laboratory investigation; a journal of technical methods and pathology

Makielski KM, Donnelly AJ, Khammanivong A, Scott MC, Ortiz AR, Galvan DC, Tomiyasu H, Amaya C, Ward KA, Montoya A, Garbe JR, Mills LJ, Cutter GR, Fenger JM, Kisseberth WC, O'Brien TD, Weigel BJ, Spector LG, Bryan BA, Subramanian S, Modiano JF.
PMID: 34489559
Lab Invest. 2021 Dec;101(12):1585-1596. doi: 10.1038/s41374-021-00655-w. Epub 2021 Sep 06.

Osteosarcoma has a guarded prognosis. A major hurdle in developing more effective osteosarcoma therapies is the lack of disease-specific biomarkers to predict risk, prognosis, or therapeutic response. Exosomes are secreted extracellular microvesicles emerging as powerful diagnostic tools. However, their...

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Makielski KM, Donnelly AJ, Khammanivong A, et al. Development of an exosomal gene signature to detect residual disease in dogs with osteosarcoma using a novel xenograft platform and machine learning. Lab Invest. 2021;101(12):1585-1596doi: 10.1038/s41374-021-00655-w.
Makielski, K. M., Donnelly, A. J., Khammanivong, A., Scott, M. C., Ortiz, A. R., Galvan, D. C., Tomiyasu, H., Amaya, C., Ward, K. A., Montoya, A., Garbe, J. R., Mills, L. J., Cutter, G. R., Fenger, J. M., Kisseberth, W. C., O'Brien, T. D., Weigel, B. J., Spector, L. G., Bryan, B. A., Subramanian, S., & Modiano, J. F. (2021). Development of an exosomal gene signature to detect residual disease in dogs with osteosarcoma using a novel xenograft platform and machine learning. Laboratory investigation; a journal of technical methods and pathology, 101(12), 1585-1596. https://doi.org/10.1038/s41374-021-00655-w
Makielski, Kelly M, et al. "Development of an exosomal gene signature to detect residual disease in dogs with osteosarcoma using a novel xenograft platform and machine learning." Laboratory investigation; a journal of technical methods and pathology vol. 101,12 (2021): 1585-1596. doi: https://doi.org/10.1038/s41374-021-00655-w
Makielski KM, Donnelly AJ, Khammanivong A, Scott MC, Ortiz AR, Galvan DC, Tomiyasu H, Amaya C, Ward KA, Montoya A, Garbe JR, Mills LJ, Cutter GR, Fenger JM, Kisseberth WC, O'Brien TD, Weigel BJ, Spector LG, Bryan BA, Subramanian S, Modiano JF. Development of an exosomal gene signature to detect residual disease in dogs with osteosarcoma using a novel xenograft platform and machine learning. Lab Invest. 2021 Dec;101(12):1585-1596. doi: 10.1038/s41374-021-00655-w. Epub 2021 Sep 06. PMID: 34489559.
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Creating tomorrow's leaders and innovators through an RN-to-bachelor of science in nursing consortium curricular model.

Journal of professional nursing : official journal of the American Association of Colleges of Nursing

Hendricks SM, Phillips JM, Narwold L, Laux M, Rouse S, Dulemba L, Makielski M, Halstead JA.
PMID: 22640948
J Prof Nurs. 2012 May-Jun;28(3):163-9. doi: 10.1016/j.profnurs.2011.11.009.

The critical need to increase the number of baccalaureate-prepared RNs to improve the safety and quality of patient care in today's complex health care system is a pressing issue in health care. One part of the solution lies in...

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Hendricks SM, Phillips JM, Narwold L, et al. Creating tomorrow's leaders and innovators through an RN-to-bachelor of science in nursing consortium curricular model. J Prof Nurs. 2012;28(3):163-9doi: 10.1016/j.profnurs.2011.11.009.
Hendricks, S. M., Phillips, J. M., Narwold, L., Laux, M., Rouse, S., Dulemba, L., Makielski, M., & Halstead, J. A. (2012). Creating tomorrow's leaders and innovators through an RN-to-bachelor of science in nursing consortium curricular model. Journal of professional nursing : official journal of the American Association of Colleges of Nursing, 28(3), 163-9. https://doi.org/10.1016/j.profnurs.2011.11.009
Hendricks, Susan M, et al. "Creating tomorrow's leaders and innovators through an RN-to-bachelor of science in nursing consortium curricular model." Journal of professional nursing : official journal of the American Association of Colleges of Nursing vol. 28,3 (2012): 163-9. doi: https://doi.org/10.1016/j.profnurs.2011.11.009
Hendricks SM, Phillips JM, Narwold L, Laux M, Rouse S, Dulemba L, Makielski M, Halstead JA. Creating tomorrow's leaders and innovators through an RN-to-bachelor of science in nursing consortium curricular model. J Prof Nurs. 2012 May-Jun;28(3):163-9. doi: 10.1016/j.profnurs.2011.11.009. PMID: 22640948.
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Does Anatomic Phenotype of Mitral Annular Disjunction Impact Survival? An Autopsy-Based Retrospective Study.

Journal of cardiovascular development and disease

Zhou N, Zhao Q, Li R, Zheng D, Xiao Y, Mao D, Wang Y, Yue J, Zhang K, Makielski JC, Cheng J.
PMID: 34940529
J Cardiovasc Dev Dis. 2021 Dec 05;8(12). doi: 10.3390/jcdd8120174.

Controversies have been raised regarding the prevalence and potential clinical significance of mitral annular disjunction (MAD). We aim to address the anatomic characteristics of MAD and their association, if any, on survival. We retrospectively reviewed 1373 consecutive dissected hearts...

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Zhou N, Zhao Q, Li R, et al. Does Anatomic Phenotype of Mitral Annular Disjunction Impact Survival? An Autopsy-Based Retrospective Study. J Cardiovasc Dev Dis. 2021;8(12)doi: 10.3390/jcdd8120174.
Zhou, N., Zhao, Q., Li, R., Zheng, D., Xiao, Y., Mao, D., Wang, Y., Yue, J., Zhang, K., Makielski, J. C., & Cheng, J. (2021). Does Anatomic Phenotype of Mitral Annular Disjunction Impact Survival? An Autopsy-Based Retrospective Study. Journal of cardiovascular development and disease, 8(12), . https://doi.org/10.3390/jcdd8120174
Zhou, Nan, et al. "Does Anatomic Phenotype of Mitral Annular Disjunction Impact Survival? An Autopsy-Based Retrospective Study." Journal of cardiovascular development and disease vol. 8,12 (2021). doi: https://doi.org/10.3390/jcdd8120174
Zhou N, Zhao Q, Li R, Zheng D, Xiao Y, Mao D, Wang Y, Yue J, Zhang K, Makielski JC, Cheng J. Does Anatomic Phenotype of Mitral Annular Disjunction Impact Survival? An Autopsy-Based Retrospective Study. J Cardiovasc Dev Dis. 2021 Dec 05;8(12). doi: 10.3390/jcdd8120174. PMID: 34940529.
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Does Anatomic Phenotype of Mitral Annular Disjunction Impact Survival? An Autopsy-Based Retrospective Study.

Journal of cardiovascular development and disease

Zhou N, Zhao Q, Li R, Zheng D, Xiao Y, Mao D, Wang Y, Yue J, Zhang K, Makielski JC, Cheng J.
PMID: 34940529
J Cardiovasc Dev Dis. 2021 Dec 05;8(12). doi: 10.3390/jcdd8120174.

Controversies have been raised regarding the prevalence and potential clinical significance of mitral annular disjunction (MAD). We aim to address the anatomic characteristics of MAD and their association, if any, on survival. We retrospectively reviewed 1373 consecutive dissected hearts...

Cite
Zhou N, Zhao Q, Li R, et al. Does Anatomic Phenotype of Mitral Annular Disjunction Impact Survival? An Autopsy-Based Retrospective Study. J Cardiovasc Dev Dis. 2021;8(12)doi: 10.3390/jcdd8120174.
Zhou, N., Zhao, Q., Li, R., Zheng, D., Xiao, Y., Mao, D., Wang, Y., Yue, J., Zhang, K., Makielski, J. C., & Cheng, J. (2021). Does Anatomic Phenotype of Mitral Annular Disjunction Impact Survival? An Autopsy-Based Retrospective Study. Journal of cardiovascular development and disease, 8(12), . https://doi.org/10.3390/jcdd8120174
Zhou, Nan, et al. "Does Anatomic Phenotype of Mitral Annular Disjunction Impact Survival? An Autopsy-Based Retrospective Study." Journal of cardiovascular development and disease vol. 8,12 (2021). doi: https://doi.org/10.3390/jcdd8120174
Zhou N, Zhao Q, Li R, Zheng D, Xiao Y, Mao D, Wang Y, Yue J, Zhang K, Makielski JC, Cheng J. Does Anatomic Phenotype of Mitral Annular Disjunction Impact Survival? An Autopsy-Based Retrospective Study. J Cardiovasc Dev Dis. 2021 Dec 05;8(12). doi: 10.3390/jcdd8120174. PMID: 34940529.
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Transgenic overexpression of the SUR2A-55 splice variant in mouse heart reduces infract size and promotes protective mitochondrial function.

Heliyon

Ramratnam M, Kenny B, Kyle JW, Wiedmeyer B, Hacker TA, Barefield DY, McNally EM, Makielski JC.
PMID: 29998196
Heliyon. 2018 Jul 04;4(7):e00677. doi: 10.1016/j.heliyon.2018.e00677. eCollection 2018 Jul.

ATP-sensitive potassium channels found in both the sarcolemma (sarcK

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Ramratnam M, Kenny B, Kyle JW, et al. Transgenic overexpression of the SUR2A-55 splice variant in mouse heart reduces infract size and promotes protective mitochondrial function. Heliyon. 2018;4(7):e00677doi: 10.1016/j.heliyon.2018.e00677.
Ramratnam, M., Kenny, B., Kyle, J. W., Wiedmeyer, B., Hacker, T. A., Barefield, D. Y., McNally, E. M., & Makielski, J. C. (2018). Transgenic overexpression of the SUR2A-55 splice variant in mouse heart reduces infract size and promotes protective mitochondrial function. Heliyon, 4(7), e00677. https://doi.org/10.1016/j.heliyon.2018.e00677
Ramratnam, Mohun, et al. "Transgenic overexpression of the SUR2A-55 splice variant in mouse heart reduces infract size and promotes protective mitochondrial function." Heliyon vol. 4,7 (2018): e00677. doi: https://doi.org/10.1016/j.heliyon.2018.e00677
Ramratnam M, Kenny B, Kyle JW, Wiedmeyer B, Hacker TA, Barefield DY, McNally EM, Makielski JC. Transgenic overexpression of the SUR2A-55 splice variant in mouse heart reduces infract size and promotes protective mitochondrial function. Heliyon. 2018 Jul 04;4(7):e00677. doi: 10.1016/j.heliyon.2018.e00677. eCollection 2018 Jul. PMID: 29998196; PMCID: PMC6037880.
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